[{"oa":1,"publication_identifier":{"issn":["01795376"]},"status":"public","month":"06","year":"2017","date_updated":"2023-09-20T12:01:28Z","issue":"4","abstract":[{"lang":"eng","text":"Let X and Y be finite simplicial sets (e.g. finite simplicial complexes), both equipped with a free simplicial action of a finite group G. Assuming that Y is d-connected and dimX≤2d, for some d≥1, we provide an algorithm that computes the set of all equivariant homotopy classes of equivariant continuous maps |X|→|Y|; the existence of such a map can be decided even for dimX≤2d+1. This yields the first algorithm for deciding topological embeddability of a k-dimensional finite simplicial complex into Rn under the condition k≤23n−1. More generally, we present an algorithm that, given a lifting-extension problem satisfying an appropriate stability assumption, computes the set of all homotopy classes of solutions. This result is new even in the non-equivariant situation."}],"_id":"1073","scopus_import":"1","main_file_link":[{"open_access":"1","url":"https://arxiv.org/abs/1307.6444"}],"citation":{"short":"M. Čadek, M. Krcál, L. Vokřínek, Discrete &#38; Computational Geometry 54 (2017) 915–965.","ieee":"M. Čadek, M. Krcál, and L. Vokřínek, “Algorithmic solvability of the lifting extension problem,” <i>Discrete &#38; Computational Geometry</i>, vol. 54, no. 4. Springer, pp. 915–965, 2017.","ama":"Čadek M, Krcál M, Vokřínek L. Algorithmic solvability of the lifting extension problem. <i>Discrete &#38; Computational Geometry</i>. 2017;54(4):915-965. doi:<a href=\"https://doi.org/10.1007/s00454-016-9855-6\">10.1007/s00454-016-9855-6</a>","chicago":"Čadek, Martin, Marek Krcál, and Lukáš Vokřínek. “Algorithmic Solvability of the Lifting Extension Problem.” <i>Discrete &#38; Computational Geometry</i>. Springer, 2017. <a href=\"https://doi.org/10.1007/s00454-016-9855-6\">https://doi.org/10.1007/s00454-016-9855-6</a>.","apa":"Čadek, M., Krcál, M., &#38; Vokřínek, L. (2017). Algorithmic solvability of the lifting extension problem. <i>Discrete &#38; Computational Geometry</i>. Springer. <a href=\"https://doi.org/10.1007/s00454-016-9855-6\">https://doi.org/10.1007/s00454-016-9855-6</a>","ista":"Čadek M, Krcál M, Vokřínek L. 2017. Algorithmic solvability of the lifting extension problem. Discrete &#38; Computational Geometry. 54(4), 915–965.","mla":"Čadek, Martin, et al. “Algorithmic Solvability of the Lifting Extension Problem.” <i>Discrete &#38; Computational Geometry</i>, vol. 54, no. 4, Springer, 2017, pp. 915–65, doi:<a href=\"https://doi.org/10.1007/s00454-016-9855-6\">10.1007/s00454-016-9855-6</a>."},"intvolume":"        54","publication_status":"published","doi":"10.1007/s00454-016-9855-6","date_published":"2017-06-01T00:00:00Z","oa_version":"Submitted Version","title":"Algorithmic solvability of the lifting extension problem","publisher":"Springer","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","day":"01","isi":1,"author":[{"last_name":"Čadek","first_name":"Martin","full_name":"Čadek, Martin"},{"first_name":"Marek","last_name":"Krcál","id":"33E21118-F248-11E8-B48F-1D18A9856A87","full_name":"Krcál, Marek"},{"first_name":"Lukáš","last_name":"Vokřínek","full_name":"Vokřínek, Lukáš"}],"publist_id":"6309","article_processing_charge":"No","external_id":{"isi":["000400072700008"]},"volume":54,"quality_controlled":"1","page":"915 - 965","date_created":"2018-12-11T11:50:00Z","publication":"Discrete & Computational Geometry","language":[{"iso":"eng"}],"department":[{"_id":"UlWa"}],"type":"journal_article"},{"type":"journal_article","department":[{"_id":"NiBa"}],"language":[{"iso":"eng"}],"publication":"Genetics","date_created":"2018-12-11T11:50:00Z","page":"1335 - 1351","volume":205,"quality_controlled":"1","external_id":{"isi":["000395807200023"]},"related_material":{"record":[{"id":"200","relation":"dissertation_contains","status":"public"}]},"article_processing_charge":"No","publist_id":"6307","isi":1,"author":[{"full_name":"Ringbauer, Harald","first_name":"Harald","orcid":"0000-0002-4884-9682","id":"417FCFF4-F248-11E8-B48F-1D18A9856A87","last_name":"Ringbauer"},{"first_name":"Graham","last_name":"Coop","full_name":"Coop, Graham"},{"id":"4880FE40-F248-11E8-B48F-1D18A9856A87","last_name":"Barton","orcid":"0000-0002-8548-5240","first_name":"Nicholas H","full_name":"Barton, Nicholas H"}],"project":[{"name":"Limits to selection in biology and in evolutionary computation","call_identifier":"FP7","_id":"25B07788-B435-11E9-9278-68D0E5697425","grant_number":"250152"}],"day":"01","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"Genetics Society of America","title":"Inferring recent demography from isolation by distance of long shared sequence blocks","oa_version":"Preprint","date_published":"2017-03-01T00:00:00Z","doi":"10.1534/genetics.116.196220","publication_status":"published","citation":{"mla":"Ringbauer, Harald, et al. “Inferring Recent Demography from Isolation by Distance of Long Shared Sequence Blocks.” <i>Genetics</i>, vol. 205, no. 3, Genetics Society of America, 2017, pp. 1335–51, doi:<a href=\"https://doi.org/10.1534/genetics.116.196220\">10.1534/genetics.116.196220</a>.","apa":"Ringbauer, H., Coop, G., &#38; Barton, N. H. (2017). Inferring recent demography from isolation by distance of long shared sequence blocks. <i>Genetics</i>. Genetics Society of America. <a href=\"https://doi.org/10.1534/genetics.116.196220\">https://doi.org/10.1534/genetics.116.196220</a>","ista":"Ringbauer H, Coop G, Barton NH. 2017. Inferring recent demography from isolation by distance of long shared sequence blocks. Genetics. 205(3), 1335–1351.","chicago":"Ringbauer, Harald, Graham Coop, and Nicholas H Barton. “Inferring Recent Demography from Isolation by Distance of Long Shared Sequence Blocks.” <i>Genetics</i>. Genetics Society of America, 2017. <a href=\"https://doi.org/10.1534/genetics.116.196220\">https://doi.org/10.1534/genetics.116.196220</a>.","short":"H. Ringbauer, G. Coop, N.H. Barton, Genetics 205 (2017) 1335–1351.","ieee":"H. Ringbauer, G. Coop, and N. H. Barton, “Inferring recent demography from isolation by distance of long shared sequence blocks,” <i>Genetics</i>, vol. 205, no. 3. Genetics Society of America, pp. 1335–1351, 2017.","ama":"Ringbauer H, Coop G, Barton NH. Inferring recent demography from isolation by distance of long shared sequence blocks. <i>Genetics</i>. 