---
_id: '93'
abstract:
- lang: eng
text: An electro-optomechanical device capable of microwave-to-optics conversion
has recently been demonstrated, with the vision of enabling optical networks of
superconducting qubits. Here we present an improved converter design that uses
a three-dimensional microwave cavity for coupling between the microwave transmission
line and an integrated LC resonator on the converter chip. The new design simplifies
the optical assembly and decouples it from the microwave part of the setup. Experimental
demonstrations show that the modular device assembly allows us to flexibly tune
the microwave coupling to the converter chip while maintaining small loss. We
also find that electromechanical experiments are not impacted by the additional
microwave cavity. Our design is compatible with a high-finesse optical cavity
and will improve optical performance.
article_number: '094701'
arxiv: 1
author:
- first_name: Tim
full_name: Menke, Tim
last_name: Menke
- first_name: Peter
full_name: Burns, Peter
last_name: Burns
- first_name: Andrew P
full_name: Higginbotham, Andrew P
id: 4AD6785A-F248-11E8-B48F-1D18A9856A87
last_name: Higginbotham
orcid: 0000-0003-2607-2363
- first_name: N S
full_name: Kampel, N S
last_name: Kampel
- first_name: Robert
full_name: Peterson, Robert
last_name: Peterson
- first_name: Katarina
full_name: Cicak, Katarina
last_name: Cicak
- first_name: Raymond
full_name: Simmonds, Raymond
last_name: Simmonds
- first_name: Cindy
full_name: Regal, Cindy
last_name: Regal
- first_name: Konrad
full_name: Lehnert, Konrad
last_name: Lehnert
citation:
ama: Menke T, Burns P, Higginbotham AP, et al. Reconfigurable re-entrant cavity
for wireless coupling to an electro-optomechanical device. Review of Scientific
Instruments. 2017;88(9). doi:10.1063/1.5000973
apa: Menke, T., Burns, P., Higginbotham, A. P., Kampel, N. S., Peterson, R., Cicak,
K., … Lehnert, K. (2017). Reconfigurable re-entrant cavity for wireless coupling
to an electro-optomechanical device. Review of Scientific Instruments.
American Institute of Physics. https://doi.org/10.1063/1.5000973
chicago: Menke, Tim, Peter Burns, Andrew P Higginbotham, N S Kampel, Robert Peterson,
Katarina Cicak, Raymond Simmonds, Cindy Regal, and Konrad Lehnert. “Reconfigurable
Re-Entrant Cavity for Wireless Coupling to an Electro-Optomechanical Device.”
Review of Scientific Instruments. American Institute of Physics, 2017.
https://doi.org/10.1063/1.5000973.
ieee: T. Menke et al., “Reconfigurable re-entrant cavity for wireless coupling
to an electro-optomechanical device,” Review of Scientific Instruments,
vol. 88, no. 9. American Institute of Physics, 2017.
ista: Menke T, Burns P, Higginbotham AP, Kampel NS, Peterson R, Cicak K, Simmonds
R, Regal C, Lehnert K. 2017. Reconfigurable re-entrant cavity for wireless coupling
to an electro-optomechanical device. Review of Scientific Instruments. 88(9),
094701.
mla: Menke, Tim, et al. “Reconfigurable Re-Entrant Cavity for Wireless Coupling
to an Electro-Optomechanical Device.” Review of Scientific Instruments,
vol. 88, no. 9, 094701, American Institute of Physics, 2017, doi:10.1063/1.5000973.
short: T. Menke, P. Burns, A.P. Higginbotham, N.S. Kampel, R. Peterson, K. Cicak,
R. Simmonds, C. Regal, K. Lehnert, Review of Scientific Instruments 88 (2017).
date_created: 2018-12-11T11:44:35Z
date_published: 2017-09-08T00:00:00Z
date_updated: 2021-01-12T08:21:59Z
day: '08'
doi: 10.1063/1.5000973
extern: '1'
external_id:
arxiv:
- '1703.06470'
pmid:
- '28964202'
intvolume: ' 88'
issue: '9'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://arxiv.org/abs/1703.06470
month: '09'
oa: 1
oa_version: Preprint
pmid: 1
publication: Review of Scientific Instruments
publication_status: published
publisher: American Institute of Physics
publist_id: '7961'
quality_controlled: '1'
status: public
title: Reconfigurable re-entrant cavity for wireless coupling to an electro-optomechanical
device
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 88
year: '2017'
...
---
_id: '934'
abstract:
- lang: eng
text: During puberty, the mouse mammary gland develops into a highly branched epithelial
network. Owing to the absence of exclusive stem cell markers, the location, multiplicity,
dynamics and fate of mammary stem cells (MaSCs), which drive branching morphogenesis,
are unknown. Here we show that morphogenesis is driven by proliferative terminal
end buds that terminate or bifurcate with near equal probability, in a stochastic
and time-invariant manner, leading to a heterogeneous epithelial network. We show
that the majority of terminal end bud cells function as highly proliferative,
lineage-committed MaSCs that are heterogeneous in their expression profile and
short-term contribution to ductal extension. Yet, through cell rearrangements
during terminal end bud bifurcation, each MaSC is able to contribute actively
to long-term growth. Our study shows that the behaviour of MaSCs is not directly
linked to a single expression profile. Instead, morphogenesis relies upon lineage-restricted
heterogeneous MaSC populations that function as single equipotent pools in the
long term.
author:
- first_name: Colinda
full_name: Scheele, Colinda
last_name: Scheele
- first_name: Edouard B
full_name: Hannezo, Edouard B
id: 3A9DB764-F248-11E8-B48F-1D18A9856A87
last_name: Hannezo
orcid: 0000-0001-6005-1561
- first_name: Mauro
full_name: Muraro, Mauro
last_name: Muraro
- first_name: Anoek
full_name: Zomer, Anoek
last_name: Zomer
- first_name: Nathalia
full_name: Langedijk, Nathalia
last_name: Langedijk
- first_name: Alexander
full_name: Van Oudenaarden, Alexander
last_name: Van Oudenaarden
- first_name: Benjamin
full_name: Simons, Benjamin
last_name: Simons
- first_name: Jacco
full_name: Van Rheenen, Jacco
last_name: Van Rheenen
citation:
ama: Scheele C, Hannezo EB, Muraro M, et al. Identity and dynamics of mammary stem
cells during branching morphogenesis. Nature. 2017;542(7641):313-317. doi:10.1038/nature21046
apa: Scheele, C., Hannezo, E. B., Muraro, M., Zomer, A., Langedijk, N., Van Oudenaarden,
A., … Van Rheenen, J. (2017). Identity and dynamics of mammary stem cells during
branching morphogenesis. Nature. Nature Publishing Group. https://doi.org/10.1038/nature21046
chicago: Scheele, Colinda, Edouard B Hannezo, Mauro Muraro, Anoek Zomer, Nathalia
Langedijk, Alexander Van Oudenaarden, Benjamin Simons, and Jacco Van Rheenen.
“Identity and Dynamics of Mammary Stem Cells during Branching Morphogenesis.”
Nature. Nature Publishing Group, 2017. https://doi.org/10.1038/nature21046.
ieee: C. Scheele et al., “Identity and dynamics of mammary stem cells during
branching morphogenesis,” Nature, vol. 542, no. 7641. Nature Publishing
Group, pp. 313–317, 2017.
ista: Scheele C, Hannezo EB, Muraro M, Zomer A, Langedijk N, Van Oudenaarden A,
Simons B, Van Rheenen J. 2017. Identity and dynamics of mammary stem cells during
branching morphogenesis. Nature. 542(7641), 313–317.
mla: Scheele, Colinda, et al. “Identity and Dynamics of Mammary Stem Cells during
Branching Morphogenesis.” Nature, vol. 542, no. 7641, Nature Publishing
Group, 2017, pp. 313–17, doi:10.1038/nature21046.
short: C. Scheele, E.B. Hannezo, M. Muraro, A. Zomer, N. Langedijk, A. Van Oudenaarden,
B. Simons, J. Van Rheenen, Nature 542 (2017) 313–317.
date_created: 2018-12-11T11:49:17Z
date_published: 2017-02-16T00:00:00Z
date_updated: 2021-01-12T08:22:01Z
day: '16'
doi: 10.1038/nature21046
extern: '1'
intvolume: ' 542'
issue: '7641'
language:
- iso: eng
month: '02'
oa_version: None
page: 313 - 317
publication: Nature
publication_identifier:
issn:
- '00280836'
publication_status: published
publisher: Nature Publishing Group
publist_id: '6505'
quality_controlled: '1'
status: public
title: Identity and dynamics of mammary stem cells during branching morphogenesis
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 542
year: '2017'
...
---
_id: '936'
abstract:
- lang: eng
text: Homeostatic replacement of epithelial cells from basal precursors is a multistep
process involving progenitor cell specification, radial intercalation and, finally,
apical surface emergence. Recent data demonstrate that actin-based pushing under
the control of the formin protein Fmn1 drives apical emergence in nascent multiciliated
epithelial cells (MCCs), but little else is known about this actin network or
the control of Fmn1. Here, we explore the role of the small GTPase RhoA in MCC
apical emergence. Disruption of RhoA function reduced the rate of apical surface
expansion and decreased the final size of the apical domain. Analysis of cell
shapes suggests that RhoA alters the balance of forces exerted on the MCC apical
surface. Finally, quantitative time-lapse imaging and fluorescence recovery after
photobleaching studies argue that RhoA works in concert with Fmn1 to control assembly
of the specialized apical actin network in MCCs. These data provide new molecular
insights into epithelial apical surface assembly and could also shed light on
mechanisms of apical lumen formation
author:
- first_name: Jakub
full_name: Sedzinski, Jakub
last_name: Sedzinski
- first_name: Edouard B
full_name: Hannezo, Edouard B
id: 3A9DB764-F248-11E8-B48F-1D18A9856A87
last_name: Hannezo
orcid: 0000-0001-6005-1561
- first_name: Fan
full_name: Tu, Fan
last_name: Tu
- first_name: Maté
full_name: Biro, Maté
last_name: Biro
- first_name: John
full_name: Wallingford, John
last_name: Wallingford
citation:
ama: Sedzinski J, Hannezo EB, Tu F, Biro M, Wallingford J. RhoA regulates actin
network dynamics during apical surface emergence in multiciliated epithelial cells
. Journal of Cell Science. 2017;130(5). doi:10.1242/jcs.202234
apa: Sedzinski, J., Hannezo, E. B., Tu, F., Biro, M., & Wallingford, J. (2017).
RhoA regulates actin network dynamics during apical surface emergence in multiciliated
epithelial cells . Journal of Cell Science. Company of Biologists. https://doi.org/10.1242/jcs.202234
chicago: Sedzinski, Jakub, Edouard B Hannezo, Fan Tu, Maté Biro, and John Wallingford.
