---
_id: '669'
abstract:
- lang: eng
  text: 'The exocyst, a eukaryotic tethering complex, coregulates targeted exocytosis
    as an effector of small GTPases in polarized cell growth. In land plants, several
    exocyst subunits are encoded by double or triple paralogs, culminating in tens
    of EXO70 paralogs. Out of 23 Arabidopsis thaliana EXO70 isoforms, we analyzed
    seven isoforms expressed in pollen. Genetic and microscopic analyses of single
    mutants in EXO70A2, EXO70C1, EXO70C2, EXO70F1, EXO70H3, EXO70H5, and EXO70H6 genes
    revealed that only a loss-of-function EXO70C2 allele resulted in a significant
    male-specific transmission defect (segregation 40%:51%:9%) due to aberrant pollen
    tube growth. Mutant pollen tubes grown in vitro exhibited an enhanced growth rate
    and a decreased thickness of the tip cell wall, causing tip bursts. However, exo70C2
    pollen tubes could frequently recover and restart their speedy elongation, resulting
    in a repetitive stop-and-go growth dynamics. A pollenspecific depletion of the
    closest paralog, EXO70C1, using artificial microRNA in the exo70C2 mutant background,
    resulted in a complete pollen-specific transmission defect, suggesting redundant
    functions of EXO70C1 and EXO70C2. Both EXO70C1 and EXO70C2, GFP tagged and expressed
    under the control of their native promoters, localized in the cytoplasm of pollen
    grains, pollen tubes, and also root trichoblast cells. The expression of EXO70C2-GFP
    complemented the aberrant growth of exo70C2 pollen tubes. The absent EXO70C2 interactions
    with core exocyst subunits in the yeast two-hybrid assay, cytoplasmic localization,
    and genetic effect suggest an unconventional EXO70 function possibly as a regulator
    of exocytosis outside the exocyst complex. In conclusion, EXO70C2 is a novel factor
    contributing to the regulation of optimal tip growth of Arabidopsis pollen tubes. '
article_processing_charge: No
article_type: original
author:
- first_name: Lukáš
  full_name: Synek, Lukáš
  last_name: Synek
- first_name: Nemanja
  full_name: Vukašinović, Nemanja
  last_name: Vukašinović
- first_name: Ivan
  full_name: Kulich, Ivan
  last_name: Kulich
- first_name: Michal
  full_name: Hála, Michal
  last_name: Hála
- first_name: Klára
  full_name: Aldorfová, Klára
  last_name: Aldorfová
- first_name: Matyas
  full_name: Fendrych, Matyas
  id: 43905548-F248-11E8-B48F-1D18A9856A87
  last_name: Fendrych
  orcid: 0000-0002-9767-8699
- first_name: Viktor
  full_name: Žárský, Viktor
  last_name: Žárský
citation:
  ama: Synek L, Vukašinović N, Kulich I, et al. EXO70C2 is a key regulatory factor
    for optimal tip growth of pollen. <i>Plant Physiology</i>. 2017;174(1):223-240.
    doi:<a href="https://doi.org/10.1104/pp.16.01282">10.1104/pp.16.01282</a>
  apa: Synek, L., Vukašinović, N., Kulich, I., Hála, M., Aldorfová, K., Fendrych,
    M., &#38; Žárský, V. (2017). EXO70C2 is a key regulatory factor for optimal tip
    growth of pollen. <i>Plant Physiology</i>. American Society of Plant Biologists.
    <a href="https://doi.org/10.1104/pp.16.01282">https://doi.org/10.1104/pp.16.01282</a>
  chicago: Synek, Lukáš, Nemanja Vukašinović, Ivan Kulich, Michal Hála, Klára Aldorfová,
    Matyas Fendrych, and Viktor Žárský. “EXO70C2 Is a Key Regulatory Factor for Optimal
    Tip Growth of Pollen.” <i>Plant Physiology</i>. American Society of Plant Biologists,
    2017. <a href="https://doi.org/10.1104/pp.16.01282">https://doi.org/10.1104/pp.16.01282</a>.
  ieee: L. Synek <i>et al.</i>, “EXO70C2 is a key regulatory factor for optimal tip
    growth of pollen,” <i>Plant Physiology</i>, vol. 174, no. 1. American Society
    of Plant Biologists, pp. 223–240, 2017.
  ista: Synek L, Vukašinović N, Kulich I, Hála M, Aldorfová K, Fendrych M, Žárský
    V. 2017. EXO70C2 is a key regulatory factor for optimal tip growth of pollen.
    Plant Physiology. 174(1), 223–240.
  mla: Synek, Lukáš, et al. “EXO70C2 Is a Key Regulatory Factor for Optimal Tip Growth
    of Pollen.” <i>Plant Physiology</i>, vol. 174, no. 1, American Society of Plant
    Biologists, 2017, pp. 223–40, doi:<a href="https://doi.org/10.1104/pp.16.01282">10.1104/pp.16.01282</a>.
  short: L. Synek, N. Vukašinović, I. Kulich, M. Hála, K. Aldorfová, M. Fendrych,
    V. Žárský, Plant Physiology 174 (2017) 223–240.
date_created: 2018-12-11T11:47:49Z
date_published: 2017-05-01T00:00:00Z
date_updated: 2021-01-12T08:08:35Z
day: '01'
ddc:
- '580'
department:
- _id: JiFr
doi: 10.1104/pp.16.01282
external_id:
  pmid:
  - '28356503'
file:
- access_level: open_access
  checksum: 97155acc6aa5f0d0a78e0589a932fe02
  content_type: application/pdf
  creator: dernst
  date_created: 2019-11-18T16:16:18Z
  date_updated: 2020-07-14T12:47:37Z
  file_id: '7041'
  file_name: 2017_PlantPhysio_Synek.pdf
  file_size: 2176903
  relation: main_file
file_date_updated: 2020-07-14T12:47:37Z
has_accepted_license: '1'
intvolume: '       174'
issue: '1'
language:
- iso: eng
month: '05'
oa: 1
oa_version: Submitted Version
page: 223 - 240
pmid: 1
publication: Plant Physiology
publication_identifier:
  issn:
  - '00320889'
publication_status: published
publisher: American Society of Plant Biologists
publist_id: '7058'
quality_controlled: '1'
scopus_import: 1
status: public
title: EXO70C2 is a key regulatory factor for optimal tip growth of pollen
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 174
year: '2017'
...
---
_id: '670'
abstract:
- lang: eng
  text: We propose an efficient method to model paper tearing in the context of interactive
    modeling. The method uses geometrical information to automatically detect potential
    starting points of tears. We further introduce a new hybrid geometrical and physical-based
    method to compute the trajectory of tears while procedurally synthesizing high
    resolution details of the tearing path using a texture based approach. The results
    obtained are compared with real paper and with previous studies on the expected
    geometric paths of paper that tears.
article_processing_charge: No
article_type: original
author:
- first_name: Camille
  full_name: Schreck, Camille
  id: 2B14B676-F248-11E8-B48F-1D18A9856A87
  last_name: Schreck
- first_name: Damien
  full_name: Rohmer, Damien
  last_name: Rohmer
- first_name: Stefanie
  full_name: Hahmann, Stefanie
  last_name: Hahmann
citation:
  ama: Schreck C, Rohmer D, Hahmann S. Interactive paper tearing. <i>Computer Graphics
    Forum</i>. 2017;36(2):95-106. doi:<a href="https://doi.org/10.1111/cgf.13110">10.1111/cgf.13110</a>
  apa: Schreck, C., Rohmer, D., &#38; Hahmann, S. (2017). Interactive paper tearing.
    <i>Computer Graphics Forum</i>. Wiley. <a href="https://doi.org/10.1111/cgf.13110">https://doi.org/10.1111/cgf.13110</a>
  chicago: Schreck, Camille, Damien Rohmer, and Stefanie Hahmann. “Interactive Paper
    Tearing.” <i>Computer Graphics Forum</i>. Wiley, 2017. <a href="https://doi.org/10.1111/cgf.13110">https://doi.org/10.1111/cgf.13110</a>.
  ieee: C. Schreck, D. Rohmer, and S. Hahmann, “Interactive paper tearing,” <i>Computer
    Graphics Forum</i>, vol. 36, no. 2. Wiley, pp. 95–106, 2017.
  ista: Schreck C, Rohmer D, Hahmann S. 2017. Interactive paper tearing. Computer
    Graphics Forum. 36(2), 95–106.
  mla: Schreck, Camille, et al. “Interactive Paper Tearing.” <i>Computer Graphics
    Forum</i>, vol. 36, no. 2, Wiley, 2017, pp. 95–106, doi:<a href="https://doi.org/10.1111/cgf.13110">10.1111/cgf.13110</a>.
  short: C. Schreck, D. Rohmer, S. Hahmann, Computer Graphics Forum 36 (2017) 95–106.
date_created: 2018-12-11T11:47:49Z
date_published: 2017-05-01T00:00:00Z
date_updated: 2021-01-12T08:08:37Z
day: '01'
ddc:
- '000'
department:
- _id: ChWo
doi: 10.1111/cgf.13110
intvolume: '        36'
issue: '2'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://hal.inria.fr/hal-01647113/file/eg_2017_schreck_paper_tearing.pdf
month: '05'
oa: 1
oa_version: Published Version
page: 95 - 106
project:
- _id: 25357BD2-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: P 24352-N23
  name: 'Deep Pictures: Creating Visual and Haptic Vector Images'
publication: Computer Graphics Forum
publication_identifier:
  issn:
  - '01677055'
publication_status: published
publisher: Wiley
publist_id: '7056'
quality_controlled: '1'
scopus_import: 1
status: public
title: Interactive paper tearing
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 36
year: '2017'
...
---
_id: '671'
abstract:
- lang: eng
  text: Humans routinely use conditionally cooperative strategies when interacting
    in repeated social dilemmas. They are more likely to cooperate if others cooperated
    before, and are ready to retaliate if others defected. To capture the emergence
    of reciprocity, most previous models consider subjects who can only choose from
    a restricted set of representative strategies, or who react to the outcome of
    the very last round only. As players memorize more rounds, the dimension of the
    strategy space increases exponentially. This increasing computational complexity
    renders simulations for individuals with higher cognitive abilities infeasible,
    especially if multiplayer interactions are taken into account. Here, we take an
    axiomatic approach instead. We propose several properties that a robust cooperative
    strategy for a repeated multiplayer dilemma should have. These properties naturally
    lead to a unique class of cooperative strategies, which contains the classical
    Win-Stay Lose-Shift rule as a special case. A comprehensive numerical analysis
    for the prisoner's dilemma and for the public goods game suggests that strategies
    of this class readily evolve across various memory-n spaces. Our results reveal
    that successful strategies depend not only on how cooperative others were in the
    past but also on the respective context of cooperation.
article_processing_charge: Yes (in subscription journal)
author:
- first_name: Christian
  full_name: Hilbe, Christian
  id: 2FDF8F3C-F248-11E8-B48F-1D18A9856A87
  last_name: Hilbe
  orcid: 0000-0001-5116-955X
- first_name: Vaquero
  full_name: Martinez, Vaquero
  last_name: Martinez
- first_name: Krishnendu
  full_name: Chatterjee, Krishnendu
  id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
  last_name: Chatterjee
  orcid: 0000-0002-4561-241X
- first_name: Martin
  full_name: Nowak, Martin
  last_name: Nowak
citation:
  ama: Hilbe C, Martinez V, Chatterjee K, Nowak M. Memory-n strategies of direct reciprocity.
    <i>PNAS</i>. 2017;114(18):4715-4720. doi:<a href="https://doi.org/10.1073/pnas.1621239114">10.1073/pnas.1621239114</a>
  apa: Hilbe, C., Martinez, V., Chatterjee, K., &#38; Nowak, M. (2017). Memory-n strategies
    of direct reciprocity. <i>PNAS</i>. National Academy of Sciences. <a href="https://doi.org/10.1073/pnas.1621239114">https://doi.org/10.1073/pnas.1621239114</a>
  chicago: Hilbe, Christian, Vaquero Martinez, Krishnendu Chatterjee, and Martin Nowak.
