---
_id: '400'
abstract:
- lang: eng
text: We consider the two-dimensional BCS functional with a radial pair interaction.
We show that the translational symmetry is not broken in a certain temperature
interval below the critical temperature. In the case of vanishing angular momentum,
our results carry over to the three-dimensional case.
article_processing_charge: Yes (via OA deal)
author:
- first_name: Andreas
full_name: Deuchert, Andreas
id: 4DA65CD0-F248-11E8-B48F-1D18A9856A87
last_name: Deuchert
orcid: 0000-0003-3146-6746
- first_name: Alissa
full_name: Geisinge, Alissa
last_name: Geisinge
- first_name: Christian
full_name: Hainzl, Christian
last_name: Hainzl
- first_name: Michael
full_name: Loss, Michael
last_name: Loss
citation:
ama: Deuchert A, Geisinge A, Hainzl C, Loss M. Persistence of translational symmetry
in the BCS model with radial pair interaction. Annales Henri Poincare.
2018;19(5):1507-1527. doi:10.1007/s00023-018-0665-7
apa: Deuchert, A., Geisinge, A., Hainzl, C., & Loss, M. (2018). Persistence
of translational symmetry in the BCS model with radial pair interaction. Annales
Henri Poincare. Springer. https://doi.org/10.1007/s00023-018-0665-7
chicago: Deuchert, Andreas, Alissa Geisinge, Christian Hainzl, and Michael Loss.
“Persistence of Translational Symmetry in the BCS Model with Radial Pair Interaction.”
Annales Henri Poincare. Springer, 2018. https://doi.org/10.1007/s00023-018-0665-7.
ieee: A. Deuchert, A. Geisinge, C. Hainzl, and M. Loss, “Persistence of translational
symmetry in the BCS model with radial pair interaction,” Annales Henri Poincare,
vol. 19, no. 5. Springer, pp. 1507–1527, 2018.
ista: Deuchert A, Geisinge A, Hainzl C, Loss M. 2018. Persistence of translational
symmetry in the BCS model with radial pair interaction. Annales Henri Poincare.
19(5), 1507–1527.
mla: Deuchert, Andreas, et al. “Persistence of Translational Symmetry in the BCS
Model with Radial Pair Interaction.” Annales Henri Poincare, vol. 19, no.
5, Springer, 2018, pp. 1507–27, doi:10.1007/s00023-018-0665-7.
short: A. Deuchert, A. Geisinge, C. Hainzl, M. Loss, Annales Henri Poincare 19 (2018)
1507–1527.
date_created: 2018-12-11T11:46:15Z
date_published: 2018-05-01T00:00:00Z
date_updated: 2023-09-15T12:04:15Z
day: '01'
ddc:
- '510'
department:
- _id: RoSe
doi: 10.1007/s00023-018-0665-7
ec_funded: 1
external_id:
isi:
- '000429799900008'
file:
- access_level: open_access
checksum: 04d2c9bd7cbf3ca1d7acaaf4e7dca3e5
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:12:47Z
date_updated: 2020-07-14T12:46:22Z
file_id: '4966'
file_name: IST-2018-1011-v1+1_2018_Deuchert_Persistence.pdf
file_size: 582680
relation: main_file
file_date_updated: 2020-07-14T12:46:22Z
has_accepted_license: '1'
intvolume: ' 19'
isi: 1
issue: '5'
language:
- iso: eng
month: '05'
oa: 1
oa_version: Published Version
page: 1507 - 1527
project:
- _id: 25C6DC12-B435-11E9-9278-68D0E5697425
call_identifier: H2020
grant_number: '694227'
name: Analysis of quantum many-body systems
- _id: B67AFEDC-15C9-11EA-A837-991A96BB2854
name: IST Austria Open Access Fund
publication: Annales Henri Poincare
publication_status: published
publisher: Springer
publist_id: '7429'
pubrep_id: '1011'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Persistence of translational symmetry in the BCS model with radial pair interaction
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 19
year: '2018'
...
---
_id: '401'
abstract:
- lang: eng
text: The actomyosin cytoskeleton, a key stress-producing unit in epithelial cells,
oscillates spontaneously in a wide variety of systems. Although much of the signal
cascade regulating myosin activity has been characterized, the origin of such
oscillatory behavior is still unclear. Here, we show that basal myosin II oscillation
in Drosophila ovarian epithelium is not controlled by actomyosin cortical tension,
but instead relies on a biochemical oscillator involving ROCK and myosin phosphatase.
Key to this oscillation is a diffusive ROCK flow, linking junctional Rho1 to medial
actomyosin cortex, and dynamically maintained by a self-activation loop reliant
on ROCK kinase activity. In response to the resulting myosin II recruitment, myosin
phosphatase is locally enriched and shuts off ROCK and myosin II signals. Coupling
Drosophila genetics, live imaging, modeling, and optogenetics, we uncover an intrinsic
biochemical oscillator at the core of myosin II regulatory network, shedding light
on the spatio-temporal dynamics of force generation.
article_number: '1210'
article_processing_charge: No
author:
- first_name: Xiang
full_name: Qin, Xiang
last_name: Qin
- first_name: Edouard B
full_name: Hannezo, Edouard B
id: 3A9DB764-F248-11E8-B48F-1D18A9856A87
last_name: Hannezo
orcid: 0000-0001-6005-1561
- first_name: Thomas
full_name: Mangeat, Thomas
last_name: Mangeat
- first_name: Chang
full_name: Liu, Chang
last_name: Liu
- first_name: Pralay
full_name: Majumder, Pralay
last_name: Majumder
- first_name: Jjiaying
full_name: Liu, Jjiaying
last_name: Liu
- first_name: Valerie
full_name: Choesmel Cadamuro, Valerie
last_name: Choesmel Cadamuro
- first_name: Jocelyn
full_name: Mcdonald, Jocelyn
last_name: Mcdonald
- first_name: Yinyao
full_name: Liu, Yinyao
last_name: Liu
- first_name: Bin
full_name: Yi, Bin
last_name: Yi
- first_name: Xiaobo
full_name: Wang, Xiaobo
last_name: Wang
citation:
ama: Qin X, Hannezo EB, Mangeat T, et al. A biochemical network controlling basal
myosin oscillation. Nature Communications. 2018;9(1). doi:10.1038/s41467-018-03574-5
apa: Qin, X., Hannezo, E. B., Mangeat, T., Liu, C., Majumder, P., Liu, J., … Wang,
X. (2018). A biochemical network controlling basal myosin oscillation. Nature
Communications. Nature Publishing Group. https://doi.org/10.1038/s41467-018-03574-5
chicago: Qin, Xiang, Edouard B Hannezo, Thomas Mangeat, Chang Liu, Pralay Majumder,
Jjiaying Liu, Valerie Choesmel Cadamuro, et al. “A Biochemical Network Controlling
Basal Myosin Oscillation.” Nature Communications. Nature Publishing Group,
2018. https://doi.org/10.1038/s41467-018-03574-5.
ieee: X. Qin et al., “A biochemical network controlling basal myosin oscillation,”
Nature Communications, vol. 9, no. 1. Nature Publishing Group, 2018.
ista: Qin X, Hannezo EB, Mangeat T, Liu C, Majumder P, Liu J, Choesmel Cadamuro
V, Mcdonald J, Liu Y, Yi B, Wang X. 2018. A biochemical network controlling basal
myosin oscillation. Nature Communications. 9(1), 1210.
mla: Qin, Xiang, et al. “A Biochemical Network Controlling Basal Myosin Oscillation.”
Nature Communications, vol. 9, no. 1, 1210, Nature Publishing Group, 2018,
doi:10.1038/s41467-018-03574-5.
short: X. Qin, E.B. Hannezo, T. Mangeat, C. Liu, P. Majumder, J. Liu, V. Choesmel
Cadamuro, J. Mcdonald, Y. Liu, B. Yi, X. Wang, Nature Communications 9 (2018).
date_created: 2018-12-11T11:46:16Z
date_published: 2018-03-23T00:00:00Z
date_updated: 2023-09-08T11:41:45Z
day: '23'
ddc:
- '539'
- '570'
department:
- _id: EdHa
doi: 10.1038/s41467-018-03574-5
external_id:
isi:
- '000428165400009'
file:
- access_level: open_access
checksum: 87a427bc2e8724be3dd22a4efdd21a33
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:11:45Z
date_updated: 2020-07-14T12:46:22Z
file_id: '4902'
file_name: IST-2018-996-v1+1_2018_Hannezo_A-biochemical.pdf
file_size: 3780491
relation: main_file
file_date_updated: 2020-07-14T12:46:22Z
has_accepted_license: '1'
intvolume: ' 9'
isi: 1
issue: '1'
language:
- iso: eng
month: '03'
oa: 1
oa_version: Published Version
publication: Nature Communications
publication_status: published
publisher: Nature Publishing Group
publist_id: '7427'
pubrep_id: '996'
quality_controlled: '1'
scopus_import: '1'
status: public
title: A biochemical network controlling basal myosin oscillation
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 9
year: '2018'
...
---
_id: '402'
abstract:
- lang: eng
text: During metastasis, malignant cells escape the primary tumor, intravasate lymphatic
vessels, and reach draining sentinel lymph nodes before they colonize distant
organs via the blood circulation. Although lymph node metastasis in cancer patients
correlates with poor prognosis, evidence is lacking as to whether and how tumor
cells enter the bloodstream via lymph nodes. To investigate this question, we
delivered carcinoma cells into the lymph nodes of mice by microinfusing the cells
into afferent lymphatic vessels. We found that tumor cells rapidly infiltrated
the lymph node parenchyma, invaded blood vessels, and seeded lung metastases without
involvement of the thoracic duct. These results suggest that the lymph node blood
vessels can serve as an exit route for systemic dissemination of cancer cells
in experimental mouse models. Whether this form of tumor cell spreading occurs
in cancer patients remains to be determined.
acknowledged_ssus:
- _id: Bio
acknowledgement: "M.B. was supported by the Cell Communication in Health and Disease
graduate study program of the Austrian Science Fund (FWF) and the Medical University
of Vienna. M.S. was supported by the European Research Council (grant ERC GA 281556)
and an FWF START award.\r\nWe thank C. Moussion for establishing the intralymphatic
injection at IST Austria and for providing anti-PNAd hybridoma supernatant, R. Förster
and A. Braun for sharing the intralymphatic injection technology, K. Vaahtomeri
for the lentiviral constructs, M. Hons for establishing in vivo multiphoton imaging,
the Sixt lab for intellectual input, M. Schunn for help with the design of the in
vivo experiments, F. Langer for technical assistance with the in vivo experiments,
the bioimaging facility of IST Austria for support, and R. Efferl for providing
the CT26 cell line."
article_processing_charge: No
article_type: original
author:
- first_name: Markus
full_name: Brown, Markus
id: 3DAB9AFC-F248-11E8-B48F-1D18A9856A87
last_name: Brown
- first_name: Frank P
full_name: Assen, Frank P
id: 3A8E7F24-F248-11E8-B48F-1D18A9856A87
last_name: Assen
orcid: 0000-0003-3470-6119
- first_name: Alexander F
full_name: Leithner, Alexander F
id: 3B1B77E4-F248-11E8-B48F-1D18A9856A87
last_name: Leithner
orcid: 0000-0002-1073-744X
- first_name: Jun
full_name: Abe, Jun
last_name: Abe
- first_name: Helga
full_name: Schachner, Helga
last_name: Schachner
- first_name: Gabriele
full_name: Asfour, Gabriele
last_name: Asfour
- first_name: Zsuzsanna
full_name: Bagó Horváth, Zsuzsanna
last_name: Bagó Horváth
- first_name: Jens
full_name: Stein, Jens
last_name: Stein
- first_name: Pavel
full_name: Uhrin, Pavel
last_name: Uhrin
- first_name: Michael K
full_name: Sixt, Michael K
id: 41E9FBEA-F248-11E8-B48F-1D18A9856A87
last_name: Sixt
orcid: 0000-0002-6620-9179
- first_name: Dontscho
full_name: Kerjaschki, Dontscho
last_name: Kerjaschki
citation:
ama: Brown M, Assen FP, Leithner AF, et al. Lymph node blood vessels provide exit
routes for metastatic tumor cell dissemination in mice. Science. 2018;359(6382):1408-1411.
doi:10.1126/science.aal3662
apa: Brown, M., Assen, F. P., Leithner, A. F., Abe, J., Schachner, H., Asfour, G.,
… Kerjaschki, D. (2018). Lymph node blood vessels provide exit routes for metastatic
tumor cell dissemination in mice. Science. American Association for the
Advancement of Science. https://doi.org/10.1126/science.aal3662
chicago: Brown, Markus, Frank P Assen, Alexander F Leithner, Jun Abe, Helga Schachner,
Gabriele Asfour, Zsuzsanna Bagó Horváth, et al. “Lymph Node Blood Vessels Provide
Exit Routes for Metastatic Tumor Cell Dissemination in Mice.” Science.