2017;205(3):1335-1351. doi:<a href=\"https://doi.org/10.1534/genetics.116.196220\">10.1534/genetics.116.196220</a>"},"intvolume":"       205","scopus_import":"1","main_file_link":[{"open_access":"1","url":"http://www.biorxiv.org/content/early/2016/09/23/076810"}],"_id":"1074","issue":"3","abstract":[{"text":"Recently it has become feasible to detect long blocks of nearly identical sequence shared between pairs of genomes. These IBD blocks are direct traces of recent coalescence events and, as such, contain ample signal to infer recent demography. Here, we examine sharing of such blocks in two-dimensional populations with local migration. Using a diffusion approximation to trace genetic ancestry, we derive analytical formulae for patterns of isolation by distance of IBD blocks, which can also incorporate recent population density changes. We introduce an inference scheme that uses a composite likelihood approach to fit these formulae. We then extensively evaluate our theory and inference method on a range of scenarios using simulated data. We first validate the diffusion approximation by showing that the theoretical results closely match the simulated block sharing patterns. We then demonstrate that our inference scheme can accurately and robustly infer dispersal rate and effective density, as well as bounds on recent dynamics of population density. To demonstrate an application, we use our estimation scheme to explore the fit of a diffusion model to Eastern European samples in the POPRES data set. We show that ancestry diffusing with a rate of σ ≈ 50–100 km/√gen during the last centuries, combined with accelerating population growth, can explain the observed exponential decay of block sharing with increasing pairwise sample distance.","lang":"eng"}],"date_updated":"2025-05-28T11:42:51Z","year":"2017","month":"03","ec_funded":1,"status":"public","publication_identifier":{"issn":["00166731"]},"oa":1},{"publication":"APS March Meeting 2017","date_created":"2022-02-08T09:49:17Z","language":[{"iso":"eng"}],"type":"conference","article_processing_charge":"No","quality_controlled":"1","volume":62,"extern":"1","day":"01","author":[{"full_name":"Polshyn, Hryhoriy","orcid":"0000-0001-8223-8896","first_name":"Hryhoriy","id":"edfc7cb1-526e-11ec-b05a-e6ecc27e4e48","last_name":"Polshyn"},{"full_name":"Naibert, Tyler","first_name":"Tyler","last_name":"Naibert"},{"first_name":"Raffi","last_name":"Budakian","full_name":"Budakian, Raffi"}],"title":" Probing and controlling fluxoid states in multiply-connected mesoscopic superconducting structures","user_id":"8b945eb4-e2f2-11eb-945a-df72226e66a9","publisher":"American Physical Society","alternative_title":["Bulletin of the American Physical Society"],"conference":{"end_date":"2017-03-17","location":"New Orleans, LA, United States","start_date":"2017-03-13","name":"APS: American Physical Society"},"publication_status":"published","date_published":"2017-03-01T00:00:00Z","oa_version":"Published Version","main_file_link":[{"open_access":"1","url":"https://meetings.aps.org/Meeting/MAR17/Session/P39.11"}],"intvolume":"        62","citation":{"ista":"Polshyn H, Naibert T, Budakian R. 2017.  Probing and controlling fluxoid states in multiply-connected mesoscopic superconducting structures. APS March Meeting 2017. APS: American Physical Society, Bulletin of the American Physical Society, vol. 62, P39.00011.","apa":"Polshyn, H., Naibert, T., &#38; Budakian, R. (2017).  Probing and controlling fluxoid states in multiply-connected mesoscopic superconducting structures. In <i>APS March Meeting 2017</i> (Vol. 62). New Orleans, LA, United States: American Physical Society.","mla":"Polshyn, Hryhoriy, et al. “ Probing and Controlling Fluxoid States in Multiply-Connected Mesoscopic Superconducting Structures.” <i>APS March Meeting 2017</i>, vol. 62, no. 4, P39.00011, American Physical Society, 2017.","chicago":"Polshyn, Hryhoriy, Tyler Naibert, and Raffi Budakian. “ Probing and Controlling Fluxoid States in Multiply-Connected Mesoscopic Superconducting Structures.” In <i>APS March Meeting 2017</i>, Vol. 62. American Physical Society, 2017.","short":"H. Polshyn, T. Naibert, R. Budakian, in:, APS March Meeting 2017, American Physical Society, 2017.","ama":"Polshyn H, Naibert T, Budakian R.  Probing and controlling fluxoid states in multiply-connected mesoscopic superconducting structures. In: <i>APS March Meeting 2017</i>. Vol 62. American Physical Society; 2017.","ieee":"H. Polshyn, T. Naibert, and R. Budakian, “ Probing and controlling fluxoid states in multiply-connected mesoscopic superconducting structures,” in <i>APS March Meeting 2017</i>, New Orleans, LA, United States, 2017, vol. 62, no. 4."},"year":"2017","abstract":[{"lang":"eng","text":"New ways to investigate and manipulate fluxoid and vortex states of mesoscopic superconducting structures are of great interest. The states with multiple vortices or winding numbers could be useful for the study of vortex interactions and interference effects, the braiding of Majorana bound states by winding vortices, and the development of novel superconducting devices. We demonstrate a methodology based on magnetic force microscopy that allows us to induce, probe and control fluxoid states in thin wall structures comprised of multiple loops. By using micro-magnet as a source of inhomogeneous magnetic field, we can efficiently explore the configuration space of fluxoid states. Scanning over the structure reveals the energy crossing points of the lowest laying fluxoid states. This is due the strong interaction of cantilever with thermally activated fluxoid transitions at points of degeneracy. We show that measured patterns of fluxoid transitions allow to identify the states, investigate their energetics, and manipulate them. Further, we show that the dynamics of driven fluxoid transitions can be described by stochastic resonance model, which provides a unique way of measuring fluxoid transition rate and related energy barrier for chosen transitions even in complicated structures"}],"issue":"4","date_updated":"2022-02-08T10:44:35Z","_id":"10745","article_number":"P39.00011","publication_identifier":{"issn":["0003-0503"]},"oa":1,"status":"public","month":"03"},{"publication_status":"published","oa_version":"None","date_published":"2017-02-01T00:00:00Z","file_date_updated":"2018-12-12T10:08:06Z","ddc":["001"],"citation":{"apa":"Wenzl, B. (2017). An American in Allied-occupied Austria: John Dos Passos Reports on &#38;quot;The Vienna Frontier&#38;quot; In J. Parker &#38; R. Poole (Eds.), <i>Austria and America: 20th-Century Cross-Cultural Encounters</i> (Vol. 15, pp. 73–80). LIT Verlag Berlin-Münster-Wien-Zürich-London.","mla":"Wenzl, Bernhard. “An American in Allied-Occupied Austria: John Dos Passos Reports on &#38;quot;The Vienna Frontier&#38;quot;” <i>Austria and America: 20th-Century Cross-Cultural Encounters</i>, edited by Joshua Parker and Ralph Poole, vol. 15, LIT Verlag Berlin-Münster-Wien-Zürich-London, 2017, pp. 73–80.","ista":"Wenzl B. 2017.An American in Allied-occupied Austria: John Dos Passos Reports on &#38;quot;The Vienna Frontier&#38;quot; In: Austria and America: 20th-Century