[{"article_number":"104203","_id":"724","date_updated":"2021-01-12T08:12:35Z","abstract":[{"text":"We investigate the stationary and dynamical behavior of an Anderson localized chain coupled to a single central bound state. Although this coupling partially dilutes the Anderson localized peaks towards nearly resonant sites, the most weight of the original peaks remains unchanged. This leads to multifractal wave functions with a frozen spectrum of fractal dimensions, which is characteristic for localized phases in models with power-law hopping. Using a perturbative approach we identify two different dynamical regimes. At weak couplings to the central site, the transport of particles and information is logarithmic in time, a feature usually attributed to many-body localization. We connect such transport to the persistence of the Poisson statistics of level spacings in parts of the spectrum. In contrast, at stronger couplings the level repulsion is established in the entire spectrum, the problem can be mapped to the Fano resonance, and the transport is ballistic.","lang":"eng"}],"issue":"10","year":"2017","month":"09","status":"public","oa":1,"publication_identifier":{"issn":["24699950"]},"oa_version":"Submitted Version","date_published":"2017-09-13T00:00:00Z","publication_status":"published","doi":"10.1103/PhysRevB.96.104203","intvolume":"        96","citation":{"ama":"Hetterich D, Serbyn M, Domínguez F, Pollmann F, Trauzettel B. Noninteracting central site model localization and logarithmic entanglement growth. <i>Physical Review B</i>. 2017;96(10). doi:<a href=\"https://doi.org/10.1103/PhysRevB.96.104203\">10.1103/PhysRevB.96.104203</a>","ieee":"D. Hetterich, M. Serbyn, F. Domínguez, F. Pollmann, and B. Trauzettel, “Noninteracting central site model localization and logarithmic entanglement growth,” <i>Physical Review B</i>, vol. 96, no. 10. American Physical Society, 2017.","short":"D. Hetterich, M. Serbyn, F. Domínguez, F. Pollmann, B. Trauzettel, Physical Review B 96 (2017).","chicago":"Hetterich, Daniel, Maksym Serbyn, Fernando Domínguez, Frank Pollmann, and Björn Trauzettel. “Noninteracting Central Site Model Localization and Logarithmic Entanglement Growth.” <i>Physical Review B</i>. American Physical Society, 2017. <a href=\"https://doi.org/10.1103/PhysRevB.96.104203\">https://doi.org/10.1103/PhysRevB.96.104203</a>.","mla":"Hetterich, Daniel, et al. “Noninteracting Central Site Model Localization and Logarithmic Entanglement Growth.” <i>Physical Review B</i>, vol. 96, no. 10, 104203, American Physical Society, 2017, doi:<a href=\"https://doi.org/10.1103/PhysRevB.96.104203\">10.1103/PhysRevB.96.104203</a>.","apa":"Hetterich, D., Serbyn, M., Domínguez, F., Pollmann, F., &#38; Trauzettel, B. (2017). Noninteracting central site model localization and logarithmic entanglement growth. <i>Physical Review B</i>. American Physical Society. <a href=\"https://doi.org/10.1103/PhysRevB.96.104203\">https://doi.org/10.1103/PhysRevB.96.104203</a>","ista":"Hetterich D, Serbyn M, Domínguez F, Pollmann F, Trauzettel B. 2017. Noninteracting central site model localization and logarithmic entanglement growth. Physical Review B. 96(10), 104203."},"main_file_link":[{"url":"https://arxiv.org/abs/1701.02744","open_access":"1"}],"scopus_import":1,"author":[{"full_name":"Hetterich, Daniel","last_name":"Hetterich","first_name":"Daniel"},{"last_name":"Serbyn","id":"47809E7E-F248-11E8-B48F-1D18A9856A87","orcid":"0000-0002-2399-5827","first_name":"Maksym","full_name":"Serbyn, Maksym"},{"full_name":"Domínguez, Fernando","last_name":"Domínguez","first_name":"Fernando"},{"first_name":"Frank","last_name":"Pollmann","full_name":"Pollmann, Frank"},{"first_name":"Björn","last_name":"Trauzettel","full_name":"Trauzettel, Björn"}],"acknowledgement":"We  would  like  to  thank  Dmitry  Abanin,  Christophe  De\r\nBeule,  Joel  Moore,  Romain  Vasseur,  and  Norman  Yao  for\r\nmany  stimulating  discussions.  Financial  support  has  been\r\nprovided  by  the  Deutsche  Forschungsgemeinschaft  (DFG)\r\nvia Grant No. TR950/8-1, SFB 1170 “ToCoTronics” and the\r\nENB  Graduate  School  on  Topological  Insulators.  M.S.  was\r\nsupported by Gordon and Betty Moore Foundation’s EPiQS\r\nInitiative through Grant No. GBMF4307. F.P. acknowledges\r\nsupport from the DFG Research Unit FOR 1807 through Grant\r\nNo. PO 1370/2-1.","day":"13","publisher":"American Physical Society","user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","title":"Noninteracting central site model localization and logarithmic entanglement growth","type":"journal_article","language":[{"iso":"eng"}],"department":[{"_id":"MaSe"}],"date_created":"2018-12-11T11:48:09Z","publication":"Physical Review B","volume":96,"quality_controlled":"1","publist_id":"6955"},{"status":"public","oa":1,"publication_identifier":{"issn":["00278424"]},"month":"09","date_updated":"2021-01-12T08:12:36Z","abstract":[{"lang":"eng","text":"Individual computations and social interactions underlying collective behavior in groups of animals are of great ethological, behavioral, and theoretical interest. While complex individual behaviors have successfully been parsed into small dictionaries of stereotyped behavioral modes, studies of collective behavior largely ignored these findings; instead, their focus was on inferring single, mode-independent social interaction rules that reproduced macroscopic and often qualitative features of group behavior. Here, we bring these two approaches together to predict individual swimming patterns of adult zebrafish in a group. We show that fish alternate between an “active” mode, in which they are sensitive to the swimming patterns of conspecifics, and a “passive” mode, where they ignore them. Using a model that accounts for these two modes explicitly, we predict behaviors of individual fish with high accuracy, outperforming previous approaches that assumed a single continuous computation by individuals and simple metric or topological weighing of neighbors’ behavior. At the group level, switching between active and passive modes is uncorrelated among fish, but correlated directional swimming behavior still emerges. Our quantitative approach for studying complex, multi-modal individual behavior jointly with emergent group behavior is readily extensible to additional behavioral modes and their neural correlates as well as to other species."}],"issue":"38","year":"2017","_id":"725","scopus_import":1,"main_file_link":[{"url":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5617265/","open_access":"1"}],"intvolume":"       114","citation":{"short":"R. Harpaz, G. Tkačik, E. Schneidman, PNAS 114 (2017) 10149–10154.","ama":"Harpaz R, Tkačik G, Schneidman E. Discrete modes of social information processing predict individual behavior of fish in a group. <i>PNAS</i>. 2017;114(38):10149-10154. doi:<a href=\"https://doi.org/10.1073/pnas.1703817114\">10.1073/pnas.1703817114</a>","ieee":"R. Harpaz, G. Tkačik, and E. Schneidman, “Discrete modes of social information processing predict individual behavior of fish in a group,” <i>PNAS</i>, vol. 114, no. 38. National Academy of Sciences, pp. 10149–10154, 2017.","mla":"Harpaz, Roy, et al. “Discrete Modes of Social Information Processing Predict Individual Behavior of Fish in a Group.” <i>PNAS</i>, vol. 114, no. 38, National Academy of Sciences, 2017, pp. 10149–54, doi:<a href=\"https://doi.org/10.1073/pnas.1703817114\">10.1073/pnas.1703817114</a>.","ista":"Harpaz R, Tkačik G, Schneidman E. 2017. Discrete modes of social information processing predict individual behavior of fish in a group. PNAS. 114(38), 10149–10154.","apa":"Harpaz, R., Tkačik, G., &#38; Schneidman, E. (2017). Discrete modes of social information processing predict individual behavior of fish in a group. <i>PNAS</i>. National Academy of Sciences. <a href=\"https://doi.org/10.1073/pnas.1703817114\">https://doi.org/10.1073/pnas.1703817114</a>","chicago":"Harpaz, Roy, Gašper Tkačik, and Elad Schneidman. “Discrete Modes of Social Information Processing Predict Individual Behavior of Fish in a Group.” <i>PNAS</i>. National Academy of Sciences, 2017. <a href=\"https://doi.org/10.1073/pnas.1703817114\">https://doi.org/10.1073/pnas.1703817114</a>."},"publication_status":"published","doi":"10.1073/pnas.1703817114","oa_version":"Submitted Version","date_published":"2017-09-19T00:00:00Z","publisher":"National Academy of Sciences","user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","title":"Discrete modes of social information processing predict individual behavior of fish in a group","pmid":1,"day":"19","author":[{"full_name":"Harpaz, Roy","last_name":"Harpaz","first_name":"Roy"},{"last_name":"Tkacik","id":"3D494DCA-F248-11E8-B48F-1D18A9856A87","first_name":"Gasper","orcid":"0000-0002-6699-1455","full_name":"Tkacik, Gasper"},{"last_name":"Schneidman","first_name":"Elad","full_name":"Schneidman, Elad"}],"publist_id":"6953","external_id":{"pmid":["28874581"]},"volume":114,"quality_controlled":"1","date_created":"2018-12-11T11:48:10Z","page":"10149 - 10154","publication":"PNAS","type":"journal_article","language":[{"iso":"eng"}],"department":[{"_id":"GaTk"}]},{"month":"09","publication_identifier":{"issn":["00928674"]},"oa":1,"status":"public","_id":"726","year":"2017","abstract":[{"lang":"eng","text":"The morphogenesis of branched organs remains a subject of abiding interest. Although much is known about the underlying signaling pathways, it remains unclear how macroscopic features of branched organs, including their size, network topology, and spatial patterning, are encoded. Here, we show that, in mouse mammary gland, kidney, and human prostate, these features can be explained quantitatively within a single unifying framework of branching and annihilating random walks. Based on quantitative analyses of large-scale organ reconstructions and proliferation kinetics measurements, we propose that morphogenesis follows from the proliferative activity of equipotent tips that stochastically branch and randomly explore their environment but compete neutrally for space, becoming proliferatively inactive when in proximity with neighboring ducts. These results show that complex branched epithelial structures develop as a self-organized process, reliant upon a strikingly simple but generic rule, without recourse to a rigid and deterministic sequence of genetically programmed events."}],"issue":"1","date_updated":"2023-09-28T11:34:17Z","citation":{"short":"E.B. Hannezo, C. Scheele, M. Moad, N. Drogo, R. Heer, R. Sampogna, J. Van Rheenen, B. Simons, Cell 171 (2017) 242–255.","ieee":"E. B. Hannezo <i>et al.</i>, “A unifying theory of branching morphogenesis,” <i>Cell</i>, vol. 171, no. 1. Cell Press, pp. 242–255, 2017.","ama":"Hannezo EB, Scheele C, Moad M, et al. A unifying theory of branching morphogenesis. <i>Cell</i>. 