---
_id: '2750'
abstract:
- lang: eng
  text: We consider random Schrödinger equations on ℝd for d ≥ 3 with a homogeneous
    Anderson-Poisson type random potential. Denote by λ the coupling constant and
    ψt the solution with initial data ψ0. The space and time variables scale as χ
    ≃λ-2-κ/2, t ≃λ-2-κ with 0 &lt; kappa; &lt; kappa;0(d). We prove that, in the limit
    λ → 0, the expectation of the Wigner distribution of ψt converges weakly to the
    solution of a heat equation in the space variable x for arbitrary L2 initial data.
    The proof is based on a rigorous analysis of Feynman diagrams. In the companion
    paper [10] the analysis of the non-repetition diagrams was presented. In this
    paper we complete the proof by estimating the recollision diagrams and showing
    that the main terms, i.e. the ladder diagrams with renormalized propagator, converge
    to the heat equation.
author:
- first_name: László
  full_name: László Erdös
  id: 4DBD5372-F248-11E8-B48F-1D18A9856A87
  last_name: Erdös
  orcid: 0000-0001-5366-9603
- first_name: Manfred
  full_name: Salmhofer, Manfred
  last_name: Salmhofer
- first_name: Horng
  full_name: Yau, Horng-Tzer
  last_name: Yau
citation:
  ama: Erdös L, Salmhofer M, Yau H. Quantum diffusion of the random Schrödinger evolution
    in the scaling limit II. The recollision diagrams. <i>Communications in Mathematical
    Physics</i>. 2007;271(1):1-53. doi:<a href="https://doi.org/10.1007/s00220-006-0158-2">10.1007/s00220-006-0158-2</a>
  apa: Erdös, L., Salmhofer, M., &#38; Yau, H. (2007). Quantum diffusion of the random
    Schrödinger evolution in the scaling limit II. The recollision diagrams. <i>Communications
    in Mathematical Physics</i>. Springer. <a href="https://doi.org/10.1007/s00220-006-0158-2">https://doi.org/10.1007/s00220-006-0158-2</a>
  chicago: Erdös, László, Manfred Salmhofer, and Horng Yau. “Quantum Diffusion of
    the Random Schrödinger Evolution in the Scaling Limit II. The Recollision Diagrams.”
    <i>Communications in Mathematical Physics</i>. Springer, 2007. <a href="https://doi.org/10.1007/s00220-006-0158-2">https://doi.org/10.1007/s00220-006-0158-2</a>.
  ieee: L. Erdös, M. Salmhofer, and H. Yau, “Quantum diffusion of the random Schrödinger
    evolution in the scaling limit II. The recollision diagrams,” <i>Communications
    in Mathematical Physics</i>, vol. 271, no. 1. Springer, pp. 1–53, 2007.
  ista: Erdös L, Salmhofer M, Yau H. 2007. Quantum diffusion of the random Schrödinger
    evolution in the scaling limit II. The recollision diagrams. Communications in
    Mathematical Physics. 271(1), 1–53.
  mla: Erdös, László, et al. “Quantum Diffusion of the Random Schrödinger Evolution
    in the Scaling Limit II. The Recollision Diagrams.” <i>Communications in Mathematical
    Physics</i>, vol. 271, no. 1, Springer, 2007, pp. 1–53, doi:<a href="https://doi.org/10.1007/s00220-006-0158-2">10.1007/s00220-006-0158-2</a>.
  short: L. Erdös, M. Salmhofer, H. Yau, Communications in Mathematical Physics 271
    (2007) 1–53.
date_created: 2018-12-11T11:59:24Z
date_published: 2007-04-01T00:00:00Z
date_updated: 2021-01-12T06:59:27Z
day: '01'
doi: 10.1007/s00220-006-0158-2
extern: 1
intvolume: '       271'
issue: '1'
month: '04'
page: 1 - 53
publication: Communications in Mathematical Physics
publication_status: published
publisher: Springer
publist_id: '4142'
quality_controlled: 0
status: public
title: Quantum diffusion of the random Schrödinger evolution in the scaling limit
  II. The recollision diagrams
type: journal_article
volume: 271
year: '2007'
...
---
_id: '2751'
abstract:
- lang: eng
  text: We consider random Schrödinger equations on ℤd for d ≥ 3 with identically
    distributed random potential. Denote by λ the coupling constant and ψ t the solution
    with initial data ψ 0. The space and time variables scale as x ∼ λ -2-κ/2,t ∼
    λ-2-κ with 0 &lt; κ &lt; κ0(d). We prove that, in the limit λ → 0, the expectation
    of the Wigner distribution of ψ t converges weakly to a solution of a heat equation
    in the space variable x for arbitrary L 2 initial data. The diffusion coefficient
    is uniquely determined by the kinetic energy associated to the momentum υ. This
    work is an extension to the lattice case of our previous result in the continuum
    [8,9]. Due to the non-convexity of the level surfaces of the dispersion relation,
    the estimates of several Feynman graphs are more involved.
author:
- first_name: László
  full_name: László Erdös
  id: 4DBD5372-F248-11E8-B48F-1D18A9856A87
  last_name: Erdös
  orcid: 0000-0001-5366-9603
- first_name: Manfred
  full_name: Salmhofer, Manfred
  last_name: Salmhofer
- first_name: Horng
  full_name: Yau, Horng-Tzer
  last_name: Yau
citation:
  ama: Erdös L, Salmhofer M, Yau H. Quantum diffusion for the Anderson model in the
    scaling limit. <i>Annales Henri Poincare</i>. 2007;8(4):621-685. doi:<a href="https://doi.org/10.1007/s00023-006-0318-0">10.1007/s00023-006-0318-0</a>
  apa: Erdös, L., Salmhofer, M., &#38; Yau, H. (2007). Quantum diffusion for the Anderson
    model in the scaling limit. <i>Annales Henri Poincare</i>. Birkhäuser. <a href="https://doi.org/10.1007/s00023-006-0318-0">https://doi.org/10.1007/s00023-006-0318-0</a>
  chicago: Erdös, László, Manfred Salmhofer, and Horng Yau. “Quantum Diffusion for
    the Anderson Model in the Scaling Limit.” <i>Annales Henri Poincare</i>. Birkhäuser,
    2007. <a href="https://doi.org/10.1007/s00023-006-0318-0">https://doi.org/10.1007/s00023-006-0318-0</a>.
  ieee: L. Erdös, M. Salmhofer, and H. Yau, “Quantum diffusion for the Anderson model
    in the scaling limit,” <i>Annales Henri Poincare</i>, vol. 8, no. 4. Birkhäuser,
    pp. 621–685, 2007.
  ista: Erdös L, Salmhofer M, Yau H. 2007. Quantum diffusion for the Anderson model
    in the scaling limit. Annales Henri Poincare. 8(4), 621–685.
  mla: Erdös, László, et al. “Quantum Diffusion for the Anderson Model in the Scaling
    Limit.” <i>Annales Henri Poincare</i>, vol. 8, no. 4, Birkhäuser, 2007, pp. 621–85,
    doi:<a href="https://doi.org/10.1007/s00023-006-0318-0">10.1007/s00023-006-0318-0</a>.
  short: L. Erdös, M. Salmhofer, H. Yau, Annales Henri Poincare 8 (2007) 621–685.
date_created: 2018-12-11T11:59:24Z
date_published: 2007-07-01T00:00:00Z
date_updated: 2021-01-12T06:59:27Z
day: '01'
doi: 10.1007/s00023-006-0318-0
extern: 1
intvolume: '         8'
issue: '4'
month: '07'
page: 621 - 685
publication: Annales Henri Poincare
publication_status: published
publisher: Birkhäuser
publist_id: '4141'
quality_controlled: 0
status: public
title: Quantum diffusion for the Anderson model in the scaling limit
type: journal_article
volume: 8
year: '2007'
...
---
_id: '2752'
abstract:
- lang: eng
  text: We prove L p -bounds on the Fourier transform of measures μ supported on two
    dimensional surfaces. Our method allows to consider surfaces whose Gauss curvature
    vanishes on a one-dimensional submanifold. Under a certain non-degeneracy condition,
    we prove that μ ∧ ε L 4+β, β &gt; 0, and we give a logarithmically divergent bound
    on the L 4-norm. We use this latter bound to estimate almost singular integrals
    involving the dispersion relation, e(p)= ∑13 [1-\cos p_j]} , of the discrete Laplace
    operator on the cubic lattice. We briefly explain our motivation for this bound
    originating in the theory of random Schrödinger operators.
author:
- first_name: László
  full_name: László Erdös
  id: 4DBD5372-F248-11E8-B48F-1D18A9856A87
  last_name: Erdös
  orcid: 0000-0001-5366-9603
- first_name: Manfred
  full_name: Salmhofer, Manfred
  last_name: Salmhofer
citation:
  ama: Erdös L, Salmhofer M. Decay of the Fourier transform of surfaces with vanishing
    curvature. <i>Mathematische Zeitschrift</i>. 2007;257(2):261-294. doi:<a href="https://doi.org/10.1007/s00209-007-0125-4">10.1007/s00209-007-0125-4</a>
  apa: Erdös, L., &#38; Salmhofer, M. (2007). Decay of the Fourier transform of surfaces
    with vanishing curvature. <i>Mathematische Zeitschrift</i>. Springer. <a href="https://doi.org/10.1007/s00209-007-0125-4">https://doi.org/10.1007/s00209-007-0125-4</a>
  chicago: Erdös, László, and Manfred Salmhofer. “Decay of the Fourier Transform of
    Surfaces with Vanishing Curvature.” <i>Mathematische Zeitschrift</i>. Springer,
    2007. <a href="https://doi.org/10.1007/s00209-007-0125-4">https://doi.org/10.1007/s00209-007-0125-4</a>.
  ieee: L. Erdös and M. Salmhofer, “Decay of the Fourier transform of surfaces with
    vanishing curvature,” <i>Mathematische Zeitschrift</i>, vol. 257, no. 2. Springer,
    pp. 261–294, 2007.
  ista: Erdös L, Salmhofer M. 2007. Decay of the Fourier transform of surfaces with
    vanishing curvature. Mathematische Zeitschrift. 257(2), 261–294.
  mla: Erdös, László, and Manfred Salmhofer. “Decay of the Fourier Transform of Surfaces
    with Vanishing Curvature.” <i>Mathematische Zeitschrift</i>, vol. 257, no. 2,
    Springer, 2007, pp. 261–94, doi:<a href="https://doi.org/10.1007/s00209-007-0125-4">10.1007/s00209-007-0125-4</a>.
  short: L. Erdös, M. Salmhofer, Mathematische Zeitschrift 257 (2007) 261–294.
date_created: 2018-12-11T11:59:25Z
date_published: 2007-01-01T00:00:00Z
date_updated: 2021-01-12T06:59:28Z
day: '01'
doi: 10.1007/s00209-007-0125-4
extern: 1
intvolume: '       257'
issue: '2'
month: '01'
page: 261 - 294
publication: Mathematische Zeitschrift
publication_status: published
publisher: Springer
publist_id: '4140'
quality_controlled: 0
status: public
title: Decay of the Fourier transform of surfaces with vanishing curvature
type: journal_article
volume: 257
year: '2007'
...