American Association for the Advancement of Science, 2018. https://doi.org/10.1126/science.aal3662.
ieee: M. Brown et al., “Lymph node blood vessels provide exit routes for
metastatic tumor cell dissemination in mice,” Science, vol. 359, no. 6382.
American Association for the Advancement of Science, pp. 1408–1411, 2018.
ista: Brown M, Assen FP, Leithner AF, Abe J, Schachner H, Asfour G, Bagó Horváth
Z, Stein J, Uhrin P, Sixt MK, Kerjaschki D. 2018. Lymph node blood vessels provide
exit routes for metastatic tumor cell dissemination in mice. Science. 359(6382),
1408–1411.
mla: Brown, Markus, et al. “Lymph Node Blood Vessels Provide Exit Routes for Metastatic
Tumor Cell Dissemination in Mice.” Science, vol. 359, no. 6382, American
Association for the Advancement of Science, 2018, pp. 1408–11, doi:10.1126/science.aal3662.
short: M. Brown, F.P. Assen, A.F. Leithner, J. Abe, H. Schachner, G. Asfour, Z.
Bagó Horváth, J. Stein, P. Uhrin, M.K. Sixt, D. Kerjaschki, Science 359 (2018)
1408–1411.
date_created: 2018-12-11T11:46:16Z
date_published: 2018-03-23T00:00:00Z
date_updated: 2024-03-25T23:30:05Z
day: '23'
department:
- _id: MiSi
doi: 10.1126/science.aal3662
ec_funded: 1
external_id:
isi:
- '000428043600047'
pmid:
- '29567714'
intvolume: ' 359'
isi: 1
issue: '6382'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://doi.org/10.1126/science.aal3662
month: '03'
oa: 1
oa_version: Published Version
page: 1408 - 1411
pmid: 1
project:
- _id: 25A8E5EA-B435-11E9-9278-68D0E5697425
call_identifier: FWF
grant_number: Y 564-B12
name: Cytoskeletal force generation and transduction of leukocytes (FWF)
- _id: 25A603A2-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '281556'
name: Cytoskeletal force generation and force transduction of migrating leukocytes
(EU)
publication: Science
publication_status: published
publisher: American Association for the Advancement of Science
publist_id: '7428'
quality_controlled: '1'
related_material:
record:
- id: '6947'
relation: dissertation_contains
status: public
scopus_import: '1'
status: public
title: Lymph node blood vessels provide exit routes for metastatic tumor cell dissemination
in mice
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 359
year: '2018'
...
---
_id: '403'
abstract:
- lang: eng
text: The ability to adapt growth and development to temperature variations is crucial
to generate plant varieties resilient to predicted temperature changes. However,
the mechanisms underlying plant response to progressive increases in temperature
have just started to be elucidated. Here, we report that the Cyclin-dependent
Kinase G1 (CDKG1) is a central element in a thermo-sensitive mRNA splicing cascade
that transduces changes in ambient temperature into differential expression of
the fundamental spliceosome component, ATU2AF65A. CDKG1 is alternatively spliced
in a temperature-dependent manner. We found that this process is partly dependent
on both the Cyclin-dependent Kinase G2 (CDKG2) and the interacting co-factor CYCLIN
L1 resulting in two distinct messenger RNAs. Relative abundance of both CDKG1
transcripts correlates with ambient temperature and possibly with different expression
levels of the associated protein isoforms. Both CDKG1 alternative transcripts
are necessary to fully complement the expression of ATU2AF65A across the temperature
range. Our data support a previously unidentified temperature-dependent mechanism
based on the alternative splicing of CDKG1 and regulated by CDKG2 and CYCLIN L1.
We propose that changes in ambient temperature affect the relative abundance of
CDKG1 transcripts and this in turn translates into differential CDKG1 protein
expression coordinating the alternative splicing of ATU2AF65A. This article is
protected by copyright. All rights reserved.
acknowledgement: CN, DD and JHD were funded by the BBSRC (grant number BB/M009459/1).
NC was funded by the VIPS Program of the Austrian Federal Ministry of Science and
Research and the City of Vienna. AB and AF were supported by the Austrian Science
Fund (FWF) [DK W1207; SFB RNAreg F43-P10]
article_processing_charge: No
author:
- first_name: Nicola
full_name: Cavallari, Nicola
id: 457160E6-F248-11E8-B48F-1D18A9856A87
last_name: Cavallari
- first_name: Candida
full_name: Nibau, Candida
last_name: Nibau
- first_name: Armin
full_name: Fuchs, Armin
last_name: Fuchs
- first_name: Despoina
full_name: Dadarou, Despoina
last_name: Dadarou
- first_name: Andrea
full_name: Barta, Andrea
last_name: Barta
- first_name: John
full_name: Doonan, John
last_name: Doonan
citation:
ama: Cavallari N, Nibau C, Fuchs A, Dadarou D, Barta A, Doonan J. The cyclin‐dependent
kinase G group defines a thermo‐sensitive alternative splicing circuit modulating
the expression of Arabidopsis ATU 2AF 65A. The Plant Journal. 2018;94(6):1010-1022.
doi:10.1111/tpj.13914
apa: Cavallari, N., Nibau, C., Fuchs, A., Dadarou, D., Barta, A., & Doonan,
J. (2018). The cyclin‐dependent kinase G group defines a thermo‐sensitive alternative
splicing circuit modulating the expression of Arabidopsis ATU 2AF 65A. The
Plant Journal. Wiley. https://doi.org/10.1111/tpj.13914
chicago: Cavallari, Nicola, Candida Nibau, Armin Fuchs, Despoina Dadarou, Andrea
Barta, and John Doonan. “The Cyclin‐dependent Kinase G Group Defines a Thermo‐sensitive
Alternative Splicing Circuit Modulating the Expression of Arabidopsis ATU 2AF
65A.” The Plant Journal. Wiley, 2018. https://doi.org/10.1111/tpj.13914.
ieee: N. Cavallari, C. Nibau, A. Fuchs, D. Dadarou, A. Barta, and J. Doonan, “The
cyclin‐dependent kinase G group defines a thermo‐sensitive alternative splicing
circuit modulating the expression of Arabidopsis ATU 2AF 65A,” The Plant Journal,
vol. 94, no. 6. Wiley, pp. 1010–1022, 2018.
ista: Cavallari N, Nibau C, Fuchs A, Dadarou D, Barta A, Doonan J. 2018. The cyclin‐dependent
kinase G group defines a thermo‐sensitive alternative splicing circuit modulating
the expression of Arabidopsis ATU 2AF 65A. The Plant Journal. 94(6), 1010–1022.
mla: Cavallari, Nicola, et al. “The Cyclin‐dependent Kinase G Group Defines a Thermo‐sensitive
Alternative Splicing Circuit Modulating the Expression of Arabidopsis ATU 2AF
65A.” The Plant Journal, vol. 94, no. 6, Wiley, 2018, pp. 1010–22, doi:10.1111/tpj.13914.
short: N. Cavallari, C. Nibau, A. Fuchs, D. Dadarou, A. Barta, J. Doonan, The Plant
Journal 94 (2018) 1010–1022.
date_created: 2018-12-11T11:46:17Z
date_published: 2018-06-01T00:00:00Z
date_updated: 2023-09-19T10:07:08Z
day: '01'
ddc:
- '580'
department:
- _id: EvBe
doi: 10.1111/tpj.13914
external_id:
isi:
- '000434365500008'
file:
- access_level: open_access
checksum: d9d3ad3215ac0e581731443fca312266
content_type: application/pdf
creator: dernst
date_created: 2019-02-06T11:40:54Z
date_updated: 2020-07-14T12:46:22Z
file_id: '5934'
file_name: 2018_PlantJourn_Cavallari.pdf
file_size: 1543354
relation: main_file
file_date_updated: 2020-07-14T12:46:22Z
has_accepted_license: '1'
intvolume: ' 94'
isi: 1
issue: '6'
language:
- iso: eng
month: '06'
oa: 1
oa_version: Published Version
page: 1010 - 1022
publication: The Plant Journal
publication_status: published
publisher: Wiley
publist_id: '7426'
quality_controlled: '1'
scopus_import: '1'
status: public
title: The cyclin‐dependent kinase G group defines a thermo‐sensitive alternative
splicing circuit modulating the expression of Arabidopsis ATU 2AF 65A
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 94
year: '2018'
...
---
_id: '404'
abstract:
- lang: eng
text: "We construct martingale solutions to stochastic thin-film equations by introducing
a (spatial) semidiscretization and establishing convergence. The discrete scheme
allows for variants of the energy and entropy estimates in the continuous setting
as long as the discrete energy does not exceed certain threshold values depending
on the spatial grid size $h$. Using a stopping time argument to prolongate high-energy
paths constant in time, arbitrary moments of coupled energy/entropy functionals
can be controlled. Having established Hölder regularity of approximate solutions,
the convergence proof is then based on compactness arguments---in particular on
Jakubowski's generalization of Skorokhod's theorem---weak convergence methods,
and recent tools on martingale convergence.\r\n\r\n"
article_processing_charge: No
article_type: original
author:
- first_name: Julian L
full_name: Fischer, Julian L
id: 2C12A0B0-F248-11E8-B48F-1D18A9856A87
last_name: Fischer
orcid: 0000-0002-0479-558X
- first_name: Günther
full_name: Grün, Günther
last_name: Grün
citation:
ama: Fischer JL, Grün G. Existence of positive solutions to stochastic thin-film
equations. SIAM Journal on Mathematical Analysis. 2018;50(1):411-455. doi:10.1137/16M1098796
apa: Fischer, J. L., & Grün, G. (2018). Existence of positive solutions to stochastic
thin-film equations. SIAM Journal on Mathematical Analysis. Society for
Industrial and Applied Mathematics . https://doi.org/10.1137/16M1098796
chicago: Fischer, Julian L, and Günther Grün. “Existence of Positive Solutions to
Stochastic Thin-Film Equations.” SIAM Journal on Mathematical Analysis.
Society for Industrial and Applied Mathematics , 2018. https://doi.org/10.1137/16M1098796.
ieee: J. L. Fischer and G. Grün, “Existence of positive solutions to stochastic
thin-film equations,” SIAM Journal on Mathematical Analysis, vol. 50, no.
1. Society for Industrial and Applied Mathematics , pp. 411–455, 2018.
ista: Fischer JL, Grün G. 2018. Existence of positive solutions to stochastic thin-film
equations. SIAM Journal on Mathematical Analysis. 50(1), 411–455.
mla: Fischer, Julian L., and Günther Grün. “Existence of Positive Solutions to Stochastic
Thin-Film Equations.” SIAM Journal on Mathematical Analysis, vol. 50, no.
1, Society for Industrial and Applied Mathematics , 2018, pp. 411–55, doi:10.1137/16M1098796.
short: J.L. Fischer, G. Grün, SIAM Journal on Mathematical Analysis 50 (2018) 411–455.
date_created: 2018-12-11T11:46:17Z
date_published: 2018-01-30T00:00:00Z
date_updated: 2023-09-11T13:59:22Z
day: '30'
ddc:
- '510'
department:
- _id: JuFi
doi: 10.1137/16M1098796
external_id:
isi:
- '000426630900015'
file:
- access_level: open_access
checksum: 89a8eae7c52bb356c04f52b44bff4b5a
content_type: application/pdf
creator: dernst
date_created: 2019-11-07T12:20:25Z
date_updated: 2020-07-14T12:46:22Z
file_id: '6992'
file_name: 2018_SIAM_Fischer.pdf
file_size: 557338
relation: main_file
file_date_updated: 2020-07-14T12:46:22Z
has_accepted_license: '1'
intvolume: ' 50'
isi: 1
issue: '1'
language:
- iso: eng
month: '01'
oa: 1
oa_version: Published Version
page: 411 - 455
publication: SIAM Journal on Mathematical Analysis
publication_status: published
publisher: 'Society for Industrial and Applied Mathematics '
publist_id: '7425'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Existence of positive solutions to stochastic thin-film equations
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 50
year: '2018'
...
