---
_id: '724'
abstract:
- lang: eng
text: We investigate the stationary and dynamical behavior of an Anderson localized
chain coupled to a single central bound state. Although this coupling partially
dilutes the Anderson localized peaks towards nearly resonant sites, the most weight
of the original peaks remains unchanged. This leads to multifractal wave functions
with a frozen spectrum of fractal dimensions, which is characteristic for localized
phases in models with power-law hopping. Using a perturbative approach we identify
two different dynamical regimes. At weak couplings to the central site, the transport
of particles and information is logarithmic in time, a feature usually attributed
to many-body localization. We connect such transport to the persistence of the
Poisson statistics of level spacings in parts of the spectrum. In contrast, at
stronger couplings the level repulsion is established in the entire spectrum,
the problem can be mapped to the Fano resonance, and the transport is ballistic.
acknowledgement: "We would like to thank Dmitry Abanin, Christophe De\r\nBeule,
\ Joel Moore, Romain Vasseur, and Norman Yao for\r\nmany stimulating discussions.
\ Financial support has been\r\nprovided by the Deutsche Forschungsgemeinschaft
\ (DFG)\r\nvia Grant No. TR950/8-1, SFB 1170 “ToCoTronics” and the\r\nENB Graduate
\ School on Topological Insulators. M.S. was\r\nsupported by Gordon and Betty
Moore Foundation’s EPiQS\r\nInitiative through Grant No. GBMF4307. F.P. acknowledges\r\nsupport
from the DFG Research Unit FOR 1807 through Grant\r\nNo. PO 1370/2-1."
article_number: '104203'
author:
- first_name: Daniel
full_name: Hetterich, Daniel
last_name: Hetterich
- first_name: Maksym
full_name: Serbyn, Maksym
id: 47809E7E-F248-11E8-B48F-1D18A9856A87
last_name: Serbyn
orcid: 0000-0002-2399-5827
- first_name: Fernando
full_name: Domínguez, Fernando
last_name: Domínguez
- first_name: Frank
full_name: Pollmann, Frank
last_name: Pollmann
- first_name: Björn
full_name: Trauzettel, Björn
last_name: Trauzettel
citation:
ama: Hetterich D, Serbyn M, Domínguez F, Pollmann F, Trauzettel B. Noninteracting
central site model localization and logarithmic entanglement growth. Physical
Review B. 2017;96(10). doi:10.1103/PhysRevB.96.104203
apa: Hetterich, D., Serbyn, M., Domínguez, F., Pollmann, F., & Trauzettel, B.
(2017). Noninteracting central site model localization and logarithmic entanglement
growth. Physical Review B. American Physical Society. https://doi.org/10.1103/PhysRevB.96.104203
chicago: Hetterich, Daniel, Maksym Serbyn, Fernando Domínguez, Frank Pollmann, and
Björn Trauzettel. “Noninteracting Central Site Model Localization and Logarithmic
Entanglement Growth.” Physical Review B. American Physical Society, 2017.
https://doi.org/10.1103/PhysRevB.96.104203.
ieee: D. Hetterich, M. Serbyn, F. Domínguez, F. Pollmann, and B. Trauzettel, “Noninteracting
central site model localization and logarithmic entanglement growth,” Physical
Review B, vol. 96, no. 10. American Physical Society, 2017.
ista: Hetterich D, Serbyn M, Domínguez F, Pollmann F, Trauzettel B. 2017. Noninteracting
central site model localization and logarithmic entanglement growth. Physical
Review B. 96(10), 104203.
mla: Hetterich, Daniel, et al. “Noninteracting Central Site Model Localization and
Logarithmic Entanglement Growth.” Physical Review B, vol. 96, no. 10, 104203,
American Physical Society, 2017, doi:10.1103/PhysRevB.96.104203.
short: D. Hetterich, M. Serbyn, F. Domínguez, F. Pollmann, B. Trauzettel, Physical
Review B 96 (2017).
date_created: 2018-12-11T11:48:09Z
date_published: 2017-09-13T00:00:00Z
date_updated: 2021-01-12T08:12:35Z
day: '13'
department:
- _id: MaSe
doi: 10.1103/PhysRevB.96.104203
intvolume: ' 96'
issue: '10'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://arxiv.org/abs/1701.02744
month: '09'
oa: 1
oa_version: Submitted Version
publication: Physical Review B
publication_identifier:
issn:
- '24699950'
publication_status: published
publisher: American Physical Society
publist_id: '6955'
quality_controlled: '1'
scopus_import: 1
status: public
title: Noninteracting central site model localization and logarithmic entanglement
growth
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 96
year: '2017'
...
---
_id: '725'
abstract:
- lang: eng
text: Individual computations and social interactions underlying collective behavior
in groups of animals are of great ethological, behavioral, and theoretical interest.
While complex individual behaviors have successfully been parsed into small dictionaries
of stereotyped behavioral modes, studies of collective behavior largely ignored
these findings; instead, their focus was on inferring single, mode-independent
social interaction rules that reproduced macroscopic and often qualitative features
of group behavior. Here, we bring these two approaches together to predict individual
swimming patterns of adult zebrafish in a group. We show that fish alternate between
an “active” mode, in which they are sensitive to the swimming patterns of conspecifics,
and a “passive” mode, where they ignore them. Using a model that accounts for
these two modes explicitly, we predict behaviors of individual fish with high
accuracy, outperforming previous approaches that assumed a single continuous computation
by individuals and simple metric or topological weighing of neighbors’ behavior.
At the group level, switching between active and passive modes is uncorrelated
among fish, but correlated directional swimming behavior still emerges. Our quantitative
approach for studying complex, multi-modal individual behavior jointly with emergent
group behavior is readily extensible to additional behavioral modes and their
neural correlates as well as to other species.
author:
- first_name: Roy
full_name: Harpaz, Roy
last_name: Harpaz
- first_name: Gasper
full_name: Tkacik, Gasper
id: 3D494DCA-F248-11E8-B48F-1D18A9856A87
last_name: Tkacik
orcid: 0000-0002-6699-1455
- first_name: Elad
full_name: Schneidman, Elad
last_name: Schneidman
citation:
ama: Harpaz R, Tkačik G, Schneidman E. Discrete modes of social information processing
predict individual behavior of fish in a group. PNAS. 2017;114(38):10149-10154.
doi:10.1073/pnas.1703817114
apa: Harpaz, R., Tkačik, G., & Schneidman, E. (2017). Discrete modes of social
information processing predict individual behavior of fish in a group. PNAS.
National Academy of Sciences. https://doi.org/10.1073/pnas.1703817114
chicago: Harpaz, Roy, Gašper Tkačik, and Elad Schneidman. “Discrete Modes of Social
Information Processing Predict Individual Behavior of Fish in a Group.” PNAS.
National Academy of Sciences, 2017. https://doi.org/10.1073/pnas.1703817114.
ieee: R. Harpaz, G. Tkačik, and E. Schneidman, “Discrete modes of social information
processing predict individual behavior of fish in a group,” PNAS, vol.
114, no. 38. National Academy of Sciences, pp. 10149–10154, 2017.
ista: Harpaz R, Tkačik G, Schneidman E. 2017. Discrete modes of social information
processing predict individual behavior of fish in a group. PNAS. 114(38), 10149–10154.
mla: Harpaz, Roy, et al. “Discrete Modes of Social Information Processing Predict
Individual Behavior of Fish in a Group.” PNAS, vol. 114, no. 38, National
Academy of Sciences, 2017, pp. 10149–54, doi:10.1073/pnas.1703817114.
short: R. Harpaz, G. Tkačik, E. Schneidman, PNAS 114 (2017) 10149–10154.
date_created: 2018-12-11T11:48:10Z
date_published: 2017-09-19T00:00:00Z
date_updated: 2021-01-12T08:12:36Z
day: '19'
department:
- _id: GaTk
doi: 10.1073/pnas.1703817114
external_id:
pmid:
- '28874581'
intvolume: ' 114'
issue: '38'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5617265/
month: '09'
oa: 1
oa_version: Submitted Version
page: 10149 - 10154
pmid: 1
publication: PNAS
publication_identifier:
issn:
- '00278424'
publication_status: published
publisher: National Academy of Sciences
publist_id: '6953'
quality_controlled: '1'
scopus_import: 1
status: public
title: Discrete modes of social information processing predict individual behavior
of fish in a group
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 114
year: '2017'
...
---
_id: '726'
abstract:
- lang: eng
text: The morphogenesis of branched organs remains a subject of abiding interest.
Although much is known about the underlying signaling pathways, it remains unclear
how macroscopic features of branched organs, including their size, network topology,
and spatial patterning, are encoded. Here, we show that, in mouse mammary gland,
kidney, and human prostate, these features can be explained quantitatively within
a single unifying framework of branching and annihilating random walks. Based
on quantitative analyses of large-scale organ reconstructions and proliferation
kinetics measurements, we propose that morphogenesis follows from the proliferative
activity of equipotent tips that stochastically branch and randomly explore their
environment but compete neutrally for space, becoming proliferatively inactive
when in proximity with neighboring ducts. These results show that complex branched
epithelial structures develop as a self-organized process, reliant upon a strikingly
simple but generic rule, without recourse to a rigid and deterministic sequence
of genetically programmed events.
article_processing_charge: No
author:
- first_name: Edouard B
full_name: Hannezo, Edouard B
id: 3A9DB764-F248-11E8-B48F-1D18A9856A87
last_name: Hannezo
orcid: 0000-0001-6005-1561
- first_name: Colinda
full_name: Scheele, Colinda
last_name: Scheele
- first_name: Mohammad
full_name: Moad, Mohammad
last_name: Moad
- first_name: Nicholas
full_name: Drogo, Nicholas
last_name: Drogo
- first_name: Rakesh
full_name: Heer, Rakesh
last_name: Heer
- first_name: Rosemary
full_name: Sampogna, Rosemary
last_name: Sampogna
- first_name: Jacco
full_name: Van Rheenen, Jacco
last_name: Van Rheenen
- first_name: Benjamin
full_name: Simons, Benjamin
last_name: Simons
citation:
ama: Hannezo EB, Scheele C, Moad M, et al. A unifying theory of branching morphogenesis.
Cell. 2017;171(1):242-255. doi:10.1016/j.cell.2017.08.026
apa: Hannezo, E. B., Scheele, C., Moad, M., Drogo, N., Heer, R., Sampogna, R., …
Simons, B. (2017). A unifying theory of branching morphogenesis. Cell.