    “Memory-n Strategies of Direct Reciprocity.” <i>PNAS</i>. National Academy of
    Sciences, 2017. <a href="https://doi.org/10.1073/pnas.1621239114">https://doi.org/10.1073/pnas.1621239114</a>.
  ieee: C. Hilbe, V. Martinez, K. Chatterjee, and M. Nowak, “Memory-n strategies of
    direct reciprocity,” <i>PNAS</i>, vol. 114, no. 18. National Academy of Sciences,
    pp. 4715–4720, 2017.
  ista: Hilbe C, Martinez V, Chatterjee K, Nowak M. 2017. Memory-n strategies of direct
    reciprocity. PNAS. 114(18), 4715–4720.
  mla: Hilbe, Christian, et al. “Memory-n Strategies of Direct Reciprocity.” <i>PNAS</i>,
    vol. 114, no. 18, National Academy of Sciences, 2017, pp. 4715–20, doi:<a href="https://doi.org/10.1073/pnas.1621239114">10.1073/pnas.1621239114</a>.
  short: C. Hilbe, V. Martinez, K. Chatterjee, M. Nowak, PNAS 114 (2017) 4715–4720.
date_created: 2018-12-11T11:47:50Z
date_published: 2017-05-02T00:00:00Z
date_updated: 2021-01-12T08:08:37Z
day: '02'
department:
- _id: KrCh
doi: 10.1073/pnas.1621239114
ec_funded: 1
external_id:
  pmid:
  - '28420786'
intvolume: '       114'
issue: '18'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5422766/
month: '05'
oa: 1
oa_version: Published Version
page: 4715 - 4720
pmid: 1
project:
- _id: 2581B60A-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '279307'
  name: 'Quantitative Graph Games: Theory and Applications'
- _id: 2584A770-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: P 23499-N23
  name: Modern Graph Algorithmic Techniques in Formal Verification
- _id: 25863FF4-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: S11407
  name: Game Theory
publication: PNAS
publication_identifier:
  issn:
  - '00278424'
publication_status: published
publisher: National Academy of Sciences
publist_id: '7053'
quality_controlled: '1'
scopus_import: 1
status: public
title: Memory-n strategies of direct reciprocity
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 114
year: '2017'
...
---
_id: '672'
abstract:
- lang: eng
  text: Trafficking cells frequently transmigrate through epithelial and endothelial
    monolayers. How monolayers cooperate with the penetrating cells to support their
    transit is poorly understood. We studied dendritic cell (DC) entry into lymphatic
    capillaries as a model system for transendothelial migration. We find that the
    chemokine CCL21, which is the decisive guidance cue for intravasation, mainly
    localizes in the trans-Golgi network and intracellular vesicles of lymphatic endothelial
    cells. Upon DC transmigration, these Golgi deposits disperse and CCL21 becomes
    extracellularly enriched at the sites of endothelial cell-cell junctions. When
    we reconstitute the transmigration process in vitro, we find that secretion of
    CCL21-positive vesicles is triggered by a DC contact-induced calcium signal, and
    selective calcium chelation in lymphatic endothelium attenuates transmigration.
    Altogether, our data demonstrate a chemokine-mediated feedback between DCs and
    lymphatic endothelium, which facilitates transendothelial migration.
article_processing_charge: Yes
author:
- first_name: Kari
  full_name: Vaahtomeri, Kari
  id: 368EE576-F248-11E8-B48F-1D18A9856A87
  last_name: Vaahtomeri
  orcid: 0000-0001-7829-3518
- first_name: Markus
  full_name: Brown, Markus
  id: 3DAB9AFC-F248-11E8-B48F-1D18A9856A87
  last_name: Brown
- first_name: Robert
  full_name: Hauschild, Robert
  id: 4E01D6B4-F248-11E8-B48F-1D18A9856A87
  last_name: Hauschild
  orcid: 0000-0001-9843-3522
- first_name: Ingrid
  full_name: De Vries, Ingrid
  id: 4C7D837E-F248-11E8-B48F-1D18A9856A87
  last_name: De Vries
- first_name: Alexander F
  full_name: Leithner, Alexander F
  id: 3B1B77E4-F248-11E8-B48F-1D18A9856A87
  last_name: Leithner
- first_name: Matthias
  full_name: Mehling, Matthias
  id: 3C23B994-F248-11E8-B48F-1D18A9856A87
  last_name: Mehling
  orcid: 0000-0001-8599-1226
- first_name: Walter
  full_name: Kaufmann, Walter
  id: 3F99E422-F248-11E8-B48F-1D18A9856A87
  last_name: Kaufmann
  orcid: 0000-0001-9735-5315
- first_name: Michael K
  full_name: Sixt, Michael K
  id: 41E9FBEA-F248-11E8-B48F-1D18A9856A87
  last_name: Sixt
  orcid: 0000-0002-6620-9179
citation:
  ama: Vaahtomeri K, Brown M, Hauschild R, et al. Locally triggered release of the
    chemokine CCL21 promotes dendritic cell transmigration across lymphatic endothelia.
    <i>Cell Reports</i>. 2017;19(5):902-909. doi:<a href="https://doi.org/10.1016/j.celrep.2017.04.027">10.1016/j.celrep.2017.04.027</a>
  apa: Vaahtomeri, K., Brown, M., Hauschild, R., de Vries, I., Leithner, A. F., Mehling,
    M., … Sixt, M. K. (2017). Locally triggered release of the chemokine CCL21 promotes
    dendritic cell transmigration across lymphatic endothelia. <i>Cell Reports</i>.
    Cell Press. <a href="https://doi.org/10.1016/j.celrep.2017.04.027">https://doi.org/10.1016/j.celrep.2017.04.027</a>
  chicago: Vaahtomeri, Kari, Markus Brown, Robert Hauschild, Ingrid de Vries, Alexander
    F Leithner, Matthias Mehling, Walter Kaufmann, and Michael K Sixt. “Locally Triggered
    Release of the Chemokine CCL21 Promotes Dendritic Cell Transmigration across Lymphatic
    Endothelia.” <i>Cell Reports</i>. Cell Press, 2017. <a href="https://doi.org/10.1016/j.celrep.2017.04.027">https://doi.org/10.1016/j.celrep.2017.04.027</a>.
  ieee: K. Vaahtomeri <i>et al.</i>, “Locally triggered release of the chemokine CCL21
    promotes dendritic cell transmigration across lymphatic endothelia,” <i>Cell Reports</i>,
    vol. 19, no. 5. Cell Press, pp. 902–909, 2017.
  ista: Vaahtomeri K, Brown M, Hauschild R, de Vries I, Leithner AF, Mehling M, Kaufmann
    W, Sixt MK. 2017. Locally triggered release of the chemokine CCL21 promotes dendritic
    cell transmigration across lymphatic endothelia. Cell Reports. 19(5), 902–909.
  mla: Vaahtomeri, Kari, et al. “Locally Triggered Release of the Chemokine CCL21
    Promotes Dendritic Cell Transmigration across Lymphatic Endothelia.” <i>Cell Reports</i>,
    vol. 19, no. 5, Cell Press, 2017, pp. 902–09, doi:<a href="https://doi.org/10.1016/j.celrep.2017.04.027">10.1016/j.celrep.2017.04.027</a>.
  short: K. Vaahtomeri, M. Brown, R. Hauschild, I. de Vries, A.F. Leithner, M. Mehling,
    W. Kaufmann, M.K. Sixt, Cell Reports 19 (2017) 902–909.
date_created: 2018-12-11T11:47:50Z
date_published: 2017-05-02T00:00:00Z
date_updated: 2023-02-23T12:50:09Z
day: '02'
ddc:
- '570'
department:
- _id: MiSi
- _id: Bio
- _id: EM-Fac
doi: 10.1016/j.celrep.2017.04.027
ec_funded: 1
file:
- access_level: open_access
  checksum: 8fdddaab1f1d76a6ec9ca94dcb6b07a2
  content_type: application/pdf
  creator: system
  date_created: 2018-12-12T10:14:54Z
  date_updated: 2020-07-14T12:47:38Z
  file_id: '5109'
  file_name: IST-2017-900-v1+1_1-s2.0-S2211124717305211-main.pdf
  file_size: 2248814
  relation: main_file
file_date_updated: 2020-07-14T12:47:38Z
has_accepted_license: '1'
intvolume: '        19'
issue: '5'
language:
- iso: eng
license: https://creativecommons.org/licenses/by-nc-nd/4.0/
month: '05'
oa: 1
oa_version: Published Version
page: 902 - 909
project:
- _id: 25A603A2-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '281556'
  name: Cytoskeletal force generation and force transduction of migrating leukocytes
    (EU)
- _id: 25A8E5EA-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: Y 564-B12
  name: Cytoskeletal force generation and transduction of leukocytes (FWF)
publication: Cell Reports
publication_identifier:
  issn:
  - '22111247'
publication_status: published
publisher: Cell Press
publist_id: '7052'
pubrep_id: '900'
quality_controlled: '1'
scopus_import: 1
status: public
title: Locally triggered release of the chemokine CCL21 promotes dendritic cell transmigration
  across lymphatic endothelia
tmp:
  image: /images/cc_by_nc_nd.png
  legal_code_url: https://creativecommons.org/licenses/by-nc-nd/4.0/legalcode
  name: Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International
    (CC BY-NC-ND 4.0)
  short: CC BY-NC-ND (4.0)
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 19
year: '2017'
...
---
_id: '6729'
abstract:
- lang: eng
  text: Consider the problem of constructing a polar code of block length N for the
    transmission over a given channel W. Typically this requires to compute the reliability
    of all the N synthetic channels and then to include those that are sufficiently
    reliable. However, we know from [1], [2] that there is a partial order among the
    synthetic channels. Hence, it is natural to ask whether we can exploit it to reduce
    the computational burden of the construction problem. We show that, if we take
    advantage of the partial order [1], [2], we can construct a polar code by computing
    the reliability of roughly N/ log 3/2 N synthetic channels. Such a set of synthetic
    channels is universal, in the sense that it allows one to construct polar codes
    for any W, and it can be identified by solving a maximum matching problem on a
    bipartite graph. Our proof technique consists in reducing the construction problem
    to the problem of computing the maximum cardinality of an antichain for a suitable
    partially ordered set. As such, this method is general and it can be used to further
    improve the complexity of the construction problem in case a new partial order
    on the synthetic channels of polar codes is discovered.
arxiv: 1
author:
- first_name: Marco
  full_name: Mondelli, Marco
  id: 27EB676C-8706-11E9-9510-7717E6697425
  last_name: Mondelli
  orcid: 0000-0002-3242-7020
- first_name: S. Hamed
  full_name: Hassani, S. Hamed
  last_name: Hassani
- first_name: Rudiger
  full_name: Urbanke, Rudiger
  last_name: Urbanke
citation:
  ama: 'Mondelli M, Hassani SH, Urbanke R. Construction of polar codes with sublinear
    complexity. In: <i>2017 IEEE International Symposium on Information Theory </i>.
    IEEE; 2017:1853-1857. doi:<a href="https://doi.org/10.1109/isit.2017.8006850">10.1109/isit.2017.8006850</a>'
  apa: 'Mondelli, M., Hassani, S. H., &#38; Urbanke, R. (2017). Construction of polar
    codes with sublinear complexity. In <i>2017 IEEE International Symposium on Information
    Theory </i> (pp. 1853–1857). Aachen, Germany: IEEE. <a href="https://doi.org/10.1109/isit.2017.8006850">https://doi.org/10.1109/isit.2017.8006850</a>'
  chicago: Mondelli, Marco, S. Hamed Hassani, and Rudiger Urbanke. “Construction of
    Polar Codes with Sublinear Complexity.” In <i>2017 IEEE International Symposium
    on Information Theory </i>, 1853–57. IEEE, 2017. <a href="https://doi.org/10.1109/isit.2017.8006850">https://doi.org/10.1109/isit.2017.8006850</a>.
  ieee: M. Mondelli, S. H. Hassani, and R. Urbanke, “Construction of polar codes with
    sublinear complexity,” in <i>2017 IEEE International Symposium on Information
    Theory </i>, Aachen, Germany, 2017, pp. 1853–1857.
  ista: 'Mondelli M, Hassani SH, Urbanke R. 2017. Construction of polar codes with
    sublinear complexity. 2017 IEEE International Symposium on Information Theory
    . ISIT: International Symposium on Information Theory, 1853–1857.'
  mla: Mondelli, Marco, et al. “Construction of Polar Codes with Sublinear Complexity.”
    <i>2017 IEEE International Symposium on Information Theory </i>, IEEE, 2017, pp.
    1853–57, doi:<a href="https://doi.org/10.1109/isit.2017.8006850">10.1109/isit.2017.8006850</a>.
  short: M. Mondelli, S.H. Hassani, R. Urbanke, in:, 2017 IEEE International Symposium
    on Information Theory , IEEE, 2017, pp. 1853–1857.
conference:
  end_date: 2017-06-30
  location: Aachen, Germany
  name: 'ISIT: International Symposium on Information Theory'
  start_date: 2017-06-25
date_created: 2019-07-30T07:14:18Z
date_published: 2017-06-15T00:00:00Z
date_updated: 2023-02-23T12:49:08Z
day: '15'
doi: 10.1109/isit.2017.8006850
extern: '1'
external_id:
  arxiv:
  - '1612.05295'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1612.05295
month: '06'
oa: 1
oa_version: Preprint
page: 1853-1857
publication: '2017 IEEE International Symposium on Information Theory '
publication_identifier:
  eissn:
  - 2157-8117
  isbn:
  - '9781509040964'
publication_status: published
publisher: IEEE
quality_controlled: '1'
related_material:
  record:
  - id: '6663'
    relation: later_version
    status: public
status: public
title: Construction of polar codes with sublinear complexity
type: conference
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
year: '2017'
...
