---
_id: '3447'
author:
- first_name: Krishnendu
full_name: Krishnendu Chatterjee
id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
last_name: Chatterjee
orcid: 0000-0002-4561-241X
- first_name: Pallab
full_name: Dasgupta, Pallab
last_name: Dasgupta
- first_name: Partha
full_name: Chakrabarti, Partha P
last_name: Chakrabarti
citation:
ama: 'Chatterjee K, Dasgupta P, Chakrabarti P. Weighted quantified computation tree
logic. In: Elsevier; 2001.'
apa: 'Chatterjee, K., Dasgupta, P., & Chakrabarti, P. (2001). Weighted quantified
computation tree logic. Presented at the CIT: Conference on Information Technology,
Elsevier.'
chicago: Chatterjee, Krishnendu, Pallab Dasgupta, and Partha Chakrabarti. “Weighted
Quantified Computation Tree Logic.” Elsevier, 2001.
ieee: 'K. Chatterjee, P. Dasgupta, and P. Chakrabarti, “Weighted quantified computation
tree logic,” presented at the CIT: Conference on Information Technology, 2001.'
ista: 'Chatterjee K, Dasgupta P, Chakrabarti P. 2001. Weighted quantified computation
tree logic. CIT: Conference on Information Technology.'
mla: Chatterjee, Krishnendu, et al. Weighted Quantified Computation Tree Logic.
Elsevier, 2001.
short: K. Chatterjee, P. Dasgupta, P. Chakrabarti, in:, Elsevier, 2001.
conference:
name: 'CIT: Conference on Information Technology'
date_created: 2018-12-11T12:03:23Z
date_published: 2001-11-14T00:00:00Z
date_updated: 2021-01-12T07:43:31Z
day: '14'
extern: 1
month: '11'
publication_status: published
publisher: Elsevier
publist_id: '2940'
quality_controlled: 0
status: public
title: Weighted quantified computation tree logic
type: conference
year: '2001'
...
---
_id: '3493'
abstract:
- lang: eng
text: Although agonists and competitive antagonists presumably occupy overlapping
binding sites on ligand-gated channels, these interactions cannot be identical
because agonists cause channel opening whereas antagonists do not. One explanation
is that only agonist binding performs enough work on the receptor to cause the
conformational changes that lead to gating. This idea is supported by agonist
binding rates at GABAA and nicotinic acetylcholine receptors that are slower than
expected for a diffusion-limited process, suggesting that agonist binding involves
an energy-requiring event. This hypothesis predicts that competitive antagonist
binding should require less activation energy than agonist binding. To test this
idea, we developed a novel deconvolution-based method to compare binding and unbinding
kinetics of GABAA receptor agonists and antagonists in outside-out patches from
rat hippocampal neurons. Agonist and antagonist unbinding rates were steeply correlated
with affinity. Unlike the agonists, three of the four antagonists tested had binding
rates that were fast, independent of affinity, and could be accounted for by diffusion-
and dehydration-limited processes. In contrast, agonist binding involved additional
energy-requiring steps, consistent with the idea that channel gating is initiated
by agonist-triggered movements within the ligand binding site. Antagonist binding
does not appear to produce such movements, and may in fact prevent them.
article_processing_charge: No
article_type: original
author:
- first_name: M.V
full_name: Jones, M.V
last_name: Jones
- first_name: Peter M
full_name: Jonas, Peter M
id: 353C1B58-F248-11E8-B48F-1D18A9856A87
last_name: Jonas
orcid: 0000-0001-5001-4804
- first_name: Y.
full_name: Sahara, Y.
last_name: Sahara
- first_name: G.
full_name: Westbrook, G.
last_name: Westbrook
citation:
ama: Jones M., Jonas PM, Sahara Y, Westbrook G. Microscopic kinetics and energetics
distinguish GABAA receptor agonists from antagonists. Biophysical Journal.
2001;81(5):2660-2670. doi:10.1016/S0006-3495(01)75909-7
apa: Jones, M. ., Jonas, P. M., Sahara, Y., & Westbrook, G. (2001). Microscopic
kinetics and energetics distinguish GABAA receptor agonists from antagonists.
Biophysical Journal. Biophysical Society. https://doi.org/10.1016/S0006-3495(01)75909-7
chicago: Jones, M.V, Peter M Jonas, Y. Sahara, and G. Westbrook. “Microscopic Kinetics
and Energetics Distinguish GABAA Receptor Agonists from Antagonists.” Biophysical
Journal. Biophysical Society, 2001. https://doi.org/10.1016/S0006-3495(01)75909-7 .
ieee: M. . Jones, P. M. Jonas, Y. Sahara, and G. Westbrook, “Microscopic kinetics
and energetics distinguish GABAA receptor agonists from antagonists,” Biophysical
Journal, vol. 81, no. 5. Biophysical Society, pp. 2660–2670, 2001.
ista: Jones M., Jonas PM, Sahara Y, Westbrook G. 2001. Microscopic kinetics and
energetics distinguish GABAA receptor agonists from antagonists. Biophysical Journal.
81(5), 2660–2670.
mla: Jones, M. .., et al. “Microscopic Kinetics and Energetics Distinguish GABAA
Receptor Agonists from Antagonists.” Biophysical Journal, vol. 81, no.
5, Biophysical Society, 2001, pp. 2660–70, doi:10.1016/S0006-3495(01)75909-7 .
short: M.. Jones, P.M. Jonas, Y. Sahara, G. Westbrook, Biophysical Journal 81 (2001)
2660–2670.
date_created: 2018-12-11T12:03:37Z
date_published: 2001-11-01T00:00:00Z
date_updated: 2023-05-15T13:50:21Z
day: '01'
doi: '10.1016/S0006-3495(01)75909-7 '
extern: '1'
external_id:
pmid:
- '11606279'
intvolume: ' 81'
issue: '5'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1301733/
month: '11'
oa: 1
oa_version: Published Version
page: 2660 - 2670
pmid: 1
publication: Biophysical Journal
publication_identifier:
issn:
- 0006-3495
publication_status: published
publisher: Biophysical Society
publist_id: '2894'
quality_controlled: '1'
status: public
title: Microscopic kinetics and energetics distinguish GABAA receptor agonists from
antagonists
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 81
year: '2001'
...
---
_id: '3494'
abstract:
- lang: eng
text: 'Mutual synaptic interactions between GABAergic interneurons are thought to
be of critical importance for the generation of network oscillations and for temporal
encoding of information in the hippocampus. However, the functional properties
of synaptic transmission between hippocampal interneurons are largely unknown.
We have made paired recordings from basket cells (BCs) in the dentate gyrus of
rat hippocampal slices, followed by correlated light and electron microscopical
analysis. Unitary GABAAreceptor-mediated IPSCs at BC–BC synapses recorded at the
soma showed a fast rise and decay, with a mean decay time constant of 2.5 ± 0.2
msec (32°C). Synaptic transmission at BC–BC synapses showed paired-pulse depression
(PPD) (32 ± 5% for 10 msec interpulse intervals) and multiple-pulse depression
during repetitive stimulation. Detailed passive cable model simulations based
on somatodendritic morphology and localization of synaptic contacts further indicated
that the conductance change at the postsynaptic site was even faster, decaying
with a mean time constant of 1.8 ± 0.6 msec. Sequential triple recordings revealed
that the decay time course of IPSCs at BC–BC synapses was approximately twofold
faster than that at BC–granule cell synapses, whereas the extent of PPD was comparable.
To examine the consequences of the fast postsynaptic conductance change for the
generation of oscillatory activity, we developed a computational model of an interneuron
network. The model showed robust oscillations at frequencies >60 Hz if the
excitatory drive was sufficiently large. Thus the fast conductance change at interneuron–interneuron
synapses may promote the generation of high-frequency oscillations observed in
the dentate gyrusin vivo. '
acknowledgement: This work was supported by grants of the Deutsche Forschungsgemeinschaft
(SFB 505/C6) and the Human Frontiers Science Program Organization (RG0017/1998-B).
We thank Drs. M. V. Jones, J. Bischofberger, and U. Kraushaar for critically reading
this manuscript. We also thank B. Taskin and A. Roth for advice in the use of reconstruction
and modeling software, and S. Nestel, M. Winter, and A. Blomenkamp for technical
assistance.
article_processing_charge: No
article_type: original
author:
- first_name: Marlene
full_name: Bartos, Marlene
last_name: Bartos
- first_name: Imre
full_name: Vida, Imre
last_name: Vida
- first_name: Michael
full_name: Frotscher, Michael
last_name: Frotscher
- first_name: Jörg
full_name: Geiger, Jörg
last_name: Geiger
- first_name: Peter M
full_name: Jonas, Peter M
id: 353C1B58-F248-11E8-B48F-1D18A9856A87
last_name: Jonas
orcid: 0000-0001-5001-4804
citation:
ama: Bartos M, Vida I, Frotscher M, Geiger J, Jonas PM. Rapid signaling at inhibitory
synapses in a dentate gyrus interneuron network. Journal of Neuroscience.
2001;21(8):2687-2698. doi:10.1523/JNEUROSCI.21-08-02687.2001
apa: Bartos, M., Vida, I., Frotscher, M., Geiger, J., & Jonas, P. M. (2001).
Rapid signaling at inhibitory synapses in a dentate gyrus interneuron network.
Journal of Neuroscience. Society for Neuroscience. https://doi.org/10.1523/JNEUROSCI.21-08-02687.2001
chicago: Bartos, Marlene, Imre Vida, Michael Frotscher, Jörg Geiger, and Peter M
Jonas. “Rapid Signaling at Inhibitory Synapses in a Dentate Gyrus Interneuron
Network.” Journal of Neuroscience. Society for Neuroscience, 2001. https://doi.org/10.1523/JNEUROSCI.21-08-02687.2001.
ieee: M. Bartos, I. Vida, M. Frotscher, J. Geiger, and P. M. Jonas, “Rapid signaling
at inhibitory synapses in a dentate gyrus interneuron network.,” Journal of
Neuroscience, vol. 21, no. 8. Society for Neuroscience, pp. 2687–2698, 2001.
ista: Bartos M, Vida I, Frotscher M, Geiger J, Jonas PM. 2001. Rapid signaling at
inhibitory synapses in a dentate gyrus interneuron network. Journal of Neuroscience.