Cell Press. https://doi.org/10.1016/j.cell.2017.08.026
chicago: Hannezo, Edouard B, Colinda Scheele, Mohammad Moad, Nicholas Drogo, Rakesh
Heer, Rosemary Sampogna, Jacco Van Rheenen, and Benjamin Simons. “A Unifying Theory
of Branching Morphogenesis.” Cell. Cell Press, 2017. https://doi.org/10.1016/j.cell.2017.08.026.
ieee: E. B. Hannezo et al., “A unifying theory of branching morphogenesis,”
Cell, vol. 171, no. 1. Cell Press, pp. 242–255, 2017.
ista: Hannezo EB, Scheele C, Moad M, Drogo N, Heer R, Sampogna R, Van Rheenen J,
Simons B. 2017. A unifying theory of branching morphogenesis. Cell. 171(1), 242–255.
mla: Hannezo, Edouard B., et al. “A Unifying Theory of Branching Morphogenesis.”
Cell, vol. 171, no. 1, Cell Press, 2017, pp. 242–55, doi:10.1016/j.cell.2017.08.026.
short: E.B. Hannezo, C. Scheele, M. Moad, N. Drogo, R. Heer, R. Sampogna, J. Van
Rheenen, B. Simons, Cell 171 (2017) 242–255.
date_created: 2018-12-11T11:48:10Z
date_published: 2017-09-21T00:00:00Z
date_updated: 2023-09-28T11:34:17Z
day: '21'
ddc:
- '539'
department:
- _id: EdHa
doi: 10.1016/j.cell.2017.08.026
external_id:
isi:
- '000411331800024'
file:
- access_level: open_access
checksum: 7a036d93a9e2e597af9bb504d6133aca
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:11:17Z
date_updated: 2020-07-14T12:47:55Z
file_id: '4870'
file_name: IST-2017-883-v1+1_PIIS0092867417309510.pdf
file_size: 12670204
relation: main_file
file_date_updated: 2020-07-14T12:47:55Z
has_accepted_license: '1'
intvolume: ' 171'
isi: 1
issue: '1'
language:
- iso: eng
month: '09'
oa: 1
oa_version: Published Version
page: 242 - 255
publication: Cell
publication_identifier:
issn:
- '00928674'
publication_status: published
publisher: Cell Press
publist_id: '6952'
pubrep_id: '883'
quality_controlled: '1'
scopus_import: '1'
status: public
title: A unifying theory of branching morphogenesis
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 171
year: '2017'
...
---
_id: '727'
abstract:
- lang: eng
text: 'Actin filaments polymerizing against membranes power endocytosis, vesicular
traffic, and cell motility. In vitro reconstitution studies suggest that the structure
and the dynamics of actin networks respond to mechanical forces. We demonstrate
that lamellipodial actin of migrating cells responds to mechanical load when membrane
tension is modulated. In a steady state, migrating cell filaments assume the canonical
dendritic geometry, defined by Arp2/3-generated 70° branch points. Increased tension
triggers a dense network with a broadened range of angles, whereas decreased tension
causes a shift to a sparse configuration dominated by filaments growing perpendicularly
to the plasma membrane. We show that these responses emerge from the geometry
of branched actin: when load per filament decreases, elongation speed increases
and perpendicular filaments gradually outcompete others because they polymerize
the shortest distance to the membrane, where they are protected from capping.
This network-intrinsic geometrical adaptation mechanism tunes protrusive force
in response to mechanical load.'
acknowledged_ssus:
- _id: ScienComp
article_processing_charge: No
author:
- first_name: Jan
full_name: Mueller, Jan
last_name: Mueller
- first_name: Gregory
full_name: Szep, Gregory
id: 4BFB7762-F248-11E8-B48F-1D18A9856A87
last_name: Szep
- first_name: Maria
full_name: Nemethova, Maria
id: 34E27F1C-F248-11E8-B48F-1D18A9856A87
last_name: Nemethova
- first_name: Ingrid
full_name: De Vries, Ingrid
id: 4C7D837E-F248-11E8-B48F-1D18A9856A87
last_name: De Vries
- first_name: Arnon
full_name: Lieber, Arnon
last_name: Lieber
- first_name: Christoph
full_name: Winkler, Christoph
last_name: Winkler
- first_name: Karsten
full_name: Kruse, Karsten
last_name: Kruse
- first_name: John
full_name: Small, John
last_name: Small
- first_name: Christian
full_name: Schmeiser, Christian
last_name: Schmeiser
- first_name: Kinneret
full_name: Keren, Kinneret
last_name: Keren
- first_name: Robert
full_name: Hauschild, Robert
id: 4E01D6B4-F248-11E8-B48F-1D18A9856A87
last_name: Hauschild
orcid: 0000-0001-9843-3522
- first_name: Michael K
full_name: Sixt, Michael K
id: 41E9FBEA-F248-11E8-B48F-1D18A9856A87
last_name: Sixt
orcid: 0000-0002-6620-9179
citation:
ama: Mueller J, Szep G, Nemethova M, et al. Load adaptation of lamellipodial actin
networks. Cell. 2017;171(1):188-200. doi:10.1016/j.cell.2017.07.051
apa: Mueller, J., Szep, G., Nemethova, M., de Vries, I., Lieber, A., Winkler, C.,
… Sixt, M. K. (2017). Load adaptation of lamellipodial actin networks. Cell.
Cell Press. https://doi.org/10.1016/j.cell.2017.07.051
chicago: Mueller, Jan, Gregory Szep, Maria Nemethova, Ingrid de Vries, Arnon Lieber,
Christoph Winkler, Karsten Kruse, et al. “Load Adaptation of Lamellipodial Actin
Networks.” Cell. Cell Press, 2017. https://doi.org/10.1016/j.cell.2017.07.051.
ieee: J. Mueller et al., “Load adaptation of lamellipodial actin networks,”
Cell, vol. 171, no. 1. Cell Press, pp. 188–200, 2017.
ista: Mueller J, Szep G, Nemethova M, de Vries I, Lieber A, Winkler C, Kruse K,
Small J, Schmeiser C, Keren K, Hauschild R, Sixt MK. 2017. Load adaptation of
lamellipodial actin networks. Cell. 171(1), 188–200.
mla: Mueller, Jan, et al. “Load Adaptation of Lamellipodial Actin Networks.” Cell,
vol. 171, no. 1, Cell Press, 2017, pp. 188–200, doi:10.1016/j.cell.2017.07.051.
short: J. Mueller, G. Szep, M. Nemethova, I. de Vries, A. Lieber, C. Winkler, K.
Kruse, J. Small, C. Schmeiser, K. Keren, R. Hauschild, M.K. Sixt, Cell 171 (2017)
188–200.
date_created: 2018-12-11T11:48:10Z
date_published: 2017-09-21T00:00:00Z
date_updated: 2023-09-28T11:33:49Z
day: '21'
department:
- _id: MiSi
- _id: Bio
doi: 10.1016/j.cell.2017.07.051
ec_funded: 1
external_id:
isi:
- '000411331800020'
intvolume: ' 171'
isi: 1
issue: '1'
language:
- iso: eng
month: '09'
oa_version: None
page: 188 - 200
project:
- _id: 25AD6156-B435-11E9-9278-68D0E5697425
grant_number: LS13-029
name: Modeling of Polarization and Motility of Leukocytes in Three-Dimensional Environments
- _id: 25A603A2-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '281556'
name: Cytoskeletal force generation and force transduction of migrating leukocytes
(EU)
publication: Cell
publication_identifier:
issn:
- '00928674'
publication_status: published
publisher: Cell Press
publist_id: '6951'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Load adaptation of lamellipodial actin networks
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 171
year: '2017'
...
---
_id: '728'
abstract:
- lang: eng
text: During animal development, cell-fate-specific changes in gene expression can
modify the material properties of a tissue and drive tissue morphogenesis. While
mechanistic insights into the genetic control of tissue-shaping events are beginning
to emerge, how tissue morphogenesis and mechanics can reciprocally impact cell-fate
specification remains relatively unexplored. Here we review recent findings reporting
how multicellular morphogenetic events and their underlying mechanical forces
can feed back into gene regulatory pathways to specify cell fate. We further discuss
emerging techniques that allow for the direct measurement and manipulation of
mechanical signals in vivo, offering unprecedented access to study mechanotransduction
during development. Examination of the mechanical control of cell fate during
tissue morphogenesis will pave the way to an integrated understanding of the design
principles that underlie robust tissue patterning in embryonic development.
article_processing_charge: No
author:
- first_name: Chii
full_name: Chan, Chii
last_name: Chan
- first_name: Carl-Philipp J
full_name: Heisenberg, Carl-Philipp J
id: 39427864-F248-11E8-B48F-1D18A9856A87
last_name: Heisenberg
orcid: 0000-0002-0912-4566
- first_name: Takashi
full_name: Hiiragi, Takashi
last_name: Hiiragi
citation:
ama: Chan C, Heisenberg C-PJ, Hiiragi T. Coordination of morphogenesis and cell
fate specification in development. Current Biology. 2017;27(18):R1024-R1035.
doi:10.1016/j.cub.2017.07.010
apa: Chan, C., Heisenberg, C.-P. J., & Hiiragi, T. (2017). Coordination of morphogenesis
and cell fate specification in development. Current Biology. Cell Press.
https://doi.org/10.1016/j.cub.2017.07.010
chicago: Chan, Chii, Carl-Philipp J Heisenberg, and Takashi Hiiragi. “Coordination
of Morphogenesis and Cell Fate Specification in Development.” Current Biology.
Cell Press, 2017. https://doi.org/10.1016/j.cub.2017.07.010.
ieee: C. Chan, C.-P. J. Heisenberg, and T. Hiiragi, “Coordination of morphogenesis
and cell fate specification in development,” Current Biology, vol. 27,
no. 18. Cell Press, pp. R1024–R1035, 2017.
ista: Chan C, Heisenberg C-PJ, Hiiragi T. 2017. Coordination of morphogenesis and
cell fate specification in development. Current Biology. 27(18), R1024–R1035.
mla: Chan, Chii, et al. “Coordination of Morphogenesis and Cell Fate Specification
in Development.” Current Biology, vol. 27, no. 18, Cell Press, 2017, pp.