---
_id: '2793'
abstract:
- lang: eng
  text: Pipe flow is a prominent example among the shear flows that undergo transition
    to turbulence without mediation by a linear instability of the laminar profile.
    Experiments on pipe flow, as well as plane Couette and plane Poiseuille flow,
    show that triggering turbulence depends sensitively on initial conditions, that
    between the laminar and the turbulent states there exists no intermediate state
    with simple spatial or temporal characteristics, and that turbulence is not persistent,
    i.e., it can decay again, if the observation time is long enough. All these features
    can consistently be explained on the assumption that the turbulent state corresponds
    to a chaotic saddle in state space. The goal of this review is to explain this
    concept, summarize the numerical and experimental evidence for pipe flow, and
    outline the consequences for related flows.
author:
- first_name: Bruno
  full_name: Eckhardt, Bruno
  last_name: Eckhardt
- first_name: Tobias
  full_name: Schneider, Tobias M
  last_name: Schneider
- first_name: Björn
  full_name: Björn Hof
  id: 3A374330-F248-11E8-B48F-1D18A9856A87
  last_name: Hof
  orcid: 0000-0003-2057-2754
- first_name: Jerry
  full_name: Westerweel, Jerry
  last_name: Westerweel
citation:
  ama: Eckhardt B, Schneider T, Hof B, Westerweel J. Turbulence transition in pipe
    flow. <i>Annual Review of Fluid Mechanics</i>. 2007;39:447-468. doi:<a href="https://doi.org/10.1146/annurev.fluid.39.050905.110308">10.1146/annurev.fluid.39.050905.110308</a>
  apa: Eckhardt, B., Schneider, T., Hof, B., &#38; Westerweel, J. (2007). Turbulence
    transition in pipe flow. <i>Annual Review of Fluid Mechanics</i>. Annual Reviews.
    <a href="https://doi.org/10.1146/annurev.fluid.39.050905.110308">https://doi.org/10.1146/annurev.fluid.39.050905.110308</a>
  chicago: Eckhardt, Bruno, Tobias Schneider, Björn Hof, and Jerry Westerweel. “Turbulence
    Transition in Pipe Flow.” <i>Annual Review of Fluid Mechanics</i>. Annual Reviews,
    2007. <a href="https://doi.org/10.1146/annurev.fluid.39.050905.110308">https://doi.org/10.1146/annurev.fluid.39.050905.110308</a>.
  ieee: B. Eckhardt, T. Schneider, B. Hof, and J. Westerweel, “Turbulence transition
    in pipe flow,” <i>Annual Review of Fluid Mechanics</i>, vol. 39. Annual Reviews,
    pp. 447–468, 2007.
  ista: Eckhardt B, Schneider T, Hof B, Westerweel J. 2007. Turbulence transition
    in pipe flow. Annual Review of Fluid Mechanics. 39, 447–468.
  mla: Eckhardt, Bruno, et al. “Turbulence Transition in Pipe Flow.” <i>Annual Review
    of Fluid Mechanics</i>, vol. 39, Annual Reviews, 2007, pp. 447–68, doi:<a href="https://doi.org/10.1146/annurev.fluid.39.050905.110308">10.1146/annurev.fluid.39.050905.110308</a>.
  short: B. Eckhardt, T. Schneider, B. Hof, J. Westerweel, Annual Review of Fluid
    Mechanics 39 (2007) 447–468.
date_created: 2018-12-11T11:59:38Z
date_published: 2007-01-01T00:00:00Z
date_updated: 2019-04-26T07:22:20Z
day: '01'
doi: 10.1146/annurev.fluid.39.050905.110308
extern: 1
intvolume: '        39'
month: '01'
page: 447 - 468
publication: Annual Review of Fluid Mechanics
publication_status: published
publisher: Annual Reviews
publist_id: '4096'
quality_controlled: 0
status: public
title: Turbulence transition in pipe flow
type: review
volume: 39
year: '2007'
...
---
_id: '2794'
alternative_title:
- Springer Proceedings in Physics
author:
- first_name: Björn
  full_name: Björn Hof
  id: 3A374330-F248-11E8-B48F-1D18A9856A87
  last_name: Hof
  orcid: 0000-0003-2057-2754
- first_name: Wilco
  full_name: Tax, Wilco
  last_name: Tax
- first_name: Jerry
  full_name: Westerweel, Jerry
  last_name: Westerweel
citation:
  ama: 'Hof B, Tax W, Westerweel J. Lifetime of turbulence in pipe flow. In: Vol 117.
    Springer; 2007:556-558. doi:<a href="https://doi.org/10.1007/978-3-540-72604-3_177">10.1007/978-3-540-72604-3_177</a>'
  apa: 'Hof, B., Tax, W., &#38; Westerweel, J. (2007). Lifetime of turbulence in pipe
    flow (Vol. 117, pp. 556–558). Presented at the EETC11: European Turbulence Conference,
    Springer. <a href="https://doi.org/10.1007/978-3-540-72604-3_177">https://doi.org/10.1007/978-3-540-72604-3_177</a>'
  chicago: Hof, Björn, Wilco Tax, and Jerry Westerweel. “Lifetime of Turbulence in
    Pipe Flow,” 117:556–58. Springer, 2007. <a href="https://doi.org/10.1007/978-3-540-72604-3_177">https://doi.org/10.1007/978-3-540-72604-3_177</a>.
  ieee: 'B. Hof, W. Tax, and J. Westerweel, “Lifetime of turbulence in pipe flow,”
    presented at the EETC11: European Turbulence Conference, 2007, vol. 117, pp. 556–558.'
  ista: 'Hof B, Tax W, Westerweel J. 2007. Lifetime of turbulence in pipe flow. EETC11:
    European Turbulence Conference, Springer Proceedings in Physics, vol. 117, 556–558.'
  mla: Hof, Björn, et al. <i>Lifetime of Turbulence in Pipe Flow</i>. Vol. 117, Springer,
    2007, pp. 556–58, doi:<a href="https://doi.org/10.1007/978-3-540-72604-3_177">10.1007/978-3-540-72604-3_177</a>.
  short: B. Hof, W. Tax, J. Westerweel, in:, Springer, 2007, pp. 556–558.
conference:
  name: 'EETC11: European Turbulence Conference'
date_created: 2018-12-11T11:59:38Z
date_published: 2007-06-01T00:00:00Z
date_updated: 2021-01-12T06:59:45Z
day: '01'
doi: 10.1007/978-3-540-72604-3_177
extern: 1
intvolume: '       117'
month: '06'
page: 556 - 558
publication_status: published
publisher: Springer
publist_id: '4095'
quality_controlled: 0
status: public
title: Lifetime of turbulence in pipe flow
type: conference
volume: 117
year: '2007'
...
---
_id: '2893'
abstract:
- lang: eng
  text: 'Summary: Regulatory CD4+ T cells, enriched in the CD25 pool of healthy individuals,
    mediate natural tolerance and prevent autoimmune diseases. Despite their fundamental
    and potential clinical significance, regulatory T (TR) cells have not yet been
    incorporated in a coherent theory of the immune system. This article reviews experimental
    evidence and theoretical arguments supporting a model of TR cell dynamics, uncovering
    some of its most relevant biological implications. According to this model, the
    persistence and expansion of TR cell populations depend strictly on specific interactions
    they make with antigen-presenting cells (APCs) and conventional effector T (TE)
    cells. This three-partner crossregulation imposes that TR cells feed on the specific
    autoimmune activities they suppress, with implications ranging from their interactions
    with other cells to their repertoire selection in the periphery and in the thymus,
    and to the relationship between these cells and the innate immune system. These
    implications stem from the basic prediction that the peripheral dynamics sort
    the CD4+ T-cell repertoire into two subsets: a less diverse set of small clones
    of autoreactive effector and regulatory cells that regulate each other’s growth,
    and a more diverse set of barely autoreactive TE cell clones, whose expansion
    is limited only by APC availability. It is argued that such partitioning of the
    repertoire sets the ground for self–non-self discrimination. '
author:
- first_name: Jorge
  full_name: Carneiro, Jorge
  last_name: Carneiro
- first_name: Kalet
  full_name: Leon, Kalet
  last_name: Leon
- first_name: Íris
  full_name: Caramalho, Íris
  last_name: Caramalho
- first_name: Carline
  full_name: Van Den Dool, Carline
  last_name: Van Den Dool
- first_name: Rui
  full_name: Gardner, Rui
  last_name: Gardner
- first_name: Vanessa
  full_name: Oliveira, Vanessa
  last_name: Oliveira
- first_name: Marie
  full_name: Bergman, Marie L
  last_name: Bergman
- first_name: Nuno
  full_name: Sepúlveda, Nuno
  last_name: Sepúlveda
- first_name: Tiago
  full_name: Tiago Paixao
  id: 2C5658E6-F248-11E8-B48F-1D18A9856A87
  last_name: Paixao
  orcid: 0000-0003-2361-3953
- first_name: Jose
  full_name: Faro, Jose
  last_name: Faro
- first_name: Jocelyne
  full_name: Demengeot, Jocelyne
  last_name: Demengeot
citation:
  ama: Carneiro J, Leon K, Caramalho Í, et al. When three is not a crowd a Crossregulation
    Model of the dynamics and repertoire selection of regulatory CD4 T cells. <i>Immunological
    Reviews</i>. 2007;216(1):48-68. doi:<a href="https://doi.org/10.1111/j.1600-065X.2007.00487.x">10.1111/j.1600-065X.2007.00487.x</a>
  apa: Carneiro, J., Leon, K., Caramalho, Í., Van Den Dool, C., Gardner, R., Oliveira,
    V., … Demengeot, J. (2007). When three is not a crowd a Crossregulation Model
    of the dynamics and repertoire selection of regulatory CD4 T cells. <i>Immunological
    Reviews</i>. Wiley-Blackwell. <a href="https://doi.org/10.1111/j.1600-065X.2007.00487.x">https://doi.org/10.1111/j.1600-065X.2007.00487.x</a>
  chicago: Carneiro, Jorge, Kalet Leon, Íris Caramalho, Carline Van Den Dool, Rui
    Gardner, Vanessa Oliveira, Marie Bergman, et al. “When Three Is Not a Crowd a
    Crossregulation Model of the Dynamics and Repertoire Selection of Regulatory CD4
    T Cells.” <i>Immunological Reviews</i>. Wiley-Blackwell, 2007. <a href="https://doi.org/10.1111/j.1600-065X.2007.00487.x">https://doi.org/10.1111/j.1600-065X.2007.00487.x</a>.
  ieee: J. Carneiro <i>et al.</i>, “When three is not a crowd a Crossregulation Model
    of the dynamics and repertoire selection of regulatory CD4 T cells,” <i>Immunological
    Reviews</i>, vol. 216, no. 1. Wiley-Blackwell, pp. 48–68, 2007.
  ista: Carneiro J, Leon K, Caramalho Í, Van Den Dool C, Gardner R, Oliveira V, Bergman
    M, Sepúlveda N, Paixao T, Faro J, Demengeot J. 2007. When three is not a crowd
    a Crossregulation Model of the dynamics and repertoire selection of regulatory
    CD4 T cells. Immunological Reviews. 216(1), 48–68.
  mla: Carneiro, Jorge, et al. “When Three Is Not a Crowd a Crossregulation Model
    of the Dynamics and Repertoire Selection of Regulatory CD4 T Cells.” <i>Immunological
    Reviews</i>, vol. 216, no. 1, Wiley-Blackwell, 2007, pp. 48–68, doi:<a href="https://doi.org/10.1111/j.1600-065X.2007.00487.x">10.1111/j.1600-065X.2007.00487.x</a>.
  short: J. Carneiro, K. Leon, Í. Caramalho, C. Van Den Dool, R. Gardner, V. Oliveira,
    M. Bergman, N. Sepúlveda, T. Paixao, J. Faro, J. Demengeot, Immunological Reviews
    216 (2007) 48–68.
date_created: 2018-12-11T12:00:11Z
date_published: 2007-01-01T00:00:00Z
date_updated: 2021-01-12T07:00:31Z
day: '01'
doi: 10.1111/j.1600-065X.2007.00487.x
extern: 1
intvolume: '       216'
issue: '1'
month: '01'
page: 48 - 68
publication: Immunological Reviews
publication_status: published
publisher: Wiley-Blackwell
publist_id: '3865'
quality_controlled: 0
status: public
title: When three is not a crowd a Crossregulation Model of the dynamics and repertoire
  selection of regulatory CD4 T cells
type: journal_article
volume: 216
year: '2007'
...