21(8), 2687–2698.
mla: Bartos, Marlene, et al. “Rapid Signaling at Inhibitory Synapses in a Dentate
Gyrus Interneuron Network.” Journal of Neuroscience, vol. 21, no. 8, Society
for Neuroscience, 2001, pp. 2687–98, doi:10.1523/JNEUROSCI.21-08-02687.2001.
short: M. Bartos, I. Vida, M. Frotscher, J. Geiger, P.M. Jonas, Journal of Neuroscience
21 (2001) 2687–2698.
date_created: 2018-12-11T12:03:37Z
date_published: 2001-04-15T00:00:00Z
date_updated: 2023-05-15T13:47:04Z
day: '15'
doi: 10.1523/JNEUROSCI.21-08-02687.2001
extern: '1'
external_id:
pmid:
- '11306622'
intvolume: ' 21'
issue: '8'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: ncbi.nlm.nih.gov/pmc/articles/PMC6762544/
month: '04'
oa: 1
oa_version: Published Version
page: 2687 - 2698
pmid: 1
publication: Journal of Neuroscience
publication_identifier:
issn:
- 0270-6474
publication_status: published
publisher: Society for Neuroscience
publist_id: '2893'
quality_controlled: '1'
status: public
title: Rapid signaling at inhibitory synapses in a dentate gyrus interneuron network.
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 21
year: '2001'
...
---
_id: '3495'
abstract:
- lang: eng
text: High Ca2+ permeability and its control by voltage-dependent Mg2+ block are
defining features of NMDA receptors. These features are lost if the principal
NR1 subunit carries an asparagine (N) to arginine (R) substitution in a critical
channel site at NR1 position 598. NR1(R) expression from a single allele in gene-targeted
NR1+/R mice is lethal soon after birth, precluding analysis of altered synaptic
functions later in life. We therefore employed the forebrain specific αCaMKII
promoter to drive tTA-mediated tetracyclin sensitive transcription of transgenes
for NR1(R) and for lacZ as reporter. Transgene expression was observed in cortex,
striatum, hippocampus, amygdala and olfactory bulb and was mosaic in all these
forebrain regions. It was highest in olfactory bulb granule cells, in most of
which Ca2+ permeability and voltage-dependent Mg2+ block of NMDA receptors were
reduced to different extents. This indicates significant impairment of NMDA receptor
function by NR1(R) in presence of the wild-type NR1 complement. Indeed, even though
NR1(R) mRNA constituted only 18% of the entire NR1 mRNA population in forebrain,
the transgenic mice died during adolescence unless transgene expression was suppressed
by doxycycline. Thus, glutamate receptor function can be altered in the mouse
by regulated NR1(R) transgene expression.
acknowledgement: 'This work was supported in part by grants from the 358 (1992) 36–41.Deutsche
Forschungsgemeinschaft (Bi 642 / 1-2) and the Volkswagen foundation. '
article_processing_charge: No
article_type: original
author:
- first_name: Jasna
full_name: Jerecic, Jasna
last_name: Jerecic
- first_name: Christian
full_name: Schulze, Christian
last_name: Schulze
- first_name: Peter M
full_name: Jonas, Peter M
id: 353C1B58-F248-11E8-B48F-1D18A9856A87
last_name: Jonas
orcid: 0000-0001-5001-4804
- first_name: Rolf
full_name: Sprengel, Rolf
last_name: Sprengel
- first_name: Peter
full_name: Seeburg, Peter
last_name: Seeburg
- first_name: Joseph
full_name: Bischofberger, Joseph
last_name: Bischofberger
citation:
ama: Jerecic J, Schulze C, Jonas PM, Sprengel R, Seeburg P, Bischofberger J. Impaired
NMDA receptor function in mouse olfactory bulb neurons by tetracycline-sensitive
NR1 (N598R) expression. Molecular Brain Research. 2001;94(1-2):96-104.
doi:10.1016/S0169-328X(01)00221-2
apa: Jerecic, J., Schulze, C., Jonas, P. M., Sprengel, R., Seeburg, P., & Bischofberger,
J. (2001). Impaired NMDA receptor function in mouse olfactory bulb neurons by
tetracycline-sensitive NR1 (N598R) expression. Molecular Brain Research.
Elsevier. https://doi.org/10.1016/S0169-328X(01)00221-2
chicago: Jerecic, Jasna, Christian Schulze, Peter M Jonas, Rolf Sprengel, Peter
Seeburg, and Joseph Bischofberger. “Impaired NMDA Receptor Function in Mouse Olfactory
Bulb Neurons by Tetracycline-Sensitive NR1 (N598R) Expression.” Molecular Brain
Research. Elsevier, 2001. https://doi.org/10.1016/S0169-328X(01)00221-2.
ieee: J. Jerecic, C. Schulze, P. M. Jonas, R. Sprengel, P. Seeburg, and J. Bischofberger,
“Impaired NMDA receptor function in mouse olfactory bulb neurons by tetracycline-sensitive
NR1 (N598R) expression,” Molecular Brain Research, vol. 94, no. 1–2. Elsevier,
pp. 96–104, 2001.
ista: Jerecic J, Schulze C, Jonas PM, Sprengel R, Seeburg P, Bischofberger J. 2001.
Impaired NMDA receptor function in mouse olfactory bulb neurons by tetracycline-sensitive
NR1 (N598R) expression. Molecular Brain Research. 94(1–2), 96–104.
mla: Jerecic, Jasna, et al. “Impaired NMDA Receptor Function in Mouse Olfactory
Bulb Neurons by Tetracycline-Sensitive NR1 (N598R) Expression.” Molecular Brain
Research, vol. 94, no. 1–2, Elsevier, 2001, pp. 96–104, doi:10.1016/S0169-328X(01)00221-2.
short: J. Jerecic, C. Schulze, P.M. Jonas, R. Sprengel, P. Seeburg, J. Bischofberger,
Molecular Brain Research 94 (2001) 96–104.
date_created: 2018-12-11T12:03:38Z
date_published: 2001-10-19T00:00:00Z
date_updated: 2023-05-15T13:42:32Z
day: '19'
doi: 10.1016/S0169-328X(01)00221-2
extern: '1'
external_id:
pmid:
- '11597769'
intvolume: ' 94'
issue: 1-2
language:
- iso: eng
month: '10'
oa_version: None
page: 96 - 104
pmid: 1
publication: Molecular Brain Research
publication_identifier:
issn:
- 0169-328X
publication_status: published
publisher: Elsevier
publist_id: '2892'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Impaired NMDA receptor function in mouse olfactory bulb neurons by tetracycline-sensitive
NR1 (N598R) expression
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 94
year: '2001'
...
---
_id: '3496'
abstract:
- lang: eng
text: 'The mossy fiber-CA3 pyramidal neuron synapse is a main component of the hippocampal
trisynaptic circuitry. Recent studies, however, suggested that inhibitory interneurons
are the major targets of the mossy fiber system. To study the regulation of mossy
fiber-interneuron excitation, we examined unitary and compound excitatory postsynaptic
currents in dentate gyrus basket cells, evoked by paired recording between granule
and basket cells or extracellular stimulation of mossy fiber collaterals. The
application of an associative high-frequency stimulation paradigm induced posttetanic
potentiation (PTP) followed by homosynaptic long-term potentiation (LTP). Analysis
of numbers of failures, coefficient of variation, and paired-pulse modulation
indicated that both PTP and LTP were expressed presynaptically. The Ca2+ chelator
1,2-bis(2-aminophenoxy)ethane-N,N,N′,N′-tetraacetic acid (BAPTA) did not affect
PTP or LTP at a concentration of 10 mM but attenuated LTP at a concentration of
30 mM. Both forskolin, an adenylyl cyclase activator, and phorbolester diacetate,
a protein kinase C stimulator, lead to a long-lasting increase in excitatory postsynaptic
current amplitude. H-89, a protein kinase A inhibitor, and bisindolylmaleimide,
a protein kinase C antagonist, reduced PTP, whereas only bisindolylmaleimide reduced
LTP. These results may suggest a differential contribution of protein kinase A
and C pathways to mossy fiber-interneuron plasticity. Interneuron PTP and LTP
may provide mechanisms to maintain the balance between synaptic excitation of
interneurons and that of principal neurons in the dentate gyrus-CA3 network. '
acknowledgement: We thank Drs. J. Bischofberger and M. Martina for critically reading
an earlier version of the manuscript and A. Blomenkamp for excellent technical assistance.
Supported by the Deutsche Forschungsgemeinschaft Sonderforschungsbereich 505/C5
and Human Frontiers Science Program Organization Grant RG0017/98.
article_processing_charge: No
article_type: original
author:
- first_name: Henrik
full_name: Alle, Henrik
last_name: Alle
- first_name: Peter M
full_name: Jonas, Peter M
id: 353C1B58-F248-11E8-B48F-1D18A9856A87
last_name: Jonas
orcid: 0000-0001-5001-4804
- first_name: Jörg
full_name: Geiger, Jörg
last_name: Geiger
citation:
ama: Alle H, Jonas PM, Geiger J. PTP and LTP at a hippocampal mossy fiber-interneuron
synapse. PNAS. 2001;98(25):14708-14713. doi:10.1073/pnas.251610898
apa: Alle, H., Jonas, P. M., & Geiger, J. (2001). PTP and LTP at a hippocampal
mossy fiber-interneuron synapse. PNAS. National Academy of Sciences. https://doi.org/10.1073/pnas.251610898
chicago: Alle, Henrik, Peter M Jonas, and Jörg Geiger. “PTP and LTP at a Hippocampal
Mossy Fiber-Interneuron Synapse.” PNAS. National Academy of Sciences, 2001.
https://doi.org/10.1073/pnas.251610898
.
ieee: H. Alle, P. M. Jonas, and J. Geiger, “PTP and LTP at a hippocampal mossy fiber-interneuron
synapse,” PNAS, vol. 98, no. 25. National Academy of Sciences, pp. 14708–14713,
2001.
ista: Alle H, Jonas PM, Geiger J. 2001. PTP and LTP at a hippocampal mossy fiber-interneuron
synapse. PNAS. 98(25), 14708–14713.
mla: Alle, Henrik, et al. “PTP and LTP at a Hippocampal Mossy Fiber-Interneuron
Synapse.” PNAS, vol. 98, no. 25, National Academy of Sciences, 2001, pp.
