---
_id: '1888'
abstract:
- lang: ger
  text: 'Im Rahmen meiner Arbeit mit der kollektiven Krankheitsabwehr in Ameisengesellschaften
    interessiert mich vor allem, wie sich die Kolonien als Ganzes gegen Krankheiten
    wehren können. Warum ist dieses Thema der Krankheitsdynamik in Gruppen so wichtig?
    Ein Vergleich von solitär lebenden Individuen mit Individuen, die in sozialen
    Gruppen zusammenleben, zeigt die Kosten und die Vorteile des Gruppenlebens: Einerseits
    haben Individuen in sozialen Gruppen aufgrund der hohen Dichte, in der die Tiere
    zusammenleben, den hohen Interaktionsraten, die sie miteinander haben, und der
    engen Verwandtschaft, die sie verbindet, ein höheres Ansteckungsrisiko. Andererseits
    kann die individuelle Krankheitsabwehr durch die kollektive Abwehr in den Gruppen
    ergänzt werden.'
alternative_title:
- Rundgespräche der Kommission für Ökologie
article_processing_charge: No
author:
- first_name: Sylvia
  full_name: Cremer, Sylvia
  id: 2F64EC8C-F248-11E8-B48F-1D18A9856A87
  last_name: Cremer
  orcid: 0000-0002-2193-3868
citation:
  ama: 'Cremer S. Soziale Immunität: Wie sich der Staat gegen Pathogene wehrt  Bayerische
    Akademie der Wissenschaften. In: <i>Soziale Insekten in Einer Sich Wandelnden
    Welt</i>. Vol 43. Verlag Dr. Friedrich Pfeil; 2014:65-72.'
  apa: 'Cremer, S. (2014). Soziale Immunität: Wie sich der Staat gegen Pathogene wehrt 
    Bayerische Akademie der Wissenschaften. In <i>Soziale Insekten in einer sich wandelnden
    Welt</i> (Vol. 43, pp. 65–72). Verlag Dr. Friedrich Pfeil.'
  chicago: 'Cremer, Sylvia. “Soziale Immunität: Wie Sich Der Staat Gegen Pathogene
    Wehrt  Bayerische Akademie Der Wissenschaften.” In <i>Soziale Insekten in Einer
    Sich Wandelnden Welt</i>, 43:65–72. Verlag Dr. Friedrich Pfeil, 2014.'
  ieee: 'S. Cremer, “Soziale Immunität: Wie sich der Staat gegen Pathogene wehrt 
    Bayerische Akademie der Wissenschaften,” in <i>Soziale Insekten in einer sich
    wandelnden Welt</i>, vol. 43, Verlag Dr. Friedrich Pfeil, 2014, pp. 65–72.'
  ista: 'Cremer S. 2014.Soziale Immunität: Wie sich der Staat gegen Pathogene wehrt 
    Bayerische Akademie der Wissenschaften. In: Soziale Insekten in einer sich wandelnden
    Welt. Rundgespräche der Kommission für Ökologie, vol. 43, 65–72.'
  mla: 'Cremer, Sylvia. “Soziale Immunität: Wie Sich Der Staat Gegen Pathogene Wehrt 
    Bayerische Akademie Der Wissenschaften.” <i>Soziale Insekten in Einer Sich Wandelnden
    Welt</i>, vol. 43, Verlag Dr. Friedrich Pfeil, 2014, pp. 65–72.'
  short: S. Cremer, in:, Soziale Insekten in Einer Sich Wandelnden Welt, Verlag Dr.
    Friedrich Pfeil, 2014, pp. 65–72.
date_created: 2018-12-11T11:54:33Z
date_published: 2014-01-01T00:00:00Z
date_updated: 2023-10-17T12:28:45Z
day: '01'
department:
- _id: SyCr
intvolume: '        43'
language:
- iso: eng
month: '01'
oa_version: None
page: 65 - 72
publication: Soziale Insekten in einer sich wandelnden Welt
publication_identifier:
  issn:
  - 2366-2875
publication_status: published
publisher: Verlag Dr. Friedrich Pfeil
publist_id: '5207'
quality_controlled: '1'
status: public
title: 'Soziale Immunität: Wie sich der Staat gegen Pathogene wehrt  Bayerische Akademie
  der Wissenschaften'
type: book_chapter
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 43
year: '2014'
...
---
_id: '1889'
abstract:
- lang: eng
  text: We study translation-invariant quasi-free states for a system of fermions
    with two-particle interactions. The associated energy functional is similar to
    the BCS functional but also includes direct and exchange energies. We show that
    for suitable short-range interactions, these latter terms only lead to a renormalization
    of the chemical potential, with the usual properties of the BCS functional left
    unchanged. Our analysis thus represents a rigorous justification of part of the
    BCS approximation. We give bounds on the critical temperature below which the
    system displays superfluidity.
acknowledgement: We would like to thank Max Lein and Andreas Deuchert for valuable
  suggestions and remarks. Partial financial support by the NSERC (R.S.) is gratefully
  acknowledged.
article_number: '1450012'
article_processing_charge: No
article_type: original
arxiv: 1
author:
- first_name: Gerhard
  full_name: Bräunlich, Gerhard
  last_name: Bräunlich
- first_name: Christian
  full_name: Hainzl, Christian
  last_name: Hainzl
- first_name: Robert
  full_name: Seiringer, Robert
  id: 4AFD0470-F248-11E8-B48F-1D18A9856A87
  last_name: Seiringer
  orcid: 0000-0002-6781-0521
citation:
  ama: Bräunlich G, Hainzl C, Seiringer R. Translation-invariant quasi-free states
    for fermionic systems and the BCS approximation. <i>Reviews in Mathematical Physics</i>.
    2014;26(7). doi:<a href="https://doi.org/10.1142/S0129055X14500123">10.1142/S0129055X14500123</a>
  apa: Bräunlich, G., Hainzl, C., &#38; Seiringer, R. (2014). Translation-invariant
    quasi-free states for fermionic systems and the BCS approximation. <i>Reviews
    in Mathematical Physics</i>. World Scientific Publishing. <a href="https://doi.org/10.1142/S0129055X14500123">https://doi.org/10.1142/S0129055X14500123</a>
  chicago: Bräunlich, Gerhard, Christian Hainzl, and Robert Seiringer. “Translation-Invariant
    Quasi-Free States for Fermionic Systems and the BCS Approximation.” <i>Reviews
    in Mathematical Physics</i>. World Scientific Publishing, 2014. <a href="https://doi.org/10.1142/S0129055X14500123">https://doi.org/10.1142/S0129055X14500123</a>.
  ieee: G. Bräunlich, C. Hainzl, and R. Seiringer, “Translation-invariant quasi-free
    states for fermionic systems and the BCS approximation,” <i>Reviews in Mathematical
    Physics</i>, vol. 26, no. 7. World Scientific Publishing, 2014.
  ista: Bräunlich G, Hainzl C, Seiringer R. 2014. Translation-invariant quasi-free
    states for fermionic systems and the BCS approximation. Reviews in Mathematical
    Physics. 26(7), 1450012.
  mla: Bräunlich, Gerhard, et al. “Translation-Invariant Quasi-Free States for Fermionic
    Systems and the BCS Approximation.” <i>Reviews in Mathematical Physics</i>, vol.
    26, no. 7, 1450012, World Scientific Publishing, 2014, doi:<a href="https://doi.org/10.1142/S0129055X14500123">10.1142/S0129055X14500123</a>.
  short: G. Bräunlich, C. Hainzl, R. Seiringer, Reviews in Mathematical Physics 26
    (2014).
date_created: 2018-12-11T11:54:33Z
date_published: 2014-08-01T00:00:00Z
date_updated: 2022-06-07T09:03:09Z
day: '01'
department:
- _id: RoSe
doi: 10.1142/S0129055X14500123
external_id:
  arxiv:
  - '1305.5135'
intvolume: '        26'
issue: '7'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: http://arxiv.org/abs/1305.5135
month: '08'
oa: 1
oa_version: Submitted Version
publication: Reviews in Mathematical Physics
publication_status: published
publisher: World Scientific Publishing
publist_id: '5206'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Translation-invariant quasi-free states for fermionic systems and the BCS approximation
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 26
year: '2014'
...
---
_id: '1890'
abstract:
- lang: eng
  text: To search for a target in a complex environment is an everyday behavior that
    ends with finding the target. When we search for two identical targets, however,
    we must continue the search after finding the first target and memorize its location.
    We used fixation-related potentials to investigate the neural correlates of different
    stages of the search, that is, before and after finding the first target. Having
    found the first target influenced subsequent distractor processing. Compared to
    distractor fixations before the first target fixation, a negative shift was observed
    for three subsequent distractor fixations. These results suggest that processing
    a target in continued search modulates the brain's response, either transiently
    by reflecting temporary working memory processes or permanently by reflecting
    working memory retention.
acknowledgement: 'Funded by Austrian Science Fund (FWF) Grant Number: P 22189-B18;
  European Union within the 6th Framework Programme Grant Number: 517590; State government
  of Styria Grant Number: PN 4055'
author:
- first_name: Christof
  full_name: Körner, Christof
  last_name: Körner
- first_name: Verena
  full_name: Braunstein, Verena
  last_name: Braunstein
- first_name: Matthias
  full_name: Stangl, Matthias
  last_name: Stangl
- first_name: Alois
  full_name: Schlögl, Alois
  id: 45BF87EE-F248-11E8-B48F-1D18A9856A87
  last_name: Schlögl
  orcid: 0000-0002-5621-8100
- first_name: Christa
  full_name: Neuper, Christa
  last_name: Neuper
- first_name: Anja
  full_name: Ischebeck, Anja
  last_name: Ischebeck
citation:
  ama: 'Körner C, Braunstein V, Stangl M, Schlögl A, Neuper C, Ischebeck A. Sequential
    effects in continued visual search: Using fixation-related potentials to compare
    distractor processing before and after target detection. <i>Psychophysiology</i>.
    2014;51(4):385-395. doi:<a href="https://doi.org/10.1111/psyp.12062">10.1111/psyp.12062</a>'
  apa: 'Körner, C., Braunstein, V., Stangl, M., Schlögl, A., Neuper, C., &#38; Ischebeck,
    A. (2014). Sequential effects in continued visual search: Using fixation-related
    potentials to compare distractor processing before and after target detection.
    <i>Psychophysiology</i>. Wiley-Blackwell. <a href="https://doi.org/10.1111/psyp.12062">https://doi.org/10.1111/psyp.12062</a>'
  chicago: 'Körner, Christof, Verena Braunstein, Matthias Stangl, Alois Schlögl, Christa
    Neuper, and Anja Ischebeck. “Sequential Effects in Continued Visual Search: Using
    Fixation-Related Potentials to Compare Distractor Processing before and after
    Target Detection.” <i>Psychophysiology</i>. Wiley-Blackwell, 2014. <a href="https://doi.org/10.1111/psyp.12062">https://doi.org/10.1111/psyp.12062</a>.'
  ieee: 'C. Körner, V. Braunstein, M. Stangl, A. Schlögl, C. Neuper, and A. Ischebeck,
    “Sequential effects in continued visual search: Using fixation-related potentials
    to compare distractor processing before and after target detection,” <i>Psychophysiology</i>,
    vol. 51, no. 4. Wiley-Blackwell, pp. 385–395, 2014.'
  ista: 'Körner C, Braunstein V, Stangl M, Schlögl A, Neuper C, Ischebeck A. 2014.
    Sequential effects in continued visual search: Using fixation-related potentials
    to compare distractor processing before and after target detection. Psychophysiology.
    51(4), 385–395.'
  mla: 'Körner, Christof, et al. “Sequential Effects in Continued Visual Search: Using
    Fixation-Related Potentials to Compare Distractor Processing before and after
    Target Detection.” <i>Psychophysiology</i>, vol. 51, no. 4, Wiley-Blackwell, 2014,
    pp. 385–95, doi:<a href="https://doi.org/10.1111/psyp.12062">10.1111/psyp.12062</a>.'
  short: C. Körner, V. Braunstein, M. Stangl, A. Schlögl, C. Neuper, A. Ischebeck,
    Psychophysiology 51 (2014) 385–395.
date_created: 2018-12-11T11:54:34Z
date_published: 2014-02-11T00:00:00Z
date_updated: 2021-01-12T06:53:52Z
day: '11'
ddc:
- '000'
department:
- _id: ScienComp
- _id: PeJo
doi: 10.1111/psyp.12062
file:
- access_level: open_access
  checksum: 4255b6185e774acce1d99f8e195c564d
  content_type: application/pdf
  creator: system
  date_created: 2018-12-12T10:16:44Z
  date_updated: 2020-07-14T12:45:20Z
  file_id: '5233'
  file_name: IST-2016-442-v1+1_K-rner_et_al-2014-Psychophysiology.pdf
  file_size: 543243
  relation: main_file
file_date_updated: 2020-07-14T12:45:20Z
has_accepted_license: '1'
intvolume: '        51'
issue: '4'
language:
- iso: eng
license: https://creativecommons.org/licenses/by/4.0/
month: '02'
oa: 1
oa_version: Published Version
page: 385 - 395
publication: Psychophysiology
publication_status: published
publisher: Wiley-Blackwell
publist_id: '5205'
pubrep_id: '442'
scopus_import: 1
status: public
title: 'Sequential effects in continued visual search: Using fixation-related potentials
  to compare distractor processing before and after target detection'
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 51
year: '2014'
...