---
_id: '2896'
abstract:
- lang: eng
  text: Gene expression from both parental alleles is beneficial by masking the effects
    of deleterious recessive mutations and by reducing the noise in gene expression
    in diploid organisms. However, a class of genes are expressed preferentially or
    strictly from a single allele. The selective advantage of avoiding biallelic expression
    is clear for allelic-excluded antigen receptor and odorant receptor genes, genes
    undergoing X-chromosome inactivation in females and parental genomic imprinted
    genes. In contrast, there is no clear biological rationale for the predominant
    and stochastic monoallelic expression of cytokine genes in the immune system,
    and the underlying mechanism is elusive and controversial. A clarification of
    the mechanism of predominant monoallelic expression would be instrumental in better
    understanding its eventual biological functional. This prompted the development
    of a quantitative framework that could describe the dynamics of the pattern of
    allele expression of the IL-10 gene, from which general quantitative insights
    could be gained. We report that the experimental observations on these patterns
    of allelic expression cannot be easily reconciled with a simple model of stochastic
    transcriptional activation, in which the two alleles are, at any time, equally
    competent for transcription. Instead, these observations call into action a general
    model of eukaryotic transcriptional regulation according to which the locus competence
    for transcription is dynamic, involving multiple, cooperative and stochastic modification
    steps. In this model, the probability that an allele becomes transcriptionally
    active is a function of the number of chromatin modifications that it accumulated.
    On the basis of the properties of this model, we argue that predominant monoallelic
    expression might have had no adaptive role, and may have evolved under indirect
    selection for low frequency of expressing cells.
acknowledgement: Calado and Paixão acknowledge the financial support of Fundação para
  a Ciência e a Tecnologia (fellowships SFRH/BD/2973/2000 and SFRH/BD/10550/2002,
  respectively).
author:
- first_name: Tiago
  full_name: Tiago Paixao
  id: 2C5658E6-F248-11E8-B48F-1D18A9856A87
  last_name: Paixao
  orcid: 0000-0003-2361-3953
- first_name: Tiago
  full_name: Carvalho, Tiago P
  last_name: Carvalho
- first_name: Dinis
  full_name: Calado, Dinis P
  last_name: Calado
- first_name: Jorge
  full_name: Carneiro, Jorge
  last_name: Carneiro
citation:
  ama: Paixao T, Carvalho T, Calado D, Carneiro J. Quantitative insights into stochastic
    monoallelic expression of cytokine genes. <i>Immunology and Cell Biology</i>.
    2007;85(4):315-322. doi:<a href="https://doi.org/10.1038/sj.icb.7100057">10.1038/sj.icb.7100057</a>
  apa: Paixao, T., Carvalho, T., Calado, D., &#38; Carneiro, J. (2007). Quantitative
    insights into stochastic monoallelic expression of cytokine genes. <i>Immunology
    and Cell Biology</i>. Nature Publishing Group. <a href="https://doi.org/10.1038/sj.icb.7100057">https://doi.org/10.1038/sj.icb.7100057</a>
  chicago: Paixao, Tiago, Tiago Carvalho, Dinis Calado, and Jorge Carneiro. “Quantitative
    Insights into Stochastic Monoallelic Expression of Cytokine Genes.” <i>Immunology
    and Cell Biology</i>. Nature Publishing Group, 2007. <a href="https://doi.org/10.1038/sj.icb.7100057">https://doi.org/10.1038/sj.icb.7100057</a>.
  ieee: T. Paixao, T. Carvalho, D. Calado, and J. Carneiro, “Quantitative insights
    into stochastic monoallelic expression of cytokine genes,” <i>Immunology and Cell
    Biology</i>, vol. 85, no. 4. Nature Publishing Group, pp. 315–322, 2007.
  ista: Paixao T, Carvalho T, Calado D, Carneiro J. 2007. Quantitative insights into
    stochastic monoallelic expression of cytokine genes. Immunology and Cell Biology.
    85(4), 315–322.
  mla: Paixao, Tiago, et al. “Quantitative Insights into Stochastic Monoallelic Expression
    of Cytokine Genes.” <i>Immunology and Cell Biology</i>, vol. 85, no. 4, Nature
    Publishing Group, 2007, pp. 315–22, doi:<a href="https://doi.org/10.1038/sj.icb.7100057">10.1038/sj.icb.7100057</a>.
  short: T. Paixao, T. Carvalho, D. Calado, J. Carneiro, Immunology and Cell Biology
    85 (2007) 315–322.
date_created: 2018-12-11T12:00:12Z
date_published: 2007-01-01T00:00:00Z
date_updated: 2021-01-12T07:00:32Z
day: '01'
doi: 10.1038/sj.icb.7100057
extern: 1
intvolume: '        85'
issue: '4'
month: '01'
page: 315 - 322
publication: Immunology and Cell Biology
publication_status: published
publisher: Nature Publishing Group
publist_id: '3862'
quality_controlled: 0
status: public
title: Quantitative insights into stochastic monoallelic expression of cytokine genes
type: journal_article
volume: 85
year: '2007'
...
---
_id: '2933'
author:
- first_name: M Pawan
  full_name: Kumar, M Pawan
  last_name: Kumar
- first_name: Vladimir
  full_name: Vladimir Kolmogorov
  id: 3D50B0BA-F248-11E8-B48F-1D18A9856A87
  last_name: Kolmogorov
- first_name: Philip
  full_name: Torr, Philip H
  last_name: Torr
citation:
  ama: 'Kumar MP, Kolmogorov V, Torr P. An Analysis of Convex Relaxations for MAP
    Estimation. In: Neural Information Processing Systems; 2007.'
  apa: Kumar, M. P., Kolmogorov, V., &#38; Torr, P. (2007). An Analysis of Convex
    Relaxations for MAP Estimation. Presented at the Neural Information Processing
    Systems, Neural Information Processing Systems.
  chicago: Kumar, M Pawan, Vladimir Kolmogorov, and Philip Torr. “An Analysis of Convex
    Relaxations for MAP Estimation.” Neural Information Processing Systems, 2007.
  ieee: M. P. Kumar, V. Kolmogorov, and P. Torr, “An Analysis of Convex Relaxations
    for MAP Estimation,” presented at the Neural Information Processing Systems, 2007.
  ista: Kumar MP, Kolmogorov V, Torr P. 2007. An Analysis of Convex Relaxations for
    MAP Estimation. Neural Information Processing Systems.
  mla: Kumar, M. Pawan, et al. <i>An Analysis of Convex Relaxations for MAP Estimation</i>.
    Neural Information Processing Systems, 2007.
  short: M.P. Kumar, V. Kolmogorov, P. Torr, in:, Neural Information Processing Systems,
    2007.
conference:
  name: Neural Information Processing Systems
date_created: 2018-12-11T12:00:25Z
date_published: 2007-12-01T00:00:00Z
date_updated: 2021-01-12T07:39:52Z
day: '01'
extern: 1
month: '12'
publication_status: published
publisher: Neural Information Processing Systems
publist_id: '3803'
quality_controlled: 0
status: public
title: An Analysis of Convex Relaxations for MAP Estimation
type: conference
year: '2007'
...
---
_id: '3019'
abstract:
- lang: eng
  text: |2-

    Directional transport of the phytohormone auxin is established primarily at the point of cellular afflux and is required for the establishment and maintenance of plant polarity. Studies in whole plants and heterologous systems indicate that PIN-FORMED (PIN) and P-glycoprotein (PGP) transport proteins mediate the cellular efflux of natural and synthetic auxins. However, aromatic anion transport resulting from PGP and PIN expression in nonplant systems was also found to lack tha high level of substrate specificity seen in planta. Furthermore, previous reports that PGP19 stabilizes PIN1 on the plasma membrane suggested that PIN-PGP interactions might regulate polar auxin efflux. Hare, we show that PGP1 and PGP19 colocalized with PIN1 in the shoot apax in Arabidopsis thaliana and with PIN1 and PIN2 in root tissues. Specific PGP-PIN interactions were seen in yeast two-hybrid and colmmunoprecipitation assays. PIN-PGP interactions appeared to enhance transport activity and, to a greater extent, substrate/inhibitor specificities when coexpressed in heterologous systems. By contrast, no interactions between PGPs and the AUXIN1 influx carrier were observed. Phenotypes of pin and pgp mutants suggest discrete functional roles in auxin transport, but pin pgp mutants exhibited phenotypes that are both additive and synergistic. These results suggest that PINs and PGPs characterize coordinated, Independent auxin transport mechanisms but also function interactively in a tissue-specific manner.
author:
- first_name: Joshua
  full_name: Blakeslee, Joshua
  last_name: Blakeslee
- first_name: Anindita
  full_name: Bandyopadhyay, Anindita
  last_name: Bandyopadhyay
- first_name: Ran
  full_name: Ok, Ran Lee
  last_name: Ok
- first_name: Jozef
  full_name: Mravec, Jozef
  last_name: Mravec
- first_name: Boosaree
  full_name: Titapiwatanakun, Boosaree
  last_name: Titapiwatanakun
- first_name: Michael
  full_name: Sauer, Michael
  last_name: Sauer
- first_name: Srinivas
  full_name: Makam, Srinivas N
  last_name: Makam
- first_name: Yan
  full_name: Cheng, Yan
  last_name: Cheng
- first_name: Rodolphe
  full_name: Bouchard, Rodolphe
  last_name: Bouchard
- first_name: Jiří
  full_name: Adamec, Jiří
  last_name: Adamec
- first_name: Markus
  full_name: Geisler, Markus
  last_name: Geisler
- first_name: Akitomo
  full_name: Nagashima, Akitomo
  last_name: Nagashima
- first_name: Tatsuya
  full_name: Sakai, Tatsuya
  last_name: Sakai
- first_name: Enrico
  full_name: Martinoia, Enrico
  last_name: Martinoia
- first_name: Jirí
  full_name: Jirí Friml
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
- first_name: Wendy
  full_name: Peer, Wendy A
  last_name: Peer
- first_name: Angus
  full_name: Murphy, Angus S
  last_name: Murphy
citation:
  ama: Blakeslee J, Bandyopadhyay A, Ok R, et al. Interactions among PIN FORMED and
    P glycoprotein auxin transporters in Arabidopsis. <i>Plant Cell</i>. 2007;19(1):131-147.
    doi:<a href="https://doi.org/10.1105/tpc.106.040782">10.1105/tpc.106.040782</a>
  apa: Blakeslee, J., Bandyopadhyay, A., Ok, R., Mravec, J., Titapiwatanakun, B.,
    Sauer, M., … Murphy, A. (2007). Interactions among PIN FORMED and P glycoprotein
    auxin transporters in Arabidopsis. <i>Plant Cell</i>. American Society of Plant
    Biologists. <a href="https://doi.org/10.1105/tpc.106.040782">https://doi.org/10.1105/tpc.106.040782</a>
  chicago: Blakeslee, Joshua, Anindita Bandyopadhyay, Ran Ok, Jozef Mravec, Boosaree
    Titapiwatanakun, Michael Sauer, Srinivas Makam, et al. “Interactions among PIN
    FORMED and P Glycoprotein Auxin Transporters in Arabidopsis.” <i>Plant Cell</i>.