---
_id: '406'
abstract:
- lang: eng
text: 'Recent developments in automated tracking allow uninterrupted, high-resolution
recording of animal trajectories, sometimes coupled with the identification of
stereotyped changes of body pose or other behaviors of interest. Analysis and
interpretation of such data represents a challenge: the timing of animal behaviors
may be stochastic and modulated by kinematic variables, by the interaction with
the environment or with the conspecifics within the animal group, and dependent
on internal cognitive or behavioral state of the individual. Existing models for
collective motion typically fail to incorporate the discrete, stochastic, and
internal-state-dependent aspects of behavior, while models focusing on individual
animal behavior typically ignore the spatial aspects of the problem. Here we propose
a probabilistic modeling framework to address this gap. Each animal can switch
stochastically between different behavioral states, with each state resulting
in a possibly different law of motion through space. Switching rates for behavioral
transitions can depend in a very general way, which we seek to identify from data,
on the effects of the environment as well as the interaction between the animals.
We represent the switching dynamics as a Generalized Linear Model and show that:
(i) forward simulation of multiple interacting animals is possible using a variant
of the Gillespie’s Stochastic Simulation Algorithm; (ii) formulated properly,
the maximum likelihood inference of switching rate functions is tractably solvable
by gradient descent; (iii) model selection can be used to identify factors that
modulate behavioral state switching and to appropriately adjust model complexity
to data. To illustrate our framework, we apply it to two synthetic models of animal
motion and to real zebrafish tracking data. '
acknowledgement: This work was supported by the Human Frontier Science Program RGP0065/2012
(GT, ES).
article_processing_charge: Yes
author:
- first_name: Katarína
full_name: Bod’Ová, Katarína
last_name: Bod’Ová
- first_name: Gabriel
full_name: Mitchell, Gabriel
id: 315BCD80-F248-11E8-B48F-1D18A9856A87
last_name: Mitchell
- first_name: Roy
full_name: Harpaz, Roy
last_name: Harpaz
- first_name: Elad
full_name: Schneidman, Elad
last_name: Schneidman
- first_name: Gasper
full_name: Tkacik, Gasper
id: 3D494DCA-F248-11E8-B48F-1D18A9856A87
last_name: Tkacik
orcid: 0000-0002-6699-1455
citation:
ama: Bod’Ová K, Mitchell G, Harpaz R, Schneidman E, Tkačik G. Probabilistic models
of individual and collective animal behavior. PLoS One. 2018;13(3). doi:10.1371/journal.pone.0193049
apa: Bod’Ová, K., Mitchell, G., Harpaz, R., Schneidman, E., & Tkačik, G. (2018).
Probabilistic models of individual and collective animal behavior. PLoS One.
Public Library of Science. https://doi.org/10.1371/journal.pone.0193049
chicago: Bod’Ová, Katarína, Gabriel Mitchell, Roy Harpaz, Elad Schneidman, and Gašper
Tkačik. “Probabilistic Models of Individual and Collective Animal Behavior.” PLoS
One. Public Library of Science, 2018. https://doi.org/10.1371/journal.pone.0193049.
ieee: K. Bod’Ová, G. Mitchell, R. Harpaz, E. Schneidman, and G. Tkačik, “Probabilistic
models of individual and collective animal behavior,” PLoS One, vol. 13,
no. 3. Public Library of Science, 2018.
ista: Bod’Ová K, Mitchell G, Harpaz R, Schneidman E, Tkačik G. 2018. Probabilistic
models of individual and collective animal behavior. PLoS One. 13(3).
mla: Bod’Ová, Katarína, et al. “Probabilistic Models of Individual and Collective
Animal Behavior.” PLoS One, vol. 13, no. 3, Public Library of Science,
2018, doi:10.1371/journal.pone.0193049.
short: K. Bod’Ová, G. Mitchell, R. Harpaz, E. Schneidman, G. Tkačik, PLoS One 13
(2018).
date_created: 2018-12-11T11:46:18Z
date_published: 2018-03-07T00:00:00Z
date_updated: 2023-09-15T12:06:19Z
day: '07'
ddc:
- '530'
- '571'
department:
- _id: GaTk
doi: 10.1371/journal.pone.0193049
external_id:
isi:
- '000426896800032'
file:
- access_level: open_access
checksum: 684229493db75b43e98a46cd922da497
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:15:43Z
date_updated: 2020-07-14T12:46:22Z
file_id: '5165'
file_name: IST-2018-995-v1+1_2018_Bodova_Probabilistic.pdf
file_size: 6887358
relation: main_file
file_date_updated: 2020-07-14T12:46:22Z
has_accepted_license: '1'
intvolume: ' 13'
isi: 1
issue: '3'
language:
- iso: eng
month: '03'
oa: 1
oa_version: Submitted Version
project:
- _id: 255008E4-B435-11E9-9278-68D0E5697425
grant_number: RGP0065/2012
name: Information processing and computation in fish groups
publication: PLoS One
publication_status: published
publisher: Public Library of Science
publist_id: '7423'
pubrep_id: '995'
quality_controlled: '1'
related_material:
record:
- id: '9831'
relation: research_data
status: public
scopus_import: '1'
status: public
title: Probabilistic models of individual and collective animal behavior
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 13
year: '2018'
...
---
_id: '407'
abstract:
- lang: eng
text: Isoprenoid cytokinins play a number of crucial roles in the regulation of
plant growth and development. To study cytokinin receptor properties in plants,
we designed and prepared fluorescent derivatives of 6-[(3-methylbut-2-en-1-yl)amino]purine
(N6-isopentenyladenine, iP) with several fluorescent labels attached to the C2
or N9 atom of the purine moiety via a 2- or 6-carbon linker. The fluorescent labels
included dansyl (DS), fluorescein (FC), 7-nitrobenzofurazan (NBD), rhodamine B
(RhoB), coumarin (Cou), 7-(diethylamino)coumarin (DEAC) and cyanine 5 dye (Cy5).
All prepared compounds were screened for affinity for the Arabidopsis thaliana
cytokinin receptor (CRE1/AHK4). Although the attachment of the fluorescent labels
to iP via the linkers mostly disrupted binding to the receptor, several fluorescent
derivatives interacted well. For this reason, three derivatives, two rhodamine
B and one 4-chloro-7-nitrobenzofurazan labeled iP were tested for their interaction
with CRE1/AHK4 and Zea mays cytokinin receptors in detail. We further showed that
the three derivatives were able to activate transcription of cytokinin response
regulator ARR5 in Arabidopsis seedlings. The activity of fluorescently labeled
cytokinins was compared with corresponding 6-dimethylaminopurine fluorescently
labeled negative controls. Selected rhodamine B C2-labeled compounds 17, 18 and
4-chloro-7-nitrobenzofurazan N9-labeled compound 28 and their respective negative
controls (19, 20 and 29, respectively) were used for in planta staining experiments
in Arabidopsis thaliana cell suspension culture using live cell confocal microscopy.
acknowledgement: "This work was supported by the Ministry of Education Youth and Sports,
Czech Republic (grant LO1204 from the National Program of Sustainability I and Agricultural
Research ) and by Czech Science Foundation grants 16-04184S , 501/10/1450 and 13-39982S
and by IGA projects IGA_PrF_2018_033 and IGA_PrF_2018_023 . We would like to thank
Jarmila Balonová, Olga Hustáková and Miroslava Šubová for their skillful technical
assistance and Mgr. Tomáš Pospíšil, Ph.D. for his measurement of 1 H NMR and analysis
of some 2D NMR spectral data. \r\n"
article_processing_charge: No
author:
- first_name: Karolina
full_name: Kubiasová, Karolina
last_name: Kubiasová
- first_name: Václav
full_name: Mik, Václav
last_name: Mik
- first_name: Jaroslav
full_name: Nisler, Jaroslav
last_name: Nisler
- first_name: Martin
full_name: Hönig, Martin
last_name: Hönig
- first_name: Alexandra
full_name: Husičková, Alexandra
last_name: Husičková
- first_name: Lukáš
full_name: Spíchal, Lukáš
last_name: Spíchal
- first_name: Zuzana
full_name: Pěkná, Zuzana
last_name: Pěkná
- first_name: Olga
full_name: Šamajová, Olga
last_name: Šamajová
- first_name: Karel
full_name: Doležal, Karel
last_name: Doležal
- first_name: Ondřej
full_name: Plíhal, Ondřej
last_name: Plíhal
- first_name: Eva
full_name: Benková, Eva
id: 38F4F166-F248-11E8-B48F-1D18A9856A87
last_name: Benková
orcid: 0000-0002-8510-9739
- first_name: Miroslav
full_name: Strnad, Miroslav
last_name: Strnad
- first_name: Lucie
full_name: Plíhalová, Lucie
last_name: Plíhalová
citation:
ama: Kubiasová K, Mik V, Nisler J, et al. Design, synthesis and perception of fluorescently
labeled isoprenoid cytokinins. Phytochemistry. 2018;150:1-11. doi:10.1016/j.phytochem.2018.02.015
apa: Kubiasová, K., Mik, V., Nisler, J., Hönig, M., Husičková, A., Spíchal, L.,
… Plíhalová, L. (2018). Design, synthesis and perception of fluorescently labeled
isoprenoid cytokinins. Phytochemistry. Elsevier. https://doi.org/10.1016/j.phytochem.2018.02.015
chicago: Kubiasová, Karolina, Václav Mik, Jaroslav Nisler, Martin Hönig, Alexandra
Husičková, Lukáš Spíchal, Zuzana Pěkná, et al. “Design, Synthesis and Perception
of Fluorescently Labeled Isoprenoid Cytokinins.” Phytochemistry. Elsevier,
2018. https://doi.org/10.1016/j.phytochem.2018.02.015.
ieee: K. Kubiasová et al., “Design, synthesis and perception of fluorescently
labeled isoprenoid cytokinins,” Phytochemistry, vol. 150. Elsevier, pp.
1–11, 2018.
ista: Kubiasová K, Mik V, Nisler J, Hönig M, Husičková A, Spíchal L, Pěkná Z, Šamajová
O, Doležal K, Plíhal O, Benková E, Strnad M, Plíhalová L. 2018. Design, synthesis
and perception of fluorescently labeled isoprenoid cytokinins. Phytochemistry.
150, 1–11.
mla: Kubiasová, Karolina, et al. “Design, Synthesis and Perception of Fluorescently
Labeled Isoprenoid Cytokinins.” Phytochemistry, vol. 150, Elsevier, 2018,
pp. 1–11, doi:10.1016/j.phytochem.2018.02.015.
short: K. Kubiasová, V. Mik, J. Nisler, M. Hönig, A. Husičková, L. Spíchal, Z. Pěkná,
O. Šamajová, K. Doležal, O. Plíhal, E. Benková, M. Strnad, L. Plíhalová, Phytochemistry
150 (2018) 1–11.
date_created: 2018-12-11T11:46:18Z
date_published: 2018-06-01T00:00:00Z
date_updated: 2023-09-11T12:53:11Z
day: '01'
department:
- _id: EvBe
doi: 10.1016/j.phytochem.2018.02.015
external_id:
isi:
- '000435623400001'
intvolume: ' 150'
isi: 1
language:
- iso: eng
month: '06'
oa_version: None
page: 1-11
publication: Phytochemistry
publication_status: published
publisher: Elsevier
publist_id: '7422'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Design, synthesis and perception of fluorescently labeled isoprenoid cytokinins
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 150
year: '2018'
...