---
_id: '1891'
abstract:
- lang: eng
  text: We provide theoretical tests of a novel experimental technique to determine
    mechanostability of proteins based on stretching a mechanically protected protein
    by single-molecule force spectroscopy. This technique involves stretching a homogeneous
    or heterogeneous chain of reference proteins (single-molecule markers) in which
    one of them acts as host to the guest protein under study. The guest protein is
    grafted into the host through genetic engineering. It is expected that unraveling
    of the host precedes the unraveling of the guest removing ambiguities in the reading
    of the force-extension patterns of the guest protein. We study examples of such
    systems within a coarse-grained structure-based model. We consider systems with
    various ratios of mechanostability for the host and guest molecules and compare
    them to experimental results involving cohesin I as the guest molecule. For a
    comparison, we also study the force-displacement patterns in proteins that are
    linked in a serial fashion. We find that the mechanostability of the guest is
    similar to that of the isolated or serially linked protein. We also demonstrate
    that the ideal configuration of this strategy would be one in which the host is
    much more mechanostable than the single-molecule markers. We finally show that
    it is troublesome to use the highly stable cystine knot proteins as a host to
    graft a guest in stretching studies because this would involve a cleaving procedure.
acknowledgement: Grant Nr. 2011/01/N/ST3/02475
author:
- first_name: Mateusz
  full_name: Chwastyk, Mateusz
  last_name: Chwastyk
- first_name: Albert
  full_name: Galera Prat, Albert
  last_name: Galera Prat
- first_name: Mateusz K
  full_name: Sikora, Mateusz K
  id: 2F74BCDE-F248-11E8-B48F-1D18A9856A87
  last_name: Sikora
- first_name: Àngel
  full_name: Gómez Sicilia, Àngel
  last_name: Gómez Sicilia
- first_name: Mariano
  full_name: Carrión Vázquez, Mariano
  last_name: Carrión Vázquez
- first_name: Marek
  full_name: Cieplak, Marek
  last_name: Cieplak
citation:
  ama: 'Chwastyk M, Galera Prat A, Sikora MK, Gómez Sicilia À, Carrión Vázquez M,
    Cieplak M. Theoretical tests of the mechanical protection strategy in protein
    nanomechanics. <i>Proteins: Structure, Function and Bioinformatics</i>. 2014;82(5):717-726.
    doi:<a href="https://doi.org/10.1002/prot.24436">10.1002/prot.24436</a>'
  apa: 'Chwastyk, M., Galera Prat, A., Sikora, M. K., Gómez Sicilia, À., Carrión Vázquez,
    M., &#38; Cieplak, M. (2014). Theoretical tests of the mechanical protection strategy
    in protein nanomechanics. <i>Proteins: Structure, Function and Bioinformatics</i>.
    Wiley-Blackwell. <a href="https://doi.org/10.1002/prot.24436">https://doi.org/10.1002/prot.24436</a>'
  chicago: 'Chwastyk, Mateusz, Albert Galera Prat, Mateusz K Sikora, Àngel Gómez Sicilia,
    Mariano Carrión Vázquez, and Marek Cieplak. “Theoretical Tests of the Mechanical
    Protection Strategy in Protein Nanomechanics.” <i>Proteins: Structure, Function
    and Bioinformatics</i>. Wiley-Blackwell, 2014. <a href="https://doi.org/10.1002/prot.24436">https://doi.org/10.1002/prot.24436</a>.'
  ieee: 'M. Chwastyk, A. Galera Prat, M. K. Sikora, À. Gómez Sicilia, M. Carrión Vázquez,
    and M. Cieplak, “Theoretical tests of the mechanical protection strategy in protein
    nanomechanics,” <i>Proteins: Structure, Function and Bioinformatics</i>, vol.
    82, no. 5. Wiley-Blackwell, pp. 717–726, 2014.'
  ista: 'Chwastyk M, Galera Prat A, Sikora MK, Gómez Sicilia À, Carrión Vázquez M,
    Cieplak M. 2014. Theoretical tests of the mechanical protection strategy in protein
    nanomechanics. Proteins: Structure, Function and Bioinformatics. 82(5), 717–726.'
  mla: 'Chwastyk, Mateusz, et al. “Theoretical Tests of the Mechanical Protection
    Strategy in Protein Nanomechanics.” <i>Proteins: Structure, Function and Bioinformatics</i>,
    vol. 82, no. 5, Wiley-Blackwell, 2014, pp. 717–26, doi:<a href="https://doi.org/10.1002/prot.24436">10.1002/prot.24436</a>.'
  short: 'M. Chwastyk, A. Galera Prat, M.K. Sikora, À. Gómez Sicilia, M. Carrión Vázquez,
    M. Cieplak, Proteins: Structure, Function and Bioinformatics 82 (2014) 717–726.'
date_created: 2018-12-11T11:54:34Z
date_published: 2014-05-01T00:00:00Z
date_updated: 2021-01-12T06:53:52Z
day: '01'
department:
- _id: CaHe
doi: 10.1002/prot.24436
intvolume: '        82'
issue: '5'
language:
- iso: eng
month: '05'
oa_version: None
page: 717 - 726
publication: 'Proteins: Structure, Function and Bioinformatics'
publication_status: published
publisher: Wiley-Blackwell
publist_id: '5204'
scopus_import: 1
status: public
title: Theoretical tests of the mechanical protection strategy in protein nanomechanics
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 82
year: '2014'
...
---
_id: '1892'
abstract:
- lang: eng
  text: Behavioural variation among conspecifics is typically contingent on individual
    state or environmental conditions. Sex-specific genetic polymorphisms are enigmatic
    because they lack conditionality, and genes causing adaptive trait variation in
    one sex may reduce Darwinian fitness in the other. One way to avoid such genetic
    antagonism is to control sex-specific traits by inheritance via sex chromosomes.
    Here, controlled laboratory crossings suggest that in snail-brooding cichlid fish
    a single locus, two-allele polymorphism located on a sex-linked chromosome of
    heterogametic males generates an extreme reproductive dimorphism. Both natural
    and sexual selection are responsible for exceptionally large body size of bourgeois
    males, creating a niche for a miniature male phenotype to evolve. This extreme
    intrasexual dimorphism results from selection on opposite size thresholds caused
    by a single ecological factor, empty snail shells used as breeding substrate.
    Paternity analyses reveal that in the field parasitic dwarf males sire the majority
    of offspring in direct sperm competition with large nest owners exceeding their
    size more than 40 times. Apparently, use of empty snail shells as breeding substrate
    and single locus sex-linked inheritance of growth are the major ecological and
    genetic mechanisms responsible for the extreme intrasexual diversity observed
    in Lamprologus callipterus.
acknowledgement: "This research was supported by grants of the Swiss National Science
  Foundation to M.T.\r\nWe thank Tetsu Sato for providing field samples, Olivier Goffinet
  for field assistance, Dolores Schütz for vital help in the field and with the manuscript,
  David Lank, Barbara Taborsky, Suzanne Alonzo and two anonymous referees for comments
  on earlier manuscript versions, and the Fisheries Department, Ministry of Agriculture
  and Livestock of Zambia, for permission and support."
article_number: '20140253'
article_processing_charge: No
article_type: original
author:
- first_name: Sabine
  full_name: Ocana, Sabine
  last_name: Ocana
- first_name: Patrick
  full_name: Meidl, Patrick
  id: 4709BCE6-F248-11E8-B48F-1D18A9856A87
  last_name: Meidl
- first_name: Danielle
  full_name: Bonfils, Danielle
  last_name: Bonfils
- first_name: Michael
  full_name: Taborsky, Michael
  last_name: Taborsky
citation:
  ama: Ocana S, Meidl P, Bonfils D, Taborsky M. Y-linked Mendelian inheritance of
    giant and dwarf male morphs in shell-brooding cichlids. <i>Proceedings of the
    Royal Society of London Series B Biological Sciences</i>. 2014;281(1794). doi:<a
    href="https://doi.org/10.1098/rspb.2014.0253">10.1098/rspb.2014.0253</a>
  apa: Ocana, S., Meidl, P., Bonfils, D., &#38; Taborsky, M. (2014). Y-linked Mendelian
    inheritance of giant and dwarf male morphs in shell-brooding cichlids. <i>Proceedings
    of the Royal Society of London Series B Biological Sciences</i>. The Royal Society.
    <a href="https://doi.org/10.1098/rspb.2014.0253">https://doi.org/10.1098/rspb.2014.0253</a>
  chicago: Ocana, Sabine, Patrick Meidl, Danielle Bonfils, and Michael Taborsky. “Y-Linked
    Mendelian Inheritance of Giant and Dwarf Male Morphs in Shell-Brooding Cichlids.”
    <i>Proceedings of the Royal Society of London Series B Biological Sciences</i>.
    The Royal Society, 2014. <a href="https://doi.org/10.1098/rspb.2014.0253">https://doi.org/10.1098/rspb.2014.0253</a>.
  ieee: S. Ocana, P. Meidl, D. Bonfils, and M. Taborsky, “Y-linked Mendelian inheritance
    of giant and dwarf male morphs in shell-brooding cichlids,” <i>Proceedings of
    the Royal Society of London Series B Biological Sciences</i>, vol. 281, no. 1794.
    The Royal Society, 2014.
  ista: Ocana S, Meidl P, Bonfils D, Taborsky M. 2014. Y-linked Mendelian inheritance
    of giant and dwarf male morphs in shell-brooding cichlids. Proceedings of the
    Royal Society of London Series B Biological Sciences. 281(1794), 20140253.
  mla: Ocana, Sabine, et al. “Y-Linked Mendelian Inheritance of Giant and Dwarf Male
    Morphs in Shell-Brooding Cichlids.” <i>Proceedings of the Royal Society of London
    Series B Biological Sciences</i>, vol. 281, no. 1794, 20140253, The Royal Society,
    2014, doi:<a href="https://doi.org/10.1098/rspb.2014.0253">10.1098/rspb.2014.0253</a>.
  short: S. Ocana, P. Meidl, D. Bonfils, M. Taborsky, Proceedings of the Royal Society
    of London Series B Biological Sciences 281 (2014).
date_created: 2018-12-11T11:54:34Z
date_published: 2014-11-07T00:00:00Z
date_updated: 2022-06-07T09:12:32Z
day: '07'
department:
- _id: CampIT
doi: 10.1098/rspb.2014.0253
external_id:
  pmid:
  - '25232141'
intvolume: '       281'
issue: '1794'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4211437/
month: '11'
oa: 1
oa_version: Submitted Version
pmid: 1
publication: Proceedings of the Royal Society of London Series B Biological Sciences
publication_status: published
publisher: The Royal Society
publist_id: '5203'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Y-linked Mendelian inheritance of giant and dwarf male morphs in shell-brooding
  cichlids
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 281
year: '2014'
...
---
_id: '1893'
abstract:
- lang: eng
  text: Phosphatidylinositol (PtdIns) is a structural phospholipid that can be phosphorylated
    into various lipid signaling molecules, designated polyphosphoinositides (PPIs).
    The reversible phosphorylation of PPIs on the 3, 4, or 5 position of inositol
    is performed by a set of organelle-specific kinases and phosphatases, and the
    characteristic head groups make these molecules ideal for regulating biological
    processes in time and space. In yeast and mammals, PtdIns3P and PtdIns(3,5)P2
    play crucial roles in trafficking toward the lytic compartments, whereas the role
    in plants is not yet fully understood. Here we identified the role of a land plant-specific
    subgroup of PPI phosphatases, the suppressor of actin 2 (SAC2) to SAC5, during
    vacuolar trafficking and morphogenesis in Arabidopsis thaliana. SAC2-SAC5 localize
    to the tonoplast along with PtdIns3P, the presumable product of their activity.
    In SAC gain- and loss-of-function mutants, the levels of PtdIns monophosphates
    and bisphosphates were changed, with opposite effects on the morphology of storage
    and lytic vacuoles, and the trafficking toward the vacuoles was defective. Moreover,
    multiple sac knockout mutants had an increased number of smaller storage and lytic
    vacuoles, whereas extralarge vacuoles were observed in the overexpression lines,
    correlating with various growth and developmental defects. The fragmented vacuolar
    phenotype of sac mutants could be mimicked by treating wild-type seedlings with
    PtdIns(3,5)P2, corroborating that this PPI is important for vacuole morphology.