“RhoA Regulates Actin Network Dynamics during Apical Surface Emergence in Multiciliated
Epithelial Cells .” Journal of Cell Science. Company of Biologists, 2017.
https://doi.org/10.1242/jcs.202234.
ieee: J. Sedzinski, E. B. Hannezo, F. Tu, M. Biro, and J. Wallingford, “RhoA regulates
actin network dynamics during apical surface emergence in multiciliated epithelial
cells ,” Journal of Cell Science, vol. 130, no. 5. Company of Biologists,
2017.
ista: Sedzinski J, Hannezo EB, Tu F, Biro M, Wallingford J. 2017. RhoA regulates
actin network dynamics during apical surface emergence in multiciliated epithelial
cells . Journal of Cell Science. 130(5).
mla: Sedzinski, Jakub, et al. “RhoA Regulates Actin Network Dynamics during Apical
Surface Emergence in Multiciliated Epithelial Cells .” Journal of Cell Science,
vol. 130, no. 5, Company of Biologists, 2017, doi:10.1242/jcs.202234.
short: J. Sedzinski, E.B. Hannezo, F. Tu, M. Biro, J. Wallingford, Journal of Cell
Science 130 (2017).
date_created: 2018-12-11T11:49:17Z
date_published: 2017-01-01T00:00:00Z
date_updated: 2021-01-12T08:22:02Z
day: '01'
doi: 10.1242/jcs.202234
extern: '1'
intvolume: ' 130'
issue: '5'
language:
- iso: eng
month: '01'
oa_version: None
publication: Journal of Cell Science
publication_status: published
publisher: Company of Biologists
publist_id: '6507'
quality_controlled: '1'
status: public
title: 'RhoA regulates actin network dynamics during apical surface emergence in multiciliated
epithelial cells '
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 130
year: '2017'
...
---
_id: '937'
abstract:
- lang: eng
text: During epithelial cytokinesis, the remodelling of adhesive cell-cell contacts
between the dividing cell and its neighbours has profound implications for the
integrity, arrangement and morphogenesis of proliferative tissues. In both vertebrates
and invertebrates, this remodelling requires the activity of non-muscle myosin
II (MyoII) in the interphasic cells neighbouring the dividing cell. However, the
mechanisms that coordinate cytokinesis and MyoII activity in the neighbours are
unknown. Here we show that in the Drosophila notum epithelium, each cell division
is associated with a mechanosensing and transmission event that controls MyoII
dynamics in neighbouring cells. We find that the ring pulling forces promote local
junction elongation, which results in local E-cadherin dilution at the ingressing
adherens junction. In turn, the reduction in E-cadherin concentration and the
contractility of the neighbouring cells promote self-organized actomyosin flows,
ultimately leading to accumulation of MyoII at the base of the ingressing junction.
Although force transduction has been extensively studied in the context of adherens
junction reinforcement to stabilize adhesive cell-cell contacts, we propose an
alternative mechanosensing mechanism that coordinates actomyosin dynamics between
epithelial cells and sustains the remodelling of the adherens junction in response
to mechanical forces.
author:
- first_name: Diana
full_name: Pinheiro, Diana
last_name: Pinheiro
- first_name: Edouard B
full_name: Hannezo, Edouard B
id: 3A9DB764-F248-11E8-B48F-1D18A9856A87
last_name: Hannezo
orcid: 0000-0001-6005-1561
- first_name: Sophie
full_name: Herszterg, Sophie
last_name: Herszterg
- first_name: Floris
full_name: Bosveld, Floris
last_name: Bosveld
- first_name: Isabelle
full_name: Gaugué, Isabelle
last_name: Gaugué
- first_name: Maria
full_name: Balakireva, Maria
last_name: Balakireva
- first_name: Zhimin
full_name: Wang, Zhimin
last_name: Wang
- first_name: Inês
full_name: Cristo, Inês
last_name: Cristo
- first_name: Stéphane
full_name: Rigaud, Stéphane
last_name: Rigaud
- first_name: Olga
full_name: Markova, Olga
last_name: Markova
- first_name: Yohanns
full_name: Bellaïche, Yohanns
last_name: Bellaïche
citation:
ama: Pinheiro D, Hannezo EB, Herszterg S, et al. Transmission of cytokinesis forces
via E cadherin dilution and actomyosin flows. Nature. 2017;545(7652):103-107.
doi:10.1038/nature22041
apa: Pinheiro, D., Hannezo, E. B., Herszterg, S., Bosveld, F., Gaugué, I., Balakireva,
M., … Bellaïche, Y. (2017). Transmission of cytokinesis forces via E cadherin
dilution and actomyosin flows. Nature. Nature Publishing Group. https://doi.org/10.1038/nature22041
chicago: Pinheiro, Diana, Edouard B Hannezo, Sophie Herszterg, Floris Bosveld, Isabelle
Gaugué, Maria Balakireva, Zhimin Wang, et al. “Transmission of Cytokinesis Forces
via E Cadherin Dilution and Actomyosin Flows.” Nature. Nature Publishing
Group, 2017. https://doi.org/10.1038/nature22041.
ieee: D. Pinheiro et al., “Transmission of cytokinesis forces via E cadherin
dilution and actomyosin flows,” Nature, vol. 545, no. 7652. Nature Publishing
Group, pp. 103–107, 2017.
ista: Pinheiro D, Hannezo EB, Herszterg S, Bosveld F, Gaugué I, Balakireva M, Wang
Z, Cristo I, Rigaud S, Markova O, Bellaïche Y. 2017. Transmission of cytokinesis
forces via E cadherin dilution and actomyosin flows. Nature. 545(7652), 103–107.
mla: Pinheiro, Diana, et al. “Transmission of Cytokinesis Forces via E Cadherin
Dilution and Actomyosin Flows.” Nature, vol. 545, no. 7652, Nature Publishing
Group, 2017, pp. 103–07, doi:10.1038/nature22041.
short: D. Pinheiro, E.B. Hannezo, S. Herszterg, F. Bosveld, I. Gaugué, M. Balakireva,
Z. Wang, I. Cristo, S. Rigaud, O. Markova, Y. Bellaïche, Nature 545 (2017) 103–107.
date_created: 2018-12-11T11:49:18Z
date_published: 2017-05-04T00:00:00Z
date_updated: 2021-01-12T08:22:02Z
day: '04'
doi: 10.1038/nature22041
extern: '1'
intvolume: ' 545'
issue: '7652'
language:
- iso: eng
month: '05'
oa_version: None
page: 103 - 107
publication: Nature
publication_identifier:
issn:
- '00280836'
publication_status: published
publisher: Nature Publishing Group
publist_id: '6504'
quality_controlled: '1'
status: public
title: Transmission of cytokinesis forces via E cadherin dilution and actomyosin flows
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 545
year: '2017'
...
---
_id: '938'
abstract:
- lang: eng
text: The thesis encompasses several topics of plant cell biology which were studied
in the model plant Arabidopsis thaliana. Chapter 1 concerns the plant hormone
auxin and its polar transport through cells and tissues. The highly controlled,
directional transport of auxin is facilitated by plasma membrane-localized transporters.
Transporters from the PIN family direct auxin transport due to their polarized
localizations at cell membranes. Substantial effort has been put into research
on cellular trafficking of PIN proteins, which is thought to underlie their polar
distribution. I participated in a forward genetic screen aimed at identifying
novel regulators of PIN polarity. The screen yielded several genes which may be
involved in PIN polarity regulation or participate in polar auxin transport by
other means. Chapter 2 focuses on the endomembrane system, with particular attention
to clathrin-mediated endocytosis. The project started with identification of several
proteins that interact with clathrin light chains. Among them, I focused on two
putative homologues of auxilin, which in non-plant systems is an endocytotic factor
known for uncoating clathrin-coated vesicles in the final step of endocytosis.
The body of my work consisted of an in-depth characterization of transgenic A.
thaliana lines overexpressing these putative auxilins in an inducible manner.
Overexpression of these proteins leads to an inhibition of endocytosis, as documented
by imaging of cargoes and clathrin-related endocytic machinery. An extension of
this work is an investigation into a concept of homeostatic regulation acting
between distinct transport processes in the endomembrane system. With auxilin
overexpressing lines, where endocytosis is blocked specifically, I made observations
on the mutual relationship between two opposite trafficking processes of secretion
and endocytosis. In Chapter 3, I analyze cortical microtubule arrays and their
relationship to auxin signaling and polarized growth in elongating cells. In plants,
microtubules are organized into arrays just below the plasma membrane, and it
is thought that their function is to guide membrane-docked cellulose synthase
complexes. These, in turn, influence cell wall structure and cell shape by directed
deposition of cellulose fibres. In elongating cells, cortical microtubule arrays
are able to reorient in relation to long cell axis, and these reorientations have
been linked to cell growth and to signaling of growth-regulating factors such
as auxin or light. In this chapter, I am addressing the causal relationship between
microtubule array reorientation, growth, and auxin signaling. I arrive at a model
where array reorientation is not guided by auxin directly, but instead is only
controlled by growth, which, in turn, is regulated by auxin.
alternative_title:
- ISTA Thesis
article_processing_charge: No
author:
- first_name: Maciek
full_name: Adamowski, Maciek
id: 45F536D2-F248-11E8-B48F-1D18A9856A87
last_name: Adamowski
orcid: 0000-0001-6463-5257
citation:
ama: Adamowski M. Investigations into cell polarity and trafficking in the plant
model Arabidopsis thaliana . 2017. doi:10.15479/AT:ISTA:th_842
apa: Adamowski, M. (2017). Investigations into cell polarity and trafficking
in the plant model Arabidopsis thaliana . Institute of Science and Technology
Austria. https://doi.org/10.15479/AT:ISTA:th_842
chicago: Adamowski, Maciek. “Investigations into Cell Polarity and Trafficking in
the Plant Model Arabidopsis Thaliana .” Institute of Science and Technology Austria,
2017. https://doi.org/10.15479/AT:ISTA:th_842.
ieee: M. Adamowski, “Investigations into cell polarity and trafficking in the plant
model Arabidopsis thaliana ,” Institute of Science and Technology Austria, 2017.
ista: Adamowski M. 2017. Investigations into cell polarity and trafficking in the
plant model Arabidopsis thaliana . Institute of Science and Technology Austria.
mla: Adamowski, Maciek. Investigations into Cell Polarity and Trafficking in
the Plant Model Arabidopsis Thaliana . Institute of Science and Technology
Austria, 2017, doi:10.15479/AT:ISTA:th_842.
short: M. Adamowski, Investigations into Cell Polarity and Trafficking in the Plant
Model Arabidopsis Thaliana , Institute of Science and Technology Austria, 2017.
date_created: 2018-12-11T11:49:18Z
date_published: 2017-06-02T00:00:00Z
date_updated: 2023-09-07T12:06:09Z
day: '02'
ddc:
- '581'
- '583'
- '580'
degree_awarded: PhD
department:
- _id: JiFr
doi: 10.15479/AT:ISTA:th_842
file:
- access_level: closed
checksum: 193425764d9aaaed3ac57062a867b315
content_type: application/vnd.openxmlformats-officedocument.wordprocessingml.document
creator: dernst
date_created: 2019-04-05T09:03:20Z
date_updated: 2020-07-14T12:48:15Z
file_id: '6215'
file_name: 2017_Adamowski-Thesis_Source.docx
file_size: 46903863
relation: source_file
- access_level: open_access
checksum: df5ab01be81f821e1b958596a1ec8d21
content_type: application/pdf
creator: dernst
date_created: 2019-04-05T09:03:19Z
date_updated: 2020-07-14T12:48:15Z
file_id: '6216'
file_name: 2017_Adamowski-Thesis.pdf
file_size: 8698888
relation: main_file
file_date_updated: 2020-07-14T12:48:15Z
has_accepted_license: '1'
language:
- iso: eng
month: '06'
oa: 1
oa_version: Published Version
page: '117'
publication_identifier:
issn:
- 2663-337X
publication_status: published
publisher: Institute of Science and Technology Austria
publist_id: '6483'
pubrep_id: '842'
related_material:
record:
- id: '1591'
relation: part_of_dissertation
status: public
status: public
supervisor:
- first_name: Jiří
full_name: Friml, Jiří
id: 4159519E-F248-11E8-B48F-1D18A9856A87
last_name: Friml
orcid: 0000-0002-8302-7596
title: 'Investigations into cell polarity and trafficking in the plant model Arabidopsis
thaliana '
type: dissertation
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
year: '2017'
...