R1024–35, doi:10.1016/j.cub.2017.07.010.
short: C. Chan, C.-P.J. Heisenberg, T. Hiiragi, Current Biology 27 (2017) R1024–R1035.
date_created: 2018-12-11T11:48:11Z
date_published: 2017-09-18T00:00:00Z
date_updated: 2023-09-28T11:33:21Z
day: '18'
department:
- _id: CaHe
doi: 10.1016/j.cub.2017.07.010
external_id:
isi:
- '000411581800019'
intvolume: ' 27'
isi: 1
issue: '18'
language:
- iso: eng
month: '09'
oa_version: None
page: R1024 - R1035
publication: Current Biology
publication_identifier:
issn:
- '09609822'
publication_status: published
publisher: Cell Press
publist_id: '6949'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Coordination of morphogenesis and cell fate specification in development
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 27
year: '2017'
...
---
_id: '729'
abstract:
- lang: eng
text: The cellular mechanisms allowing tissues to efficiently regenerate are not
fully understood. In this issue of Developmental Cell, Cao et al. (2017)) discover
that during zebrafish heart regeneration, epicardial cells at the leading edge
of regenerating tissue undergo endoreplication, possibly due to increased tissue
tension, thereby boosting their regenerative capacity.
article_processing_charge: No
author:
- first_name: Zoltan P
full_name: Spiro, Zoltan P
id: 426AD026-F248-11E8-B48F-1D18A9856A87
last_name: Spiro
- first_name: Carl-Philipp J
full_name: Heisenberg, Carl-Philipp J
id: 39427864-F248-11E8-B48F-1D18A9856A87
last_name: Heisenberg
orcid: 0000-0002-0912-4566
citation:
ama: Spiro ZP, Heisenberg C-PJ. Regeneration tensed up polyploidy takes the lead.
Developmental Cell. 2017;42(6):559-560. doi:10.1016/j.devcel.2017.09.008
apa: Spiro, Z. P., & Heisenberg, C.-P. J. (2017). Regeneration tensed up polyploidy
takes the lead. Developmental Cell. Cell Press. https://doi.org/10.1016/j.devcel.2017.09.008
chicago: Spiro, Zoltan P, and Carl-Philipp J Heisenberg. “Regeneration Tensed up
Polyploidy Takes the Lead.” Developmental Cell. Cell Press, 2017. https://doi.org/10.1016/j.devcel.2017.09.008.
ieee: Z. P. Spiro and C.-P. J. Heisenberg, “Regeneration tensed up polyploidy takes
the lead,” Developmental Cell, vol. 42, no. 6. Cell Press, pp. 559–560,
2017.
ista: Spiro ZP, Heisenberg C-PJ. 2017. Regeneration tensed up polyploidy takes the
lead. Developmental Cell. 42(6), 559–560.
mla: Spiro, Zoltan P., and Carl-Philipp J. Heisenberg. “Regeneration Tensed up Polyploidy
Takes the Lead.” Developmental Cell, vol. 42, no. 6, Cell Press, 2017,
pp. 559–60, doi:10.1016/j.devcel.2017.09.008.
short: Z.P. Spiro, C.-P.J. Heisenberg, Developmental Cell 42 (2017) 559–560.
date_created: 2018-12-11T11:48:11Z
date_published: 2017-01-01T00:00:00Z
date_updated: 2023-09-28T11:32:49Z
day: '01'
department:
- _id: CaHe
doi: 10.1016/j.devcel.2017.09.008
external_id:
isi:
- '000411582800003'
intvolume: ' 42'
isi: 1
issue: '6'
language:
- iso: eng
month: '01'
oa_version: None
page: 559 - 560
publication: Developmental Cell
publication_identifier:
issn:
- '15345807'
publication_status: published
publisher: Cell Press
publist_id: '6948'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Regeneration tensed up polyploidy takes the lead
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 42
year: '2017'
...
---
_id: '730'
abstract:
- lang: eng
text: Neural responses are highly structured, with population activity restricted
to a small subset of the astronomical range of possible activity patterns. Characterizing
these statistical regularities is important for understanding circuit computation,
but challenging in practice. Here we review recent approaches based on the maximum
entropy principle used for quantifying collective behavior in neural activity.
We highlight recent models that capture population-level statistics of neural
data, yielding insights into the organization of the neural code and its biological
substrate. Furthermore, the MaxEnt framework provides a general recipe for constructing
surrogate ensembles that preserve aspects of the data, but are otherwise maximally
unstructured. This idea can be used to generate a hierarchy of controls against
which rigorous statistical tests are possible.
article_processing_charge: No
author:
- first_name: Cristina
full_name: Savin, Cristina
id: 3933349E-F248-11E8-B48F-1D18A9856A87
last_name: Savin
- first_name: Gasper
full_name: Tkacik, Gasper
id: 3D494DCA-F248-11E8-B48F-1D18A9856A87
last_name: Tkacik
orcid: 0000-0002-6699-1455
citation:
ama: Savin C, Tkačik G. Maximum entropy models as a tool for building precise neural
controls. Current Opinion in Neurobiology. 2017;46:120-126. doi:10.1016/j.conb.2017.08.001
apa: Savin, C., & Tkačik, G. (2017). Maximum entropy models as a tool for building
precise neural controls. Current Opinion in Neurobiology. Elsevier. https://doi.org/10.1016/j.conb.2017.08.001
chicago: Savin, Cristina, and Gašper Tkačik. “Maximum Entropy Models as a Tool for
Building Precise Neural Controls.” Current Opinion in Neurobiology. Elsevier,
2017. https://doi.org/10.1016/j.conb.2017.08.001.
ieee: C. Savin and G. Tkačik, “Maximum entropy models as a tool for building precise
neural controls,” Current Opinion in Neurobiology, vol. 46. Elsevier, pp.
120–126, 2017.
ista: Savin C, Tkačik G. 2017. Maximum entropy models as a tool for building precise
neural controls. Current Opinion in Neurobiology. 46, 120–126.
mla: Savin, Cristina, and Gašper Tkačik. “Maximum Entropy Models as a Tool for Building
Precise Neural Controls.” Current Opinion in Neurobiology, vol. 46, Elsevier,
2017, pp. 120–26, doi:10.1016/j.conb.2017.08.001.
short: C. Savin, G. Tkačik, Current Opinion in Neurobiology 46 (2017) 120–126.
date_created: 2018-12-11T11:48:11Z
date_published: 2017-10-01T00:00:00Z
date_updated: 2023-09-28T11:32:22Z
day: '01'
department:
- _id: GaTk
doi: 10.1016/j.conb.2017.08.001
ec_funded: 1
external_id:
isi:
- '000416196400016'
intvolume: ' 46'
isi: 1
language:
- iso: eng
month: '10'
oa_version: None
page: 120 - 126
project:
- _id: 25681D80-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '291734'
name: International IST Postdoc Fellowship Programme
publication: Current Opinion in Neurobiology
publication_identifier:
issn:
- '09594388'
publication_status: published
publisher: Elsevier
publist_id: '6943'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Maximum entropy models as a tool for building precise neural controls
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 46
year: '2017'
...
---
_id: '731'
abstract:
- lang: eng
text: Genetic variations in the oxytocin receptor gene affect patients with ASD
and ADHD differently.
article_number: eaap8168
author:
- first_name: Gaia
full_name: Novarino, Gaia
id: 3E57A680-F248-11E8-B48F-1D18A9856A87
last_name: Novarino
orcid: 0000-0002-7673-7178
citation:
ama: Novarino G. The science of love in ASD and ADHD. Science Translational Medicine.
2017;9(411). doi:10.1126/scitranslmed.aap8168
apa: Novarino, G. (2017). The science of love in ASD and ADHD. Science Translational
Medicine. American Association for the Advancement of Science. https://doi.org/10.1126/scitranslmed.aap8168
chicago: Novarino, Gaia. “The Science of Love in ASD and ADHD.” Science Translational
Medicine. American Association for the Advancement of Science, 2017. https://doi.org/10.1126/scitranslmed.aap8168.
ieee: G. Novarino, “The science of love in ASD and ADHD,” Science Translational
Medicine, vol. 9, no. 411. American Association for the Advancement of Science,
2017.
ista: Novarino G. 2017. The science of love in ASD and ADHD. Science Translational
Medicine. 9(411), eaap8168.
mla: Novarino, Gaia. “The Science of Love in ASD and ADHD.” Science Translational
Medicine, vol. 9, no. 411, eaap8168, American Association for the Advancement
of Science, 2017, doi:10.1126/scitranslmed.aap8168.
short: G. Novarino, Science Translational Medicine 9 (2017).
date_created: 2018-12-11T11:48:12Z
date_published: 2017-10-11T00:00:00Z
date_updated: 2021-01-12T08:12:57Z
day: '11'
department:
- _id: GaNo
doi: 10.1126/scitranslmed.aap8168
intvolume: ' 9'
issue: '411'
language:
- iso: eng
month: '10'
oa_version: None
publication: Science Translational Medicine
publication_identifier:
issn:
- '19466234'
publication_status: published
publisher: American Association for the Advancement of Science
publist_id: '6938'
quality_controlled: '1'
scopus_import: 1
status: public
title: The science of love in ASD and ADHD
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 9
year: '2017'
...
---
_id: '732'
abstract:
- lang: eng
text: 'Background: Social insects form densely crowded societies in environments
with high pathogen loads, but have evolved collective defences that mitigate the
impact of disease. However, colony-founding queens lack this protection and suffer
high rates of mortality. The impact of pathogens may be exacerbated in species
where queens found colonies together, as healthy individuals may contract pathogens
from infectious co-founders. Therefore, we tested whether ant queens avoid founding
colonies with pathogen-exposed conspecifics and how they might limit disease transmission
from infectious individuals. Results: Using Lasius Niger queens and a naturally
infecting fungal pathogen Metarhizium brunneum, we observed that queens were equally
likely to found colonies with another pathogen-exposed or sham-treated queen.