    American Society of Plant Biologists, 2007. <a href="https://doi.org/10.1105/tpc.106.040782">https://doi.org/10.1105/tpc.106.040782</a>.
  ieee: J. Blakeslee <i>et al.</i>, “Interactions among PIN FORMED and P glycoprotein
    auxin transporters in Arabidopsis,” <i>Plant Cell</i>, vol. 19, no. 1. American
    Society of Plant Biologists, pp. 131–147, 2007.
  ista: Blakeslee J, Bandyopadhyay A, Ok R, Mravec J, Titapiwatanakun B, Sauer M,
    Makam S, Cheng Y, Bouchard R, Adamec J, Geisler M, Nagashima A, Sakai T, Martinoia
    E, Friml J, Peer W, Murphy A. 2007. Interactions among PIN FORMED and P glycoprotein
    auxin transporters in Arabidopsis. Plant Cell. 19(1), 131–147.
  mla: Blakeslee, Joshua, et al. “Interactions among PIN FORMED and P Glycoprotein
    Auxin Transporters in Arabidopsis.” <i>Plant Cell</i>, vol. 19, no. 1, American
    Society of Plant Biologists, 2007, pp. 131–47, doi:<a href="https://doi.org/10.1105/tpc.106.040782">10.1105/tpc.106.040782</a>.
  short: J. Blakeslee, A. Bandyopadhyay, R. Ok, J. Mravec, B. Titapiwatanakun, M.
    Sauer, S. Makam, Y. Cheng, R. Bouchard, J. Adamec, M. Geisler, A. Nagashima, T.
    Sakai, E. Martinoia, J. Friml, W. Peer, A. Murphy, Plant Cell 19 (2007) 131–147.
date_created: 2018-12-11T12:00:54Z
date_published: 2007-01-01T00:00:00Z
date_updated: 2021-01-12T07:40:29Z
day: '01'
doi: 10.1105/tpc.106.040782
extern: 1
intvolume: '        19'
issue: '1'
month: '01'
page: 131 - 147
publication: Plant Cell
publication_status: published
publisher: American Society of Plant Biologists
publist_id: '3682'
quality_controlled: 0
status: public
title: Interactions among PIN FORMED and P glycoprotein auxin transporters in Arabidopsis
type: journal_article
volume: 19
year: '2007'
...
---
_id: '3021'
abstract:
- lang: eng
  text: Polarized transport of the plant hormone auxin influences multiple growth
    processes in plants and is regulated by plasma-membrane-localized efflux and uptake
    carriers. The PGP (P-glycoprotein) ABC transporters (ATP-binding-cassette transporters),
    PIN (pin-formed) subfamily of major facilitator proteins and members of AUX/LAX
    families have been shown to independently transport auxin both in planta and in
    heterologous systems. However, PIN- and PGP-mediated transport in heterologous
    systems exhibits decreased substrate specificity and inhibitor-sensitivity compared
    with what is seen in plants and plant cells. To determine whether PIN-PGP interactions
    enhance transport specificity, we analysed interactions of the representative
    auxin-transporting PGPs with PIN1 and AUX1 in planta and in heterologous systems.
    Here, we provide evidence that PINs and PGPs interact and function both independently
    and co-ordinately to control polar auxin transport and impart transport specificity
    and directionality. These interactions take place in protein complexes stabilized
    by PGPs in detergent-resistant microdomains.
alternative_title:
- Biochemical Society Transactions
author:
- first_name: Anindita
  full_name: Bandyopadhyay, Anindita
  last_name: Bandyopadhyay
- first_name: Joshua
  full_name: Blakeslee, Joshua
  last_name: Blakeslee
- first_name: Ok
  full_name: Lee, Ok Ran
  last_name: Lee
- first_name: Jozef
  full_name: Mravec, Jozef
  last_name: Mravec
- first_name: Michael
  full_name: Sauer, Michael
  last_name: Sauer
- first_name: Boosaree
  full_name: Titapiwatanakun, Boosaree
  last_name: Titapiwatanakun
- first_name: Srinivas
  full_name: Makam, Srinivas N
  last_name: Makam
- first_name: Rodolphe
  full_name: Bouchard, Rodolphe
  last_name: Bouchard
- first_name: Markus
  full_name: Geisler, Markus
  last_name: Geisler
- first_name: Enrico
  full_name: Martinoia, Enrico
  last_name: Martinoia
- first_name: Jirí
  full_name: Jirí Friml
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
- first_name: Wendy
  full_name: Peer, Wendy A
  last_name: Peer
- first_name: Angus
  full_name: Murphy, Angus S
  last_name: Murphy
citation:
  ama: 'Bandyopadhyay A, Blakeslee J, Lee O, et al. Interactions of PIN and PGP auxin
    transport mechanisms. In: Vol 35. Portland Press; 2007:137-141. doi:<a href="https://doi.org/10.1042/BST0350137">10.1042/BST0350137</a>'
  apa: Bandyopadhyay, A., Blakeslee, J., Lee, O., Mravec, J., Sauer, M., Titapiwatanakun,
    B., … Murphy, A. (2007). Interactions of PIN and PGP auxin transport mechanisms
    (Vol. 35, pp. 137–141). Presented at the Intercellular Signalling in Plants, Portland
    Press. <a href="https://doi.org/10.1042/BST0350137">https://doi.org/10.1042/BST0350137</a>
  chicago: Bandyopadhyay, Anindita, Joshua Blakeslee, Ok Lee, Jozef Mravec, Michael
    Sauer, Boosaree Titapiwatanakun, Srinivas Makam, et al. “Interactions of PIN and
    PGP Auxin Transport Mechanisms,” 35:137–41. Portland Press, 2007. <a href="https://doi.org/10.1042/BST0350137">https://doi.org/10.1042/BST0350137</a>.
  ieee: A. Bandyopadhyay <i>et al.</i>, “Interactions of PIN and PGP auxin transport
    mechanisms,” presented at the Intercellular Signalling in Plants, 2007, vol. 35,
    no. 1, pp. 137–141.
  ista: Bandyopadhyay A, Blakeslee J, Lee O, Mravec J, Sauer M, Titapiwatanakun B,
    Makam S, Bouchard R, Geisler M, Martinoia E, Friml J, Peer W, Murphy A. 2007.
    Interactions of PIN and PGP auxin transport mechanisms. Intercellular Signalling
    in Plants, Biochemical Society Transactions, vol. 35, 137–141.
  mla: Bandyopadhyay, Anindita, et al. <i>Interactions of PIN and PGP Auxin Transport
    Mechanisms</i>. Vol. 35, no. 1, Portland Press, 2007, pp. 137–41, doi:<a href="https://doi.org/10.1042/BST0350137">10.1042/BST0350137</a>.
  short: A. Bandyopadhyay, J. Blakeslee, O. Lee, J. Mravec, M. Sauer, B. Titapiwatanakun,
    S. Makam, R. Bouchard, M. Geisler, E. Martinoia, J. Friml, W. Peer, A. Murphy,
    in:, Portland Press, 2007, pp. 137–141.
conference:
  name: Intercellular Signalling in Plants
date_created: 2018-12-11T12:00:54Z
date_published: 2007-02-01T00:00:00Z
date_updated: 2021-01-12T07:40:30Z
day: '01'
doi: 10.1042/BST0350137
extern: 1
intvolume: '        35'
issue: '1'
month: '02'
page: 137 - 141
publication_status: published
publisher: Portland Press
publist_id: '3681'
quality_controlled: 0
status: public
title: Interactions of PIN and PGP auxin transport mechanisms
type: conference
volume: 35
year: '2007'
...
---
_id: '3022'
abstract:
- lang: eng
  text: Endocytosis is an essential process by which eukaryotic cells internalize
    exogenous material or regulate signaling at the cell surface [1]. Different endocytic
    pathways are well established in yeast and animals; prominent among them is clathrin-dependent
    endocytosis [2, 3]. In plants, endocytosis is poorly defined, and no molecular
    mechanism for cargo internalization has been demonstrated so far [4, 5], although
    the internalization of receptor-ligand complexes at the plant plasma membrane
    has recently been shown [6]. Here we demonstrate by means of a green-to-red photoconvertible
    fluorescent reporter, EosFP [7], the constitutive endocytosis of PIN auxin efflux
    carriers [8] and their recycling to the plasma membrane. Using a plant clathrin-specific
    antibody, we show the presence of clathrin at different stages of coated-vesicle
    formation at the plasma membrane in Arabidopsis. Genetic interference with clathrin
    function inhibits PIN internalization and endocytosis in general. Furthermore,
    pharmacological interference with cargo recruitment into the clathrin pathway
    blocks internalization of PINs and other plasma-membrane proteins. Our data demonstrate
    that clathrin-dependent endocytosis is operational in plants and constitutes the
    predominant pathway for the internalization of numerous plasma-membrane-resident
    proteins including PIN auxin efflux carriers.
author:
- first_name: Pankaj
  full_name: Dhonukshe, Pankaj
  last_name: Dhonukshe
- first_name: Fernando
  full_name: Aniento, Fernando
  last_name: Aniento
- first_name: Inhwan
  full_name: Hwang, Inhwan
  last_name: Hwang
- first_name: David
  full_name: Robinson, David G
  last_name: Robinson
- first_name: Jozef
  full_name: Mravec, Jozef
  last_name: Mravec
- first_name: York
  full_name: Stierhof, York-Dieter
  last_name: Stierhof
- first_name: Jirí
  full_name: Jirí Friml
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
citation:
  ama: Dhonukshe P, Aniento F, Hwang I, et al. Clathrin-mediated constitutive endocytosis
    of PIN auxin efflux carriers in Arabidopsis. <i>Current Biology</i>. 2007;17(6):520-527.
    doi:<a href="https://doi.org/10.1016/j.cub.2007.01.052">10.1016/j.cub.2007.01.052</a>
  apa: Dhonukshe, P., Aniento, F., Hwang, I., Robinson, D., Mravec, J., Stierhof,
    Y., &#38; Friml, J. (2007). Clathrin-mediated constitutive endocytosis of PIN
    auxin efflux carriers in Arabidopsis. <i>Current Biology</i>. Cell Press. <a href="https://doi.org/10.1016/j.cub.2007.01.052">https://doi.org/10.1016/j.cub.2007.01.052</a>
  chicago: Dhonukshe, Pankaj, Fernando Aniento, Inhwan Hwang, David Robinson, Jozef
    Mravec, York Stierhof, and Jiří Friml. “Clathrin-Mediated Constitutive Endocytosis
    of PIN Auxin Efflux Carriers in Arabidopsis.” <i>Current Biology</i>. Cell Press,
    2007. <a href="https://doi.org/10.1016/j.cub.2007.01.052">https://doi.org/10.1016/j.cub.2007.01.052</a>.
  ieee: P. Dhonukshe <i>et al.</i>, “Clathrin-mediated constitutive endocytosis of
    PIN auxin efflux carriers in Arabidopsis,” <i>Current Biology</i>, vol. 17, no.