    Taken together, these results provide evidence that PPIs, together with their
    metabolic enzymes SAC2-SAC5, are crucial for vacuolar trafficking and for vacuolar
    morphology and function in plants.
acknowledgement: This work was supported by grants from the Research Foundation-Flanders
  (Odysseus).
author:
- first_name: Petra
  full_name: Nováková, Petra
  id: 44E59624-F248-11E8-B48F-1D18A9856A87
  last_name: Nováková
- first_name: Sibylle
  full_name: Hirsch, Sibylle
  last_name: Hirsch
- first_name: Elena
  full_name: Feraru, Elena
  last_name: Feraru
- first_name: Ricardo
  full_name: Tejos, Ricardo
  last_name: Tejos
- first_name: Ringo
  full_name: Van Wijk, Ringo
  last_name: Van Wijk
- first_name: Tom
  full_name: Viaene, Tom
  last_name: Viaene
- first_name: Mareike
  full_name: Heilmann, Mareike
  last_name: Heilmann
- first_name: Jennifer
  full_name: Lerche, Jennifer
  last_name: Lerche
- first_name: Riet
  full_name: De Rycke, Riet
  last_name: De Rycke
- first_name: Mugurel
  full_name: Feraru, Mugurel
  last_name: Feraru
- first_name: Peter
  full_name: Grones, Peter
  id: 399876EC-F248-11E8-B48F-1D18A9856A87
  last_name: Grones
- first_name: Marc
  full_name: Van Montagu, Marc
  last_name: Van Montagu
- first_name: Ingo
  full_name: Heilmann, Ingo
  last_name: Heilmann
- first_name: Teun
  full_name: Munnik, Teun
  last_name: Munnik
- first_name: Jirí
  full_name: Friml, Jirí
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
citation:
  ama: Marhavá P, Hirsch S, Feraru E, et al. SAC phosphoinositide phosphatases at
    the tonoplast mediate vacuolar function in Arabidopsis. <i>PNAS</i>. 2014;111(7):2818-2823.
    doi:<a href="https://doi.org/10.1073/pnas.1324264111">10.1073/pnas.1324264111</a>
  apa: Marhavá, P., Hirsch, S., Feraru, E., Tejos, R., Van Wijk, R., Viaene, T., …
    Friml, J. (2014). SAC phosphoinositide phosphatases at the tonoplast mediate vacuolar
    function in Arabidopsis. <i>PNAS</i>. National Academy of Sciences. <a href="https://doi.org/10.1073/pnas.1324264111">https://doi.org/10.1073/pnas.1324264111</a>
  chicago: Marhavá, Petra, Sibylle Hirsch, Elena Feraru, Ricardo Tejos, Ringo Van
    Wijk, Tom Viaene, Mareike Heilmann, et al. “SAC Phosphoinositide Phosphatases
    at the Tonoplast Mediate Vacuolar Function in Arabidopsis.” <i>PNAS</i>. National
    Academy of Sciences, 2014. <a href="https://doi.org/10.1073/pnas.1324264111">https://doi.org/10.1073/pnas.1324264111</a>.
  ieee: P. Marhavá <i>et al.</i>, “SAC phosphoinositide phosphatases at the tonoplast
    mediate vacuolar function in Arabidopsis,” <i>PNAS</i>, vol. 111, no. 7. National
    Academy of Sciences, pp. 2818–2823, 2014.
  ista: Marhavá P, Hirsch S, Feraru E, Tejos R, Van Wijk R, Viaene T, Heilmann M,
    Lerche J, De Rycke R, Feraru M, Grones P, Van Montagu M, Heilmann I, Munnik T,
    Friml J. 2014. SAC phosphoinositide phosphatases at the tonoplast mediate vacuolar
    function in Arabidopsis. PNAS. 111(7), 2818–2823.
  mla: Marhavá, Petra, et al. “SAC Phosphoinositide Phosphatases at the Tonoplast
    Mediate Vacuolar Function in Arabidopsis.” <i>PNAS</i>, vol. 111, no. 7, National
    Academy of Sciences, 2014, pp. 2818–23, doi:<a href="https://doi.org/10.1073/pnas.1324264111">10.1073/pnas.1324264111</a>.
  short: P. Marhavá, S. Hirsch, E. Feraru, R. Tejos, R. Van Wijk, T. Viaene, M. Heilmann,
    J. Lerche, R. De Rycke, M. Feraru, P. Grones, M. Van Montagu, I. Heilmann, T.
    Munnik, J. Friml, PNAS 111 (2014) 2818–2823.
date_created: 2018-12-11T11:54:34Z
date_published: 2014-02-18T00:00:00Z
date_updated: 2021-01-12T06:53:53Z
day: '18'
department:
- _id: JiFr
doi: 10.1073/pnas.1324264111
ec_funded: 1
intvolume: '       111'
issue: '7'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3932866/
month: '02'
oa: 1
oa_version: Submitted Version
page: 2818 - 2823
project:
- _id: 25716A02-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '282300'
  name: Polarity and subcellular dynamics in plants
publication: PNAS
publication_status: published
publisher: National Academy of Sciences
publist_id: '5202'
scopus_import: 1
status: public
title: SAC phosphoinositide phosphatases at the tonoplast mediate vacuolar function
  in Arabidopsis
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 111
year: '2014'
...
---
_id: '1894'
abstract:
- lang: eng
  text: 'Background: Bacterial Dsb enzymes are involved in the oxidative folding of
    many proteins, through the formation of disulfide bonds between their cysteine
    residues. The Dsb protein network has been well characterized in cells of the
    model microorganism Escherichia coli. To gain insight into the functioning of
    the Dsb system in epsilon-Proteobacteria, where it plays an important role in
    the colonization process, we studied two homologs of the main Escherichia coli
    Dsb oxidase (EcDsbA) that are present in the cells of the enteric pathogen Campylobacter
    jejuni, the most frequently reported bacterial cause of human enteritis in the
    world. Methods and Results: Phylogenetic analysis suggests the horizontal transfer
    of the epsilon-Proteobacterial DsbAs from a common ancestor to gamma-Proteobacteria,
    which then gave rise to the DsbL lineage. Phenotype and enzymatic assays suggest
    that the two C. jejuni DsbAs play different roles in bacterial cells and have
    divergent substrate spectra. CjDsbA1 is essential for the motility and autoagglutination
    phenotypes, while CjDsbA2 has no impact on those processes. CjDsbA1 plays a critical
    role in the oxidative folding that ensures the activity of alkaline phosphatase
    CjPhoX, whereas CjDsbA2 is crucial for the activity of arylsulfotransferase CjAstA,
    encoded within the dsbA2-dsbB-astA operon. Conclusions: Our results show that
    CjDsbA1 is the primary thiol-oxidoreductase affecting life processes associated
    with bacterial spread and host colonization, as well as ensuring the oxidative
    folding of particular protein substrates. In contrast, CjDsbA2 activity does not
    affect the same processes and so far its oxidative folding activity has been demonstrated
    for one substrate, arylsulfotransferase CjAstA. The results suggest the cooperation
    between CjDsbA2 and CjDsbB. In the case of the CjDsbA1, this cooperation is not
    exclusive and there is probably another protein to be identified in C. jejuni
    cells that acts to re-oxidize CjDsbA1. Altogether the data presented here constitute
    the considerable insight to the Epsilonproteobacterial Dsb systems, which have
    been poorly understood so far.'
article_number: e106247
author:
- first_name: Anna
  full_name: Grabowska, Anna
  last_name: Grabowska
- first_name: Ewa
  full_name: Wywiał, Ewa
  last_name: Wywiał
- first_name: Stanislaw
  full_name: Dunin Horkawicz, Stanislaw
  last_name: Dunin Horkawicz
- first_name: Anna
  full_name: Łasica, Anna
  last_name: Łasica
- first_name: Marc
  full_name: Wösten, Marc
  last_name: Wösten
- first_name: Anna A
  full_name: Nagy-Staron, Anna A
  id: 3ABC5BA6-F248-11E8-B48F-1D18A9856A87
  last_name: Nagy-Staron
- first_name: Renata
  full_name: Godlewska, Renata
  last_name: Godlewska
- first_name: Katarzyna
  full_name: Bocian Ostrzycka, Katarzyna
  last_name: Bocian Ostrzycka
- first_name: Katarzyna
  full_name: Pieńkowska, Katarzyna
  last_name: Pieńkowska
- first_name: Paweł
  full_name: Łaniewski, Paweł
  last_name: Łaniewski
- first_name: Janusz
  full_name: Bujnicki, Janusz
  last_name: Bujnicki
- first_name: Jos
  full_name: Van Putten, Jos
  last_name: Van Putten
- first_name: Elzbieta
  full_name: Jagusztyn Krynicka, Elzbieta
  last_name: Jagusztyn Krynicka
citation:
  ama: Grabowska A, Wywiał E, Dunin Horkawicz S, et al. Functional and bioinformatics
    analysis of two Campylobacter jejuni homologs of the thiol-disulfide oxidoreductase,
    DsbA. <i>PLoS One</i>. 2014;9(9). doi:<a href="https://doi.org/10.1371/journal.pone.0106247">10.1371/journal.pone.0106247</a>
  apa: Grabowska, A., Wywiał, E., Dunin Horkawicz, S., Łasica, A., Wösten, M., Nagy-Staron,
    A. A., … Jagusztyn Krynicka, E. (2014). Functional and bioinformatics analysis
    of two Campylobacter jejuni homologs of the thiol-disulfide oxidoreductase, DsbA.
    <i>PLoS One</i>. Public Library of Science. <a href="https://doi.org/10.1371/journal.pone.0106247">https://doi.org/10.1371/journal.pone.0106247</a>
  chicago: Grabowska, Anna, Ewa Wywiał, Stanislaw Dunin Horkawicz, Anna Łasica, Marc
    Wösten, Anna A Nagy-Staron, Renata Godlewska, et al. “Functional and Bioinformatics
    Analysis of Two Campylobacter Jejuni Homologs of the Thiol-Disulfide Oxidoreductase,
    DsbA.” <i>PLoS One</i>. Public Library of Science, 2014. <a href="https://doi.org/10.1371/journal.pone.0106247">https://doi.org/10.1371/journal.pone.0106247</a>.
  ieee: A. Grabowska <i>et al.</i>, “Functional and bioinformatics analysis of two
    Campylobacter jejuni homologs of the thiol-disulfide oxidoreductase, DsbA,” <i>PLoS
    One</i>, vol. 9, no. 9. Public Library of Science, 2014.
  ista: Grabowska A, Wywiał E, Dunin Horkawicz S, Łasica A, Wösten M, Nagy-Staron
    AA, Godlewska R, Bocian Ostrzycka K, Pieńkowska K, Łaniewski P, Bujnicki J, Van
    Putten J, Jagusztyn Krynicka E. 2014. Functional and bioinformatics analysis of
    two Campylobacter jejuni homologs of the thiol-disulfide oxidoreductase, DsbA.
    PLoS One. 9(9), e106247.
  mla: Grabowska, Anna, et al. “Functional and Bioinformatics Analysis of Two Campylobacter
    Jejuni Homologs of the Thiol-Disulfide Oxidoreductase, DsbA.” <i>PLoS One</i>,
    vol. 9, no. 9, e106247, Public Library of Science, 2014, doi:<a href="https://doi.org/10.1371/journal.pone.0106247">10.1371/journal.pone.0106247</a>.
  short: A. Grabowska, E. Wywiał, S. Dunin Horkawicz, A. Łasica, M. Wösten, A.A. Nagy-Staron,
    R. Godlewska, K. Bocian Ostrzycka, K. Pieńkowska, P. Łaniewski, J. Bujnicki, J.
    Van Putten, E. Jagusztyn Krynicka, PLoS One 9 (2014).
date_created: 2018-12-11T11:54:35Z
date_published: 2014-09-02T00:00:00Z
date_updated: 2021-01-12T06:53:54Z
day: '02'
ddc:
- '570'
department:
- _id: CaGu
doi: 10.1371/journal.pone.0106247
file:
- access_level: open_access
  checksum: 7d02c3da7f72b82bb5d7932d80c3251f
  content_type: application/pdf
  creator: system
  date_created: 2018-12-12T10:16:19Z
  date_updated: 2020-07-14T12:45:20Z
  file_id: '5205'
  file_name: IST-2016-438-v1+1_journal.pone.0106247.pdf
  file_size: 4248801
  relation: main_file
file_date_updated: 2020-07-14T12:45:20Z
has_accepted_license: '1'
intvolume: '         9'
issue: '9'
language:
- iso: eng
month: '09'
oa: 1
oa_version: Published Version
publication: PLoS One
publication_status: published
publisher: Public Library of Science
publist_id: '5201'
pubrep_id: '438'
quality_controlled: '1'
scopus_import: 1
status: public
title: Functional and bioinformatics analysis of two Campylobacter jejuni homologs
  of the thiol-disulfide oxidoreductase, DsbA
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 9
year: '2014'
...