---
_id: '939'
abstract:
- lang: eng
text: We reveal the existence of continuous families of guided single-mode solitons
in planar waveguides with weakly nonlinear active core and absorbing boundaries.
Stable propagation of TE and TM-polarized solitons is accompanied by attenuation
of all other modes, i.e., the waveguide features properties of conservative and
dissipative systems. If the linear spectrum of the waveguide possesses exceptional
points, which occurs in the case of TM polarization, an originally focusing (defocusing)
material nonlinearity may become effectively defocusing (focusing). This occurs
due to the geometric phase of the carried eigenmode when the surface impedance
encircles the exceptional point. In its turn, the change of the effective nonlinearity
ensures the existence of dark (bright) solitons in spite of focusing (defocusing)
Kerr nonlinearity of the core. The existence of an exceptional point can also
result in anomalous enhancement of the effective nonlinearity. In terms of practical
applications, the nonlinearity of the reported waveguide can be manipulated by
controlling the properties of the absorbing cladding.
article_number: '033905'
article_processing_charge: No
author:
- first_name: Bikashkali
full_name: Midya, Bikashkali
id: 456187FC-F248-11E8-B48F-1D18A9856A87
last_name: Midya
- first_name: Vladimir
full_name: Konotop, Vladimir
last_name: Konotop
citation:
ama: 'Midya B, Konotop V. Waveguides with absorbing boundaries: Nonlinearity controlled
by an exceptional point and solitons. Physical Review Letters. 2017;119(3).
doi:10.1103/PhysRevLett.119.033905'
apa: 'Midya, B., & Konotop, V. (2017). Waveguides with absorbing boundaries:
Nonlinearity controlled by an exceptional point and solitons. Physical Review
Letters. American Physical Society. https://doi.org/10.1103/PhysRevLett.119.033905'
chicago: 'Midya, Bikashkali, and Vladimir Konotop. “Waveguides with Absorbing Boundaries:
Nonlinearity Controlled by an Exceptional Point and Solitons.” Physical Review
Letters. American Physical Society, 2017. https://doi.org/10.1103/PhysRevLett.119.033905.'
ieee: 'B. Midya and V. Konotop, “Waveguides with absorbing boundaries: Nonlinearity
controlled by an exceptional point and solitons,” Physical Review Letters,
vol. 119, no. 3. American Physical Society, 2017.'
ista: 'Midya B, Konotop V. 2017. Waveguides with absorbing boundaries: Nonlinearity
controlled by an exceptional point and solitons. Physical Review Letters. 119(3),
033905.'
mla: 'Midya, Bikashkali, and Vladimir Konotop. “Waveguides with Absorbing Boundaries:
Nonlinearity Controlled by an Exceptional Point and Solitons.” Physical Review
Letters, vol. 119, no. 3, 033905, American Physical Society, 2017, doi:10.1103/PhysRevLett.119.033905.'
short: B. Midya, V. Konotop, Physical Review Letters 119 (2017).
date_created: 2018-12-11T11:49:18Z
date_published: 2017-07-18T00:00:00Z
date_updated: 2023-09-26T15:39:46Z
day: '18'
department:
- _id: MiLe
doi: 10.1103/PhysRevLett.119.033905
ec_funded: 1
external_id:
isi:
- '000405718200012'
intvolume: ' 119'
isi: 1
issue: '3'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: 'https://arxiv.org/abs/1706.04085 '
month: '07'
oa: 1
oa_version: Submitted Version
project:
- _id: 25681D80-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '291734'
name: International IST Postdoc Fellowship Programme
publication: Physical Review Letters
publication_identifier:
issn:
- '00319007'
publication_status: published
publisher: American Physical Society
publist_id: '6481'
quality_controlled: '1'
scopus_import: '1'
status: public
title: 'Waveguides with absorbing boundaries: Nonlinearity controlled by an exceptional
point and solitons'
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 119
year: '2017'
...
---
_id: '94'
abstract:
- lang: eng
text: We introduce a method for breaking Lorentz reciprocity based upon the noncommutation
of frequency conversion and delay. The method requires no magnetic materials or
resonant physics, allowing for the design of scalable and broadband nonreciprocal
circuits. With this approach, two types of gyrators - universal building blocks
for linear, nonreciprocal circuits - are constructed. Using one of these gyrators,
we create a circulator with >15 dB of isolation across the 5-9 GHz band. Our
designs may be readily extended to any platform with suitable frequency conversion
elements, including semiconducting devices for telecommunication or an on-chip
superconducting implementation for quantum information processing.
article_number: '147703'
arxiv: 1
author:
- first_name: Eric
full_name: Rosenthal, Eric
last_name: Rosenthal
- first_name: Benjamin
full_name: Chapman, Benjamin
last_name: Chapman
- first_name: Andrew P
full_name: Higginbotham, Andrew P
id: 4AD6785A-F248-11E8-B48F-1D18A9856A87
last_name: Higginbotham
orcid: 0000-0003-2607-2363
- first_name: Joseph
full_name: Kerckhoff, Joseph
last_name: Kerckhoff
- first_name: Konrad
full_name: Lehnert, Konrad
last_name: Lehnert
citation:
ama: Rosenthal E, Chapman B, Higginbotham AP, Kerckhoff J, Lehnert K. Breaking Lorentz
reciprocity with frequency conversion and delay. APS Physics, Physical Review
Letters. 2017;119(14). doi:10.1103/PhysRevLett.119.147703
apa: Rosenthal, E., Chapman, B., Higginbotham, A. P., Kerckhoff, J., & Lehnert,
K. (2017). Breaking Lorentz reciprocity with frequency conversion and delay. APS
Physics, Physical Review Letters. American Physical Society. https://doi.org/10.1103/PhysRevLett.119.147703
chicago: Rosenthal, Eric, Benjamin Chapman, Andrew P Higginbotham, Joseph Kerckhoff,
and Konrad Lehnert. “Breaking Lorentz Reciprocity with Frequency Conversion and
Delay.” APS Physics, Physical Review Letters. American Physical Society,
2017. https://doi.org/10.1103/PhysRevLett.119.147703.
ieee: E. Rosenthal, B. Chapman, A. P. Higginbotham, J. Kerckhoff, and K. Lehnert,
“Breaking Lorentz reciprocity with frequency conversion and delay,” APS Physics,
Physical Review Letters, vol. 119, no. 14. American Physical Society, 2017.
ista: Rosenthal E, Chapman B, Higginbotham AP, Kerckhoff J, Lehnert K. 2017. Breaking
Lorentz reciprocity with frequency conversion and delay. APS Physics, Physical
Review Letters. 119(14), 147703.
mla: Rosenthal, Eric, et al. “Breaking Lorentz Reciprocity with Frequency Conversion
and Delay.” APS Physics, Physical Review Letters, vol. 119, no. 14, 147703,
American Physical Society, 2017, doi:10.1103/PhysRevLett.119.147703.
short: E. Rosenthal, B. Chapman, A.P. Higginbotham, J. Kerckhoff, K. Lehnert, APS
Physics, Physical Review Letters 119 (2017).
date_created: 2018-12-11T11:44:35Z
date_published: 2017-10-06T00:00:00Z
date_updated: 2021-01-12T08:22:04Z
day: '06'
doi: 10.1103/PhysRevLett.119.147703
extern: '1'
external_id:
arxiv:
- '1705.09548'
intvolume: ' 119'
issue: '14'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://arxiv.org/abs/1705.09548
month: '10'
oa: 1
oa_version: Submitted Version
publication: APS Physics, Physical Review Letters
publication_status: published
publisher: American Physical Society
publist_id: '7960'
quality_controlled: '1'
status: public
title: Breaking Lorentz reciprocity with frequency conversion and delay
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 119
year: '2017'
...
---
_id: '693'
abstract:
- lang: eng
text: 'Many central synapses contain a single presynaptic active zone and a single
postsynaptic density. Vesicular release statistics at such “simple synapses” indicate
that they contain a small complement of docking sites where vesicles repetitively
dock and fuse. In this work, we investigate functional and morphological aspects
of docking sites at simple synapses made between cerebellar parallel fibers and
molecular layer interneurons. Using immunogold labeling of SDS-treated freeze-fracture
replicas, we find that Cav2.1 channels form several clusters per active zone with
about nine channels per cluster. The mean value and range of intersynaptic variation
are similar for Cav2.1 cluster numbers and for functional estimates of docking-site
numbers obtained from the maximum numbers of released vesicles per action potential.
Both numbers grow in relation with synaptic size and decrease by a similar extent
with age between 2 wk and 4 wk postnatal. Thus, the mean docking-site numbers
were 3.15 at 2 wk (range: 1–10) and 2.03 at 4 wk (range: 1–4), whereas the mean
numbers of Cav2.1 clusters were 2.84 at 2 wk (range: 1–8) and 2.37 at 4 wk (range:
1–5). These changes were accompanied by decreases of miniature current amplitude
(from 93 pA to 56 pA), active-zone surface area (from 0.0427 μm2 to 0.0234 μm2),
and initial success rate (from 0.609 to 0.353), indicating a tightening of synaptic
transmission with development. Altogether, these results suggest a close correspondence
between the number of functionally defined vesicular docking sites and that of
clusters of voltage-gated calcium channels. '
article_processing_charge: Yes (in subscription journal)
author:
- first_name: Takafumi
full_name: Miki, Takafumi
last_name: Miki
- first_name: Walter
full_name: Kaufmann, Walter
id: 3F99E422-F248-11E8-B48F-1D18A9856A87
last_name: Kaufmann
orcid: 0000-0001-9735-5315
- first_name: Gerardo
full_name: Malagon, Gerardo
last_name: Malagon
- first_name: Laura
full_name: Gomez, Laura
last_name: Gomez
- first_name: Katsuhiko
full_name: Tabuchi, Katsuhiko
last_name: Tabuchi
- first_name: Masahiko
full_name: Watanabe, Masahiko
last_name: Watanabe
- first_name: Ryuichi
full_name: Shigemoto, Ryuichi
id: 499F3ABC-F248-11E8-B48F-1D18A9856A87
last_name: Shigemoto
orcid: 0000-0001-8761-9444
- first_name: Alain
full_name: Marty, Alain
last_name: Marty
citation:
ama: Miki T, Kaufmann W, Malagon G, et al. Numbers of presynaptic Ca2+ channel clusters
match those of functionally defined vesicular docking sites in single central
synapses. PNAS. 2017;114(26):E5246-E5255. doi:10.1073/pnas.1704470114
apa: Miki, T., Kaufmann, W., Malagon, G., Gomez, L., Tabuchi, K., Watanabe, M.,
… Marty, A. (2017). Numbers of presynaptic Ca2+ channel clusters match those of
functionally defined vesicular docking sites in single central synapses. PNAS.