Cross-Cultural Encounters. American Studies in Austria, vol. 15, 73–80.","chicago":"Wenzl, Bernhard. “An American in Allied-Occupied Austria: John Dos Passos Reports on &#38;quot;The Vienna Frontier&#38;quot;” In <i>Austria and America: 20th-Century Cross-Cultural Encounters</i>, edited by Joshua Parker and Ralph Poole, 15:73–80. LIT Verlag Berlin-Münster-Wien-Zürich-London, 2017.","short":"B. Wenzl, in:, J. Parker, R. Poole (Eds.), Austria and America: 20th-Century Cross-Cultural Encounters, LIT Verlag Berlin-Münster-Wien-Zürich-London, 2017, pp. 73–80.","ieee":"B. Wenzl, “An American in Allied-occupied Austria: John Dos Passos Reports on &#38;quot;The Vienna Frontier&#38;quot;,” in <i>Austria and America: 20th-Century Cross-Cultural Encounters</i>, vol. 15, J. Parker and R. Poole, Eds. LIT Verlag Berlin-Münster-Wien-Zürich-London, 2017, pp. 73–80.","ama":"Wenzl B. An American in Allied-occupied Austria: John Dos Passos Reports on &#38;quot;The Vienna Frontier&#38;quot; In: Parker J, Poole R, eds. <i>Austria and America: 20th-Century Cross-Cultural Encounters</i>. Vol 15. LIT Verlag Berlin-Münster-Wien-Zürich-London; 2017:73-80."},"intvolume":"        15","date_updated":"2021-01-12T06:48:06Z","year":"2017","_id":"1075","status":"public","oa":1,"publication_identifier":{"isbn":["978-3643908124"]},"month":"02","has_accepted_license":"1","publication":"Austria and America: 20th-Century Cross-Cultural Encounters","page":"73 - 80","date_created":"2018-12-11T11:50:00Z","type":"book_chapter","language":[{"iso":"eng"}],"publist_id":"6306","volume":15,"editor":[{"last_name":"Parker","first_name":"Joshua","full_name":"Parker, Joshua"},{"full_name":"Poole, Ralph","last_name":"Poole","first_name":"Ralph"}],"extern":"1","day":"01","author":[{"first_name":"Bernhard","last_name":"Wenzl","id":"479E9046-F248-11E8-B48F-1D18A9856A87","full_name":"Wenzl, Bernhard"}],"file":[{"relation":"main_file","content_type":"application/pdf","file_size":380624,"file_name":"IST-2017-732-v1+1_Austria_and_America_Cross-Cultural_Encounters.pdf","date_updated":"2018-12-12T10:08:06Z","date_created":"2018-12-12T10:08:06Z","creator":"system","file_id":"4666","access_level":"open_access"}],"user_id":"2EBD1598-F248-11E8-B48F-1D18A9856A87","publisher":"LIT Verlag Berlin-Münster-Wien-Zürich-London","title":"An American in Allied-occupied Austria: John Dos Passos Reports on &quot;The Vienna Frontier&quot;","alternative_title":["American Studies in Austria"]},{"status":"public","publication_identifier":{"issn":["24699926"]},"oa":1,"month":"02","ec_funded":1,"date_updated":"2023-09-20T11:57:23Z","issue":"2","abstract":[{"lang":"eng","text":"Signatures of the Coulomb corrections in the photoelectron momentum distribution during laser-induced ionization of atoms or ions in tunneling and multiphoton regimes are investigated analytically in the case of a one-dimensional problem. A high-order Coulomb-corrected strong-field approximation is applied, where the exact continuum state in the S matrix is approximated by the eikonal Coulomb-Volkov state including the second-order corrections to the eikonal. Although without high-order corrections our theory coincides with the known analytical R-matrix (ARM) theory, we propose a simplified procedure for the matrix element derivation. Rather than matching the eikonal Coulomb-Volkov wave function with the bound state as in the ARM theory to remove the Coulomb singularity, we calculate the matrix element via the saddle-point integration method by time as well as by coordinate, and in this way avoiding the Coulomb singularity. The momentum shift in the photoelectron momentum distribution with respect to the ARM theory due to high-order corrections is analyzed for tunneling and multiphoton regimes. The relation of the quantum corrections to the tunneling delay time is discussed."}],"year":"2017","article_number":"023403","_id":"1076","main_file_link":[{"open_access":"1","url":"https://arxiv.org/abs/1609.07018"}],"scopus_import":"1","citation":{"chicago":"Klaiber, Michael, Jiří Daněk, Enderalp Yakaboylu, Karen Hatsagortsyan, and Christoph Keitel. “Strong-Field Ionization via a High-Order Coulomb-Corrected Strong-Field Approximation.” <i> Physical Review A - Atomic, Molecular, and Optical Physics</i>. American Physical Society, 2017. <a href=\"https://doi.org/10.1103/PhysRevA.95.023403\">https://doi.org/10.1103/PhysRevA.95.023403</a>.","apa":"Klaiber, M., Daněk, J., Yakaboylu, E., Hatsagortsyan, K., &#38; Keitel, C. (2017). Strong-field ionization via a high-order Coulomb-corrected strong-field approximation. <i> Physical Review A - Atomic, Molecular, and Optical Physics</i>. American Physical Society. <a href=\"https://doi.org/10.1103/PhysRevA.95.023403\">https://doi.org/10.1103/PhysRevA.95.023403</a>","mla":"Klaiber, Michael, et al. “Strong-Field Ionization via a High-Order Coulomb-Corrected Strong-Field Approximation.” <i> Physical Review A - Atomic, Molecular, and Optical Physics</i>, vol. 95, no. 2, 023403, American Physical Society, 2017, doi:<a href=\"https://doi.org/10.1103/PhysRevA.95.023403\">10.1103/PhysRevA.95.023403</a>.","ista":"Klaiber M, Daněk J, Yakaboylu E, Hatsagortsyan K, Keitel C. 2017. Strong-field ionization via a high-order Coulomb-corrected strong-field approximation.  Physical Review A - Atomic, Molecular, and Optical Physics. 95(2), 023403.","ieee":"M. Klaiber, J. Daněk, E. Yakaboylu, K. Hatsagortsyan, and C. Keitel, “Strong-field ionization via a high-order Coulomb-corrected strong-field approximation,” <i> Physical Review A - Atomic, Molecular, and Optical Physics</i>, vol. 95, no. 2. American Physical Society, 2017.","ama":"Klaiber M, Daněk J, Yakaboylu E, Hatsagortsyan K, Keitel C. Strong-field ionization via a high-order Coulomb-corrected strong-field approximation. <i> Physical Review A - Atomic, Molecular, and Optical Physics</i>. 2017;95(2). doi:<a href=\"https://doi.org/10.1103/PhysRevA.95.023403\">10.1103/PhysRevA.95.023403</a>","short":"M. Klaiber, J. Daněk, E. Yakaboylu, K. Hatsagortsyan, C. Keitel,  Physical Review A - Atomic, Molecular, and Optical Physics 95 (2017)."