---
_id: '673'
abstract:
- lang: eng
  text: We present a numerical study of wavy supercritical cylindrical Couette flow
    between counter-rotating cylinders in which the wavy pattern propagates either
    prograde with the inner cylinder or retrograde opposite the rotation of the inner
    cylinder. The wave propagation reversals from prograde to retrograde and vice
    versa occur at distinct values of the inner cylinder Reynolds number when the
    associated frequency of the wavy instability vanishes. The reversal occurs for
    both twofold and threefold symmetric wavy vortices. Moreover, the wave propagation
    reversal only occurs for sufficiently strong counter-rotation. The flow pattern
    reversal appears to be intrinsic in the system as either periodic boundary conditions
    or fixed end wall boundary conditions for different system sizes always result
    in the wave propagation reversal. We present a detailed bifurcation sequence and
    parameter space diagram with respect to retrograde behavior of wavy flows. The
    retrograde propagation of the instability occurs when the inner Reynolds number
    is about two times the outer Reynolds number. The mechanism for the retrograde
    propagation is associated with the inviscidly unstable region near the inner cylinder
    and the direction of the global average azimuthal velocity. Flow dynamics, spatio-temporal
    behavior, global mean angular velocity, and torque of the flow with the wavy pattern
    are explored.
article_number: '053103'
article_processing_charge: No
author:
- first_name: Sebastian
  full_name: Altmeyer, Sebastian
  id: 2EE67FDC-F248-11E8-B48F-1D18A9856A87
  last_name: Altmeyer
  orcid: 0000-0001-5964-0203
- first_name: Richard
  full_name: Lueptow, Richard
  last_name: Lueptow
citation:
  ama: Altmeyer S, Lueptow R. Wave propagation reversal for wavy vortices in wide
    gap counter rotating cylindrical Couette flow. <i>Physical Review E</i>. 2017;95(5).
    doi:<a href="https://doi.org/10.1103/PhysRevE.95.053103">10.1103/PhysRevE.95.053103</a>
  apa: Altmeyer, S., &#38; Lueptow, R. (2017). Wave propagation reversal for wavy
    vortices in wide gap counter rotating cylindrical Couette flow. <i>Physical Review
    E</i>. American Physical Society. <a href="https://doi.org/10.1103/PhysRevE.95.053103">https://doi.org/10.1103/PhysRevE.95.053103</a>
  chicago: Altmeyer, Sebastian, and Richard Lueptow. “Wave Propagation Reversal for
    Wavy Vortices in Wide Gap Counter Rotating Cylindrical Couette Flow.” <i>Physical
    Review E</i>. American Physical Society, 2017. <a href="https://doi.org/10.1103/PhysRevE.95.053103">https://doi.org/10.1103/PhysRevE.95.053103</a>.
  ieee: S. Altmeyer and R. Lueptow, “Wave propagation reversal for wavy vortices in
    wide gap counter rotating cylindrical Couette flow,” <i>Physical Review E</i>,
    vol. 95, no. 5. American Physical Society, 2017.
  ista: Altmeyer S, Lueptow R. 2017. Wave propagation reversal for wavy vortices in
    wide gap counter rotating cylindrical Couette flow. Physical Review E. 95(5),
    053103.
  mla: Altmeyer, Sebastian, and Richard Lueptow. “Wave Propagation Reversal for Wavy
    Vortices in Wide Gap Counter Rotating Cylindrical Couette Flow.” <i>Physical Review
    E</i>, vol. 95, no. 5, 053103, American Physical Society, 2017, doi:<a href="https://doi.org/10.1103/PhysRevE.95.053103">10.1103/PhysRevE.95.053103</a>.
  short: S. Altmeyer, R. Lueptow, Physical Review E 95 (2017).
date_created: 2018-12-11T11:47:50Z
date_published: 2017-05-10T00:00:00Z
date_updated: 2023-10-10T13:30:03Z
day: '10'
department:
- _id: BjHo
doi: 10.1103/PhysRevE.95.053103
intvolume: '        95'
issue: '5'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/pdf/physics/0505164.pdf
month: '05'
oa: 1
oa_version: Submitted Version
publication: Physical Review E
publication_identifier:
  issn:
  - 2470-0045
publication_status: published
publisher: American Physical Society
publist_id: '7049'
scopus_import: '1'
status: public
title: Wave propagation reversal for wavy vortices in wide gap counter rotating cylindrical
  Couette flow
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 95
year: '2017'
...
---
_id: '6730'
abstract:
- lang: eng
  text: We introduce a new approach to proving that a sequence of deterministic linear
    codes achieves capacity on an erasure channel under maximum a posteriori decoding.
    Rather than relying on the precise structure of the codes, our method exploits
    code symmetry. In particular, the technique applies to any sequence of linear
    codes where the blocklengths are strictly increasing, the code rates converge,
    and the permutation group of each code is doubly transitive. In other words, we
    show that symmetry alone implies near-optimal performance. An important consequence
    of this result is that a sequence of Reed-Muller codes with increasing block length
    and converging rate achieves capacity. This possibility has been suggested previously
    in the literature but it has only been proven for cases where the limiting code
    rate is 0 or 1. Moreover, these results extend naturally to all affine-invariant
    codes and, thus, to extended primitive narrow-sense BCH codes. This also resolves,
    in the affirmative, the existence question for capacity-achieving sequences of
    binary cyclic codes. The primary tools used in the proof are the sharp threshold
    property for symmetric monotone Boolean functions and the area theorem for extrinsic
    information transfer functions.
arxiv: 1
author:
- first_name: Shrinivas
  full_name: Kudekar, Shrinivas
  last_name: Kudekar
- first_name: Santhosh
  full_name: Kumar, Santhosh
  last_name: Kumar
- first_name: Marco
  full_name: Mondelli, Marco
  id: 27EB676C-8706-11E9-9510-7717E6697425
  last_name: Mondelli
  orcid: 0000-0002-3242-7020
- first_name: Henry D.
  full_name: Pfister, Henry D.
  last_name: Pfister
- first_name: Eren
  full_name: Sasoglu, Eren
  last_name: Sasoglu
- first_name: Ridiger L.
  full_name: Urbanke, Ridiger L.
  last_name: Urbanke
citation:
  ama: Kudekar S, Kumar S, Mondelli M, Pfister HD, Sasoglu E, Urbanke RL. Reed–Muller
    codes achieve capacity on erasure channels. <i>IEEE Transactions on Information
    Theory</i>. 2017;63(7):4298-4316. doi:<a href="https://doi.org/10.1109/tit.2017.2673829">10.1109/tit.2017.2673829</a>
  apa: Kudekar, S., Kumar, S., Mondelli, M., Pfister, H. D., Sasoglu, E., &#38; Urbanke,
    R. L. (2017). Reed–Muller codes achieve capacity on erasure channels. <i>IEEE
    Transactions on Information Theory</i>. IEEE. <a href="https://doi.org/10.1109/tit.2017.2673829">https://doi.org/10.1109/tit.2017.2673829</a>
  chicago: Kudekar, Shrinivas, Santhosh Kumar, Marco Mondelli, Henry D. Pfister, Eren
    Sasoglu, and Ridiger L. Urbanke. “Reed–Muller Codes Achieve Capacity on Erasure
    Channels.” <i>IEEE Transactions on Information Theory</i>. IEEE, 2017. <a href="https://doi.org/10.1109/tit.2017.2673829">https://doi.org/10.1109/tit.2017.2673829</a>.
  ieee: S. Kudekar, S. Kumar, M. Mondelli, H. D. Pfister, E. Sasoglu, and R. L. Urbanke,
    “Reed–Muller codes achieve capacity on erasure channels,” <i>IEEE Transactions
    on Information Theory</i>, vol. 63, no. 7. IEEE, pp. 4298–4316, 2017.
  ista: Kudekar S, Kumar S, Mondelli M, Pfister HD, Sasoglu E, Urbanke RL. 2017. Reed–Muller
    codes achieve capacity on erasure channels. IEEE Transactions on Information Theory.
    63(7), 4298–4316.
  mla: Kudekar, Shrinivas, et al. “Reed–Muller Codes Achieve Capacity on Erasure Channels.”
    <i>IEEE Transactions on Information Theory</i>, vol. 63, no. 7, IEEE, 2017, pp.
    4298–316, doi:<a href="https://doi.org/10.1109/tit.2017.2673829">10.1109/tit.2017.2673829</a>.
  short: S. Kudekar, S. Kumar, M. Mondelli, H.D. Pfister, E. Sasoglu, R.L. Urbanke,
    IEEE Transactions on Information Theory 63 (2017) 4298–4316.
date_created: 2019-07-30T07:18:11Z
date_published: 2017-07-01T00:00:00Z
date_updated: 2021-01-12T08:08:43Z
day: '01'
doi: 10.1109/tit.2017.2673829
extern: '1'
external_id:
  arxiv:
  - '1601.04689'
intvolume: '        63'
issue: '7'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1601.04689
month: '07'
oa: 1
oa_version: Preprint
page: 4298-4316
publication: IEEE Transactions on Information Theory
publication_identifier:
  eissn:
  - 1557-9654
  issn:
  - 0018-9448
publication_status: published
publisher: IEEE
quality_controlled: '1'
status: public
title: Reed–Muller codes achieve capacity on erasure channels
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 63
year: '2017'
...
---
_id: '6731'
abstract:
- lang: eng
  text: 'We present a rate-compatible polar coding scheme that achieves the capacity
    of any family of channels. Our solution generalizes the previous results [1],
    [2] that provide capacity-achieving rate-compatible polar codes for a degraded
    family of channels. The motivation for our extension comes from the fact that
    in many practical scenarios, e.g., MIMO systems and non-Gaussian interference,
    the channels cannot be ordered by degradation. The main technical contribution
    of this paper consists in removing the degradation condition. To do so, we exploit
    the ideas coming from the construction of universal polar codes. Our scheme possesses
    the usual attractive features of polar codes: low complexity code construction,
    encoding, and decoding; super-polynomial scaling of the error probability with
    the block length; and absence of error floors. On the negative side, the scaling
    of the gap to capacity with the block length is slower than in standard polar
    codes, and we prove an upper bound on the scaling exponent.'
article_number: '7919107'
arxiv: 1
author:
- first_name: Marco
  full_name: Mondelli, Marco
  id: 27EB676C-8706-11E9-9510-7717E6697425
  last_name: Mondelli
  orcid: 0000-0002-3242-7020
- first_name: Hamed
  full_name: Hassani, Hamed
  last_name: Hassani
- first_name: Ivana
  full_name: Maric, Ivana
  last_name: Maric
- first_name: Dennis
  full_name: Hui, Dennis
  last_name: Hui
- first_name: Song-Nam
  full_name: Hong, Song-Nam
  last_name: Hong
citation:
  ama: 'Mondelli M, Hassani H, Maric I, Hui D, Hong S-N. Capacity-achieving rate-compatible
    polar codes for general channels. In: <i>2017 IEEE Wireless Communications and
    Networking Conference Workshops </i>. IEEE; 2017. doi:<a href="https://doi.org/10.1109/wcncw.2017.7919107">10.1109/wcncw.2017.7919107</a>'
  apa: 'Mondelli, M., Hassani, H., Maric, I., Hui, D., &#38; Hong, S.-N. (2017). Capacity-achieving
    rate-compatible polar codes for general channels. In <i>2017 IEEE Wireless Communications
    and Networking Conference Workshops </i>. San Francisco, CA, USA: IEEE. <a href="https://doi.org/10.1109/wcncw.2017.7919107">https://doi.org/10.1109/wcncw.2017.7919107</a>'
  chicago: Mondelli, Marco, Hamed Hassani, Ivana Maric, Dennis Hui, and Song-Nam Hong.
    “Capacity-Achieving Rate-Compatible Polar Codes for General Channels.” In <i>2017
    IEEE Wireless Communications and Networking Conference Workshops </i>. IEEE, 2017.