National Academy of Sciences. https://doi.org/10.1073/pnas.1704470114
chicago: Miki, Takafumi, Walter Kaufmann, Gerardo Malagon, Laura Gomez, Katsuhiko
Tabuchi, Masahiko Watanabe, Ryuichi Shigemoto, and Alain Marty. “Numbers of Presynaptic
Ca2+ Channel Clusters Match Those of Functionally Defined Vesicular Docking Sites
in Single Central Synapses.” PNAS. National Academy of Sciences, 2017.
https://doi.org/10.1073/pnas.1704470114.
ieee: T. Miki et al., “Numbers of presynaptic Ca2+ channel clusters match
those of functionally defined vesicular docking sites in single central synapses,”
PNAS, vol. 114, no. 26. National Academy of Sciences, pp. E5246–E5255,
2017.
ista: Miki T, Kaufmann W, Malagon G, Gomez L, Tabuchi K, Watanabe M, Shigemoto R,
Marty A. 2017. Numbers of presynaptic Ca2+ channel clusters match those of functionally
defined vesicular docking sites in single central synapses. PNAS. 114(26), E5246–E5255.
mla: Miki, Takafumi, et al. “Numbers of Presynaptic Ca2+ Channel Clusters Match
Those of Functionally Defined Vesicular Docking Sites in Single Central Synapses.”
PNAS, vol. 114, no. 26, National Academy of Sciences, 2017, pp. E5246–55,
doi:10.1073/pnas.1704470114.
short: T. Miki, W. Kaufmann, G. Malagon, L. Gomez, K. Tabuchi, M. Watanabe, R. Shigemoto,
A. Marty, PNAS 114 (2017) E5246–E5255.
date_created: 2018-12-11T11:47:57Z
date_published: 2017-06-27T00:00:00Z
date_updated: 2023-02-23T12:54:57Z
day: '27'
ddc:
- '570'
department:
- _id: EM-Fac
- _id: RySh
doi: 10.1073/pnas.1704470114
external_id:
pmid:
- '28607047'
file:
- access_level: open_access
checksum: 2ab75d554f3df4a34d20fa8040589b7e
content_type: application/pdf
creator: kschuh
date_created: 2020-01-03T13:27:29Z
date_updated: 2020-07-14T12:47:44Z
file_id: '7223'
file_name: 2017_PNAS_Miki.pdf
file_size: 2721544
relation: main_file
file_date_updated: 2020-07-14T12:47:44Z
has_accepted_license: '1'
intvolume: ' 114'
issue: '26'
language:
- iso: eng
month: '06'
oa: 1
oa_version: Published Version
page: E5246 - E5255
pmid: 1
publication: PNAS
publication_identifier:
issn:
- '00278424'
publication_status: published
publisher: National Academy of Sciences
publist_id: '7013'
quality_controlled: '1'
scopus_import: 1
status: public
title: Numbers of presynaptic Ca2+ channel clusters match those of functionally defined
vesicular docking sites in single central synapses
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 114
year: '2017'
...
---
_id: '6932'
abstract:
- lang: eng
text: "LCLs or locally checkable labelling problems (e.g. maximal independent set,
maximal matching, and vertex colouring) in the LOCAL model of computation are
very well-understood in cycles (toroidal 1-dimensional grids): every problem has
a complexity of O(1), Θ(log* n), or Θ(n), and the design of optimal algorithms
can be fully automated. This work develops the complexity theory of LCL problems
for toroidal 2-dimensional grids. The complexity classes are the same as in the
1-dimensional case: O(1), Θ(log* n), and Θ(n). However, given an LCL problem it
is undecidable whether its complexity is Θ(log* n) or Θ(n) in 2-dimensional grids.\r\nNevertheless,
if we correctly guess that the complexity of a problem is Θ(log* n), we can completely
automate the design of optimal algorithms. For any problem we can find an algorithm
that is of a normal form A' o Sk, where A' is a finite function, Sk is an algorithm
for finding a maximal independent set in kth power of the grid, and k is a constant.\r\nFinally,
partially with the help of automated design tools, we classify the complexity
of several concrete LCL problems related to colourings and orientations."
article_processing_charge: No
author:
- first_name: Sebastian
full_name: Brandt, Sebastian
last_name: Brandt
- first_name: Juho
full_name: Hirvonen, Juho
last_name: Hirvonen
- first_name: Janne H.
full_name: Korhonen, Janne H.
last_name: Korhonen
- first_name: Tuomo
full_name: Lempiäinen, Tuomo
last_name: Lempiäinen
- first_name: Patric R.J.
full_name: Östergård, Patric R.J.
last_name: Östergård
- first_name: Christopher
full_name: Purcell, Christopher
last_name: Purcell
- first_name: Joel
full_name: Rybicki, Joel
id: 334EFD2E-F248-11E8-B48F-1D18A9856A87
last_name: Rybicki
orcid: 0000-0002-6432-6646
- first_name: Jukka
full_name: Suomela, Jukka
last_name: Suomela
- first_name: Przemysław
full_name: Uznański, Przemysław
last_name: Uznański
citation:
ama: 'Brandt S, Hirvonen J, Korhonen JH, et al. LCL problems on grids. In: ACM Press;
2017:101-110. doi:10.1145/3087801.3087833'
apa: 'Brandt, S., Hirvonen, J., Korhonen, J. H., Lempiäinen, T., Östergård, P. R.
J., Purcell, C., … Uznański, P. (2017). LCL problems on grids (pp. 101–110). Presented
at the PODC: Principles of Distributed Computing, Washington, DC, United States:
ACM Press. https://doi.org/10.1145/3087801.3087833'
chicago: Brandt, Sebastian, Juho Hirvonen, Janne H. Korhonen, Tuomo Lempiäinen,
Patric R.J. Östergård, Christopher Purcell, Joel Rybicki, Jukka Suomela, and Przemysław
Uznański. “LCL Problems on Grids,” 101–10. ACM Press, 2017. https://doi.org/10.1145/3087801.3087833.
ieee: 'S. Brandt et al., “LCL problems on grids,” presented at the PODC:
Principles of Distributed Computing, Washington, DC, United States, 2017, pp.
101–110.'
ista: 'Brandt S, Hirvonen J, Korhonen JH, Lempiäinen T, Östergård PRJ, Purcell C,
Rybicki J, Suomela J, Uznański P. 2017. LCL problems on grids. PODC: Principles
of Distributed Computing, 101–110.'
mla: Brandt, Sebastian, et al. LCL Problems on Grids. ACM Press, 2017, pp.
101–10, doi:10.1145/3087801.3087833.
short: S. Brandt, J. Hirvonen, J.H. Korhonen, T. Lempiäinen, P.R.J. Östergård, C.
Purcell, J. Rybicki, J. Suomela, P. Uznański, in:, ACM Press, 2017, pp. 101–110.
conference:
end_date: 2017-07-27
location: Washington, DC, United States
name: 'PODC: Principles of Distributed Computing'
start_date: 2017-07-25
date_created: 2019-10-08T12:47:46Z
date_published: 2017-07-01T00:00:00Z
date_updated: 2021-01-12T08:09:39Z
day: '01'
doi: 10.1145/3087801.3087833
extern: '1'
language:
- iso: eng
month: '07'
oa_version: None
page: 101-110
publication_identifier:
isbn:
- '9781450349925'
publication_status: published
publisher: ACM Press
quality_controlled: '1'
status: public
title: LCL problems on grids
type: conference
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
year: '2017'
...
---
_id: '694'
abstract:
- lang: eng
text: A change regarding the extent of adhesion - hereafter referred to as adhesion
plasticity - between adhesive and less-adhesive states of mammalian cells is important
for their behavior. To investigate adhesion plasticity, we have selected a stable
isogenic subpopulation of human MDA-MB-468 breast carcinoma cells growing in suspension.
These suspension cells are unable to re-adhere to various matrices or to contract
three-dimensional collagen lattices. By using transcriptome analysis, we identified
the focal adhesion protein tensin3 (Tns3) as a determinant of adhesion plasticity.
Tns3 is strongly reduced at mRNA and protein levels in suspension cells. Furthermore,
by transiently challenging breast cancer cells to grow under non-adherent conditions
markedly reduces Tns3 protein expression, which is regained upon re-adhesion.
Stable knockdown of Tns3 in parental MDA-MB-468 cells results in defective adhesion,
spreading and migration. Tns3-knockdown cells display impaired structure and dynamics
of focal adhesion complexes as determined by immunostaining. Restoration of Tns3
protein expression in suspension cells partially rescues adhesion and focal contact
composition. Our work identifies Tns3 as a crucial focal adhesion component regulated
by, and functionally contributing to, the switch between adhesive and non-adhesive
states in MDA-MB-468 cancer cells.
article_type: original
author:
- first_name: Astrid
full_name: Veß, Astrid
last_name: Veß
- first_name: Ulrich
full_name: Blache, Ulrich
last_name: Blache
- first_name: Laura
full_name: Leitner, Laura
last_name: Leitner
- first_name: Angela
full_name: Kurz, Angela
last_name: Kurz
- first_name: Anja
full_name: Ehrenpfordt, Anja
last_name: Ehrenpfordt
- first_name: Michael K
full_name: Sixt, Michael K
id: 41E9FBEA-F248-11E8-B48F-1D18A9856A87
last_name: Sixt
orcid: 0000-0002-6620-9179
- first_name: Guido
full_name: Posern, Guido
last_name: Posern
citation:
ama: Veß A, Blache U, Leitner L, et al. A dual phenotype of MDA MB 468 cancer cells
reveals mutual regulation of tensin3 and adhesion plasticity. Journal of Cell
Science. 2017;130(13):2172-2184. doi:10.1242/jcs.200899
apa: Veß, A., Blache, U., Leitner, L., Kurz, A., Ehrenpfordt, A., Sixt, M. K., &
Posern, G. (2017). A dual phenotype of MDA MB 468 cancer cells reveals mutual
regulation of tensin3 and adhesion plasticity. Journal of Cell Science.
Company of Biologists. https://doi.org/10.1242/jcs.200899
chicago: Veß, Astrid, Ulrich Blache, Laura Leitner, Angela Kurz, Anja Ehrenpfordt,
Michael K Sixt, and Guido Posern. “A Dual Phenotype of MDA MB 468 Cancer Cells
Reveals Mutual Regulation of Tensin3 and Adhesion Plasticity.” Journal of Cell
Science. Company of Biologists, 2017. https://doi.org/10.1242/jcs.200899.
ieee: A. Veß et al., “A dual phenotype of MDA MB 468 cancer cells reveals
mutual regulation of tensin3 and adhesion plasticity,” Journal of Cell Science,
vol. 130, no. 13. Company of Biologists, pp. 2172–2184, 2017.
ista: Veß A, Blache U, Leitner L, Kurz A, Ehrenpfordt A, Sixt MK, Posern G. 2017.