---
_id: '408'
abstract:
- lang: eng
text: Adventitious roots (AR) are de novo formed roots that emerge from any part
of the plant or from callus in tissue culture, except root tissue. The plant tissue
origin and the method by which they are induced determine the physiological properties
of emerged ARs. Hence, a standard method encompassing all types of AR does not
exist. Here we describe a method for the induction and analysis of AR that emerge
from the etiolated hypocotyl of dicot plants. The hypocotyl is formed during embryogenesis
and shows a determined developmental pattern which usually does not involve AR
formation. However, the hypocotyl shows propensity to form de novo roots under
specific circumstances such as removal of the root system, high humidity or flooding,
or during de-etiolation. The hypocotyl AR emerge from a pericycle-like cell layer
surrounding the vascular tissue of the central cylinder, which is reminiscent
to the developmental program of lateral roots. Here we propose an easy protocol
for in vitro hypocotyl AR induction from etiolated Arabidopsis seedlings.
alternative_title:
- MIMB
article_processing_charge: No
author:
- first_name: Hoang
full_name: Trinh, Hoang
last_name: Trinh
- first_name: Inge
full_name: Verstraeten, Inge
id: 362BF7FE-F248-11E8-B48F-1D18A9856A87
last_name: Verstraeten
orcid: 0000-0001-7241-2328
- first_name: Danny
full_name: Geelen, Danny
last_name: Geelen
citation:
ama: 'Trinh H, Verstraeten I, Geelen D. In vitro assay for induction of adventitious
rooting on intact arabidopsis hypocotyls. In: Root Development . Vol 1761.
Springer Nature; 2018:95-102. doi:10.1007/978-1-4939-7747-5_7'
apa: Trinh, H., Verstraeten, I., & Geelen, D. (2018). In vitro assay for induction
of adventitious rooting on intact arabidopsis hypocotyls. In Root Development
(Vol. 1761, pp. 95–102). Springer Nature. https://doi.org/10.1007/978-1-4939-7747-5_7
chicago: Trinh, Hoang, Inge Verstraeten, and Danny Geelen. “In Vitro Assay for Induction
of Adventitious Rooting on Intact Arabidopsis Hypocotyls.” In Root Development
, 1761:95–102. Springer Nature, 2018. https://doi.org/10.1007/978-1-4939-7747-5_7.
ieee: H. Trinh, I. Verstraeten, and D. Geelen, “In vitro assay for induction of
adventitious rooting on intact arabidopsis hypocotyls,” in Root Development
, vol. 1761, Springer Nature, 2018, pp. 95–102.
ista: 'Trinh H, Verstraeten I, Geelen D. 2018.In vitro assay for induction of adventitious
rooting on intact arabidopsis hypocotyls. In: Root Development . MIMB, vol. 1761,
95–102.'
mla: Trinh, Hoang, et al. “In Vitro Assay for Induction of Adventitious Rooting
on Intact Arabidopsis Hypocotyls.” Root Development , vol. 1761, Springer
Nature, 2018, pp. 95–102, doi:10.1007/978-1-4939-7747-5_7.
short: H. Trinh, I. Verstraeten, D. Geelen, in:, Root Development , Springer Nature,
2018, pp. 95–102.
date_created: 2018-12-11T11:46:18Z
date_published: 2018-03-01T00:00:00Z
date_updated: 2021-01-12T07:54:21Z
day: '01'
department:
- _id: JiFr
doi: 10.1007/978-1-4939-7747-5_7
external_id:
pmid:
- '29525951'
intvolume: ' 1761'
language:
- iso: eng
month: '03'
oa_version: None
page: 95 - 102
pmid: 1
publication: 'Root Development '
publication_identifier:
issn:
- 1064-3745
publication_status: published
publisher: Springer Nature
publist_id: '7421'
quality_controlled: '1'
scopus_import: '1'
status: public
title: In vitro assay for induction of adventitious rooting on intact arabidopsis
hypocotyls
type: book_chapter
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 1761
year: '2018'
...
---
_id: '409'
abstract:
- lang: eng
text: We give a simple proof of T. Stehling's result [4], whereby in any normal
tiling of the plane with convex polygons with number of sides not less than six,
all tiles except a finite number are hexagons.
article_processing_charge: No
article_type: original
arxiv: 1
author:
- first_name: Arseniy
full_name: Akopyan, Arseniy
id: 430D2C90-F248-11E8-B48F-1D18A9856A87
last_name: Akopyan
orcid: 0000-0002-2548-617X
citation:
ama: Akopyan A. On the number of non-hexagons in a planar tiling. Comptes Rendus
Mathematique. 2018;356(4):412-414. doi:10.1016/j.crma.2018.03.005
apa: Akopyan, A. (2018). On the number of non-hexagons in a planar tiling. Comptes
Rendus Mathematique. Elsevier. https://doi.org/10.1016/j.crma.2018.03.005
chicago: Akopyan, Arseniy. “On the Number of Non-Hexagons in a Planar Tiling.” Comptes
Rendus Mathematique. Elsevier, 2018. https://doi.org/10.1016/j.crma.2018.03.005.
ieee: A. Akopyan, “On the number of non-hexagons in a planar tiling,” Comptes
Rendus Mathematique, vol. 356, no. 4. Elsevier, pp. 412–414, 2018.
ista: Akopyan A. 2018. On the number of non-hexagons in a planar tiling. Comptes
Rendus Mathematique. 356(4), 412–414.
mla: Akopyan, Arseniy. “On the Number of Non-Hexagons in a Planar Tiling.” Comptes
Rendus Mathematique, vol. 356, no. 4, Elsevier, 2018, pp. 412–14, doi:10.1016/j.crma.2018.03.005.
short: A. Akopyan, Comptes Rendus Mathematique 356 (2018) 412–414.
date_created: 2018-12-11T11:46:19Z
date_published: 2018-04-01T00:00:00Z
date_updated: 2023-09-13T09:34:12Z
day: '01'
department:
- _id: HeEd
doi: 10.1016/j.crma.2018.03.005
external_id:
arxiv:
- '1805.01652'
isi:
- '000430402700009'
intvolume: ' 356'
isi: 1
issue: '4'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://arxiv.org/abs/1805.01652
month: '04'
oa: 1
oa_version: Preprint
page: 412-414
publication: Comptes Rendus Mathematique
publication_identifier:
issn:
- 1631073X
publication_status: published
publisher: Elsevier
publist_id: '7420'
quality_controlled: '1'
scopus_import: '1'
status: public
title: On the number of non-hexagons in a planar tiling
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 356
year: '2018'
...
---
_id: '41'
abstract:
- lang: eng
text: 'The small-conductance, Ca2+-activated K+ (SK) channel subtype SK2 regulates
the spike rate and firing frequency, as well as Ca2+ transients in Purkinje cells
(PCs). To understand the molecular basis by which SK2 channels mediate these functions,
we analyzed the exact location and densities of SK2 channels along the neuronal
surface of the mouse cerebellar PCs using SDS-digested freeze-fracture replica
labeling (SDS-FRL) of high sensitivity combined with quantitative analyses. Immunogold
particles for SK2 were observed on post- and pre-synaptic compartments showing
both scattered and clustered distribution patterns. We found an axo-somato-dendritic
gradient of the SK2 particle density increasing 12-fold from soma to dendritic
spines. Using two different immunogold approaches, we also found that SK2 immunoparticles
were frequently adjacent to, but never overlap with, the postsynaptic density
of excitatory synapses in PC spines. Co-immunoprecipitation analysis demonstrated
that SK2 channels form macromolecular complexes with two types of proteins that
mobilize Ca2+: CaV2.1 channels and mGlu1α receptors in the cerebellum. Freeze-fracture
replica double-labeling showed significant co-clustering of particles for SK2
with those for CaV2.1 channels and mGlu1α receptors. SK2 channels were also detected
at presynaptic sites, mostly at the presynaptic active zone (AZ), where they are
close to CaV2.1 channels, though they are not significantly co-clustered. These
data demonstrate that SK2 channels located in different neuronal compartments
can associate with distinct proteins mobilizing Ca2+, and suggest that the ultrastructural
association of SK2 with CaV2.1 and mGlu1α provides the mechanism that ensures
voltage (excitability) regulation by distinct intracellular Ca2+ transients in
PCs.'
article_number: '311'
article_processing_charge: No
article_type: original
author:
- first_name: Rafæl
full_name: Luján, Rafæl
last_name: Luján
- first_name: Carolina
full_name: Aguado, Carolina
last_name: Aguado
- first_name: Francisco
full_name: Ciruela, Francisco
last_name: Ciruela
- first_name: Xavier
full_name: Arus, Xavier
last_name: Arus
- first_name: Alejandro
full_name: Martín Belmonte, Alejandro
last_name: Martín Belmonte
- first_name: Rocío
full_name: Alfaro Ruiz, Rocío
last_name: Alfaro Ruiz
- first_name: Jesus
full_name: Martinez Gomez, Jesus
last_name: Martinez Gomez
- first_name: Luis
full_name: De La Ossa, Luis
last_name: De La Ossa
- first_name: Masahiko
full_name: Watanabe, Masahiko
last_name: Watanabe
- first_name: John
full_name: Adelman, John
last_name: Adelman
- first_name: Ryuichi
full_name: Shigemoto, Ryuichi
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Yugo
full_name: Fukazawa, Yugo
last_name: Fukazawa
citation:
ama: Luján R, Aguado C, Ciruela F, et al. Sk2 channels associate with mGlu1α receptors
and CaV2.1 channels in Purkinje cells. Frontiers in Cellular Neuroscience.
2018;12. doi:10.3389/fncel.2018.00311
apa: Luján, R., Aguado, C., Ciruela, F., Arus, X., Martín Belmonte, A., Alfaro Ruiz,
R., … Fukazawa, Y. (2018). Sk2 channels associate with mGlu1α receptors and CaV2.1
channels in Purkinje cells. Frontiers in Cellular Neuroscience. Frontiers
Media. https://doi.org/10.3389/fncel.2018.00311
chicago: Luján, Rafæl, Carolina Aguado, Francisco Ciruela, Xavier Arus, Alejandro
Martín Belmonte, Rocío Alfaro Ruiz, Jesus Martinez Gomez, et al. “Sk2 Channels
Associate with MGlu1α Receptors and CaV2.1 Channels in Purkinje Cells.” Frontiers
in Cellular Neuroscience. Frontiers Media, 2018. https://doi.org/10.3389/fncel.2018.00311.
ieee: R. Luján et al., “Sk2 channels associate with mGlu1α receptors and
CaV2.1 channels in Purkinje cells,” Frontiers in Cellular Neuroscience,
vol. 12. Frontiers Media, 2018.
ista: Luján R, Aguado C, Ciruela F, Arus X, Martín Belmonte A, Alfaro Ruiz R, Martinez
Gomez J, De La Ossa L, Watanabe M, Adelman J, Shigemoto R, Fukazawa Y. 2018. Sk2
channels associate with mGlu1α receptors and CaV2.1 channels in Purkinje cells.
Frontiers in Cellular Neuroscience. 12, 311.
mla: Luján, Rafæl, et al. “Sk2 Channels Associate with MGlu1α Receptors and CaV2.1
Channels in Purkinje Cells.” Frontiers in Cellular Neuroscience, vol. 12,
311, Frontiers Media, 2018, doi:10.3389/fncel.2018.00311.
short: R. Luján, C. Aguado, F. Ciruela, X. Arus, A. Martín Belmonte, R. Alfaro Ruiz,
J. Martinez Gomez, L. De La Ossa, M. Watanabe, J. Adelman, R. Shigemoto, Y. Fukazawa,
Frontiers in Cellular Neuroscience 12 (2018).
date_created: 2018-12-11T11:44:19Z
date_published: 2018-09-19T00:00:00Z
date_updated: 2023-09-18T09:31:18Z
day: '19'
ddc:
- '570'
department:
- _id: RySh
doi: 10.3389/fncel.2018.00311
ec_funded: 1
external_id:
isi:
- '000445090100002'
file:
- access_level: open_access
checksum: 0bcaec8d596162af0b7fe3f31325d480
content_type: application/pdf
creator: dernst
date_created: 2018-12-17T08:49:03Z
date_updated: 2020-07-14T12:46:23Z
file_id: '5684'
file_name: fncel-12-00311.pdf
file_size: 6834251
relation: main_file
file_date_updated: 2020-07-14T12:46:23Z
has_accepted_license: '1'
intvolume: ' 12'
isi: 1
language:
- iso: eng
month: '09'
oa: 1
oa_version: Published Version
project:
- _id: 25CBA828-B435-11E9-9278-68D0E5697425
call_identifier: H2020
grant_number: '720270'
name: Human Brain Project Specific Grant Agreement 1 (HBP SGA 1)
publication: Frontiers in Cellular Neuroscience
publication_identifier:
issn:
- '16625102'
publication_status: published
publisher: Frontiers Media
publist_id: '8013'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Sk2 channels associate with mGlu1α receptors and CaV2.1 channels in Purkinje
cells
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 12
year: '2018'
...