---
_id: '1895'
abstract:
- lang: eng
  text: Major histocompatibility complex class I (MHCI) molecules were recently identified
    as novel regulators of synaptic plasticity. These molecules are expressed in various
    brain areas, especially in regions undergoing activity-dependent synaptic plasticity,
    but their role in the nucleus accumbens (NAc) is unknown. In this study, we investigated
    the effects of genetic disruption of MHCI function, through deletion of β2-microblobulin,
    which causes lack of cell surface expression of MHCI. First, we confirmed that
    MHCI molecules are expressed in the NAc core in wild-type mice. Second, we performed
    electrophysiological recordings with NAc core slices from wild-type and β2-microglobulin
    knock-out mice lacking cell surface expression of MHCI. We found that low frequency
    stimulation induced long-term depression in wild-type but not knock-out mice,
    whereas high frequency stimulation induced long-term potentiation in both genotypes,
    with a larger magnitude in knock-out mice. Furthermore, we demonstrated that knock-out
    mice showed more persistent behavioral sensitization to cocaine, which is a NAc-related
    behavior. Using this model, we analyzed the density of total AMPA receptors and
    their subunits GluR1 and GluR2 in the NAc core, by SDS-digested freeze-fracture
    replica labeling. After repeated cocaine exposure, the density of GluR1 was increased,
    but there was no change in total AMPA receptors and GluR2 levels in wildtype mice.
    In contrast, following repeated cocaine exposure, increased densities of total
    AMPA receptors, GluR1 and GluR2 were observed in knock-out mice. These results
    indicate that functional deficiency of MHCI enhances synaptic potentiation, induced
    by electrical and pharmacological stimulation.
acknowledgement: This work was supported in part by a Grant-in-Aid for Scientific
  Research on Innovative Areas (Comprehensive Brain Science Network) and (B) 17330153,
  from the Ministry of Education, Culture, Sports, Science and Technology of Japan.
article_number: e107099
author:
- first_name: Mitsuhiro
  full_name: Edamura, Mitsuhiro
  last_name: Edamura
- first_name: Gen
  full_name: Murakami, Gen
  last_name: Murakami
- first_name: Hongrui
  full_name: Meng, Hongrui
  last_name: Meng
- first_name: Makoto
  full_name: Itakura, Makoto
  last_name: Itakura
- first_name: Ryuichi
  full_name: Shigemoto, Ryuichi
  id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
  last_name: Shigemoto
  orcid: 0000-0001-8761-9444
- first_name: Atsuo
  full_name: Fukuda, Atsuo
  last_name: Fukuda
- first_name: Daiichiro
  full_name: Nakahara, Daiichiro
  last_name: Nakahara
citation:
  ama: Edamura M, Murakami G, Meng H, et al. Functional deficiency of MHC class i
    enhances LTP and abolishes LTD in the nucleus accumbens of mice. <i>PLoS One</i>.
    2014;9(9). doi:<a href="https://doi.org/10.1371/journal.pone.0107099">10.1371/journal.pone.0107099</a>
  apa: Edamura, M., Murakami, G., Meng, H., Itakura, M., Shigemoto, R., Fukuda, A.,
    &#38; Nakahara, D. (2014). Functional deficiency of MHC class i enhances LTP and
    abolishes LTD in the nucleus accumbens of mice. <i>PLoS One</i>. Public Library
    of Science. <a href="https://doi.org/10.1371/journal.pone.0107099">https://doi.org/10.1371/journal.pone.0107099</a>
  chicago: Edamura, Mitsuhiro, Gen Murakami, Hongrui Meng, Makoto Itakura, Ryuichi
    Shigemoto, Atsuo Fukuda, and Daiichiro Nakahara. “Functional Deficiency of MHC
    Class i Enhances LTP and Abolishes LTD in the Nucleus Accumbens of Mice.” <i>PLoS
    One</i>. Public Library of Science, 2014. <a href="https://doi.org/10.1371/journal.pone.0107099">https://doi.org/10.1371/journal.pone.0107099</a>.
  ieee: M. Edamura <i>et al.</i>, “Functional deficiency of MHC class i enhances LTP
    and abolishes LTD in the nucleus accumbens of mice,” <i>PLoS One</i>, vol. 9,
    no. 9. Public Library of Science, 2014.
  ista: Edamura M, Murakami G, Meng H, Itakura M, Shigemoto R, Fukuda A, Nakahara
    D. 2014. Functional deficiency of MHC class i enhances LTP and abolishes LTD in
    the nucleus accumbens of mice. PLoS One. 9(9), e107099.
  mla: Edamura, Mitsuhiro, et al. “Functional Deficiency of MHC Class i Enhances LTP
    and Abolishes LTD in the Nucleus Accumbens of Mice.” <i>PLoS One</i>, vol. 9,
    no. 9, e107099, Public Library of Science, 2014, doi:<a href="https://doi.org/10.1371/journal.pone.0107099">10.1371/journal.pone.0107099</a>.
  short: M. Edamura, G. Murakami, H. Meng, M. Itakura, R. Shigemoto, A. Fukuda, D.
    Nakahara, PLoS One 9 (2014).
date_created: 2018-12-11T11:54:35Z
date_published: 2014-09-30T00:00:00Z
date_updated: 2021-01-12T06:53:54Z
day: '30'
ddc:
- '570'
department:
- _id: RySh
doi: 10.1371/journal.pone.0107099
file:
- access_level: open_access
  checksum: 1f3be936be93114596d61ba44cacee69
  content_type: application/pdf
  creator: system
  date_created: 2018-12-12T10:09:01Z
  date_updated: 2020-07-14T12:45:20Z
  file_id: '4724'
  file_name: IST-2016-439-v1+1_journal.pone.0107099.pdf
  file_size: 6262085
  relation: main_file
file_date_updated: 2020-07-14T12:45:20Z
has_accepted_license: '1'
intvolume: '         9'
issue: '9'
language:
- iso: eng
month: '09'
oa: 1
oa_version: Published Version
publication: PLoS One
publication_status: published
publisher: Public Library of Science
publist_id: '5200'
pubrep_id: '439'
scopus_import: 1
status: public
title: Functional deficiency of MHC class i enhances LTP and abolishes LTD in the
  nucleus accumbens of mice
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 9
year: '2014'
...
---
_id: '1896'
abstract:
- lang: eng
  text: 'Biopolymer length regulation is a complex process that involves a large number
    of biological, chemical, and physical subprocesses acting simultaneously across
    multiple spatial and temporal scales. An illustrative example important for genomic
    stability is the length regulation of telomeres - nucleoprotein structures at
    the ends of linear chromosomes consisting of tandemly repeated DNA sequences and
    a specialized set of proteins. Maintenance of telomeres is often facilitated by
    the enzyme telomerase but, particularly in telomerase-free systems, the maintenance
    of chromosomal termini depends on alternative lengthening of telomeres (ALT) mechanisms
    mediated by recombination. Various linear and circular DNA structures were identified
    to participate in ALT, however, dynamics of the whole process is still poorly
    understood. We propose a chemical kinetics model of ALT with kinetic rates systematically
    derived from the biophysics of DNA diffusion and looping. The reaction system
    is reduced to a coagulation-fragmentation system by quasi-steady-state approximation.
    The detailed treatment of kinetic rates yields explicit formulas for expected
    size distributions of telomeres that demonstrate the key role played by the J
    factor, a quantitative measure of bending of polymers. The results are in agreement
    with experimental data and point out interesting phenomena: an appearance of very
    long telomeric circles if the total telomere density exceeds a critical value
    (excess mass) and a nonlinear response of the telomere size distributions to the
    amount of telomeric DNA in the system. The results can be of general importance
    for understanding dynamics of telomeres in telomerase-independent systems as this
    mode of telomere maintenance is similar to the situation in tumor cells lacking
    telomerase activity. Furthermore, due to its universality, the model may also
    serve as a prototype of an interaction between linear and circular DNA structures
    in various settings.'
acknowledgement: The work was supported by the VEGA Grant No. 1/0459/13 (R.K. and
  K.B.).
article_number: '032701'
article_processing_charge: No
author:
- first_name: Richard
  full_name: Kollár, Richard
  last_name: Kollár
- first_name: Katarína
  full_name: Bod'ová, Katarína
  id: 2BA24EA0-F248-11E8-B48F-1D18A9856A87
  last_name: Bod'ová
  orcid: 0000-0002-7214-0171
- first_name: Jozef
  full_name: Nosek, Jozef
  last_name: Nosek
- first_name: Ľubomír
  full_name: Tomáška, Ľubomír
  last_name: Tomáška
citation:
  ama: Kollár R, Bodova K, Nosek J, Tomáška Ľ. Mathematical model of alternative mechanism
    of telomere length maintenance. <i>Physical Review E Statistical Nonlinear and
    Soft Matter Physics</i>. 2014;89(3). doi:<a href="https://doi.org/10.1103/PhysRevE.89.032701">10.1103/PhysRevE.89.032701</a>
  apa: Kollár, R., Bodova, K., Nosek, J., &#38; Tomáška, Ľ. (2014). Mathematical model
    of alternative mechanism of telomere length maintenance. <i>Physical Review E
    Statistical Nonlinear and Soft Matter Physics</i>. American Institute of Physics.
    <a href="https://doi.org/10.1103/PhysRevE.89.032701">https://doi.org/10.1103/PhysRevE.89.032701</a>
  chicago: Kollár, Richard, Katarina Bodova, Jozef Nosek, and Ľubomír Tomáška. “Mathematical
    Model of Alternative Mechanism of Telomere Length Maintenance.” <i>Physical Review
    E Statistical Nonlinear and Soft Matter Physics</i>. American Institute of Physics,
    2014. <a href="https://doi.org/10.1103/PhysRevE.89.032701">https://doi.org/10.1103/PhysRevE.89.032701</a>.
  ieee: R. Kollár, K. Bodova, J. Nosek, and Ľ. Tomáška, “Mathematical model of alternative
    mechanism of telomere length maintenance,” <i>Physical Review E Statistical Nonlinear
    and Soft Matter Physics</i>, vol. 89, no. 3. American Institute of Physics, 2014.
  ista: Kollár R, Bodova K, Nosek J, Tomáška Ľ. 2014. Mathematical model of alternative
    mechanism of telomere length maintenance. Physical Review E Statistical Nonlinear
    and Soft Matter Physics. 89(3), 032701.
  mla: Kollár, Richard, et al. “Mathematical Model of Alternative Mechanism of Telomere
    Length Maintenance.” <i>Physical Review E Statistical Nonlinear and Soft Matter
    Physics</i>, vol. 89, no. 3, 032701, American Institute of Physics, 2014, doi:<a
    href="https://doi.org/10.1103/PhysRevE.89.032701">10.1103/PhysRevE.89.032701</a>.
  short: R. Kollár, K. Bodova, J. Nosek, Ľ. Tomáška, Physical Review E Statistical
    Nonlinear and Soft Matter Physics 89 (2014).
date_created: 2018-12-11T11:54:35Z
date_published: 2014-03-04T00:00:00Z
date_updated: 2022-08-01T10:50:10Z
day: '04'
department:
- _id: NiBa
- _id: GaTk
doi: 10.1103/PhysRevE.89.032701
intvolume: '        89'
issue: '3'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: http://arxiv.org/abs/1402.0430
month: '03'
oa: 1
oa_version: Submitted Version
publication: Physical Review E Statistical Nonlinear and Soft Matter Physics
publication_status: published
publisher: American Institute of Physics
publist_id: '5198'
scopus_import: '1'
status: public
title: Mathematical model of alternative mechanism of telomere length maintenance
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 89
year: '2014'
...