However, when one queen died, the surviving individual performed biting, burial
and removal of the corpse. These undertaking behaviours were performed prophylactically,
i.e. targeted equally towards non-infected and infected corpses, as well as carried
out before infected corpses became infectious. Biting and burial reduced the risk
of the queens contracting and dying from disease from an infectious corpse of
a dead co-foundress. Conclusions: We show that co-founding ant queens express
undertaking behaviours that, in mature colonies, are performed exclusively by
workers. Such infection avoidance behaviours act before the queens can contract
the disease and will therefore improve the overall chance of colony founding success
in ant queens.'
article_number: '219'
article_processing_charge: Yes
article_type: original
author:
- first_name: Christopher
full_name: Pull, Christopher
id: 3C7F4840-F248-11E8-B48F-1D18A9856A87
last_name: Pull
orcid: 0000-0003-1122-3982
- first_name: Sylvia
full_name: Cremer, Sylvia
id: 2F64EC8C-F248-11E8-B48F-1D18A9856A87
last_name: Cremer
orcid: 0000-0002-2193-3868
citation:
ama: Pull C, Cremer S. Co-founding ant queens prevent disease by performing prophylactic
undertaking behaviour. BMC Evolutionary Biology. 2017;17(1). doi:10.1186/s12862-017-1062-4
apa: Pull, C., & Cremer, S. (2017). Co-founding ant queens prevent disease by
performing prophylactic undertaking behaviour. BMC Evolutionary Biology.
BioMed Central. https://doi.org/10.1186/s12862-017-1062-4
chicago: Pull, Christopher, and Sylvia Cremer. “Co-Founding Ant Queens Prevent Disease
by Performing Prophylactic Undertaking Behaviour.” BMC Evolutionary Biology.
BioMed Central, 2017. https://doi.org/10.1186/s12862-017-1062-4.
ieee: C. Pull and S. Cremer, “Co-founding ant queens prevent disease by performing
prophylactic undertaking behaviour,” BMC Evolutionary Biology, vol. 17,
no. 1. BioMed Central, 2017.
ista: Pull C, Cremer S. 2017. Co-founding ant queens prevent disease by performing
prophylactic undertaking behaviour. BMC Evolutionary Biology. 17(1), 219.
mla: Pull, Christopher, and Sylvia Cremer. “Co-Founding Ant Queens Prevent Disease
by Performing Prophylactic Undertaking Behaviour.” BMC Evolutionary Biology,
vol. 17, no. 1, 219, BioMed Central, 2017, doi:10.1186/s12862-017-1062-4.
short: C. Pull, S. Cremer, BMC Evolutionary Biology 17 (2017).
date_created: 2018-12-11T11:48:12Z
date_published: 2017-10-13T00:00:00Z
date_updated: 2023-09-28T11:31:32Z
day: '13'
ddc:
- '576'
- '592'
department:
- _id: SyCr
doi: 10.1186/s12862-017-1062-4
ec_funded: 1
external_id:
isi:
- '000412816800001'
file:
- access_level: open_access
checksum: 3e24a2cfd48f49f7b3643d08d30fb480
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:17:18Z
date_updated: 2020-07-14T12:47:55Z
file_id: '5271'
file_name: IST-2017-882-v1+1_12862_2017_Article_1062.pdf
file_size: 949857
relation: main_file
file_date_updated: 2020-07-14T12:47:55Z
has_accepted_license: '1'
intvolume: ' 17'
isi: 1
issue: '1'
language:
- iso: eng
month: '10'
oa: 1
oa_version: Published Version
project:
- _id: 25DC711C-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '243071'
name: 'Social Vaccination in Ant Colonies: from Individual Mechanisms to Society
Effects'
publication: BMC Evolutionary Biology
publication_identifier:
issn:
- '14712148'
publication_status: published
publisher: BioMed Central
publist_id: '6937'
pubrep_id: '882'
quality_controlled: '1'
related_material:
record:
- id: '819'
relation: dissertation_contains
status: public
scopus_import: '1'
status: public
title: Co-founding ant queens prevent disease by performing prophylactic undertaking
behaviour
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 17
year: '2017'
...
---
_id: '733'
abstract:
- lang: eng
text: Let A and B be two N by N deterministic Hermitian matrices and let U be an
N by N Haar distributed unitary matrix. It is well known that the spectral distribution
of the sum H = A + UBU∗ converges weakly to the free additive convolution of the
spectral distributions of A and B, as N tends to infinity. We establish the optimal
convergence rate in the bulk of the spectrum.
acknowledgement: Partially supported by ERC Advanced Grant RANMAT No. 338804, Hong
Kong RGC grant ECS 26301517, and the Göran Gustafsson Foundation
article_processing_charge: No
author:
- first_name: Zhigang
full_name: Bao, Zhigang
id: 442E6A6C-F248-11E8-B48F-1D18A9856A87
last_name: Bao
orcid: 0000-0003-3036-1475
- first_name: László
full_name: Erdös, László
id: 4DBD5372-F248-11E8-B48F-1D18A9856A87
last_name: Erdös
orcid: 0000-0001-5366-9603
- first_name: Kevin
full_name: Schnelli, Kevin
id: 434AD0AE-F248-11E8-B48F-1D18A9856A87
last_name: Schnelli
orcid: 0000-0003-0954-3231
citation:
ama: Bao Z, Erdös L, Schnelli K. Convergence rate for spectral distribution of addition
of random matrices. Advances in Mathematics. 2017;319:251-291. doi:10.1016/j.aim.2017.08.028
apa: Bao, Z., Erdös, L., & Schnelli, K. (2017). Convergence rate for spectral
distribution of addition of random matrices. Advances in Mathematics. Academic
Press. https://doi.org/10.1016/j.aim.2017.08.028
chicago: Bao, Zhigang, László Erdös, and Kevin Schnelli. “Convergence Rate for Spectral
Distribution of Addition of Random Matrices.” Advances in Mathematics.
Academic Press, 2017. https://doi.org/10.1016/j.aim.2017.08.028.
ieee: Z. Bao, L. Erdös, and K. Schnelli, “Convergence rate for spectral distribution
of addition of random matrices,” Advances in Mathematics, vol. 319. Academic
Press, pp. 251–291, 2017.
ista: Bao Z, Erdös L, Schnelli K. 2017. Convergence rate for spectral distribution
of addition of random matrices. Advances in Mathematics. 319, 251–291.
mla: Bao, Zhigang, et al. “Convergence Rate for Spectral Distribution of Addition
of Random Matrices.” Advances in Mathematics, vol. 319, Academic Press,
2017, pp. 251–91, doi:10.1016/j.aim.2017.08.028.
short: Z. Bao, L. Erdös, K. Schnelli, Advances in Mathematics 319 (2017) 251–291.
date_created: 2018-12-11T11:48:13Z
date_published: 2017-10-15T00:00:00Z
date_updated: 2023-09-28T11:30:42Z
day: '15'
department:
- _id: LaEr
doi: 10.1016/j.aim.2017.08.028
ec_funded: 1
external_id:
isi:
- '000412150400010'
intvolume: ' 319'
isi: 1
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://arxiv.org/abs/1606.03076
month: '10'
oa: 1
oa_version: Submitted Version
page: 251 - 291
project:
- _id: 258DCDE6-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '338804'
name: Random matrices, universality and disordered quantum systems
publication: Advances in Mathematics
publication_status: published
publisher: Academic Press
publist_id: '6935'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Convergence rate for spectral distribution of addition of random matrices
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 319
year: '2017'
...
---
_id: '734'
abstract:
- lang: eng
text: 'Social insect societies are long-standing models for understanding social
behaviour and evolution. Unlike other advanced biological societies (such as the
multicellular body), the component parts of social insect societies can be easily
deconstructed and manipulated. Recent methodological and theoretical innovations
have exploited this trait to address an expanded range of biological questions.
We illustrate the broadening range of biological insight coming from social insect
biology with four examples. These new frontiers promote open-minded, interdisciplinary
exploration of one of the richest and most complex of biological phenomena: sociality.'
article_processing_charge: No
article_type: original
author:
- first_name: Patrick
full_name: Kennedy, Patrick
last_name: Kennedy
- first_name: Gemma
full_name: Baron, Gemma
last_name: Baron
- first_name: Bitao
full_name: Qiu, Bitao
last_name: Qiu
- first_name: Dalial
full_name: Freitak, Dalial
last_name: Freitak
- first_name: Heikki
full_name: Helantera, Heikki
last_name: Helantera
- first_name: Edmund
full_name: Hunt, Edmund
last_name: Hunt
- first_name: Fabio
full_name: Manfredini, Fabio
last_name: Manfredini
- first_name: Thomas
full_name: O'Shea Wheller, Thomas
last_name: O'Shea Wheller
- first_name: Solenn
full_name: Patalano, Solenn
last_name: Patalano
- first_name: Christopher
full_name: Pull, Christopher
id: 3C7F4840-F248-11E8-B48F-1D18A9856A87
last_name: Pull
orcid: 0000-0003-1122-3982
- first_name: Takao
full_name: Sasaki, Takao
last_name: Sasaki
- first_name: Daisy
full_name: Taylor, Daisy
last_name: Taylor
- first_name: Christopher
full_name: Wyatt, Christopher
last_name: Wyatt
- first_name: Seirian
full_name: Sumner, Seirian
last_name: Sumner
citation:
ama: Kennedy P, Baron G, Qiu B, et al. Deconstructing superorganisms and societies
to address big questions in biology. Trends in Ecology and Evolution. 2017;32(11):861-872.
doi:10.1016/j.tree.2017.08.004
apa: Kennedy, P., Baron, G., Qiu, B., Freitak, D., Helantera, H., Hunt, E., … Sumner,
S. (2017). Deconstructing superorganisms and societies to address big questions
in biology. Trends in Ecology and Evolution. Cell Press. https://doi.org/10.1016/j.tree.2017.08.004
chicago: Kennedy, Patrick, Gemma Baron, Bitao Qiu, Dalial Freitak, Heikki Helantera,
Edmund Hunt, Fabio Manfredini, et al. “Deconstructing Superorganisms and Societies
to Address Big Questions in Biology.” Trends in Ecology and Evolution.
Cell Press, 2017. https://doi.org/10.1016/j.tree.2017.08.004.
ieee: P. Kennedy et al., “Deconstructing superorganisms and societies to
address big questions in biology,” Trends in Ecology and Evolution, vol.
32, no. 11. Cell Press, pp. 861–872, 2017.
ista: Kennedy P, Baron G, Qiu B, Freitak D, Helantera H, Hunt E, Manfredini F, O’Shea
Wheller T, Patalano S, Pull C, Sasaki T, Taylor D, Wyatt C, Sumner S. 2017. Deconstructing
superorganisms and societies to address big questions in biology. Trends in Ecology
and Evolution. 32(11), 861–872.
mla: Kennedy, Patrick, et al. “Deconstructing Superorganisms and Societies to Address
Big Questions in Biology.” Trends in Ecology and Evolution, vol. 32, no.