},"intvolume":"        95","publication_status":"published","doi":"10.1103/PhysRevA.95.023403","oa_version":"Submitted Version","date_published":"2017-02-01T00:00:00Z","publisher":"American Physical Society","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","title":"Strong-field ionization via a high-order Coulomb-corrected strong-field approximation","day":"01","project":[{"_id":"25681D80-B435-11E9-9278-68D0E5697425","grant_number":"291734","name":"International IST Postdoc Fellowship Programme","call_identifier":"FP7"}],"author":[{"full_name":"Klaiber, Michael","first_name":"Michael","last_name":"Klaiber"},{"last_name":"Daněk","first_name":"Jiří","full_name":"Daněk, Jiří"},{"full_name":"Yakaboylu, Enderalp","first_name":"Enderalp","orcid":"0000-0001-5973-0874","id":"38CB71F6-F248-11E8-B48F-1D18A9856A87","last_name":"Yakaboylu"},{"full_name":"Hatsagortsyan, Karen","first_name":"Karen","last_name":"Hatsagortsyan"},{"first_name":"Christoph","last_name":"Keitel","full_name":"Keitel, Christoph"}],"isi":1,"publist_id":"6305","article_processing_charge":"No","external_id":{"isi":["000400571700011"]},"volume":95,"quality_controlled":"1","date_created":"2018-12-11T11:50:01Z","publication":" Physical Review A - Atomic, Molecular, and Optical Physics","type":"journal_article","language":[{"iso":"eng"}],"department":[{"_id":"MiLe"}]},{"month":"01","ec_funded":1,"status":"public","oa":1,"publication_identifier":{"issn":["17425689"]},"article_number":"20160139","_id":"1077","abstract":[{"lang":"eng","text":"Viral capsids are structurally constrained by interactions among the amino acids (AAs) of their constituent proteins. Therefore, epistasis is expected to evolve among physically interacting sites and to influence the rates of substitution. To study the evolution of epistasis, we focused on the major structural protein of the fX174 phage family by first reconstructing the ancestral protein sequences of 18 species using a Bayesian statistical framework. The inferred ancestral reconstruction differed at eight AAs, for a total of 256 possible ancestral haplotypes. For each ancestral haplotype and the extant species, we estimated, in silico, the distribution of free energies and epistasis of the capsid structure. We found that free energy has not significantly increased but epistasis has. We decomposed epistasis up to fifth order and found that higher-order epistasis sometimes compensates pairwise interactions making the free energy seem additive. The dN/dS ratio is low, suggesting strong purifying selection, and that structure is under stabilizing selection. We synthesized phages carrying ancestral haplotypes of the coat protein gene and measured their fitness experimentally. Our findings indicate that stabilizing mutations can have higher fitness, and that fitness optima do not necessarily coincide with energy minima."}],"issue":"126","date_updated":"2025-05-28T11:42:51Z","year":"2017","intvolume":"        14","citation":{"apa":"Fernandes Redondo, R. A., de Vladar, H., Włodarski, T., &#38; Bollback, J. P. (2017). Evolutionary interplay between structure, energy and epistasis in the coat protein of the ϕX174 phage family. <i>Journal of the Royal Society Interface</i>. Royal Society of London. <a href=\"https://doi.org/10.1098/rsif.2016.0139\">https://doi.org/10.1098/rsif.2016.0139</a>","mla":"Fernandes Redondo, Rodrigo A., et al. “Evolutionary Interplay between Structure, Energy and Epistasis in the Coat Protein of the ΦX174 Phage Family.” <i>Journal of the Royal Society Interface</i>, vol. 14, no. 126, 20160139, Royal Society of London, 2017, doi:<a href=\"https://doi.org/10.1098/rsif.2016.0139\">10.1098/rsif.2016.0139</a>.","ista":"Fernandes Redondo RA, de Vladar H, Włodarski T, Bollback JP. 2017. Evolutionary interplay between structure, energy and epistasis in the coat protein of the ϕX174 phage family. Journal of the Royal Society Interface. 14(126), 20160139.","chicago":"Fernandes Redondo, Rodrigo A, Harold de Vladar, Tomasz Włodarski, and Jonathan P Bollback. “Evolutionary Interplay between Structure, Energy and Epistasis in the Coat Protein of the ΦX174 Phage Family.” <i>Journal of the Royal Society Interface</i>. Royal Society of London, 2017. <a href=\"https://doi.org/10.1098/rsif.2016.0139\">https://doi.org/10.1098/rsif.2016.0139</a>.","short":"R.A. Fernandes Redondo, H. de Vladar, T. Włodarski, J.P. Bollback, Journal of the Royal Society Interface 14 (2017).","ama":"Fernandes Redondo RA, de Vladar H, Włodarski T, Bollback JP. Evolutionary interplay between structure, energy and epistasis in the coat protein of the ϕX174 phage family. <i>Journal of the Royal Society Interface</i>. 2017;14(126). doi:<a href=\"https://doi.org/10.1098/rsif.2016.0139\">10.1098/rsif.2016.0139</a>","ieee":"R. A. Fernandes Redondo, H. de Vladar, T. Włodarski, and J. P. Bollback, “Evolutionary interplay between structure, energy and epistasis in the coat protein of the ϕX174 phage family,” <i>Journal of the Royal Society Interface</i>, vol. 14, no. 126. Royal Society of London, 2017."},"scopus_import":"1","file_date_updated":"2019-01-18T09:14:02Z","ddc":["570"],"oa_version":"Published Version","date_published":"2017-01-04T00:00:00Z","doi":"10.1098/rsif.2016.0139","publication_status":"published","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"Royal Society of London","title":"Evolutionary interplay between structure, energy and epistasis in the coat protein of the ϕX174 phage family","author":[{"full_name":"Fernandes Redondo, Rodrigo A","id":"409D5C96-F248-11E8-B48F-1D18A9856A87","last_name":"Fernandes Redondo","orcid":"0000-0002-5837-2793","first_name":"Rodrigo A"},{"full_name":"Vladar, Harold","orcid":"0000-0002-5985-7653","first_name":"Harold","last_name":"Vladar","id":"2A181218-F248-11E8-B48F-1D18A9856A87"},{"full_name":"Włodarski, Tomasz","last_name":"Włodarski","first_name":"Tomasz"},{"full_name":"Bollback, Jonathan P","orcid":"0000-0002-4624-4612","first_name":"Jonathan P","last_name":"Bollback","id":"2C6FA9CC-F248-11E8-B48F-1D18A9856A87"}],"isi":1,"project":[{"_id":"25B07788-B435-11E9-9278-68D0E5697425","grant_number":"250152","call_identifier":"FP7","name":"Limits to selection in biology and in evolutionary computation"},{"name":"Selective Barriers to Horizontal Gene Transfer","call_identifier":"H2020","grant_number":"648440","_id":"2578D616-B435-11E9-9278-68D0E5697425"}],"file":[{"file_size":1092015,"success":1,"relation":"main_file","content_type":"application/pdf","date_created":"2019-01-18T09:14:02Z","date_updated":"2019-01-18T09:14:02Z","file_name":"2017_JRSI_Redondo.pdf","access_level":"open_access","creator":"dernst","file_id":"5843"}],"day":"04","tmp":{"short":"CC BY (4.0)","image":"/images/cc_by.png","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode"},"quality_controlled":"1","volume":14,"external_id":{"isi":["000393380400001"]},"related_material":{"record":[{"status":"public","relation":"research_data","id":"9864"}]},"article_processing_charge":"Yes (in subscription journal)","publist_id":"6303","type":"journal_article","department":[{"_id":"NiBa"},{"_id":"JoBo"}],"language":[{"iso":"eng"}],"has_accepted_license":"1","publication":"Journal of the Royal Society