A dual phenotype of MDA MB 468 cancer cells reveals mutual regulation of tensin3
and adhesion plasticity. Journal of Cell Science. 130(13), 2172–2184.
mla: Veß, Astrid, et al. “A Dual Phenotype of MDA MB 468 Cancer Cells Reveals Mutual
Regulation of Tensin3 and Adhesion Plasticity.” Journal of Cell Science,
vol. 130, no. 13, Company of Biologists, 2017, pp. 2172–84, doi:10.1242/jcs.200899.
short: A. Veß, U. Blache, L. Leitner, A. Kurz, A. Ehrenpfordt, M.K. Sixt, G. Posern,
Journal of Cell Science 130 (2017) 2172–2184.
date_created: 2018-12-11T11:47:58Z
date_published: 2017-07-01T00:00:00Z
date_updated: 2021-01-12T08:09:41Z
day: '01'
ddc:
- '570'
department:
- _id: MiSi
doi: 10.1242/jcs.200899
external_id:
pmid:
- '28515231'
file:
- access_level: open_access
checksum: 42c81a0a4fc3128883b391c3af3f74bc
content_type: application/pdf
creator: dernst
date_created: 2019-10-24T09:43:56Z
date_updated: 2020-07-14T12:47:45Z
file_id: '6966'
file_name: 2017_CellScience_Vess.pdf
file_size: 10847596
relation: main_file
file_date_updated: 2020-07-14T12:47:45Z
has_accepted_license: '1'
intvolume: ' 130'
issue: '13'
language:
- iso: eng
month: '07'
oa: 1
oa_version: Published Version
page: 2172 - 2184
pmid: 1
publication: Journal of Cell Science
publication_identifier:
issn:
- '00219533'
publication_status: published
publisher: Company of Biologists
publist_id: '7008'
quality_controlled: '1'
scopus_import: 1
status: public
title: A dual phenotype of MDA MB 468 cancer cells reveals mutual regulation of tensin3
and adhesion plasticity
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 130
year: '2017'
...
---
_id: '695'
abstract:
- lang: eng
text: It has been known since Stefan Vogel's observations in 1969 that solitary
female oil bees collect fatty floral oils from specialized oil-secreting plants
with the aid of hairy patches on either their legs or abdomen, a reward used as
food for their larvae and/or to line their brood cells. Similar adaptations are
also known from male oil bees, although the purpose of their oil-collecting behavior
has not yet been clarified. Here, we describe a novel pollination system involving
male Paratetrapedia oil bees and the tropical herb Anthurium acutifolium. We present
ultrastructural morphological details of bee and plant structures involved in
this interaction and the composition of floral scents likely mediating pollinator
attraction. Inflorescences of A. acutifolium were visited almost exclusively by
male P. chocoensis oil bees. The bees mopped with a hairy patch of their abdominal
sterna 3 across the inflorescence surface. During this activity on both staminate
and pistillate stage inflorescences, bees’ abdomens and legs became loaded with
pollen and contacted receptive stigmas. In contrast to what has been observed
in other angiosperms visited for the collection of fatty floral oils, the inflorescences/flowers
of A. acutifolium do not have structures specialized in oil secretion, i.e., elaiophores.
These inflorescences, nonetheless, were strongly scented during the time interval
they were visited by the bees. Gas chromatography/mass spectrometry (GC/MS) analyses
of dynamic headspace floral samples revealed that inflorescences of both anthetic
phases emitted scent bouquets consisting mainly of aliphatic esters, indole and
uncommmon terpenoids (megastigmanes). Interestingly enough, our data suggest that
the unusual floral scent of A. acutifolium is a perfume reward collected by male
P. chocoensis oil bees. This pollination system thus bears a remarkable resemblence
with the interactions between perfume-collecting male euglossine bees and their
preferred flowers, discovered by Stefan Vogel half a century ago.
author:
- first_name: Florian
full_name: Etl, Florian
last_name: Etl
- first_name: Anna
full_name: Franschitz, Anna
id: 480826C8-F248-11E8-B48F-1D18A9856A87
last_name: Franschitz
- first_name: Antonio
full_name: Aguiar, Antonio
last_name: Aguiar
- first_name: Jürg
full_name: Schönenberger, Jürg
last_name: Schönenberger
- first_name: Stefan
full_name: Dötterl, Stefan
last_name: Dötterl
citation:
ama: 'Etl F, Franschitz A, Aguiar A, Schönenberger J, Dötterl S. A perfume collecting
male oil bee? Evidences of a novel pollination system involving Anthurium acutifolium
Araceae and Paratetrapedia chocoensis Apidae Tapinotaspidini. Flora: Morphology,
Distribution, Functional Ecology of Plants. 2017;232:7-15. doi:10.1016/j.flora.2017.02.020'
apa: 'Etl, F., Franschitz, A., Aguiar, A., Schönenberger, J., & Dötterl, S.
(2017). A perfume collecting male oil bee? Evidences of a novel pollination system
involving Anthurium acutifolium Araceae and Paratetrapedia chocoensis Apidae Tapinotaspidini.
Flora: Morphology, Distribution, Functional Ecology of Plants. Elsevier.
https://doi.org/10.1016/j.flora.2017.02.020'
chicago: 'Etl, Florian, Anna Franschitz, Antonio Aguiar, Jürg Schönenberger, and
Stefan Dötterl. “A Perfume Collecting Male Oil Bee? Evidences of a Novel Pollination
System Involving Anthurium Acutifolium Araceae and Paratetrapedia Chocoensis Apidae
Tapinotaspidini.” Flora: Morphology, Distribution, Functional Ecology of Plants.
Elsevier, 2017. https://doi.org/10.1016/j.flora.2017.02.020.'
ieee: 'F. Etl, A. Franschitz, A. Aguiar, J. Schönenberger, and S. Dötterl, “A perfume
collecting male oil bee? Evidences of a novel pollination system involving Anthurium
acutifolium Araceae and Paratetrapedia chocoensis Apidae Tapinotaspidini,” Flora:
Morphology, Distribution, Functional Ecology of Plants, vol. 232. Elsevier,
pp. 7–15, 2017.'
ista: 'Etl F, Franschitz A, Aguiar A, Schönenberger J, Dötterl S. 2017. A perfume
collecting male oil bee? Evidences of a novel pollination system involving Anthurium
acutifolium Araceae and Paratetrapedia chocoensis Apidae Tapinotaspidini. Flora:
Morphology, Distribution, Functional Ecology of Plants. 232, 7–15.'
mla: 'Etl, Florian, et al. “A Perfume Collecting Male Oil Bee? Evidences of a Novel
Pollination System Involving Anthurium Acutifolium Araceae and Paratetrapedia
Chocoensis Apidae Tapinotaspidini.” Flora: Morphology, Distribution, Functional
Ecology of Plants, vol. 232, Elsevier, 2017, pp. 7–15, doi:10.1016/j.flora.2017.02.020.'
short: 'F. Etl, A. Franschitz, A. Aguiar, J. Schönenberger, S. Dötterl, Flora: Morphology,
Distribution, Functional Ecology of Plants 232 (2017) 7–15.'
date_created: 2018-12-11T11:47:58Z
date_published: 2017-07-01T00:00:00Z
date_updated: 2021-01-12T08:09:44Z
day: '01'
doi: 10.1016/j.flora.2017.02.020
extern: '1'
intvolume: ' 232'
language:
- iso: eng
month: '07'
oa_version: None
page: 7 - 15
publication: 'Flora: Morphology, Distribution, Functional Ecology of Plants'
publication_identifier:
issn:
- '03672530'
publication_status: published
publisher: Elsevier
publist_id: '7007'
quality_controlled: '1'
status: public
title: A perfume collecting male oil bee? Evidences of a novel pollination system
involving Anthurium acutifolium Araceae and Paratetrapedia chocoensis Apidae Tapinotaspidini
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 232
year: '2017'
...
---
_id: '696'
abstract:
- lang: eng
text: Mutator strains are expected to evolve when the availability and effect of
beneficial mutations are high enough to counteract the disadvantage from deleterious
mutations that will inevitably accumulate. As the population becomes more adapted
to its environment, both availability and effect of beneficial mutations necessarily
decrease and mutation rates are predicted to decrease. It has been shown that
certain molecular mechanisms can lead to increased mutation rates when the organism
finds itself in a stressful environment. While this may be a correlated response
to other functions, it could also be an adaptive mechanism, raising mutation rates
only when it is most advantageous. Here, we use a mathematical model to investigate
the plausibility of the adaptive hypothesis. We show that such a mechanism can
be mantained if the population is subjected to diverse stresses. By simulating
various antibiotic treatment schemes, we find that combination treatments can
reduce the effectiveness of second-order selection on stress-induced mutagenesis.
We discuss the implications of our results to strategies of antibiotic therapy.
article_number: e1005609
article_type: original
author:
- first_name: Marta
full_name: Lukacisinova, Marta
id: 4342E402-F248-11E8-B48F-1D18A9856A87
last_name: Lukacisinova
orcid: 0000-0002-2519-8004
- first_name: Sebastian
full_name: Novak, Sebastian
id: 461468AE-F248-11E8-B48F-1D18A9856A87
last_name: Novak
orcid: 0000-0002-2519-824X
- first_name: Tiago
full_name: Paixao, Tiago
id: 2C5658E6-F248-11E8-B48F-1D18A9856A87
last_name: Paixao
orcid: 0000-0003-2361-3953
citation:
ama: 'Lukacisinova M, Novak S, Paixao T. Stress induced mutagenesis: Stress diversity
facilitates the persistence of mutator genes. PLoS Computational Biology.
2017;13(7). doi:10.1371/journal.pcbi.1005609'
apa: 'Lukacisinova, M., Novak, S., & Paixao, T. (2017). Stress induced mutagenesis:
Stress diversity facilitates the persistence of mutator genes. PLoS Computational
Biology. Public Library of Science. https://doi.org/10.1371/journal.pcbi.1005609'
chicago: 'Lukacisinova, Marta, Sebastian Novak, and Tiago Paixao. “Stress Induced
Mutagenesis: Stress Diversity Facilitates the Persistence of Mutator Genes.” PLoS
Computational Biology. Public Library of Science, 2017. https://doi.org/10.1371/journal.pcbi.1005609.'
ieee: 'M. Lukacisinova, S. Novak, and T. Paixao, “Stress induced mutagenesis: Stress
diversity facilitates the persistence of mutator genes,” PLoS Computational
Biology, vol. 13, no. 7. Public Library of Science, 2017.'
ista: 'Lukacisinova M, Novak S, Paixao T. 2017. Stress induced mutagenesis: Stress
diversity facilitates the persistence of mutator genes. PLoS Computational Biology.
13(7), e1005609.'
mla: 'Lukacisinova, Marta, et al. “Stress Induced Mutagenesis: Stress Diversity
Facilitates the Persistence of Mutator Genes.” PLoS Computational Biology,
vol. 13, no. 7, e1005609, Public Library of Science, 2017, doi:10.1371/journal.pcbi.1005609.'
short: M. Lukacisinova, S. Novak, T. Paixao, PLoS Computational Biology 13 (2017).
date_created: 2018-12-11T11:47:58Z
date_published: 2017-07-18T00:00:00Z
date_updated: 2024-03-25T23:30:14Z
day: '18'
ddc:
- '576'
department:
- _id: ToBo
- _id: NiBa
- _id: CaGu
doi: 10.1371/journal.pcbi.1005609
ec_funded: 1
file:
- access_level: open_access
checksum: 9143c290fa6458ed2563bff4b295554a
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:15:01Z
date_updated: 2020-07-14T12:47:46Z
file_id: '5117'
file_name: IST-2017-894-v1+1_journal.pcbi.1005609.pdf
file_size: 3775716
relation: main_file
file_date_updated: 2020-07-14T12:47:46Z
has_accepted_license: '1'
intvolume: ' 13'
issue: '7'
language:
- iso: eng
license: https://creativecommons.org/licenses/by/4.0/
month: '07'
oa: 1
oa_version: Published Version
project:
- _id: 25B1EC9E-B435-11E9-9278-68D0E5697425
call_identifier: FP7
grant_number: '618091'
name: Speed of Adaptation in Population Genetics and Evolutionary Computation
publication: PLoS Computational Biology
publication_identifier:
issn:
- 1553734X
publication_status: published
publisher: Public Library of Science
publist_id: '7004'
pubrep_id: '894'
quality_controlled: '1'
related_material:
record:
- id: '9849'
relation: research_data
status: public
- id: '9850'
relation: research_data
status: public
- id: '9851'
relation: research_data
status: public
- id: '9852'
relation: research_data
status: public
- id: '6263'
relation: dissertation_contains
status: public
scopus_import: 1
status: public
title: 'Stress induced mutagenesis: Stress diversity facilitates the persistence of
mutator genes'
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 13
year: '2017'
...