11, Cell Press, 2017, pp. 861–72, doi:10.1016/j.tree.2017.08.004.
short: P. Kennedy, G. Baron, B. Qiu, D. Freitak, H. Helantera, E. Hunt, F. Manfredini,
T. O’Shea Wheller, S. Patalano, C. Pull, T. Sasaki, D. Taylor, C. Wyatt, S. Sumner,
Trends in Ecology and Evolution 32 (2017) 861–872.
date_created: 2018-12-11T11:48:13Z
date_published: 2017-11-01T00:00:00Z
date_updated: 2023-09-27T14:15:15Z
day: '01'
ddc:
- '570'
department:
- _id: SyCr
doi: 10.1016/j.tree.2017.08.004
external_id:
isi:
- '000413231900011'
file:
- access_level: open_access
checksum: c8f49309ed9436201814fa7153d66a99
content_type: application/pdf
creator: dernst
date_created: 2020-05-14T16:22:27Z
date_updated: 2020-07-14T12:47:56Z
file_id: '7842'
file_name: 2017_TrendsEcology_Kennedy.pdf
file_size: 15018382
relation: main_file
file_date_updated: 2020-07-14T12:47:56Z
has_accepted_license: '1'
intvolume: ' 32'
isi: 1
issue: '11'
language:
- iso: eng
month: '11'
oa: 1
oa_version: Submitted Version
page: 861 - 872
publication: Trends in Ecology and Evolution
publication_identifier:
issn:
- '01695347'
publication_status: published
publisher: Cell Press
publist_id: '6933'
quality_controlled: '1'
related_material:
record:
- id: '819'
relation: dissertation_contains
status: public
scopus_import: '1'
status: public
title: Deconstructing superorganisms and societies to address big questions in biology
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 32
year: '2017'
...
---
_id: '735'
abstract:
- lang: eng
text: Cell-cell contact formation constitutes an essential step in evolution, leading
to the differentiation of specialized cell types. However, remarkably little is
known about whether and how the interplay between contact formation and fate specification
affects development. Here, we identify a positive feedback loop between cell-cell
contact duration, morphogen signaling, and mesendoderm cell-fate specification
during zebrafish gastrulation. We show that long-lasting cell-cell contacts enhance
the competence of prechordal plate (ppl) progenitor cells to respond to Nodal
signaling, required for ppl cell-fate specification. We further show that Nodal
signaling promotes ppl cell-cell contact duration, generating a positive feedback
loop between ppl cell-cell contact duration and cell-fate specification. Finally,
by combining mathematical modeling and experimentation, we show that this feedback
determines whether anterior axial mesendoderm cells become ppl or, instead, turn
into endoderm. Thus, the interdependent activities of cell-cell signaling and
contact formation control fate diversification within the developing embryo.
article_processing_charge: No
author:
- first_name: Vanessa
full_name: Barone, Vanessa
id: 419EECCC-F248-11E8-B48F-1D18A9856A87
last_name: Barone
orcid: 0000-0003-2676-3367
- first_name: Moritz
full_name: Lang, Moritz
id: 29E0800A-F248-11E8-B48F-1D18A9856A87
last_name: Lang
- first_name: Gabriel
full_name: Krens, Gabriel
id: 2B819732-F248-11E8-B48F-1D18A9856A87
last_name: Krens
orcid: 0000-0003-4761-5996
- first_name: Saurabh
full_name: Pradhan, Saurabh
last_name: Pradhan
- first_name: Shayan
full_name: Shamipour, Shayan
id: 40B34FE2-F248-11E8-B48F-1D18A9856A87
last_name: Shamipour
- first_name: Keisuke
full_name: Sako, Keisuke
id: 3BED66BE-F248-11E8-B48F-1D18A9856A87
last_name: Sako
orcid: 0000-0002-6453-8075
- first_name: Mateusz K
full_name: Sikora, Mateusz K
id: 2F74BCDE-F248-11E8-B48F-1D18A9856A87
last_name: Sikora
- first_name: Calin C
full_name: Guet, Calin C
id: 47F8433E-F248-11E8-B48F-1D18A9856A87
last_name: Guet
orcid: 0000-0001-6220-2052
- first_name: Carl-Philipp J
full_name: Heisenberg, Carl-Philipp J
id: 39427864-F248-11E8-B48F-1D18A9856A87
last_name: Heisenberg
orcid: 0000-0002-0912-4566
citation:
ama: Barone V, Lang M, Krens G, et al. An effective feedback loop between cell-cell
contact duration and morphogen signaling determines cell fate. Developmental
Cell. 2017;43(2):198-211. doi:10.1016/j.devcel.2017.09.014
apa: Barone, V., Lang, M., Krens, G., Pradhan, S., Shamipour, S., Sako, K., … Heisenberg,
C.-P. J. (2017). An effective feedback loop between cell-cell contact duration
and morphogen signaling determines cell fate. Developmental Cell. Cell
Press. https://doi.org/10.1016/j.devcel.2017.09.014
chicago: Barone, Vanessa, Moritz Lang, Gabriel Krens, Saurabh Pradhan, Shayan Shamipour,
Keisuke Sako, Mateusz K Sikora, Calin C Guet, and Carl-Philipp J Heisenberg. “An
Effective Feedback Loop between Cell-Cell Contact Duration and Morphogen Signaling
Determines Cell Fate.” Developmental Cell. Cell Press, 2017. https://doi.org/10.1016/j.devcel.2017.09.014.
ieee: V. Barone et al., “An effective feedback loop between cell-cell contact
duration and morphogen signaling determines cell fate,” Developmental Cell,
vol. 43, no. 2. Cell Press, pp. 198–211, 2017.
ista: Barone V, Lang M, Krens G, Pradhan S, Shamipour S, Sako K, Sikora MK, Guet
CC, Heisenberg C-PJ. 2017. An effective feedback loop between cell-cell contact
duration and morphogen signaling determines cell fate. Developmental Cell. 43(2),
198–211.
mla: Barone, Vanessa, et al. “An Effective Feedback Loop between Cell-Cell Contact
Duration and Morphogen Signaling Determines Cell Fate.” Developmental Cell,
vol. 43, no. 2, Cell Press, 2017, pp. 198–211, doi:10.1016/j.devcel.2017.09.014.
short: V. Barone, M. Lang, G. Krens, S. Pradhan, S. Shamipour, K. Sako, M.K. Sikora,
C.C. Guet, C.-P.J. Heisenberg, Developmental Cell 43 (2017) 198–211.
date_created: 2018-12-11T11:48:13Z
date_published: 2017-10-23T00:00:00Z
date_updated: 2024-03-25T23:30:21Z
day: '23'
department:
- _id: CaHe
- _id: CaGu
- _id: GaTk
doi: 10.1016/j.devcel.2017.09.014
ec_funded: 1
external_id:
isi:
- '000413443700011'
intvolume: ' 43'
isi: 1
issue: '2'
language:
- iso: eng
month: '10'
oa_version: None
page: 198 - 211
project:
- _id: 25681D80-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '291734'
name: International IST Postdoc Fellowship Programme
- _id: 252DD2A6-B435-11E9-9278-68D0E5697425
call_identifier: FWF
grant_number: I2058
name: 'Cell segregation in gastrulation: the role of cell fate specification'
publication: Developmental Cell
publication_identifier:
issn:
- '15345807'
publication_status: published
publisher: Cell Press
publist_id: '6934'
quality_controlled: '1'
related_material:
record:
- id: '961'
relation: dissertation_contains
status: public
- id: '8350'
relation: dissertation_contains
status: public
scopus_import: '1'
status: public
title: An effective feedback loop between cell-cell contact duration and morphogen
signaling determines cell fate
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 43
year: '2017'
...
---
_id: '736'
abstract:
- lang: eng
text: The neurotransmitter receptor subtype, number, density, and distribution relative
to the location of transmitter release sites are key determinants of signal transmission.
AMPA-type ionotropic glutamate receptors (AMPARs) containing GluA3 and GluA4 subunits
are prominently expressed in subsets of neurons capable of firing action potentials
at high frequencies, such as auditory relay neurons. The auditory nerve (AN) forms
glutamatergic synapses on two types of relay neurons, bushy cells (BCs) and fusiform
cells (FCs) of the cochlear nucleus. AN-BC and AN-FC synapses have distinct kinetics;
thus, we investigated whether the number, density, and localization of GluA3 and
GluA4 subunits in these synapses are differentially organized using quantitative
freeze-fracture replica immunogold labeling. We identify a positive correlation
between the number of AMPARs and the size of AN-BC and AN-FC synapses. Both types
of AN synapses have similar numbers of AMPARs; however, the AN-BC have a higher
density of AMPARs than AN-FC synapses, because the AN-BC synapses are smaller.
A higher number and density of GluA3 subunits are observed at AN-BC synapses,
whereas a higher number and density of GluA4 subunits are observed at AN-FC synapses.
The intrasynaptic distribution of immunogold labeling revealed that AMPAR subunits,
particularly GluA3, are concentrated at the center of the AN-BC synapses. The
central distribution of AMPARs is absent in GluA3-knockout mice, and gold particles
are evenly distributed along the postsynaptic density. GluA4 gold labeling was
homogenously distributed along both synapse types. Thus, GluA3 and GluA4 subunits
are distributed at AN synapses in a target-cell-dependent manner.
article_processing_charge: No
author:
- first_name: María
full_name: Rubio, María
last_name: Rubio
- first_name: Ko
full_name: Matsui, Ko
last_name: Matsui
- first_name: Yugo
full_name: Fukazawa, Yugo
last_name: Fukazawa
- first_name: Naomi
full_name: Kamasawa, Naomi
last_name: Kamasawa
- first_name: Harumi
full_name: Harada, Harumi
id: 2E55CDF2-F248-11E8-B48F-1D18A9856A87
last_name: Harada
orcid: 0000-0001-7429-7896
- first_name: Makoto
full_name: Itakura, Makoto
last_name: Itakura
- first_name: Elek
full_name: Molnár, Elek
last_name: Molnár
- first_name: Manabu
full_name: Abe, Manabu
last_name: Abe
- first_name: Kenji
full_name: Sakimura, Kenji
last_name: Sakimura
- first_name: Ryuichi
full_name: Shigemoto, Ryuichi
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
citation:
ama: Rubio M, Matsui K, Fukazawa Y, et al. The number and distribution of AMPA receptor
channels containing fast kinetic GluA3 and GluA4 subunits at auditory nerve synapses
depend on the target cells. Brain Structure and Function. 2017;222(8):3375-3393.
doi:10.1007/s00429-017-1408-0
apa: Rubio, M., Matsui, K., Fukazawa, Y., Kamasawa, N., Harada, H., Itakura, M.,
… Shigemoto, R. (2017). The number and distribution of AMPA receptor channels
containing fast kinetic GluA3 and GluA4 subunits at auditory nerve synapses depend
on the target cells. Brain Structure and Function. Springer. https://doi.org/10.1007/s00429-017-1408-0
chicago: Rubio, María, Ko Matsui, Yugo Fukazawa, Naomi Kamasawa, Harumi Harada,
Makoto Itakura, Elek Molnár, Manabu Abe, Kenji Sakimura, and Ryuichi Shigemoto.