---
_id: '410'
abstract:
- lang: eng
text: Lesion verification and quantification is traditionally done via histological
examination of sectioned brains, a time-consuming process that relies heavily
on manual estimation. Such methods are particularly problematic in posterior cortical
regions (e.g. visual cortex), where sectioning leads to significant damage and
distortion of tissue. Even more challenging is the post hoc localization of micro-electrodes,
which relies on the same techniques, suffers from similar drawbacks and requires
even higher precision. Here, we propose a new, simple method for quantitative
lesion characterization and electrode localization that is less labor-intensive
and yields more detailed results than conventional methods. We leverage staining
techniques standard in electron microscopy with the use of commodity micro-CT
imaging. We stain whole rat and zebra finch brains in osmium tetroxide, embed
these in resin and scan entire brains in a micro-CT machine. The scans result
in 3D reconstructions of the brains with section thickness dependent on sample
size (12–15 and 5–6 microns for rat and zebra finch respectively) that can be
segmented manually or automatically. Because the method captures the entire intact
brain volume, comparisons within and across studies are more tractable, and the
extent of lesions and electrodes may be studied with higher accuracy than with
current methods.
article_number: '5184'
article_processing_charge: No
author:
- first_name: Javier
full_name: Masís, Javier
last_name: Masís
- first_name: David
full_name: Mankus, David
last_name: Mankus
- first_name: Steffen
full_name: Wolff, Steffen
last_name: Wolff
- first_name: Grigori
full_name: Guitchounts, Grigori
last_name: Guitchounts
- first_name: Maximilian A
full_name: Jösch, Maximilian A
id: 2BD278E6-F248-11E8-B48F-1D18A9856A87
last_name: Jösch
orcid: 0000-0002-3937-1330
- first_name: David
full_name: Cox, David
last_name: Cox
citation:
ama: Masís J, Mankus D, Wolff S, Guitchounts G, Jösch MA, Cox D. A micro-CT-based
method for quantitative brain lesion characterization and electrode localization.
Scientific Reports. 2018;8(1). doi:10.1038/s41598-018-23247-z
apa: Masís, J., Mankus, D., Wolff, S., Guitchounts, G., Jösch, M. A., & Cox,
D. (2018). A micro-CT-based method for quantitative brain lesion characterization
and electrode localization. Scientific Reports. Nature Publishing Group.
https://doi.org/10.1038/s41598-018-23247-z
chicago: Masís, Javier, David Mankus, Steffen Wolff, Grigori Guitchounts, Maximilian
A Jösch, and David Cox. “A Micro-CT-Based Method for Quantitative Brain Lesion
Characterization and Electrode Localization.” Scientific Reports. Nature
Publishing Group, 2018. https://doi.org/10.1038/s41598-018-23247-z.
ieee: J. Masís, D. Mankus, S. Wolff, G. Guitchounts, M. A. Jösch, and D. Cox, “A
micro-CT-based method for quantitative brain lesion characterization and electrode
localization,” Scientific Reports, vol. 8, no. 1. Nature Publishing Group,
2018.
ista: Masís J, Mankus D, Wolff S, Guitchounts G, Jösch MA, Cox D. 2018. A micro-CT-based
method for quantitative brain lesion characterization and electrode localization.
Scientific Reports. 8(1), 5184.
mla: Masís, Javier, et al. “A Micro-CT-Based Method for Quantitative Brain Lesion
Characterization and Electrode Localization.” Scientific Reports, vol.
8, no. 1, 5184, Nature Publishing Group, 2018, doi:10.1038/s41598-018-23247-z.
short: J. Masís, D. Mankus, S. Wolff, G. Guitchounts, M.A. Jösch, D. Cox, Scientific
Reports 8 (2018).
date_created: 2018-12-11T11:46:19Z
date_published: 2018-03-26T00:00:00Z
date_updated: 2023-09-08T11:48:39Z
day: '26'
ddc:
- '571'
- '572'
department:
- _id: MaJö
doi: 10.1038/s41598-018-23247-z
external_id:
isi:
- '000428234100005'
file:
- access_level: open_access
checksum: 653fcb852f899c75b00ceee2a670d738
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:10:42Z
date_updated: 2020-07-14T12:46:23Z
file_id: '4831'
file_name: IST-2018-994-v1+1_2018_Joesch_A-micro-CT-based.pdf
file_size: 2359430
relation: main_file
file_date_updated: 2020-07-14T12:46:23Z
has_accepted_license: '1'
intvolume: ' 8'
isi: 1
issue: '1'
language:
- iso: eng
month: '03'
oa: 1
oa_version: Published Version
publication: Scientific Reports
publication_status: published
publisher: Nature Publishing Group
publist_id: '7419'
pubrep_id: '994'
quality_controlled: '1'
scopus_import: '1'
status: public
title: A micro-CT-based method for quantitative brain lesion characterization and
electrode localization
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 8
year: '2018'
...
---
_id: '411'
abstract:
- lang: eng
text: Immunolocalization is a valuable tool for cell biology research that allows
to rapidly determine the localization and expression levels of endogenous proteins.
In plants, whole-mount in situ immunolocalization remains a challenging method,
especially in tissues protected by waxy layers and complex cell wall carbohydrates.
Here, we present a robust method for whole-mount in situ immunolocalization in
primary root meristems and lateral root primordia in Arabidopsis thaliana. For
good epitope preservation, fixation is done in an alkaline paraformaldehyde/glutaraldehyde
mixture. This fixative is suitable for detecting a wide range of proteins, including
integral transmembrane proteins and proteins peripherally attached to the plasma
membrane. From initiation until emergence from the primary root, lateral root
primordia are surrounded by several layers of differentiated tissues with a complex
cell wall composition that interferes with the efficient penetration of all buffers.
Therefore, immunolocalization in early lateral root primordia requires a modified
method, including a strong solvent treatment for removal of hydrophobic barriers
and a specific cocktail of cell wall-degrading enzymes. The presented method allows
for easy, reliable, and high-quality in situ detection of the subcellular localization
of endogenous proteins in primary and lateral root meristems without the need
of time-consuming crosses or making translational fusions to fluorescent proteins.
alternative_title:
- Methods in Molecular Biology
author:
- first_name: Michael
full_name: Karampelias, Michael
last_name: Karampelias
- first_name: Ricardo
full_name: Tejos, Ricardo
last_name: Tejos
- first_name: Jirí
full_name: Friml, Jirí
id: 4159519E-F248-11E8-B48F-1D18A9856A87
last_name: Friml
orcid: 0000-0002-8302-7596
- first_name: Steffen
full_name: Vanneste, Steffen
last_name: Vanneste
citation:
ama: 'Karampelias M, Tejos R, Friml J, Vanneste S. Optimized whole mount in situ
immunolocalization for Arabidopsis thaliana root meristems and lateral root primordia.
In: Ristova D, Barbez E, eds. Root Development. Methods and Protocols.
Vol 1761. MIMB. Springer; 2018:131-143. doi:10.1007/978-1-4939-7747-5_10'
apa: Karampelias, M., Tejos, R., Friml, J., & Vanneste, S. (2018). Optimized
whole mount in situ immunolocalization for Arabidopsis thaliana root meristems
and lateral root primordia. In D. Ristova & E. Barbez (Eds.), Root Development.
Methods and Protocols (Vol. 1761, pp. 131–143). Springer. https://doi.org/10.1007/978-1-4939-7747-5_10
chicago: Karampelias, Michael, Ricardo Tejos, Jiří Friml, and Steffen Vanneste.
“Optimized Whole Mount in Situ Immunolocalization for Arabidopsis Thaliana Root
Meristems and Lateral Root Primordia.” In Root Development. Methods and Protocols,
edited by Daniela Ristova and Elke Barbez, 1761:131–43. MIMB. Springer, 2018.
https://doi.org/10.1007/978-1-4939-7747-5_10.
ieee: M. Karampelias, R. Tejos, J. Friml, and S. Vanneste, “Optimized whole mount
in situ immunolocalization for Arabidopsis thaliana root meristems and lateral
root primordia,” in Root Development. Methods and Protocols, vol. 1761,
D. Ristova and E. Barbez, Eds. Springer, 2018, pp. 131–143.
ista: 'Karampelias M, Tejos R, Friml J, Vanneste S. 2018.Optimized whole mount in
situ immunolocalization for Arabidopsis thaliana root meristems and lateral root
primordia. In: Root Development. Methods and Protocols. Methods in Molecular Biology,
vol. 1761, 131–143.'
mla: Karampelias, Michael, et al. “Optimized Whole Mount in Situ Immunolocalization
for Arabidopsis Thaliana Root Meristems and Lateral Root Primordia.” Root
Development. Methods and Protocols, edited by Daniela Ristova and Elke Barbez,
vol. 1761, Springer, 2018, pp. 131–43, doi:10.1007/978-1-4939-7747-5_10.
short: M. Karampelias, R. Tejos, J. Friml, S. Vanneste, in:, D. Ristova, E. Barbez
(Eds.), Root Development. Methods and Protocols, Springer, 2018, pp. 131–143.
date_created: 2018-12-11T11:46:20Z
date_published: 2018-03-11T00:00:00Z
date_updated: 2021-01-12T07:54:34Z
day: '11'
department:
- _id: JiFr
doi: 10.1007/978-1-4939-7747-5_10
editor:
- first_name: Daniela
full_name: Ristova, Daniela
last_name: Ristova
- first_name: Elke
full_name: Barbez, Elke
last_name: Barbez
intvolume: ' 1761'
language:
- iso: eng
month: '03'
oa_version: None
page: 131 - 143
publication: Root Development. Methods and Protocols
publication_status: published
publisher: Springer
publist_id: '7418'
quality_controlled: '1'
scopus_import: 1
series_title: MIMB
status: public
title: Optimized whole mount in situ immunolocalization for Arabidopsis thaliana root
meristems and lateral root primordia
type: book_chapter
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 1761
year: '2018'
...
---
_id: '412'
abstract:
- lang: eng
text: Clathrin-mediated endocytosis (CME) is a cellular trafficking process in which
cargoes and lipids are internalized from the plasma membrane into vesicles coated
with clathrin and adaptor proteins. CME is essential for many developmental and
physiological processes in plants, but its underlying mechanism is not well characterised
compared to that in yeast and animal systems. Here, we searched for new factors
involved in CME in Arabidopsis thaliana by performing Tandem Affinity Purification
of proteins that interact with clathrin light chain, a principal component of
the clathrin coat. Among the confirmed interactors, we found two putative homologues
of the clathrin-coat uncoating factor auxilin previously described in non-plant
systems. Overexpression of AUXILIN-LIKE1 and AUXILIN-LIKE2 in A. thaliana caused
an arrest of seedling growth and development. This was concomitant with inhibited
endocytosis due to blocking of clathrin recruitment after the initial step of
adaptor protein binding to the plasma membrane. By contrast, auxilin-like(1/2)
loss-of-function lines did not present endocytosis-related developmental or cellular
phenotypes under normal growth conditions. This work contributes to the on-going
characterization of the endocytotic machinery in plants and provides a robust
tool for conditionally and specifically interfering with CME in A. thaliana.
acknowledgement: We thank James Matthew Watson, Monika Borowska, and Peggy Stolt-Bergner
at ProTech Facility of the Vienna Biocenter Core Facilities for the CRISPR/CAS9
construct; Anna Müller for assistance with molecular cloning; Sebastian Bednarek,
Liwen Jiang, and Daniël Van Damme for sharing published material; Matyáš Fendrych,
Daniël Van Damme, and Lindy Abas for valuable discussions; and Martine De Cock for
help with correcting the manuscript. This work was supported by the European Research
Council under the European Union Seventh Framework Programme (FP7/2007-2013)/ERC
Grant 282300 and by the Ministry of Education of the Czech Republic/MŠMT project
NPUI-LO1417.
article_processing_charge: No
article_type: original
author:
- first_name: Maciek
full_name: Adamowski, Maciek
id: 45F536D2-F248-11E8-B48F-1D18A9856A87
last_name: Adamowski
orcid: 0000-0001-6463-5257
- first_name: Madhumitha
full_name: Narasimhan, Madhumitha
id: 44BF24D0-F248-11E8-B48F-1D18A9856A87
last_name: Narasimhan
orcid: 0000-0002-8600-0671
- first_name: Urszula
full_name: Kania, Urszula
id: 4AE5C486-F248-11E8-B48F-1D18A9856A87
last_name: Kania
- first_name: Matous
full_name: Glanc, Matous
id: 1AE1EA24-02D0-11E9-9BAA-DAF4881429F2
last_name: Glanc
orcid: 0000-0003-0619-7783
- first_name: Geert
full_name: De Jaeger, Geert
last_name: De Jaeger
- first_name: Jirí
full_name: Friml, Jirí
id: 4159519E-F248-11E8-B48F-1D18A9856A87
last_name: Friml
orcid: 0000-0002-8302-7596
citation:
ama: Adamowski M, Narasimhan M, Kania U, Glanc M, De Jaeger G, Friml J. A functional
study of AUXILIN LIKE1 and 2 two putative clathrin uncoating factors in Arabidopsis.