---
_id: '697'
abstract:
- lang: eng
text: 'De, Trevisan and Tulsiani [CRYPTO 2010] show that every distribution over
n-bit strings which has constant statistical distance to uniform (e.g., the output
of a pseudorandom generator mapping n-1 to n bit strings), can be distinguished
from the uniform distribution with advantage epsilon by a circuit of size O( 2^n
epsilon^2). We generalize this result, showing that a distribution which has less
than k bits of min-entropy, can be distinguished from any distribution with k
bits of delta-smooth min-entropy with advantage epsilon by a circuit of size O(2^k
epsilon^2/delta^2). As a special case, this implies that any distribution with
support at most 2^k (e.g., the output of a pseudoentropy generator mapping k to
n bit strings) can be distinguished from any given distribution with min-entropy
k+1 with advantage epsilon by a circuit of size O(2^k epsilon^2). Our result thus
shows that pseudoentropy distributions face basically the same non-uniform attacks
as pseudorandom distributions. '
alternative_title:
- LIPIcs
article_number: '39'
author:
- first_name: Krzysztof Z
full_name: Pietrzak, Krzysztof Z
id: 3E04A7AA-F248-11E8-B48F-1D18A9856A87
last_name: Pietrzak
orcid: 0000-0002-9139-1654
- first_name: Maciej
full_name: Skórski, Maciej
id: EC09FA6A-02D0-11E9-8223-86B7C91467DD
last_name: Skórski
citation:
ama: 'Pietrzak KZ, Skórski M. Non uniform attacks against pseudoentropy. In: Vol
80. Schloss Dagstuhl - Leibniz-Zentrum für Informatik; 2017. doi:10.4230/LIPIcs.ICALP.2017.39'
apa: 'Pietrzak, K. Z., & Skórski, M. (2017). Non uniform attacks against pseudoentropy
(Vol. 80). Presented at the ICALP: International Colloquium on Automata, Languages,
and Programming, Warsaw, Poland: Schloss Dagstuhl - Leibniz-Zentrum für Informatik.
https://doi.org/10.4230/LIPIcs.ICALP.2017.39'
chicago: Pietrzak, Krzysztof Z, and Maciej Skórski. “Non Uniform Attacks against
Pseudoentropy,” Vol. 80. Schloss Dagstuhl - Leibniz-Zentrum für Informatik, 2017.
https://doi.org/10.4230/LIPIcs.ICALP.2017.39.
ieee: 'K. Z. Pietrzak and M. Skórski, “Non uniform attacks against pseudoentropy,”
presented at the ICALP: International Colloquium on Automata, Languages, and Programming,
Warsaw, Poland, 2017, vol. 80.'
ista: 'Pietrzak KZ, Skórski M. 2017. Non uniform attacks against pseudoentropy.
ICALP: International Colloquium on Automata, Languages, and Programming, LIPIcs,
vol. 80, 39.'
mla: Pietrzak, Krzysztof Z., and Maciej Skórski. Non Uniform Attacks against
Pseudoentropy. Vol. 80, 39, Schloss Dagstuhl - Leibniz-Zentrum für Informatik,
2017, doi:10.4230/LIPIcs.ICALP.2017.39.
short: K.Z. Pietrzak, M. Skórski, in:, Schloss Dagstuhl - Leibniz-Zentrum für Informatik,
2017.
conference:
end_date: 2017-07-14
location: Warsaw, Poland
name: 'ICALP: International Colloquium on Automata, Languages, and Programming'
start_date: 2017-07-10
date_created: 2018-12-11T11:47:59Z
date_published: 2017-07-01T00:00:00Z
date_updated: 2021-01-12T08:11:15Z
day: '01'
ddc:
- '005'
department:
- _id: KrPi
doi: 10.4230/LIPIcs.ICALP.2017.39
ec_funded: 1
file:
- access_level: open_access
checksum: e95618a001692f1af2d68f5fde43bc1f
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:08:40Z
date_updated: 2020-07-14T12:47:46Z
file_id: '4701'
file_name: IST-2017-893-v1+1_LIPIcs-ICALP-2017-39.pdf
file_size: 601004
relation: main_file
file_date_updated: 2020-07-14T12:47:46Z
has_accepted_license: '1'
intvolume: ' 80'
language:
- iso: eng
month: '07'
oa: 1
oa_version: Published Version
project:
- _id: 258AA5B2-B435-11E9-9278-68D0E5697425
call_identifier: H2020
grant_number: '682815'
name: Teaching Old Crypto New Tricks
publication_identifier:
issn:
- '18688969'
publication_status: published
publisher: Schloss Dagstuhl - Leibniz-Zentrum für Informatik
publist_id: '7003'
pubrep_id: '893'
quality_controlled: '1'
scopus_import: 1
status: public
title: Non uniform attacks against pseudoentropy
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: conference
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 80
year: '2017'
...
---
_id: '698'
abstract:
- lang: eng
text: 'Extracellular matrix signals from the microenvironment regulate gene expression
patterns and cell behavior. Using a combination of experiments and geometric models,
we demonstrate correlations between cell geometry, three-dimensional (3D) organization
of chromosome territories, and gene expression. Fluorescence in situ hybridization
experiments showed that micropatterned fibroblasts cultured on anisotropic versus
isotropic substrates resulted in repositioning of specific chromosomes, which
contained genes that were differentially regulated by cell geometries. Experiments
combined with ellipsoid packing models revealed that the mechanosensitivity of
chromosomes was correlated with their orientation in the nucleus. Transcription
inhibition experiments suggested that the intermingling degree was more sensitive
to global changes in transcription than to chromosome radial positioning and its
orientations. These results suggested that cell geometry modulated 3D chromosome
arrangement, and their neighborhoods correlated with gene expression patterns
in a predictable manner. This is central to understanding geometric control of
genetic programs involved in cellular homeostasis and the associated diseases. '
author:
- first_name: Yejun
full_name: Wang, Yejun
last_name: Wang
- first_name: Mallika
full_name: Nagarajan, Mallika
last_name: Nagarajan
- first_name: Caroline
full_name: Uhler, Caroline
id: 49ADD78E-F248-11E8-B48F-1D18A9856A87
last_name: Uhler
orcid: 0000-0002-7008-0216
- first_name: Gv
full_name: Shivashankar, Gv
last_name: Shivashankar
citation:
ama: Wang Y, Nagarajan M, Uhler C, Shivashankar G. Orientation and repositioning
of chromosomes correlate with cell geometry dependent gene expression. Molecular
Biology of the Cell. 2017;28(14):1997-2009. doi:10.1091/mbc.E16-12-0825
apa: Wang, Y., Nagarajan, M., Uhler, C., & Shivashankar, G. (2017). Orientation
and repositioning of chromosomes correlate with cell geometry dependent gene expression.
Molecular Biology of the Cell. American Society for Cell Biology. https://doi.org/10.1091/mbc.E16-12-0825
chicago: Wang, Yejun, Mallika Nagarajan, Caroline Uhler, and Gv Shivashankar. “Orientation
and Repositioning of Chromosomes Correlate with Cell Geometry Dependent Gene Expression.”
Molecular Biology of the Cell. American Society for Cell Biology, 2017.
https://doi.org/10.1091/mbc.E16-12-0825.
ieee: Y. Wang, M. Nagarajan, C. Uhler, and G. Shivashankar, “Orientation and repositioning
of chromosomes correlate with cell geometry dependent gene expression,” Molecular
Biology of the Cell, vol. 28, no. 14. American Society for Cell Biology, pp.
1997–2009, 2017.
ista: Wang Y, Nagarajan M, Uhler C, Shivashankar G. 2017. Orientation and repositioning
of chromosomes correlate with cell geometry dependent gene expression. Molecular
Biology of the Cell. 28(14), 1997–2009.
mla: Wang, Yejun, et al. “Orientation and Repositioning of Chromosomes Correlate
with Cell Geometry Dependent Gene Expression.” Molecular Biology of the Cell,
vol. 28, no. 14, American Society for Cell Biology, 2017, pp. 1997–2009, doi:10.1091/mbc.E16-12-0825.
short: Y. Wang, M. Nagarajan, C. Uhler, G. Shivashankar, Molecular Biology of the
Cell 28 (2017) 1997–2009.
date_created: 2018-12-11T11:47:59Z
date_published: 2017-07-07T00:00:00Z
date_updated: 2021-01-12T08:11:17Z
day: '07'
ddc:
- '519'
department:
- _id: CaUh
doi: 10.1091/mbc.E16-12-0825
file:
- access_level: open_access
checksum: de01dac9e30970cfa6ae902480a4e04d
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:10:53Z
date_updated: 2020-07-14T12:47:46Z
file_id: '4844'
file_name: IST-2017-892-v1+1_Mol._Biol._Cell-2017-Wang-1997-2009.pdf
file_size: 1086097
relation: main_file
file_date_updated: 2020-07-14T12:47:46Z
has_accepted_license: '1'
intvolume: ' 28'
issue: '14'
language:
- iso: eng
license: https://creativecommons.org/licenses/by-nc-sa/4.0/
month: '07'
oa: 1
oa_version: Published Version
page: 1997 - 2009
project:
- _id: 2530CA10-B435-11E9-9278-68D0E5697425
call_identifier: FWF
grant_number: Y 903-N35
name: 'Gaussian Graphical Models: Theory and Applications'
publication: Molecular Biology of the Cell
publication_identifier:
issn:
- '10591524'
publication_status: published
publisher: American Society for Cell Biology
publist_id: '7001'
pubrep_id: '892'
quality_controlled: '1'
scopus_import: 1
status: public
title: Orientation and repositioning of chromosomes correlate with cell geometry dependent
gene expression
tmp:
image: /images/cc_by_nc_sa.png
legal_code_url: https://creativecommons.org/licenses/by-nc-sa/4.0/legalcode
name: Creative Commons Attribution-NonCommercial-ShareAlike 4.0 International (CC
BY-NC-SA 4.0)
short: CC BY-NC-SA (4.0)
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 28
year: '2017'
...