---
_id: '1897'
abstract:
- lang: eng
  text: GNOM is one of the most characterized membrane trafficking regulators in plants,
    with crucial roles in development. GNOM encodes an ARF-guanine nucleotide exchange
    factor (ARF-GEF) that activates small GTPases of the ARF (ADP ribosylation factor)
    class to mediate vesicle budding at endomembranes. The crucial role of GNOM in
    recycling of PIN auxin transporters and other proteins to the plasma membrane
    was identified in studies using the ARF-GEF inhibitor brefeldin A (BFA). GNOM,
    the most prominent regulator of recycling in plants, has been proposed to act
    and localize at so far elusive recycling endosomes. Here, we report the GNOM localization
    in context of its cellular function in Arabidopsis thaliana. State-of-the-art
    imaging, pharmacological interference, and ultrastructure analysis show that GNOM
    predominantly localizes to Golgi apparatus. Super-resolution confocal live imaging
    microscopy identified GNOM and its closest homolog GNOM-like 1 at distinct subdomains
    on Golgi cisternae. Short-term BFA treatment stabilizes GNOM at the Golgi apparatus,
    whereas prolonged exposures results in GNOM translocation to trans-Golgi network
    (TGN)/early endosomes (EEs). Malformed TGN/EE in gnom mutants suggests a role
    for GNOM in maintaining TGN/EE function. Our results redefine the subcellular
    action of GNOM and reevaluate the identity and function of recycling endosomes
    in plants.
acknowledgement: This work was supported by the Odysseus Program of the Research Foundation-Flanders
  (J.F.).
author:
- first_name: Satoshi
  full_name: Naramoto, Satoshi
  last_name: Naramoto
- first_name: Marisa
  full_name: Otegui, Marisa
  last_name: Otegui
- first_name: Natsumaro
  full_name: Kutsuna, Natsumaro
  last_name: Kutsuna
- first_name: Riet
  full_name: De Rycke, Riet
  last_name: De Rycke
- first_name: Tomoko
  full_name: Dainobu, Tomoko
  last_name: Dainobu
- first_name: Michael
  full_name: Karampelias, Michael
  last_name: Karampelias
- first_name: Masaru
  full_name: Fujimoto, Masaru
  last_name: Fujimoto
- first_name: Elena
  full_name: Feraru, Elena
  last_name: Feraru
- first_name: Daisuke
  full_name: Miki, Daisuke
  last_name: Miki
- first_name: Hiroo
  full_name: Fukuda, Hiroo
  last_name: Fukuda
- first_name: Akihiko
  full_name: Nakano, Akihiko
  last_name: Nakano
- first_name: Jirí
  full_name: Friml, Jirí
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
citation:
  ama: Naramoto S, Otegui M, Kutsuna N, et al. Insights into the localization and
    function of the membrane trafficking regulator GNOM ARF-GEF at the Golgi apparatus
    in Arabidopsis. <i>Plant Cell</i>. 2014;26(7):3062-3076. doi:<a href="https://doi.org/10.1105/tpc.114.125880">10.1105/tpc.114.125880</a>
  apa: Naramoto, S., Otegui, M., Kutsuna, N., De Rycke, R., Dainobu, T., Karampelias,
    M., … Friml, J. (2014). Insights into the localization and function of the membrane
    trafficking regulator GNOM ARF-GEF at the Golgi apparatus in Arabidopsis. <i>Plant
    Cell</i>. American Society of Plant Biologists. <a href="https://doi.org/10.1105/tpc.114.125880">https://doi.org/10.1105/tpc.114.125880</a>
  chicago: Naramoto, Satoshi, Marisa Otegui, Natsumaro Kutsuna, Riet De Rycke, Tomoko
    Dainobu, Michael Karampelias, Masaru Fujimoto, et al. “Insights into the Localization
    and Function of the Membrane Trafficking Regulator GNOM ARF-GEF at the Golgi Apparatus
    in Arabidopsis.” <i>Plant Cell</i>. American Society of Plant Biologists, 2014.
    <a href="https://doi.org/10.1105/tpc.114.125880">https://doi.org/10.1105/tpc.114.125880</a>.
  ieee: S. Naramoto <i>et al.</i>, “Insights into the localization and function of
    the membrane trafficking regulator GNOM ARF-GEF at the Golgi apparatus in Arabidopsis,”
    <i>Plant Cell</i>, vol. 26, no. 7. American Society of Plant Biologists, pp. 3062–3076,
    2014.
  ista: Naramoto S, Otegui M, Kutsuna N, De Rycke R, Dainobu T, Karampelias M, Fujimoto
    M, Feraru E, Miki D, Fukuda H, Nakano A, Friml J. 2014. Insights into the localization
    and function of the membrane trafficking regulator GNOM ARF-GEF at the Golgi apparatus
    in Arabidopsis. Plant Cell. 26(7), 3062–3076.
  mla: Naramoto, Satoshi, et al. “Insights into the Localization and Function of the
    Membrane Trafficking Regulator GNOM ARF-GEF at the Golgi Apparatus in Arabidopsis.”
    <i>Plant Cell</i>, vol. 26, no. 7, American Society of Plant Biologists, 2014,
    pp. 3062–76, doi:<a href="https://doi.org/10.1105/tpc.114.125880">10.1105/tpc.114.125880</a>.
  short: S. Naramoto, M. Otegui, N. Kutsuna, R. De Rycke, T. Dainobu, M. Karampelias,
    M. Fujimoto, E. Feraru, D. Miki, H. Fukuda, A. Nakano, J. Friml, Plant Cell 26
    (2014) 3062–3076.
date_created: 2018-12-11T11:54:36Z
date_published: 2014-07-01T00:00:00Z
date_updated: 2021-01-12T06:53:55Z
day: '01'
department:
- _id: JiFr
doi: 10.1105/tpc.114.125880
intvolume: '        26'
issue: '7'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4145132/
month: '07'
oa: 1
oa_version: Submitted Version
page: 3062 - 3076
publication: Plant Cell
publication_status: published
publisher: American Society of Plant Biologists
publist_id: '5199'
scopus_import: 1
status: public
title: Insights into the localization and function of the membrane trafficking regulator
  GNOM ARF-GEF at the Golgi apparatus in Arabidopsis
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 26
year: '2014'
...
---
_id: '1898'
abstract:
- lang: eng
  text: Fast synaptic transmission is important for rapid information processing.
    To explore the maximal rate of neuronal signaling and to analyze the presynaptic
    mechanisms, we focused on the input layer of the cerebellar cortex, where exceptionally
    high action potential (AP) frequencies have been reported invivo. With paired
    recordings between presynaptic cerebellar mossy fiber boutons and postsynaptic
    granule cells, we demonstrate reliable neurotransmission upto ~1 kHz. Presynaptic
    APs are ultrafast, with ~100μs half-duration. Both Kv1 and Kv3 potassium channels
    mediate the fast repolarization, rapidly inactivating sodium channels ensure metabolic
    efficiency, and little AP broadening occurs during bursts of up to 1.5 kHz. Presynaptic
    Cav2.1 (P/Q-type) calcium channels open efficiently during ultrafast APs. Furthermore,
    a subset of synaptic vesicles is tightly coupled to Ca2+ channels, and vesicles
    are rapidly recruited to the release site. These data reveal mechanisms of presynaptic
    AP generation and transmitter release underlying neuronal kHz signaling.
author:
- first_name: Andreas
  full_name: Ritzau Jost, Andreas
  last_name: Ritzau Jost
- first_name: Igor
  full_name: Delvendahl, Igor
  last_name: Delvendahl
- first_name: Annika
  full_name: Rings, Annika
  last_name: Rings
- first_name: Niklas
  full_name: Byczkowicz, Niklas
  last_name: Byczkowicz
- first_name: Harumi
  full_name: Harada, Harumi
  id: 2E55CDF2-F248-11E8-B48F-1D18A9856A87
  last_name: Harada
  orcid: 0000-0001-7429-7896
- first_name: Ryuichi
  full_name: Shigemoto, Ryuichi
  id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
  last_name: Shigemoto
  orcid: 0000-0001-8761-9444
- first_name: Johannes
  full_name: Hirrlinger, Johannes
  last_name: Hirrlinger
- first_name: Jens
  full_name: Eilers, Jens
  last_name: Eilers
- first_name: Stefan
  full_name: Hallermann, Stefan
  last_name: Hallermann
citation:
  ama: Ritzau Jost A, Delvendahl I, Rings A, et al. Ultrafast action potentials mediate
    kilohertz signaling at a central synapse. <i>Neuron</i>. 2014;84(1):152-163. doi:<a
    href="https://doi.org/10.1016/j.neuron.2014.08.036">10.1016/j.neuron.2014.08.036</a>
  apa: Ritzau Jost, A., Delvendahl, I., Rings, A., Byczkowicz, N., Harada, H., Shigemoto,
    R., … Hallermann, S. (2014). Ultrafast action potentials mediate kilohertz signaling
    at a central synapse. <i>Neuron</i>. Elsevier. <a href="https://doi.org/10.1016/j.neuron.2014.08.036">https://doi.org/10.1016/j.neuron.2014.08.036</a>
  chicago: Ritzau Jost, Andreas, Igor Delvendahl, Annika Rings, Niklas Byczkowicz,
    Harumi Harada, Ryuichi Shigemoto, Johannes Hirrlinger, Jens Eilers, and Stefan
    Hallermann. “Ultrafast Action Potentials Mediate Kilohertz Signaling at a Central
    Synapse.” <i>Neuron</i>. Elsevier, 2014. <a href="https://doi.org/10.1016/j.neuron.2014.08.036">https://doi.org/10.1016/j.neuron.2014.08.036</a>.
  ieee: A. Ritzau Jost <i>et al.</i>, “Ultrafast action potentials mediate kilohertz
    signaling at a central synapse,” <i>Neuron</i>, vol. 84, no. 1. Elsevier, pp.
    152–163, 2014.
  ista: Ritzau Jost A, Delvendahl I, Rings A, Byczkowicz N, Harada H, Shigemoto R,
    Hirrlinger J, Eilers J, Hallermann S. 2014. Ultrafast action potentials mediate
    kilohertz signaling at a central synapse. Neuron. 84(1), 152–163.
  mla: Ritzau Jost, Andreas, et al. “Ultrafast Action Potentials Mediate Kilohertz
    Signaling at a Central Synapse.” <i>Neuron</i>, vol. 84, no. 1, Elsevier, 2014,
    pp. 152–63, doi:<a href="https://doi.org/10.1016/j.neuron.2014.08.036">10.1016/j.neuron.2014.08.036</a>.
  short: A. Ritzau Jost, I. Delvendahl, A. Rings, N. Byczkowicz, H. Harada, R. Shigemoto,
    J. Hirrlinger, J. Eilers, S. Hallermann, Neuron 84 (2014) 152–163.
date_created: 2018-12-11T11:54:36Z
date_published: 2014-10-01T00:00:00Z
date_updated: 2021-01-12T06:53:55Z
day: '01'
department:
- _id: RySh
doi: 10.1016/j.neuron.2014.08.036
intvolume: '        84'
issue: '1'
language:
- iso: eng
month: '10'
oa_version: None
page: 152 - 163
publication: Neuron
publication_status: published
publisher: Elsevier
publist_id: '5197'
quality_controlled: '1'
scopus_import: 1
status: public
title: Ultrafast action potentials mediate kilohertz signaling at a central synapse
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 84
year: '2014'
...
---
_id: '1899'
abstract:
- lang: eng
  text: Asymmetric cell divisions allow stem cells to balance proliferation and differentiation.
    During embryogenesis, murine epidermis expands rapidly from a single layer of
    unspecified basal layer progenitors to a stratified, differentiated epithelium.
    Morphogenesis involves perpendicular (asymmetric) divisions and the spindle orientation
    protein LGN, but little is known about how the apical localization of LGN is regulated.
    Here, we combine conventional genetics and lentiviral-mediated in vivo RNAi to
    explore the functions of the LGN-interacting proteins Par3, mInsc and Gα i3. Whereas
    loss of each gene alone leads to randomized division angles, combined loss of
    Gnai3 and mInsc causes a phenotype of mostly planar divisions, akin to loss of
    LGN. These findings lend experimental support for the hitherto untested model
    that Par3-mInsc and Gα i3 act cooperatively to polarize LGN and promote perpendicular
    divisions. Finally, we uncover a developmental switch between delamination-driven
    early stratification and spindle-orientation-dependent differentiation that occurs
    around E15, revealing a two-step mechanism underlying epidermal maturation.
article_processing_charge: No
article_type: original
author:
- first_name: Scott
  full_name: Williams, Scott
  last_name: Williams
- first_name: Lyndsay
  full_name: Ratliff, Lyndsay
  last_name: Ratliff
- first_name: Maria P
  full_name: Postiglione, Maria P
  id: 2C67902A-F248-11E8-B48F-1D18A9856A87
  last_name: Postiglione
- first_name: Juergen
  full_name: Knoblich, Juergen
  last_name: Knoblich
- first_name: Elaine
  full_name: Fuchs, Elaine
  last_name: Fuchs
citation:
  ama: Williams S, Ratliff L, Postiglione MP, Knoblich J, Fuchs E. Par3-mInsc and
    Gα i3 cooperate to promote oriented epidermal cell divisions through LGN. <i>Nature
    Cell Biology</i>. 2014;16(8):758-769. doi:<a href="https://doi.org/10.1038/ncb3001">10.1038/ncb3001</a>
  apa: Williams, S., Ratliff, L., Postiglione, M. P., Knoblich, J., &#38; Fuchs, E.
    (2014). Par3-mInsc and Gα i3 cooperate to promote oriented epidermal cell divisions
    through LGN. <i>Nature Cell Biology</i>. Nature Publishing Group. <a href="https://doi.org/10.1038/ncb3001">https://doi.org/10.1038/ncb3001</a>
  chicago: Williams, Scott, Lyndsay Ratliff, Maria P Postiglione, Juergen Knoblich,
    and Elaine Fuchs. “Par3-MInsc and Gα I3 Cooperate to Promote Oriented Epidermal
    Cell Divisions through LGN.” <i>Nature Cell Biology</i>. Nature Publishing Group,
    2014. <a href="https://doi.org/10.1038/ncb3001">https://doi.org/10.1038/ncb3001</a>.
  ieee: S. Williams, L. Ratliff, M. P. Postiglione, J. Knoblich, and E. Fuchs, “Par3-mInsc
    and Gα i3 cooperate to promote oriented epidermal cell divisions through LGN,”
    <i>Nature Cell Biology</i>, vol. 16, no. 8. Nature Publishing Group, pp. 758–769,
    2014.
  ista: Williams S, Ratliff L, Postiglione MP, Knoblich J, Fuchs E. 2014. Par3-mInsc
    and Gα i3 cooperate to promote oriented epidermal cell divisions through LGN.