14708–13, doi:10.1073/pnas.251610898
.
short: H. Alle, P.M. Jonas, J. Geiger, PNAS 98 (2001) 14708–14713.
date_created: 2018-12-11T12:03:38Z
date_published: 2001-12-04T00:00:00Z
date_updated: 2023-05-15T11:08:08Z
day: '04'
doi: '10.1073/pnas.251610898 '
extern: '1'
external_id:
pmid:
- '11734656'
intvolume: ' 98'
issue: '25'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC64746/
month: '12'
oa: 1
oa_version: None
page: 14708 - 14713
pmid: 1
publication: PNAS
publication_identifier:
issn:
- 0027-8424
publication_status: published
publisher: National Academy of Sciences
publist_id: '2891'
quality_controlled: '1'
scopus_import: '1'
status: public
title: PTP and LTP at a hippocampal mossy fiber-interneuron synapse
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 98
year: '2001'
...
---
_id: '3507'
abstract:
- lang: eng
text: A molecular classification method is based on a space filling description
of a molecule. The three dimensional body corresponding to the space filling molecular
structure is divided into Voronoi regions to provide a basis for efficiently processing
local structural information. A Delaunay triangulation provides a basis for systematically
processing information relating to the Voronoi regions into shape descriptors
in the form of topological elements. Preferably, additional shape and/or property
descriptors are included in the classification method. The classification methods
generally are used to identify similarities between molecules that can be used
as property predictors for a variety of applications. Generally, the property
predictions are the basis for selection of compounds for incorporation into efficacy
evaluations.
applicant:
- Jie Liang, Herbert Edelsbrunner
article_processing_charge: No
author:
- first_name: Jie
full_name: Liang, Jie
last_name: Liang
- first_name: Herbert
full_name: Edelsbrunner, Herbert
id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
last_name: Edelsbrunner
orcid: 0000-0002-9823-6833
citation:
ama: Liang J, Edelsbrunner H. Molecular classification for property prediction.
2001.
apa: Liang, J., & Edelsbrunner, H. (2001). Molecular classification for property
prediction.
chicago: Liang, Jie, and Herbert Edelsbrunner. “Molecular Classification for Property
Prediction,” 2001.
ieee: J. Liang and H. Edelsbrunner, “Molecular classification for property prediction.”
2001.
ista: Liang J, Edelsbrunner H. 2001. Molecular classification for property prediction.
mla: Liang, Jie, and Herbert Edelsbrunner. Molecular Classification for Property
Prediction. 2001.
short: J. Liang, H. Edelsbrunner, (2001).
date_created: 2018-12-11T12:03:41Z
date_published: 2001-01-30T00:00:00Z
date_updated: 2022-01-05T15:12:42Z
day: '30'
extern: '1'
ipc: G16C20/70 ; G16B15/00
ipn: US6182016B1
main_file_link:
- open_access: '1'
url: https://patents.google.com/patent/US6182016B1
month: '01'
oa: 1
oa_version: Published Version
publication_date: 2001-01-30
publist_id: '2880'
status: public
title: Molecular classification for property prediction
type: patent
user_id: 8b945eb4-e2f2-11eb-945a-df72226e66a9
year: '2001'
...
---
_id: '3517'
abstract:
- lang: eng
text: 'A modular multichannel microdrive (''hyperdrive'') is described. The microdrive
uses printed circuit board technology and flexible fused silica capillaries. The
modular design allows for the fabrication of 4-32 independently movable electrodes
or `tetrodes''. The drives are re-usable and re-loading the drive with electrodes
is simple. '
article_processing_charge: No
article_type: original
author:
- first_name: Imre
full_name: Szabo, Imre
last_name: Szabo
- first_name: András
full_name: Czurkó, András
last_name: Czurkó
- first_name: Jozsef L
full_name: Csicsvari, Jozsef L
id: 3FA14672-F248-11E8-B48F-1D18A9856A87
last_name: Csicsvari
orcid: 0000-0002-5193-4036
- first_name: Hajima
full_name: Hirase, Hajima
last_name: Hirase
- first_name: Xavier
full_name: Leinekugel, Xavier
last_name: Leinekugel
- first_name: György
full_name: Buzsáki, György
last_name: Buzsáki
citation:
ama: Szabo I, Czurkó A, Csicsvari JL, Hirase H, Leinekugel X, Buzsáki G. The application
of printed circuit board technology for fabrication of multi-channel micro-drives.
Journal of Neuroscience Methods. 2001;105(1):105-110. doi:10.1016/S0165-0270(00)00362-9
apa: Szabo, I., Czurkó, A., Csicsvari, J. L., Hirase, H., Leinekugel, X., &
Buzsáki, G. (2001). The application of printed circuit board technology for fabrication
of multi-channel micro-drives. Journal of Neuroscience Methods. Elsevier.
https://doi.org/10.1016/S0165-0270(00)00362-9
chicago: Szabo, Imre, András Czurkó, Jozsef L Csicsvari, Hajima Hirase, Xavier Leinekugel,
and György Buzsáki. “The Application of Printed Circuit Board Technology for Fabrication
of Multi-Channel Micro-Drives.” Journal of Neuroscience Methods. Elsevier,
2001. https://doi.org/10.1016/S0165-0270(00)00362-9.
ieee: I. Szabo, A. Czurkó, J. L. Csicsvari, H. Hirase, X. Leinekugel, and G. Buzsáki,
“The application of printed circuit board technology for fabrication of multi-channel
micro-drives,” Journal of Neuroscience Methods, vol. 105, no. 1. Elsevier,
pp. 105–110, 2001.
ista: Szabo I, Czurkó A, Csicsvari JL, Hirase H, Leinekugel X, Buzsáki G. 2001.
The application of printed circuit board technology for fabrication of multi-channel
micro-drives. Journal of Neuroscience Methods. 105(1), 105–110.
mla: Szabo, Imre, et al. “The Application of Printed Circuit Board Technology for
Fabrication of Multi-Channel Micro-Drives.” Journal of Neuroscience Methods,
vol. 105, no. 1, Elsevier, 2001, pp. 105–10, doi:10.1016/S0165-0270(00)00362-9.
short: I. Szabo, A. Czurkó, J.L. Csicsvari, H. Hirase, X. Leinekugel, G. Buzsáki,
Journal of Neuroscience Methods 105 (2001) 105–110.
date_created: 2018-12-11T12:03:45Z
date_published: 2001-01-30T00:00:00Z
date_updated: 2023-05-15T10:50:39Z
day: '30'
doi: 10.1016/S0165-0270(00)00362-9
extern: '1'
external_id:
pmid:
- '11166371'
intvolume: ' 105'
issue: '1'
language:
- iso: eng
month: '01'
oa_version: None
page: 105 - 110
pmid: 1
publication: Journal of Neuroscience Methods
publication_identifier:
issn:
- 0165-0270
publication_status: published
publisher: Elsevier
publist_id: '2868'
quality_controlled: '1'
scopus_import: '1'
status: public
title: The application of printed circuit board technology for fabrication of multi-channel
micro-drives
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 105
year: '2001'
...
---
_id: '3540'
abstract:
- lang: eng
text: What determines the firing rate of cortical neurons in the absence of external
sensory input or motor behavior, such as during sleep? Hero we report that, in
a familiar environment, the discharge frequency of simultaneously recorded individual
CA1 pyramidal neurons and the coactivation of cell pairs remain highly correlated
across sleep-wake-steep sequences. However, both measures were affected when new
sets of neurons were activated in a novel environment. Nevertheless, the grand
mean firing rate of the whole pyramidal cell population remained constant across
behavioral states and testing conditions. The findings suggest that long-term
firing patterns of single cells can be modified by experience. We hypothesize
that increased firing rates of recently used neurons are associated with a concomitant
decrease in the discharge activity of the remaining population, leaving the mean
excitability of the hippocampal network unaltered.
acknowledgement: This work was supported by National Institutes of Health Grants NS34994
and MH54671, the F. M. Kirby Foundation, the Human Frontier Science Program (X.L.),
and the Uehara Memorial Foundation (H.H.).
article_processing_charge: No
article_type: original
author:
- first_name: Hajima
full_name: Hirase, Hajima
last_name: Hirase
- first_name: Xavier
full_name: Leinekugel, Xavier
last_name: Leinekugel
- first_name: András
full_name: Czurkó, András
last_name: Czurkó
- first_name: Jozsef L
full_name: Csicsvari, Jozsef L
id: 3FA14672-F248-11E8-B48F-1D18A9856A87
last_name: Csicsvari
orcid: 0000-0002-5193-4036
- first_name: György
full_name: Buzsáki, György
last_name: Buzsáki
citation:
ama: Hirase H, Leinekugel X, Czurkó A, Csicsvari JL, Buzsáki G. Firing rates of
hippocampal neurons are preserved during subsequent sleep episodes and modified
by novel awake experience. PNAS. 2001;98(16):9386-9390. doi:10.1073/pnas.161274398
apa: Hirase, H., Leinekugel, X., Czurkó, A., Csicsvari, J. L., & Buzsáki, G.