2017;171(1):242-255. doi:<a href=\"https://doi.org/10.1016/j.cell.2017.08.026\">10.1016/j.cell.2017.08.026</a>","apa":"Hannezo, E. B., Scheele, C., Moad, M., Drogo, N., Heer, R., Sampogna, R., … Simons, B. (2017). A unifying theory of branching morphogenesis. <i>Cell</i>. Cell Press. <a href=\"https://doi.org/10.1016/j.cell.2017.08.026\">https://doi.org/10.1016/j.cell.2017.08.026</a>","mla":"Hannezo, Edouard B., et al. “A Unifying Theory of Branching Morphogenesis.” <i>Cell</i>, vol. 171, no. 1, Cell Press, 2017, pp. 242–55, doi:<a href=\"https://doi.org/10.1016/j.cell.2017.08.026\">10.1016/j.cell.2017.08.026</a>.","ista":"Hannezo EB, Scheele C, Moad M, Drogo N, Heer R, Sampogna R, Van Rheenen J, Simons B. 2017. A unifying theory of branching morphogenesis. Cell. 171(1), 242–255.","chicago":"Hannezo, Edouard B, Colinda Scheele, Mohammad Moad, Nicholas Drogo, Rakesh Heer, Rosemary Sampogna, Jacco Van Rheenen, and Benjamin Simons. “A Unifying Theory of Branching Morphogenesis.” <i>Cell</i>. Cell Press, 2017. <a href=\"https://doi.org/10.1016/j.cell.2017.08.026\">https://doi.org/10.1016/j.cell.2017.08.026</a>."},"intvolume":"       171","ddc":["539"],"scopus_import":"1","file_date_updated":"2020-07-14T12:47:55Z","date_published":"2017-09-21T00:00:00Z","oa_version":"Published Version","doi":"10.1016/j.cell.2017.08.026","publication_status":"published","license":"https://creativecommons.org/licenses/by/4.0/","title":"A unifying theory of branching morphogenesis","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"Cell Press","file":[{"file_size":12670204,"relation":"main_file","content_type":"application/pdf","date_created":"2018-12-12T10:11:17Z","file_name":"IST-2017-883-v1+1_PIIS0092867417309510.pdf","date_updated":"2020-07-14T12:47:55Z","access_level":"open_access","file_id":"4870","checksum":"7a036d93a9e2e597af9bb504d6133aca","creator":"system"}],"author":[{"id":"3A9DB764-F248-11E8-B48F-1D18A9856A87","last_name":"Hannezo","first_name":"Edouard B","orcid":"0000-0001-6005-1561","full_name":"Hannezo, Edouard B"},{"first_name":"Colinda","last_name":"Scheele","full_name":"Scheele, Colinda"},{"first_name":"Mohammad","last_name":"Moad","full_name":"Moad, Mohammad"},{"first_name":"Nicholas","last_name":"Drogo","full_name":"Drogo, Nicholas"},{"full_name":"Heer, Rakesh","last_name":"Heer","first_name":"Rakesh"},{"full_name":"Sampogna, Rosemary","last_name":"Sampogna","first_name":"Rosemary"},{"full_name":"Van Rheenen, Jacco","first_name":"Jacco","last_name":"Van Rheenen"},{"full_name":"Simons, Benjamin","first_name":"Benjamin","last_name":"Simons"}],"isi":1,"day":"21","tmp":{"short":"CC BY (4.0)","image":"/images/cc_by.png","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode"},"volume":171,"quality_controlled":"1","external_id":{"isi":["000411331800024"]},"pubrep_id":"883","article_processing_charge":"No","publist_id":"6952","department":[{"_id":"EdHa"}],"language":[{"iso":"eng"}],"type":"journal_article","publication":"Cell","page":"242 - 255","date_created":"2018-12-11T11:48:10Z","has_accepted_license":"1"},{"publisher":"Cell Press","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","title":"Load adaptation of lamellipodial actin networks","project":[{"grant_number":"LS13-029","_id":"25AD6156-B435-11E9-9278-68D0E5697425","name":"Modeling of Polarization and Motility of Leukocytes in Three-Dimensional Environments"},{"call_identifier":"FP7","name":"Cytoskeletal force generation and force transduction of migrating leukocytes (EU)","grant_number":"281556","_id":"25A603A2-B435-11E9-9278-68D0E5697425"}],"author":[{"full_name":"Mueller, Jan","first_name":"Jan","last_name":"Mueller"},{"id":"4BFB7762-F248-11E8-B48F-1D18A9856A87","last_name":"Szep","first_name":"Gregory","full_name":"Szep, Gregory"},{"full_name":"Nemethova, Maria","id":"34E27F1C-F248-11E8-B48F-1D18A9856A87","last_name":"Nemethova","first_name":"Maria"},{"first_name":"Ingrid","last_name":"De Vries","id":"4C7D837E-F248-11E8-B48F-1D18A9856A87","full_name":"De Vries, Ingrid"},{"first_name":"Arnon","last_name":"Lieber","full_name":"Lieber, Arnon"},{"full_name":"Winkler, Christoph","first_name":"Christoph","last_name":"Winkler"},{"full_name":"Kruse, Karsten","first_name":"Karsten","last_name":"Kruse"},{"full_name":"Small, John","last_name":"Small","first_name":"John"},{"full_name":"Schmeiser, Christian","last_name":"Schmeiser","first_name":"Christian"},{"first_name":"Kinneret","last_name":"Keren","full_name":"Keren, Kinneret"},{"full_name":"Hauschild, Robert","id":"4E01D6B4-F248-11E8-B48F-1D18A9856A87","last_name":"Hauschild","first_name":"Robert","orcid":"0000-0001-9843-3522"},{"first_name":"Michael K","orcid":"0000-0002-6620-9179","last_name":"Sixt","id":"41E9FBEA-F248-11E8-B48F-1D18A9856A87","full_name":"Sixt, Michael K"}],"isi":1,"day":"21","external_id":{"isi":["000411331800020"]},"volume":171,"quality_controlled":"1","publist_id":"6951","article_processing_charge":"No","type":"journal_article","language":[{"iso":"eng"}],"department":[{"_id":"MiSi"},{"_id":"Bio"}],"date_created":"2018-12-11T11:48:10Z","page":"188 - 200","publication":"Cell","month":"09","ec_funded":1,"status":"public","publication_identifier":{"issn":["00928674"]},"acknowledged_ssus":[{"_id":"ScienComp"}],"_id":"727","date_updated":"2023-09-28T11:33:49Z","issue":"1","abstract":[{"text":"Actin filaments polymerizing against membranes power endocytosis, vesicular traffic, and cell motility. In vitro reconstitution studies suggest that the structure and the dynamics of actin networks respond to mechanical forces. We demonstrate that lamellipodial actin of migrating cells responds to mechanical load when membrane tension is modulated. In a steady state, migrating cell filaments assume the canonical dendritic geometry, defined by Arp2/3-generated 70° branch points. Increased tension triggers a dense network with a broadened range of angles, whereas decreased tension causes a shift to a sparse configuration dominated by filaments growing perpendicularly to the plasma membrane. We show that these responses emerge from the geometry of branched actin: when load per filament decreases, elongation speed increases and perpendicular filaments gradually outcompete others because they polymerize the shortest distance to the membrane, where they are protected from capping. This network-intrinsic geometrical adaptation mechanism tunes protrusive force in response to mechanical load.","lang":"eng"}],"year":"2017","intvolume":"       171","citation":{"ieee":"J. Mueller <i>et al.</i>, “Load adaptation of lamellipodial actin networks,” <i>Cell</i>, vol. 171, no. 1. Cell Press, pp. 188–200, 2017.","ama":"Mueller J, Szep G, Nemethova M, et al. Load adaptation of lamellipodial actin networks. <i>Cell</i>. 2017;171(1):188-200. doi:<a href=\"https://doi.org/10.1016/j.cell.2017.07.051\">10.1016/j.cell.2017.07.051</a>","short":"J. Mueller, G. Szep, M. Nemethova, I. de Vries, A. Lieber, C. Winkler, K. Kruse, J. Small, C. Schmeiser, K. Keren, R. Hauschild, M.K. Sixt, Cell 171 (2017) 188–200.","apa":"Mueller, J., Szep, G., Nemethova, M., de Vries, I., Lieber, A., Winkler, C., … Sixt, M. K. (2017). Load adaptation of lamellipodial actin networks. <i>Cell</i>. Cell Press. <a href=\"https://doi.org/10.1016/j.cell.2017.07.051\">https://doi.org/10.1016/j.cell.2017.07.051</a>","mla":"Mueller, Jan, et al. “Load Adaptation of Lamellipodial Actin Networks.” <i>Cell</i>, vol. 171, no. 1, Cell Press, 2017, pp. 188–200, doi:<a href=\"https://doi.org/10.1016/j.cell.2017.07.051\">10.1016/j.cell.2017.07.051</a>.","ista":"Mueller J, Szep G, Nemethova M, de Vries I, Lieber A, Winkler C, Kruse K, Small J, Schmeiser C, Keren K, Hauschild R, Sixt MK. 2017. Load adaptation of lamellipodial actin networks. Cell. 171(1), 188–200.","chicago":"Mueller, Jan, Gregory Szep, Maria Nemethova, Ingrid de Vries, Arnon Lieber, Christoph Winkler, Karsten Kruse, et al. “Load Adaptation of Lamellipodial Actin Networks.” <i>Cell</i>. Cell Press, 2017. <a href=\"https://doi.org/10.1016/j.cell.2017.07.051\">https://doi.org/10.1016/j.cell.2017.07.051</a>."},"scopus_import":"1","oa_version":"None","date_published":"2017-09-21T00:00:00Z","publication_status":"published","doi":"10.1016/j.cell.2017.07.051"},{"title":"Coordination of morphogenesis and cell fate specification in development","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"Cell Press","isi":1,"author":[{"full_name":"Chan, Chii","last_name":"Chan","first_name":"Chii"},{"id":"39427864-F248-11E8-B48F-1D18A9856A87","last_name":"Heisenberg","orcid":"0000-0002-0912-4566","first_name":"Carl-Philipp J","full_name":"Heisenberg, Carl-Philipp J"},{"last_name":"Hiiragi","first_name":"Takashi","full_name":"Hiiragi, Takashi"}],"day":"18","volume":27,"quality_controlled":"1","external_id":{"isi":["000411581800019"]},"article_processing_charge":"No","publist_id":"6949","department":[{"_id":"CaHe"}],"language":[{"iso":"eng"}],"type":"journal_article","publication":"Current Biology","page":"R1024 - R1035","date_created":"2018-12-11T11:48:11Z","month":"09","publication_identifier":{"issn":["09609822"]},"status":"public","_id":"728","year":"2017","abstract":[{"text":"During animal development, cell-fate-specific changes in gene expression can modify the material properties of a tissue and drive tissue morphogenesis. While mechanistic insights into the genetic control of tissue-shaping events are beginning to emerge, how tissue morphogenesis and mechanics can reciprocally impact cell-fate specification remains relatively unexplored. Here we review recent findings reporting how multicellular morphogenetic events and their underlying mechanical forces can feed back into gene regulatory pathways to specify cell fate. We further discuss emerging techniques that allow for the direct measurement and manipulation of mechanical signals in vivo, offering unprecedented access to study mechanotransduction during development. Examination of the mechanical control of cell fate during tissue morphogenesis will pave the way to an integrated understanding of the design principles that underlie robust tissue patterning in embryonic development.","lang":"eng"}],"issue":"18","date_updated":"2023-09-28T11:33:21Z","intvolume":"        27","citation":{"short":"C. Chan, C.-P.J. Heisenberg, T. Hiiragi, Current Biology 27 (2017) R1024–R1035.","ama":"Chan C, Heisenberg C-PJ, Hiiragi T. Coordination of morphogenesis and cell fate specification in development. <i>Current Biology</i>. 2017;27(18):R1024-R1035. doi:<a href=\"https://doi.org/10.1016/j.cub.2017.07.010\">10.1016/j.cub.2017.07.010</a>","ieee":"C. Chan, C.-P. J. Heisenberg, and T. Hiiragi, “Coordination of morphogenesis and cell fate specification in development,” <i>Current Biology</i>, vol. 27, no. 18. Cell Press, pp. R1024–R1035, 2017.","chicago":"Chan, Chii, Carl-Philipp J Heisenberg, and Takashi Hiiragi. “Coordination of Morphogenesis and Cell Fate Specification in Development.” <i>Current Biology</i>. Cell Press, 2017. <a href=\"https://doi.org/10.1016/j.cub.2017.07.010\">https://doi.org/10.1016/j.cub.2017.07.010</a>.","ista":"Chan C, Heisenberg C-PJ, Hiiragi T. 2017. Coordination of morphogenesis and cell fate specification in development. Current Biology. 