    <a href="https://doi.org/10.1109/wcncw.2017.7919107">https://doi.org/10.1109/wcncw.2017.7919107</a>.
  ieee: M. Mondelli, H. Hassani, I. Maric, D. Hui, and S.-N. Hong, “Capacity-achieving
    rate-compatible polar codes for general channels,” in <i>2017 IEEE Wireless Communications
    and Networking Conference Workshops </i>, San Francisco, CA, USA, 2017.
  ista: 'Mondelli M, Hassani H, Maric I, Hui D, Hong S-N. 2017. Capacity-achieving
    rate-compatible polar codes for general channels. 2017 IEEE Wireless Communications
    and Networking Conference Workshops . WCNCW: Wireless communications and networking
    conference workshops, 7919107.'
  mla: Mondelli, Marco, et al. “Capacity-Achieving Rate-Compatible Polar Codes for
    General Channels.” <i>2017 IEEE Wireless Communications and Networking Conference
    Workshops </i>, 7919107, IEEE, 2017, doi:<a href="https://doi.org/10.1109/wcncw.2017.7919107">10.1109/wcncw.2017.7919107</a>.
  short: M. Mondelli, H. Hassani, I. Maric, D. Hui, S.-N. Hong, in:, 2017 IEEE Wireless
    Communications and Networking Conference Workshops , IEEE, 2017.
conference:
  end_date: 2017-03-22
  location: San Francisco, CA, USA
  name: 'WCNCW: Wireless communications and networking conference workshops'
  start_date: 2017-03-19
date_created: 2019-07-31T05:56:58Z
date_published: 2017-05-04T00:00:00Z
date_updated: 2021-01-12T08:08:43Z
day: '04'
doi: 10.1109/wcncw.2017.7919107
extern: '1'
external_id:
  arxiv:
  - '1611.01199'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1611.01199
month: '05'
oa: 1
oa_version: Preprint
publication: '2017 IEEE Wireless Communications and Networking Conference Workshops '
publication_identifier:
  isbn:
  - '9781509059089'
publication_status: published
publisher: IEEE
quality_controlled: '1'
status: public
title: Capacity-achieving rate-compatible polar codes for general channels
type: conference
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
year: '2017'
...
---
_id: '674'
abstract:
- lang: eng
  text: Navigation of cells along gradients of guidance cues is a determining step
    in many developmental and immunological processes. Gradients can either be soluble
    or immobilized to tissues as demonstrated for the haptotactic migration of dendritic
    cells (DCs) toward higher concentrations of immobilized chemokine CCL21. To elucidate
    how gradient characteristics govern cellular response patterns, we here introduce
    an in vitro system allowing to track migratory responses of DCs to precisely controlled
    immobilized gradients of CCL21. We find that haptotactic sensing depends on the
    absolute CCL21 concentration and local steepness of the gradient, consistent with
    a scenario where DC directionality is governed by the signal-to-noise ratio of
    CCL21 binding to the receptor CCR7. We find that the conditions for optimal DC
    guidance are perfectly provided by the CCL21 gradients we measure in vivo. Furthermore,
    we find that CCR7 signal termination by the G-protein-coupled receptor kinase
    6 (GRK6) is crucial for haptotactic but dispensable for chemotactic CCL21 gradient
    sensing in vitro and confirm those observations in vivo. These findings suggest
    that stable, tissue-bound CCL21 gradients as sustainable “roads” ensure optimal
    guidance in vivo.
author:
- first_name: Jan
  full_name: Schwarz, Jan
  id: 346C1EC6-F248-11E8-B48F-1D18A9856A87
  last_name: Schwarz
- first_name: Veronika
  full_name: Bierbaum, Veronika
  id: 3FD04378-F248-11E8-B48F-1D18A9856A87
  last_name: Bierbaum
- first_name: Kari
  full_name: Vaahtomeri, Kari
  id: 368EE576-F248-11E8-B48F-1D18A9856A87
  last_name: Vaahtomeri
  orcid: 0000-0001-7829-3518
- first_name: Robert
  full_name: Hauschild, Robert
  id: 4E01D6B4-F248-11E8-B48F-1D18A9856A87
  last_name: Hauschild
  orcid: 0000-0001-9843-3522
- first_name: Markus
  full_name: Brown, Markus
  id: 3DAB9AFC-F248-11E8-B48F-1D18A9856A87
  last_name: Brown
- first_name: Ingrid
  full_name: De Vries, Ingrid
  id: 4C7D837E-F248-11E8-B48F-1D18A9856A87
  last_name: De Vries
- first_name: Alexander F
  full_name: Leithner, Alexander F
  id: 3B1B77E4-F248-11E8-B48F-1D18A9856A87
  last_name: Leithner
- first_name: Anne
  full_name: Reversat, Anne
  id: 35B76592-F248-11E8-B48F-1D18A9856A87
  last_name: Reversat
  orcid: 0000-0003-0666-8928
- first_name: Jack
  full_name: Merrin, Jack
  id: 4515C308-F248-11E8-B48F-1D18A9856A87
  last_name: Merrin
  orcid: 0000-0001-5145-4609
- first_name: Teresa
  full_name: Tarrant, Teresa
  last_name: Tarrant
- first_name: Tobias
  full_name: Bollenbach, Tobias
  id: 3E6DB97A-F248-11E8-B48F-1D18A9856A87
  last_name: Bollenbach
  orcid: 0000-0003-4398-476X
- first_name: Michael K
  full_name: Sixt, Michael K
  id: 41E9FBEA-F248-11E8-B48F-1D18A9856A87
  last_name: Sixt
  orcid: 0000-0002-6620-9179
citation:
  ama: Schwarz J, Bierbaum V, Vaahtomeri K, et al. Dendritic cells interpret haptotactic
    chemokine gradients in a manner governed by signal to noise ratio and dependent
    on GRK6. <i>Current Biology</i>. 2017;27(9):1314-1325. doi:<a href="https://doi.org/10.1016/j.cub.2017.04.004">10.1016/j.cub.2017.04.004</a>
  apa: Schwarz, J., Bierbaum, V., Vaahtomeri, K., Hauschild, R., Brown, M., de Vries,
    I., … Sixt, M. K. (2017). Dendritic cells interpret haptotactic chemokine gradients
    in a manner governed by signal to noise ratio and dependent on GRK6. <i>Current
    Biology</i>. Cell Press. <a href="https://doi.org/10.1016/j.cub.2017.04.004">https://doi.org/10.1016/j.cub.2017.04.004</a>
  chicago: Schwarz, Jan, Veronika Bierbaum, Kari Vaahtomeri, Robert Hauschild, Markus
    Brown, Ingrid de Vries, Alexander F Leithner, et al. “Dendritic Cells Interpret
    Haptotactic Chemokine Gradients in a Manner Governed by Signal to Noise Ratio
    and Dependent on GRK6.” <i>Current Biology</i>. Cell Press, 2017. <a href="https://doi.org/10.1016/j.cub.2017.04.004">https://doi.org/10.1016/j.cub.2017.04.004</a>.
  ieee: J. Schwarz <i>et al.</i>, “Dendritic cells interpret haptotactic chemokine
    gradients in a manner governed by signal to noise ratio and dependent on GRK6,”
    <i>Current Biology</i>, vol. 27, no. 9. Cell Press, pp. 1314–1325, 2017.
  ista: Schwarz J, Bierbaum V, Vaahtomeri K, Hauschild R, Brown M, de Vries I, Leithner
    AF, Reversat A, Merrin J, Tarrant T, Bollenbach MT, Sixt MK. 2017. Dendritic cells
    interpret haptotactic chemokine gradients in a manner governed by signal to noise
    ratio and dependent on GRK6. Current Biology. 27(9), 1314–1325.
  mla: Schwarz, Jan, et al. “Dendritic Cells Interpret Haptotactic Chemokine Gradients
    in a Manner Governed by Signal to Noise Ratio and Dependent on GRK6.” <i>Current
    Biology</i>, vol. 27, no. 9, Cell Press, 2017, pp. 1314–25, doi:<a href="https://doi.org/10.1016/j.cub.2017.04.004">10.1016/j.cub.2017.04.004</a>.
  short: J. Schwarz, V. Bierbaum, K. Vaahtomeri, R. Hauschild, M. Brown, I. de Vries,
    A.F. Leithner, A. Reversat, J. Merrin, T. Tarrant, M.T. Bollenbach, M.K. Sixt,
    Current Biology 27 (2017) 1314–1325.
date_created: 2018-12-11T11:47:51Z
date_published: 2017-05-09T00:00:00Z
date_updated: 2023-02-23T12:50:44Z
day: '09'
department:
- _id: MiSi
- _id: Bio
- _id: NanoFab
doi: 10.1016/j.cub.2017.04.004
ec_funded: 1
intvolume: '        27'
issue: '9'
language:
- iso: eng
month: '05'
oa_version: None
page: 1314 - 1325
project:
- _id: 25681D80-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '291734'
  name: International IST Postdoc Fellowship Programme
- _id: 25A8E5EA-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: Y 564-B12
  name: Cytoskeletal force generation and transduction of leukocytes (FWF)
publication: Current Biology
publication_identifier:
  issn:
  - '09609822'
publication_status: published
publisher: Cell Press
publist_id: '7050'
quality_controlled: '1'
scopus_import: 1
status: public
title: Dendritic cells interpret haptotactic chemokine gradients in a manner governed
  by signal to noise ratio and dependent on GRK6
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 27
year: '2017'
...
---
_id: '675'
abstract:
- lang: eng
  text: 'We report the enhancement of infrared absorption of chemisorbed carbon monoxide
    on platinum in the gap of plasmonic nanoantennas. Our method is based on the self-assembled
    formation of platinum nanoislands on nanoscopic dipole antenna arrays manufactured
    via electron beam lithography. We employ systematic variations of the plasmonic
    antenna resonance to precisely couple to the molecular stretch vibration of carbon
    monoxide adsorbed on the platinum nanoislands. Ultimately, we reach more than
    1500-fold infrared absorption enhancements, allowing for an ultrasensitive detection
    of a monolayer of chemisorbed carbon monoxide. The developed procedure can be
    adapted to other metal adsorbents and molecular species and could be utilized
    for coverage sensing in surface catalytic reactions. '
article_processing_charge: No
article_type: original
author:
- first_name: Johannes
  full_name: Haase, Johannes
  last_name: Haase
- first_name: Salvatore
  full_name: Bagiante, Salvatore
  id: 38ED402E-F248-11E8-B48F-1D18A9856A87
  last_name: Bagiante
  orcid: 0000-0002-0122-9603
- first_name: Hans
  full_name: Sigg, Hans
  last_name: Sigg
- first_name: Jeroen
  full_name: Van Bokhoven, Jeroen
  last_name: Van Bokhoven
citation:
  ama: Haase J, Bagiante S, Sigg H, Van Bokhoven J. Surface enhanced infrared absorption
    of chemisorbed carbon monoxide using plasmonic nanoantennas. <i>Optics Letters</i>.
    2017;42(10):1931-1934. doi:<a href="https://doi.org/10.1364/OL.42.001931">10.1364/OL.42.001931</a>
  apa: Haase, J., Bagiante, S., Sigg, H., &#38; Van Bokhoven, J. (2017). Surface enhanced
    infrared absorption of chemisorbed carbon monoxide using plasmonic nanoantennas.
    <i>Optics Letters</i>. Optica Publishing Group. <a href="https://doi.org/10.1364/OL.42.001931">https://doi.org/10.1364/OL.42.001931</a>
  chicago: Haase, Johannes, Salvatore Bagiante, Hans Sigg, and Jeroen Van Bokhoven.
    “Surface Enhanced Infrared Absorption of Chemisorbed Carbon Monoxide Using Plasmonic
    Nanoantennas.” <i>Optics Letters</i>. Optica Publishing Group, 2017. <a href="https://doi.org/10.1364/OL.42.001931">https://doi.org/10.1364/OL.42.001931</a>.
  ieee: J. Haase, S. Bagiante, H. Sigg, and J. Van Bokhoven, “Surface enhanced infrared
    absorption of chemisorbed carbon monoxide using plasmonic nanoantennas,” <i>Optics
    Letters</i>, vol. 42, no. 10. Optica Publishing Group, pp. 1931–1934, 2017.
  ista: Haase J, Bagiante S, Sigg H, Van Bokhoven J. 2017. Surface enhanced infrared
    absorption of chemisorbed carbon monoxide using plasmonic nanoantennas. Optics
    Letters. 42(10), 1931–1934.
  mla: Haase, Johannes, et al. “Surface Enhanced Infrared Absorption of Chemisorbed
    Carbon Monoxide Using Plasmonic Nanoantennas.” <i>Optics Letters</i>, vol. 42,
    no. 10, Optica Publishing Group, 2017, pp. 1931–34, doi:<a href="https://doi.org/10.1364/OL.42.001931">10.1364/OL.42.001931</a>.
  short: J. Haase, S. Bagiante, H. Sigg, J. Van Bokhoven, Optics Letters 42 (2017)
    1931–1934.
date_created: 2018-12-11T11:47:51Z
date_published: 2017-05-15T00:00:00Z
date_updated: 2023-10-17T12:16:02Z
day: '15'
ddc:
- '530'
department:
- _id: NanoFab
doi: 10.1364/OL.42.001931
intvolume: '        42'
issue: '10'
language:
- iso: eng
month: '05'
oa_version: None
page: 1931 - 1934
publication: Optics Letters
publication_status: published
publisher: Optica Publishing Group
publist_id: '7048'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Surface enhanced infrared absorption of chemisorbed carbon monoxide using plasmonic
  nanoantennas
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 42
year: '2017'
...