(2001). Firing rates of hippocampal neurons are preserved during subsequent sleep
episodes and modified by novel awake experience. PNAS. National Academy
of Sciences. https://doi.org/10.1073/pnas.161274398
chicago: Hirase, Hajima, Xavier Leinekugel, András Czurkó, Jozsef L Csicsvari, and
György Buzsáki. “Firing Rates of Hippocampal Neurons Are Preserved during Subsequent
Sleep Episodes and Modified by Novel Awake Experience.” PNAS. National
Academy of Sciences, 2001. https://doi.org/10.1073/pnas.161274398.
ieee: H. Hirase, X. Leinekugel, A. Czurkó, J. L. Csicsvari, and G. Buzsáki, “Firing
rates of hippocampal neurons are preserved during subsequent sleep episodes and
modified by novel awake experience,” PNAS, vol. 98, no. 16. National Academy
of Sciences, pp. 9386–9390, 2001.
ista: Hirase H, Leinekugel X, Czurkó A, Csicsvari JL, Buzsáki G. 2001. Firing rates
of hippocampal neurons are preserved during subsequent sleep episodes and modified
by novel awake experience. PNAS. 98(16), 9386–9390.
mla: Hirase, Hajima, et al. “Firing Rates of Hippocampal Neurons Are Preserved during
Subsequent Sleep Episodes and Modified by Novel Awake Experience.” PNAS,
vol. 98, no. 16, National Academy of Sciences, 2001, pp. 9386–90, doi:10.1073/pnas.161274398.
short: H. Hirase, X. Leinekugel, A. Czurkó, J.L. Csicsvari, G. Buzsáki, PNAS 98
(2001) 9386–9390.
date_created: 2018-12-11T12:03:52Z
date_published: 2001-07-31T00:00:00Z
date_updated: 2023-05-12T10:07:41Z
day: '31'
doi: 10.1073/pnas.161274398
extern: '1'
external_id:
pmid:
- '11470910'
intvolume: ' 98'
issue: '16'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC55430/
month: '07'
oa: 1
oa_version: Published Version
page: 9386 - 9390
pmid: 1
publication: PNAS
publication_identifier:
issn:
- 0027-8424
publication_status: published
publisher: National Academy of Sciences
publist_id: '2846'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Firing rates of hippocampal neurons are preserved during subsequent sleep episodes
and modified by novel awake experience
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 98
year: '2001'
...
---
_id: '3546'
abstract:
- lang: eng
text: Local versus distant coherence of hippocampal CA1 pyramidal cells was investigated
in the behaving rat. Temporal cross-correlation of pyramidal cells revealed a
significantly stronger relationship among local (<140 <mu>m) pyramidal
neurons compared with distant (>300 mum) neurons during non-theta-associated
immobility and sleep but not during theta-associated running and walking. In contrast,
cross-correlation between local pyramidal cell-interneuron pairs was significantly
stronger than between distant pairs during theta oscillations but were similar
during non-theta-associated behaviors. We suggest that network state-dependent
functional clustering of neuronal activity emerges because of the differential
contribution of the main excitatory inputs, the perforant path, and Schaffer collaterals
during theta and non-theta behaviors.
article_processing_charge: No
article_type: original
author:
- first_name: Hajima
full_name: Hirase, Hajima
last_name: Hirase
- first_name: Xavier
full_name: Leinekugel, Xavier
last_name: Leinekugel
- first_name: Jozsef L
full_name: Csicsvari, Jozsef L
id: 3FA14672-F248-11E8-B48F-1D18A9856A87
last_name: Csicsvari
orcid: 0000-0002-5193-4036
- first_name: András
full_name: Czurkó, András
last_name: Czurkó
- first_name: György
full_name: Buzsáki, György
last_name: Buzsáki
citation:
ama: Hirase H, Leinekugel X, Csicsvari JL, Czurkó A, Buzsáki G. Behavior-dependent
states of the hippocampal network affect functional clustering of neurons. Journal
of Neuroscience. 2001;21(10). doi:10.1523/JNEUROSCI.21-10-j0003.2001
apa: Hirase, H., Leinekugel, X., Csicsvari, J. L., Czurkó, A., & Buzsáki, G.
(2001). Behavior-dependent states of the hippocampal network affect functional
clustering of neurons. Journal of Neuroscience. Society for Neuroscience.
https://doi.org/10.1523/JNEUROSCI.21-10-j0003.2001
chicago: Hirase, Hajima, Xavier Leinekugel, Jozsef L Csicsvari, András Czurkó, and
György Buzsáki. “Behavior-Dependent States of the Hippocampal Network Affect Functional
Clustering of Neurons.” Journal of Neuroscience. Society for Neuroscience,
2001. https://doi.org/10.1523/JNEUROSCI.21-10-j0003.2001.
ieee: H. Hirase, X. Leinekugel, J. L. Csicsvari, A. Czurkó, and G. Buzsáki, “Behavior-dependent
states of the hippocampal network affect functional clustering of neurons,” Journal
of Neuroscience, vol. 21, no. 10. Society for Neuroscience, 2001.
ista: Hirase H, Leinekugel X, Csicsvari JL, Czurkó A, Buzsáki G. 2001. Behavior-dependent
states of the hippocampal network affect functional clustering of neurons. Journal
of Neuroscience. 21(10).
mla: Hirase, Hajima, et al. “Behavior-Dependent States of the Hippocampal Network
Affect Functional Clustering of Neurons.” Journal of Neuroscience, vol.
21, no. 10, Society for Neuroscience, 2001, doi:10.1523/JNEUROSCI.21-10-j0003.2001.
short: H. Hirase, X. Leinekugel, J.L. Csicsvari, A. Czurkó, G. Buzsáki, Journal
of Neuroscience 21 (2001).
date_created: 2018-12-11T12:03:54Z
date_published: 2001-05-15T00:00:00Z
date_updated: 2023-05-12T09:47:39Z
day: '15'
doi: 10.1523/JNEUROSCI.21-10-j0003.2001
extern: '1'
external_id:
pmid:
- '11319243'
intvolume: ' 21'
issue: '10'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://pubmed.ncbi.nlm.nih.gov/11319243/
month: '05'
oa: 1
oa_version: Published Version
pmid: 1
publication: Journal of Neuroscience
publication_identifier:
issn:
- 0270-6474
publication_status: published
publisher: Society for Neuroscience
publist_id: '2839'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Behavior-dependent states of the hippocampal network affect functional clustering
of neurons
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 21
year: '2001'
...
---
_id: '3586'
abstract:
- lang: eng
text: The book combines topics in mathematics (geometry and topology), computer
science (algorithms), and engineering (mesh generation). The original motivation
for these topics was the difficulty faced (both conceptually and in the technical
execution) in any attempt to combine elements of combinatorial and of numerical
algorithms. Mesh generation is a topic where a meaningful combination of these
different approaches to problem solving is inevitable. The book develops methods
from both areas that are amenable to combination, and explains recent breakthrough
solutions to meshing that fit into this category.The book should be an ideal graduate
text for courses on mesh generation. The specific material is selected giving
preference to topics that are elementary, attractive, lend themselves to teaching,
useful, and interesting.
article_processing_charge: No
author:
- first_name: Herbert
full_name: Edelsbrunner, Herbert
id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
last_name: Edelsbrunner
orcid: 0000-0002-9823-6833
citation:
ama: Edelsbrunner H. Geometry and Topology for Mesh Generation. Vol 7. Cambridge
University Press; 2001. doi:10.1017/CBO9780511530067
apa: Edelsbrunner, H. (2001). Geometry and Topology for Mesh Generation (Vol.
7). Cambridge University Press. https://doi.org/10.1017/CBO9780511530067
chicago: Edelsbrunner, Herbert. Geometry and Topology for Mesh Generation.
Vol. 7. Cambridge Monographs on Applied and Computational Mathematics. Cambridge
University Press, 2001. https://doi.org/10.1017/CBO9780511530067.
ieee: H. Edelsbrunner, Geometry and Topology for Mesh Generation, vol. 7.
Cambridge University Press, 2001.
ista: Edelsbrunner H. 2001. Geometry and Topology for Mesh Generation, Cambridge
University Press, 190p.
mla: Edelsbrunner, Herbert. Geometry and Topology for Mesh Generation. Vol.
7, Cambridge University Press, 2001, doi:10.1017/CBO9780511530067.
short: H. Edelsbrunner, Geometry and Topology for Mesh Generation, Cambridge University
Press, 2001.
date_created: 2018-12-11T12:04:06Z
date_published: 2001-05-28T00:00:00Z
date_updated: 2021-12-22T08:46:46Z
day: '28'
doi: 10.1017/CBO9780511530067
extern: '1'
intvolume: ' 7'
language:
- iso: eng
month: '05'
oa_version: None
page: '190'
publication_identifier:
eisbn:
- ' 978-0-5115-3006-7'
isbn:
- 0-521-79309-2
publication_status: published
publisher: Cambridge University Press
publist_id: '2798'
quality_controlled: '1'
related_material:
link:
- description: available via catalog IST BookList
relation: other
url: https://koha.app.ist.ac.at/cgi-bin/koha/opac-detail.pl?biblionumber=3508
series_title: Cambridge Monographs on Applied and Computational Mathematics
status: public
title: Geometry and Topology for Mesh Generation
type: book
user_id: 8b945eb4-e2f2-11eb-945a-df72226e66a9
volume: 7
year: '2001'
...
---
_id: '3596'
article_processing_charge: No
author:
- first_name: Nicholas H
full_name: Barton, Nicholas H
id: 4880FE40-F248-11E8-B48F-1D18A9856A87
last_name: Barton
orcid: 0000-0002-8548-5240
citation:
ama: Barton NH. Mendel and mathematics. Trends in Genetics. 2001;17:420-420.
doi:10.1016/S0168-9525(01)02315-0
apa: Barton, N. H. (2001). Mendel and mathematics. Trends in Genetics. Elsevier.
https://doi.org/10.1016/S0168-9525(01)02315-0
chicago: Barton, Nicholas H. “Mendel and Mathematics.” Trends in Genetics.