“The Number and Distribution of AMPA Receptor Channels Containing Fast Kinetic
GluA3 and GluA4 Subunits at Auditory Nerve Synapses Depend on the Target Cells.”
Brain Structure and Function. Springer, 2017. https://doi.org/10.1007/s00429-017-1408-0.
ieee: M. Rubio et al., “The number and distribution of AMPA receptor channels
containing fast kinetic GluA3 and GluA4 subunits at auditory nerve synapses depend
on the target cells,” Brain Structure and Function, vol. 222, no. 8. Springer,
pp. 3375–3393, 2017.
ista: Rubio M, Matsui K, Fukazawa Y, Kamasawa N, Harada H, Itakura M, Molnár E,
Abe M, Sakimura K, Shigemoto R. 2017. The number and distribution of AMPA receptor
channels containing fast kinetic GluA3 and GluA4 subunits at auditory nerve synapses
depend on the target cells. Brain Structure and Function. 222(8), 3375–3393.
mla: Rubio, María, et al. “The Number and Distribution of AMPA Receptor Channels
Containing Fast Kinetic GluA3 and GluA4 Subunits at Auditory Nerve Synapses Depend
on the Target Cells.” Brain Structure and Function, vol. 222, no. 8, Springer,
2017, pp. 3375–93, doi:10.1007/s00429-017-1408-0.
short: M. Rubio, K. Matsui, Y. Fukazawa, N. Kamasawa, H. Harada, M. Itakura, E.
Molnár, M. Abe, K. Sakimura, R. Shigemoto, Brain Structure and Function 222 (2017)
3375–3393.
date_created: 2018-12-11T11:48:14Z
date_published: 2017-11-01T00:00:00Z
date_updated: 2023-09-27T14:14:51Z
day: '01'
ddc:
- '571'
department:
- _id: RySh
doi: 10.1007/s00429-017-1408-0
external_id:
isi:
- '000414761700002'
file:
- access_level: open_access
checksum: 73787a22507de8fb585bb598e1418ca7
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:10:20Z
date_updated: 2020-07-14T12:47:56Z
file_id: '4806'
file_name: IST-2017-881-v1+1_s00429-017-1408-0.pdf
file_size: 4011126
relation: main_file
file_date_updated: 2020-07-14T12:47:56Z
has_accepted_license: '1'
intvolume: ' 222'
isi: 1
issue: '8'
language:
- iso: eng
month: '11'
oa: 1
oa_version: Published Version
page: 3375 - 3393
publication: Brain Structure and Function
publication_identifier:
issn:
- '18632653'
publication_status: published
publisher: Springer
publist_id: '6932'
pubrep_id: '881'
quality_controlled: '1'
scopus_import: '1'
status: public
title: The number and distribution of AMPA receptor channels containing fast kinetic
GluA3 and GluA4 subunits at auditory nerve synapses depend on the target cells
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 222
year: '2017'
...
---
_id: '7360'
abstract:
- lang: eng
text: Inflammation, which is a highly regulated host response against danger signals,
may be harmful if it is excessive and deregulated. Ideally, anti-inflammatory
therapy should autonomously commence as soon as possible after the onset of inflammation,
should be controllable by a physician, and should not systemically block beneficial
immune response in the long term. We describe a genetically encoded anti-inflammatory
mammalian cell device based on a modular engineered genetic circuit comprising
a sensor, an amplifier, a “thresholder” to restrict activation of a positive-feedback
loop, a combination of advanced clinically used biopharmaceutical proteins, and
orthogonal regulatory elements that linked modules into the functional device.
This genetic circuit was autonomously activated by inflammatory signals, including
endogenous cecal ligation and puncture (CLP)-induced inflammation in mice and
serum from a systemic juvenile idiopathic arthritis (sIJA) patient, and could
be reset externally by a chemical signal. The microencapsulated anti-inflammatory
device significantly reduced the pathology in dextran sodium sulfate (DSS)-induced
acute murine colitis, demonstrating a synthetic immunological approach for autonomous
anti-inflammatory therapy.
article_processing_charge: No
article_type: original
author:
- first_name: Anže
full_name: Smole, Anže
last_name: Smole
- first_name: Duško
full_name: Lainšček, Duško
last_name: Lainšček
- first_name: Urban
full_name: Bezeljak, Urban
id: 2A58201A-F248-11E8-B48F-1D18A9856A87
last_name: Bezeljak
orcid: 0000-0003-1365-5631
- first_name: Simon
full_name: Horvat, Simon
last_name: Horvat
- first_name: Roman
full_name: Jerala, Roman
last_name: Jerala
citation:
ama: Smole A, Lainšček D, Bezeljak U, Horvat S, Jerala R. A synthetic mammalian
therapeutic gene circuit for sensing and suppressing inflammation. Molecular
Therapy. 2017;25(1):102-119. doi:10.1016/j.ymthe.2016.10.005
apa: Smole, A., Lainšček, D., Bezeljak, U., Horvat, S., & Jerala, R. (2017).
A synthetic mammalian therapeutic gene circuit for sensing and suppressing inflammation.
Molecular Therapy. Elsevier. https://doi.org/10.1016/j.ymthe.2016.10.005
chicago: Smole, Anže, Duško Lainšček, Urban Bezeljak, Simon Horvat, and Roman Jerala.
“A Synthetic Mammalian Therapeutic Gene Circuit for Sensing and Suppressing Inflammation.”
Molecular Therapy. Elsevier, 2017. https://doi.org/10.1016/j.ymthe.2016.10.005.
ieee: A. Smole, D. Lainšček, U. Bezeljak, S. Horvat, and R. Jerala, “A synthetic
mammalian therapeutic gene circuit for sensing and suppressing inflammation,”
Molecular Therapy, vol. 25, no. 1. Elsevier, pp. 102–119, 2017.
ista: Smole A, Lainšček D, Bezeljak U, Horvat S, Jerala R. 2017. A synthetic mammalian
therapeutic gene circuit for sensing and suppressing inflammation. Molecular Therapy.
25(1), 102–119.
mla: Smole, Anže, et al. “A Synthetic Mammalian Therapeutic Gene Circuit for Sensing
and Suppressing Inflammation.” Molecular Therapy, vol. 25, no. 1, Elsevier,
2017, pp. 102–19, doi:10.1016/j.ymthe.2016.10.005.
short: A. Smole, D. Lainšček, U. Bezeljak, S. Horvat, R. Jerala, Molecular Therapy
25 (2017) 102–119.
date_created: 2020-01-25T15:55:39Z
date_published: 2017-01-01T00:00:00Z
date_updated: 2021-01-12T08:13:14Z
day: '01'
ddc:
- '570'
department:
- _id: MaLo
doi: 10.1016/j.ymthe.2016.10.005
external_id:
pmid:
- '28129106'
file:
- access_level: open_access
checksum: ea8b1b28606dd1edab7379ba4fa3641f
content_type: application/pdf
creator: dernst
date_created: 2020-03-03T10:55:13Z
date_updated: 2020-07-14T12:47:56Z
file_id: '7561'
file_name: 2017_MolecularTherapy_Smole.pdf
file_size: 3404806
relation: main_file
file_date_updated: 2020-07-14T12:47:56Z
has_accepted_license: '1'
intvolume: ' 25'
issue: '1'
language:
- iso: eng
license: https://creativecommons.org/licenses/by-nc-nd/4.0/
month: '01'
oa: 1
oa_version: Published Version
page: 102-119
pmid: 1
publication: Molecular Therapy
publication_identifier:
issn:
- 1525-0016
publication_status: published
publisher: Elsevier
quality_controlled: '1'
status: public
title: A synthetic mammalian therapeutic gene circuit for sensing and suppressing
inflammation
tmp:
image: /images/cc_by_nc_nd.png
legal_code_url: https://creativecommons.org/licenses/by-nc-nd/4.0/legalcode
name: Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International
(CC BY-NC-ND 4.0)
short: CC BY-NC-ND (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 25
year: '2017'
...
---
_id: '737'
abstract:
- lang: eng
text: We generalize Brazas’ topology on the fundamental group to the whole universal
path space X˜ i.e., to the set of homotopy classes of all based paths. We develop
basic properties of the new notion and provide a complete comparison of the obtained
topology with the established topologies, in particular with the Lasso topology
and the CO topology, i.e., the topology that is induced by the compact-open topology.
It turns out that the new topology is the finest topology contained in the CO
topology, for which the action of the fundamental group on the universal path
space is a continuous group action.
article_processing_charge: No
author:
- first_name: Ziga
full_name: Virk, Ziga
id: 2E36B656-F248-11E8-B48F-1D18A9856A87
last_name: Virk
- first_name: Andreas
full_name: Zastrow, Andreas
last_name: Zastrow
citation:
ama: Virk Z, Zastrow A. A new topology on the universal path space. Topology
and its Applications. 2017;231:186-196. doi:10.1016/j.topol.2017.09.015
apa: Virk, Z., & Zastrow, A. (2017). A new topology on the universal path space.
Topology and Its Applications. Elsevier. https://doi.org/10.1016/j.topol.2017.09.015
chicago: Virk, Ziga, and Andreas Zastrow. “A New Topology on the Universal Path
Space.” Topology and Its Applications. Elsevier, 2017. https://doi.org/10.1016/j.topol.2017.09.015.
ieee: Z. Virk and A. Zastrow, “A new topology on the universal path space,” Topology
and its Applications, vol. 231. Elsevier, pp. 186–196, 2017.
ista: Virk Z, Zastrow A. 2017. A new topology on the universal path space. Topology
and its Applications. 231, 186–196.
mla: Virk, Ziga, and Andreas Zastrow. “A New Topology on the Universal Path Space.”