Interface","date_created":"2018-12-11T11:50:01Z"},{"file":[{"date_updated":"2018-12-12T10:16:31Z","file_name":"IST-2017-808-v1+1_2017_VWangenheim_list.pdf","date_created":"2018-12-12T10:16:31Z","creator":"system","file_id":"5219","access_level":"open_access","relation":"main_file","content_type":"application/pdf","file_size":57678},{"date_created":"2018-12-12T10:16:32Z","file_name":"IST-2017-808-v1+2_2017_VWangenheim_article.pdf","date_updated":"2018-12-12T10:16:32Z","access_level":"open_access","file_id":"5220","creator":"system","file_size":1317820,"relation":"main_file","content_type":"application/pdf"}],"project":[{"grant_number":"291734","_id":"25681D80-B435-11E9-9278-68D0E5697425","call_identifier":"FP7","name":"International IST Postdoc Fellowship Programme"},{"grant_number":"282300","_id":"25716A02-B435-11E9-9278-68D0E5697425","call_identifier":"FP7","name":"Polarity and subcellular dynamics in plants"}],"isi":1,"author":[{"full_name":"Von Wangenheim, Daniel","first_name":"Daniel","orcid":"0000-0002-6862-1247","id":"49E91952-F248-11E8-B48F-1D18A9856A87","last_name":"Von Wangenheim"},{"full_name":"Hauschild, Robert","first_name":"Robert","orcid":"0000-0001-9843-3522","id":"4E01D6B4-F248-11E8-B48F-1D18A9856A87","last_name":"Hauschild"},{"full_name":"Friml, Jirí","last_name":"Friml","id":"4159519E-F248-11E8-B48F-1D18A9856A87","first_name":"Jirí","orcid":"0000-0002-8302-7596"}],"day":"18","title":"Light sheet fluorescence microscopy of plant roots growing on the surface of a gel","publisher":"Journal of Visualized Experiments","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","language":[{"iso":"eng"}],"department":[{"_id":"JiFr"},{"_id":"Bio"}],"type":"journal_article","date_created":"2018-12-11T11:50:01Z","publication":"Journal of visualized experiments JoVE","has_accepted_license":"1","related_material":{"record":[{"relation":"popular_science","status":"public","id":"5565"}]},"external_id":{"isi":["000397847200041"]},"volume":2017,"publist_id":"6302","article_processing_charge":"No","pubrep_id":"808","_id":"1078","article_number":"e55044","acknowledged_ssus":[{"_id":"M-Shop"},{"_id":"Bio"}],"year":"2017","date_updated":"2025-05-07T11:12:33Z","issue":"119","abstract":[{"lang":"eng","text":"One of the key questions in understanding plant development is how single cells behave in a larger context of the tissue. Therefore, it requires the observation of the whole organ with a high spatial- as well as temporal resolution over prolonged periods of time, which may cause photo-toxic effects. This protocol shows a plant sample preparation method for light-sheet microscopy, which is characterized by mounting the plant vertically on the surface of a gel. The plant is mounted in such a way that the roots are submerged in a liquid medium while the leaves remain in the air. In order to ensure photosynthetic activity of the plant, a custom-made lighting system illuminates the leaves. To keep the roots in darkness the water surface is covered with sheets of black plastic foil. This method allows long-term imaging of plant organ development in standardized conditions. "}],"ec_funded":1,"month":"01","oa":1,"status":"public","date_published":"2017-01-18T00:00:00Z","oa_version":"Published Version","publication_status":"published","doi":"10.3791/55044","citation":{"apa":"von Wangenheim, D., Hauschild, R., &#38; Friml, J. (2017). Light sheet fluorescence microscopy of plant roots growing on the surface of a gel. <i>Journal of Visualized Experiments JoVE</i>. Journal of Visualized Experiments. <a href=\"https://doi.org/10.3791/55044\">https://doi.org/10.3791/55044</a>","mla":"von Wangenheim, Daniel, et al. “Light Sheet Fluorescence Microscopy of Plant Roots Growing on the Surface of a Gel.” <i>Journal of Visualized Experiments JoVE</i>, vol. 2017, no. 119, e55044, Journal of Visualized Experiments, 2017, doi:<a href=\"https://doi.org/10.3791/55044\">10.3791/55044</a>.","ista":"von Wangenheim D, Hauschild R, Friml J. 2017. Light sheet fluorescence microscopy of plant roots growing on the surface of a gel. Journal of visualized experiments JoVE. 2017(119), e55044.","chicago":"Wangenheim, Daniel von, Robert Hauschild, and Jiří Friml. “Light Sheet Fluorescence Microscopy of Plant Roots Growing on the Surface of a Gel.” <i>Journal of Visualized Experiments JoVE</i>. Journal of Visualized Experiments, 2017. <a href=\"https://doi.org/10.3791/55044\">https://doi.org/10.3791/55044</a>.","short":"D. von Wangenheim, R. Hauschild, J. Friml, Journal of Visualized Experiments JoVE 2017 (2017).","ieee":"D. von Wangenheim, R. Hauschild, and J. Friml, “Light sheet fluorescence microscopy of plant roots growing on the surface of a gel,” <i>Journal of visualized experiments JoVE</i>, vol. 2017, no. 119. Journal of Visualized Experiments, 2017.","ama":"von Wangenheim D, Hauschild R, Friml J. Light sheet fluorescence microscopy of plant roots growing on the surface of a gel. <i>Journal of visualized experiments JoVE</i>. 2017;2017(119). doi:<a href=\"https://doi.org/10.3791/55044\">10.3791/55044</a>"},"intvolume":"      2017","ddc":["580"],"scopus_import":"1","file_date_updated":"2018-12-12T10:16:32Z"},{"user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"Springer","title":"Nonexistence in Thomas Fermi-Dirac-von Weizsäcker theory with small nuclear charges","author":[{"first_name":"Phan","last_name":"Nam","id":"404092F4-F248-11E8-B48F-1D18A9856A87","full_name":"Nam, Phan"},{"last_name":"Van Den Bosch","first_name":"Hanne","full_name":"Van Den Bosch, Hanne"}],"isi":1,"project":[{"name":"Structure of the Excitation Spectrum for Many-Body Quantum Systems","call_identifier":"FWF","grant_number":"P27533_N27","_id":"25C878CE-B435-11E9-9278-68D0E5697425"}],"day":"01","volume":20,"quality_controlled":"1","external_id":{"isi":["000401270000004"]},"article_processing_charge":"No","publist_id":"6300","type":"journal_article","department":[{"_id":"RoSe"}],"language":[{"iso":"eng"}],"publication":"Mathematical Physics, Analysis and Geometry","date_created":"2018-12-11T11:50:02Z","month":"06","status":"public","publication_identifier":{"issn":["13850172"]},"oa":1,"article_number":"6","_id":"1079","abstract":[{"text":"We study the ionization problem in the Thomas-Fermi-Dirac-von Weizsäcker theory for atoms and molecules. We prove the nonexistence of minimizers for the energy functional when the number of electrons is large and the total nuclear charge is small. This nonexistence result also applies to external potentials decaying faster than the Coulomb potential. In the case of arbitrary nuclear charges, we obtain the nonexistence of stable minimizers and radial minimizers.","lang":"eng"}],"issue":"2","date_updated":"2023-09-20T11:53:35Z","year":"2017","citation":{"short":"P. Nam, H. Van Den Bosch, Mathematical Physics, Analysis and Geometry 20 (2017).","ama":"Nam P, Van Den Bosch H. Nonexistence in Thomas Fermi-Dirac-von Weizsäcker theory with small nuclear charges. <i>Mathematical Physics, Analysis and Geometry</i>. 