27(18), R1024–R1035.","apa":"Chan, C., Heisenberg, C.-P. J., &#38; Hiiragi, T. (2017). Coordination of morphogenesis and cell fate specification in development. <i>Current Biology</i>. Cell Press. <a href=\"https://doi.org/10.1016/j.cub.2017.07.010\">https://doi.org/10.1016/j.cub.2017.07.010</a>","mla":"Chan, Chii, et al. “Coordination of Morphogenesis and Cell Fate Specification in Development.” <i>Current Biology</i>, vol. 27, no. 18, Cell Press, 2017, pp. R1024–35, doi:<a href=\"https://doi.org/10.1016/j.cub.2017.07.010\">10.1016/j.cub.2017.07.010</a>."},"scopus_import":"1","date_published":"2017-09-18T00:00:00Z","oa_version":"None","doi":"10.1016/j.cub.2017.07.010","publication_status":"published"},{"_id":"729","year":"2017","date_updated":"2023-09-28T11:32:49Z","abstract":[{"lang":"eng","text":"The cellular mechanisms allowing tissues to efficiently regenerate are not fully understood. In this issue of Developmental Cell, Cao et al. (2017)) discover that during zebrafish heart regeneration, epicardial cells at the leading edge of regenerating tissue undergo endoreplication, possibly due to increased tissue tension, thereby boosting their regenerative capacity."}],"issue":"6","month":"01","publication_identifier":{"issn":["15345807"]},"status":"public","date_published":"2017-01-01T00:00:00Z","oa_version":"None","publication_status":"published","doi":"10.1016/j.devcel.2017.09.008","citation":{"ama":"Spiro ZP, Heisenberg C-PJ. Regeneration tensed up polyploidy takes the lead. <i>Developmental Cell</i>. 2017;42(6):559-560. doi:<a href=\"https://doi.org/10.1016/j.devcel.2017.09.008\">10.1016/j.devcel.2017.09.008</a>","ieee":"Z. P. Spiro and C.-P. J. Heisenberg, “Regeneration tensed up polyploidy takes the lead,” <i>Developmental Cell</i>, vol. 42, no. 6. Cell Press, pp. 559–560, 2017.","short":"Z.P. Spiro, C.-P.J. Heisenberg, Developmental Cell 42 (2017) 559–560.","chicago":"Spiro, Zoltan P, and Carl-Philipp J Heisenberg. “Regeneration Tensed up Polyploidy Takes the Lead.” <i>Developmental Cell</i>. Cell Press, 2017. <a href=\"https://doi.org/10.1016/j.devcel.2017.09.008\">https://doi.org/10.1016/j.devcel.2017.09.008</a>.","apa":"Spiro, Z. P., &#38; Heisenberg, C.-P. J. (2017). Regeneration tensed up polyploidy takes the lead. <i>Developmental Cell</i>. Cell Press. <a href=\"https://doi.org/10.1016/j.devcel.2017.09.008\">https://doi.org/10.1016/j.devcel.2017.09.008</a>","ista":"Spiro ZP, Heisenberg C-PJ. 2017. Regeneration tensed up polyploidy takes the lead. Developmental Cell. 42(6), 559–560.","mla":"Spiro, Zoltan P., and Carl-Philipp J. Heisenberg. “Regeneration Tensed up Polyploidy Takes the Lead.” <i>Developmental Cell</i>, vol. 42, no. 6, Cell Press, 2017, pp. 559–60, doi:<a href=\"https://doi.org/10.1016/j.devcel.2017.09.008\">10.1016/j.devcel.2017.09.008</a>."},"intvolume":"        42","scopus_import":"1","author":[{"full_name":"Spiro, Zoltan P","last_name":"Spiro","id":"426AD026-F248-11E8-B48F-1D18A9856A87","first_name":"Zoltan P"},{"first_name":"Carl-Philipp J","orcid":"0000-0002-0912-4566","last_name":"Heisenberg","id":"39427864-F248-11E8-B48F-1D18A9856A87","full_name":"Heisenberg, Carl-Philipp J"}],"isi":1,"day":"01","title":"Regeneration tensed up polyploidy takes the lead","publisher":"Cell Press","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","language":[{"iso":"eng"}],"department":[{"_id":"CaHe"}],"type":"journal_article","page":"559 - 560","date_created":"2018-12-11T11:48:11Z","publication":"Developmental Cell","external_id":{"isi":["000411582800003"]},"quality_controlled":"1","volume":42,"publist_id":"6948","article_processing_charge":"No"},{"oa_version":"None","date_published":"2017-10-01T00:00:00Z","publication_status":"published","doi":"10.1016/j.conb.2017.08.001","intvolume":"        46","citation":{"ieee":"C. Savin and G. Tkačik, “Maximum entropy models as a tool for building precise neural controls,” <i>Current Opinion in Neurobiology</i>, vol. 46. Elsevier, pp. 120–126, 2017.","ama":"Savin C, Tkačik G. Maximum entropy models as a tool for building precise neural controls. <i>Current Opinion in Neurobiology</i>. 2017;46:120-126. doi:<a href=\"https://doi.org/10.1016/j.conb.2017.08.001\">10.1016/j.conb.2017.08.001</a>","short":"C. Savin, G. Tkačik, Current Opinion in Neurobiology 46 (2017) 120–126.","mla":"Savin, Cristina, and Gašper Tkačik. “Maximum Entropy Models as a Tool for Building Precise Neural Controls.” <i>Current Opinion in Neurobiology</i>, vol. 46, Elsevier, 2017, pp. 120–26, doi:<a href=\"https://doi.org/10.1016/j.conb.2017.08.001\">10.1016/j.conb.2017.08.001</a>.","apa":"Savin, C., &#38; Tkačik, G. (2017). Maximum entropy models as a tool for building precise neural controls. <i>Current Opinion in Neurobiology</i>. Elsevier. <a href=\"https://doi.org/10.1016/j.conb.2017.08.001\">https://doi.org/10.1016/j.conb.2017.08.001</a>","ista":"Savin C, Tkačik G. 2017. Maximum entropy models as a tool for building precise neural controls. Current Opinion in Neurobiology. 46, 120–126.","chicago":"Savin, Cristina, and Gašper Tkačik. “Maximum Entropy Models as a Tool for Building Precise Neural Controls.” <i>Current Opinion in Neurobiology</i>. Elsevier, 2017. <a href=\"https://doi.org/10.1016/j.conb.2017.08.001\">https://doi.org/10.1016/j.conb.2017.08.001</a>."},"scopus_import":"1","_id":"730","date_updated":"2023-09-28T11:32:22Z","abstract":[{"lang":"eng","text":"Neural responses are highly structured, with population activity restricted to a small subset of the astronomical range of possible activity patterns. Characterizing these statistical regularities is important for understanding circuit computation, but challenging in practice. Here we review recent approaches based on the maximum entropy principle used for quantifying collective behavior in neural activity. We highlight recent models that capture population-level statistics of neural data, yielding insights into the organization of the neural code and its biological substrate. Furthermore, the MaxEnt framework provides a general recipe for constructing surrogate ensembles that preserve aspects of the data, but are otherwise maximally unstructured. This idea can be used to generate a hierarchy of controls against which rigorous statistical tests are possible."}],"year":"2017","month":"10","ec_funded":1,"status":"public","publication_identifier":{"issn":["09594388"]},"type":"journal_article","language":[{"iso":"eng"}],"department":[{"_id":"GaTk"}],"page":"120 - 126","date_created":"2018-12-11T11:48:11Z","publication":"Current Opinion in Neurobiology","external_id":{"isi":["000416196400016"]},"quality_controlled":"1","volume":46,"publist_id":"6943","article_processing_charge":"No","project":[{"call_identifier":"FP7","name":"International IST Postdoc Fellowship Programme","_id":"25681D80-B435-11E9-9278-68D0E5697425","grant_number":"291734"}],"author":[{"last_name":"Savin","id":"3933349E-F248-11E8-B48F-1D18A9856A87","first_name":"Cristina","full_name":"Savin, Cristina"},{"last_name":"Tkacik","id":"3D494DCA-F248-11E8-B48F-1D18A9856A87","first_name":"Gasper","orcid":"0000-0002-6699-1455","full_name":"Tkacik, Gasper"}],"isi":1,"day":"01","publisher":"Elsevier","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","title":"Maximum entropy models as a tool for building precise neural controls"},{"day":"11","year":"2017","date_updated":"2021-01-12T08:12:57Z","issue":"411","abstract":[{"text":"Genetic variations in the oxytocin receptor gene affect patients with ASD and ADHD differently.","lang":"eng"}],"_id":"731","article_number":"eaap8168","author":[{"full_name":"Novarino, Gaia","id":"3E57A680-F248-11E8-B48F-1D18A9856A87","last_name":"Novarino","orcid":"0000-0002-7673-7178","first_name":"Gaia"}],"title":"The science of love in ASD and ADHD","publication_identifier":{"issn":["19466234"]},"publisher":"American Association for the Advancement of Science","status":"public","user_id":"3E5EF7F0-F248-11E8-B48F-1D18A9856A87","month":"10","date_created":"2018-12-11T11:48:12Z","publication":"Science Translational Medicine","publication_status":"published","doi":"10.1126/scitranslmed.aap8168","language":[{"iso":"eng"}],"date_published":"2017-10-11T00:00:00Z","department":[{"_id":"GaNo"}],"type":"journal_article","oa_version":"None","scopus_import":1,"publist_id":"6938","quality_controlled":"1","volume":9,"intvolume":"         9","citation":{"ama":"Novarino G. The science of love in ASD and ADHD. <i>Science Translational Medicine</i>. 2017;9(411). doi:<a href=\"https://doi.org/10.1126/scitranslmed.aap8168\">10.1126/scitranslmed.aap8168</a>","ieee":"G. Novarino, “The science of love in ASD and ADHD,” <i>Science Translational Medicine</i>, vol. 9, no. 411. American Association for the Advancement of Science, 2017.","short":"G. Novarino, Science Translational Medicine 9 (2017).","chicago":"Novarino, Gaia. “The Science of Love in ASD and ADHD.” <i>Science Translational Medicine</i>. American Association for the Advancement of Science, 2017. <a href=\"https://doi.org/10.1126/scitranslmed.aap8168\">https://doi.org/10.1126/scitranslmed.aap8168</a>.","mla":"Novarino, Gaia. “The Science of Love in ASD and ADHD.” <i>Science Translational Medicine</i>, vol. 9, no. 411, eaap8168, American Association for the Advancement of Science, 2017, doi:<a href=\"https://doi.org/10.1126/scitranslmed.aap8168\">10.1126/scitranslmed.aap8168</a>.","ista":"Novarino G. 2017. The science of love in ASD and ADHD. Science Translational Medicine. 