Topology and Its Applications, vol. 231, Elsevier, 2017, pp. 186–96, doi:10.1016/j.topol.2017.09.015.
short: Z. Virk, A. Zastrow, Topology and Its Applications 231 (2017) 186–196.
date_created: 2018-12-11T11:48:14Z
date_published: 2017-11-01T00:00:00Z
date_updated: 2023-09-27T12:53:01Z
day: '01'
department:
- _id: HeEd
doi: 10.1016/j.topol.2017.09.015
external_id:
isi:
- '000413889100012'
intvolume: ' 231'
isi: 1
language:
- iso: eng
month: '11'
oa_version: None
page: 186 - 196
publication: Topology and its Applications
publication_identifier:
issn:
- '01668641'
publication_status: published
publisher: Elsevier
publist_id: '6930'
quality_controlled: '1'
scopus_import: '1'
status: public
title: A new topology on the universal path space
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 231
year: '2017'
...
---
_id: '739'
abstract:
- lang: eng
text: We study the norm approximation to the Schrödinger dynamics of N bosons in
with an interaction potential of the form . Assuming that in the initial state
the particles outside of the condensate form a quasi-free state with finite kinetic
energy, we show that in the large N limit, the fluctuations around the condensate
can be effectively described using Bogoliubov approximation for all . The range
of β is expected to be optimal for this large class of initial states.
article_processing_charge: No
author:
- first_name: Phan
full_name: Nam, Phan
id: 404092F4-F248-11E8-B48F-1D18A9856A87
last_name: Nam
- first_name: Marcin M
full_name: Napiórkowski, Marcin M
id: 4197AD04-F248-11E8-B48F-1D18A9856A87
last_name: Napiórkowski
citation:
ama: Nam P, Napiórkowski MM. A note on the validity of Bogoliubov correction to
mean field dynamics. Journal de Mathématiques Pures et Appliquées. 2017;108(5):662-688.
doi:10.1016/j.matpur.2017.05.013
apa: Nam, P., & Napiórkowski, M. M. (2017). A note on the validity of Bogoliubov
correction to mean field dynamics. Journal de Mathématiques Pures et Appliquées.
Elsevier. https://doi.org/10.1016/j.matpur.2017.05.013
chicago: Nam, Phan, and Marcin M Napiórkowski. “A Note on the Validity of Bogoliubov
Correction to Mean Field Dynamics.” Journal de Mathématiques Pures et Appliquées.
Elsevier, 2017. https://doi.org/10.1016/j.matpur.2017.05.013.
ieee: P. Nam and M. M. Napiórkowski, “A note on the validity of Bogoliubov correction
to mean field dynamics,” Journal de Mathématiques Pures et Appliquées,
vol. 108, no. 5. Elsevier, pp. 662–688, 2017.
ista: Nam P, Napiórkowski MM. 2017. A note on the validity of Bogoliubov correction
to mean field dynamics. Journal de Mathématiques Pures et Appliquées. 108(5),
662–688.
mla: Nam, Phan, and Marcin M. Napiórkowski. “A Note on the Validity of Bogoliubov
Correction to Mean Field Dynamics.” Journal de Mathématiques Pures et Appliquées,
vol. 108, no. 5, Elsevier, 2017, pp. 662–88, doi:10.1016/j.matpur.2017.05.013.
short: P. Nam, M.M. Napiórkowski, Journal de Mathématiques Pures et Appliquées 108
(2017) 662–688.
date_created: 2018-12-11T11:48:15Z
date_published: 2017-11-01T00:00:00Z
date_updated: 2023-09-27T12:52:07Z
day: '01'
department:
- _id: RoSe
doi: 10.1016/j.matpur.2017.05.013
external_id:
isi:
- '000414113600003'
intvolume: ' 108'
isi: 1
issue: '5'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://arxiv.org/abs/1604.05240
month: '11'
oa: 1
oa_version: Submitted Version
page: 662 - 688
project:
- _id: 25C878CE-B435-11E9-9278-68D0E5697425
call_identifier: FWF
grant_number: P27533_N27
name: Structure of the Excitation Spectrum for Many-Body Quantum Systems
publication: Journal de Mathématiques Pures et Appliquées
publication_identifier:
issn:
- '00217824'
publication_status: published
publisher: Elsevier
publist_id: '6928'
quality_controlled: '1'
scopus_import: '1'
status: public
title: A note on the validity of Bogoliubov correction to mean field dynamics
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 108
year: '2017'
...
---
_id: '740'
abstract:
- lang: eng
text: 'Developments in bioengineering and molecular biology have introduced a palette
of genetically encoded probes for identification of specific cell populations
in electron microscopy. These probes can be targeted to distinct cellular compartments,
rendering them electron dense through a subsequent chemical reaction. These electron
densities strongly increase the local contrast in samples prepared for electron
microscopy, allowing three major advances in ultrastructural mapping of circuits:
genetic identification of circuit components, targeted imaging of regions of interest
and automated analysis of the tagged circuits. Together, the gains from these
advances can decrease the time required for the analysis of targeted circuit motifs
by over two orders of magnitude. These genetic encoded tags for electron microscopy
promise to simplify the analysis of circuit motifs and become a central tool for
structure‐function studies of synaptic connections in the brain. We review the
current state‐of‐the‐art with an emphasis on connectomics, the quantitative analysis
of neuronal structures and motifs.'
article_number: e288
article_processing_charge: No
article_type: original
author:
- first_name: Ryuichi
full_name: Shigemoto, Ryuichi
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Maximilian A
full_name: Jösch, Maximilian A
id: 2BD278E6-F248-11E8-B48F-1D18A9856A87
last_name: Jösch
orcid: 0000-0002-3937-1330
citation:
ama: Shigemoto R, Jösch MA. The genetic encoded toolbox for electron microscopy
and connectomics. WIREs Developmental Biology. 2017;6(6). doi:10.1002/wdev.288
apa: Shigemoto, R., & Jösch, M. A. (2017). The genetic encoded toolbox for electron
microscopy and connectomics. WIREs Developmental Biology. Wiley-Blackwell.
https://doi.org/10.1002/wdev.288
chicago: Shigemoto, Ryuichi, and Maximilian A Jösch. “The Genetic Encoded Toolbox
for Electron Microscopy and Connectomics.” WIREs Developmental Biology.
Wiley-Blackwell, 2017. https://doi.org/10.1002/wdev.288.
ieee: R. Shigemoto and M. A. Jösch, “The genetic encoded toolbox for electron microscopy
and connectomics,” WIREs Developmental Biology, vol. 6, no. 6. Wiley-Blackwell,
2017.
ista: Shigemoto R, Jösch MA. 2017. The genetic encoded toolbox for electron microscopy
and connectomics. WIREs Developmental Biology. 6(6), e288.
mla: Shigemoto, Ryuichi, and Maximilian A. Jösch. “The Genetic Encoded Toolbox for
Electron Microscopy and Connectomics.” WIREs Developmental Biology, vol.
6, no. 6, e288, Wiley-Blackwell, 2017, doi:10.1002/wdev.288.
short: R. Shigemoto, M.A. Jösch, WIREs Developmental Biology 6 (2017).
date_created: 2018-12-11T11:48:15Z
date_published: 2017-08-11T00:00:00Z
date_updated: 2023-09-27T12:51:41Z
day: '11'
ddc:
- '570'
department:
- _id: RySh
- _id: MaJö
doi: 10.1002/wdev.288
external_id:
isi:
- '000412827400005'
pmid:
- '28800674'
file:
- access_level: open_access
checksum: a9370f27b1591773b7a0de299bc81c8c
content_type: application/pdf
creator: dernst
date_created: 2019-11-19T07:36:18Z
date_updated: 2020-07-14T12:47:57Z
file_id: '7045'
file_name: 2017_WIREs_Shigemoto.pdf
file_size: 1647787
relation: main_file
file_date_updated: 2020-07-14T12:47:57Z
has_accepted_license: '1'
intvolume: ' 6'
isi: 1
issue: '6'
language:
- iso: eng
month: '08'
oa: 1
oa_version: Submitted Version
pmid: 1
publication: WIREs Developmental Biology
publication_identifier:
issn:
- '17597684'
publication_status: published
publisher: Wiley-Blackwell
publist_id: '6927'
quality_controlled: '1'
scopus_import: '1'
status: public
title: The genetic encoded toolbox for electron microscopy and connectomics
tmp:
image: /images/cc_by_nc.png
legal_code_url: https://creativecommons.org/licenses/by-nc/4.0/legalcode
name: Creative Commons Attribution-NonCommercial 4.0 International (CC BY-NC 4.0)
short: CC BY-NC (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 6
year: '2017'
...
---
_id: '741'
abstract:
- lang: eng
text: We prove that a system of N fermions interacting with an additional particle
via point interactions is stable if the ratio of the mass of the additional particle
to the one of the fermions is larger than some critical m*. The value of m* is
independent of N and turns out to be less than 1. This fact has important implications
for the stability of the unitary Fermi gas. We also characterize the domain of
the Hamiltonian of this model, and establish the validity of the Tan relations
for all wave functions in the domain.
article_processing_charge: No
author:
- first_name: Thomas
full_name: Moser, Thomas
id: 2B5FC9A4-F248-11E8-B48F-1D18A9856A87
last_name: Moser
- first_name: Robert
full_name: Seiringer, Robert
id: 4AFD0470-F248-11E8-B48F-1D18A9856A87
last_name: Seiringer
orcid: 0000-0002-6781-0521
citation:
ama: Moser T, Seiringer R. Stability of a fermionic N+1 particle system with point
interactions. Communications in Mathematical Physics. 2017;356(1):329-355.
doi:10.1007/s00220-017-2980-0
apa: Moser, T., & Seiringer, R. (2017). Stability of a fermionic N+1 particle
system with point interactions. Communications in Mathematical Physics.
Springer. https://doi.org/10.1007/s00220-017-2980-0
chicago: Moser, Thomas, and Robert Seiringer. “Stability of a Fermionic N+1 Particle
System with Point Interactions.” Communications in Mathematical Physics.