The Plant Cell. 2018;30(3):700-716. doi:10.1105/tpc.17.00785
apa: Adamowski, M., Narasimhan, M., Kania, U., Glanc, M., De Jaeger, G., & Friml,
J. (2018). A functional study of AUXILIN LIKE1 and 2 two putative clathrin uncoating
factors in Arabidopsis. The Plant Cell. American Society of Plant Biologists.
https://doi.org/10.1105/tpc.17.00785
chicago: Adamowski, Maciek, Madhumitha Narasimhan, Urszula Kania, Matous Glanc,
Geert De Jaeger, and Jiří Friml. “A Functional Study of AUXILIN LIKE1 and 2 Two
Putative Clathrin Uncoating Factors in Arabidopsis.” The Plant Cell. American
Society of Plant Biologists, 2018. https://doi.org/10.1105/tpc.17.00785.
ieee: M. Adamowski, M. Narasimhan, U. Kania, M. Glanc, G. De Jaeger, and J. Friml,
“A functional study of AUXILIN LIKE1 and 2 two putative clathrin uncoating factors
in Arabidopsis,” The Plant Cell, vol. 30, no. 3. American Society of Plant
Biologists, pp. 700–716, 2018.
ista: Adamowski M, Narasimhan M, Kania U, Glanc M, De Jaeger G, Friml J. 2018. A
functional study of AUXILIN LIKE1 and 2 two putative clathrin uncoating factors
in Arabidopsis. The Plant Cell. 30(3), 700–716.
mla: Adamowski, Maciek, et al. “A Functional Study of AUXILIN LIKE1 and 2 Two Putative
Clathrin Uncoating Factors in Arabidopsis.” The Plant Cell, vol. 30, no.
3, American Society of Plant Biologists, 2018, pp. 700–16, doi:10.1105/tpc.17.00785.
short: M. Adamowski, M. Narasimhan, U. Kania, M. Glanc, G. De Jaeger, J. Friml,
The Plant Cell 30 (2018) 700–716.
date_created: 2018-12-11T11:46:20Z
date_published: 2018-04-09T00:00:00Z
date_updated: 2025-05-07T11:12:27Z
day: '09'
ddc:
- '580'
department:
- _id: JiFr
doi: 10.1105/tpc.17.00785
ec_funded: 1
external_id:
isi:
- '000429441400018'
pmid:
- '29511054'
file:
- access_level: open_access
checksum: 4e165e653b67d3f0684697f21aace5a1
content_type: application/pdf
creator: dernst
date_created: 2022-05-23T09:12:38Z
date_updated: 2022-05-23T09:12:38Z
file_id: '11406'
file_name: 2018_PlantCell_Adamowski.pdf
file_size: 4407538
relation: main_file
success: 1
file_date_updated: 2022-05-23T09:12:38Z
has_accepted_license: '1'
intvolume: ' 30'
isi: 1
issue: '3'
language:
- iso: eng
month: '04'
oa: 1
oa_version: Published Version
page: 700 - 716
pmid: 1
project:
- _id: 25716A02-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '282300'
name: Polarity and subcellular dynamics in plants
publication: The Plant Cell
publication_identifier:
eissn:
- 1532-298X
issn:
- 1040-4651
publication_status: published
publisher: American Society of Plant Biologists
publist_id: '7417'
quality_controlled: '1'
related_material:
record:
- id: '6269'
relation: dissertation_contains
status: public
scopus_import: '1'
status: public
title: A functional study of AUXILIN LIKE1 and 2 two putative clathrin uncoating factors
in Arabidopsis
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 30
year: '2018'
...
---
_id: '413'
abstract:
- lang: eng
text: Being cared for when sick is a benefit of sociality that can reduce disease
and improve survival of group members. However, individuals providing care risk
contracting infectious diseases themselves. If they contract a low pathogen dose,
they may develop low-level infections that do not cause disease but still affect
host immunity by either decreasing or increasing the host’s vulnerability to subsequent
infections. Caring for contagious individuals can thus significantly alter the
future disease susceptibility of caregivers. Using ants and their fungal pathogens
as a model system, we tested if the altered disease susceptibility of experienced
caregivers, in turn, affects their expression of sanitary care behavior. We found
that low-level infections contracted during sanitary care had protective or neutral
effects on secondary exposure to the same (homologous) pathogen but consistently
caused high mortality on superinfection with a different (heterologous) pathogen.
In response to this risk, the ants selectively adjusted the expression of their
sanitary care. Specifically, the ants performed less grooming and more antimicrobial
disinfection when caring for nestmates contaminated with heterologous pathogens
compared with homologous ones. By modulating the components of sanitary care in
this way the ants acquired less infectious particles of the heterologous pathogens,
resulting in reduced superinfection. The performance of risk-adjusted sanitary
care reveals the remarkable capacity of ants to react to changes in their disease
susceptibility, according to their own infection history and to flexibly adjust
collective care to individual risk.
article_processing_charge: No
author:
- first_name: Matthias
full_name: Konrad, Matthias
id: 46528076-F248-11E8-B48F-1D18A9856A87
last_name: Konrad
- first_name: Christopher
full_name: Pull, Christopher
id: 3C7F4840-F248-11E8-B48F-1D18A9856A87
last_name: Pull
orcid: 0000-0003-1122-3982
- first_name: Sina
full_name: Metzler, Sina
id: 48204546-F248-11E8-B48F-1D18A9856A87
last_name: Metzler
orcid: 0000-0002-9547-2494
- first_name: Katharina
full_name: Seif, Katharina
id: 90F7894A-02CF-11E9-976E-E38CFE5CBC1D
last_name: Seif
- first_name: Elisabeth
full_name: Naderlinger, Elisabeth
id: 31757262-F248-11E8-B48F-1D18A9856A87
last_name: Naderlinger
- first_name: Anna V
full_name: Grasse, Anna V
id: 406F989C-F248-11E8-B48F-1D18A9856A87
last_name: Grasse
- first_name: Sylvia
full_name: Cremer, Sylvia
id: 2F64EC8C-F248-11E8-B48F-1D18A9856A87
last_name: Cremer
orcid: 0000-0002-2193-3868
citation:
ama: Konrad M, Pull C, Metzler S, et al. Ants avoid superinfections by performing
risk-adjusted sanitary care. PNAS. 2018;115(11):2782-2787. doi:10.1073/pnas.1713501115
apa: Konrad, M., Pull, C., Metzler, S., Seif, K., Naderlinger, E., Grasse, A. V.,
& Cremer, S. (2018). Ants avoid superinfections by performing risk-adjusted
sanitary care. PNAS. National Academy of Sciences. https://doi.org/10.1073/pnas.1713501115
chicago: Konrad, Matthias, Christopher Pull, Sina Metzler, Katharina Seif, Elisabeth
Naderlinger, Anna V Grasse, and Sylvia Cremer. “Ants Avoid Superinfections by
Performing Risk-Adjusted Sanitary Care.” PNAS. National Academy of Sciences,
2018. https://doi.org/10.1073/pnas.1713501115.
ieee: M. Konrad et al., “Ants avoid superinfections by performing risk-adjusted
sanitary care,” PNAS, vol. 115, no. 11. National Academy of Sciences, pp.
2782–2787, 2018.
ista: Konrad M, Pull C, Metzler S, Seif K, Naderlinger E, Grasse AV, Cremer S. 2018.
Ants avoid superinfections by performing risk-adjusted sanitary care. PNAS. 115(11),
2782–2787.
mla: Konrad, Matthias, et al. “Ants Avoid Superinfections by Performing Risk-Adjusted
Sanitary Care.” PNAS, vol. 115, no. 11, National Academy of Sciences, 2018,
pp. 2782–87, doi:10.1073/pnas.1713501115.
short: M. Konrad, C. Pull, S. Metzler, K. Seif, E. Naderlinger, A.V. Grasse, S.
Cremer, PNAS 115 (2018) 2782–2787.
date_created: 2018-12-11T11:46:20Z
date_published: 2018-03-13T00:00:00Z
date_updated: 2023-09-08T13:22:21Z
day: '13'
department:
- _id: SyCr
doi: 10.1073/pnas.1713501115
ec_funded: 1
external_id:
isi:
- '000427245400069'
pmid:
- '29463746'
intvolume: ' 115'
isi: 1
issue: '11'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://www.ncbi.nlm.nih.gov/pubmed/29463746
month: '03'
oa: 1
oa_version: Published Version
page: 2782 - 2787
pmid: 1
project:
- _id: 25DC711C-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '243071'
name: 'Social Vaccination in Ant Colonies: from Individual Mechanisms to Society
Effects'
publication: PNAS
publication_status: published
publisher: National Academy of Sciences
publist_id: '7416'
quality_controlled: '1'
related_material:
link:
- description: News on IST Homepage
relation: press_release
url: https://ist.ac.at/en/news/helping-in-spite-of-risk-ants-perform-risk-averse-sanitary-care-of-infectious-nest-mates/
scopus_import: '1'
status: public
title: Ants avoid superinfections by performing risk-adjusted sanitary care
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 115
year: '2018'
...
---
_id: '415'
abstract:
- lang: eng
text: Recently it was shown that a molecule rotating in a quantum solvent can be
described in terms of the “angulon” quasiparticle [M. Lemeshko, Phys. Rev. Lett.
118, 095301 (2017)]. Here we extend the angulon theory to the case of molecules
possessing an additional spin-1/2 degree of freedom and study the behavior of
the system in the presence of a static magnetic field. We show that exchange of
angular momentum between the molecule and the solvent can be altered by the field,
even though the solvent itself is non-magnetic. In particular, we demonstrate
a possibility to control resonant emission of phonons with a given angular momentum
using a magnetic field.
acknowledgement: "We acknowledge insightful discussions with Giacomo Bighin, Igor
Cherepanov, Johan Mentink, and Enderalp Yakaboylu. This work was supported by the
Austrian Science Fund (FWF), Project No. P29902-N27. W.R. was supported by the Polish
Ministry of Science and Higher Education Grant No. MNISW/2016/DIR/285/NN and by
the European Union’s Horizon 2020 research and innovation programme under the Marie
Skłodowska-Curie Grant Agreement No. 665385.\r\n"
article_number: '104307'
article_processing_charge: No
article_type: original
arxiv: 1
author:
- first_name: Wojciech
full_name: Rzadkowski, Wojciech
id: 48C55298-F248-11E8-B48F-1D18A9856A87
last_name: Rzadkowski
orcid: 0000-0002-1106-4419
- first_name: Mikhail
full_name: Lemeshko, Mikhail
id: 37CB05FA-F248-11E8-B48F-1D18A9856A87
last_name: Lemeshko
orcid: 0000-0002-6990-7802
citation:
ama: Rzadkowski W, Lemeshko M. Effect of a magnetic field on molecule–solvent angular
momentum transfer. The Journal of Chemical Physics. 2018;148(10). doi:10.1063/1.5017591
apa: Rzadkowski, W., & Lemeshko, M. (2018). Effect of a magnetic field on molecule–solvent
angular momentum transfer. The Journal of Chemical Physics. AIP Publishing.
https://doi.org/10.1063/1.5017591
chicago: Rzadkowski, Wojciech, and Mikhail Lemeshko. “Effect of a Magnetic Field
on Molecule–Solvent Angular Momentum Transfer.” The Journal of Chemical Physics.