Elsevier, 2001. https://doi.org/10.1016/S0168-9525(01)02315-0.
ieee: N. H. Barton, “Mendel and mathematics,” Trends in Genetics, vol. 17.
Elsevier, pp. 420–420, 2001.
ista: Barton NH. 2001. Mendel and mathematics. Trends in Genetics. 17, 420–420.
mla: Barton, Nicholas H. “Mendel and Mathematics.” Trends in Genetics, vol.
17, Elsevier, 2001, pp. 420–420, doi:10.1016/S0168-9525(01)02315-0.
short: N.H. Barton, Trends in Genetics 17 (2001) 420–420.
date_created: 2018-12-11T12:04:09Z
date_published: 2001-07-01T00:00:00Z
date_updated: 2023-05-11T13:50:32Z
day: '01'
doi: 10.1016/S0168-9525(01)02315-0
extern: '1'
intvolume: ' 17'
language:
- iso: eng
month: '07'
oa_version: None
page: 420 - 420
publication: Trends in Genetics
publication_identifier:
issn:
- 0168-9479
publication_status: published
publisher: Elsevier
publist_id: '2787'
quality_controlled: '1'
status: public
title: Mendel and mathematics
type: review
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 17
year: '2001'
...
---
_id: '3622'
abstract:
- lang: eng
text: The extent of genetic variation in fitness and its components and genetic
variation's dependence on environmental conditions remain key issues in evolutionary
biology. We present measurements of genetic variation in preadult viability in
a laboratory-adapted population of Drosophila melanogaster, made at four different
densities. By crossing flies heterozygous for a wild-type chromosome and one of
two different balancers (TM1, TM2), we measure both heterozygous (TM1/+, TM2/+)
and homozygous (+/+) viability relative to a standard genotype (TM1/TM2). Forty
wild-type chromosomes were tested, of which 10 were chosen to be homozygous viable.
The mean numbers produced varied significantly between chromosome lines, with
an estimated between-line variance in loge numbers of 0.013. Relative viabilities
also varied significantly across chromosome lines, with a variance in loge homozygous
viability of 1.76 and of loge heterozygous viability of 0.165. The between-line
variance for numbers emerging increased with density, from 0.009 at lowest density
to 0.079 at highest. The genetic variance in relative viability increases with
density, but not significantly. Overall, the effects of different chromosomes
on relative viability were remarkably consistent across densities and across the
two heterozygous genotypes (TM1, TM2). The 10 lines that carried homozygous viable
wild-type chromosomes produced significantly more adults than the 30 lethal lines
at low density and significantly fewer adults at the highest density. Similarly,
there was a positive correlation between heterozygous viability and mean numbers
at low density, but a negative correlation at high density.
acknowledgement: We thank SERC and BBSRC for financial support and R.Miah, G. Geddes,
and E. Garcia for technical assistance.
article_processing_charge: No
article_type: original
author:
- first_name: Michael
full_name: Gardner, Michael
last_name: Gardner
- first_name: Kevin
full_name: Fowler, Kevin
last_name: Fowler
- first_name: Linda
full_name: Patridge, Linda
last_name: Patridge
- first_name: Nicholas H
full_name: Barton, Nicholas H
id: 4880FE40-F248-11E8-B48F-1D18A9856A87
last_name: Barton
orcid: 0000-0002-8548-5240
citation:
ama: Gardner M, Fowler K, Patridge L, Barton NH. Genetic variation for preadult
viability in Drosophila melanogaster. Evolution. 2001;55(8):1609-1620.
doi:10.1111/j.0014-3820.2001.tb00680.x
apa: Gardner, M., Fowler, K., Patridge, L., & Barton, N. H. (2001). Genetic
variation for preadult viability in Drosophila melanogaster. Evolution.
Wiley-Blackwell. https://doi.org/10.1111/j.0014-3820.2001.tb00680.x
chicago: Gardner, Michael, Kevin Fowler, Linda Patridge, and Nicholas H Barton.
“Genetic Variation for Preadult Viability in Drosophila Melanogaster.” Evolution.
Wiley-Blackwell, 2001. https://doi.org/10.1111/j.0014-3820.2001.tb00680.x.
ieee: M. Gardner, K. Fowler, L. Patridge, and N. H. Barton, “Genetic variation for
preadult viability in Drosophila melanogaster,” Evolution, vol. 55, no.
8. Wiley-Blackwell, pp. 1609–1620, 2001.
ista: Gardner M, Fowler K, Patridge L, Barton NH. 2001. Genetic variation for preadult
viability in Drosophila melanogaster. Evolution. 55(8), 1609–1620.
mla: Gardner, Michael, et al. “Genetic Variation for Preadult Viability in Drosophila
Melanogaster.” Evolution, vol. 55, no. 8, Wiley-Blackwell, 2001, pp. 1609–20,
doi:10.1111/j.0014-3820.2001.tb00680.x.
short: M. Gardner, K. Fowler, L. Patridge, N.H. Barton, Evolution 55 (2001) 1609–1620.
date_created: 2018-12-11T12:04:18Z
date_published: 2001-08-01T00:00:00Z
date_updated: 2023-05-11T13:43:30Z
day: '01'
doi: 10.1111/j.0014-3820.2001.tb00680.x
extern: '1'
external_id:
pmid:
- '11580020'
intvolume: ' 55'
issue: '8'
language:
- iso: eng
main_file_link:
- url: http://www.jstor.org/stable/2680379
month: '08'
oa_version: None
page: 1609 - 1620
pmid: 1
publication: Evolution
publication_identifier:
issn:
- 0014-3820
publication_status: published
publisher: Wiley-Blackwell
publist_id: '2761'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Genetic variation for preadult viability in Drosophila melanogaster
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 55
year: '2001'
...
---
_id: '3927'
abstract:
- lang: eng
text: TNF-alpha has been clearly identified as central mediator of T cell activation-induced
acute hepatic injury in mice, e.g., Con A hepatitis. In this model, liver injury
depends on both TNFRs, i.e., the 55-kDa TNFR1 as well as the 75-kDa TNFR2. We
show in this report that the hepatic TNFRs are not transcriptionally regulated,
but are regulated by receptor shedding. TNF directly mediates hepatocellular death
by activation of TNFR1 but also induces the expression of inflammatory proteins,
such as cytokines and adhesion molecules. Here we provide evidence that resistance
of TNFR1(-/-) and TNFR2(-/-) mice against Con A hepatitis is not due to an impaired
production of the central mediators TNF and IFN-gamma. Con A injection results
in a massive induction of ICAM-1, VCAM-1, and E-selectin in the liver. Lack of
either one of both TNFRs did not change adhesion molecule expression in the livers
of Con A-treated mice, presumably reflecting the fact that other endothelial cell-activating
cytokines up-regulated adhesion molecule expression. However, treatment of TNFR1(-/-)
and TNFR2(-/-) mice with murine rTNF revealed a predominant role for TNFR1 for
the induction of hepatic adhesion molecule expression. Pretreatment with blocking
Abs against E- and P-selectin or of ICAM(-/-) mice with anti-VCAM-1 Abs failed
to prevent Con A hepatitis, although accumulation of the critical cell population,
i.e., CD4(+) T cells was significantly inhibited. Hence, up-regulation of adhesion
molecules during acute hepatitis unlikely contributes to organ injury but rather
represents a defense mechanism.
acknowledgement: We thank Dr. H. Bluethmann (F. Hoffmann-LaRoche AG, Basle, Switzerland)
for kindly providing us TNFR knockout mice. We are indebted to Dr. G. R. Adolf (Bender
& Co Vienna, Austria) for providing recombinant murine TNF. We are also indebted
to Dr. D. Vestweber for providing anti-P-selectin mAb (23). We thank Dr. W. Neuhuber
(Institute of Anatomy, University of Erlangen-NÜrnberg, Erlangen, Germany) for
experimental support regarding confocal laser scanning microscopy. The perfect technical
assistance of Andrea Agli is gratefully acknowledged.
article_processing_charge: No
article_type: original
author:
- first_name: Dominik
full_name: Wolf, Dominik
last_name: Wolf
- first_name: Rupert
full_name: Hallmann, Rupert
last_name: Hallmann
- first_name: Gabriele
full_name: Sass, Gabriele
last_name: Sass
- first_name: Michael K
full_name: Sixt, Michael K
id: 41E9FBEA-F248-11E8-B48F-1D18A9856A87
last_name: Sixt
orcid: 0000-0002-6620-9179
- first_name: Sabine
full_name: Küsters, Sabine
last_name: Küsters
- first_name: Bastian
full_name: Fregien, Bastian
last_name: Fregien
- first_name: Christian
full_name: Trautwein, Christian
last_name: Trautwein
- first_name: Gisa
full_name: Tiegs, Gisa
last_name: Tiegs
citation:
ama: Wolf D, Hallmann R, Sass G, et al. TNF-α-induced expression of adhesion molecules
in the liver is under the control of TNFR1--relevance for concanavalin A-induced
hepatitis. Journal of Immunology. 2001;166(2):1300-1307. doi:10.4049/jimmunol.166.2.1300
apa: Wolf, D., Hallmann, R., Sass, G., Sixt, M. K., Küsters, S., Fregien, B., …
Tiegs, G. (2001). TNF-α-induced expression of adhesion molecules in the liver
is under the control of TNFR1--relevance for concanavalin A-induced hepatitis.