---
_id: '699'
abstract:
- lang: eng
text: 'In antagonistic symbioses, such as host–parasite interactions, one population’s
success is the other’s loss. In mutualistic symbioses, such as division of labor,
both parties can gain, but they might have different preferences over the possible
mutualistic arrangements. The rates of evolution of the two populations in a symbiosis
are important determinants of which population will be more successful: Faster
evolution is thought to be favored in antagonistic symbioses (the “Red Queen effect”),
but disfavored in certain mutualistic symbioses (the “Red King effect”). However,
it remains unclear which biological parameters drive these effects. Here, we analyze
the effects of the various determinants of evolutionary rate: generation time,
mutation rate, population size, and the intensity of natural selection. Our main
results hold for the case where mutation is infrequent. Slower evolution causes
a long-term advantage in an important class of mutualistic interactions. Surprisingly,
less intense selection is the strongest driver of this Red King effect, whereas
relative mutation rates and generation times have little effect. In antagonistic
interactions, faster evolution by any means is beneficial. Our results provide
insight into the demographic evolution of symbionts. '
author:
- first_name: Carl
full_name: Veller, Carl
last_name: Veller
- first_name: Laura
full_name: Hayward, Laura
last_name: Hayward
- first_name: Martin
full_name: Nowak, Martin
last_name: Nowak
- first_name: Christian
full_name: Hilbe, Christian
id: 2FDF8F3C-F248-11E8-B48F-1D18A9856A87
last_name: Hilbe
orcid: 0000-0001-5116-955X
citation:
ama: Veller C, Hayward L, Nowak M, Hilbe C. The red queen and king in finite populations.
PNAS. 2017;114(27):E5396-E5405. doi:10.1073/pnas.1702020114
apa: Veller, C., Hayward, L., Nowak, M., & Hilbe, C. (2017). The red queen and
king in finite populations. PNAS. National Academy of Sciences. https://doi.org/10.1073/pnas.1702020114
chicago: Veller, Carl, Laura Hayward, Martin Nowak, and Christian Hilbe. “The Red
Queen and King in Finite Populations.” PNAS. National Academy of Sciences,
2017. https://doi.org/10.1073/pnas.1702020114.
ieee: C. Veller, L. Hayward, M. Nowak, and C. Hilbe, “The red queen and king in
finite populations,” PNAS, vol. 114, no. 27. National Academy of Sciences,
pp. E5396–E5405, 2017.
ista: Veller C, Hayward L, Nowak M, Hilbe C. 2017. The red queen and king in finite
populations. PNAS. 114(27), E5396–E5405.
mla: Veller, Carl, et al. “The Red Queen and King in Finite Populations.” PNAS,
vol. 114, no. 27, National Academy of Sciences, 2017, pp. E5396–405, doi:10.1073/pnas.1702020114.
short: C. Veller, L. Hayward, M. Nowak, C. Hilbe, PNAS 114 (2017) E5396–E5405.
date_created: 2018-12-11T11:48:00Z
date_published: 2017-07-03T00:00:00Z
date_updated: 2021-01-12T08:11:21Z
day: '03'
department:
- _id: KrCh
doi: 10.1073/pnas.1702020114
external_id:
pmid:
- '28630336'
intvolume: ' 114'
issue: '27'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5502615/
month: '07'
oa: 1
oa_version: Submitted Version
page: E5396 - E5405
pmid: 1
publication: PNAS
publication_identifier:
issn:
- '00278424'
publication_status: published
publisher: National Academy of Sciences
publist_id: '7002'
quality_controlled: '1'
scopus_import: 1
status: public
title: The red queen and king in finite populations
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 114
year: '2017'
...
---
_id: '700'
abstract:
- lang: eng
text: Microtubules provide the mechanical force required for chromosome separation
during mitosis. However, little is known about the dynamic (high-frequency) mechanical
properties of microtubules. Here, we theoretically propose to control the vibrations
of a doubly clamped microtubule by tip electrodes and to detect its motion via
the optomechanical coupling between the vibrational modes of the microtubule and
an optical cavity. In the presence of a red-detuned strong pump laser, this coupling
leads to optomechanical-induced transparency of an optical probe field, which
can be detected with state-of-the art technology. The center frequency and line
width of the transparency peak give the resonance frequency and damping rate of
the microtubule, respectively, while the height of the peak reveals information
about the microtubule-cavity field coupling. Our method opens the new possibilities
to gain information about the physical properties of microtubules, which will
enhance our capability to design physical cancer treatment protocols as alternatives
to chemotherapeutic drugs.
article_number: '012404'
author:
- first_name: Shabir
full_name: Barzanjeh, Shabir
id: 2D25E1F6-F248-11E8-B48F-1D18A9856A87
last_name: Barzanjeh
orcid: 0000-0003-0415-1423
- first_name: Vahid
full_name: Salari, Vahid
last_name: Salari
- first_name: Jack
full_name: Tuszynski, Jack
last_name: Tuszynski
- first_name: Michal
full_name: Cifra, Michal
last_name: Cifra
- first_name: Christoph
full_name: Simon, Christoph
last_name: Simon
citation:
ama: Barzanjeh S, Salari V, Tuszynski J, Cifra M, Simon C. Optomechanical proposal
for monitoring microtubule mechanical vibrations. Physical Review E Statistical
Nonlinear and Soft Matter Physics . 2017;96(1). doi:10.1103/PhysRevE.96.012404
apa: Barzanjeh, S., Salari, V., Tuszynski, J., Cifra, M., & Simon, C. (2017).
Optomechanical proposal for monitoring microtubule mechanical vibrations.
Physical Review E Statistical Nonlinear and Soft Matter Physics . American
Institute of Physics. https://doi.org/10.1103/PhysRevE.96.012404
chicago: Barzanjeh, Shabir, Vahid Salari, Jack Tuszynski, Michal Cifra, and Christoph
Simon. “Optomechanical Proposal for Monitoring Microtubule Mechanical Vibrations.”
Physical Review E Statistical Nonlinear and Soft Matter Physics . American
Institute of Physics, 2017. https://doi.org/10.1103/PhysRevE.96.012404.
ieee: S. Barzanjeh, V. Salari, J. Tuszynski, M. Cifra, and C. Simon, “Optomechanical
proposal for monitoring microtubule mechanical vibrations,” Physical Review
E Statistical Nonlinear and Soft Matter Physics , vol. 96, no. 1. American
Institute of Physics, 2017.
ista: Barzanjeh S, Salari V, Tuszynski J, Cifra M, Simon C. 2017. Optomechanical
proposal for monitoring microtubule mechanical vibrations. Physical Review E
Statistical Nonlinear and Soft Matter Physics . 96(1), 012404.
mla: Barzanjeh, Shabir, et al. “Optomechanical Proposal for Monitoring Microtubule
Mechanical Vibrations.” Physical Review E Statistical Nonlinear and Soft Matter
Physics , vol. 96, no. 1, 012404, American Institute of Physics, 2017, doi:10.1103/PhysRevE.96.012404.
short: S. Barzanjeh, V. Salari, J. Tuszynski, M. Cifra, C. Simon, Physical Review
E Statistical Nonlinear and Soft Matter Physics 96 (2017).
date_created: 2018-12-11T11:48:00Z
date_published: 2017-07-12T00:00:00Z
date_updated: 2023-02-23T12:56:35Z
day: '12'
department:
- _id: JoFi
doi: 10.1103/PhysRevE.96.012404
ec_funded: 1
intvolume: ' 96'
issue: '1'
language:
- iso: eng
main_file_link:
- open_access: '1'
url: https://arxiv.org/pdf/1612.07061.pdf
month: '07'
oa: 1
oa_version: Submitted Version
project:
- _id: 258047B6-B435-11E9-9278-68D0E5697425
call_identifier: H2020
grant_number: '707438'
name: 'Microwave-to-Optical Quantum Link: Quantum Teleportation and Quantum Illumination
with cavity Optomechanics'
publication: ' Physical Review E Statistical Nonlinear and Soft Matter Physics '
publication_identifier:
issn:
- '24700045'
publication_status: published
publisher: American Institute of Physics
publist_id: '6997'
quality_controlled: '1'
scopus_import: 1
status: public
title: Optomechanical proposal for monitoring microtubule mechanical vibrations
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 96
year: '2017'
...
---
_id: '701'
abstract:
- lang: eng
text: A d-dimensional simplex S is called a k-reptile (or a k-reptile simplex) if
it can be tiled by k simplices with disjoint interiors that are all mutually congruent
and similar to S. For d = 2, triangular k-reptiles exist for all k of the form
a^2, 3a^2 or a^2+b^2 and they have been completely characterized by Snover, Waiveris,
and Williams. On the other hand, the only k-reptile simplices that are known for
d ≥ 3, have k = m^d, where m is a positive integer. We substantially simplify
the proof by Matoušek and the second author that for d = 3, k-reptile tetrahedra
can exist only for k = m^3. We then prove a weaker analogue of this result for
d = 4 by showing that four-dimensional k-reptile simplices can exist only for
k = m^2.
author:
- first_name: Jan
full_name: Kynčl, Jan
last_name: Kynčl
- first_name: Zuzana
full_name: Patakova, Zuzana
id: 48B57058-F248-11E8-B48F-1D18A9856A87
last_name: Patakova
orcid: 0000-0002-3975-1683
citation:
ama: Kynčl J, Patakova Z. On the nonexistence of k reptile simplices in ℝ^3 and
ℝ^4. The Electronic Journal of Combinatorics. 2017;24(3):1-44.
apa: Kynčl, J., & Patakova, Z. (2017). On the nonexistence of k reptile simplices
in ℝ^3 and ℝ^4. The Electronic Journal of Combinatorics. International
Press.
chicago: Kynčl, Jan, and Zuzana Patakova. “On the Nonexistence of k Reptile Simplices
in ℝ^3 and ℝ^4.” The Electronic Journal of Combinatorics. International
Press, 2017.
ieee: J. Kynčl and Z. Patakova, “On the nonexistence of k reptile simplices in ℝ^3
and ℝ^4,” The Electronic Journal of Combinatorics, vol. 24, no. 3. International
Press, pp. 1–44, 2017.
ista: Kynčl J, Patakova Z. 2017. On the nonexistence of k reptile simplices in ℝ^3
and ℝ^4. The Electronic Journal of Combinatorics. 24(3), 1–44.
mla: Kynčl, Jan, and Zuzana Patakova. “On the Nonexistence of k Reptile Simplices
in ℝ^3 and ℝ^4.” The Electronic Journal of Combinatorics, vol. 24, no.
3, International Press, 2017, pp. 1–44.
short: J. Kynčl, Z. Patakova, The Electronic Journal of Combinatorics 24 (2017)
1–44.
date_created: 2018-12-11T11:48:00Z
date_published: 2017-07-14T00:00:00Z
date_updated: 2021-01-12T08:11:28Z
day: '14'
ddc:
- '500'
department:
- _id: UlWa
file:
- access_level: open_access
checksum: a431e573e31df13bc0f66de3061006ec
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:14:25Z
date_updated: 2020-07-14T12:47:47Z
file_id: '5077'
file_name: IST-2018-984-v1+1_Patakova_on_the_nonexistence_of_k-reptile_simplices_in_R_3_and_R_4_2017.pdf
file_size: 544042
relation: main_file
file_date_updated: 2020-07-14T12:47:47Z
has_accepted_license: '1'
intvolume: ' 24'
issue: '3'
language:
- iso: eng
month: '07'
oa: 1
oa_version: Submitted Version
page: 1-44
publication: The Electronic Journal of Combinatorics
publication_identifier:
issn:
- '10778926'
publication_status: published
publisher: International Press
publist_id: '6996'
pubrep_id: '984'
quality_controlled: '1'
status: public
title: On the nonexistence of k reptile simplices in ℝ^3 and ℝ^4
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 24
year: '2017'
...
---
_id: '702'
abstract:
- lang: eng
text: "Leading autism-associated mutation in mouse partially mimics human disorder.\r\n\r\n"
author:
- first_name: Gaia
full_name: Novarino, Gaia
id: 3E57A680-F248-11E8-B48F-1D18A9856A87
last_name: Novarino
orcid: 0000-0002-7673-7178
citation:
ama: Novarino G. The riddle of CHD8 haploinsufficiency in autism spectrum disorder.