9(411), eaap8168.","apa":"Novarino, G. (2017). The science of love in ASD and ADHD. <i>Science Translational Medicine</i>. American Association for the Advancement of Science. <a href=\"https://doi.org/10.1126/scitranslmed.aap8168\">https://doi.org/10.1126/scitranslmed.aap8168</a>"}},{"title":"Co-founding ant queens prevent disease by performing prophylactic undertaking behaviour","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"BioMed Central","file":[{"file_name":"IST-2017-882-v1+1_12862_2017_Article_1062.pdf","date_updated":"2020-07-14T12:47:55Z","date_created":"2018-12-12T10:17:18Z","creator":"system","checksum":"3e24a2cfd48f49f7b3643d08d30fb480","file_id":"5271","access_level":"open_access","content_type":"application/pdf","relation":"main_file","file_size":949857}],"isi":1,"author":[{"full_name":"Pull, Christopher","last_name":"Pull","id":"3C7F4840-F248-11E8-B48F-1D18A9856A87","orcid":"0000-0003-1122-3982","first_name":"Christopher"},{"last_name":"Cremer","id":"2F64EC8C-F248-11E8-B48F-1D18A9856A87","first_name":"Sylvia","orcid":"0000-0002-2193-3868","full_name":"Cremer, Sylvia"}],"project":[{"call_identifier":"FP7","name":"Social Vaccination in Ant Colonies: from Individual Mechanisms to Society Effects","grant_number":"243071","_id":"25DC711C-B435-11E9-9278-68D0E5697425"}],"day":"13","tmp":{"short":"CC BY (4.0)","image":"/images/cc_by.png","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode"},"related_material":{"record":[{"status":"public","relation":"dissertation_contains","id":"819"}]},"quality_controlled":"1","volume":17,"external_id":{"isi":["000412816800001"]},"article_processing_charge":"Yes","article_type":"original","pubrep_id":"882","publist_id":"6937","department":[{"_id":"SyCr"}],"language":[{"iso":"eng"}],"type":"journal_article","publication":"BMC Evolutionary Biology","date_created":"2018-12-11T11:48:12Z","has_accepted_license":"1","ec_funded":1,"month":"10","publication_identifier":{"issn":["14712148"]},"oa":1,"status":"public","_id":"732","article_number":"219","year":"2017","abstract":[{"text":"Background: Social insects form densely crowded societies in environments with high pathogen loads, but have evolved collective defences that mitigate the impact of disease. However, colony-founding queens lack this protection and suffer high rates of mortality. The impact of pathogens may be exacerbated in species where queens found colonies together, as healthy individuals may contract pathogens from infectious co-founders. Therefore, we tested whether ant queens avoid founding colonies with pathogen-exposed conspecifics and how they might limit disease transmission from infectious individuals. Results: Using Lasius Niger queens and a naturally infecting fungal pathogen Metarhizium brunneum, we observed that queens were equally likely to found colonies with another pathogen-exposed or sham-treated queen. However, when one queen died, the surviving individual performed biting, burial and removal of the corpse. These undertaking behaviours were performed prophylactically, i.e. targeted equally towards non-infected and infected corpses, as well as carried out before infected corpses became infectious. Biting and burial reduced the risk of the queens contracting and dying from disease from an infectious corpse of a dead co-foundress. Conclusions: We show that co-founding ant queens express undertaking behaviours that, in mature colonies, are performed exclusively by workers. Such infection avoidance behaviours act before the queens can contract the disease and will therefore improve the overall chance of colony founding success in ant queens.","lang":"eng"}],"issue":"1","date_updated":"2023-09-28T11:31:32Z","citation":{"chicago":"Pull, Christopher, and Sylvia Cremer. “Co-Founding Ant Queens Prevent Disease by Performing Prophylactic Undertaking Behaviour.” <i>BMC Evolutionary Biology</i>. BioMed Central, 2017. <a href=\"https://doi.org/10.1186/s12862-017-1062-4\">https://doi.org/10.1186/s12862-017-1062-4</a>.","apa":"Pull, C., &#38; Cremer, S. (2017). Co-founding ant queens prevent disease by performing prophylactic undertaking behaviour. <i>BMC Evolutionary Biology</i>. BioMed Central. <a href=\"https://doi.org/10.1186/s12862-017-1062-4\">https://doi.org/10.1186/s12862-017-1062-4</a>","ista":"Pull C, Cremer S. 2017. Co-founding ant queens prevent disease by performing prophylactic undertaking behaviour. BMC Evolutionary Biology. 17(1), 219.","mla":"Pull, Christopher, and Sylvia Cremer. “Co-Founding Ant Queens Prevent Disease by Performing Prophylactic Undertaking Behaviour.” <i>BMC Evolutionary Biology</i>, vol. 17, no. 1, 219, BioMed Central, 2017, doi:<a href=\"https://doi.org/10.1186/s12862-017-1062-4\">10.1186/s12862-017-1062-4</a>.","ama":"Pull C, Cremer S. Co-founding ant queens prevent disease by performing prophylactic undertaking behaviour. <i>BMC Evolutionary Biology</i>. 2017;17(1). doi:<a href=\"https://doi.org/10.1186/s12862-017-1062-4\">10.1186/s12862-017-1062-4</a>","ieee":"C. Pull and S. Cremer, “Co-founding ant queens prevent disease by performing prophylactic undertaking behaviour,” <i>BMC Evolutionary Biology</i>, vol. 17, no. 1. BioMed Central, 2017.","short":"C. Pull, S. Cremer, BMC Evolutionary Biology 17 (2017)."},"intvolume":"        17","ddc":["576","592"],"file_date_updated":"2020-07-14T12:47:55Z","scopus_import":"1","date_published":"2017-10-13T00:00:00Z","oa_version":"Published Version","doi":"10.1186/s12862-017-1062-4","publication_status":"published"},{"article_processing_charge":"No","publist_id":"6935","volume":319,"quality_controlled":"1","external_id":{"isi":["000412150400010"]},"publication":"Advances in Mathematics","page":"251 - 291","date_created":"2018-12-11T11:48:13Z","type":"journal_article","department":[{"_id":"LaEr"}],"language":[{"iso":"eng"}],"user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"Academic Press","title":"Convergence rate for spectral distribution of addition of random matrices","acknowledgement":"Partially supported by ERC Advanced Grant RANMAT No. 338804, Hong Kong RGC grant ECS 26301517, and the Göran Gustafsson Foundation","day":"15","isi":1,"author":[{"orcid":"0000-0003-3036-1475","first_name":"Zhigang","last_name":"Bao","id":"442E6A6C-F248-11E8-B48F-1D18A9856A87","full_name":"Bao, Zhigang"},{"full_name":"Erdös, László","first_name":"László","orcid":"0000-0001-5366-9603","id":"4DBD5372-F248-11E8-B48F-1D18A9856A87","last_name":"Erdös"},{"last_name":"Schnelli","id":"434AD0AE-F248-11E8-B48F-1D18A9856A87","first_name":"Kevin","orcid":"0000-0003-0954-3231","full_name":"Schnelli, Kevin"}],"project":[{"grant_number":"338804","_id":"258DCDE6-B435-11E9-9278-68D0E5697425","name":"Random matrices, universality and disordered quantum systems","call_identifier":"FP7"}],"main_file_link":[{"url":"https://arxiv.org/abs/1606.03076","open_access":"1"}],"scopus_import":"1","citation":{"ieee":"Z. Bao, L. Erdös, and K. Schnelli, “Convergence rate for spectral distribution of addition of random matrices,” <i>Advances in Mathematics</i>, vol. 319. Academic Press, pp. 251–291, 2017.","ama":"Bao Z, Erdös L, Schnelli K. Convergence rate for spectral distribution of addition of random matrices. <i>Advances in Mathematics</i>. 2017;319:251-291. doi:<a href=\"https://doi.org/10.1016/j.aim.2017.08.028\">10.1016/j.aim.2017.08.028</a>","short":"Z. Bao, L. Erdös, K. Schnelli, Advances in Mathematics 319 (2017) 251–291.","mla":"Bao, Zhigang, et al. “Convergence Rate for Spectral Distribution of Addition of Random Matrices.” <i>Advances in Mathematics</i>, vol. 319, Academic Press, 2017, pp. 251–91, doi:<a href=\"https://doi.org/10.1016/j.aim.2017.08.028\">10.1016/j.aim.2017.08.028</a>.","apa":"Bao, Z., Erdös, L., &#38; Schnelli, K. (2017). Convergence rate for spectral distribution of addition of random matrices. <i>Advances in Mathematics</i>. Academic Press. <a href=\"https://doi.org/10.1016/j.aim.2017.08.028\">https://doi.org/10.1016/j.aim.2017.08.028</a>","ista":"Bao Z, Erdös L, Schnelli K. 2017. Convergence rate for spectral distribution of addition of random matrices. Advances in Mathematics. 319, 251–291.","chicago":"Bao, Zhigang, László Erdös, and Kevin Schnelli. “Convergence Rate for Spectral Distribution of Addition of Random Matrices.” <i>Advances in Mathematics</i>. Academic Press, 2017. <a href=\"https://doi.org/10.1016/j.aim.2017.08.028\">https://doi.org/10.1016/j.aim.2017.08.028</a>."},"intvolume":"       319","doi":"10.1016/j.aim.2017.08.028","publication_status":"published","oa_version":"Submitted Version","date_published":"2017-10-15T00:00:00Z","status":"public","oa":1,"month":"10","ec_funded":1,"abstract":[{"lang":"eng","text":"Let A and B be two N by N deterministic Hermitian matrices and let U be an N by N Haar distributed unitary matrix. It is well known that the spectral distribution of the sum H = A + UBU∗ converges weakly to the free additive convolution of the spectral distributions of A and B, as N tends to infinity. We establish the optimal convergence rate in the bulk of the spectrum."}],"date_updated":"2023-09-28T11:30:42Z","year":"2017","_id":"733"},{"doi":"10.1016/j.tree.2017.08.004","publication_status":"published","date_published":"2017-11-01T00:00:00Z","oa_version":"Submitted Version","ddc":["570"],"scopus_import":"1","file_date_updated":"2020-07-14T12:47:56Z","intvolume":"        32","citation":{"short":"P. Kennedy, G. Baron, B. Qiu, D. Freitak, H. Helantera, E. Hunt, F. Manfredini, T. O’Shea Wheller, S. Patalano, C. Pull, T. Sasaki, D. Taylor, C. Wyatt, S. Sumner, Trends in Ecology and Evolution 32 (2017) 861–872.","ieee":"P. Kennedy <i>et al.</i>, “Deconstructing superorganisms and societies to address big questions in biology,” <i>Trends in Ecology and Evolution</i>, vol. 32, no. 11. Cell Press, pp. 861–872, 2017.","ama":"Kennedy P, Baron G, Qiu B, et al. Deconstructing superorganisms and societies to address big questions in biology. <i>Trends in Ecology and Evolution</i>. 2017;32(11):861-872. doi:<a href=\"https://doi.org/10.1016/j.tree.2017.08.004\">10.1016/j.tree.2017.08.004</a>","chicago":"Kennedy, Patrick, Gemma Baron, Bitao Qiu, Dalial Freitak, Heikki Helantera, Edmund Hunt, Fabio Manfredini, et al. “Deconstructing Superorganisms and Societies to Address Big Questions in Biology.” <i>Trends in Ecology and Evolution</i>. Cell Press, 2017. <a href=\"https://doi.org/10.1016/j.tree.2017.08.004\">https://doi.org/10.1016/j.tree.2017.08.004</a>.","ista":"Kennedy P, Baron G, Qiu B, Freitak D, Helantera H, Hunt E, Manfredini F, O’Shea Wheller T, Patalano S, Pull C, Sasaki T, Taylor D, Wyatt C, Sumner S. 2017. Deconstructing superorganisms and societies to address big questions in biology. Trends in Ecology and Evolution. 32(11), 861–872.","mla":"Kennedy, Patrick, et al. “Deconstructing Superorganisms and Societies to Address Big Questions in Biology.” <i>Trends in Ecology and Evolution</i>, vol. 32, no. 11, Cell Press, 2017, pp. 861–72, doi:<a href=\"https://doi.org/10.1016/j.tree.2017.08.004\">10.1016/j.tree.2017.08.004</a>.","apa":"Kennedy, P., Baron, G., Qiu, B., Freitak, D., Helantera, H., Hunt, E., … Sumner, S. (2017). Deconstructing superorganisms and societies to address big questions in biology. <i>Trends in Ecology and Evolution</i>. Cell Press. <a href=\"https://doi.org/10.1016/j.tree.2017.08.004\">https://doi.org/10.1016/j.tree.2017.08.004</a>"},"year":"2017","abstract":[{"lang":"eng","text":"Social insect societies are long-standing models for understanding social behaviour and evolution. Unlike other advanced biological societies (such as the multicellular body), the component parts of social insect societies can be easily deconstructed and manipulated. Recent methodological and theoretical innovations have exploited this trait to address an expanded range of biological questions. We illustrate the broadening range of biological insight coming from social insect biology with four examples. These new frontiers promote open-minded, interdisciplinary exploration of one of the richest and most complex of biological phenomena: sociality."