    6. Cell Press, pp. 520–527, 2007.
  ista: Dhonukshe P, Aniento F, Hwang I, Robinson D, Mravec J, Stierhof Y, Friml J.
    2007. Clathrin-mediated constitutive endocytosis of PIN auxin efflux carriers
    in Arabidopsis. Current Biology. 17(6), 520–527.
  mla: Dhonukshe, Pankaj, et al. “Clathrin-Mediated Constitutive Endocytosis of PIN
    Auxin Efflux Carriers in Arabidopsis.” <i>Current Biology</i>, vol. 17, no. 6,
    Cell Press, 2007, pp. 520–27, doi:<a href="https://doi.org/10.1016/j.cub.2007.01.052">10.1016/j.cub.2007.01.052</a>.
  short: P. Dhonukshe, F. Aniento, I. Hwang, D. Robinson, J. Mravec, Y. Stierhof,
    J. Friml, Current Biology 17 (2007) 520–527.
date_created: 2018-12-11T12:00:55Z
date_published: 2007-03-24T00:00:00Z
date_updated: 2021-01-12T07:40:31Z
day: '24'
doi: 10.1016/j.cub.2007.01.052
extern: 1
intvolume: '        17'
issue: '6'
month: '03'
page: 520 - 527
publication: Current Biology
publication_status: published
publisher: Cell Press
publist_id: '3680'
quality_controlled: 0
status: public
title: Clathrin-mediated constitutive endocytosis of PIN auxin efflux carriers in
  Arabidopsis
type: journal_article
volume: 17
year: '2007'
...
---
_id: '3023'
abstract:
- lang: eng
  text: 'Cytokinesis ensures proper partitioning of the nucleocytoplasmic contents
    into two daughter cells. It has generally been thought that cytokinesis is accomplished
    differently in animals and plants because of the differences in the preparatory
    phases, into the centrosomal or acentrosomal nature of the process, the presence
    or absence of rigid cell walls, and on the basis of ‘outside‐in’ or ‘inside‐out’
    mechanism. However, this long‐standing paradigm needs further reevaluation based
    on new findings. Recent advances reveal that plant cells, similarly to animal
    cells, possess astral microtubules that regulate the cell division plane. Furthermore,
    endocytosis has been found to be important for cytokinesis in animal and plant
    cells: vesicles containing endocytosed cargo provide material for the cell plate
    formation in plants and for closure of the midbody channel in animals. Thus, although
    the preparatory phases of the cell division process differ between plant and animal
    cells, the later phases show similarities. We unify these findings in a model
    that suggests a conserved mode of cytokinesis.'
author:
- first_name: Pankaj
  full_name: Dhonukshe, Pankaj
  last_name: Dhonukshe
- first_name: Jozef
  full_name: Šamaj, Jozef
  last_name: Šamaj
- first_name: František
  full_name: Baluška, František
  last_name: Baluška
- first_name: Jirí
  full_name: Friml, Jirí
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
citation:
  ama: 'Dhonukshe P, Šamaj J, Baluška F, Friml J. A unifying new model of cytokinesis
    for the dividing plant and animal cells. <i>Bioessays : News and Reviews in Molecular,
    Cellular and Developmental Biology</i>. 2007;29(4):371-381. doi:<a href="https://doi.org/10.1002/bies.20559">10.1002/bies.20559</a>'
  apa: 'Dhonukshe, P., Šamaj, J., Baluška, F., &#38; Friml, J. (2007). A unifying
    new model of cytokinesis for the dividing plant and animal cells. <i>Bioessays :
    News and Reviews in Molecular, Cellular and Developmental Biology</i>. Wiley-Blackwell.
    <a href="https://doi.org/10.1002/bies.20559">https://doi.org/10.1002/bies.20559</a>'
  chicago: 'Dhonukshe, Pankaj, Jozef Šamaj, František Baluška, and Jiří Friml. “A
    Unifying New Model of Cytokinesis for the Dividing Plant and Animal Cells.” <i>Bioessays :
    News and Reviews in Molecular, Cellular and Developmental Biology</i>. Wiley-Blackwell,
    2007. <a href="https://doi.org/10.1002/bies.20559">https://doi.org/10.1002/bies.20559</a>.'
  ieee: 'P. Dhonukshe, J. Šamaj, F. Baluška, and J. Friml, “A unifying new model of
    cytokinesis for the dividing plant and animal cells,” <i>Bioessays : News and
    Reviews in Molecular, Cellular and Developmental Biology</i>, vol. 29, no. 4.
    Wiley-Blackwell, pp. 371–381, 2007.'
  ista: 'Dhonukshe P, Šamaj J, Baluška F, Friml J. 2007. A unifying new model of cytokinesis
    for the dividing plant and animal cells. Bioessays : News and Reviews in Molecular,
    Cellular and Developmental Biology. 29(4), 371–381.'
  mla: 'Dhonukshe, Pankaj, et al. “A Unifying New Model of Cytokinesis for the Dividing
    Plant and Animal Cells.” <i>Bioessays : News and Reviews in Molecular, Cellular
    and Developmental Biology</i>, vol. 29, no. 4, Wiley-Blackwell, 2007, pp. 371–81,
    doi:<a href="https://doi.org/10.1002/bies.20559">10.1002/bies.20559</a>.'
  short: 'P. Dhonukshe, J. Šamaj, F. Baluška, J. Friml, Bioessays : News and Reviews
    in Molecular, Cellular and Developmental Biology 29 (2007) 371–381.'
date_created: 2018-12-11T12:00:55Z
date_published: 2007-04-01T00:00:00Z
date_updated: 2021-01-12T07:40:31Z
day: '01'
doi: 10.1002/bies.20559
extern: '1'
external_id:
  pmid:
  - '    17373659'
intvolume: '        29'
issue: '4'
language:
- iso: eng
month: '04'
oa_version: None
page: 371 - 381
pmid: 1
publication: 'Bioessays : News and Reviews in Molecular, Cellular and Developmental
  Biology'
publication_status: published
publisher: Wiley-Blackwell
publist_id: '3678'
quality_controlled: '1'
status: public
title: A unifying new model of cytokinesis for the dividing plant and animal cells
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 29
year: '2007'
...
---
_id: '3024'
abstract:
- lang: eng
  text: The plant hormone auxin is frequently observed to be asymmetrically distributed
    across adjacent cells during crucial stages of growth and development. These auxin
    gradients depend on polar transport and regulate a wide variety of processes,
    including embryogenesis, organogenesis, vascular tissue differentiation, root
    meristem maintenance and tropic growth. Auxin can mediate such a perplexing array
    of developmental processes by acting as a general trigger for the change in developmental
    program in cells where it accumulates and by providing vectorial information to
    the tissues by its polar intercellular flow. In recent years, a wealth of molecular
    data on the mechanism of auxin transport and its regulation has been generated,
    providing significant insights into the action of this versatile coordinative
    signal.
author:
- first_name: Anne
  full_name: Vieten, Anne
  last_name: Vieten
- first_name: Michael
  full_name: Sauer, Michael
  last_name: Sauer
- first_name: Philip
  full_name: Brewer, Philip
  last_name: Brewer
- first_name: Jirí
  full_name: Friml, Jirí
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
citation:
  ama: Vieten A, Sauer M, Brewer P, Friml J. Molecular and cellular aspects of auxin-transport-mediated
    development. <i>Trends in Plant Science</i>. 2007;12(4):160-168. doi:<a href="https://doi.org/10.1016/j.tplants.2007.03.006">10.1016/j.tplants.2007.03.006</a>
  apa: Vieten, A., Sauer, M., Brewer, P., &#38; Friml, J. (2007). Molecular and cellular
    aspects of auxin-transport-mediated development. <i>Trends in Plant Science</i>.
    Cell Press. <a href="https://doi.org/10.1016/j.tplants.2007.03.006">https://doi.org/10.1016/j.tplants.2007.03.006</a>
  chicago: Vieten, Anne, Michael Sauer, Philip Brewer, and Jiří Friml. “Molecular
    and Cellular Aspects of Auxin-Transport-Mediated Development.” <i>Trends in Plant
    Science</i>. Cell Press, 2007. <a href="https://doi.org/10.1016/j.tplants.2007.03.006">https://doi.org/10.1016/j.tplants.2007.03.006</a>.
  ieee: A. Vieten, M. Sauer, P. Brewer, and J. Friml, “Molecular and cellular aspects
    of auxin-transport-mediated development,” <i>Trends in Plant Science</i>, vol.
    12, no. 4. Cell Press, pp. 160–168, 2007.
  ista: Vieten A, Sauer M, Brewer P, Friml J. 2007. Molecular and cellular aspects
    of auxin-transport-mediated development. Trends in Plant Science. 12(4), 160–168.
  mla: Vieten, Anne, et al. “Molecular and Cellular Aspects of Auxin-Transport-Mediated
    Development.” <i>Trends in Plant Science</i>, vol. 12, no. 4, Cell Press, 2007,
    pp. 160–68, doi:<a href="https://doi.org/10.1016/j.tplants.2007.03.006">10.1016/j.tplants.2007.03.006</a>.
  short: A. Vieten, M. Sauer, P. Brewer, J. Friml, Trends in Plant Science 12 (2007)
    160–168.
date_created: 2018-12-11T12:00:55Z
date_published: 2007-04-01T00:00:00Z
date_updated: 2021-01-12T07:40:31Z
day: '01'
doi: 10.1016/j.tplants.2007.03.006
extern: '1'
external_id:
  pmid:
  - '    17369077'
intvolume: '        12'
issue: '4'
language:
- iso: eng
month: '04'
oa_version: None
page: 160 - 168
pmid: 1
publication: Trends in Plant Science
publication_status: published
publisher: Cell Press
publist_id: '3679'
quality_controlled: '1'
status: public
title: Molecular and cellular aspects of auxin-transport-mediated development
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 12
year: '2007'
...