Journal of Immunology. American Association of Immunologists. https://doi.org/10.4049/jimmunol.166.2.1300
chicago: Wolf, Dominik, Rupert Hallmann, Gabriele Sass, Michael K Sixt, Sabine Küsters,
Bastian Fregien, Christian Trautwein, and Gisa Tiegs. “TNF-α-Induced Expression
of Adhesion Molecules in the Liver Is under the Control of TNFR1--Relevance for
Concanavalin A-Induced Hepatitis.” Journal of Immunology. American Association
of Immunologists, 2001. https://doi.org/10.4049/jimmunol.166.2.1300.
ieee: D. Wolf et al., “TNF-α-induced expression of adhesion molecules in
the liver is under the control of TNFR1--relevance for concanavalin A-induced
hepatitis,” Journal of Immunology, vol. 166, no. 2. American Association
of Immunologists, pp. 1300–1307, 2001.
ista: Wolf D, Hallmann R, Sass G, Sixt MK, Küsters S, Fregien B, Trautwein C, Tiegs
G. 2001. TNF-α-induced expression of adhesion molecules in the liver is under
the control of TNFR1--relevance for concanavalin A-induced hepatitis. Journal
of Immunology. 166(2), 1300–1307.
mla: Wolf, Dominik, et al. “TNF-α-Induced Expression of Adhesion Molecules in the
Liver Is under the Control of TNFR1--Relevance for Concanavalin A-Induced Hepatitis.”
Journal of Immunology, vol. 166, no. 2, American Association of Immunologists,
2001, pp. 1300–07, doi:10.4049/jimmunol.166.2.1300.
short: D. Wolf, R. Hallmann, G. Sass, M.K. Sixt, S. Küsters, B. Fregien, C. Trautwein,
G. Tiegs, Journal of Immunology 166 (2001) 1300–1307.
date_created: 2018-12-11T12:05:56Z
date_published: 2001-01-15T00:00:00Z
date_updated: 2023-05-11T13:37:29Z
day: '15'
doi: 10.4049/jimmunol.166.2.1300
extern: '1'
external_id:
pmid:
- '11145713'
intvolume: ' 166'
issue: '2'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: http://www.jimmunol.org/content/166/2/1300.long
month: '01'
oa: 1
oa_version: None
page: 1300 - 1307
pmid: 1
publication: Journal of Immunology
publication_identifier:
issn:
- 0022-1767
publication_status: published
publisher: American Association of Immunologists
publist_id: '2200'
quality_controlled: '1'
status: public
title: TNF-α-induced expression of adhesion molecules in the liver is under the control
of TNFR1--relevance for concanavalin A-induced hepatitis
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 166
year: '2001'
...
---
_id: '3928'
abstract:
- lang: eng
text: Regulated adhesion of leukocytes to the extracellular matrix is essential
for transmigration of blood vessels and subsequent migration into the stroma of
inflamed tissues. Although beta(2)-integrins play an indisputable role in adhesion
of polymorphonuclear granulocytes (PMN) to endothelium, we show here that beta(1)-
and beta(3)-integrins but not beta(2)-integrin are essential for the adhesion
to and migration on extracellular matrix molecules of the endothelial cell basement
membrane and subjacent interstitial matrix. Mouse wild type and beta(2)-integrin
null PMN and the progranulocytic cell line 32DC13 were employed in in vitro adhesion
and migration assays using extracellular matrix molecules expressed at sites of
extravasation in vivo, in particular the endothelial cell laminins 8 and 10. Wild
type and beta(2)-integrin null PMN showed the same pattern of ECM binding, indicating
that beta(2)-integrins do not mediate specific adhesion of PMN to the extracellular
matrix molecules tested; binding was observed to the interstitial matrix molecules,
fibronectin and vitronectin, via integrins alpha(5)beta(1) and alpha(v)beta(3),
respectively; to laminin 10 via alpha(6)beta(1); but not to laminins 1, 2, and
8, collagen type I and IV, perlecan, or tenascin-C. PMN binding to laminins 1,
2, and 8 could not be induced despite surface expression of functionally active
integrin alpha(6)beta(1), a major laminin receptor, demonstrating that expression
of alpha(6)beta(1) alone is insufficient for ligand binding and suggesting the
involvement of accessory factors. Nevertheless, laminins 1, 8, and 10 supported
PMN migration, indicating that differential cellular signaling via laminins is
independent of the extent of adhesion. The data demonstrate that adhesive and
nonadhesive interactions with components of the endothelial cell basement membrane
and subjacent interstitium play decisive roles in controlling PMN movement into
sites of inflammation and illustrate that beta(2)-integrins are not essential
for such interactions.
acknowledgement: We thank Dr. T. Winkler for carrying out flow cytometry analysis,
Dr. Simon Goodman for providing cyclic RGD peptides and helpful discussions, and
Stefanie Karosi and Thomas Samson for critical review of the manuscript. This work
would not have been possible without the expert technical assistance of Friederike
Pausch.
article_processing_charge: No
article_type: original
author:
- first_name: Michael K
full_name: Sixt, Michael K
id: 41E9FBEA-F248-11E8-B48F-1D18A9856A87
last_name: Sixt
orcid: 0000-0002-6620-9179
- first_name: Rupert
full_name: Hallmann, Rupert
last_name: Hallmann
- first_name: Olaf
full_name: Wendler, Olaf
last_name: Wendler
- first_name: Karin
full_name: Scharffetter Kochanek, Karin
last_name: Scharffetter Kochanek
- first_name: Lydia
full_name: Sorokin, Lydia
last_name: Sorokin
citation:
ama: Sixt MK, Hallmann R, Wendler O, Scharffetter Kochanek K, Sorokin L. Cell adhesion
and migration properties of β2-integrin negative polymorphonuclear granulocytes
on defined extracellular matrix molecules. Relevance for leukocyte extravasation.
Journal of Biological Chemistry. 2001;276(22):18878-18887. doi:10.1074/jbc.M010898200
apa: Sixt, M. K., Hallmann, R., Wendler, O., Scharffetter Kochanek, K., & Sorokin,
L. (2001). Cell adhesion and migration properties of β2-integrin negative polymorphonuclear
granulocytes on defined extracellular matrix molecules. Relevance for leukocyte
extravasation. Journal of Biological Chemistry. American Society for Biochemistry
and Molecular Biology. https://doi.org/10.1074/jbc.M010898200
chicago: Sixt, Michael K, Rupert Hallmann, Olaf Wendler, Karin Scharffetter Kochanek,
and Lydia Sorokin. “Cell Adhesion and Migration Properties of Β2-Integrin Negative
Polymorphonuclear Granulocytes on Defined Extracellular Matrix Molecules. Relevance
for Leukocyte Extravasation.” Journal of Biological Chemistry. American
Society for Biochemistry and Molecular Biology, 2001. https://doi.org/10.1074/jbc.M010898200.
ieee: M. K. Sixt, R. Hallmann, O. Wendler, K. Scharffetter Kochanek, and L. Sorokin,
“Cell adhesion and migration properties of β2-integrin negative polymorphonuclear
granulocytes on defined extracellular matrix molecules. Relevance for leukocyte
extravasation,” Journal of Biological Chemistry, vol. 276, no. 22. American
Society for Biochemistry and Molecular Biology, pp. 18878–18887, 2001.
ista: Sixt MK, Hallmann R, Wendler O, Scharffetter Kochanek K, Sorokin L. 2001.
Cell adhesion and migration properties of β2-integrin negative polymorphonuclear
granulocytes on defined extracellular matrix molecules. Relevance for leukocyte
extravasation. Journal of Biological Chemistry. 276(22), 18878–18887.
mla: Sixt, Michael K., et al. “Cell Adhesion and Migration Properties of Β2-Integrin
Negative Polymorphonuclear Granulocytes on Defined Extracellular Matrix Molecules.
Relevance for Leukocyte Extravasation.” Journal of Biological Chemistry,
vol. 276, no. 22, American Society for Biochemistry and Molecular Biology, 2001,
pp. 18878–87, doi:10.1074/jbc.M010898200.
short: M.K. Sixt, R. Hallmann, O. Wendler, K. Scharffetter Kochanek, L. Sorokin,
Journal of Biological Chemistry 276 (2001) 18878–18887.
date_created: 2018-12-11T12:05:56Z
date_published: 2001-06-01T00:00:00Z
date_updated: 2023-05-11T12:54:06Z
day: '01'
doi: 10.1074/jbc.M010898200
extern: '1'
external_id:
pmid:
- '11278780'
intvolume: ' 276'
issue: '22'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://www.sciencedirect.com/science/article/pii/S0021925819670134?via%3Dihub
month: '06'
oa: 1
oa_version: Published Version
page: 18878 - 18887
pmid: 1
publication: Journal of Biological Chemistry
publication_identifier:
issn:
- 0021-9258
publication_status: published
publisher: American Society for Biochemistry and Molecular Biology
publist_id: '2199'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Cell adhesion and migration properties of β2-integrin negative polymorphonuclear
granulocytes on defined extracellular matrix molecules. Relevance for leukocyte
extravasation
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 276
year: '2001'
...
---
_id: '3930'
abstract:
- lang: eng
text: 'An active involvement of blood-brain barrier endothelial cell basement membranes
in development of inflammatory lesions in the central nervous system (CNS) has
not been considered to date. Here we investigated the molecular composition and
possible function of the extracellular matrix encountered by extravasating T lymphocytes
during experimental autoimmune encephalomyelitis (EAE). Endothelial basement membranes
contained laminin 8 (alpha4beta1gamma1) and/or 10 (alpha5beta1gamma1) and their
expression was influenced by proinflammatory cytokines or angiostatic agents.