}],"issue":"11","date_updated":"2023-09-27T14:15:15Z","_id":"734","publication_identifier":{"issn":["01695347"]},"oa":1,"status":"public","month":"11","publication":"Trends in Ecology and Evolution","date_created":"2018-12-11T11:48:13Z","page":"861 - 872","has_accepted_license":"1","department":[{"_id":"SyCr"}],"language":[{"iso":"eng"}],"type":"journal_article","article_type":"original","article_processing_charge":"No","publist_id":"6933","related_material":{"record":[{"relation":"dissertation_contains","status":"public","id":"819"}]},"quality_controlled":"1","volume":32,"external_id":{"isi":["000413231900011"]},"day":"01","file":[{"content_type":"application/pdf","relation":"main_file","file_size":15018382,"file_name":"2017_TrendsEcology_Kennedy.pdf","date_updated":"2020-07-14T12:47:56Z","date_created":"2020-05-14T16:22:27Z","creator":"dernst","file_id":"7842","checksum":"c8f49309ed9436201814fa7153d66a99","access_level":"open_access"}],"isi":1,"author":[{"first_name":"Patrick","last_name":"Kennedy","full_name":"Kennedy, Patrick"},{"full_name":"Baron, Gemma","first_name":"Gemma","last_name":"Baron"},{"full_name":"Qiu, Bitao","last_name":"Qiu","first_name":"Bitao"},{"full_name":"Freitak, Dalial","first_name":"Dalial","last_name":"Freitak"},{"last_name":"Helantera","first_name":"Heikki","full_name":"Helantera, Heikki"},{"first_name":"Edmund","last_name":"Hunt","full_name":"Hunt, Edmund"},{"full_name":"Manfredini, Fabio","last_name":"Manfredini","first_name":"Fabio"},{"full_name":"O'Shea Wheller, Thomas","first_name":"Thomas","last_name":"O'Shea Wheller"},{"full_name":"Patalano, Solenn","last_name":"Patalano","first_name":"Solenn"},{"orcid":"0000-0003-1122-3982","first_name":"Christopher","id":"3C7F4840-F248-11E8-B48F-1D18A9856A87","last_name":"Pull","full_name":"Pull, Christopher"},{"full_name":"Sasaki, Takao","first_name":"Takao","last_name":"Sasaki"},{"full_name":"Taylor, Daisy","last_name":"Taylor","first_name":"Daisy"},{"first_name":"Christopher","last_name":"Wyatt","full_name":"Wyatt, Christopher"},{"full_name":"Sumner, Seirian","first_name":"Seirian","last_name":"Sumner"}],"title":"Deconstructing superorganisms and societies to address big questions in biology","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"Cell Press"},{"page":"198 - 211","date_created":"2018-12-11T11:48:13Z","publication":"Developmental Cell","type":"journal_article","language":[{"iso":"eng"}],"department":[{"_id":"CaHe"},{"_id":"CaGu"},{"_id":"GaTk"}],"publist_id":"6934","article_processing_charge":"No","external_id":{"isi":["000413443700011"]},"quality_controlled":"1","volume":43,"related_material":{"record":[{"id":"961","status":"public","relation":"dissertation_contains"},{"id":"8350","status":"public","relation":"dissertation_contains"}]},"day":"23","project":[{"name":"International IST Postdoc Fellowship Programme","call_identifier":"FP7","grant_number":"291734","_id":"25681D80-B435-11E9-9278-68D0E5697425"},{"name":"Cell segregation in gastrulation: the role of cell fate specification","call_identifier":"FWF","grant_number":"I2058","_id":"252DD2A6-B435-11E9-9278-68D0E5697425"}],"isi":1,"author":[{"orcid":"0000-0003-2676-3367","first_name":"Vanessa","id":"419EECCC-F248-11E8-B48F-1D18A9856A87","last_name":"Barone","full_name":"Barone, Vanessa"},{"full_name":"Lang, Moritz","first_name":"Moritz","last_name":"Lang","id":"29E0800A-F248-11E8-B48F-1D18A9856A87"},{"full_name":"Krens, Gabriel","id":"2B819732-F248-11E8-B48F-1D18A9856A87","last_name":"Krens","first_name":"Gabriel","orcid":"0000-0003-4761-5996"},{"full_name":"Pradhan, Saurabh","last_name":"Pradhan","first_name":"Saurabh"},{"full_name":"Shamipour, Shayan","id":"40B34FE2-F248-11E8-B48F-1D18A9856A87","last_name":"Shamipour","first_name":"Shayan"},{"id":"3BED66BE-F248-11E8-B48F-1D18A9856A87","last_name":"Sako","orcid":"0000-0002-6453-8075","first_name":"Keisuke","full_name":"Sako, Keisuke"},{"full_name":"Sikora, Mateusz K","first_name":"Mateusz K","id":"2F74BCDE-F248-11E8-B48F-1D18A9856A87","last_name":"Sikora"},{"full_name":"Guet, Calin C","last_name":"Guet","id":"47F8433E-F248-11E8-B48F-1D18A9856A87","first_name":"Calin C","orcid":"0000-0001-6220-2052"},{"id":"39427864-F248-11E8-B48F-1D18A9856A87","last_name":"Heisenberg","orcid":"0000-0002-0912-4566","first_name":"Carl-Philipp J","full_name":"Heisenberg, Carl-Philipp J"}],"publisher":"Cell Press","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","title":"An effective feedback loop between cell-cell contact duration and morphogen signaling determines cell fate","publication_status":"published","doi":"10.1016/j.devcel.2017.09.014","oa_version":"None","date_published":"2017-10-23T00:00:00Z","scopus_import":"1","citation":{"chicago":"Barone, Vanessa, Moritz Lang, Gabriel Krens, Saurabh Pradhan, Shayan Shamipour, Keisuke Sako, Mateusz K Sikora, Calin C Guet, and Carl-Philipp J Heisenberg. “An Effective Feedback Loop between Cell-Cell Contact Duration and Morphogen Signaling Determines Cell Fate.” <i>Developmental Cell</i>. Cell Press, 2017. <a href=\"https://doi.org/10.1016/j.devcel.2017.09.014\">https://doi.org/10.1016/j.devcel.2017.09.014</a>.","mla":"Barone, Vanessa, et al. “An Effective Feedback Loop between Cell-Cell Contact Duration and Morphogen Signaling Determines Cell Fate.” <i>Developmental Cell</i>, vol. 43, no. 2, Cell Press, 2017, pp. 198–211, doi:<a href=\"https://doi.org/10.1016/j.devcel.2017.09.014\">10.1016/j.devcel.2017.09.014</a>.","apa":"Barone, V., Lang, M., Krens, G., Pradhan, S., Shamipour, S., Sako, K., … Heisenberg, C.-P. J. (2017). An effective feedback loop between cell-cell contact duration and morphogen signaling determines cell fate. <i>Developmental Cell</i>. Cell Press. <a href=\"https://doi.org/10.1016/j.devcel.2017.09.014\">https://doi.org/10.1016/j.devcel.2017.09.014</a>","ista":"Barone V, Lang M, Krens G, Pradhan S, Shamipour S, Sako K, Sikora MK, Guet CC, Heisenberg C-PJ. 2017. An effective feedback loop between cell-cell contact duration and morphogen signaling determines cell fate. Developmental Cell. 43(2), 198–211.","ieee":"V. Barone <i>et al.</i>, “An effective feedback loop between cell-cell contact duration and morphogen signaling determines cell fate,” <i>Developmental Cell</i>, vol. 43, no. 2. Cell Press, pp. 198–211, 2017.","ama":"Barone V, Lang M, Krens G, et al. An effective feedback loop between cell-cell contact duration and morphogen signaling determines cell fate. <i>Developmental Cell</i>. 2017;43(2):198-211. doi:<a href=\"https://doi.org/10.1016/j.devcel.2017.09.014\">10.1016/j.devcel.2017.09.014</a>","short":"V. Barone, M. Lang, G. Krens, S. Pradhan, S. Shamipour, K. Sako, M.K. Sikora, C.C. Guet, C.-P.J. Heisenberg, Developmental Cell 43 (2017) 198–211."},"intvolume":"        43","date_updated":"2024-03-25T23:30:21Z","issue":"2","abstract":[{"text":"Cell-cell contact formation constitutes an essential step in evolution, leading to the differentiation of specialized cell types. However, remarkably little is known about whether and how the interplay between contact formation and fate specification affects development. Here, we identify a positive feedback loop between cell-cell contact duration, morphogen signaling, and mesendoderm cell-fate specification during zebrafish gastrulation. We show that long-lasting cell-cell contacts enhance the competence of prechordal plate (ppl) progenitor cells to respond to Nodal signaling, required for ppl cell-fate specification. We further show that Nodal signaling promotes ppl cell-cell contact duration, generating a positive feedback loop between ppl cell-cell contact duration and cell-fate specification. Finally, by combining mathematical modeling and experimentation, we show that this feedback determines whether anterior axial mesendoderm cells become ppl or, instead, turn into endoderm. Thus, the interdependent activities of cell-cell signaling and contact formation control fate diversification within the developing embryo.","lang":"eng"}],"year":"2017","_id":"735","status":"public","publication_identifier":{"issn":["15345807"]},"month":"10","ec_funded":1},{"title":"The number and distribution of AMPA receptor channels containing fast kinetic GluA3 and GluA4 subunits at auditory nerve synapses depend on the target cells","publisher":"Springer","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","file":[{"date_created":"2018-12-12T10:10:20Z","date_updated":"2020-07-14T12:47:56Z","file_name":"IST-2017-881-v1+1_s00429-017-1408-0.pdf","access_level":"open_access","file_id":"4806","creator":"system","checksum":"73787a22507de8fb585bb598e1418ca7","file_size":4011126,"content_type":"application/pdf","relation":"main_file"}],"author":[{"full_name":"Rubio, María","last_name":"Rubio","first_name":"María"},{"full_name":"Matsui, Ko","first_name":"Ko","last_name":"Matsui"},{"last_name":"Fukazawa","first_name":"Yugo","full_name":"Fukazawa, Yugo"},{"last_name":"Kamasawa","first_name":"Naomi","full_name":"Kamasawa, Naomi"},{"full_name":"Harada, Harumi","orcid":"0000-0001-7429-7896","first_name":"Harumi","id":"2E55CDF2-F248-11E8-B48F-1D18A9856A87","last_name":"Harada"},{"first_name":"Makoto","last_name":"Itakura","full_name":"Itakura, Makoto"},{"full_name":"Molnár, Elek","first_name":"Elek","last_name":"Molnár"},{"first_name":"Manabu","last_name":"Abe","full_name":"Abe, Manabu"},{"first_name":"Kenji","last_name":"Sakimura","full_name":"Sakimura, Kenji"},{"first_name":"Ryuichi","orcid":"0000-0001-8761-9444","last_name":"Shigemoto","id":"499F3ABC-F248-11E8-B48F-1D18A9856A87","full_name":"Shigemoto, Ryuichi"}],"isi":1,"tmp":{"short":"CC BY (4.0)","image":"/images/cc_by.png","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode"},"day":"01","external_id":{"isi":["000414761700002"]},"quality_controlled":"1","volume":222,"publist_id":"6932","article_processing_charge":"No","pubrep_id":"881","language":[{"iso":"eng"}],"department":[{"_id":"RySh"}],"type":"journal_article","page":"3375 - 3393","date_created":"2018-12-11T11:48:14Z","publication":"Brain Structure and Function","has_accepted_license":"1","month":"11","publication_identifier":{"issn":["18632653"]},"oa":1,"status":"public","_id":"736","year":"2017","date_updated":"2023-09-27T14:14:51Z","issue":"8","abstract":[{"lang":"eng","text":"The neurotransmitter receptor subtype, number, density, and distribution relative to the location of transmitter release sites are key determinants of signal transmission. AMPA-type ionotropic glutamate receptors (AMPARs) containing GluA3 and GluA4 subunits are prominently expressed in subsets of neurons capable of firing action potentials at high frequencies, such as auditory relay neurons. The auditory nerve (AN) forms glutamatergic synapses on two types of relay neurons, bushy cells (BCs) and fusiform cells (FCs) of the cochlear nucleus. AN-BC and AN-FC synapses have distinct kinetics; thus, we investigated whether the number, density, and localization of GluA3 and GluA4 subunits in these synapses are differentially organized using quantitative freeze-fracture replica immunogold labeling. We identify a positive correlation between the number of AMPARs and the size of AN-BC and AN-FC synapses. Both types of AN synapses have similar numbers of AMPARs; however, the AN-BC have a higher density of AMPARs than AN-FC synapses, because the AN-BC synapses are smaller. A higher number and density of GluA3 subunits are observed at AN-BC synapses, whereas a higher number and density of GluA4 subunits are observed at AN-FC synapses. The intrasynaptic distribution of immunogold labeling revealed that AMPAR subunits, particularly GluA3, are concentrated at the center of the AN-BC synapses. The central distribution of AMPARs is absent in GluA3-knockout mice, and gold particles are evenly distributed along the postsynaptic density. GluA4 gold labeling was homogenously distributed along both synapse types. Thus, GluA3 and GluA4 subunits are distributed at AN synapses in a target-cell-dependent manner."