---
_id: '3026'
abstract:
- lang: eng
  text: In plants, each developmental process integrates a network of signaling events
    that are regulated by different phytohormones, and interactions among hormonal
    pathways are essential to modulate their effect. Continuous growth of roots results
    from the postembryonic activity of cells within the root meristem that is controlled
    by the coordinated action of several phytohormones, including auxin and ethylene.
    Although their interaction has been studied intensively, the molecular and cellular
    mechanisms underlying this interplay are unknown. We show that the effect of ethylene
    on root growth is largely mediated by the regulation of the auxin biosynthesis
    and transport-dependent local auxin distribution. Ethylene stimulates auxin biosynthesis
    and basipetal auxin transport toward the elongation zone, where it activates a
    local auxin response leading to inhibition of cell elongation. Consistently, in
    mutants affected in auxin perception or basipetal auxin transport, ethylene cannot
    activate the auxin response nor regulate the root growth. In addition, ethylene
    modulates the transcription of several components of the auxin transport machinery.
    Thus, ethylene achieves a local activation of the auxin signaling pathway and
    regulates root growth by both stimulating the auxin biosynthesis and by modulating
    the auxin transport machinery.
author:
- first_name: Kamil
  full_name: Růžička, Kamil
  last_name: Růžička
- first_name: Karin
  full_name: Ljung, Karin
  last_name: Ljung
- first_name: Steffen
  full_name: Vanneste, Steffen
  last_name: Vanneste
- first_name: Radka
  full_name: Podhorská, Radka
  last_name: Podhorská
- first_name: Tom
  full_name: Beeckman, Tom
  last_name: Beeckman
- first_name: Jirí
  full_name: Jirí Friml
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
- first_name: Eva
  full_name: Eva Benková
  id: 38F4F166-F248-11E8-B48F-1D18A9856A87
  last_name: Benková
  orcid: 0000-0002-8510-9739
citation:
  ama: Růžička K, Ljung K, Vanneste S, et al. Ethylene regulates root growth through
    effects on auxin biosynthesis and transport dependent auxin distribution. <i>Plant
    Cell</i>. 2007;19(7):2197-2212. doi:<a href="https://doi.org/10.1105/tpc.107.052126">10.1105/tpc.107.052126</a>
  apa: Růžička, K., Ljung, K., Vanneste, S., Podhorská, R., Beeckman, T., Friml, J.,
    &#38; Benková, E. (2007). Ethylene regulates root growth through effects on auxin
    biosynthesis and transport dependent auxin distribution. <i>Plant Cell</i>. American
    Society of Plant Biologists. <a href="https://doi.org/10.1105/tpc.107.052126">https://doi.org/10.1105/tpc.107.052126</a>
  chicago: Růžička, Kamil, Karin Ljung, Steffen Vanneste, Radka Podhorská, Tom Beeckman,
    Jiří Friml, and Eva Benková. “Ethylene Regulates Root Growth through Effects on
    Auxin Biosynthesis and Transport Dependent Auxin Distribution.” <i>Plant Cell</i>.
    American Society of Plant Biologists, 2007. <a href="https://doi.org/10.1105/tpc.107.052126">https://doi.org/10.1105/tpc.107.052126</a>.
  ieee: K. Růžička <i>et al.</i>, “Ethylene regulates root growth through effects
    on auxin biosynthesis and transport dependent auxin distribution,” <i>Plant Cell</i>,
    vol. 19, no. 7. American Society of Plant Biologists, pp. 2197–2212, 2007.
  ista: Růžička K, Ljung K, Vanneste S, Podhorská R, Beeckman T, Friml J, Benková
    E. 2007. Ethylene regulates root growth through effects on auxin biosynthesis
    and transport dependent auxin distribution. Plant Cell. 19(7), 2197–2212.
  mla: Růžička, Kamil, et al. “Ethylene Regulates Root Growth through Effects on Auxin
    Biosynthesis and Transport Dependent Auxin Distribution.” <i>Plant Cell</i>, vol.
    19, no. 7, American Society of Plant Biologists, 2007, pp. 2197–212, doi:<a href="https://doi.org/10.1105/tpc.107.052126">10.1105/tpc.107.052126</a>.
  short: K. Růžička, K. Ljung, S. Vanneste, R. Podhorská, T. Beeckman, J. Friml, E.
    Benková, Plant Cell 19 (2007) 2197–2212.
date_created: 2018-12-11T12:00:56Z
date_published: 2007-07-01T00:00:00Z
date_updated: 2021-01-12T07:40:32Z
day: '01'
doi: 10.1105/tpc.107.052126
extern: 1
intvolume: '        19'
issue: '7'
month: '07'
page: 2197 - 2212
publication: Plant Cell
publication_status: published
publisher: American Society of Plant Biologists
publist_id: '3676'
quality_controlled: 0
status: public
title: Ethylene regulates root growth through effects on auxin biosynthesis and transport
  dependent auxin distribution
type: journal_article
volume: 19
year: '2007'
...
---
_id: '3027'
abstract:
- lang: eng
  text: Polar transport of the phytohormone auxin controls numerous growth responses
    in plants. Molecular characterization of auxin transport in Arabidopsis thaliana
    has provided important insights into the mechanisms underlying the regulation
    of auxin distribution. In particular, the control of subcellular localization
    and expression of PIN-type auxin efflux components appears to be fundamental for
    orchestrated distribution of the growth regulator throughout the entire plant
    body. Here we describe the identification of two Arabidopsis loci, MOP2 and MOP3
    (for MODULATOR OF PIN), that are involved in control of the steady-state levels
    of PIN protein. Mutations in both loci result in defects in auxin distribution
    and polar auxin transport, and cause phenotypes consistent with a reduction of
    PIN protein levels. Genetic interaction between PIN2 and both MOP loci is suggestive
    of functional cross-talk, which is further substantiated by findings demonstrating
    that ectopic PIN up-regulation is compensated in the mop background. Thus, in
    addition to pathways that control PIN localization and transcription, MOP2 and
    MOP3 appear to be involved in fine-tuning of auxin distribution via post-transcriptional
    regulation of PIN expression.
author:
- first_name: Nenad
  full_name: Malenica, Nenad
  last_name: Malenica
- first_name: Lindy
  full_name: Abas, Lindy
  last_name: Abas
- first_name: René
  full_name: Benjamins, René
  last_name: Benjamins
- first_name: Saeko
  full_name: Kitakura, Saeko
  last_name: Kitakura
- first_name: Harald
  full_name: Sigmund, Harald F
  last_name: Sigmund
- first_name: Kim
  full_name: Jun, Kim S
  last_name: Jun
- first_name: Marie
  full_name: Hauser, Marie-Theres
  last_name: Hauser
- first_name: Jirí
  full_name: Jirí Friml
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
- first_name: Christian
  full_name: Luschnig, Christian
  last_name: Luschnig
citation:
  ama: Malenica N, Abas L, Benjamins R, et al. MODULATOR of PIN genes control steady
    state levels of Arabidopsis PIN proteins. <i>Plant Journal</i>. 2007;51(4):537-550.
    doi:<a href="https://doi.org/10.1111/j.1365-313X.2007.03158.x">10.1111/j.1365-313X.2007.03158.x</a>
  apa: Malenica, N., Abas, L., Benjamins, R., Kitakura, S., Sigmund, H., Jun, K.,
    … Luschnig, C. (2007). MODULATOR of PIN genes control steady state levels of Arabidopsis
    PIN proteins. <i>Plant Journal</i>. Wiley-Blackwell. <a href="https://doi.org/10.1111/j.1365-313X.2007.03158.x">https://doi.org/10.1111/j.1365-313X.2007.03158.x</a>
  chicago: Malenica, Nenad, Lindy Abas, René Benjamins, Saeko Kitakura, Harald Sigmund,
    Kim Jun, Marie Hauser, Jiří Friml, and Christian Luschnig. “MODULATOR of PIN Genes
    Control Steady State Levels of Arabidopsis PIN Proteins.” <i>Plant Journal</i>.
    Wiley-Blackwell, 2007. <a href="https://doi.org/10.1111/j.1365-313X.2007.03158.x">https://doi.org/10.1111/j.1365-313X.2007.03158.x</a>.
  ieee: N. Malenica <i>et al.</i>, “MODULATOR of PIN genes control steady state levels
    of Arabidopsis PIN proteins,” <i>Plant Journal</i>, vol. 51, no. 4. Wiley-Blackwell,
    pp. 537–550, 2007.
  ista: Malenica N, Abas L, Benjamins R, Kitakura S, Sigmund H, Jun K, Hauser M, Friml
    J, Luschnig C. 2007. MODULATOR of PIN genes control steady state levels of Arabidopsis
    PIN proteins. Plant Journal. 51(4), 537–550.
  mla: Malenica, Nenad, et al. “MODULATOR of PIN Genes Control Steady State Levels
    of Arabidopsis PIN Proteins.” <i>Plant Journal</i>, vol. 51, no. 4, Wiley-Blackwell,
    2007, pp. 537–50, doi:<a href="https://doi.org/10.1111/j.1365-313X.2007.03158.x">10.1111/j.1365-313X.2007.03158.x</a>.
  short: N. Malenica, L. Abas, R. Benjamins, S. Kitakura, H. Sigmund, K. Jun, M. Hauser,
    J. Friml, C. Luschnig, Plant Journal 51 (2007) 537–550.
date_created: 2018-12-11T12:00:56Z
date_published: 2007-08-01T00:00:00Z
date_updated: 2021-01-12T07:40:32Z
day: '01'
doi: 10.1111/j.1365-313X.2007.03158.x
extern: 1
intvolume: '        51'
issue: '4'
month: '08'
page: 537 - 550
publication: Plant Journal
publication_status: published
publisher: Wiley-Blackwell
publist_id: '3675'
quality_controlled: 0
status: public
title: MODULATOR of PIN genes control steady state levels of Arabidopsis PIN proteins
type: journal_article
volume: 51
year: '2007'
...
---
_id: '3028'
abstract:
- lang: eng
  text: In plants, cell polarity and tissue patterning are connected by intercellular
    flow of the phytohormone auxin, whose directional signaling depends on polar subcellular
    localization of PIN auxin transport proteins. The mechanism of polar targeting
    of PINs or other cargos in plants is largely unidentified, with the PINOID kinase
    being the only known molecular component. Here, we identify PP2A phosphatase as
    an important regulator of PIN apical-basal targeting and auxin distribution. Genetic
    analysis, localization, and phosphorylation studies demonstrate that PP2A and
    PINOID both partially colocalize with PINs and act antagonistically on the phosphorylation
    state of their central hydrophilic loop, hence mediating PIN apical-basal polar
    targeting. Thus, in plants, polar sorting by the reversible phosphorylation of
    cargos allows for their conditional delivery to specific intracellular destinations.