T cells emigrating into the CNS during EAE encountered two biochemically distinct
basement membranes, the endothelial (containing laminins 8 and 10) and the parenchymal
(containing laminins 1 and 2) basement membranes. However, inflammatory cuffs
occurred exclusively around endothelial basement membranes containing laminin
8, whereas in the presence of laminin 10 no infiltration was detectable. In vitro
assays using encephalitogenic T cell lines revealed adhesion to laminins 8 and
10, whereas binding to laminins 1 and 2 could not be induced. Downregulation of
integrin alpha6 on cerebral endothelium at sites of T cell infiltration, plus
a high turnover of laminin 8 at these sites, suggested two possible roles for
laminin 8 in the endothelial basement membrane: one at the level of the endothelial
cells resulting in reduced adhesion and, thereby, increased penetrability of the
monolayer; and secondly at the level of the T cells providing direct signals to
the transmigrating cells.'
acknowledgement: The authors thank Stefanie Karosi for careful and critical reading
of the manuscript and Monika Bruckner for expert technical assistance. We are particularly
grateful to Winfried Neuhuber for help with confocal microscopy and interpretation
of the data. This work was supported by Deutsche Forschungsgemeinschaft grants So285/1-3
and So285/1-4 to L.M. Sorokin.
article_processing_charge: No
article_type: original
author:
- first_name: Michael K
full_name: Sixt, Michael K
id: 41E9FBEA-F248-11E8-B48F-1D18A9856A87
last_name: Sixt
orcid: 0000-0002-6620-9179
- first_name: Britta
full_name: Engelhardt, Britta
last_name: Engelhardt
- first_name: Friederike
full_name: Pausch, Friederike
last_name: Pausch
- first_name: Rupert
full_name: Hallmann, Rupert
last_name: Hallmann
- first_name: Olaf
full_name: Wendler, Olaf
last_name: Wendler
- first_name: Lydia
full_name: Sorokin, Lydia
last_name: Sorokin
citation:
ama: Sixt MK, Engelhardt B, Pausch F, Hallmann R, Wendler O, Sorokin L. Endothelial
cell laminin isoforms, laminins 8 and 10, play decisive roles in T cell recruitment
across the blood-brain barrier in experimental autoimmune encephalomyelitis. Journal
of Cell Biology. 2001;153(5):933-946. doi:10.1083/jcb.153.5.933
apa: Sixt, M. K., Engelhardt, B., Pausch, F., Hallmann, R., Wendler, O., & Sorokin,
L. (2001). Endothelial cell laminin isoforms, laminins 8 and 10, play decisive
roles in T cell recruitment across the blood-brain barrier in experimental autoimmune
encephalomyelitis. Journal of Cell Biology. Rockefeller University Press.
https://doi.org/10.1083/jcb.153.5.933
chicago: Sixt, Michael K, Britta Engelhardt, Friederike Pausch, Rupert Hallmann,
Olaf Wendler, and Lydia Sorokin. “Endothelial Cell Laminin Isoforms, Laminins
8 and 10, Play Decisive Roles in T Cell Recruitment across the Blood-Brain Barrier
in Experimental Autoimmune Encephalomyelitis.” Journal of Cell Biology.
Rockefeller University Press, 2001. https://doi.org/10.1083/jcb.153.5.933 .
ieee: M. K. Sixt, B. Engelhardt, F. Pausch, R. Hallmann, O. Wendler, and L. Sorokin,
“Endothelial cell laminin isoforms, laminins 8 and 10, play decisive roles in
T cell recruitment across the blood-brain barrier in experimental autoimmune encephalomyelitis,”
Journal of Cell Biology, vol. 153, no. 5. Rockefeller University Press,
pp. 933–946, 2001.
ista: Sixt MK, Engelhardt B, Pausch F, Hallmann R, Wendler O, Sorokin L. 2001. Endothelial
cell laminin isoforms, laminins 8 and 10, play decisive roles in T cell recruitment
across the blood-brain barrier in experimental autoimmune encephalomyelitis. Journal
of Cell Biology. 153(5), 933–946.
mla: Sixt, Michael K., et al. “Endothelial Cell Laminin Isoforms, Laminins 8 and
10, Play Decisive Roles in T Cell Recruitment across the Blood-Brain Barrier in
Experimental Autoimmune Encephalomyelitis.” Journal of Cell Biology, vol.
153, no. 5, Rockefeller University Press, 2001, pp. 933–46, doi:10.1083/jcb.153.5.933 .
short: M.K. Sixt, B. Engelhardt, F. Pausch, R. Hallmann, O. Wendler, L. Sorokin,
Journal of Cell Biology 153 (2001) 933–946.
date_created: 2018-12-11T12:05:57Z
date_published: 2001-05-21T00:00:00Z
date_updated: 2023-05-11T12:19:36Z
day: '21'
doi: '10.1083/jcb.153.5.933 '
extern: '1'
external_id:
pmid:
- '11381080'
intvolume: ' 153'
issue: '5'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2174323/
month: '05'
oa: 1
oa_version: Published Version
page: 933 - 946
pmid: 1
publication: Journal of Cell Biology
publication_identifier:
issn:
- 0021-9525
publication_status: published
publisher: Rockefeller University Press
publist_id: '2198'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Endothelial cell laminin isoforms, laminins 8 and 10, play decisive roles in
T cell recruitment across the blood-brain barrier in experimental autoimmune encephalomyelitis
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 153
year: '2001'
...
---
_id: '4001'
abstract:
- lang: eng
text: 'The construction of shape spaces is studied from a mathematical and a computational
viewpoint. A program is outlined reducing the problem to four tasks: the representation
of geometry, the canonical deformation of geometry, the measuring of distance
in shape space, and the selection of base shapes. The technical part of this paper
focuses on the second task: the specification of a deformation mixing two or more
shapes in continuously changing proportions. (C) 2001 Elsevier Science B.V All
rights reserved.'
acknowledgement: National Science Foundation under grants CCR-96-19542 and CCR-97-12088,
and by the Army Research Office under grant DAAG55-98-1-0177.
article_processing_charge: No
article_type: original
author:
- first_name: Ho
full_name: Cheng, Ho
last_name: Cheng
- first_name: Herbert
full_name: Edelsbrunner, Herbert
id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
last_name: Edelsbrunner
orcid: 0000-0002-9823-6833
- first_name: Ping
full_name: Fu, Ping
last_name: Fu
citation:
ama: 'Cheng H, Edelsbrunner H, Fu P. Shape space from deformation. Computational
Geometry: Theory and Applications. 2001;19(2-3):191-204. doi:10.1016/S0925-7721(01)00021-9'
apa: 'Cheng, H., Edelsbrunner, H., & Fu, P. (2001). Shape space from deformation.
Computational Geometry: Theory and Applications. Elsevier. https://doi.org/10.1016/S0925-7721(01)00021-9'
chicago: 'Cheng, Ho, Herbert Edelsbrunner, and Ping Fu. “Shape Space from Deformation.”
Computational Geometry: Theory and Applications. Elsevier, 2001. https://doi.org/10.1016/S0925-7721(01)00021-9.'
ieee: 'H. Cheng, H. Edelsbrunner, and P. Fu, “Shape space from deformation,” Computational
Geometry: Theory and Applications, vol. 19, no. 2–3. Elsevier, pp. 191–204,
2001.'
ista: 'Cheng H, Edelsbrunner H, Fu P. 2001. Shape space from deformation. Computational
Geometry: Theory and Applications. 19(2–3), 191–204.'
mla: 'Cheng, Ho, et al. “Shape Space from Deformation.” Computational Geometry:
Theory and Applications, vol. 19, no. 2–3, Elsevier, 2001, pp. 191–204, doi:10.1016/S0925-7721(01)00021-9.'
short: 'H. Cheng, H. Edelsbrunner, P. Fu, Computational Geometry: Theory and Applications
19 (2001) 191–204.'
date_created: 2018-12-11T12:06:22Z
date_published: 2001-07-01T00:00:00Z
date_updated: 2023-05-10T12:57:14Z
day: '01'
doi: 10.1016/S0925-7721(01)00021-9
extern: '1'
intvolume: ' 19'
issue: 2-3
language:
- iso: eng
month: '07'
oa_version: None
page: 191 - 204
publication: 'Computational Geometry: Theory and Applications'
publication_identifier:
issn:
- 0925-7721
publication_status: published
publisher: Elsevier
publist_id: '2123'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Shape space from deformation
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 19
year: '2001'
...
---
_id: '4002'
abstract:
- lang: eng
text: Shape deformation refers to the continuous change of one geometric object
to another. We develop a software tool for planning, analyzing and visualizing
deformations between two shapes in R-2. The deformation is generated automatically
without any user intervention or specification of feature correspondences. A unique
property of the tool is the explicit availability of a two-dimensional shape space,
which can be used for designing the deformation either automatically by following
constraints and objectives or manually by drawing deformation paths.
acknowledgement: NSF under grants CCR-96-19542 and CCR-97-12088.
article_processing_charge: No
article_type: original
author:
- first_name: Siu
full_name: Cheng, Siu
last_name: Cheng
- first_name: Herbert
full_name: Edelsbrunner, Herbert
id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
last_name: Edelsbrunner
orcid: 0000-0002-9823-6833
- first_name: Ping
full_name: Fu, Ping
last_name: Fu
- first_name: Ka
full_name: Lam, Ka
last_name: Lam
citation:
ama: 'Cheng S, Edelsbrunner H, Fu P, Lam K. Design and analysis of planar shape
deformation. Computational Geometry: Theory and Applications. 2001;19(2-3):205-218.
doi:10.1016/S0925-7721(01)00020-7'
apa: 'Cheng, S., Edelsbrunner, H., Fu, P., & Lam, K. (2001). Design and analysis
of planar shape deformation. Computational Geometry: Theory and Applications.
Elsevier. https://doi.org/10.1016/S0925-7721(01)00020-7'
chicago: 'Cheng, Siu, Herbert Edelsbrunner, Ping Fu, and Ka Lam. “Design and Analysis
of Planar Shape Deformation.” Computational Geometry: Theory and Applications.
Elsevier, 2001. https://doi.org/10.1016/S0925-7721(01)00020-7.'
ieee: 'S. Cheng, H. Edelsbrunner, P. Fu, and K. Lam, “Design and analysis of planar
shape deformation,” Computational Geometry: Theory and Applications, vol.