    Nature Cell Biology. 16(8), 758–769.
  mla: Williams, Scott, et al. “Par3-MInsc and Gα I3 Cooperate to Promote Oriented
    Epidermal Cell Divisions through LGN.” <i>Nature Cell Biology</i>, vol. 16, no.
    8, Nature Publishing Group, 2014, pp. 758–69, doi:<a href="https://doi.org/10.1038/ncb3001">10.1038/ncb3001</a>.
  short: S. Williams, L. Ratliff, M.P. Postiglione, J. Knoblich, E. Fuchs, Nature
    Cell Biology 16 (2014) 758–769.
date_created: 2018-12-11T11:54:36Z
date_published: 2014-07-13T00:00:00Z
date_updated: 2021-01-12T06:53:55Z
day: '13'
department:
- _id: SiHi
doi: 10.1038/ncb3001
external_id:
  pmid:
  - '25016959'
intvolume: '        16'
issue: '8'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4159251/
month: '07'
oa: 1
oa_version: Submitted Version
page: 758 - 769
pmid: 1
publication: Nature Cell Biology
publication_status: published
publisher: Nature Publishing Group
publist_id: '5196'
quality_controlled: '1'
scopus_import: 1
status: public
title: Par3-mInsc and Gα i3 cooperate to promote oriented epidermal cell divisions
  through LGN
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 16
year: '2014'
...
---
_id: '1900'
abstract:
- lang: eng
  text: Epithelial cell layers need to be tightly regulated to maintain their integrity
    and correct function. Cell integration into epithelial sheets is now shown to
    depend on the N-WASP-regulated stabilization of cortical F-actin, which generates
    distinct patterns of apical-lateral contractility at E-cadherin-based cell-cell
    junctions.
author:
- first_name: Martin
  full_name: Behrndt, Martin
  id: 3ECECA3A-F248-11E8-B48F-1D18A9856A87
  last_name: Behrndt
- first_name: Carl-Philipp J
  full_name: Heisenberg, Carl-Philipp J
  id: 39427864-F248-11E8-B48F-1D18A9856A87
  last_name: Heisenberg
  orcid: 0000-0002-0912-4566
citation:
  ama: Behrndt M, Heisenberg C-PJ. Lateral junction dynamics lead the way out. <i>Nature
    Cell Biology</i>. 2014;16(2):127-129. doi:<a href="https://doi.org/10.1038/ncb2913">10.1038/ncb2913</a>
  apa: Behrndt, M., &#38; Heisenberg, C.-P. J. (2014). Lateral junction dynamics lead
    the way out. <i>Nature Cell Biology</i>. Nature Publishing Group. <a href="https://doi.org/10.1038/ncb2913">https://doi.org/10.1038/ncb2913</a>
  chicago: Behrndt, Martin, and Carl-Philipp J Heisenberg. “Lateral Junction Dynamics
    Lead the Way Out.” <i>Nature Cell Biology</i>. Nature Publishing Group, 2014.
    <a href="https://doi.org/10.1038/ncb2913">https://doi.org/10.1038/ncb2913</a>.
  ieee: M. Behrndt and C.-P. J. Heisenberg, “Lateral junction dynamics lead the way
    out,” <i>Nature Cell Biology</i>, vol. 16, no. 2. Nature Publishing Group, pp.
    127–129, 2014.
  ista: Behrndt M, Heisenberg C-PJ. 2014. Lateral junction dynamics lead the way out.
    Nature Cell Biology. 16(2), 127–129.
  mla: Behrndt, Martin, and Carl-Philipp J. Heisenberg. “Lateral Junction Dynamics
    Lead the Way Out.” <i>Nature Cell Biology</i>, vol. 16, no. 2, Nature Publishing
    Group, 2014, pp. 127–29, doi:<a href="https://doi.org/10.1038/ncb2913">10.1038/ncb2913</a>.
  short: M. Behrndt, C.-P.J. Heisenberg, Nature Cell Biology 16 (2014) 127–129.
date_created: 2018-12-11T11:54:37Z
date_published: 2014-01-31T00:00:00Z
date_updated: 2021-01-12T06:53:56Z
day: '31'
department:
- _id: CaHe
doi: 10.1038/ncb2913
intvolume: '        16'
issue: '2'
language:
- iso: eng
month: '01'
oa_version: None
page: 127 - 129
publication: Nature Cell Biology
publication_status: published
publisher: Nature Publishing Group
publist_id: '5195'
quality_controlled: '1'
scopus_import: 1
status: public
title: Lateral junction dynamics lead the way out
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 16
year: '2014'
...
---
_id: '1901'
abstract:
- lang: eng
  text: In plants, the patterning of stem cell-enriched meristems requires a graded
    auxin response maximum that emerges from the concerted action of polar auxin transport,
    auxin biosynthesis, auxin metabolism, and cellular auxin response machinery. However,
    mechanisms underlying this auxin response maximum-mediated root stem cell maintenance
    are not fully understood. Here, we present unexpected evidence that WUSCHEL-RELATED
    HOMEOBOX 5 (WOX5) transcription factor modulates expression of auxin biosynthetic
    genes in the quiescent center (QC) of the root and thus provides a robust mechanism
    for the maintenance of auxin response maximum in the root tip. This WOX5 action
    is balanced through the activity of indole-3-acetic acid 17 (IAA17) auxin response
    repressor. Our combined genetic, cell biology, and computational modeling studies
    revealed a previously uncharacterized feedback loop linking WOX5-mediated auxin
    production to IAA17-dependent repression of auxin responses. This WOX5-IAA17 feedback
    circuit further assures the maintenance of auxin response maximum in the root
    tip and thereby contributes to the maintenance of distal stem cell (DSC) populations.
    Our experimental studies and in silico computer simulations both demonstrate that
    the WOX5-IAA17 feedback circuit is essential for the maintenance of auxin gradient
    in the root tip and the auxin-mediated root DSC differentiation.
acknowledgement: "This work was supported by funding from the projects CZ.1.07/2.3.00/20.0043
  and CZ.1.05/1.1.00/02.0068 (to CEITEC, Central European Institute of Technology)
  and the Odysseus program of the Research Foundation-Flanders to J.F\r\n"
author:
- first_name: Huiyu
  full_name: Tian, Huiyu
  last_name: Tian
- first_name: Krzysztof T
  full_name: Wabnik, Krzysztof T
  last_name: Wabnik
- first_name: Tiantian
  full_name: Niu, Tiantian
  last_name: Niu
- first_name: Hongjiang
  full_name: Li, Hongjiang
  last_name: Li
- first_name: Qianqian
  full_name: Yu, Qianqian
  last_name: Yu
- first_name: Stephan
  full_name: Pollmann, Stephan
  last_name: Pollmann
- first_name: Steffen
  full_name: Vanneste, Steffen
  last_name: Vanneste
- first_name: Willy
  full_name: Govaerts, Willy
  last_name: Govaerts
- first_name: Jakub
  full_name: Rolčík, Jakub
  last_name: Rolčík
- first_name: Markus
  full_name: Geisler, Markus
  last_name: Geisler
- first_name: Jirí
  full_name: Friml, Jirí
  id: 4159519E-F248-11E8-B48F-1D18A9856A87
  last_name: Friml
  orcid: 0000-0002-8302-7596
- first_name: Zhaojun
  full_name: Ding, Zhaojun
  last_name: Ding
citation:
  ama: Tian H, Wabnik KT, Niu T, et al. WOX5-IAA17 feedback circuit-mediated cellular
    auxin response is crucial for the patterning of root stem cell niches in arabidopsis.
    <i>Molecular Plant</i>. 2014;7(2):277-289. doi:<a href="https://doi.org/10.1093/mp/sst118">10.1093/mp/sst118</a>
  apa: Tian, H., Wabnik, K. T., Niu, T., Li, H., Yu, Q., Pollmann, S., … Ding, Z.
    (2014). WOX5-IAA17 feedback circuit-mediated cellular auxin response is crucial
    for the patterning of root stem cell niches in arabidopsis. <i>Molecular Plant</i>.
    Oxford University Press. <a href="https://doi.org/10.1093/mp/sst118">https://doi.org/10.1093/mp/sst118</a>
  chicago: Tian, Huiyu, Krzysztof T Wabnik, Tiantian Niu, Hongjiang Li, Qianqian Yu,
    Stephan Pollmann, Steffen Vanneste, et al. “WOX5-IAA17 Feedback Circuit-Mediated
    Cellular Auxin Response Is Crucial for the Patterning of Root Stem Cell Niches
    in Arabidopsis.” <i>Molecular Plant</i>. Oxford University Press, 2014. <a href="https://doi.org/10.1093/mp/sst118">https://doi.org/10.1093/mp/sst118</a>.
  ieee: H. Tian <i>et al.</i>, “WOX5-IAA17 feedback circuit-mediated cellular auxin
    response is crucial for the patterning of root stem cell niches in arabidopsis,”
    <i>Molecular Plant</i>, vol. 7, no. 2. Oxford University Press, pp. 277–289, 2014.
  ista: Tian H, Wabnik KT, Niu T, Li H, Yu Q, Pollmann S, Vanneste S, Govaerts W,
    Rolčík J, Geisler M, Friml J, Ding Z. 2014. WOX5-IAA17 feedback circuit-mediated
    cellular auxin response is crucial for the patterning of root stem cell niches
    in arabidopsis. Molecular Plant. 7(2), 277–289.
  mla: Tian, Huiyu, et al. “WOX5-IAA17 Feedback Circuit-Mediated Cellular Auxin Response
    Is Crucial for the Patterning of Root Stem Cell Niches in Arabidopsis.” <i>Molecular
    Plant</i>, vol. 7, no. 2, Oxford University Press, 2014, pp. 277–89, doi:<a href="https://doi.org/10.1093/mp/sst118">10.1093/mp/sst118</a>.
  short: H. Tian, K.T. Wabnik, T. Niu, H. Li, Q. Yu, S. Pollmann, S. Vanneste, W.
    Govaerts, J. Rolčík, M. Geisler, J. Friml, Z. Ding, Molecular Plant 7 (2014) 277–289.
date_created: 2018-12-11T11:54:37Z
date_published: 2014-02-01T00:00:00Z
date_updated: 2021-01-12T06:53:57Z
day: '01'
department:
- _id: JiFr
doi: 10.1093/mp/sst118
intvolume: '         7'
issue: '2'
language:
- iso: eng
month: '02'
oa_version: None
page: 277 - 289
publication: Molecular Plant
publication_status: published
publisher: Oxford University Press
publist_id: '5194'
scopus_import: 1
status: public
title: WOX5-IAA17 feedback circuit-mediated cellular auxin response is crucial for
  the patterning of root stem cell niches in arabidopsis
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 7
year: '2014'
...
---
_id: '1902'
abstract:
- lang: eng
  text: In the 1960s-1980s, determination of bacterial growth rates was an important
    tool in microbial genetics, biochemistry, molecular biology, and microbial physiology.
    The exciting technical developments of the 1990s and the 2000s eclipsed that tool;
    as a result, many investigators today lack experience with growth rate measurements.
    Recently, investigators in a number of areas have started to use measurements
    of bacterial growth rates for a variety of purposes. Those measurements have been
    greatly facilitated by the availability of microwell plate readers that permit
    the simultaneous measurements on up to 384 different cultures. Only the exponential
    (logarithmic) portions of the resulting growth curves are useful for determining
    growth rates, and manual determination of that portion and calculation of growth
    rates can be tedious for high-throughput purposes. Here, we introduce the program
    GrowthRates that uses plate reader output files to automatically determine the
    exponential portion of the curve and to automatically calculate the growth rate,
    the maximum culture density, and the duration of the growth lag phase. GrowthRates
    is freely available for Macintosh, Windows, and Linux.We discuss the effects of
    culture volume, the classical bacterial growth curve, and the differences between
    determinations in rich media and minimal (mineral salts) media. This protocol
    covers calibration of the plate reader, growth of culture inocula for both rich
    and minimal media, and experimental setup. As a guide to reliability, we report
    typical day-to-day variation in growth rates and variation within experiments
    with respect to position of wells within the plates.
article_processing_charge: No
article_type: original
author:
- first_name: Barry
  full_name: Hall, Barry
  last_name: Hall
- first_name: Hande
  full_name: Acar, Hande
  id: 2DDF136A-F248-11E8-B48F-1D18A9856A87
  last_name: Acar
  orcid: 0000-0003-1986-9753
- first_name: Anna
  full_name: Nandipati, Anna
  last_name: Nandipati
- first_name: Miriam
  full_name: Barlow, Miriam
  last_name: Barlow
citation:
  ama: Hall B, Acar H, Nandipati A, Barlow M. Growth rates made easy. <i>Molecular
    Biology and Evolution</i>. 2014;31(1):232-238. doi:<a href="https://doi.org/10.1093/molbev/mst187">10.1093/molbev/mst187</a>
  apa: Hall, B., Acar, H., Nandipati, A., &#38; Barlow, M. (2014). Growth rates made
    easy. <i>Molecular Biology and Evolution</i>. Oxford University Press. <a href="https://doi.org/10.1093/molbev/mst187">https://doi.org/10.1093/molbev/mst187</a>
  chicago: Hall, Barry, Hande Acar, Anna Nandipati, and Miriam Barlow. “Growth Rates
    Made Easy.” <i>Molecular Biology and Evolution</i>. Oxford University Press, 2014.