    In the case of PIN proteins, this mechanism enables switches in the direction
    of intercellular auxin fluxes, which mediate differential growth, tissue patterning,
    and organogenesis.
author:
- first_name: Marta
  full_name: Michniewicz, Marta
  last_name: Michniewicz
- first_name: Marcelo
  full_name: Zago, Marcelo K
  last_name: Zago
- first_name: Lindy
  full_name: Abas, Lindy
  last_name: Abas
- first_name: Dolf
  full_name: Weijers, Dolf
  last_name: Weijers
- first_name: Alois
  full_name: Schweighofer, Alois
  last_name: Schweighofer
- first_name: Irute
  full_name: Meskiene, Irute
  last_name: Meskiene
- first_name: Marcus
  full_name: Heisler, Marcus G
  last_name: Heisler
- first_name: Carolyn
  full_name: Ohno, Carolyn
  last_name: Ohno
- first_name: Jing
  full_name: Zhang, Jing
  last_name: Zhang
- first_name: Fang
  full_name: Huang, Fang
  last_name: Huang
- first_name: Rebecca
  full_name: Schwab, Rebecca
  last_name: Schwab
- first_name: Detlef
  full_name: Weigel, Detlef
  last_name: Weigel
- first_name: Elliot
  full_name: Meyerowitz, Elliot M
  last_name: Meyerowitz
- first_name: Christian
  full_name: Luschnig, Christian
  last_name: Luschnig
- first_name: Remko
  full_name: Offringa, Remko
  last_name: Offringa
- first_name: Jirí
  full_name: Jirí Friml
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
citation:
  ama: Michniewicz M, Zago M, Abas L, et al. Antagonistic regulation of PIN phosphorylation
    by PP2A and PINOID directs auxin flux. <i>Cell</i>. 2007;130(6):1044-1056. doi:<a
    href="https://doi.org/10.1016/j.cell.2007.07.033">10.1016/j.cell.2007.07.033</a>
  apa: Michniewicz, M., Zago, M., Abas, L., Weijers, D., Schweighofer, A., Meskiene,
    I., … Friml, J. (2007). Antagonistic regulation of PIN phosphorylation by PP2A
    and PINOID directs auxin flux. <i>Cell</i>. Cell Press. <a href="https://doi.org/10.1016/j.cell.2007.07.033">https://doi.org/10.1016/j.cell.2007.07.033</a>
  chicago: Michniewicz, Marta, Marcelo Zago, Lindy Abas, Dolf Weijers, Alois Schweighofer,
    Irute Meskiene, Marcus Heisler, et al. “Antagonistic Regulation of PIN Phosphorylation
    by PP2A and PINOID Directs Auxin Flux.” <i>Cell</i>. Cell Press, 2007. <a href="https://doi.org/10.1016/j.cell.2007.07.033">https://doi.org/10.1016/j.cell.2007.07.033</a>.
  ieee: M. Michniewicz <i>et al.</i>, “Antagonistic regulation of PIN phosphorylation
    by PP2A and PINOID directs auxin flux,” <i>Cell</i>, vol. 130, no. 6. Cell Press,
    pp. 1044–1056, 2007.
  ista: Michniewicz M, Zago M, Abas L, Weijers D, Schweighofer A, Meskiene I, Heisler
    M, Ohno C, Zhang J, Huang F, Schwab R, Weigel D, Meyerowitz E, Luschnig C, Offringa
    R, Friml J. 2007. Antagonistic regulation of PIN phosphorylation by PP2A and PINOID
    directs auxin flux. Cell. 130(6), 1044–1056.
  mla: Michniewicz, Marta, et al. “Antagonistic Regulation of PIN Phosphorylation
    by PP2A and PINOID Directs Auxin Flux.” <i>Cell</i>, vol. 130, no. 6, Cell Press,
    2007, pp. 1044–56, doi:<a href="https://doi.org/10.1016/j.cell.2007.07.033">10.1016/j.cell.2007.07.033</a>.
  short: M. Michniewicz, M. Zago, L. Abas, D. Weijers, A. Schweighofer, I. Meskiene,
    M. Heisler, C. Ohno, J. Zhang, F. Huang, R. Schwab, D. Weigel, E. Meyerowitz,
    C. Luschnig, R. Offringa, J. Friml, Cell 130 (2007) 1044–1056.
date_created: 2018-12-11T12:00:57Z
date_published: 2007-09-21T00:00:00Z
date_updated: 2021-01-12T07:40:33Z
day: '21'
doi: 10.1016/j.cell.2007.07.033
extern: 1
intvolume: '       130'
issue: '6'
month: '09'
page: 1044 - 1056
publication: Cell
publication_status: published
publisher: Cell Press
publist_id: '3674'
quality_controlled: 0
status: public
title: Antagonistic regulation of PIN phosphorylation by PP2A and PINOID directs auxin
  flux
type: journal_article
volume: 130
year: '2007'
...
---
_id: '3029'
abstract:
- lang: eng
  text: In Arabidopsis thaliana, lateral roots are formed from root pericycle cells
    adjacent to the xylem poles. Lateral root development is regulated antagonistically
    by the plant hormones auxin and cytokinin. While a great deal is known about how
    auxin promotes lateral root development, the mechanism of cytokinin repression
    is still unclear. Elevating cytokinin levels was observed to disrupt lateral root
    initiation and the regular pattern of divisions that characterizes lateral root
    development in Arabidopsis. To identify the stage of lateral root development
    that is sensitive to cytokinins, we targeted the expression of the Agrobacterium
    tumefaciens cytokinin biosynthesis enzyme isopentenyltransferase to either xylem-pole
    pericycle cells or young lateral root primordia using GAL4-GFP enhancer trap lines.
    Transactivation experiments revealed that xylem-pole pericycle cells are sensitive
    to cytokinins, whereas young lateral root primordia are not. This effect is physiologically
    significant because transactivation of the Arabidopsis cytokinin degrading enzyme
    cytokinin oxidase 1 in lateral root founder cells results in increased lateral
    root formation. We observed that cytokinins perturb the expression of PIN genes
    in lateral root founder cells and prevent the formation of an auxin gradient that
    is required to pattern lateral root primordia.
author:
- first_name: Laurent
  full_name: Laplaze, Laurent
  last_name: Laplaze
- first_name: Eva
  full_name: Eva Benková
  id: 38F4F166-F248-11E8-B48F-1D18A9856A87
  last_name: Benková
  orcid: 0000-0002-8510-9739
- first_name: Ilda
  full_name: Casimiro, Ilda
  last_name: Casimiro
- first_name: Lies
  full_name: Maes, Lies
  last_name: Maes
- first_name: Steffen
  full_name: Vanneste, Steffen
  last_name: Vanneste
- first_name: Ranjan
  full_name: Swarup, Ranjan
  last_name: Swarup
- first_name: Dolf
  full_name: Weijers, Dolf
  last_name: Weijers
- first_name: Vanessa
  full_name: Calvo, Vanessa
  last_name: Calvo
- first_name: Boris
  full_name: Parizot, Boris
  last_name: Parizot
- first_name: Maria
  full_name: Herrera-Rodriguez, Maria Begoña
  last_name: Herrera Rodriguez
- first_name: Remko
  full_name: Offringa, Remko
  last_name: Offringa
- first_name: Neil
  full_name: Graham, Neil
  last_name: Graham
- first_name: Patrick
  full_name: Doumas, Patrick
  last_name: Doumas
- first_name: Jirí
  full_name: Jirí Friml
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
- first_name: Didier
  full_name: Bogusz, Didier
  last_name: Bogusz
- first_name: Tom
  full_name: Beeckman, Tom
  last_name: Beeckman
- first_name: Malcolm
  full_name: Bennett, Malcolm
  last_name: Bennett
citation:
  ama: Laplaze L, Benková E, Casimiro I, et al. Cytokinins act directly on lateral
    root founder cells to inhibit root initiation. <i>Plant Cell</i>. 2007;19(12):3889-3900.
    doi:<a href="https://doi.org/10.1105/tpc.107.055863">10.1105/tpc.107.055863</a>
  apa: Laplaze, L., Benková, E., Casimiro, I., Maes, L., Vanneste, S., Swarup, R.,
    … Bennett, M. (2007). Cytokinins act directly on lateral root founder cells to
    inhibit root initiation. <i>Plant Cell</i>. American Society of Plant Biologists.
    <a href="https://doi.org/10.1105/tpc.107.055863">https://doi.org/10.1105/tpc.107.055863</a>
  chicago: Laplaze, Laurent, Eva Benková, Ilda Casimiro, Lies Maes, Steffen Vanneste,
    Ranjan Swarup, Dolf Weijers, et al. “Cytokinins Act Directly on Lateral Root Founder
    Cells to Inhibit Root Initiation.” <i>Plant Cell</i>. American Society of Plant
    Biologists, 2007. <a href="https://doi.org/10.1105/tpc.107.055863">https://doi.org/10.1105/tpc.107.055863</a>.
  ieee: L. Laplaze <i>et al.</i>, “Cytokinins act directly on lateral root founder
    cells to inhibit root initiation,” <i>Plant Cell</i>, vol. 19, no. 12. American
    Society of Plant Biologists, pp. 3889–3900, 2007.
  ista: Laplaze L, Benková E, Casimiro I, Maes L, Vanneste S, Swarup R, Weijers D,
    Calvo V, Parizot B, Herrera Rodriguez M, Offringa R, Graham N, Doumas P, Friml
    J, Bogusz D, Beeckman T, Bennett M. 2007. Cytokinins act directly on lateral root
    founder cells to inhibit root initiation. Plant Cell. 19(12), 3889–3900.
  mla: Laplaze, Laurent, et al. “Cytokinins Act Directly on Lateral Root Founder Cells
    to Inhibit Root Initiation.” <i>Plant Cell</i>, vol. 19, no. 12, American Society
    of Plant Biologists, 2007, pp. 3889–900, doi:<a href="https://doi.org/10.1105/tpc.107.055863">10.1105/tpc.107.055863</a>.
  short: L. Laplaze, E. Benková, I. Casimiro, L. Maes, S. Vanneste, R. Swarup, D.
    Weijers, V. Calvo, B. Parizot, M. Herrera Rodriguez, R. Offringa, N. Graham, P.
    Doumas, J. Friml, D. Bogusz, T. Beeckman, M. Bennett, Plant Cell 19 (2007) 3889–3900.
date_created: 2018-12-11T12:00:57Z
date_published: 2007-12-01T00:00:00Z
date_updated: 2021-01-12T07:40:33Z
day: '01'
doi: 10.1105/tpc.107.055863
extern: 1
intvolume: '        19'
issue: '12'
month: '12'
page: 3889 - 3900
publication: Plant Cell
publication_status: published
publisher: American Society of Plant Biologists
publist_id: '3673'
quality_controlled: 0
status: public
title: Cytokinins act directly on lateral root founder cells to inhibit root initiation
type: journal_article
volume: 19
year: '2007'
...