Science Translational Medicine. 2017;9(399):eaao0972. doi:10.1126/scitranslmed.aao0972
apa: Novarino, G. (2017). The riddle of CHD8 haploinsufficiency in autism spectrum
disorder. Science Translational Medicine. American Association for the
Advancement of Science. https://doi.org/10.1126/scitranslmed.aao0972
chicago: Novarino, Gaia. “The Riddle of CHD8 Haploinsufficiency in Autism Spectrum
Disorder.” Science Translational Medicine. American Association for the
Advancement of Science, 2017. https://doi.org/10.1126/scitranslmed.aao0972.
ieee: G. Novarino, “The riddle of CHD8 haploinsufficiency in autism spectrum disorder,”
Science Translational Medicine, vol. 9, no. 399. American Association for
the Advancement of Science, p. eaao0972, 2017.
ista: Novarino G. 2017. The riddle of CHD8 haploinsufficiency in autism spectrum
disorder. Science Translational Medicine. 9(399), eaao0972.
mla: Novarino, Gaia. “The Riddle of CHD8 Haploinsufficiency in Autism Spectrum Disorder.”
Science Translational Medicine, vol. 9, no. 399, American Association for
the Advancement of Science, 2017, p. eaao0972, doi:10.1126/scitranslmed.aao0972.
short: G. Novarino, Science Translational Medicine 9 (2017) eaao0972.
date_created: 2018-12-11T11:48:01Z
date_published: 2017-07-19T00:00:00Z
date_updated: 2021-01-12T08:11:31Z
day: '19'
department:
- _id: GaNo
doi: 10.1126/scitranslmed.aao0972
intvolume: ' 9'
issue: '399'
language:
- iso: eng
month: '07'
oa_version: None
page: eaao0972
publication: Science Translational Medicine
publication_identifier:
issn:
- '19466234'
publication_status: published
publisher: American Association for the Advancement of Science
publist_id: '6993'
quality_controlled: '1'
scopus_import: 1
status: public
title: The riddle of CHD8 haploinsufficiency in autism spectrum disorder
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 9
year: '2017'
...
---
_id: '704'
abstract:
- lang: eng
text: 'How the organization of genes on a chromosome shapes adaptation is essential
for understanding evolutionary paths. Here, we investigate how adaptation to rapidly
increasing levels of antibiotic depends on the chromosomal neighborhood of a drug-resistance
gene inserted at different positions of the Escherichia coli chromosome. Using
a dual-fluorescence reporter that allows us to distinguish gene amplifications
from other up-mutations, we track in real-time adaptive changes in expression
of the drug-resistance gene. We find that the relative contribution of several
mutation types differs systematically between loci due to properties of neighboring
genes: essentiality, expression, orientation, termination, and presence of duplicates.
These properties determine rate and fitness effects of gene amplification, deletions,
and mutations compromising transcriptional termination. Thus, the adaptive potential
of a gene under selection is a system-property with a complex genetic basis that
is specific for each chromosomal locus, and it can be inferred from detailed functional
and genomic data.'
article_number: e25100
author:
- first_name: Magdalena
full_name: Steinrück, Magdalena
id: 2C023F40-F248-11E8-B48F-1D18A9856A87
last_name: Steinrück
orcid: 0000-0003-1229-9719
- first_name: Calin C
full_name: Guet, Calin C
id: 47F8433E-F248-11E8-B48F-1D18A9856A87
last_name: Guet
orcid: 0000-0001-6220-2052
citation:
ama: Steinrück M, Guet CC. Complex chromosomal neighborhood effects determine the
adaptive potential of a gene under selection. eLife. 2017;6. doi:10.7554/eLife.25100
apa: Steinrück, M., & Guet, C. C. (2017). Complex chromosomal neighborhood effects
determine the adaptive potential of a gene under selection. ELife. eLife
Sciences Publications. https://doi.org/10.7554/eLife.25100
chicago: Steinrück, Magdalena, and Calin C Guet. “Complex Chromosomal Neighborhood
Effects Determine the Adaptive Potential of a Gene under Selection.” ELife.
eLife Sciences Publications, 2017. https://doi.org/10.7554/eLife.25100.
ieee: M. Steinrück and C. C. Guet, “Complex chromosomal neighborhood effects determine
the adaptive potential of a gene under selection,” eLife, vol. 6. eLife
Sciences Publications, 2017.
ista: Steinrück M, Guet CC. 2017. Complex chromosomal neighborhood effects determine
the adaptive potential of a gene under selection. eLife. 6, e25100.
mla: Steinrück, Magdalena, and Calin C. Guet. “Complex Chromosomal Neighborhood
Effects Determine the Adaptive Potential of a Gene under Selection.” ELife,
vol. 6, e25100, eLife Sciences Publications, 2017, doi:10.7554/eLife.25100.
short: M. Steinrück, C.C. Guet, ELife 6 (2017).
date_created: 2018-12-11T11:48:01Z
date_published: 2017-07-25T00:00:00Z
date_updated: 2024-03-25T23:30:14Z
day: '25'
ddc:
- '576'
department:
- _id: CaGu
doi: 10.7554/eLife.25100
file:
- access_level: open_access
checksum: 6b908b5db9f61f6820ebd7f8fa815571
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:12:54Z
date_updated: 2020-07-14T12:47:48Z
file_id: '4975'
file_name: IST-2017-890-v1+1_elife-25100-v1.pdf
file_size: 2092088
relation: main_file
- access_level: open_access
checksum: ca21530389b720243552678125fdba35
content_type: application/pdf
creator: system
date_created: 2018-12-12T10:12:55Z
date_updated: 2020-07-14T12:47:48Z
file_id: '4976'
file_name: IST-2017-890-v1+2_elife-25100-figures-v1.pdf
file_size: 3428681
relation: main_file
file_date_updated: 2020-07-14T12:47:48Z
has_accepted_license: '1'
intvolume: ' 6'
language:
- iso: eng
month: '07'
oa: 1
oa_version: Published Version
publication: eLife
publication_identifier:
issn:
- 2050084X
publication_status: published
publisher: eLife Sciences Publications
publist_id: '6990'
pubrep_id: '890'
quality_controlled: '1'
related_material:
record:
- id: '5564'
relation: popular_science
status: public
- id: '26'
relation: dissertation_contains
status: public
scopus_import: 1
status: public
title: Complex chromosomal neighborhood effects determine the adaptive potential of
a gene under selection
tmp:
image: /images/cc_by.png
legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
short: CC BY (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 6
year: '2017'
...
---
_id: '706'
abstract:
- lang: eng
text: A hippocampal mossy fiber synapse has a complex structure and is implicated
in learning and memory. In this synapse, the mossy fiber boutons attach to the
dendritic shaft by puncta adherentia junctions and wrap around a multiply-branched
spine, forming synaptic junctions. We have recently shown using transmission electron
microscopy, immunoelectron microscopy and serial block face-scanning electron
microscopy that atypical puncta adherentia junctions are formed in the afadin-deficient
mossy fiber synapse and that the complexity of postsynaptic spines and mossy fiber
boutons, the number of spine heads, the area of postsynaptic densities and the
density of synaptic vesicles docked to active zones are decreased in the afadin-deficient
synapse. We investigated here the roles of afadin in the functional differentiations
of the mossy fiber synapse using the afadin-deficient mice. The electrophysiological
studies showed that both the release probability of glutamate and the postsynaptic
responsiveness to glutamate were markedly reduced, but not completely lost, in
the afadin-deficient mossy fiber synapse, whereas neither long-term potentiation
nor long-term depression was affected. These results indicate that afadin plays
roles in the functional differentiations of the presynapse and the postsynapse
of the hippocampal mossy fiber synapse.
author:
- first_name: Xiaoqi
full_name: Geng, Xiaoqi
id: 3395256A-F248-11E8-B48F-1D18A9856A87
last_name: Geng
- first_name: Tomohiko
full_name: Maruo, Tomohiko
last_name: Maruo
- first_name: Kenji
full_name: Mandai, Kenji
last_name: Mandai
- first_name: Irwan
full_name: Supriyanto, Irwan
last_name: Supriyanto
- first_name: Muneaki
full_name: Miyata, Muneaki
last_name: Miyata
- first_name: Shotaro
full_name: Sakakibara, Shotaro
last_name: Sakakibara
- first_name: Akira
full_name: Mizoguchi, Akira
last_name: Mizoguchi
- first_name: Yoshimi
full_name: Takai, Yoshimi
last_name: Takai
- first_name: Masahiro
full_name: Mori, Masahiro
last_name: Mori
citation:
ama: Geng X, Maruo T, Mandai K, et al. Roles of afadin in functional differentiations
of hippocampal mossy fiber synapse. Genes to Cells. 2017;22(8):715-722.
doi:10.1111/gtc.12508
apa: Geng, X., Maruo, T., Mandai, K., Supriyanto, I., Miyata, M., Sakakibara, S.,
… Mori, M. (2017). Roles of afadin in functional differentiations of hippocampal
mossy fiber synapse. Genes to Cells. Wiley-Blackwell. https://doi.org/10.1111/gtc.12508
chicago: Geng, Xiaoqi, Tomohiko Maruo, Kenji Mandai, Irwan Supriyanto, Muneaki Miyata,
Shotaro Sakakibara, Akira Mizoguchi, Yoshimi Takai, and Masahiro Mori. “Roles
of Afadin in Functional Differentiations of Hippocampal Mossy Fiber Synapse.”
Genes to Cells. Wiley-Blackwell, 2017. https://doi.org/10.1111/gtc.12508.
ieee: X. Geng et al., “Roles of afadin in functional differentiations of
hippocampal mossy fiber synapse,” Genes to Cells, vol. 22, no. 8. Wiley-Blackwell,
pp. 715–722, 2017.
ista: Geng X, Maruo T, Mandai K, Supriyanto I, Miyata M, Sakakibara S, Mizoguchi
A, Takai Y, Mori M. 2017. Roles of afadin in functional differentiations of hippocampal
mossy fiber synapse. Genes to Cells. 22(8), 715–722.
mla: Geng, Xiaoqi, et al. “Roles of Afadin in Functional Differentiations of Hippocampal
Mossy Fiber Synapse.” Genes to Cells, vol. 22, no. 8, Wiley-Blackwell,
2017, pp. 715–22, doi:10.1111/gtc.12508.
short: X. Geng, T. Maruo, K. Mandai, I. Supriyanto, M. Miyata, S. Sakakibara, A.
Mizoguchi, Y. Takai, M. Mori, Genes to Cells 22 (2017) 715–722.
date_created: 2018-12-11T11:48:02Z
date_published: 2017-08-01T00:00:00Z
date_updated: 2021-01-12T08:11:37Z
day: '01'
department:
- _id: PeJo
doi: 10.1111/gtc.12508
intvolume: ' 22'
issue: '8'
language:
- iso: eng
month: '08'
oa_version: None
page: 715 - 722
publication: Genes to Cells
publication_identifier:
issn:
- '13569597'
publication_status: published
publisher: Wiley-Blackwell
publist_id: '6987'
quality_controlled: '1'
scopus_import: 1
status: public
title: Roles of afadin in functional differentiations of hippocampal mossy fiber synapse
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 22
year: '2017'
...