2017;20(2). doi:<a href=\"https://doi.org/10.1007/s11040-017-9238-0\">10.1007/s11040-017-9238-0</a>","ieee":"P. Nam and H. Van Den Bosch, “Nonexistence in Thomas Fermi-Dirac-von Weizsäcker theory with small nuclear charges,” <i>Mathematical Physics, Analysis and Geometry</i>, vol. 20, no. 2. Springer, 2017.","ista":"Nam P, Van Den Bosch H. 2017. Nonexistence in Thomas Fermi-Dirac-von Weizsäcker theory with small nuclear charges. Mathematical Physics, Analysis and Geometry. 20(2), 6.","apa":"Nam, P., &#38; Van Den Bosch, H. (2017). Nonexistence in Thomas Fermi-Dirac-von Weizsäcker theory with small nuclear charges. <i>Mathematical Physics, Analysis and Geometry</i>. Springer. <a href=\"https://doi.org/10.1007/s11040-017-9238-0\">https://doi.org/10.1007/s11040-017-9238-0</a>","mla":"Nam, Phan, and Hanne Van Den Bosch. “Nonexistence in Thomas Fermi-Dirac-von Weizsäcker Theory with Small Nuclear Charges.” <i>Mathematical Physics, Analysis and Geometry</i>, vol. 20, no. 2, 6, Springer, 2017, doi:<a href=\"https://doi.org/10.1007/s11040-017-9238-0\">10.1007/s11040-017-9238-0</a>.","chicago":"Nam, Phan, and Hanne Van Den Bosch. “Nonexistence in Thomas Fermi-Dirac-von Weizsäcker Theory with Small Nuclear Charges.” <i>Mathematical Physics, Analysis and Geometry</i>. Springer, 2017. <a href=\"https://doi.org/10.1007/s11040-017-9238-0\">https://doi.org/10.1007/s11040-017-9238-0</a>."},"intvolume":"        20","main_file_link":[{"url":"https://arxiv.org/abs/1603.07368","open_access":"1"}],"scopus_import":"1","oa_version":"Submitted Version","date_published":"2017-06-01T00:00:00Z","doi":"10.1007/s11040-017-9238-0","publication_status":"published"},{"_id":"1080","article_number":"14114","year":"2017","date_updated":"2023-09-20T11:55:31Z","abstract":[{"text":"Reconstructing the evolutionary history of metastases is critical for understanding their basic biological principles and has profound clinical implications. Genome-wide sequencing data has enabled modern phylogenomic methods to accurately dissect subclones and their phylogenies from noisy and impure bulk tumour samples at unprecedented depth. However, existing methods are not designed to infer metastatic seeding patterns. Here we develop a tool, called Treeomics, to reconstruct the phylogeny of metastases and map subclones to their anatomic locations. Treeomics infers comprehensive seeding patterns for pancreatic, ovarian, and prostate cancers. Moreover, Treeomics correctly disambiguates true seeding patterns from sequencing artifacts; 7% of variants were misclassified by conventional statistical methods. These artifacts can skew phylogenies by creating illusory tumour heterogeneity among distinct samples. In silico benchmarking on simulated tumour phylogenies across a wide range of sample purities (15–95%) and sequencing depths (25-800 × ) demonstrates the accuracy of Treeomics compared with existing methods.","lang":"eng"}],"ec_funded":1,"month":"01","oa":1,"publication_identifier":{"issn":["20411723"]},"status":"public","date_published":"2017-01-31T00:00:00Z","oa_version":"Published Version","publication_status":"published","doi":"10.1038/ncomms14114","intvolume":"         8","citation":{"ista":"Reiter J, Makohon Moore A, Gerold J, Božić I, Chatterjee K, Iacobuzio Donahue C, Vogelstein B, Nowak M. 2017. Reconstructing metastatic seeding patterns of human cancers. Nature Communications. 8, 14114.","mla":"Reiter, Johannes, et al. “Reconstructing Metastatic Seeding Patterns of Human Cancers.” <i>Nature Communications</i>, vol. 8, 14114, Nature Publishing Group, 2017, doi:<a href=\"https://doi.org/10.1038/ncomms14114\">10.1038/ncomms14114</a>.","apa":"Reiter, J., Makohon Moore, A., Gerold, J., Božić, I., Chatterjee, K., Iacobuzio Donahue, C., … Nowak, M. (2017). Reconstructing metastatic seeding patterns of human cancers. <i>Nature Communications</i>. Nature Publishing Group. <a href=\"https://doi.org/10.1038/ncomms14114\">https://doi.org/10.1038/ncomms14114</a>","chicago":"Reiter, Johannes, Alvin Makohon Moore, Jeffrey Gerold, Ivana Božić, Krishnendu Chatterjee, Christine Iacobuzio Donahue, Bert Vogelstein, and Martin Nowak. “Reconstructing Metastatic Seeding Patterns of Human Cancers.” <i>Nature Communications</i>. Nature Publishing Group, 2017. <a href=\"https://doi.org/10.1038/ncomms14114\">https://doi.org/10.1038/ncomms14114</a>.","short":"J. Reiter, A. Makohon Moore, J. Gerold, I. Božić, K. Chatterjee, C. Iacobuzio Donahue, B. Vogelstein, M. Nowak, Nature Communications 8 (2017).","ieee":"J. Reiter <i>et al.</i>, “Reconstructing metastatic seeding patterns of human cancers,” <i>Nature Communications</i>, vol. 8. Nature Publishing Group, 2017.","ama":"Reiter J, Makohon Moore A, Gerold J, et al. Reconstructing metastatic seeding patterns of human cancers. <i>Nature Communications</i>. 2017;8. doi:<a href=\"https://doi.org/10.1038/ncomms14114\">10.1038/ncomms14114</a>"},"ddc":["004","006"],"file_date_updated":"2018-12-12T10:15:15Z","scopus_import":"1","file":[{"access_level":"open_access","creator":"system","file_id":"5133","date_created":"2018-12-12T10:15:15Z","date_updated":"2018-12-12T10:15:15Z","file_name":"IST-2017-786-v1+1_ncomms14114.pdf","file_size":897050,"relation":"main_file","content_type":"application/pdf"}],"project":[{"name":"Quantitative Graph Games: Theory and Applications","call_identifier":"FP7","grant_number":"279307","_id":"2581B60A-B435-11E9-9278-68D0E5697425"},{"_id":"2584A770-B435-11E9-9278-68D0E5697425","grant_number":"P 23499-N23","name":"Modern Graph Algorithmic Techniques in Formal Verification","call_identifier":"FWF"},{"grant_number":"S11407","_id":"25863FF4-B435-11E9-9278-68D0E5697425","call_identifier":"FWF","name":"Game Theory"}],"isi":1,"author":[{"full_name":"Reiter, Johannes","first_name":"Johannes","orcid":"0000-0002-0170-7353","id":"4A918E98-F248-11E8-B48F-1D18A9856A87","last_name":"Reiter"},{"first_name":"Alvin","last_name":"Makohon Moore","full_name":"Makohon Moore, Alvin"},{"full_name":"Gerold, Jeffrey","last_name":"Gerold","first_name":"Jeffrey"},{"full_name":"Božić, Ivana","first_name":"Ivana","last_name":"Božić"},{"full_name":"Chatterjee, Krishnendu","id":"2E5DCA20-F248-11E8-B48F-1D18A9856A87","last_name":"Chatterjee","first_name":"Krishnendu","orcid":"0000-0002-4561-241X"},{"last_name":"Iacobuzio Donahue","first_name":"Christine","full_name":"Iacobuzio Donahue, Christine"},{"last_name":"Vogelstein","first_name":"Bert","full_name":"Vogelstein, Bert"},{"last_name":"Nowak","first_name":"Martin","full_name":"Nowak, Martin"}],"day":"31","tmp":{"short":"CC BY (4.0)","image":"/images/cc_by.png","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode"},"title":"Reconstructing metastatic seeding patterns of human cancers","publisher":"Nature Publishing Group","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","language":[{"iso":"eng"}],"department":[{"_id":"KrCh"}],"type":"journal_article","date_created":"2018-12-11T11:50:02Z","publication":"Nature Communications","has_accepted_license":"1","external_id":{"isi":["000393096600001"]},"volume":8,"quality_controlled":"1","publist_id":"6301","article_processing_charge":"No","pubrep_id":"786"},{"date_published":"2017-04-01T00:00:00Z","oa_version":"None","publication_status":"published","doi":"10.1111/mmi.13597","citation":{"ieee":"C. Fang <i>et al.