AIP Publishing, 2018. https://doi.org/10.1063/1.5017591.
ieee: W. Rzadkowski and M. Lemeshko, “Effect of a magnetic field on molecule–solvent
angular momentum transfer,” The Journal of Chemical Physics, vol. 148,
no. 10. AIP Publishing, 2018.
ista: Rzadkowski W, Lemeshko M. 2018. Effect of a magnetic field on molecule–solvent
angular momentum transfer. The Journal of Chemical Physics. 148(10), 104307.
mla: Rzadkowski, Wojciech, and Mikhail Lemeshko. “Effect of a Magnetic Field on
Molecule–Solvent Angular Momentum Transfer.” The Journal of Chemical Physics,
vol. 148, no. 10, 104307, AIP Publishing, 2018, doi:10.1063/1.5017591.
short: W. Rzadkowski, M. Lemeshko, The Journal of Chemical Physics 148 (2018).
date_created: 2018-12-11T11:46:21Z
date_published: 2018-03-14T00:00:00Z
date_updated: 2024-02-28T13:01:59Z
day: '14'
department:
- _id: MiLe
doi: 10.1063/1.5017591
ec_funded: 1
external_id:
arxiv:
- '1711.09904'
isi:
- '000427517200065'
intvolume: ' 148'
isi: 1
issue: '10'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://arxiv.org/abs/1711.09904
month: '03'
oa: 1
oa_version: Preprint
project:
- _id: 26031614-B435-11E9-9278-68D0E5697425
call_identifier: FWF
grant_number: P29902
name: Quantum rotations in the presence of a many-body environment
- _id: 2564DBCA-B435-11E9-9278-68D0E5697425
call_identifier: H2020
grant_number: '665385'
name: International IST Doctoral Program
publication: The Journal of Chemical Physics
publication_status: published
publisher: AIP Publishing
publist_id: '7408'
quality_controlled: '1'
related_material:
record:
- id: '10759'
relation: dissertation_contains
status: public
scopus_import: '1'
status: public
title: Effect of a magnetic field on molecule–solvent angular momentum transfer
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 148
year: '2018'
...
---
_id: '417'
abstract:
- lang: eng
text: 'We introduce a Diagrammatic Monte Carlo (DiagMC) approach to complex molecular
impurities with rotational degrees of freedom interacting with a many-particle
environment. The treatment is based on the diagrammatic expansion that merges
the usual Feynman diagrams with the angular momentum diagrams known from atomic
and nuclear structure theory, thereby incorporating the non-Abelian algebra inherent
to quantum rotations. Our approach works at arbitrary coupling, is free of systematic
errors and of finite size effects, and naturally provides access to the impurity
Green function. We exemplify the technique by obtaining an all-coupling solution
of the angulon model, however, the method is quite general and can be applied
to a broad variety of quantum impurities possessing angular momentum degrees of
freedom. '
article_number: '165301'
article_processing_charge: No
arxiv: 1
author:
- first_name: Giacomo
full_name: Bighin, Giacomo
id: 4CA96FD4-F248-11E8-B48F-1D18A9856A87
last_name: Bighin
orcid: 0000-0001-8823-9777
- first_name: Timur
full_name: Tscherbul, Timur
last_name: Tscherbul
- first_name: Mikhail
full_name: Lemeshko, Mikhail
id: 37CB05FA-F248-11E8-B48F-1D18A9856A87
last_name: Lemeshko
orcid: 0000-0002-6990-7802
citation:
ama: Bighin G, Tscherbul T, Lemeshko M. Diagrammatic Monte Carlo approach to rotating
molecular impurities. Physical Review Letters. 2018;121(16). doi:10.1103/PhysRevLett.121.165301
apa: Bighin, G., Tscherbul, T., & Lemeshko, M. (2018). Diagrammatic Monte Carlo
approach to rotating molecular impurities. Physical Review Letters. American
Physical Society. https://doi.org/10.1103/PhysRevLett.121.165301
chicago: Bighin, Giacomo, Timur Tscherbul, and Mikhail Lemeshko. “Diagrammatic Monte
Carlo Approach to Rotating Molecular Impurities.” Physical Review Letters.
American Physical Society, 2018. https://doi.org/10.1103/PhysRevLett.121.165301.
ieee: G. Bighin, T. Tscherbul, and M. Lemeshko, “Diagrammatic Monte Carlo approach
to rotating molecular impurities,” Physical Review Letters, vol. 121, no.
16. American Physical Society, 2018.
ista: Bighin G, Tscherbul T, Lemeshko M. 2018. Diagrammatic Monte Carlo approach
to rotating molecular impurities. Physical Review Letters. 121(16), 165301.
mla: Bighin, Giacomo, et al. “Diagrammatic Monte Carlo Approach to Rotating Molecular
Impurities.” Physical Review Letters, vol. 121, no. 16, 165301, American
Physical Society, 2018, doi:10.1103/PhysRevLett.121.165301.
short: G. Bighin, T. Tscherbul, M. Lemeshko, Physical Review Letters 121 (2018).
date_created: 2018-12-11T11:46:22Z
date_published: 2018-10-16T00:00:00Z
date_updated: 2024-02-28T13:14:53Z
day: '16'
department:
- _id: MiLe
doi: 10.1103/PhysRevLett.121.165301
external_id:
arxiv:
- '1803.07990'
intvolume: ' 121'
issue: '16'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://arxiv.org/abs/1803.07990
month: '10'
oa: 1
oa_version: Preprint
project:
- _id: 26031614-B435-11E9-9278-68D0E5697425
call_identifier: FWF
grant_number: P29902
name: Quantum rotations in the presence of a many-body environment
publication: Physical Review Letters
publication_status: published
publisher: American Physical Society
publist_id: '8025'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Diagrammatic Monte Carlo approach to rotating molecular impurities
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 121
year: '2018'
...
---
_id: '418'
abstract:
- lang: eng
text: "The aim of this thesis was the development of new strategies for optical
and optogenetic control of proliferative and pro-survival signaling, and characterizing
them from the molecular mechanism up to cellular effects. These new light-based
methods have unique features, such as red light as an activator, or the avoidance
of gene delivery, which enable to overcome current limitations, such as light
delivery to target tissues and feasibility as therapeutic approach. A special
focus was placed on implementing these new light-based approaches in pancreatic
β-cells, as β-cells are the key players in diabetes and especially their loss
in number negatively affects disease progression. Currently no treatment options
are available to compensate the lack of functional β-cells in diabetic patients.\r\nIn
a first approach, red-light-activated growth factor receptors, in particular receptor
tyrosine kinases were engineered and characterized. Receptor activation with light
allows spatio-temporal control compared to ligand-based activation, and especially
red light exhibits deeper tissue penetration than other wavelengths of the visible
spectrum. Red-light-activated receptor tyrosine kinases robustly activated major
growth factor related signaling pathways with a high temporal resolution. Moreover,
the remote activation of the proliferative MAPK/Erk pathway by red-light-activated
receptor tyrosine kinases in a pancreatic β-cell line was also achieved, through
one centimeter thick mouse tissue. Although red-light-activated receptor tyrosine
kinases are particularly attractive for applications in animal models due to the
deep tissue penetration of red light, a drawback, especially with regard to translation
into humans, is the requirement of gene therapy.\r\nIn a second approach an endogenous
light-sensitive mechanism was identified and its potential to promote proliferative
and pro-survival signals was explored, towards light-based tissue regeneration
without the need for gene transfer. Blue-green light illumination was found to
be sufficient for the activation of proliferation and survival promoting signaling
pathways in primary pancreatic murine and human islets. Blue-green light also
led to an increase in proliferation of primary islet cells, an effect which was
shown to be mostly β-cell specific in human islets. Moreover, it was demonstrated
that this approach of pancreatic β-cell expansion did not have any negative effect
on the β-cell function, in particular on their insulin secretion capacity. In
contrast, a trend for enhanced insulin secretion under high glucose conditions
after illumination was detected. In order to unravel the detailed characteristics
of this endogenous light-sensitive mechanism, the precise light requirements were
determined. In addition, the expression of light sensing proteins, OPN3 and rhodopsin,
was detected. The observed effects were found to be independent of handling effects
such as temperature differences and cytochrome c oxidase dependent ATP increase,
but they were found to be enhanced through the knockout of OPN3. The exact mechanism
of how islets cells sense light and the identity of the photoreceptor remains
unknown.\r\nSummarized two new light-based systems with unique features were established
that enable the activation of proliferative and pro-survival signaling pathways.
While red-light-activated receptor tyrosine kinases open a new avenue for optogenetics
research, by allowing non-invasive control of signaling in vivo, the identified
endogenous light-sensitive mechanism has the potential to be the basis of a gene
therapy-free therapeutical approach for light-based β-cell expansion."
alternative_title:
- ISTA Thesis
article_processing_charge: No
author:
- first_name: Eva
full_name: Gschaider-Reichhart, Eva
id: 3FEE232A-F248-11E8-B48F-1D18A9856A87
last_name: Gschaider-Reichhart
orcid: 0000-0002-7218-7738
citation:
ama: Gschaider-Reichhart E. Optical and optogenetic control of proliferation and
survival . 2018. doi:10.15479/AT:ISTA:th_913
apa: Gschaider-Reichhart, E. (2018). Optical and optogenetic control of proliferation
and survival . Institute of Science and Technology Austria. https://doi.org/10.15479/AT:ISTA:th_913
chicago: Gschaider-Reichhart, Eva. “Optical and Optogenetic Control of Proliferation
and Survival .” Institute of Science and Technology Austria, 2018. https://doi.org/10.15479/AT:ISTA:th_913.
ieee: E. Gschaider-Reichhart, “Optical and optogenetic control of proliferation
and survival ,” Institute of Science and Technology Austria, 2018.
ista: Gschaider-Reichhart E. 2018. Optical and optogenetic control of proliferation
and survival . Institute of Science and Technology Austria.
mla: Gschaider-Reichhart, Eva. Optical and Optogenetic Control of Proliferation
and Survival . Institute of Science and Technology Austria, 2018, doi:10.15479/AT:ISTA:th_913.
short: E. Gschaider-Reichhart, Optical and Optogenetic Control of Proliferation
and Survival , Institute of Science and Technology Austria, 2018.
date_created: 2018-12-11T11:46:22Z
date_published: 2018-01-08T00:00:00Z
date_updated: 2023-09-22T09:20:10Z
day: '08'
ddc:
- '571'
- '570'
degree_awarded: PhD
department:
- _id: HaJa
doi: 10.15479/AT:ISTA:th_913
file:
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language:
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month: '01'
oa: 1
oa_version: Published Version
page: '107'
publication_identifier:
issn:
- 2663-337X
publication_status: published
publisher: Institute of Science and Technology Austria
publist_id: '7405'
pubrep_id: '913'
related_material:
record:
- id: '1441'
relation: part_of_dissertation
status: public
- id: '1678'
relation: part_of_dissertation
status: public
- id: '2084'
relation: part_of_dissertation
status: public
- id: '1028'
relation: part_of_dissertation
status: public
status: public
supervisor:
- first_name: Harald L
full_name: Janovjak, Harald L
id: 33BA6C30-F248-11E8-B48F-1D18A9856A87
last_name: Janovjak
orcid: 0000-0002-8023-9315
title: 'Optical and optogenetic control of proliferation and survival '
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: dissertation
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
year: '2018'
...
---
_id: '419'
abstract:
- lang: eng
text: 'Reciprocity is a major factor in human social life and accounts for a large
part of cooperation in our communities. Direct reciprocity arises when repeated
interactions occur between the same individuals. The framework of iterated games
formalizes this phenomenon. Despite being introduced more than five decades ago,
the concept keeps offering beautiful surprises. Recent theoretical research driven
by new mathematical tools has proposed a remarkable dichotomy among the crucial
strategies: successful individuals either act as partners or as rivals. Rivals
strive for unilateral advantages by applying selfish or extortionate strategies.
Partners aim to share the payoff for mutual cooperation, but are ready to fight
back when being exploited. Which of these behaviours evolves depends on the environment.
Whereas small population sizes and a limited number of rounds favour rivalry,
partner strategies are selected when populations are large and relationships stable.
Only partners allow for evolution of cooperation, while the rivals’ attempt to
put themselves first leads to defection. Hilbe et al. synthesize recent theoretical
work on zero-determinant and ‘rival’ versus ‘partner’ strategies in social dilemmas.
They describe the environments under which these contrasting selfish or cooperative
strategies emerge in evolution.'
article_processing_charge: No
article_type: review
author:
- first_name: Christian
full_name: Hilbe, Christian
id: 2FDF8F3C-F248-11E8-B48F-1D18A9856A87
last_name: Hilbe
orcid: 0000-0001-5116-955X
- first_name: Krishnendu
full_name: Chatterjee, Krishnendu
id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
last_name: Chatterjee
orcid: 0000-0002-4561-241X
- first_name: Martin
full_name: Nowak, Martin
last_name: Nowak
citation:
ama: Hilbe C, Chatterjee K, Nowak M. Partners and rivals in direct reciprocity.