---
_id: '676'
abstract:
- lang: eng
  text: The segregation of different cell types into distinct tissues is a fundamental
    process in metazoan development. Differences in cell adhesion and cortex tension
    are commonly thought to drive cell sorting by regulating tissue surface tension
    (TST). However, the role that differential TST plays in cell segregation within
    the developing embryo is as yet unclear. Here, we have analyzed the role of differential
    TST for germ layer progenitor cell segregation during zebrafish gastrulation.
    Contrary to previous observations that differential TST drives germ layer progenitor
    cell segregation in vitro, we show that germ layers display indistinguishable
    TST within the gastrulating embryo, arguing against differential TST driving germ
    layer progenitor cell segregation in vivo. We further show that the osmolarity
    of the interstitial fluid (IF) is an important factor that influences germ layer
    TST in vivo, and that lower osmolarity of the IF compared with standard cell culture
    medium can explain why germ layers display differential TST in culture but not
    in vivo. Finally, we show that directed migration of mesendoderm progenitors is
    required for germ layer progenitor cell segregation and germ layer formation.
article_processing_charge: No
article_type: original
author:
- first_name: Gabriel
  full_name: Krens, Gabriel
  id: 2B819732-F248-11E8-B48F-1D18A9856A87
  last_name: Krens
  orcid: 0000-0003-4761-5996
- first_name: Jim
  full_name: Veldhuis, Jim
  last_name: Veldhuis
- first_name: Vanessa
  full_name: Barone, Vanessa
  id: 419EECCC-F248-11E8-B48F-1D18A9856A87
  last_name: Barone
  orcid: 0000-0003-2676-3367
- first_name: Daniel
  full_name: Capek, Daniel
  id: 31C42484-F248-11E8-B48F-1D18A9856A87
  last_name: Capek
  orcid: 0000-0001-5199-9940
- first_name: Jean-Léon
  full_name: Maître, Jean-Léon
  id: 48F1E0D8-F248-11E8-B48F-1D18A9856A87
  last_name: Maître
  orcid: 0000-0002-3688-1474
- first_name: Wayne
  full_name: Brodland, Wayne
  last_name: Brodland
- first_name: Carl-Philipp J
  full_name: Heisenberg, Carl-Philipp J
  id: 39427864-F248-11E8-B48F-1D18A9856A87
  last_name: Heisenberg
  orcid: 0000-0002-0912-4566
citation:
  ama: Krens G, Veldhuis J, Barone V, et al. Interstitial fluid osmolarity modulates
    the action of differential tissue surface tension in progenitor cell segregation
    during gastrulation. <i>Development</i>. 2017;144(10):1798-1806. doi:<a href="https://doi.org/10.1242/dev.144964">10.1242/dev.144964</a>
  apa: Krens, G., Veldhuis, J., Barone, V., Capek, D., Maître, J.-L., Brodland, W.,
    &#38; Heisenberg, C.-P. J. (2017). Interstitial fluid osmolarity modulates the
    action of differential tissue surface tension in progenitor cell segregation during
    gastrulation. <i>Development</i>. Company of Biologists. <a href="https://doi.org/10.1242/dev.144964">https://doi.org/10.1242/dev.144964</a>
  chicago: Krens, Gabriel, Jim Veldhuis, Vanessa Barone, Daniel Capek, Jean-Léon Maître,
    Wayne Brodland, and Carl-Philipp J Heisenberg. “Interstitial Fluid Osmolarity
    Modulates the Action of Differential Tissue Surface Tension in Progenitor Cell
    Segregation during Gastrulation.” <i>Development</i>. Company of Biologists, 2017.
    <a href="https://doi.org/10.1242/dev.144964">https://doi.org/10.1242/dev.144964</a>.
  ieee: G. Krens <i>et al.</i>, “Interstitial fluid osmolarity modulates the action
    of differential tissue surface tension in progenitor cell segregation during gastrulation,”
    <i>Development</i>, vol. 144, no. 10. Company of Biologists, pp. 1798–1806, 2017.
  ista: Krens G, Veldhuis J, Barone V, Capek D, Maître J-L, Brodland W, Heisenberg
    C-PJ. 2017. Interstitial fluid osmolarity modulates the action of differential
    tissue surface tension in progenitor cell segregation during gastrulation. Development.
    144(10), 1798–1806.
  mla: Krens, Gabriel, et al. “Interstitial Fluid Osmolarity Modulates the Action
    of Differential Tissue Surface Tension in Progenitor Cell Segregation during Gastrulation.”
    <i>Development</i>, vol. 144, no. 10, Company of Biologists, 2017, pp. 1798–806,
    doi:<a href="https://doi.org/10.1242/dev.144964">10.1242/dev.144964</a>.
  short: G. Krens, J. Veldhuis, V. Barone, D. Capek, J.-L. Maître, W. Brodland, C.-P.J.
    Heisenberg, Development 144 (2017) 1798–1806.
date_created: 2018-12-11T11:47:52Z
date_published: 2017-05-15T00:00:00Z
date_updated: 2024-03-25T23:30:13Z
day: '15'
ddc:
- '570'
department:
- _id: Bio
- _id: CaHe
doi: 10.1242/dev.144964
external_id:
  pmid:
  - '28512197'
file:
- access_level: open_access
  checksum: bc25125fb664706cdf180e061429f91d
  content_type: application/pdf
  creator: dernst
  date_created: 2019-09-24T06:56:22Z
  date_updated: 2020-07-14T12:47:39Z
  file_id: '6905'
  file_name: 2017_Development_Krens.pdf
  file_size: 8194516
  relation: main_file
file_date_updated: 2020-07-14T12:47:39Z
has_accepted_license: '1'
intvolume: '       144'
issue: '10'
language:
- iso: eng
month: '05'
oa: 1
oa_version: Published Version
page: 1798 - 1806
pmid: 1
publication: Development
publication_identifier:
  issn:
  - '09501991'
publication_status: published
publisher: Company of Biologists
publist_id: '7047'
quality_controlled: '1'
related_material:
  record:
  - id: '961'
    relation: dissertation_contains
    status: public
  - id: '50'
    relation: dissertation_contains
    status: public
scopus_import: 1
status: public
title: Interstitial fluid osmolarity modulates the action of differential tissue surface
  tension in progenitor cell segregation during gastrulation
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 144
year: '2017'
...
---
_id: '677'
abstract:
- lang: eng
  text: The INO80 complex (INO80-C) is an evolutionarily conserved nucleosome remodeler
    that acts in transcription, replication, and genome stability. It is required
    for resistance against genotoxic agents and is involved in the repair of DNA double-strand
    breaks (DSBs) by homologous recombination (HR). However, the causes of the HR
    defect in INO80-C mutant cells are controversial. Here, we unite previous findings
    using a system to study HR with high spatial resolution in budding yeast. We find
    that INO80-C has at least two distinct functions during HR—DNA end resection and
    presynaptic filament formation. Importantly, the second function is linked to
    the histone variant H2A.Z. In the absence of H2A.Z, presynaptic filament formation
    and HR are restored in INO80-C-deficient mutants, suggesting that presynaptic
    filament formation is the crucial INO80-C function during HR.
author:
- first_name: Claudio
  full_name: Lademann, Claudio
  last_name: Lademann
- first_name: Jörg
  full_name: Renkawitz, Jörg
  id: 3F0587C8-F248-11E8-B48F-1D18A9856A87
  last_name: Renkawitz
  orcid: 0000-0003-2856-3369
- first_name: Boris
  full_name: Pfander, Boris
  last_name: Pfander
- first_name: Stefan
  full_name: Jentsch, Stefan
  last_name: Jentsch
citation:
  ama: Lademann C, Renkawitz J, Pfander B, Jentsch S. The INO80 complex removes H2A.Z
    to promote presynaptic filament formation during homologous recombination. <i>Cell
    Reports</i>. 2017;19(7):1294-1303. doi:<a href="https://doi.org/10.1016/j.celrep.2017.04.051">10.1016/j.celrep.2017.04.051</a>
  apa: Lademann, C., Renkawitz, J., Pfander, B., &#38; Jentsch, S. (2017). The INO80
    complex removes H2A.Z to promote presynaptic filament formation during homologous
    recombination. <i>Cell Reports</i>. Cell Press. <a href="https://doi.org/10.1016/j.celrep.2017.04.051">https://doi.org/10.1016/j.celrep.2017.04.051</a>
  chicago: Lademann, Claudio, Jörg Renkawitz, Boris Pfander, and Stefan Jentsch. “The
    INO80 Complex Removes H2A.Z to Promote Presynaptic Filament Formation during Homologous
    Recombination.” <i>Cell Reports</i>. Cell Press, 2017. <a href="https://doi.org/10.1016/j.celrep.2017.04.051">https://doi.org/10.1016/j.celrep.2017.04.051</a>.
  ieee: C. Lademann, J. Renkawitz, B. Pfander, and S. Jentsch, “The INO80 complex
    removes H2A.Z to promote presynaptic filament formation during homologous recombination,”
    <i>Cell Reports</i>, vol. 19, no. 7. Cell Press, pp. 1294–1303, 2017.
  ista: Lademann C, Renkawitz J, Pfander B, Jentsch S. 2017. The INO80 complex removes
    H2A.Z to promote presynaptic filament formation during homologous recombination.
    Cell Reports. 19(7), 1294–1303.
  mla: Lademann, Claudio, et al. “The INO80 Complex Removes H2A.Z to Promote Presynaptic
    Filament Formation during Homologous Recombination.” <i>Cell Reports</i>, vol.
    19, no. 7, Cell Press, 2017, pp. 1294–303, doi:<a href="https://doi.org/10.1016/j.celrep.2017.04.051">10.1016/j.celrep.2017.04.051</a>.
  short: C. Lademann, J. Renkawitz, B. Pfander, S. Jentsch, Cell Reports 19 (2017)
    1294–1303.
date_created: 2018-12-11T11:47:52Z
date_published: 2017-05-16T00:00:00Z
date_updated: 2021-01-12T08:08:57Z
day: '16'
ddc:
- '570'
department:
- _id: MiSi
doi: 10.1016/j.celrep.2017.04.051
file:
- access_level: open_access
  checksum: efc7287d9c6354983cb151880e9ad72a
  content_type: application/pdf
  creator: system
  date_created: 2018-12-12T10:15:48Z
  date_updated: 2020-07-14T12:47:40Z
  file_id: '5171'
  file_name: IST-2017-899-v1+1_1-s2.0-S2211124717305454-main.pdf
  file_size: 3005610
  relation: main_file
file_date_updated: 2020-07-14T12:47:40Z
has_accepted_license: '1'
intvolume: '        19'
issue: '7'
language:
- iso: eng
month: '05'
oa: 1
oa_version: Published Version
page: 1294 - 1303
publication: Cell Reports
publication_identifier:
  issn:
  - '22111247'
publication_status: published
publisher: Cell Press
publist_id: '7046'
pubrep_id: '899'
quality_controlled: '1'
scopus_import: 1
status: public
title: The INO80 complex removes H2A.Z to promote presynaptic filament formation during
  homologous recombination
tmp:
  image: /images/cc_by_nc_nd.png
  legal_code_url: https://creativecommons.org/licenses/by-nc-nd/4.0/legalcode
  name: Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International
    (CC BY-NC-ND 4.0)
  short: CC BY-NC-ND (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 19
year: '2017'
...
---
_id: '678'
abstract:
- lang: eng
  text: The seminal observation that mechanical signals can elicit changes in biochemical
    signalling within cells, a process commonly termed mechanosensation and mechanotransduction,
    has revolutionized our understanding of the role of cell mechanics in various
    fundamental biological processes, such as cell motility, adhesion, proliferation
    and differentiation. In this Review, we will discuss how the interplay and feedback
    between mechanical and biochemical signals control tissue morphogenesis and cell
    fate specification in embryonic development.
author:
- first_name: Nicoletta
  full_name: Petridou, Nicoletta
  id: 2A003F6C-F248-11E8-B48F-1D18A9856A87
  last_name: Petridou
  orcid: 0000-0002-8451-1195
- first_name: Zoltan P
  full_name: Spiro, Zoltan P
  id: 426AD026-F248-11E8-B48F-1D18A9856A87
  last_name: Spiro
- first_name: Carl-Philipp J
  full_name: Heisenberg, Carl-Philipp J
  id: 39427864-F248-11E8-B48F-1D18A9856A87
  last_name: Heisenberg
  orcid: 0000-0002-0912-4566
citation:
  ama: Petridou N, Spiro ZP, Heisenberg C-PJ. Multiscale force sensing in development.