}],"citation":{"chicago":"Rubio, María, Ko Matsui, Yugo Fukazawa, Naomi Kamasawa, Harumi Harada, Makoto Itakura, Elek Molnár, Manabu Abe, Kenji Sakimura, and Ryuichi Shigemoto. “The Number and Distribution of AMPA Receptor Channels Containing Fast Kinetic GluA3 and GluA4 Subunits at Auditory Nerve Synapses Depend on the Target Cells.” <i>Brain Structure and Function</i>. Springer, 2017. <a href=\"https://doi.org/10.1007/s00429-017-1408-0\">https://doi.org/10.1007/s00429-017-1408-0</a>.","apa":"Rubio, M., Matsui, K., Fukazawa, Y., Kamasawa, N., Harada, H., Itakura, M., … Shigemoto, R. (2017). The number and distribution of AMPA receptor channels containing fast kinetic GluA3 and GluA4 subunits at auditory nerve synapses depend on the target cells. <i>Brain Structure and Function</i>. Springer. <a href=\"https://doi.org/10.1007/s00429-017-1408-0\">https://doi.org/10.1007/s00429-017-1408-0</a>","mla":"Rubio, María, et al. “The Number and Distribution of AMPA Receptor Channels Containing Fast Kinetic GluA3 and GluA4 Subunits at Auditory Nerve Synapses Depend on the Target Cells.” <i>Brain Structure and Function</i>, vol. 222, no. 8, Springer, 2017, pp. 3375–93, doi:<a href=\"https://doi.org/10.1007/s00429-017-1408-0\">10.1007/s00429-017-1408-0</a>.","ista":"Rubio M, Matsui K, Fukazawa Y, Kamasawa N, Harada H, Itakura M, Molnár E, Abe M, Sakimura K, Shigemoto R. 2017. The number and distribution of AMPA receptor channels containing fast kinetic GluA3 and GluA4 subunits at auditory nerve synapses depend on the target cells. Brain Structure and Function. 222(8), 3375–3393.","short":"M. Rubio, K. Matsui, Y. Fukazawa, N. Kamasawa, H. Harada, M. Itakura, E. Molnár, M. Abe, K. Sakimura, R. Shigemoto, Brain Structure and Function 222 (2017) 3375–3393.","ama":"Rubio M, Matsui K, Fukazawa Y, et al. The number and distribution of AMPA receptor channels containing fast kinetic GluA3 and GluA4 subunits at auditory nerve synapses depend on the target cells. <i>Brain Structure and Function</i>. 2017;222(8):3375-3393. doi:<a href=\"https://doi.org/10.1007/s00429-017-1408-0\">10.1007/s00429-017-1408-0</a>","ieee":"M. Rubio <i>et al.</i>, “The number and distribution of AMPA receptor channels containing fast kinetic GluA3 and GluA4 subunits at auditory nerve synapses depend on the target cells,” <i>Brain Structure and Function</i>, vol. 222, no. 8. Springer, pp. 3375–3393, 2017."},"intvolume":"       222","ddc":["571"],"file_date_updated":"2020-07-14T12:47:56Z","scopus_import":"1","date_published":"2017-11-01T00:00:00Z","oa_version":"Published Version","publication_status":"published","doi":"10.1007/s00429-017-1408-0"},{"quality_controlled":"1","volume":25,"external_id":{"pmid":["28129106"]},"article_processing_charge":"No","article_type":"original","type":"journal_article","department":[{"_id":"MaLo"}],"language":[{"iso":"eng"}],"has_accepted_license":"1","publication":"Molecular Therapy","page":"102-119","date_created":"2020-01-25T15:55:39Z","license":"https://creativecommons.org/licenses/by-nc-nd/4.0/","user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","publisher":"Elsevier","title":"A synthetic mammalian therapeutic gene circuit for sensing and suppressing inflammation","author":[{"last_name":"Smole","first_name":"Anže","full_name":"Smole, Anže"},{"full_name":"Lainšček, Duško","last_name":"Lainšček","first_name":"Duško"},{"last_name":"Bezeljak","id":"2A58201A-F248-11E8-B48F-1D18A9856A87","orcid":"0000-0003-1365-5631","first_name":"Urban","full_name":"Bezeljak, Urban"},{"full_name":"Horvat, Simon","last_name":"Horvat","first_name":"Simon"},{"full_name":"Jerala, Roman","first_name":"Roman","last_name":"Jerala"}],"file":[{"checksum":"ea8b1b28606dd1edab7379ba4fa3641f","creator":"dernst","file_id":"7561","access_level":"open_access","file_name":"2017_MolecularTherapy_Smole.pdf","date_updated":"2020-07-14T12:47:56Z","date_created":"2020-03-03T10:55:13Z","content_type":"application/pdf","relation":"main_file","file_size":3404806}],"pmid":1,"day":"01","tmp":{"name":"Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International (CC BY-NC-ND 4.0)","legal_code_url":"https://creativecommons.org/licenses/by-nc-nd/4.0/legalcode","short":"CC BY-NC-ND (4.0)","image":"/images/cc_by_nc_nd.png"},"intvolume":"        25","citation":{"mla":"Smole, Anže, et al. “A Synthetic Mammalian Therapeutic Gene Circuit for Sensing and Suppressing Inflammation.” <i>Molecular Therapy</i>, vol. 25, no. 1, Elsevier, 2017, pp. 102–19, doi:<a href=\"https://doi.org/10.1016/j.ymthe.2016.10.005\">10.1016/j.ymthe.2016.10.005</a>.","ista":"Smole A, Lainšček D, Bezeljak U, Horvat S, Jerala R. 2017. A synthetic mammalian therapeutic gene circuit for sensing and suppressing inflammation. Molecular Therapy. 25(1), 102–119.","apa":"Smole, A., Lainšček, D., Bezeljak, U., Horvat, S., &#38; Jerala, R. (2017). A synthetic mammalian therapeutic gene circuit for sensing and suppressing inflammation. <i>Molecular Therapy</i>. Elsevier. <a href=\"https://doi.org/10.1016/j.ymthe.2016.10.005\">https://doi.org/10.1016/j.ymthe.2016.10.005</a>","chicago":"Smole, Anže, Duško Lainšček, Urban Bezeljak, Simon Horvat, and Roman Jerala. “A Synthetic Mammalian Therapeutic Gene Circuit for Sensing and Suppressing Inflammation.” <i>Molecular Therapy</i>. Elsevier, 2017. <a href=\"https://doi.org/10.1016/j.ymthe.2016.10.005\">https://doi.org/10.1016/j.ymthe.2016.10.005</a>.","ama":"Smole A, Lainšček D, Bezeljak U, Horvat S, Jerala R. A synthetic mammalian therapeutic gene circuit for sensing and suppressing inflammation. <i>Molecular Therapy</i>. 2017;25(1):102-119. doi:<a href=\"https://doi.org/10.1016/j.ymthe.2016.10.005\">10.1016/j.ymthe.2016.10.005</a>","ieee":"A. Smole, D. Lainšček, U. Bezeljak, S. Horvat, and R. Jerala, “A synthetic mammalian therapeutic gene circuit for sensing and suppressing inflammation,” <i>Molecular Therapy</i>, vol. 25, no. 1. Elsevier, pp. 102–119, 2017.","short":"A. Smole, D. Lainšček, U. Bezeljak, S. Horvat, R. Jerala, Molecular Therapy 25 (2017) 102–119."},"file_date_updated":"2020-07-14T12:47:56Z","ddc":["570"],"oa_version":"Published Version","date_published":"2017-01-01T00:00:00Z","doi":"10.1016/j.ymthe.2016.10.005","publication_status":"published","month":"01","status":"public","oa":1,"publication_identifier":{"issn":["1525-0016"]},"_id":"7360","issue":"1","abstract":[{"text":"Inflammation, which is a highly regulated host response against danger signals, may be harmful if it is excessive and deregulated. Ideally, anti-inflammatory therapy should autonomously commence as soon as possible after the onset of inflammation, should be controllable by a physician, and should not systemically block beneficial immune response in the long term. We describe a genetically encoded anti-inflammatory mammalian cell device based on a modular engineered genetic circuit comprising a sensor, an amplifier, a “thresholder” to restrict activation of a positive-feedback loop, a combination of advanced clinically used biopharmaceutical proteins, and orthogonal regulatory elements that linked modules into the functional device. This genetic circuit was autonomously activated by inflammatory signals, including endogenous cecal ligation and puncture (CLP)-induced inflammation in mice and serum from a systemic juvenile idiopathic arthritis (sIJA) patient, and could be reset externally by a chemical signal. The microencapsulated anti-inflammatory device significantly reduced the pathology in dextran sodium sulfate (DSS)-induced acute murine colitis, demonstrating a synthetic immunological approach for autonomous anti-inflammatory therapy.","lang":"eng"}],"date_updated":"2021-01-12T08:13:14Z","year":"2017"},{"isi":1,"author":[{"id":"2E36B656-F248-11E8-B48F-1D18A9856A87","last_name":"Virk","first_name":"Ziga","full_name":"Virk, Ziga"},{"first_name":"Andreas","last_name":"Zastrow","full_name":"Zastrow, Andreas"}],"day":"01","title":"A new topology on the universal path space","publisher":"Elsevier","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","language":[{"iso":"eng"}],"department":[{"_id":"HeEd"}],"type":"journal_article","date_created":"2018-12-11T11:48:14Z","page":"186 - 196","publication":"Topology and its Applications","external_id":{"isi":["000413889100012"]},"volume":231,"quality_controlled":"1","publist_id":"6930","article_processing_charge":"No","_id":"737","year":"2017","date_updated":"2023-09-27T12:53:01Z","abstract":[{"text":"We generalize Brazas’ topology on the fundamental group to the whole universal path space X˜ i.e., to the set of homotopy classes of all based paths. We develop basic properties of the new notion and provide a complete comparison of the obtained topology with the established topologies, in particular with the Lasso topology and the CO topology, i.e., the topology that is induced by the compact-open topology. It turns out that the new topology is the finest topology contained in the CO topology, for which the action of the fundamental group on the universal path space is a continuous group action.","lang":"eng"}],"month":"11","publication_identifier":{"issn":["01668641"]},"status":"public","date_published":"2017-11-01T00:00:00Z","oa_version":"None","publication_status":"published","doi":"10.1016/j.topol.2017.09.015","citation":{"short":"Z. Virk, A. Zastrow, Topology and Its Applications 231 (2017) 186–196.","ieee":"Z. Virk and A. Zastrow, “A new topology on the universal path space,” <i>Topology and its Applications</i>, vol. 231. Elsevier, pp. 186–196, 2017.","ama":"Virk Z, Zastrow A. A new topology on the universal path space. <i>Topology and its Applications</i>. 2017;231:186-196. doi:<a href=\"https://doi.org/10.1016/j.topol.2017.09.015\">10.1016/j.topol.2017.09.015</a>","chicago":"Virk, Ziga, and Andreas Zastrow. “A New Topology on the Universal Path Space.” <i>Topology and Its Applications</i>. Elsevier, 2017. <a href=\"https://doi.org/10.1016/j.topol.2017.09.015\">https://doi.org/10.1016/j.topol.2017.09.015</a>.","mla":"Virk, Ziga, and Andreas Zastrow. “A New Topology on the Universal Path Space.” <i>Topology and Its Applications</i>, vol. 231, Elsevier, 2017, pp. 186–96, doi:<a href=\"https://doi.org/10.1016/j.topol.2017.09.015\">10.1016/j.topol.2017.09.015</a>.","ista":"Virk Z, Zastrow A. 2017. A new topology on the universal path space. Topology and its Applications. 