Springer, 2017. https://doi.org/10.1007/s00220-017-2980-0.
ieee: T. Moser and R. Seiringer, “Stability of a fermionic N+1 particle system with
point interactions,” Communications in Mathematical Physics, vol. 356,
no. 1. Springer, pp. 329–355, 2017.
ista: Moser T, Seiringer R. 2017. Stability of a fermionic N+1 particle system with
point interactions. Communications in Mathematical Physics. 356(1), 329–355.
mla: Moser, Thomas, and Robert Seiringer. “Stability of a Fermionic N+1 Particle
System with Point Interactions.” Communications in Mathematical Physics,
vol. 356, no. 1, Springer, 2017, pp. 329–55, doi:10.1007/s00220-017-2980-0.
short: T. Moser, R. Seiringer, Communications in Mathematical Physics 356 (2017)
329–355.
date_created: 2018-12-11T11:48:15Z
date_published: 2017-11-01T00:00:00Z
date_updated: 2023-09-27T12:34:15Z
day: '01'
ddc:
- '539'
department:
- _id: RoSe
doi: 10.1007/s00220-017-2980-0
ec_funded: 1
external_id:
isi:
- '000409821300010'
file:
- access_level: open_access
checksum: 0fd9435400f91e9b3c5346319a2d24e3
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:10:50Z
date_updated: 2020-07-14T12:47:57Z
file_id: '4841'
file_name: IST-2017-880-v1+1_s00220-017-2980-0.pdf
file_size: 952639
relation: main_file
file_date_updated: 2020-07-14T12:47:57Z
has_accepted_license: '1'
intvolume: ' 356'
isi: 1
issue: '1'
language:
- iso: eng
month: '11'
oa: 1
oa_version: Published Version
page: 329 - 355
project:
- _id: 25C6DC12-B435-11E9-9278-68D0E5697425
call_identifier: H2020
grant_number: '694227'
name: Analysis of quantum many-body systems
- _id: 25C878CE-B435-11E9-9278-68D0E5697425
call_identifier: FWF
grant_number: P27533_N27
name: Structure of the Excitation Spectrum for Many-Body Quantum Systems
publication: Communications in Mathematical Physics
publication_identifier:
issn:
- '00103616'
publication_status: published
publisher: Springer
publist_id: '6926'
pubrep_id: '880'
quality_controlled: '1'
related_material:
record:
- id: '52'
relation: dissertation_contains
status: public
scopus_import: '1'
status: public
title: Stability of a fermionic N+1 particle system with point interactions
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 356
year: '2017'
...
---
_id: '743'
abstract:
- lang: eng
text: "This special issue of the Journal on Formal Methods in System Design is dedicated
to Prof. Helmut Veith, who unexpectedly passed away in March 2016. Helmut Veith
was a brilliant researcher, inspiring collaborator, passionate mentor, generous
friend, and valued member of the formal methods community. Helmut was not only
known for his numerous and influential contributions in the field of automated
verification (most prominently his work on Counterexample-Guided Abstraction Refinement
[1,2]), but also for his untiring and passionate efforts for the logic community:
he co-organized the Vienna Summer of Logic (an event comprising twelve conferences
and numerous workshops which attracted thousands of researchers from all over
the world), he initiated the Vienna Center for Logic and Algorithms (which promotes
international collaboration on logic and algorithms and organizes outreach events
such as the LogicLounge), and he coordinated the Doctoral Program on Logical Methods
in Computer Science at TU Wien (currently educating more than 40 doctoral students)
and a National Research Network on Rigorous Systems Engineering (uniting fifteen
researchers in Austria to address the challenge of building reliable and safe
computer\r\nsystems). With his enthusiasm and commitment, Helmut completely reshaped
the Austrian research landscape in the field of logic and verification in his
few years as a full professor at TU Wien."
article_processing_charge: No
author:
- first_name: Georg
full_name: Gottlob, Georg
last_name: Gottlob
- first_name: Thomas A
full_name: Henzinger, Thomas A
id: 40876CD8-F248-11E8-B48F-1D18A9856A87
last_name: Henzinger
orcid: 0000−0002−2985−7724
- first_name: Georg
full_name: Weißenbacher, Georg
last_name: Weißenbacher
citation:
ama: Gottlob G, Henzinger TA, Weißenbacher G. Preface of the special issue in memoriam
Helmut Veith. Formal Methods in System Design. 2017;51(2):267-269. doi:10.1007/s10703-017-0307-6
apa: Gottlob, G., Henzinger, T. A., & Weißenbacher, G. (2017). Preface of the
special issue in memoriam Helmut Veith. Formal Methods in System Design.
Springer. https://doi.org/10.1007/s10703-017-0307-6
chicago: Gottlob, Georg, Thomas A Henzinger, and Georg Weißenbacher. “Preface of
the Special Issue in Memoriam Helmut Veith.” Formal Methods in System Design.
Springer, 2017. https://doi.org/10.1007/s10703-017-0307-6.
ieee: G. Gottlob, T. A. Henzinger, and G. Weißenbacher, “Preface of the special
issue in memoriam Helmut Veith,” Formal Methods in System Design, vol.
51, no. 2. Springer, pp. 267–269, 2017.
ista: Gottlob G, Henzinger TA, Weißenbacher G. 2017. Preface of the special issue
in memoriam Helmut Veith. Formal Methods in System Design. 51(2), 267–269.
mla: Gottlob, Georg, et al. “Preface of the Special Issue in Memoriam Helmut Veith.”
Formal Methods in System Design, vol. 51, no. 2, Springer, 2017, pp. 267–69,
doi:10.1007/s10703-017-0307-6.
short: G. Gottlob, T.A. Henzinger, G. Weißenbacher, Formal Methods in System Design
51 (2017) 267–269.
date_created: 2018-12-11T11:48:16Z
date_published: 2017-11-14T00:00:00Z
date_updated: 2023-09-27T12:29:29Z
day: '14'
department:
- _id: ToHe
doi: 10.1007/s10703-017-0307-6
external_id:
isi:
- '000415615600001'
intvolume: ' 51'
isi: 1
issue: '2'
language:
- iso: eng
month: '11'
oa_version: None
page: 267 - 269
publication: Formal Methods in System Design
publication_status: published
publisher: Springer
publist_id: '6924'
quality_controlled: '1'
status: public
title: Preface of the special issue in memoriam Helmut Veith
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 51
year: '2017'
...
---
_id: '744'
abstract:
- lang: eng
text: In evolutionary game theory interactions between individuals are often assumed
obligatory. However, in many real-life situations, individuals can decide to opt
out of an interaction depending on the information they have about the opponent.
We consider a simple evolutionary game theoretic model to study such a scenario,
where at each encounter between two individuals the type of the opponent (cooperator/defector)
is known with some probability, and where each individual either accepts or opts
out of the interaction. If the type of the opponent is unknown, a trustful individual
accepts the interaction, whereas a suspicious individual opts out of the interaction.
If either of the two individuals opt out both individuals remain without an interaction.
We show that in the prisoners dilemma optional interactions along with suspicious
behaviour facilitates the emergence of trustful cooperation.
article_processing_charge: No
article_type: original
author:
- first_name: Tadeas
full_name: Priklopil, Tadeas
id: 3C869AA0-F248-11E8-B48F-1D18A9856A87
last_name: Priklopil
- first_name: Krishnendu
full_name: Chatterjee, Krishnendu
id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
last_name: Chatterjee
orcid: 0000-0002-4561-241X
- first_name: Martin
full_name: Nowak, Martin
last_name: Nowak
citation:
ama: Priklopil T, Chatterjee K, Nowak M. Optional interactions and suspicious behaviour
facilitates trustful cooperation in prisoners dilemma. Journal of Theoretical
Biology. 2017;433:64-72. doi:10.1016/j.jtbi.2017.08.025
apa: Priklopil, T., Chatterjee, K., & Nowak, M. (2017). Optional interactions
and suspicious behaviour facilitates trustful cooperation in prisoners dilemma.
Journal of Theoretical Biology. Elsevier. https://doi.org/10.1016/j.jtbi.2017.08.025
chicago: Priklopil, Tadeas, Krishnendu Chatterjee, and Martin Nowak. “Optional Interactions
and Suspicious Behaviour Facilitates Trustful Cooperation in Prisoners Dilemma.”
Journal of Theoretical Biology. Elsevier, 2017. https://doi.org/10.1016/j.jtbi.2017.08.025.
ieee: T. Priklopil, K. Chatterjee, and M. Nowak, “Optional interactions and suspicious
behaviour facilitates trustful cooperation in prisoners dilemma,” Journal
of Theoretical Biology, vol. 433. Elsevier, pp. 64–72, 2017.
ista: Priklopil T, Chatterjee K, Nowak M. 2017. Optional interactions and suspicious
behaviour facilitates trustful cooperation in prisoners dilemma. Journal of Theoretical
Biology. 433, 64–72.
mla: Priklopil, Tadeas, et al. “Optional Interactions and Suspicious Behaviour Facilitates
Trustful Cooperation in Prisoners Dilemma.” Journal of Theoretical Biology,
vol. 433, Elsevier, 2017, pp. 64–72, doi:10.1016/j.jtbi.2017.08.025.
short: T. Priklopil, K. Chatterjee, M. Nowak, Journal of Theoretical Biology 433
(2017) 64–72.
date_created: 2018-12-11T11:48:16Z
date_published: 2017-11-21T00:00:00Z
date_updated: 2023-09-27T12:29:02Z
day: '21'
ddc:
- '000'
- '570'
department:
- _id: KrCh
doi: 10.1016/j.jtbi.2017.08.025
ec_funded: 1
external_id:
isi:
- '000412039800007'
pmid:
- '28867224'
file:
- access_level: open_access
checksum: 4b43af1615ebf1a861840cb03d8a320c
content_type: application/pdf
creator: dernst
date_created: 2019-11-19T07:57:39Z
date_updated: 2020-07-14T12:47:58Z
file_id: '7047'
file_name: 2017_JournTheoretBio_Priklopil.pdf
file_size: 537323
relation: main_file
file_date_updated: 2020-07-14T12:47:58Z
has_accepted_license: '1'
intvolume: ' 433'
isi: 1
language:
- iso: eng
month: '11'
oa: 1
oa_version: Submitted Version
page: 64 - 72
pmid: 1
project:
- _id: 25681D80-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '291734'
name: International IST Postdoc Fellowship Programme
- _id: 2581B60A-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '279307'
name: 'Quantitative Graph Games: Theory and Applications'
publication: ' Journal of Theoretical Biology'
publication_identifier:
issn:
- '00225193'
publication_status: published
publisher: Elsevier
publist_id: '6923'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Optional interactions and suspicious behaviour facilitates trustful cooperation
in prisoners dilemma
tmp:
image: /images/cc_by_nc_nd.png
legal_code_url: https://creativecommons.org/licenses/by-nc-nd/4.0/legalcode
name: Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International
(CC BY-NC-ND 4.0)
short: CC BY-NC-ND (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 433
year: '2017'
...