    <i>Nature Cell Biology</i>. 2017;19(6):581-588. doi:<a href="https://doi.org/10.1038/ncb3524">10.1038/ncb3524</a>
  apa: Petridou, N., Spiro, Z. P., &#38; Heisenberg, C.-P. J. (2017). Multiscale force
    sensing in development. <i>Nature Cell Biology</i>. Nature Publishing Group. <a
    href="https://doi.org/10.1038/ncb3524">https://doi.org/10.1038/ncb3524</a>
  chicago: Petridou, Nicoletta, Zoltan P Spiro, and Carl-Philipp J Heisenberg. “Multiscale
    Force Sensing in Development.” <i>Nature Cell Biology</i>. Nature Publishing Group,
    2017. <a href="https://doi.org/10.1038/ncb3524">https://doi.org/10.1038/ncb3524</a>.
  ieee: N. Petridou, Z. P. Spiro, and C.-P. J. Heisenberg, “Multiscale force sensing
    in development,” <i>Nature Cell Biology</i>, vol. 19, no. 6. Nature Publishing
    Group, pp. 581–588, 2017.
  ista: Petridou N, Spiro ZP, Heisenberg C-PJ. 2017. Multiscale force sensing in development.
    Nature Cell Biology. 19(6), 581–588.
  mla: Petridou, Nicoletta, et al. “Multiscale Force Sensing in Development.” <i>Nature
    Cell Biology</i>, vol. 19, no. 6, Nature Publishing Group, 2017, pp. 581–88, doi:<a
    href="https://doi.org/10.1038/ncb3524">10.1038/ncb3524</a>.
  short: N. Petridou, Z.P. Spiro, C.-P.J. Heisenberg, Nature Cell Biology 19 (2017)
    581–588.
date_created: 2018-12-11T11:47:53Z
date_published: 2017-05-31T00:00:00Z
date_updated: 2021-01-12T08:08:59Z
day: '31'
department:
- _id: CaHe
doi: 10.1038/ncb3524
intvolume: '        19'
issue: '6'
language:
- iso: eng
month: '05'
oa_version: None
page: 581 - 588
project:
- _id: 25236028-B435-11E9-9278-68D0E5697425
  grant_number: ALTF534-2016
  name: The generation and function of anisotropic tissue tension in zebrafish epiboly
    (EMBO Fellowship)
publication: Nature Cell Biology
publication_identifier:
  issn:
  - '14657392'
publication_status: published
publisher: Nature Publishing Group
publist_id: '7040'
quality_controlled: '1'
scopus_import: 1
status: public
title: Multiscale force sensing in development
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 19
year: '2017'
...
---
_id: '679'
abstract:
- lang: eng
  text: Protective responses against pathogens require a rapid mobilization of resting
    neutrophils and the timely removal of activated ones. Neutrophils are exceptionally
    short-lived leukocytes, yet it remains unclear whether the lifespan of pathogen-engaged
    neutrophils is regulated differently from that in the circulating steady-state
    pool. Here, we have found that under homeostatic conditions, the mRNA-destabilizing
    protein tristetraprolin (TTP) regulates apoptosis and the numbers of activated
    infiltrating murine neutrophils but not neutrophil cellularity. Activated TTP-deficient
    neutrophils exhibited decreased apoptosis and enhanced accumulation at the infection
    site. In the context of myeloid-specific deletion of Ttp, the potentiation of
    neutrophil deployment protected mice against lethal soft tissue infection with
    Streptococcus pyogenes and prevented bacterial dissemination. Neutrophil transcriptome
    analysis revealed that decreased apoptosis of TTP-deficient neutrophils was specifically
    associated with elevated expression of myeloid cell leukemia 1 (Mcl1) but not
    other antiapoptotic B cell leukemia/ lymphoma 2 (Bcl2) family members. Higher
    Mcl1 expression resulted from stabilization of Mcl1 mRNA in the absence of TTP.
    The low apoptosis rate of infiltrating TTP-deficient neutrophils was comparable
    to that of transgenic Mcl1-overexpressing neutrophils. Our study demonstrates
    that posttranscriptional gene regulation by TTP schedules the termination of the
    antimicrobial engagement of neutrophils. The balancing role of TTP comes at the
    cost of an increased risk of bacterial infections.
acknowledgement: This work was supported by grants from the Austrian Science Fund
  (FWF) (P27538-B21, I1621-B22, and SFB 43, to PK); by funding from the European Union
  Seventh Framework Programme Marie Curie Initial Training Networks (FP7-PEOPLE-2012-ITN)
  for the project INBIONET (INfection BIOlogy Training NETwork under grant agreement
  PITN-GA-2012-316682; and by a joint research cluster initiative of the University
  of Vienna and the Medical University of Vienna.
author:
- first_name: Florian
  full_name: Ebner, Florian
  last_name: Ebner
- first_name: Vitaly
  full_name: Sedlyarov, Vitaly
  last_name: Sedlyarov
- first_name: Saren
  full_name: Tasciyan, Saren
  id: 4323B49C-F248-11E8-B48F-1D18A9856A87
  last_name: Tasciyan
  orcid: 0000-0003-1671-393X
- first_name: Masa
  full_name: Ivin, Masa
  last_name: Ivin
- first_name: Franz
  full_name: Kratochvill, Franz
  last_name: Kratochvill
- first_name: Nina
  full_name: Gratz, Nina
  last_name: Gratz
- first_name: Lukas
  full_name: Kenner, Lukas
  last_name: Kenner
- first_name: Andreas
  full_name: Villunger, Andreas
  last_name: Villunger
- first_name: Michael K
  full_name: Sixt, Michael K
  id: 41E9FBEA-F248-11E8-B48F-1D18A9856A87
  last_name: Sixt
  orcid: 0000-0002-6620-9179
- first_name: Pavel
  full_name: Kovarik, Pavel
  last_name: Kovarik
citation:
  ama: Ebner F, Sedlyarov V, Tasciyan S, et al. The RNA-binding protein tristetraprolin
    schedules apoptosis of pathogen-engaged neutrophils during bacterial infection.
    <i>The Journal of Clinical Investigation</i>. 2017;127(6):2051-2065. doi:<a href="https://doi.org/10.1172/JCI80631">10.1172/JCI80631</a>
  apa: Ebner, F., Sedlyarov, V., Tasciyan, S., Ivin, M., Kratochvill, F., Gratz, N.,
    … Kovarik, P. (2017). The RNA-binding protein tristetraprolin schedules apoptosis
    of pathogen-engaged neutrophils during bacterial infection. <i>The Journal of
    Clinical Investigation</i>. American Society for Clinical Investigation. <a href="https://doi.org/10.1172/JCI80631">https://doi.org/10.1172/JCI80631</a>
  chicago: Ebner, Florian, Vitaly Sedlyarov, Saren Tasciyan, Masa Ivin, Franz Kratochvill,
    Nina Gratz, Lukas Kenner, Andreas Villunger, Michael K Sixt, and Pavel Kovarik.
    “The RNA-Binding Protein Tristetraprolin Schedules Apoptosis of Pathogen-Engaged
    Neutrophils during Bacterial Infection.” <i>The Journal of Clinical Investigation</i>.
    American Society for Clinical Investigation, 2017. <a href="https://doi.org/10.1172/JCI80631">https://doi.org/10.1172/JCI80631</a>.
  ieee: F. Ebner <i>et al.</i>, “The RNA-binding protein tristetraprolin schedules
    apoptosis of pathogen-engaged neutrophils during bacterial infection,” <i>The
    Journal of Clinical Investigation</i>, vol. 127, no. 6. American Society for Clinical
    Investigation, pp. 2051–2065, 2017.
  ista: Ebner F, Sedlyarov V, Tasciyan S, Ivin M, Kratochvill F, Gratz N, Kenner L,
    Villunger A, Sixt MK, Kovarik P. 2017. The RNA-binding protein tristetraprolin
    schedules apoptosis of pathogen-engaged neutrophils during bacterial infection.
    The Journal of Clinical Investigation. 127(6), 2051–2065.
  mla: Ebner, Florian, et al. “The RNA-Binding Protein Tristetraprolin Schedules Apoptosis
    of Pathogen-Engaged Neutrophils during Bacterial Infection.” <i>The Journal of
    Clinical Investigation</i>, vol. 127, no. 6, American Society for Clinical Investigation,
    2017, pp. 2051–65, doi:<a href="https://doi.org/10.1172/JCI80631">10.1172/JCI80631</a>.
  short: F. Ebner, V. Sedlyarov, S. Tasciyan, M. Ivin, F. Kratochvill, N. Gratz, L.
    Kenner, A. Villunger, M.K. Sixt, P. Kovarik, The Journal of Clinical Investigation
    127 (2017) 2051–2065.
date_created: 2018-12-11T11:47:53Z
date_published: 2017-06-01T00:00:00Z
date_updated: 2024-03-25T23:30:12Z
day: '01'
department:
- _id: MiSi
doi: 10.1172/JCI80631
external_id:
  pmid:
  - '28504646'
intvolume: '       127'
issue: '6'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5451238/
month: '06'
oa: 1
oa_version: Submitted Version
page: 2051 - 2065
pmid: 1
project:
- _id: 25985A36-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: T00817-B21
  name: The biochemical basis of PAR polarization
- _id: 25E9AF9E-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: P27201-B22
  name: Revealing the mechanisms underlying drug interactions
publication: The Journal of Clinical Investigation
publication_identifier:
  issn:
  - '00219738'
publication_status: published
publisher: American Society for Clinical Investigation
publist_id: '7038'
quality_controlled: '1'
related_material:
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  - id: '12401'
    relation: dissertation_contains
    status: public
scopus_import: 1
status: public
title: The RNA-binding protein tristetraprolin schedules apoptosis of pathogen-engaged
  neutrophils during bacterial infection
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 127
year: '2017'
...
---
_id: '680'
abstract:
- lang: eng
  text: In order to respond reliably to specific features of their environment, sensory
    neurons need to integrate multiple incoming noisy signals. Crucially, they also
    need to compete for the interpretation of those signals with other neurons representing
    similar features. The form that this competition should take depends critically
    on the noise corrupting these signals. In this study we show that for the type
    of noise commonly observed in sensory systems, whose variance scales with the
    mean signal, sensory neurons should selectively divide their input signals by
    their predictions, suppressing ambiguous cues while amplifying others. Any change
    in the stimulus context alters which inputs are suppressed, leading to a deep
    dynamic reshaping of neural receptive fields going far beyond simple surround
    suppression. Paradoxically, these highly variable receptive fields go alongside
    and are in fact required for an invariant representation of external sensory features.
    In addition to offering a normative account of context-dependent changes in sensory
    responses, perceptual inference in the presence of signal-dependent noise accounts
    for ubiquitous features of sensory neurons such as divisive normalization, gain
    control and contrast dependent temporal dynamics.
article_number: e1005582
author:
- first_name: Matthew J
  full_name: Chalk, Matthew J
  id: 2BAAC544-F248-11E8-B48F-1D18A9856A87
  last_name: Chalk
  orcid: 0000-0001-7782-4436
- first_name: Paul
  full_name: Masset, Paul
  last_name: Masset
- first_name: Boris
  full_name: Gutkin, Boris
  last_name: Gutkin
- first_name: Sophie
  full_name: Denève, Sophie
  last_name: Denève
citation:
  ama: Chalk MJ, Masset P, Gutkin B, Denève S. Sensory noise predicts divisive reshaping
    of receptive fields. <i>PLoS Computational Biology</i>. 2017;13(6). doi:<a href="https://doi.org/10.1371/journal.pcbi.1005582">10.1371/journal.pcbi.1005582</a>
  apa: Chalk, M. J., Masset, P., Gutkin, B., &#38; Denève, S. (2017). Sensory noise
    predicts divisive reshaping of receptive fields. <i>PLoS Computational Biology</i>.