231, 186–196.","apa":"Virk, Z., &#38; Zastrow, A. (2017). A new topology on the universal path space. <i>Topology and Its Applications</i>. Elsevier. <a href=\"https://doi.org/10.1016/j.topol.2017.09.015\">https://doi.org/10.1016/j.topol.2017.09.015</a>"},"intvolume":"       231","scopus_import":"1"},{"_id":"739","year":"2017","date_updated":"2023-09-27T12:52:07Z","issue":"5","abstract":[{"text":"We study the norm approximation to the Schrödinger dynamics of N bosons in with an interaction potential of the form . Assuming that in the initial state the particles outside of the condensate form a quasi-free state with finite kinetic energy, we show that in the large N limit, the fluctuations around the condensate can be effectively described using Bogoliubov approximation for all . The range of β is expected to be optimal for this large class of initial states.","lang":"eng"}],"month":"11","publication_identifier":{"issn":["00217824"]},"oa":1,"status":"public","date_published":"2017-11-01T00:00:00Z","oa_version":"Submitted Version","publication_status":"published","doi":"10.1016/j.matpur.2017.05.013","citation":{"chicago":"Nam, Phan, and Marcin M Napiórkowski. “A Note on the Validity of Bogoliubov Correction to Mean Field Dynamics.” <i>Journal de Mathématiques Pures et Appliquées</i>. Elsevier, 2017. <a href=\"https://doi.org/10.1016/j.matpur.2017.05.013\">https://doi.org/10.1016/j.matpur.2017.05.013</a>.","ista":"Nam P, Napiórkowski MM. 2017. A note on the validity of Bogoliubov correction to mean field dynamics. Journal de Mathématiques Pures et Appliquées. 108(5), 662–688.","mla":"Nam, Phan, and Marcin M. Napiórkowski. “A Note on the Validity of Bogoliubov Correction to Mean Field Dynamics.” <i>Journal de Mathématiques Pures et Appliquées</i>, vol. 108, no. 5, Elsevier, 2017, pp. 662–88, doi:<a href=\"https://doi.org/10.1016/j.matpur.2017.05.013\">10.1016/j.matpur.2017.05.013</a>.","apa":"Nam, P., &#38; Napiórkowski, M. M. (2017). A note on the validity of Bogoliubov correction to mean field dynamics. <i>Journal de Mathématiques Pures et Appliquées</i>. Elsevier. <a href=\"https://doi.org/10.1016/j.matpur.2017.05.013\">https://doi.org/10.1016/j.matpur.2017.05.013</a>","ama":"Nam P, Napiórkowski MM. A note on the validity of Bogoliubov correction to mean field dynamics. <i>Journal de Mathématiques Pures et Appliquées</i>. 2017;108(5):662-688. doi:<a href=\"https://doi.org/10.1016/j.matpur.2017.05.013\">10.1016/j.matpur.2017.05.013</a>","ieee":"P. Nam and M. M. Napiórkowski, “A note on the validity of Bogoliubov correction to mean field dynamics,” <i>Journal de Mathématiques Pures et Appliquées</i>, vol. 108, no. 5. Elsevier, pp. 662–688, 2017.","short":"P. Nam, M.M. Napiórkowski, Journal de Mathématiques Pures et Appliquées 108 (2017) 662–688."},"intvolume":"       108","scopus_import":"1","main_file_link":[{"open_access":"1","url":"https://arxiv.org/abs/1604.05240"}],"project":[{"call_identifier":"FWF","name":"Structure of the Excitation Spectrum for Many-Body Quantum Systems","_id":"25C878CE-B435-11E9-9278-68D0E5697425","grant_number":"P27533_N27"}],"isi":1,"author":[{"full_name":"Nam, Phan","first_name":"Phan","id":"404092F4-F248-11E8-B48F-1D18A9856A87","last_name":"Nam"},{"first_name":"Marcin M","last_name":"Napiórkowski","id":"4197AD04-F248-11E8-B48F-1D18A9856A87","full_name":"Napiórkowski, Marcin M"}],"day":"01","title":"A note on the validity of Bogoliubov correction to mean field dynamics","publisher":"Elsevier","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","language":[{"iso":"eng"}],"department":[{"_id":"RoSe"}],"type":"journal_article","page":"662 - 688","date_created":"2018-12-11T11:48:15Z","publication":"Journal de Mathématiques Pures et Appliquées","external_id":{"isi":["000414113600003"]},"volume":108,"quality_controlled":"1","publist_id":"6928","article_processing_charge":"No"},{"doi":"10.1002/wdev.288","publication_status":"published","oa_version":"Submitted Version","date_published":"2017-08-11T00:00:00Z","file_date_updated":"2020-07-14T12:47:57Z","scopus_import":"1","ddc":["570"],"citation":{"ama":"Shigemoto R, Jösch MA. The genetic encoded toolbox for electron microscopy and connectomics. <i>WIREs Developmental Biology</i>. 2017;6(6). doi:<a href=\"https://doi.org/10.1002/wdev.288\">10.1002/wdev.288</a>","ieee":"R. Shigemoto and M. A. Jösch, “The genetic encoded toolbox for electron microscopy and connectomics,” <i>WIREs Developmental Biology</i>, vol. 6, no. 6. Wiley-Blackwell, 2017.","short":"R. Shigemoto, M.A. Jösch, WIREs Developmental Biology 6 (2017).","apa":"Shigemoto, R., &#38; Jösch, M. A. (2017). The genetic encoded toolbox for electron microscopy and connectomics. <i>WIREs Developmental Biology</i>. Wiley-Blackwell. <a href=\"https://doi.org/10.1002/wdev.288\">https://doi.org/10.1002/wdev.288</a>","ista":"Shigemoto R, Jösch MA. 2017. The genetic encoded toolbox for electron microscopy and connectomics. WIREs Developmental Biology. 6(6), e288.","mla":"Shigemoto, Ryuichi, and Maximilian A. Jösch. “The Genetic Encoded Toolbox for Electron Microscopy and Connectomics.” <i>WIREs Developmental Biology</i>, vol. 6, no. 6, e288, Wiley-Blackwell, 2017, doi:<a href=\"https://doi.org/10.1002/wdev.288\">10.1002/wdev.288</a>.","chicago":"Shigemoto, Ryuichi, and Maximilian A Jösch. “The Genetic Encoded Toolbox for Electron Microscopy and Connectomics.” <i>WIREs Developmental Biology</i>. Wiley-Blackwell, 2017. <a href=\"https://doi.org/10.1002/wdev.288\">https://doi.org/10.1002/wdev.288</a>."},"intvolume":"         6","issue":"6","abstract":[{"lang":"eng","text":"Developments in bioengineering and molecular biology have introduced a palette of genetically encoded probes for identification of specific cell populations in electron microscopy. These probes can be targeted to distinct cellular compartments, rendering them electron dense through a subsequent chemical reaction. These electron densities strongly increase the local contrast in samples prepared for electron microscopy, allowing three major advances in ultrastructural mapping of circuits: genetic identification of circuit components, targeted imaging of regions of interest and automated analysis of the tagged circuits. Together, the gains from these advances can decrease the time required for the analysis of targeted circuit motifs by over two orders of magnitude. These genetic encoded tags for electron microscopy promise to simplify the analysis of circuit motifs and become a central tool for structure‐function studies of synaptic connections in the brain. We review the current state‐of‐the‐art with an emphasis on connectomics, the quantitative analysis of neuronal structures and motifs."}],"date_updated":"2023-09-27T12:51:41Z","year":"2017","article_number":"e288","_id":"740","status":"public","oa":1,"publication_identifier":{"issn":["17597684"]},"month":"08","has_accepted_license":"1","publication":"WIREs Developmental Biology","date_created":"2018-12-11T11:48:15Z","type":"journal_article","department":[{"_id":"RySh"},{"_id":"MaJö"}],"language":[{"iso":"eng"}],"article_processing_charge":"No","article_type":"original","publist_id":"6927","volume":6,"quality_controlled":"1","external_id":{"pmid":["28800674"],"isi":["000412827400005"]},"pmid":1,"day":"11","tmp":{"image":"/images/cc_by_nc.png","short":"CC BY-NC (4.0)","legal_code_url":"https://creativecommons.org/licenses/by-nc/4.0/legalcode","name":"Creative Commons Attribution-NonCommercial 4.0 International (CC BY-NC 4.0)"},"author":[{"full_name":"Shigemoto, Ryuichi","last_name":"Shigemoto","id":"499F3ABC-F248-11E8-B48F-1D18A9856A87","first_name":"Ryuichi","orcid":"0000-0001-8761-9444"},{"orcid":"0000-0002-3937-1330","first_name":"Maximilian A","id":"2BD278E6-F248-11E8-B48F-1D18A9856A87","last_name":"Jösch","full_name":"Jösch, Maximilian A"}],"isi":1,"file":[{"file_size":1647787,"content_type":"application/pdf","relation":"main_file","access_level":"open_access","file_id":"7045","checksum":"a9370f27b1591773b7a0de299bc81c8c","creator":"dernst","date_created":"2019-11-19T07:36:18Z","date_updated":"2020-07-14T12:47:57Z","file_name":"2017_WIREs_Shigemoto.pdf"}],"user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"Wiley-Blackwell","title":"The genetic encoded toolbox for electron microscopy and connectomics","license":"https://creativecommons.org/licenses/by-nc/4.0/"},{"external_id":{"isi":["000409821300010"]},"quality_controlled":"1","volume":356,"related_material":{"record":[{"id":"52","status":"public","relation":"dissertation_contains"}]},"publist_id":"6926","article_processing_charge":"No","pubrep_id":"880","type":"journal_article","language":[{"iso":"eng"}],"department":[{"_id":"RoSe"}],"has_accepted_license":"1","page":"329 - 355","date_created":"2018-12-11T11:48:15Z","publication":"Communications in Mathematical Physics","publisher":"Springer","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","title":"Stability of a fermionic N+1 particle system with point interactions","project":[{"_id":"25C6DC12-B435-11E9-9278-68D0E5697425","grant_number":"694227","call_identifier":"H2020","name":"Analysis of quantum many-body systems"},{"call_identifier":"FWF","name":"Structure of the Excitation Spectrum for Many-Body Quantum Systems","_id":"25C878CE-B435-11E9-9278-68D0E5697425","grant_number":"P27533_N27"}],"isi":1,"author":[{"full_name":"Moser, Thomas","first_name":"Thomas","id":"2B5FC9A4-F248-11E8-B48F-1D18A9856A87","last_name":"Moser"},{"last_name":"Seiringer","id":"4AFD0470-F248-11E8-B48F-1D18A9856A87","orcid":"0000-0002-6781-0521","first_name":"Robert","full_name":"Seiringer, Robert"}],"file":[{"file_name":"IST-2017-880-v1+1_s00220-017-2980-0.pdf","date_updated":"2020-07-14T12:47:57Z","date_created":"2018-12-12T10:10:50Z","checksum":"0fd9435400f91e9b3c5346319a2d24e3","file_id":"4841","creator":"system","access_level":"open_access","relation":"main_file","content_type":"application/pdf","file_size":952639}],"tmp":{"short":"CC BY (4.0)","image":"/images/cc_by.png","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode"},"day":"01","citation":{"chicago":"Moser, Thomas, and Robert Seiringer. “Stability of a Fermionic N+1 Particle System with Point Interactions.” <i>Communications in Mathematical Physics</i>. Springer, 2017. <a href=\"https://doi.org/10.1007/s00220-017-2980-0\">https://doi.org/10.1007/s00220-017-2980-0</a>.","mla":"Moser, Thomas, and Robert Seiringer. “Stability of a Fermionic N+1 Particle System with Point Interactions.” <i>Communications in Mathematical Physics</i>, vol. 356, no. 1, Springer, 2017, pp. 329–55, doi:<a href=\"https://doi.org/10.1007/s00220-017-2980-0\">10.1007/s00220-017-2980-0</a>.","apa":"Moser, T., &#38; Seiringer, R. (2017). Stability of a fermionic N+1 particle system with point interactions. <i>Communications in Mathematical Physics</i>. Springer. <a href=\"https://doi.org/10.1007/s00220-017-2980-0\">https://doi.org/10.1007/s00220-017-2980-0</a>","ista":"Moser T, Seiringer R. 2017. Stability of a fermionic N+1 particle system with point interactions. Communications in Mathematical Physics. 