</i>, “Insulation and wiring specificity of BceR like response regulators and their target promoters in Bacillus subtilis,” <i>Molecular Microbiology</i>, vol. 104, no. 1. Wiley-Blackwell, pp. 16–31, 2017.","ama":"Fang C, Nagy-Staron AA, Grafe M, et al. Insulation and wiring specificity of BceR like response regulators and their target promoters in Bacillus subtilis. <i>Molecular Microbiology</i>. 2017;104(1):16-31. doi:<a href=\"https://doi.org/10.1111/mmi.13597\">10.1111/mmi.13597</a>","short":"C. Fang, A.A. Nagy-Staron, M. Grafe, R. Heermann, K. Jung, S. Gebhard, T. Mascher, Molecular Microbiology 104 (2017) 16–31.","chicago":"Fang, Chong, Anna A Nagy-Staron, Martin Grafe, Ralf Heermann, Kirsten Jung, Susanne Gebhard, and Thorsten Mascher. “Insulation and Wiring Specificity of BceR like Response Regulators and Their Target Promoters in Bacillus Subtilis.” <i>Molecular Microbiology</i>. Wiley-Blackwell, 2017. <a href=\"https://doi.org/10.1111/mmi.13597\">https://doi.org/10.1111/mmi.13597</a>.","apa":"Fang, C., Nagy-Staron, A. A., Grafe, M., Heermann, R., Jung, K., Gebhard, S., &#38; Mascher, T. (2017). Insulation and wiring specificity of BceR like response regulators and their target promoters in Bacillus subtilis. <i>Molecular Microbiology</i>. Wiley-Blackwell. <a href=\"https://doi.org/10.1111/mmi.13597\">https://doi.org/10.1111/mmi.13597</a>","ista":"Fang C, Nagy-Staron AA, Grafe M, Heermann R, Jung K, Gebhard S, Mascher T. 2017. Insulation and wiring specificity of BceR like response regulators and their target promoters in Bacillus subtilis. Molecular Microbiology. 104(1), 16–31.","mla":"Fang, Chong, et al. “Insulation and Wiring Specificity of BceR like Response Regulators and Their Target Promoters in Bacillus Subtilis.” <i>Molecular Microbiology</i>, vol. 104, no. 1, Wiley-Blackwell, 2017, pp. 16–31, doi:<a href=\"https://doi.org/10.1111/mmi.13597\">10.1111/mmi.13597</a>."},"intvolume":"       104","scopus_import":"1","_id":"1084","year":"2017","date_updated":"2023-09-20T11:48:43Z","abstract":[{"text":"BceRS and PsdRS are paralogous two-component systems in Bacillus subtilis controlling the response to antimicrobial peptides. In the presence of extracellular bacitracin and nisin, respectively, the two response regulators (RRs) bind their target promoters, PbceA or PpsdA, resulting in a strong up-regulation of target gene expression and ultimately antibiotic resistance. Despite high sequence similarity between the RRs BceR and PsdR and their known binding sites, no cross-regulation has been observed between them. We therefore investigated the specificity determinants of PbceA and PpsdA that ensure the insulation of these two paralogous pathways at the RR–promoter interface. In vivo and in vitro analyses demonstrate that the regulatory regions within these two promoters contain three important elements: in addition to the known (main) binding site, we identified a linker region and a secondary binding site that are crucial for functionality. Initial binding to the high-affinity, low-specificity main binding site is a prerequisite for the subsequent highly specific binding of a second RR dimer to the low-affinity secondary binding site. In addition to this hierarchical cooperative binding, discrimination requires a competition of the two RRs for their respective binding site mediated by only slight differences in binding affinities.","lang":"eng"}],"issue":"1","month":"04","publication_identifier":{"issn":[" 0950382X"]},"status":"public","language":[{"iso":"eng"}],"department":[{"_id":"CaGu"}],"type":"journal_article","date_created":"2018-12-11T11:50:03Z","page":"16 - 31","publication":"Molecular Microbiology","external_id":{"isi":["000398059200002"]},"volume":104,"quality_controlled":"1","publist_id":"6294","article_processing_charge":"No","author":[{"first_name":"Chong","last_name":"Fang","full_name":"Fang, Chong"},{"full_name":"Nagy-Staron, Anna A","orcid":"0000-0002-1391-8377","first_name":"Anna A","last_name":"Nagy-Staron","id":"3ABC5BA6-F248-11E8-B48F-1D18A9856A87"},{"first_name":"Martin","last_name":"Grafe","full_name":"Grafe, Martin"},{"full_name":"Heermann, Ralf","last_name":"Heermann","first_name":"Ralf"},{"full_name":"Jung, Kirsten","first_name":"Kirsten","last_name":"Jung"},{"last_name":"Gebhard","first_name":"Susanne","full_name":"Gebhard, Susanne"},{"full_name":"Mascher, Thorsten","last_name":"Mascher","first_name":"Thorsten"}],"isi":1,"day":"01","title":"Insulation and wiring specificity of BceR like response regulators and their target promoters in Bacillus subtilis","publisher":"Wiley-Blackwell","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1"},{"publication":"Nature Communications","date_created":"2018-12-11T11:50:04Z","has_accepted_license":"1","department":[{"_id":"BeVi"}],"language":[{"iso":"eng"}],"type":"journal_article","article_processing_charge":"No","pubrep_id":"791","publist_id":"6292","quality_controlled":"1","volume":8,"external_id":{"isi":["000392953700001"]},"tmp":{"short":"CC BY (4.0)","image":"/images/cc_by.png","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode"},"day":"31","file":[{"file_size":955256,"content_type":"application/pdf","relation":"main_file","date_created":"2018-12-12T10:15:22Z","file_name":"IST-2017-791-v1+1_ncomms14251.pdf","date_updated":"2018-12-12T10:15:22Z","access_level":"open_access","file_id":"5141","creator":"system"}],"isi":1,"author":[{"full_name":"Wright, Alison","last_name":"Wright","first_name":"Alison"},{"full_name":"Darolti, Iulia","first_name":"Iulia","last_name":"Darolti"},{"full_name":"Bloch, Natasha","last_name":"Bloch","first_name":"Natasha"},{"full_name":"Oostra, Vicencio","first_name":"Vicencio","last_name":"Oostra"},{"first_name":"Benjamin","last_name":"Sandkam","full_name":"Sandkam, Benjamin"},{"full_name":"Buechel, Séverine","last_name":"Buechel","first_name":"Séverine"},{"full_name":"Kolm, Niclas","first_name":"Niclas","last_name":"Kolm"},{"last_name":"Breden","first_name":"Felix","full_name":"Breden, Felix"},{"full_name":"Vicoso, Beatriz","id":"49E1C5C6-F248-11E8-B48F-1D18A9856A87","last_name":"Vicoso","first_name":"Beatriz","orcid":"0000-0002-4579-8306"},{"first_name":"Judith","last_name":"Mank","full_name":"Mank, Judith"}],"title":"Convergent