19, no. 2–3. Elsevier, pp. 205–218, 2001.'
ista: 'Cheng S, Edelsbrunner H, Fu P, Lam K. 2001. Design and analysis of planar
shape deformation. Computational Geometry: Theory and Applications. 19(2–3), 205–218.'
mla: 'Cheng, Siu, et al. “Design and Analysis of Planar Shape Deformation.” Computational
Geometry: Theory and Applications, vol. 19, no. 2–3, Elsevier, 2001, pp. 205–18,
doi:10.1016/S0925-7721(01)00020-7.'
short: 'S. Cheng, H. Edelsbrunner, P. Fu, K. Lam, Computational Geometry: Theory
and Applications 19 (2001) 205–218.'
date_created: 2018-12-11T12:06:22Z
date_published: 2001-07-01T00:00:00Z
date_updated: 2023-05-10T14:21:31Z
day: '01'
doi: 10.1016/S0925-7721(01)00020-7
extern: '1'
intvolume: ' 19'
issue: 2-3
language:
- iso: eng
month: '07'
oa_version: None
page: 205 - 218
publication: 'Computational Geometry: Theory and Applications'
publication_identifier:
issn:
- 0925-7721
publication_status: published
publisher: Elsevier
publist_id: '2124'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Design and analysis of planar shape deformation
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 19
year: '2001'
...
---
_id: '4005'
abstract:
- lang: eng
text: This paper describes an algorithm for maintaining an approximating triangulation
of a deforming surface in R-3. The triangulation adapts dynamically to changing
shape, curvature, and topology of the surface.
article_processing_charge: No
author:
- first_name: Ho
full_name: Cheng, Ho
last_name: Cheng
- first_name: Tamal
full_name: Dey, Tamal
last_name: Dey
- first_name: Herbert
full_name: Edelsbrunner, Herbert
id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
last_name: Edelsbrunner
orcid: 0000-0002-9823-6833
- first_name: John
full_name: Sullivan, John
last_name: Sullivan
citation:
ama: 'Cheng H, Dey T, Edelsbrunner H, Sullivan J. Dynamic skin triangulation. In:
Proceedings of the 12th Annual ACM-SIAM Symposium on Discrete Algorithms.
SIAM; 2001:47-56.'
apa: 'Cheng, H., Dey, T., Edelsbrunner, H., & Sullivan, J. (2001). Dynamic skin
triangulation. In Proceedings of the 12th annual ACM-SIAM symposium on Discrete
algorithms (pp. 47–56). Washington, DC, USA : SIAM.'
chicago: Cheng, Ho, Tamal Dey, Herbert Edelsbrunner, and John Sullivan. “Dynamic
Skin Triangulation.” In Proceedings of the 12th Annual ACM-SIAM Symposium on
Discrete Algorithms, 47–56. SIAM, 2001.
ieee: H. Cheng, T. Dey, H. Edelsbrunner, and J. Sullivan, “Dynamic skin triangulation,”
in Proceedings of the 12th annual ACM-SIAM symposium on Discrete algorithms,
Washington, DC, USA , 2001, pp. 47–56.
ista: 'Cheng H, Dey T, Edelsbrunner H, Sullivan J. 2001. Dynamic skin triangulation.
Proceedings of the 12th annual ACM-SIAM symposium on Discrete algorithms. SODA:
Symposium on Discrete Algorithms, 47–56.'
mla: Cheng, Ho, et al. “Dynamic Skin Triangulation.” Proceedings of the 12th
Annual ACM-SIAM Symposium on Discrete Algorithms, SIAM, 2001, pp. 47–56.
short: H. Cheng, T. Dey, H. Edelsbrunner, J. Sullivan, in:, Proceedings of the 12th
Annual ACM-SIAM Symposium on Discrete Algorithms, SIAM, 2001, pp. 47–56.
conference:
end_date: 2001-01-09
location: 'Washington, DC, USA '
name: 'SODA: Symposium on Discrete Algorithms'
start_date: 2001-01-07
date_created: 2018-12-11T12:06:23Z
date_published: 2001-01-09T00:00:00Z
date_updated: 2023-05-10T12:35:44Z
day: '09'
extern: '1'
language:
- iso: eng
main_file_link:
- url: https://dl.acm.org/doi/10.5555/365411.365418
month: '01'
oa_version: None
page: 47 - 56
publication: Proceedings of the 12th annual ACM-SIAM symposium on Discrete algorithms
publication_identifier:
isbn:
- '9780898714906'
publication_status: published
publisher: SIAM
publist_id: '2120'
quality_controlled: '1'
status: public
title: Dynamic skin triangulation
type: conference
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
year: '2001'
...
---
_id: '4006'
abstract:
- lang: eng
text: The 180 models collected in this paper are produced by sampling and wrapping
point sets on tubes. The surfaces are represented as triangulated 2-manifolds
and available as st1-files from the author's web site at www.cs.duke.edu/similar
toedels. Each tube is obtained by thickening a circle or a smooth torus knot,
and for some we use the degrees of freedom in the thickening process to encode
meaningful information, such as curvature or torsion.
article_processing_charge: No
article_type: original
author:
- first_name: Herbert
full_name: Edelsbrunner, Herbert
id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
last_name: Edelsbrunner
orcid: 0000-0002-9823-6833
citation:
ama: Edelsbrunner H. 180 wrapped tubes. Journal of Universal Computer Science.
2001;7(5):379-399. doi:10.3217/jucs-007-05-0379
apa: Edelsbrunner, H. (2001). 180 wrapped tubes. Journal of Universal Computer
Science. Springer. https://doi.org/10.3217/jucs-007-05-0379
chicago: Edelsbrunner, Herbert. “180 Wrapped Tubes.” Journal of Universal Computer
Science. Springer, 2001. https://doi.org/10.3217/jucs-007-05-0379.
ieee: H. Edelsbrunner, “180 wrapped tubes,” Journal of Universal Computer Science,
vol. 7, no. 5. Springer, pp. 379–399, 2001.
ista: Edelsbrunner H. 2001. 180 wrapped tubes. Journal of Universal Computer Science.
7(5), 379–399.
mla: Edelsbrunner, Herbert. “180 Wrapped Tubes.” Journal of Universal Computer
Science, vol. 7, no. 5, Springer, 2001, pp. 379–99, doi:10.3217/jucs-007-05-0379.
short: H. Edelsbrunner, Journal of Universal Computer Science 7 (2001) 379–399.
date_created: 2018-12-11T12:06:24Z
date_published: 2001-05-28T00:00:00Z
date_updated: 2023-05-10T12:39:54Z
day: '28'
doi: 10.3217/jucs-007-05-0379
extern: '1'
intvolume: ' 7'
issue: '5'
language:
- iso: eng
month: '05'
oa_version: None
page: 379 - 399
publication: Journal of Universal Computer Science
publication_identifier:
issn:
- 0948-695X
publication_status: published
publisher: Springer
publist_id: '2121'
quality_controlled: '1'
status: public
title: 180 wrapped tubes
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 7
year: '2001'
...
---
_id: '4007'
abstract:
- lang: eng
text: 'This paper describes an algorithm for maintaining an approximating triangulation
of a deforming surface in R 3 . The surface is the envelope of an infinite family
of spheres defined and controlled by a finite collection of weighted points. The
triangulation adapts dynamically to changing shape, curvature, and topology of
the surface. '
acknowledgement: NSF under grant DMS- 98-73945, ARO under grant DAAG55-98-1-0177,
NSF under grants CCR-96- 19542 and CCR-97-12088.
article_processing_charge: No
article_type: original
author:
- first_name: Ho
full_name: Cheng, Ho
last_name: Cheng
- first_name: Tamal
full_name: Dey, Tamal
last_name: Dey
- first_name: Herbert
full_name: Edelsbrunner, Herbert
id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
last_name: Edelsbrunner
orcid: 0000-0002-9823-6833
- first_name: John
full_name: Sullivan, John
last_name: Sullivan
citation:
ama: Cheng H, Dey T, Edelsbrunner H, Sullivan J. Dynamic skin triangulation. Discrete
& Computational Geometry. 2001;25(4):525-568. doi:10.1007/s00454-001-0007-1
apa: Cheng, H., Dey, T., Edelsbrunner, H., & Sullivan, J. (2001). Dynamic skin
triangulation. Discrete & Computational Geometry. Springer. https://doi.org/10.1007/s00454-001-0007-1
chicago: Cheng, Ho, Tamal Dey, Herbert Edelsbrunner, and John Sullivan. “Dynamic
Skin Triangulation.” Discrete & Computational Geometry. Springer, 2001.
https://doi.org/10.1007/s00454-001-0007-1.
ieee: H. Cheng, T. Dey, H. Edelsbrunner, and J. Sullivan, “Dynamic skin triangulation,”
Discrete & Computational Geometry, vol. 25, no. 4. Springer, pp. 525–568,
2001.
ista: Cheng H, Dey T, Edelsbrunner H, Sullivan J. 2001. Dynamic skin triangulation.
Discrete & Computational Geometry. 25(4), 525–568.
mla: Cheng, Ho, et al. “Dynamic Skin Triangulation.” Discrete & Computational
Geometry, vol. 25, no. 4, Springer, 2001, pp. 525–68, doi:10.1007/s00454-001-0007-1.
short: H. Cheng, T. Dey, H. Edelsbrunner, J. Sullivan, Discrete & Computational
Geometry 25 (2001) 525–568.
date_created: 2018-12-11T12:06:24Z
date_published: 2001-04-04T00:00:00Z
date_updated: 2023-05-10T12:45:59Z
day: '04'
doi: 10.1007/s00454-001-0007-1
extern: '1'
intvolume: ' 25'
issue: '4'
language:
- iso: eng
month: '04'
oa_version: None
page: 525 - 568
publication: Discrete & Computational Geometry
publication_identifier:
issn:
- 0179-5376
publication_status: published
publisher: Springer
publist_id: '2122'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Dynamic skin triangulation
type: journal_article
user_id: ea97e931-d5af-11eb-85d4-e6957dddbf17
volume: 25
year: '2001'
...