---
_id: '3144'
abstract:
- lang: eng
  text: Accumulation of specific proteins at synaptic structures is essential for
    synapse assembly and function, but mechanisms regulating local protein enrichment
    remain poorly understood. At the neuromuscular junction (NMJ), subsynaptic nuclei
    underlie motor axon terminals within extrafusal muscle fibers and are transcriptionally
    distinct from neighboring nuclei. In this study, we show that expression of the
    ETS transcription factor Erm is highly concentrated at subsynaptic nuclei, and
    its mutation in mice leads to severe downregulation of many genes with normally
    enriched subsynaptic expression. Erm mutant mice display an expansion of the muscle
    central domain in which acetylcholine receptor (AChR) clusters accumulate, show
    gradual fragmentation of AChR clusters, and exhibit symptoms of muscle weakness
    mimicking congenital myasthenic syndrome (CMS). Together, our findings define
    Erm as an upstream regulator of a transcriptional program selective to subsynaptic
    nuclei at the NMJ and underscore the importance of transcriptional control of
    local synaptic protein accumulation.
author:
- first_name: Simon
  full_name: Simon Hippenmeyer
  id: 37B36620-F248-11E8-B48F-1D18A9856A87
  last_name: Hippenmeyer
  orcid: 0000-0003-2279-1061
- first_name: Roland
  full_name: Huber, Roland M
  last_name: Huber
- first_name: David
  full_name: Ladle, David R
  last_name: Ladle
- first_name: Kenneth
  full_name: Murphy, Kenneth
  last_name: Murphy
- first_name: Silvia
  full_name: Arber, Silvia
  last_name: Arber
citation:
  ama: Hippenmeyer S, Huber R, Ladle D, Murphy K, Arber S. ETS transcription factor
    Erm controls subsynaptic gene expression in skeletal muscles. <i>Neuron</i>. 2007;55(5):726-740.
    doi:<a href="https://doi.org/10.1016/j.neuron.2007.07.028">10.1016/j.neuron.2007.07.028</a>
  apa: Hippenmeyer, S., Huber, R., Ladle, D., Murphy, K., &#38; Arber, S. (2007).
    ETS transcription factor Erm controls subsynaptic gene expression in skeletal
    muscles. <i>Neuron</i>. Elsevier. <a href="https://doi.org/10.1016/j.neuron.2007.07.028">https://doi.org/10.1016/j.neuron.2007.07.028</a>
  chicago: Hippenmeyer, Simon, Roland Huber, David Ladle, Kenneth Murphy, and Silvia
    Arber. “ETS Transcription Factor Erm Controls Subsynaptic Gene Expression in Skeletal
    Muscles.” <i>Neuron</i>. Elsevier, 2007. <a href="https://doi.org/10.1016/j.neuron.2007.07.028">https://doi.org/10.1016/j.neuron.2007.07.028</a>.
  ieee: S. Hippenmeyer, R. Huber, D. Ladle, K. Murphy, and S. Arber, “ETS transcription
    factor Erm controls subsynaptic gene expression in skeletal muscles,” <i>Neuron</i>,
    vol. 55, no. 5. Elsevier, pp. 726–740, 2007.
  ista: Hippenmeyer S, Huber R, Ladle D, Murphy K, Arber S. 2007. ETS transcription
    factor Erm controls subsynaptic gene expression in skeletal muscles. Neuron. 55(5),
    726–740.
  mla: Hippenmeyer, Simon, et al. “ETS Transcription Factor Erm Controls Subsynaptic
    Gene Expression in Skeletal Muscles.” <i>Neuron</i>, vol. 55, no. 5, Elsevier,
    2007, pp. 726–40, doi:<a href="https://doi.org/10.1016/j.neuron.2007.07.028">10.1016/j.neuron.2007.07.028</a>.
  short: S. Hippenmeyer, R. Huber, D. Ladle, K. Murphy, S. Arber, Neuron 55 (2007)
    726–740.
date_created: 2018-12-11T12:01:39Z
date_published: 2007-09-06T00:00:00Z
date_updated: 2021-01-12T07:41:22Z
day: '06'
doi: 10.1016/j.neuron.2007.07.028
extern: 1
intvolume: '        55'
issue: '5'
month: '09'
page: 726 - 740
publication: Neuron
publication_status: published
publisher: Elsevier
publist_id: '3551'
quality_controlled: 0
status: public
title: ETS transcription factor Erm controls subsynaptic gene expression in skeletal
  muscles
type: journal_article
volume: 55
year: '2007'
...
---
_id: '3187'
abstract:
- lang: eng
  text: Stereo vision has numerous applications in robotics, graphics, inspection
    and other areas. A prime application, one which has driven work on stereo in our
    laboratory, is teleconferencing in which the use of a stereo webcam already makes
    possible various transformations of the video stream. These include digital camera
    control, insertion of virtual objects, background substitution, and eye-gaze correction
    [9, 8].
author:
- first_name: Andrew
  full_name: Blake, Andrew
  last_name: Blake
- first_name: Antonio
  full_name: Criminisi, Antonio
  last_name: Criminisi
- first_name: Geoffrey
  full_name: Cross, Geoffrey
  last_name: Cross
- first_name: Vladimir
  full_name: Vladimir Kolmogorov
  id: 3D50B0BA-F248-11E8-B48F-1D18A9856A87
  last_name: Kolmogorov
- first_name: Carsten
  full_name: Rother, Carsten
  last_name: Rother
citation:
  ama: Blake A, Criminisi A, Cross G, Kolmogorov V, Rother C. Fusion of stereo colour
    and contrast. <i>Springer Tracts in Advanced Robotics</i>. 2007;28:295-304. doi:<a
    href="https://doi.org/10.1007/978-3-540-48113-3_27">10.1007/978-3-540-48113-3_27</a>
  apa: Blake, A., Criminisi, A., Cross, G., Kolmogorov, V., &#38; Rother, C. (2007).
    Fusion of stereo colour and contrast. <i>Springer Tracts in Advanced Robotics</i>.
    Springer. <a href="https://doi.org/10.1007/978-3-540-48113-3_27">https://doi.org/10.1007/978-3-540-48113-3_27</a>
  chicago: Blake, Andrew, Antonio Criminisi, Geoffrey Cross, Vladimir Kolmogorov,
    and Carsten Rother. “Fusion of Stereo Colour and Contrast.” <i>Springer Tracts
    in Advanced Robotics</i>. Springer, 2007. <a href="https://doi.org/10.1007/978-3-540-48113-3_27">https://doi.org/10.1007/978-3-540-48113-3_27</a>.
  ieee: A. Blake, A. Criminisi, G. Cross, V. Kolmogorov, and C. Rother, “Fusion of
    stereo colour and contrast,” <i>Springer Tracts in Advanced Robotics</i>, vol.
    28. Springer, pp. 295–304, 2007.
  ista: Blake A, Criminisi A, Cross G, Kolmogorov V, Rother C. 2007. Fusion of stereo
    colour and contrast. Springer Tracts in Advanced Robotics. 28, 295–304.
  mla: Blake, Andrew, et al. “Fusion of Stereo Colour and Contrast.” <i>Springer Tracts
    in Advanced Robotics</i>, vol. 28, Springer, 2007, pp. 295–304, doi:<a href="https://doi.org/10.1007/978-3-540-48113-3_27">10.1007/978-3-540-48113-3_27</a>.
  short: A. Blake, A. Criminisi, G. Cross, V. Kolmogorov, C. Rother, Springer Tracts
    in Advanced Robotics 28 (2007) 295–304.
date_created: 2018-12-11T12:01:54Z
date_published: 2007-02-05T00:00:00Z
date_updated: 2021-01-12T07:41:40Z
day: '05'
doi: 10.1007/978-3-540-48113-3_27
extern: 1
intvolume: '        28'
main_file_link:
- open_access: '0'
  url: http://research.microsoft.com/pubs/67417/criminisi_isrr2005.pdf
month: '02'
page: 295 - 304
publication: Springer Tracts in Advanced Robotics
publication_status: published
publisher: Springer
publist_id: '3492'
quality_controlled: 0
status: public
title: Fusion of stereo colour and contrast
type: journal_article
volume: 28
year: '2007'
...
---
_id: '3191'
abstract:
- lang: eng
  text: 'The maximum flow algorithm for minimizing energy functions of binary variables
    has become a standard tool in computer vision. In many cases, unary costs of the
    energy depend linearly on parameter lambda. In this paper we study vision applications
    for which it is important to solve the maxflow problem for different lambda''s.
    An example is a weighting between data and regularization terms in image segmentation
    or stereo: it is desirable to vary it both during training (to learn lambda from
    ground truth data) and testing (to select best lambda using high-knowledge constraints,
    e.g. user input). We review algorithmic aspects of this parametric maximum flow
    problem previously unknown in vision, such as the ability to compute all breakpoints
    of lambda and corresponding optimal configurations infinite time. These results
    allow, in particular, to minimize the ratio of some geometric functional, such
    as flux of a vector field over length (or area). Previously, such functional were
    tackled with shortest path techniques applicable only in 2D. We give theoretical
    improvements for &quot;PDE cuts&quot; [5]. We present experimental results for
    image segmentation, 3D reconstruction, and the cosegmentation problem.'
author:
- first_name: Vladimir
  full_name: Vladimir Kolmogorov
  id: 3D50B0BA-F248-11E8-B48F-1D18A9856A87
  last_name: Kolmogorov
- first_name: Yuri
  full_name: Boykov, Yuri
  last_name: Boykov
- first_name: Carsten
  full_name: Rother, Carsten
  last_name: Rother
citation:
  ama: 'Kolmogorov V, Boykov Y, Rother C. Applications of parametric maxflow in computer
    vision. In: IEEE; 2007. doi:<a href="https://doi.org/10.1109/ICCV.2007.4408910">10.1109/ICCV.2007.4408910</a>'
  apa: 'Kolmogorov, V., Boykov, Y., &#38; Rother, C. (2007). Applications of parametric
    maxflow in computer vision. Presented at the ICCV: International Conference on
    Computer Vision, IEEE. <a href="https://doi.org/10.1109/ICCV.2007.4408910">https://doi.org/10.1109/ICCV.2007.4408910</a>'
  chicago: Kolmogorov, Vladimir, Yuri Boykov, and Carsten Rother. “Applications of
    Parametric Maxflow in Computer Vision.” IEEE, 2007. <a href="https://doi.org/10.1109/ICCV.2007.4408910">https://doi.org/10.1109/ICCV.2007.4408910</a>.
  ieee: 'V. Kolmogorov, Y. Boykov, and C. Rother, “Applications of parametric maxflow
    in computer vision,” presented at the ICCV: International Conference on Computer
    Vision, 2007.'
  ista: 'Kolmogorov V, Boykov Y, Rother C. 2007. Applications of parametric maxflow
    in computer vision. ICCV: International Conference on Computer Vision.'
  mla: Kolmogorov, Vladimir, et al. <i>Applications of Parametric Maxflow in Computer
    Vision</i>. IEEE, 2007, doi:<a href="https://doi.org/10.1109/ICCV.2007.4408910">10.1109/ICCV.2007.4408910</a>.
  short: V. Kolmogorov, Y. Boykov, C. Rother, in:, IEEE, 2007.
conference:
  name: 'ICCV: International Conference on Computer Vision'
date_created: 2018-12-11T12:01:55Z
date_published: 2007-12-26T00:00:00Z
date_updated: 2021-01-12T07:41:41Z
day: '26'
doi: 10.1109/ICCV.2007.4408910
extern: 1
main_file_link:
- open_access: '0'
  url: http://research.microsoft.com/pubs/70474/iccv07-paramaxflow.pdf
month: '12'
publication_status: published
publisher: IEEE
publist_id: '3489'
quality_controlled: 0
status: public
title: Applications of parametric maxflow in computer vision
type: conference
year: '2007'
...