    Public Library of Science. <a href="https://doi.org/10.1371/journal.pcbi.1005582">https://doi.org/10.1371/journal.pcbi.1005582</a>
  chicago: Chalk, Matthew J, Paul Masset, Boris Gutkin, and Sophie Denève. “Sensory
    Noise Predicts Divisive Reshaping of Receptive Fields.” <i>PLoS Computational
    Biology</i>. Public Library of Science, 2017. <a href="https://doi.org/10.1371/journal.pcbi.1005582">https://doi.org/10.1371/journal.pcbi.1005582</a>.
  ieee: M. J. Chalk, P. Masset, B. Gutkin, and S. Denève, “Sensory noise predicts
    divisive reshaping of receptive fields,” <i>PLoS Computational Biology</i>, vol.
    13, no. 6. Public Library of Science, 2017.
  ista: Chalk MJ, Masset P, Gutkin B, Denève S. 2017. Sensory noise predicts divisive
    reshaping of receptive fields. PLoS Computational Biology. 13(6), e1005582.
  mla: Chalk, Matthew J., et al. “Sensory Noise Predicts Divisive Reshaping of Receptive
    Fields.” <i>PLoS Computational Biology</i>, vol. 13, no. 6, e1005582, Public Library
    of Science, 2017, doi:<a href="https://doi.org/10.1371/journal.pcbi.1005582">10.1371/journal.pcbi.1005582</a>.
  short: M.J. Chalk, P. Masset, B. Gutkin, S. Denève, PLoS Computational Biology 13
    (2017).
date_created: 2018-12-11T11:47:53Z
date_published: 2017-06-01T00:00:00Z
date_updated: 2023-02-23T14:10:54Z
day: '01'
ddc:
- '571'
department:
- _id: GaTk
doi: 10.1371/journal.pcbi.1005582
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month: '06'
oa: 1
oa_version: Published Version
publication: PLoS Computational Biology
publication_identifier:
  issn:
  - 1553734X
publication_status: published
publisher: Public Library of Science
publist_id: '7035'
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status: public
title: Sensory noise predicts divisive reshaping of receptive fields
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 13
year: '2017'
...
---
_id: '681'
abstract:
- lang: eng
  text: Two-player games on graphs provide the theoretical framework for many important
    problems such as reactive synthesis. While the traditional study of two-player
    zero-sum games has been extended to multi-player games with several notions of
    equilibria, they are decidable only for perfect-information games, whereas several
    applications require imperfect-information. In this paper we propose a new notion
    of equilibria, called doomsday equilibria, which is a strategy profile where all
    players satisfy their own objective, and if any coalition of players deviates
    and violates even one of the players' objective, then the objective of every player
    is violated. We present algorithms and complexity results for deciding the existence
    of doomsday equilibria for various classes of ω-regular objectives, both for imperfect-information
    games, and for perfect-information games. We provide optimal complexity bounds
    for imperfect-information games, and in most cases for perfect-information games.
article_processing_charge: No
article_type: original
arxiv: 1
author:
- first_name: Krishnendu
  full_name: Chatterjee, Krishnendu
  id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
  last_name: Chatterjee
  orcid: 0000-0002-4561-241X
- first_name: Laurent
  full_name: Doyen, Laurent
  last_name: Doyen
- first_name: Emmanuel
  full_name: Filiot, Emmanuel
  last_name: Filiot
- first_name: Jean
  full_name: Raskin, Jean
  last_name: Raskin
citation:
  ama: Chatterjee K, Doyen L, Filiot E, Raskin J. Doomsday equilibria for omega-regular
    games. <i>Information and Computation</i>. 2017;254:296-315. doi:<a href="https://doi.org/10.1016/j.ic.2016.10.012">10.1016/j.ic.2016.10.012</a>
  apa: Chatterjee, K., Doyen, L., Filiot, E., &#38; Raskin, J. (2017). Doomsday equilibria
    for omega-regular games. <i>Information and Computation</i>. Elsevier. <a href="https://doi.org/10.1016/j.ic.2016.10.012">https://doi.org/10.1016/j.ic.2016.10.012</a>
  chicago: Chatterjee, Krishnendu, Laurent Doyen, Emmanuel Filiot, and Jean Raskin.
    “Doomsday Equilibria for Omega-Regular Games.” <i>Information and Computation</i>.
    Elsevier, 2017. <a href="https://doi.org/10.1016/j.ic.2016.10.012">https://doi.org/10.1016/j.ic.2016.10.012</a>.
  ieee: K. Chatterjee, L. Doyen, E. Filiot, and J. Raskin, “Doomsday equilibria for
    omega-regular games,” <i>Information and Computation</i>, vol. 254. Elsevier,
    pp. 296–315, 2017.
  ista: Chatterjee K, Doyen L, Filiot E, Raskin J. 2017. Doomsday equilibria for omega-regular
    games. Information and Computation. 254, 296–315.
  mla: Chatterjee, Krishnendu, et al. “Doomsday Equilibria for Omega-Regular Games.”
    <i>Information and Computation</i>, vol. 254, Elsevier, 2017, pp. 296–315, doi:<a
    href="https://doi.org/10.1016/j.ic.2016.10.012">10.1016/j.ic.2016.10.012</a>.
  short: K. Chatterjee, L. Doyen, E. Filiot, J. Raskin, Information and Computation
    254 (2017) 296–315.
date_created: 2018-12-11T11:47:53Z
date_published: 2017-06-01T00:00:00Z
date_updated: 2023-02-21T16:06:02Z
day: '01'
department:
- _id: KrCh
doi: 10.1016/j.ic.2016.10.012
ec_funded: 1
external_id:
  arxiv:
  - '1311.3238'
intvolume: '       254'
language:
- iso: eng
main_file_link:
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  url: https://arxiv.org/abs/1311.3238
month: '06'
oa: 1
oa_version: Submitted Version
page: 296 - 315
project:
- _id: 2584A770-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: P 23499-N23
  name: Modern Graph Algorithmic Techniques in Formal Verification
- _id: 25863FF4-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: S11407
  name: Game Theory
- _id: 2581B60A-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '279307'
  name: 'Quantitative Graph Games: Theory and Applications'
- _id: 25681D80-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '291734'
  name: International IST Postdoc Fellowship Programme
publication: Information and Computation
publication_identifier:
  issn:
  - '08905401'
publication_status: published
publisher: Elsevier
publist_id: '7036'
quality_controlled: '1'
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scopus_import: '1'
status: public
title: Doomsday equilibria for omega-regular games
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 254
year: '2017'
...
---
_id: '682'
abstract:
- lang: eng
  text: Left-right asymmetry is a fundamental feature of higher-order brain structure;
    however, the molecular basis of brain asymmetry remains unclear. We recently identified
    structural and functional asymmetries in mouse hippocampal circuitry that result
    from the asymmetrical distribution of two distinct populations of pyramidal cell
    synapses that differ in the density of the NMDA receptor subunit GluRε2 (also
    known as NR2B, GRIN2B or GluN2B). By examining the synaptic distribution of ε2
    subunits, we previously found that β2-microglobulin-deficient mice, which lack
    cell surface expression of the vast majority of major histocompatibility complex
    class I (MHCI) proteins, do not exhibit circuit asymmetry. In the present study,
    we conducted electrophysiological and anatomical analyses on the hippocampal circuitry
    of mice with a knockout of the paired immunoglobulin-like receptor B (PirB), an
    MHCI receptor. As in β2-microglobulin-deficient mice, the PirB-deficient hippocampus
    lacked circuit asymmetries. This finding that MHCI loss-of-function mice and PirB
    knockout mice have identical phenotypes suggests that MHCI signals that produce
    hippocampal asymmetries are transduced through PirB. Our results provide evidence
    for a critical role of the MHCI/PirB signaling system in the generation of asymmetries
    in hippocampal circuitry.
article_number: e0179377
article_type: original
author:
- first_name: Hikari
  full_name: Ukai, Hikari
  last_name: Ukai
- first_name: Aiko
  full_name: Kawahara, Aiko
  last_name: Kawahara
- first_name: Keiko
  full_name: Hirayama, Keiko
  last_name: Hirayama
- first_name: Matthew J
  full_name: Case, Matthew J
  id: 44B7CA5A-F248-11E8-B48F-1D18A9856A87
  last_name: Case
- first_name: Shotaro
  full_name: Aino, Shotaro
  last_name: Aino
- first_name: Masahiro
  full_name: Miyabe, Masahiro
  last_name: Miyabe
- first_name: Ken
  full_name: Wakita, Ken
  last_name: Wakita
- first_name: Ryohei
  full_name: Oogi, Ryohei
  last_name: Oogi
- first_name: Michiyo
  full_name: Kasayuki, Michiyo
  last_name: Kasayuki
- first_name: Shihomi
  full_name: Kawashima, Shihomi
  last_name: Kawashima
- first_name: Shunichi
  full_name: Sugimoto, Shunichi
  last_name: Sugimoto
- first_name: Kanako
  full_name: Chikamatsu, Kanako
  last_name: Chikamatsu
- first_name: Noritaka
  full_name: Nitta, Noritaka
  last_name: Nitta
- first_name: Tsuneyuki
  full_name: Koga, Tsuneyuki
  last_name: Koga
- first_name: Ryuichi
  full_name: Shigemoto, Ryuichi
  id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
  last_name: Shigemoto
  orcid: 0000-0001-8761-9444
- first_name: Toshiyuki
  full_name: Takai, Toshiyuki
  last_name: Takai
- first_name: Isao
  full_name: Ito, Isao
  last_name: Ito
citation:
  ama: Ukai H, Kawahara A, Hirayama K, et al. PirB regulates asymmetries in hippocampal
    circuitry. <i>PLoS One</i>. 2017;12(6). doi:<a href="https://doi.org/10.1371/journal.pone.0179377">10.1371/journal.pone.0179377</a>
  apa: Ukai, H., Kawahara, A., Hirayama, K., Case, M. J., Aino, S., Miyabe, M., …
    Ito, I. (2017). PirB regulates asymmetries in hippocampal circuitry. <i>PLoS One</i>.
    Public Library of Science. <a href="https://doi.org/10.1371/journal.pone.0179377">https://doi.org/10.1371/journal.pone.0179377</a>
  chicago: Ukai, Hikari, Aiko Kawahara, Keiko Hirayama, Matthew J Case, Shotaro Aino,
    Masahiro Miyabe, Ken Wakita, et al. “PirB Regulates Asymmetries in Hippocampal
    Circuitry.” <i>PLoS One</i>. Public Library of Science, 2017. <a href="https://doi.org/10.1371/journal.pone.0179377">https://doi.org/10.1371/journal.pone.0179377</a>.
  ieee: H. Ukai <i>et al.</i>, “PirB regulates asymmetries in hippocampal circuitry,”
    <i>PLoS One</i>, vol. 12, no. 6. Public Library of Science, 2017.
  ista: Ukai H, Kawahara A, Hirayama K, Case MJ, Aino S, Miyabe M, Wakita K, Oogi
    R, Kasayuki M, Kawashima S, Sugimoto S, Chikamatsu K, Nitta N, Koga T, Shigemoto
    R, Takai T, Ito I. 2017. PirB regulates asymmetries in hippocampal circuitry.
    PLoS One. 12(6), e0179377.
  mla: Ukai, Hikari, et al. “PirB Regulates Asymmetries in Hippocampal Circuitry.”
    <i>PLoS One</i>, vol. 12, no. 6, e0179377, Public Library of Science, 2017, doi:<a
    href="https://doi.org/10.1371/journal.pone.0179377">10.1371/journal.pone.0179377</a>.
  short: H. Ukai, A. Kawahara, K. Hirayama, M.J. Case, S. Aino, M. Miyabe, K. Wakita,
    R. Oogi, M. Kasayuki, S. Kawashima, S. Sugimoto, K. Chikamatsu, N. Nitta, T. Koga,
    R. Shigemoto, T. Takai, I. Ito, PLoS One 12 (2017).
date_created: 2018-12-11T11:47:54Z
date_published: 2017-06-01T00:00:00Z
date_updated: 2024-03-25T23:30:07Z
day: '01'
ddc:
- '571'
department:
- _id: RySh
doi: 10.1371/journal.pone.0179377
file:
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- iso: eng
month: '06'
oa: 1
oa_version: Published Version
publication: PLoS One
publication_identifier:
  issn:
  - '19326203'
publication_status: published
publisher: Public Library of Science
publist_id: '7034'
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related_material:
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    status: public
scopus_import: 1
status: public
title: PirB regulates asymmetries in hippocampal circuitry
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 12
year: '2017'
...
---
_id: '683'
abstract:
- lang: eng
  text: 'Given a triangulation of a point set in the plane, a flip deletes an edge
    e whose removal leaves a convex quadrilateral, and replaces e by the opposite
    diagonal of the quadrilateral. It is well known that any triangulation of a point
    set can be reconfigured to any other triangulation by some sequence of flips.