    <a href="https://doi.org/10.1093/molbev/mst187">https://doi.org/10.1093/molbev/mst187</a>.
  ieee: B. Hall, H. Acar, A. Nandipati, and M. Barlow, “Growth rates made easy,” <i>Molecular
    Biology and Evolution</i>, vol. 31, no. 1. Oxford University Press, pp. 232–238,
    2014.
  ista: Hall B, Acar H, Nandipati A, Barlow M. 2014. Growth rates made easy. Molecular
    Biology and Evolution. 31(1), 232–238.
  mla: Hall, Barry, et al. “Growth Rates Made Easy.” <i>Molecular Biology and Evolution</i>,
    vol. 31, no. 1, Oxford University Press, 2014, pp. 232–38, doi:<a href="https://doi.org/10.1093/molbev/mst187">10.1093/molbev/mst187</a>.
  short: B. Hall, H. Acar, A. Nandipati, M. Barlow, Molecular Biology and Evolution
    31 (2014) 232–238.
date_created: 2018-12-11T11:54:37Z
date_published: 2014-01-01T00:00:00Z
date_updated: 2022-06-07T11:08:13Z
day: '01'
department:
- _id: JoBo
doi: 10.1093/molbev/mst187
external_id:
  pmid:
  - '24170494'
intvolume: '        31'
issue: '1'
language:
- iso: eng
month: '01'
oa_version: None
page: 232 - 238
pmid: 1
publication: Molecular Biology and Evolution
publication_identifier:
  eissn:
  - 1537-1719
  issn:
  - 0737-4038
publication_status: published
publisher: Oxford University Press
publist_id: '5193'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Growth rates made easy
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 31
year: '2014'
...
---
_id: '1903'
abstract:
- lang: eng
  text: 'We consider two-player zero-sum partial-observation stochastic games on graphs.
    Based on the information available to the players these games can be classified
    as follows: (a) general partial-observation (both players have partial view of
    the game); (b) one-sided partial-observation (one player has partial-observation
    and the other player has complete-observation); and (c) perfect-observation (both
    players have complete view of the game). The one-sided partial-observation games
    subsumes the important special case of one-player partial-observation stochastic
    games (or partial-observation Markov decision processes (POMDPs)). Based on the
    randomization available for the strategies, (a) the players may not be allowed
    to use randomization (pure strategies), or (b) they may choose a probability distribution
    over actions but the actual random choice is external and not visible to the player
    (actions invisible), or (c) they may use full randomization. We consider all these
    classes of games with reachability, and parity objectives that can express all
    ω-regular objectives. The analysis problems are classified into the qualitative
    analysis that asks for the existence of a strategy that ensures the objective
    with probability 1; and the quantitative analysis that asks for the existence
    of a strategy that ensures the objective with probability at least λ (0,1). In
    this talk we will cover a wide range of results: for perfect-observation games;
    for POMDPs; for one-sided partial-observation games; and for general partial-observation
    games.'
alternative_title:
- LNCS
author:
- first_name: Krishnendu
  full_name: Chatterjee, Krishnendu
  id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
  last_name: Chatterjee
  orcid: 0000-0002-4561-241X
citation:
  ama: 'Chatterjee K. Partial-observation stochastic reachability and parity games.
    In: Vol 8634. Springer; 2014:1-4. doi:<a href="https://doi.org/10.1007/978-3-662-44522-8_1">10.1007/978-3-662-44522-8_1</a>'
  apa: 'Chatterjee, K. (2014). Partial-observation stochastic reachability and parity
    games (Vol. 8634, pp. 1–4). Presented at the MFCS: Mathematical Foundations of
    Computer Science, Budapest, Hungary: Springer. <a href="https://doi.org/10.1007/978-3-662-44522-8_1">https://doi.org/10.1007/978-3-662-44522-8_1</a>'
  chicago: Chatterjee, Krishnendu. “Partial-Observation Stochastic Reachability and
    Parity Games,” 8634:1–4. Springer, 2014. <a href="https://doi.org/10.1007/978-3-662-44522-8_1">https://doi.org/10.1007/978-3-662-44522-8_1</a>.
  ieee: 'K. Chatterjee, “Partial-observation stochastic reachability and parity games,”
    presented at the MFCS: Mathematical Foundations of Computer Science, Budapest,
    Hungary, 2014, vol. 8634, no. PART 1, pp. 1–4.'
  ista: 'Chatterjee K. 2014. Partial-observation stochastic reachability and parity
    games. MFCS: Mathematical Foundations of Computer Science, LNCS, vol. 8634, 1–4.'
  mla: Chatterjee, Krishnendu. <i>Partial-Observation Stochastic Reachability and
    Parity Games</i>. Vol. 8634, no. PART 1, Springer, 2014, pp. 1–4, doi:<a href="https://doi.org/10.1007/978-3-662-44522-8_1">10.1007/978-3-662-44522-8_1</a>.
  short: K. Chatterjee, in:, Springer, 2014, pp. 1–4.
conference:
  end_date: 2014-08-29
  location: Budapest, Hungary
  name: 'MFCS: Mathematical Foundations of Computer Science'
  start_date: 2014-08-25
date_created: 2018-12-11T11:54:38Z
date_published: 2014-01-01T00:00:00Z
date_updated: 2023-02-23T12:23:43Z
day: '01'
department:
- _id: KrCh
doi: 10.1007/978-3-662-44522-8_1
ec_funded: 1
intvolume: '      8634'
issue: PART 1
language:
- iso: eng
month: '01'
oa_version: None
page: 1 - 4
project:
- _id: 2584A770-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: P 23499-N23
  name: Modern Graph Algorithmic Techniques in Formal Verification
- _id: 25863FF4-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: S11407
  name: Game Theory
- _id: 2581B60A-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '279307'
  name: 'Quantitative Graph Games: Theory and Applications'
- _id: 2587B514-B435-11E9-9278-68D0E5697425
  name: Microsoft Research Faculty Fellowship
publication_status: published
publisher: Springer
publist_id: '5192'
pubrep_id: '141'
quality_controlled: '1'
related_material:
  record:
  - id: '2211'
    relation: later_version
    status: public
  - id: '5381'
    relation: earlier_version
    status: public
scopus_import: 1
status: public
title: Partial-observation stochastic reachability and parity games
type: conference
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 8634
year: '2014'
...
---
_id: '1904'
abstract:
- lang: eng
  text: We prove a Strichartz inequality for a system of orthonormal functions, with
    an optimal behavior of the constant in the limit of a large number of functions.
    The estimate generalizes the usual Strichartz inequality, in the same fashion
    as the Lieb-Thirring inequality generalizes the Sobolev inequality. As an application,
    we consider the Schrödinger equation with a time-dependent potential and we show
    the existence of the wave operator in Schatten spaces.
author:
- first_name: Rupert
  full_name: Frank, Rupert
  last_name: Frank
- first_name: Mathieu
  full_name: Lewin, Mathieu
  last_name: Lewin
- first_name: Élliott
  full_name: Lieb, Élliott
  last_name: Lieb
- first_name: Robert
  full_name: Seiringer, Robert
  id: 4AFD0470-F248-11E8-B48F-1D18A9856A87
  last_name: Seiringer
  orcid: 0000-0002-6781-0521
citation:
  ama: Frank R, Lewin M, Lieb É, Seiringer R. Strichartz inequality for orthonormal
    functions. <i>Journal of the European Mathematical Society</i>. 2014;16(7):1507-1526.
    doi:<a href="https://doi.org/10.4171/JEMS/467">10.4171/JEMS/467</a>
  apa: Frank, R., Lewin, M., Lieb, É., &#38; Seiringer, R. (2014). Strichartz inequality
    for orthonormal functions. <i>Journal of the European Mathematical Society</i>.
    European Mathematical Society. <a href="https://doi.org/10.4171/JEMS/467">https://doi.org/10.4171/JEMS/467</a>
  chicago: Frank, Rupert, Mathieu Lewin, Élliott Lieb, and Robert Seiringer. “Strichartz
    Inequality for Orthonormal Functions.” <i>Journal of the European Mathematical
    Society</i>. European Mathematical Society, 2014. <a href="https://doi.org/10.4171/JEMS/467">https://doi.org/10.4171/JEMS/467</a>.
  ieee: R. Frank, M. Lewin, É. Lieb, and R. Seiringer, “Strichartz inequality for
    orthonormal functions,” <i>Journal of the European Mathematical Society</i>, vol.
    16, no. 7. European Mathematical Society, pp. 1507–1526, 2014.
  ista: Frank R, Lewin M, Lieb É, Seiringer R. 2014. Strichartz inequality for orthonormal
    functions. Journal of the European Mathematical Society. 16(7), 1507–1526.
  mla: Frank, Rupert, et al. “Strichartz Inequality for Orthonormal Functions.” <i>Journal
    of the European Mathematical Society</i>, vol. 16, no. 7, European Mathematical
    Society, 2014, pp. 1507–26, doi:<a href="https://doi.org/10.4171/JEMS/467">10.4171/JEMS/467</a>.
  short: R. Frank, M. Lewin, É. Lieb, R. Seiringer, Journal of the European Mathematical
    Society 16 (2014) 1507–1526.
date_created: 2018-12-11T11:54:38Z
date_published: 2014-08-23T00:00:00Z
date_updated: 2021-01-12T06:53:58Z
day: '23'
department:
- _id: RoSe
doi: 10.4171/JEMS/467
intvolume: '        16'
issue: '7'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: http://arxiv.org/abs/1306.1309
month: '08'
oa: 1
oa_version: Submitted Version
page: 1507 - 1526
project:
- _id: 26450934-B435-11E9-9278-68D0E5697425
  name: NSERC Postdoctoral fellowship
publication: Journal of the European Mathematical Society
publication_status: published
publisher: European Mathematical Society
publist_id: '5191'
quality_controlled: '1'
scopus_import: 1
status: public
title: Strichartz inequality for orthonormal functions
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 16
year: '2014'
...
---
_id: '1905'
abstract:
- lang: eng
  text: The unprecedented polymorphism in the major histocompatibility complex (MHC)
    genes is thought to be maintained by balancing selection from parasites. However,
    do parasites also drive divergence at MHC loci between host populations, or do
    the effects of balancing selection maintain similarities among populations? We
    examined MHC variation in populations of the livebearing fish Poecilia mexicana
    and characterized their parasite communities. Poecilia mexicana populations in
    the Cueva del Azufre system are locally adapted to darkness and the presence of
    toxic hydrogen sulphide, representing highly divergent ecotypes or incipient species.
    Parasite communities differed significantly across populations, and populations
    with higher parasite loads had higher levels of diversity at class II MHC genes.