356(1), 329–355.","short":"T. Moser, R. Seiringer, Communications in Mathematical Physics 356 (2017) 329–355.","ieee":"T. Moser and R. Seiringer, “Stability of a fermionic N+1 particle system with point interactions,” <i>Communications in Mathematical Physics</i>, vol. 356, no. 1. Springer, pp. 329–355, 2017.","ama":"Moser T, Seiringer R. Stability of a fermionic N+1 particle system with point interactions. <i>Communications in Mathematical Physics</i>. 2017;356(1):329-355. doi:<a href=\"https://doi.org/10.1007/s00220-017-2980-0\">10.1007/s00220-017-2980-0</a>"},"intvolume":"       356","scopus_import":"1","file_date_updated":"2020-07-14T12:47:57Z","ddc":["539"],"oa_version":"Published Version","date_published":"2017-11-01T00:00:00Z","publication_status":"published","doi":"10.1007/s00220-017-2980-0","month":"11","ec_funded":1,"status":"public","oa":1,"publication_identifier":{"issn":["00103616"]},"_id":"741","date_updated":"2023-09-27T12:34:15Z","issue":"1","abstract":[{"lang":"eng","text":"We prove that a system of N fermions interacting with an additional particle via point interactions is stable if the ratio of the mass of the additional particle to the one of the fermions is larger than some critical m*. The value of m* is independent of N and turns out to be less than 1. This fact has important implications for the stability of the unitary Fermi gas. We also characterize the domain of the Hamiltonian of this model, and establish the validity of the Tan relations for all wave functions in the domain."}],"year":"2017"},{"volume":51,"citation":{"mla":"Gottlob, Georg, et al. “Preface of the Special Issue in Memoriam Helmut Veith.” <i>Formal Methods in System Design</i>, vol. 51, no. 2, Springer, 2017, pp. 267–69, doi:<a href=\"https://doi.org/10.1007/s10703-017-0307-6\">10.1007/s10703-017-0307-6</a>.","apa":"Gottlob, G., Henzinger, T. A., &#38; Weißenbacher, G. (2017). Preface of the special issue in memoriam Helmut Veith. <i>Formal Methods in System Design</i>. Springer. <a href=\"https://doi.org/10.1007/s10703-017-0307-6\">https://doi.org/10.1007/s10703-017-0307-6</a>","ista":"Gottlob G, Henzinger TA, Weißenbacher G. 2017. Preface of the special issue in memoriam Helmut Veith. Formal Methods in System Design. 51(2), 267–269.","chicago":"Gottlob, Georg, Thomas A Henzinger, and Georg Weißenbacher. “Preface of the Special Issue in Memoriam Helmut Veith.” <i>Formal Methods in System Design</i>. Springer, 2017. <a href=\"https://doi.org/10.1007/s10703-017-0307-6\">https://doi.org/10.1007/s10703-017-0307-6</a>.","ieee":"G. Gottlob, T. A. Henzinger, and G. Weißenbacher, “Preface of the special issue in memoriam Helmut Veith,” <i>Formal Methods in System Design</i>, vol. 51, no. 2. Springer, pp. 267–269, 2017.","ama":"Gottlob G, Henzinger TA, Weißenbacher G. Preface of the special issue in memoriam Helmut Veith. <i>Formal Methods in System Design</i>. 2017;51(2):267-269. doi:<a href=\"https://doi.org/10.1007/s10703-017-0307-6\">10.1007/s10703-017-0307-6</a>","short":"G. Gottlob, T.A. Henzinger, G. Weißenbacher, Formal Methods in System Design 51 (2017) 267–269."},"quality_controlled":"1","intvolume":"        51","external_id":{"isi":["000415615600001"]},"article_processing_charge":"No","publist_id":"6924","date_published":"2017-11-14T00:00:00Z","department":[{"_id":"ToHe"}],"language":[{"iso":"eng"}],"oa_version":"None","type":"journal_article","publication":"Formal Methods in System Design","date_created":"2018-12-11T11:48:16Z","page":"267 - 269","doi":"10.1007/s10703-017-0307-6","publication_status":"published","month":"11","title":"Preface of the special issue in memoriam Helmut Veith","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"Springer","status":"public","_id":"743","isi":1,"author":[{"full_name":"Gottlob, Georg","last_name":"Gottlob","first_name":"Georg"},{"full_name":"Henzinger, Thomas A","orcid":"0000−0002−2985−7724","first_name":"Thomas A","last_name":"Henzinger","id":"40876CD8-F248-11E8-B48F-1D18A9856A87"},{"full_name":"Weißenbacher, Georg","first_name":"Georg","last_name":"Weißenbacher"}],"year":"2017","day":"14","issue":"2","abstract":[{"text":"This special issue of the Journal on Formal Methods in System Design is dedicated to Prof. Helmut Veith, who unexpectedly passed away in March 2016. Helmut Veith was a brilliant researcher, inspiring collaborator, passionate mentor, generous friend, and valued member of the formal methods community. Helmut was not only known for his numerous and influential contributions in the field of automated verification (most prominently his work on Counterexample-Guided Abstraction Refinement [1,2]), but also for his untiring and passionate efforts for the logic community: he co-organized the Vienna Summer of Logic (an event comprising twelve conferences and numerous workshops which attracted thousands of researchers from all over the world), he initiated the Vienna Center for Logic and Algorithms (which promotes international collaboration on logic and algorithms and organizes outreach events such as the LogicLounge), and he coordinated the Doctoral Program on Logical Methods in Computer Science at TU Wien (currently educating more than 40 doctoral students) and a National Research Network on Rigorous Systems Engineering (uniting fifteen researchers in Austria to address the challenge of building reliable and safe computer\r\nsystems). With his enthusiasm and commitment, Helmut completely reshaped the Austrian research landscape in the field of logic and verification in his few years as a full professor at TU Wien.","lang":"eng"}],"date_updated":"2023-09-27T12:29:29Z"},{"file_date_updated":"2020-07-14T12:47:58Z","scopus_import":"1","ddc":["000","570"],"citation":{"chicago":"Priklopil, Tadeas, Krishnendu Chatterjee, and Martin Nowak. “Optional Interactions and Suspicious Behaviour Facilitates Trustful Cooperation in Prisoners Dilemma.” <i> Journal of Theoretical Biology</i>. Elsevier, 2017. <a href=\"https://doi.org/10.1016/j.jtbi.2017.08.025\">https://doi.org/10.1016/j.jtbi.2017.08.025</a>.","mla":"Priklopil, Tadeas, et al. “Optional Interactions and Suspicious Behaviour Facilitates Trustful Cooperation in Prisoners Dilemma.” <i> Journal of Theoretical Biology</i>, vol. 433, Elsevier, 2017, pp. 64–72, doi:<a href=\"https://doi.org/10.1016/j.jtbi.2017.08.025\">10.1016/j.jtbi.2017.08.025</a>.","apa":"Priklopil, T., Chatterjee, K., &#38; Nowak, M. (2017). Optional interactions and suspicious behaviour facilitates trustful cooperation in prisoners dilemma. <i> Journal of Theoretical Biology</i>. Elsevier. <a href=\"https://doi.org/10.1016/j.jtbi.2017.08.025\">https://doi.org/10.1016/j.jtbi.2017.08.025</a>","ista":"Priklopil T, Chatterjee K, Nowak M. 2017. Optional interactions and suspicious behaviour facilitates trustful cooperation in prisoners dilemma.  Journal of Theoretical Biology. 433, 64–72.","short":"T. Priklopil, K. Chatterjee, M. Nowak,  Journal of Theoretical Biology 433 (2017) 64–72.","ama":"Priklopil T, Chatterjee K, Nowak M. Optional interactions and suspicious behaviour facilitates trustful cooperation in prisoners dilemma. <i> Journal of Theoretical Biology</i>. 2017;433:64-72. doi:<a href=\"https://doi.org/10.1016/j.jtbi.2017.08.025\">10.1016/j.jtbi.2017.08.025</a>","ieee":"T. Priklopil, K. Chatterjee, and M. Nowak, “Optional interactions and suspicious behaviour facilitates trustful cooperation in prisoners dilemma,” <i> Journal of Theoretical Biology</i>, vol. 433. Elsevier, pp. 64–72, 2017."},"intvolume":"       433","publication_status":"published","doi":"10.1016/j.jtbi.2017.08.025","oa_version":"Submitted Version","date_published":"2017-11-21T00:00:00Z","status":"public","publication_identifier":{"issn":["00225193"]},"oa":1,"month":"11","ec_funded":1,"date_updated":"2023-09-27T12:29:02Z","abstract":[{"text":"In evolutionary game theory interactions between individuals are often assumed obligatory. However, in many real-life situations, individuals can decide to opt out of an interaction depending on the information they have about the opponent. We consider a simple evolutionary game theoretic model to study such a scenario, where at each encounter between two individuals the type of the opponent (cooperator/defector) is known with some probability, and where each individual either accepts or opts out of the interaction. If the type of the opponent is unknown, a trustful individual accepts the interaction, whereas a suspicious individual opts out of the interaction. If either of the two individuals opt out both individuals remain without an interaction. We show that in the prisoners dilemma optional interactions along with suspicious behaviour facilitates the emergence of trustful cooperation.","lang":"eng"}],"year":"2017","_id":"744","publist_id":"6923","article_type":"original","article_processing_charge":"No","external_id":{"isi":["000412039800007"],"pmid":["28867224"]},"volume":433,"quality_controlled":"1","has_accepted_license":"1","date_created":"2018-12-11T11:48:16Z","page":"64 - 72","publication":" Journal of Theoretical Biology","type":"journal_article","language":[{"iso":"eng"}],"department":[{"_id":"KrCh"}],"publisher":"Elsevier","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","title":"Optional interactions and suspicious behaviour facilitates trustful cooperation in prisoners dilemma","pmid":1,"tmp":{"name":"Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International (CC BY-NC-ND 4.0)","legal_code_url":"https://creativecommons.org/licenses/by-nc-nd/4.0/legalcode","short":"CC BY-NC-ND (4.0)","image":"/images/cc_by_nc_nd.png"},"day":"21","project":[{"_id":"25681D80-B435-11E9-9278-68D0E5697425","grant_number":"291734","call_identifier":"FP7","name":"International IST Postdoc Fellowship Programme"},{"grant_number":"279307","_id":"2581B60A-B435-11E9-9278-68D0E5697425","call_identifier":"FP7","name":"Quantitative Graph Games: Theory and Applications"}],"isi":1,"author":[{"last_name":"Priklopil","id":"3C869AA0-F248-11E8-B48F-1D18A9856A87","first_name":"Tadeas","full_name":"Priklopil, Tadeas"},{"full_name":"Chatterjee, Krishnendu","first_name":"Krishnendu","orcid":"0000-0002-4561-241X","id":"2E5DCA20-F248-11E8-B48F-1D18A9856A87","last_name":"Chatterjee"},{"full_name":"Nowak, Martin","last_name":"Nowak","first_name":"Martin"}],"file":[{"checksum":"4b43af1615ebf1a861840cb03d8a320c","creator":"dernst","file_id":"7047","access_level":"open_access","date_updated":"2020-07-14T12:47:58Z","file_name":"2017_JournTheoretBio_Priklopil.pdf","date_created":"2019-11-19T07:57:39Z","content_type":"application/pdf","relation":"main_file","file_size":537323}]}]