    We explore this question in the setting where each edge of a triangulation has
    a label, and a flip transfers the label of the removed edge to the new edge. It
    is not true that every labelled triangulation of a point set can be reconfigured
    to every other labelled triangulation via a sequence of flips, but we characterize
    when this is possible. There is an obvious necessary condition: for each label
    l, if edge e has label l in the first triangulation and edge f has label l in
    the second triangulation, then there must be some sequence of flips that moves
    label l from e to f, ignoring all other labels. Bose, Lubiw, Pathak and Verdonschot
    formulated the Orbit Conjecture, which states that this necessary condition is
    also sufficient, i.e. that all labels can be simultaneously mapped to their destination
    if and only if each label individually can be mapped to its destination. We prove
    this conjecture. Furthermore, we give a polynomial-time algorithm to find a sequence
    of flips to reconfigure one labelled triangulation to another, if such a sequence
    exists, and we prove an upper bound of O(n7) on the length of the flip sequence.
    Our proof uses the topological result that the sets of pairwise non-crossing edges
    on a planar point set form a simplicial complex that is homeomorphic to a high-dimensional
    ball (this follows from a result of Orden and Santos; we give a different proof
    based on a shelling argument). The dual cell complex of this simplicial ball,
    called the flip complex, has the usual flip graph as its 1-skeleton. We use properties
    of the 2-skeleton of the flip complex to prove the Orbit Conjecture.'
alternative_title:
- LIPIcs
article_number: '49'
author:
- first_name: Anna
  full_name: Lubiw, Anna
  last_name: Lubiw
- first_name: Zuzana
  full_name: Masárová, Zuzana
  id: 45CFE238-F248-11E8-B48F-1D18A9856A87
  last_name: Masárová
  orcid: 0000-0002-6660-1322
- first_name: Uli
  full_name: Wagner, Uli
  id: 36690CA2-F248-11E8-B48F-1D18A9856A87
  last_name: Wagner
  orcid: 0000-0002-1494-0568
citation:
  ama: 'Lubiw A, Masárová Z, Wagner U. A proof of the orbit conjecture for flipping
    edge labelled triangulations. In: Vol 77. Schloss Dagstuhl - Leibniz-Zentrum für
    Informatik; 2017. doi:<a href="https://doi.org/10.4230/LIPIcs.SoCG.2017.49">10.4230/LIPIcs.SoCG.2017.49</a>'
  apa: 'Lubiw, A., Masárová, Z., &#38; Wagner, U. (2017). A proof of the orbit conjecture
    for flipping edge labelled triangulations (Vol. 77). Presented at the SoCG: Symposium
    on Computational Geometry, Brisbane, Australia: Schloss Dagstuhl - Leibniz-Zentrum
    für Informatik. <a href="https://doi.org/10.4230/LIPIcs.SoCG.2017.49">https://doi.org/10.4230/LIPIcs.SoCG.2017.49</a>'
  chicago: Lubiw, Anna, Zuzana Masárová, and Uli Wagner. “A Proof of the Orbit Conjecture
    for Flipping Edge Labelled Triangulations,” Vol. 77. Schloss Dagstuhl - Leibniz-Zentrum
    für Informatik, 2017. <a href="https://doi.org/10.4230/LIPIcs.SoCG.2017.49">https://doi.org/10.4230/LIPIcs.SoCG.2017.49</a>.
  ieee: 'A. Lubiw, Z. Masárová, and U. Wagner, “A proof of the orbit conjecture for
    flipping edge labelled triangulations,” presented at the SoCG: Symposium on Computational
    Geometry, Brisbane, Australia, 2017, vol. 77.'
  ista: 'Lubiw A, Masárová Z, Wagner U. 2017. A proof of the orbit conjecture for
    flipping edge labelled triangulations. SoCG: Symposium on Computational Geometry,
    LIPIcs, vol. 77, 49.'
  mla: Lubiw, Anna, et al. <i>A Proof of the Orbit Conjecture for Flipping Edge Labelled
    Triangulations</i>. Vol. 77, 49, Schloss Dagstuhl - Leibniz-Zentrum für Informatik,
    2017, doi:<a href="https://doi.org/10.4230/LIPIcs.SoCG.2017.49">10.4230/LIPIcs.SoCG.2017.49</a>.
  short: A. Lubiw, Z. Masárová, U. Wagner, in:, Schloss Dagstuhl - Leibniz-Zentrum
    für Informatik, 2017.
conference:
  end_date: 2017-07-07
  location: Brisbane, Australia
  name: 'SoCG: Symposium on Computational Geometry'
  start_date: 2017-07-04
date_created: 2018-12-11T11:47:54Z
date_published: 2017-06-01T00:00:00Z
date_updated: 2023-09-05T15:01:43Z
day: '01'
ddc:
- '514'
- '516'
department:
- _id: UlWa
doi: 10.4230/LIPIcs.SoCG.2017.49
file:
- access_level: open_access
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  creator: system
  date_created: 2018-12-12T10:17:12Z
  date_updated: 2020-07-14T12:47:41Z
  file_id: '5265'
  file_name: IST-2017-896-v1+1_LIPIcs-SoCG-2017-49.pdf
  file_size: 710007
  relation: main_file
file_date_updated: 2020-07-14T12:47:41Z
has_accepted_license: '1'
intvolume: '        77'
language:
- iso: eng
month: '06'
oa: 1
oa_version: Published Version
publication_status: published
publisher: Schloss Dagstuhl - Leibniz-Zentrum für Informatik
publist_id: '7033'
pubrep_id: '896'
quality_controlled: '1'
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scopus_import: 1
status: public
title: A proof of the orbit conjecture for flipping edge labelled triangulations
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: conference
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 77
year: '2017'
...
---
_id: '684'
abstract:
- lang: eng
  text: We generalize winning conditions in two-player games by adding a structural
    acceptance condition called obligations. Obligations are orthogonal to the linear
    winning conditions that define whether a play is winning. Obligations are a declaration
    that player 0 can achieve a certain value from a configuration. If the obligation
    is met, the value of that configuration for player 0 is 1. We define the value
    in such games and show that obligation games are determined. For Markov chains
    with Borel objectives and obligations, and finite turn-based stochastic parity
    games with obligations we give an alternative and simpler characterization of
    the value function. Based on this simpler definition we show that the decision
    problem of winning finite turn-based stochastic parity games with obligations
    is in NP∩co-NP. We also show that obligation games provide a game framework for
    reasoning about p-automata. © 2017 The Association for Symbolic Logic.
article_processing_charge: No
author:
- first_name: Krishnendu
  full_name: Chatterjee, Krishnendu
  id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
  last_name: Chatterjee
  orcid: 0000-0002-4561-241X
- first_name: Nir
  full_name: Piterman, Nir
  last_name: Piterman
citation:
  ama: Chatterjee K, Piterman N. Obligation blackwell games and p-automata. <i>Journal
    of Symbolic Logic</i>. 2017;82(2):420-452. doi:<a href="https://doi.org/10.1017/jsl.2016.71">10.1017/jsl.2016.71</a>
  apa: Chatterjee, K., &#38; Piterman, N. (2017). Obligation blackwell games and p-automata.
    <i>Journal of Symbolic Logic</i>. Cambridge University Press. <a href="https://doi.org/10.1017/jsl.2016.71">https://doi.org/10.1017/jsl.2016.71</a>
  chicago: Chatterjee, Krishnendu, and Nir Piterman. “Obligation Blackwell Games and
    P-Automata.” <i>Journal of Symbolic Logic</i>. Cambridge University Press, 2017.
    <a href="https://doi.org/10.1017/jsl.2016.71">https://doi.org/10.1017/jsl.2016.71</a>.
  ieee: K. Chatterjee and N. Piterman, “Obligation blackwell games and p-automata,”
    <i>Journal of Symbolic Logic</i>, vol. 82, no. 2. Cambridge University Press,
    pp. 420–452, 2017.
  ista: Chatterjee K, Piterman N. 2017. Obligation blackwell games and p-automata.
    Journal of Symbolic Logic. 82(2), 420–452.
  mla: Chatterjee, Krishnendu, and Nir Piterman. “Obligation Blackwell Games and P-Automata.”
    <i>Journal of Symbolic Logic</i>, vol. 82, no. 2, Cambridge University Press,
    2017, pp. 420–52, doi:<a href="https://doi.org/10.1017/jsl.2016.71">10.1017/jsl.2016.71</a>.
  short: K. Chatterjee, N. Piterman, Journal of Symbolic Logic 82 (2017) 420–452.
date_created: 2018-12-11T11:47:54Z
date_published: 2017-06-01T00:00:00Z
date_updated: 2021-04-16T12:10:53Z
day: '01'
department:
- _id: KrCh
doi: 10.1017/jsl.2016.71
intvolume: '        82'
issue: '2'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1206.5174
month: '06'
oa: 1
oa_version: Submitted Version
page: 420 - 452
publication: Journal of Symbolic Logic
publication_identifier:
  eissn:
  - 1943-5886
  issn:
  - 0022-4812
publication_status: published
publisher: Cambridge University Press
publist_id: '7026'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Obligation blackwell games and p-automata
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 82
year: '2017'
...
---
_id: '6841'
abstract:
- lang: eng
  text: In classical machine learning, regression is treated as a black box process
    of identifying a suitable function from a hypothesis set without attempting to
    gain insight into the mechanism connecting inputs and outputs. In the natural
    sciences, however, finding an interpretable function for a phenomenon is the prime
    goal as it allows to understand and generalize results. This paper proposes a
    novel type of function learning network, called equation learner (EQL), that can
    learn analytical expressions and is able to extrapolate to unseen domains. It
    is implemented as an end-to-end differentiable feed-forward network and allows
    for efficient gradient based training. Due to sparsity regularization concise
    interpretable expressions can be obtained. Often the true underlying source expression
    is identified.
arxiv: 1
author:
- first_name: Georg S
  full_name: Martius, Georg S
  id: 3A276B68-F248-11E8-B48F-1D18A9856A87
  last_name: Martius
- first_name: Christoph
  full_name: Lampert, Christoph
  id: 40C20FD2-F248-11E8-B48F-1D18A9856A87
  last_name: Lampert
  orcid: 0000-0001-8622-7887
citation:
  ama: 'Martius GS, Lampert C. Extrapolation and learning equations. In: <i>5th International
    Conference on Learning Representations, ICLR 2017 - Workshop Track Proceedings</i>.
    International Conference on Learning Representations; 2017.'
  apa: 'Martius, G. S., &#38; Lampert, C. (2017). Extrapolation and learning equations.
    In <i>5th International Conference on Learning Representations, ICLR 2017 - Workshop
    Track Proceedings</i>. Toulon, France: International Conference on Learning Representations.'
  chicago: Martius, Georg S, and Christoph Lampert. “Extrapolation and Learning Equations.”
    In <i>5th International Conference on Learning Representations, ICLR 2017 - Workshop
    Track Proceedings</i>. International Conference on Learning Representations, 2017.
  ieee: G. S. Martius and C. Lampert, “Extrapolation and learning equations,” in <i>5th
    International Conference on Learning Representations, ICLR 2017 - Workshop Track
    Proceedings</i>, Toulon, France, 2017.
  ista: 'Martius GS, Lampert C. 2017. Extrapolation and learning equations. 5th International
    Conference on Learning Representations, ICLR 2017 - Workshop Track Proceedings.
    ICLR: International Conference on Learning Representations.'
  mla: Martius, Georg S., and Christoph Lampert. “Extrapolation and Learning Equations.”
    <i>5th International Conference on Learning Representations, ICLR 2017 - Workshop
    Track Proceedings</i>, International Conference on Learning Representations, 2017.
  short: G.S. Martius, C. Lampert, in:, 5th International Conference on Learning Representations,
    ICLR 2017 - Workshop Track Proceedings, International Conference on Learning Representations,
    2017.
conference:
  end_date: 2017-04-26
  location: Toulon, France
  name: 'ICLR: International Conference on Learning Representations'
  start_date: 2017-04-24
date_created: 2019-09-01T22:01:00Z
date_published: 2017-02-21T00:00:00Z
date_updated: 2021-01-12T08:09:17Z
day: '21'
department:
- _id: ChLa
ec_funded: 1
external_id:
  arxiv:
  - '1610.02995'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1610.02995
month: '02'
oa: 1
oa_version: Preprint
project:
- _id: 2532554C-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '308036'
  name: Lifelong Learning of Visual Scene Understanding
publication: 5th International Conference on Learning Representations, ICLR 2017 -
  Workshop Track Proceedings
publication_status: published
publisher: International Conference on Learning Representations
quality_controlled: '1'
scopus_import: 1
status: public
title: Extrapolation and learning equations
type: conference
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
year: '2017'
...