    However, despite different parasite communities, marked divergence in adaptive
    traits and in neutral genetic markers, we found MHC alleles to be remarkably similar
    among host populations. Our findings indicate that balancing selection from parasites
    maintains immunogenetic diversity of hosts, but this process does not promote
    MHC divergence in this system. On the contrary, we suggest that balancing selection
    on immunogenetic loci may outweigh divergent selection causing divergence, thereby
    hindering host divergence and speciation. Our findings support the hypothesis
    that balancing selection maintains MHC similarities among lineages during and
    after speciation (trans-species evolution).
acknowledgement: This study was funded by grants from the National Science Foundation
  (NSF) to MT (IOS-1121832) and IS (DEB-0743406) and from the German Science Foundation
  (DFG; PL 470/1-2) and ‘LOEWE − Landesoffensive zur Entwicklung wissenschaftlich-ökonomischer
  Exzellenz’ of Hesse's Ministry of Higher Education, Research, and the Arts, to MP.
article_processing_charge: No
article_type: original
author:
- first_name: Michael
  full_name: Tobler, Michael
  last_name: Tobler
- first_name: Martin
  full_name: Plath, Martin
  last_name: Plath
- first_name: Rüdiger
  full_name: Riesch, Rüdiger
  last_name: Riesch
- first_name: Ingo
  full_name: Schlupp, Ingo
  last_name: Schlupp
- first_name: Anna V
  full_name: Grasse, Anna V
  id: 406F989C-F248-11E8-B48F-1D18A9856A87
  last_name: Grasse
- first_name: Gopi
  full_name: Munimanda, Gopi
  last_name: Munimanda
- first_name: C
  full_name: Setzer, C
  last_name: Setzer
- first_name: Dustin
  full_name: Penn, Dustin
  last_name: Penn
- first_name: Yoshan
  full_name: Moodley, Yoshan
  last_name: Moodley
citation:
  ama: Tobler M, Plath M, Riesch R, et al. Selection from parasites favours immunogenetic
    diversity but not divergence among locally adapted host populations. <i>Journal
    of Evolutionary Biology</i>. 2014;27(5):960-974. doi:<a href="https://doi.org/10.1111/jeb.12370">10.1111/jeb.12370</a>
  apa: Tobler, M., Plath, M., Riesch, R., Schlupp, I., Grasse, A. V., Munimanda, G.,
    … Moodley, Y. (2014). Selection from parasites favours immunogenetic diversity
    but not divergence among locally adapted host populations. <i>Journal of Evolutionary
    Biology</i>. Wiley. <a href="https://doi.org/10.1111/jeb.12370">https://doi.org/10.1111/jeb.12370</a>
  chicago: Tobler, Michael, Martin Plath, Rüdiger Riesch, Ingo Schlupp, Anna V Grasse,
    Gopi Munimanda, C Setzer, Dustin Penn, and Yoshan Moodley. “Selection from Parasites
    Favours Immunogenetic Diversity but Not Divergence among Locally Adapted Host
    Populations.” <i>Journal of Evolutionary Biology</i>. Wiley, 2014. <a href="https://doi.org/10.1111/jeb.12370">https://doi.org/10.1111/jeb.12370</a>.
  ieee: M. Tobler <i>et al.</i>, “Selection from parasites favours immunogenetic diversity
    but not divergence among locally adapted host populations,” <i>Journal of Evolutionary
    Biology</i>, vol. 27, no. 5. Wiley, pp. 960–974, 2014.
  ista: Tobler M, Plath M, Riesch R, Schlupp I, Grasse AV, Munimanda G, Setzer C,
    Penn D, Moodley Y. 2014. Selection from parasites favours immunogenetic diversity
    but not divergence among locally adapted host populations. Journal of Evolutionary
    Biology. 27(5), 960–974.
  mla: Tobler, Michael, et al. “Selection from Parasites Favours Immunogenetic Diversity
    but Not Divergence among Locally Adapted Host Populations.” <i>Journal of Evolutionary
    Biology</i>, vol. 27, no. 5, Wiley, 2014, pp. 960–74, doi:<a href="https://doi.org/10.1111/jeb.12370">10.1111/jeb.12370</a>.
  short: M. Tobler, M. Plath, R. Riesch, I. Schlupp, A.V. Grasse, G. Munimanda, C.
    Setzer, D. Penn, Y. Moodley, Journal of Evolutionary Biology 27 (2014) 960–974.
date_created: 2018-12-11T11:54:38Z
date_published: 2014-04-12T00:00:00Z
date_updated: 2022-06-07T09:22:20Z
day: '12'
department:
- _id: SyCr
doi: 10.1111/jeb.12370
external_id:
  pmid:
  - '24725091'
intvolume: '        27'
issue: '5'
language:
- iso: eng
month: '04'
oa_version: None
page: 960 - 974
pmid: 1
publication: Journal of Evolutionary Biology
publication_identifier:
  eissn:
  - 1420-9101
  issn:
  - 1010-061X
publication_status: published
publisher: Wiley
publist_id: '5190'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Selection from parasites favours immunogenetic diversity but not divergence
  among locally adapted host populations
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 27
year: '2014'
...
---
_id: '1906'
abstract:
- lang: eng
  text: In this paper, we introduce a novel scene representation for the visualization
    of large-scale point clouds accompanied by a set of high-resolution photographs.
    Many real-world applications deal with very densely sampled point-cloud data,
    which are augmented with photographs that often reveal lighting variations and
    inaccuracies in registration. Consequently, the high-quality representation of
    the captured data, i.e., both point clouds and photographs together, is a challenging
    and time-consuming task. We propose a two-phase approach, in which the first (preprocessing)
    phase generates multiple overlapping surface patches and handles the problem of
    seamless texture generation locally for each patch. The second phase stitches
    these patches at render-time to produce a high-quality visualization of the data.
    As a result of the proposed localization of the global texturing problem, our
    algorithm is more than an order of magnitude faster than equivalent mesh-based
    texturing techniques. Furthermore, since our preprocessing phase requires only
    a minor fraction of the whole data set at once, we provide maximum flexibility
    when dealing with growing data sets.
acknowledgement: This research was supported by the Austrian Research Promotion Agency
  (FFG) project REPLICATE (no. 835948), the EU FP7 project HARVEST4D (no. 323567).
author:
- first_name: Murat
  full_name: Arikan, Murat
  last_name: Arikan
- first_name: Reinhold
  full_name: Preiner, Reinhold
  last_name: Preiner
- first_name: Claus
  full_name: Scheiblauer, Claus
  last_name: Scheiblauer
- first_name: Stefan
  full_name: Jeschke, Stefan
  id: 44D6411A-F248-11E8-B48F-1D18A9856A87
  last_name: Jeschke
- first_name: Michael
  full_name: Wimmer, Michael
  last_name: Wimmer
citation:
  ama: Arikan M, Preiner R, Scheiblauer C, Jeschke S, Wimmer M. Large-scale point-cloud
    visualization through localized textured surface reconstruction. <i>IEEE Transactions
    on Visualization and Computer Graphics</i>. 2014;20(9):1280-1292. doi:<a href="https://doi.org/10.1109/TVCG.2014.2312011">10.1109/TVCG.2014.2312011</a>
  apa: Arikan, M., Preiner, R., Scheiblauer, C., Jeschke, S., &#38; Wimmer, M. (2014).
    Large-scale point-cloud visualization through localized textured surface reconstruction.
    <i>IEEE Transactions on Visualization and Computer Graphics</i>. IEEE. <a href="https://doi.org/10.1109/TVCG.2014.2312011">https://doi.org/10.1109/TVCG.2014.2312011</a>
  chicago: Arikan, Murat, Reinhold Preiner, Claus Scheiblauer, Stefan Jeschke, and
    Michael Wimmer. “Large-Scale Point-Cloud Visualization through Localized Textured
    Surface Reconstruction.” <i>IEEE Transactions on Visualization and Computer Graphics</i>.
    IEEE, 2014. <a href="https://doi.org/10.1109/TVCG.2014.2312011">https://doi.org/10.1109/TVCG.2014.2312011</a>.
  ieee: M. Arikan, R. Preiner, C. Scheiblauer, S. Jeschke, and M. Wimmer, “Large-scale
    point-cloud visualization through localized textured surface reconstruction,”
    <i>IEEE Transactions on Visualization and Computer Graphics</i>, vol. 20, no.
    9. IEEE, pp. 1280–1292, 2014.
  ista: Arikan M, Preiner R, Scheiblauer C, Jeschke S, Wimmer M. 2014. Large-scale
    point-cloud visualization through localized textured surface reconstruction. IEEE
    Transactions on Visualization and Computer Graphics. 20(9), 1280–1292.
  mla: Arikan, Murat, et al. “Large-Scale Point-Cloud Visualization through Localized
    Textured Surface Reconstruction.” <i>IEEE Transactions on Visualization and Computer
    Graphics</i>, vol. 20, no. 9, IEEE, 2014, pp. 1280–92, doi:<a href="https://doi.org/10.1109/TVCG.2014.2312011">10.1109/TVCG.2014.2312011</a>.
  short: M. Arikan, R. Preiner, C. Scheiblauer, S. Jeschke, M. Wimmer, IEEE Transactions
    on Visualization and Computer Graphics 20 (2014) 1280–1292.
date_created: 2018-12-11T11:54:39Z
date_published: 2014-09-09T00:00:00Z
date_updated: 2021-01-12T06:53:59Z
day: '09'
ddc:
- '000'
department:
- _id: ChWo
doi: 10.1109/TVCG.2014.2312011
file:
- access_level: open_access
  checksum: 5bf58942d2eb20adf03c7f9ea2e68124
  content_type: application/pdf
  creator: system
  date_created: 2018-12-12T10:17:41Z
  date_updated: 2020-07-14T12:45:20Z
  file_id: '5297'
  file_name: IST-2016-573-v1+1_arikan-2014-pcvis-draft.pdf
  file_size: 13594598
  relation: main_file
file_date_updated: 2020-07-14T12:45:20Z
has_accepted_license: '1'
intvolume: '        20'
issue: '9'
language:
- iso: eng
month: '09'
oa: 1
oa_version: Submitted Version
page: 1280 - 1292
project:
- _id: 25357BD2-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: P 24352-N23
  name: 'Deep Pictures: Creating Visual and Haptic Vector Images'
publication: IEEE Transactions on Visualization and Computer Graphics
publication_status: published
publisher: IEEE
publist_id: '5189'
pubrep_id: '573'
scopus_import: 1
status: public
title: Large-scale point-cloud visualization through localized textured surface reconstruction
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 20
year: '2014'
...
---
_id: '1907'
abstract:
- lang: eng
  text: 'Most cryptographic security proofs require showing that two systems are indistinguishable.
    A central tool in such proofs is that of a game, where winning the game means
    provoking a certain condition, and it is shown that the two systems considered
    cannot be distinguished unless this condition is provoked. Upper bounding the
    probability of winning such a game, i.e., provoking this condition, for an arbitrary
    strategy is usually hard, except in the special case where the best strategy for
    winning such a game is known to be non-adaptive. A sufficient criterion for ensuring
    the optimality of non-adaptive strategies is that of conditional equivalence to
    a system, a notion introduced in [1]. In this paper, we show that this criterion
    is not necessary to ensure the optimality of non-adaptive strategies by giving
    two results of independent interest: 1) the optimality of non-adaptive strategies
    is not preserved under parallel composition; 2) in contrast, conditional equivalence
    is preserved under parallel composition.'
article_number: '6875125'
author:
- first_name: Grégory
  full_name: Demay, Grégory
  last_name: Demay
- first_name: Peter
  full_name: Gazi, Peter
  id: 3E0BFE38-F248-11E8-B48F-1D18A9856A87
  last_name: Gazi
- first_name: Ueli
  full_name: Maurer, Ueli
  last_name: Maurer
- first_name: Björn
  full_name: Tackmann, Björn
  last_name: Tackmann
citation:
  ama: 'Demay G, Gazi P, Maurer U, Tackmann B. Optimality of non-adaptive strategies:
    The case of parallel games. In: <i>IEEE International Symposium on Information
    Theory</i>. IEEE; 2014. doi:<a href="https://doi.org/10.1109/ISIT.2014.6875125">10.1109/ISIT.2014.6875125</a>'
  apa: 'Demay, G., Gazi, P., Maurer, U., &#38; Tackmann, B. (2014). Optimality of
    non-adaptive strategies: The case of parallel games. In <i>IEEE International
    Symposium on Information Theory</i>. Honolulu, USA: IEEE. <a href="https://doi.org/10.1109/ISIT.2014.6875125">https://doi.org/10.1109/ISIT.2014.6875125</a>'
  chicago: 'Demay, Grégory, Peter Gazi, Ueli Maurer, and Björn Tackmann. “Optimality
    of Non-Adaptive Strategies: The Case of Parallel Games.” In <i>IEEE International
    Symposium on Information Theory</i>. IEEE, 2014. <a href="https://doi.org/10.1109/ISIT.2014.6875125">https://doi.org/10.1109/ISIT.2014.6875125</a>.'
  ieee: 'G. Demay, P. Gazi, U. Maurer, and B. Tackmann, “Optimality of non-adaptive
    strategies: The case of parallel games,” in <i>IEEE International Symposium on
    Information Theory</i>, Honolulu, USA, 2014.'
  ista: 'Demay G, Gazi P, Maurer U, Tackmann B. 2014. Optimality of non-adaptive strategies:
    The case of parallel games. IEEE International Symposium on Information Theory.
    IEEE International Symposium on Information Theory Proceedings, 6875125.'
  mla: 'Demay, Grégory, et al. “Optimality of Non-Adaptive Strategies: The Case of
    Parallel Games.” <i>IEEE International Symposium on Information Theory</i>, 6875125,
    IEEE, 2014, doi:<a href="https://doi.org/10.1109/ISIT.2014.6875125">10.1109/ISIT.2014.6875125</a>.'
  short: G. Demay, P. Gazi, U. Maurer, B. Tackmann, in:, IEEE International Symposium
    on Information Theory, IEEE, 2014.
conference:
  end_date: 2014-07-04
  location: Honolulu, USA
  name: IEEE International Symposium on Information Theory Proceedings
  start_date: 2014-06-29
date_created: 2018-12-11T11:54:39Z
date_published: 2014-01-01T00:00:00Z
date_updated: 2021-01-12T06:53:59Z
day: '01'
department:
- _id: KrPi
doi: 10.1109/ISIT.2014.6875125
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://eprint.iacr.org/2014/299
month: '01'
oa: 1
oa_version: Submitted Version
publication: IEEE International Symposium on Information Theory
publication_status: published
publisher: IEEE
publist_id: '5188'
quality_controlled: '1'
scopus_import: 1
status: public
title: 'Optimality of non-adaptive strategies: The case of parallel games'
type: conference
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
year: '2014'
...