Nature Human Behaviour. 2018;2:469–477. doi:10.1038/s41562-018-0320-9
apa: Hilbe, C., Chatterjee, K., & Nowak, M. (2018). Partners and rivals in direct
reciprocity. Nature Human Behaviour. Nature Publishing Group. https://doi.org/10.1038/s41562-018-0320-9
chicago: Hilbe, Christian, Krishnendu Chatterjee, and Martin Nowak. “Partners and
Rivals in Direct Reciprocity.” Nature Human Behaviour. Nature Publishing
Group, 2018. https://doi.org/10.1038/s41562-018-0320-9.
ieee: C. Hilbe, K. Chatterjee, and M. Nowak, “Partners and rivals in direct reciprocity,”
Nature Human Behaviour, vol. 2. Nature Publishing Group, pp. 469–477, 2018.
ista: Hilbe C, Chatterjee K, Nowak M. 2018. Partners and rivals in direct reciprocity.
Nature Human Behaviour. 2, 469–477.
mla: Hilbe, Christian, et al. “Partners and Rivals in Direct Reciprocity.” Nature
Human Behaviour, vol. 2, Nature Publishing Group, 2018, pp. 469–477, doi:10.1038/s41562-018-0320-9.
short: C. Hilbe, K. Chatterjee, M. Nowak, Nature Human Behaviour 2 (2018) 469–477.
date_created: 2018-12-11T11:46:22Z
date_published: 2018-03-19T00:00:00Z
date_updated: 2023-09-13T09:38:54Z
day: '19'
ddc:
- '000'
department:
- _id: KrCh
doi: 10.1038/s41562-018-0320-9
ec_funded: 1
external_id:
isi:
- '000446612000016'
file:
- access_level: open_access
checksum: 571b8cc0ba14e8d5d8b18e439a9835eb
content_type: application/pdf
creator: dernst
date_created: 2019-11-19T08:19:51Z
date_updated: 2020-07-14T12:46:25Z
file_id: '7052'
file_name: 2018_NatureHumanBeh_Hilbe.pdf
file_size: 598033
relation: main_file
file_date_updated: 2020-07-14T12:46:25Z
has_accepted_license: '1'
intvolume: ' 2'
isi: 1
language:
- iso: eng
month: '03'
oa: 1
oa_version: Submitted Version
page: 469–477
project:
- _id: 2581B60A-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '279307'
name: 'Quantitative Graph Games: Theory and Applications'
- _id: 2584A770-B435-11E9-9278-68D0E5697425
call_identifier: FWF
grant_number: P 23499-N23
name: Modern Graph Algorithmic Techniques in Formal Verification
- _id: 25832EC2-B435-11E9-9278-68D0E5697425
call_identifier: FWF
grant_number: S 11407_N23
name: Rigorous Systems Engineering
- _id: 25681D80-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '291734'
name: International IST Postdoc Fellowship Programme
publication: Nature Human Behaviour
publication_status: published
publisher: Nature Publishing Group
publist_id: '7404'
quality_controlled: '1'
related_material:
link:
- relation: erratum
url: http://doi.org/10.1038/s41562-018-0342-3
scopus_import: '1'
status: public
title: Partners and rivals in direct reciprocity
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 2
year: '2018'
...
---
_id: '42'
abstract:
- lang: eng
text: Seeds derive from ovules upon fertilization and therefore the total number
of ovules determines the final seed yield, a fundamental trait in crop plants.
Among the factors that co-ordinate the process of ovule formation, the transcription
factors CUP-SHAPED COTYLEDON 1 (CUC1) and CUC2 and the hormone cytokinin (CK)
have a particularly prominent role. Indeed, the absence of both CUC1 and CUC2
causes a severe reduction in ovule number, a phenotype that can be rescued by
CK treatment. In this study, we combined CK quantification with an integrative
genome-wide target identification approach to select Arabidopsis genes regulated
by CUCs that are also involved in CK metabolism. We focused our attention on the
functional characterization of UDP-GLUCOSYL TRANSFERASE 85A3 (UGT85A3) and UGT73C1,
which are up-regulated in the absence of CUC1 and CUC2 and encode enzymes able
to catalyse CK inactivation by O-glucosylation. Our results demonstrate a role
for these UGTs as a link between CUCs and CK homeostasis, and highlight the importance
of CUCs and CKs in the determination of seed yield.
acknowledgement: This work was funded by the Ministry of Education, Youth and Sports
of the Czech Republic through the National Program of Sustainability (grant no.
LO1204).
article_processing_charge: No
author:
- first_name: Mara
full_name: Cucinotta, Mara
last_name: Cucinotta
- first_name: Silvia
full_name: Manrique, Silvia
last_name: Manrique
- first_name: Candela
full_name: Cuesta, Candela
id: 33A3C818-F248-11E8-B48F-1D18A9856A87
last_name: Cuesta
orcid: 0000-0003-1923-2410
- first_name: Eva
full_name: Benková, Eva
id: 38F4F166-F248-11E8-B48F-1D18A9856A87
last_name: Benková
orcid: 0000-0002-8510-9739
- first_name: Ondřej
full_name: Novák, Ondřej
last_name: Novák
- first_name: Lucia
full_name: Colombo, Lucia
last_name: Colombo
citation:
ama: Cucinotta M, Manrique S, Cuesta C, Benková E, Novák O, Colombo L. Cup-shaped
Cotyledon1 (CUC1) and CU2 regulate cytokinin homeostasis to determine ovule number
in arabidopsis. Journal of Experimental Botany. 2018;69(21):5169-5176.
doi:10.1093/jxb/ery281
apa: Cucinotta, M., Manrique, S., Cuesta, C., Benková, E., Novák, O., & Colombo,
L. (2018). Cup-shaped Cotyledon1 (CUC1) and CU2 regulate cytokinin homeostasis
to determine ovule number in arabidopsis. Journal of Experimental Botany.
Oxford University Press. https://doi.org/10.1093/jxb/ery281
chicago: Cucinotta, Mara, Silvia Manrique, Candela Cuesta, Eva Benková, Ondřej Novák,
and Lucia Colombo. “Cup-Shaped Cotyledon1 (CUC1) and CU2 Regulate Cytokinin Homeostasis
to Determine Ovule Number in Arabidopsis.” Journal of Experimental Botany.
Oxford University Press, 2018. https://doi.org/10.1093/jxb/ery281.
ieee: M. Cucinotta, S. Manrique, C. Cuesta, E. Benková, O. Novák, and L. Colombo,
“Cup-shaped Cotyledon1 (CUC1) and CU2 regulate cytokinin homeostasis to determine
ovule number in arabidopsis,” Journal of Experimental Botany, vol. 69,
no. 21. Oxford University Press, pp. 5169–5176, 2018.
ista: Cucinotta M, Manrique S, Cuesta C, Benková E, Novák O, Colombo L. 2018. Cup-shaped
Cotyledon1 (CUC1) and CU2 regulate cytokinin homeostasis to determine ovule number
in arabidopsis. Journal of Experimental Botany. 69(21), 5169–5176.
mla: Cucinotta, Mara, et al. “Cup-Shaped Cotyledon1 (CUC1) and CU2 Regulate Cytokinin
Homeostasis to Determine Ovule Number in Arabidopsis.” Journal of Experimental
Botany, vol. 69, no. 21, Oxford University Press, 2018, pp. 5169–76, doi:10.1093/jxb/ery281.
short: M. Cucinotta, S. Manrique, C. Cuesta, E. Benková, O. Novák, L. Colombo, Journal
of Experimental Botany 69 (2018) 5169–5176.
date_created: 2018-12-11T11:44:19Z
date_published: 2018-07-26T00:00:00Z
date_updated: 2023-09-11T12:52:03Z
day: '26'
ddc:
- '575'
department:
- _id: EvBe
doi: 10.1093/jxb/ery281
external_id:
isi:
- '000448163900015'
file:
- access_level: open_access
checksum: ca3b6711040b1662488aeb3d1f961f13
content_type: application/pdf
creator: dernst
date_created: 2018-12-17T10:44:16Z
date_updated: 2020-07-14T12:46:25Z
file_id: '5691'
file_name: 2018_JournalExperimBotany_Cucinotta.pdf
file_size: 1292128
relation: main_file
file_date_updated: 2020-07-14T12:46:25Z
has_accepted_license: '1'
intvolume: ' 69'
isi: 1
issue: '21'
language:
- iso: eng
month: '07'
oa: 1
oa_version: Published Version
page: 5169 - 5176
publication: Journal of Experimental Botany
publication_status: published
publisher: Oxford University Press
publist_id: '8012'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Cup-shaped Cotyledon1 (CUC1) and CU2 regulate cytokinin homeostasis to determine
ovule number in arabidopsis
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 69
year: '2018'
...
---
_id: '420'
abstract:
- lang: eng
text: We analyze the theoretical derivation of the beyond-mean-field equation of
state for two-dimensional gas of dilute, ultracold alkali-metal atoms in the Bardeen–Cooper–Schrieffer
(BCS) to Bose–Einstein condensate (BEC) crossover. We show that at zero temperature
our theory — considering Gaussian fluctuations on top of the mean-field equation
of state — is in very good agreement with experimental data. Subsequently, we
investigate the superfluid density at finite temperature and its renormalization
due to the proliferation of vortex–antivortex pairs. By doing so, we determine
the Berezinskii–Kosterlitz–Thouless (BKT) critical temperature — at which the
renormalized superfluid density jumps to zero — as a function of the inter-atomic
potential strength. We find that the Nelson–Kosterlitz criterion overestimates
the BKT temperature with respect to the renormalization group equations, this
effect being particularly relevant in the intermediate regime of the crossover.
article_processing_charge: No
author:
- first_name: Giacomo
full_name: Bighin, Giacomo
id: 4CA96FD4-F248-11E8-B48F-1D18A9856A87
last_name: Bighin
orcid: 0000-0001-8823-9777
- first_name: Luca
full_name: Salasnich, Luca
last_name: Salasnich
citation:
ama: Bighin G, Salasnich L. Renormalization of the superfluid density in the two-dimensional
BCS-BEC crossover. International Journal of Modern Physics B. 2018;32(17):1840022.
doi:10.1142/S0217979218400222
apa: Bighin, G., & Salasnich, L. (2018). Renormalization of the superfluid density
in the two-dimensional BCS-BEC crossover. International Journal of Modern Physics
B. World Scientific Publishing. https://doi.org/10.1142/S0217979218400222
chicago: Bighin, Giacomo, and Luca Salasnich. “Renormalization of the Superfluid
Density in the Two-Dimensional BCS-BEC Crossover.” International Journal of
Modern Physics B. World Scientific Publishing, 2018. https://doi.org/10.1142/S0217979218400222.
ieee: G. Bighin and L. Salasnich, “Renormalization of the superfluid density in
the two-dimensional BCS-BEC crossover,” International Journal of Modern Physics
B, vol. 32, no. 17. World Scientific Publishing, p. 1840022, 2018.
ista: Bighin G, Salasnich L. 2018. Renormalization of the superfluid density in
the two-dimensional BCS-BEC crossover. International Journal of Modern Physics
B. 32(17), 1840022.
mla: Bighin, Giacomo, and Luca Salasnich. “Renormalization of the Superfluid Density
in the Two-Dimensional BCS-BEC Crossover.” International Journal of Modern
Physics B, vol. 32, no. 17, World Scientific Publishing, 2018, p. 1840022,
doi:10.1142/S0217979218400222.
short: G. Bighin, L. Salasnich, International Journal of Modern Physics B 32 (2018)
1840022.
date_created: 2018-12-11T11:46:22Z
date_published: 2018-07-10T00:00:00Z
date_updated: 2023-09-18T08:09:59Z
day: '10'
department:
- _id: MiLe
doi: 10.1142/S0217979218400222
external_id:
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intvolume: ' 32'
isi: 1
issue: '17'
language:
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url: https://arxiv.org/abs/1710.11171
month: '07'
oa: 1
oa_version: Preprint
page: '1840022'
publication: International Journal of Modern Physics B
publication_status: published
publisher: World Scientific Publishing
publist_id: '7402'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Renormalization of the superfluid density in the two-dimensional BCS-BEC crossover
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 32
year: '2018'
...