recombination suppression suggests role of sexual selection in guppy sex chromosome formation","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"Nature Publishing Group","doi":"10.1038/ncomms14251","publication_status":"published","date_published":"2017-01-31T00:00:00Z","oa_version":"Published Version","ddc":["570","576"],"file_date_updated":"2018-12-12T10:15:22Z","scopus_import":"1","intvolume":"         8","citation":{"chicago":"Wright, Alison, Iulia Darolti, Natasha Bloch, Vicencio Oostra, Benjamin Sandkam, Séverine Buechel, Niclas Kolm, Felix Breden, Beatriz Vicoso, and Judith Mank. “Convergent Recombination Suppression Suggests Role of Sexual Selection in Guppy Sex Chromosome Formation.” <i>Nature Communications</i>. Nature Publishing Group, 2017. <a href=\"https://doi.org/10.1038/ncomms14251\">https://doi.org/10.1038/ncomms14251</a>.","apa":"Wright, A., Darolti, I., Bloch, N., Oostra, V., Sandkam, B., Buechel, S., … Mank, J. (2017). Convergent recombination suppression suggests role of sexual selection in guppy sex chromosome formation. <i>Nature Communications</i>. Nature Publishing Group. <a href=\"https://doi.org/10.1038/ncomms14251\">https://doi.org/10.1038/ncomms14251</a>","ista":"Wright A, Darolti I, Bloch N, Oostra V, Sandkam B, Buechel S, Kolm N, Breden F, Vicoso B, Mank J. 2017. Convergent recombination suppression suggests role of sexual selection in guppy sex chromosome formation. Nature Communications. 8, 14251.","mla":"Wright, Alison, et al. “Convergent Recombination Suppression Suggests Role of Sexual Selection in Guppy Sex Chromosome Formation.” <i>Nature Communications</i>, vol. 8, 14251, Nature Publishing Group, 2017, doi:<a href=\"https://doi.org/10.1038/ncomms14251\">10.1038/ncomms14251</a>.","ieee":"A. Wright <i>et al.</i>, “Convergent recombination suppression suggests role of sexual selection in guppy sex chromosome formation,” <i>Nature Communications</i>, vol. 8. Nature Publishing Group, 2017.","ama":"Wright A, Darolti I, Bloch N, et al. Convergent recombination suppression suggests role of sexual selection in guppy sex chromosome formation. <i>Nature Communications</i>. 2017;8. doi:<a href=\"https://doi.org/10.1038/ncomms14251\">10.1038/ncomms14251</a>","short":"A. Wright, I. Darolti, N. Bloch, V. Oostra, B. Sandkam, S. Buechel, N. Kolm, F. Breden, B. Vicoso, J. Mank, Nature Communications 8 (2017)."},"year":"2017","abstract":[{"text":"Sex chromosomes evolve once recombination is halted between a homologous pair of chromosomes. The dominant model of sex chromosome evolution posits that recombination is suppressed between emerging X and Y chromosomes in order to resolve sexual conflict. Here we test this model using whole genome and transcriptome resequencing data in the guppy, a model for sexual selection with many Y-linked colour traits. We show that although the nascent Y chromosome encompasses nearly half of the linkage group, there has been no perceptible degradation of Y chromosome gene content or activity. Using replicate wild populations with differing levels of sexually antagonistic selection for colour, we also show that sexual selection leads to greater expansion of the non-recombining region and increased Y chromosome divergence. These results provide empirical support for longstanding models of sex chromosome catalysis, and suggest an important role for sexual selection and sexual conflict in genome evolution.","lang":"eng"}],"date_updated":"2023-09-20T11:48:16Z","_id":"1085","article_number":"14251","oa":1,"publication_identifier":{"issn":["20411723"]},"status":"public","month":"01"},{"oa":1,"status":"public","month":"02","year":"2017","date_updated":"2023-09-20T11:47:47Z","abstract":[{"lang":"eng","text":"Characterisation of G protein-coupled receptors (GPCR) relies on the availability of a toolbox of ligands that selectively modulate different functional states of the receptors. To uncover such molecules, we explored a unique strategy for ligand discovery that takes advantage of the evolutionary conservation of the 600-million-year-old oxytocin/vasopressin signalling system. We isolated the insect oxytocin/vasopressin orthologue inotocin from the black garden ant (Lasius niger), identified and cloned its cognate receptor and determined its pharmacological properties on the insect and human oxytocin/vasopressin receptors. Subsequently, we identified a functional dichotomy: inotocin activated the insect inotocin and the human vasopressin V1b receptors, but inhibited the human V1aR. Replacement of Arg8 of inotocin by D-Arg8 led to a potent, stable and competitive V1aR-antagonist ([D-Arg8]-inotocin) with a 3,000-fold binding selectivity for the human V1aR over the other three subtypes, OTR, V1bR and V2R. The Arg8/D-Arg8 ligand-pair was further investigated to gain novel insights into the oxytocin/vasopressin peptide-receptor interaction, which led to the identification of key residues of the receptors that are important for ligand functionality and selectivity. These observations could play an important role for development of oxytocin/vasopressin receptor modulators that would enable clear distinction of the physiological and pathological responses of the individual receptor subtypes."}],"_id":"1086","ddc":["570","590"],"file_date_updated":"2018-12-12T10:14:59Z","scopus_import":"1","citation":{"ieee":"M. Di Giglio <i>et al.</i>, “Development of a human vasopressin V1a-receptor antagonist from an evolutionary-related insect neuropeptide,” <i>Scientific Reports</i>, vol. 7. Nature Publishing Group, p. 41002, 2017.","ama":"Di Giglio M, Muttenthaler M, Harpsøe K, et al. Development of a human vasopressin V1a-receptor antagonist from an evolutionary-related insect neuropeptide. <i>Scientific Reports</i>. 2017;7:41002. doi:<a href=\"https://doi.org/10.1038/srep41002\">10.1038/srep41002</a>","short":"M. Di Giglio, M. Muttenthaler, K. Harpsøe, Z. Liutkeviciute, P. Keov, T. Eder, T. Rattei, S. Arrowsmith, S. Wray, A. Marek, T. Elbert, P. Alewood, D. Gloriam, C. Gruber, Scientific Reports 7 (2017) 41002.","chicago":"Di Giglio, Maria, Markus Muttenthaler, Kasper Harpsøe, Zita Liutkeviciute, Peter Keov, Thomas Eder, Thomas Rattei, et al. “Development of a Human Vasopressin V1a-Receptor Antagonist from an Evolutionary-Related Insect Neuropeptide.” <i>Scientific Reports</i>. Nature Publishing Group, 2017. <a href=\"https://doi.org/10.1038/srep41002\">https://doi.org/10.1038/srep41002</a>.","mla":"Di Giglio, Maria, et al. “Development of a Human Vasopressin V1a-Receptor Antagonist from an Evolutionary-Related Insect Neuropeptide.” <i>Scientific Reports</i>, vol. 7, Nature Publishing Group, 2017, p. 41002, doi:<a href=\"https://doi.org/10.1038/srep41002\">10.1038/srep41002</a>.","ista":"Di Giglio M, Muttenthaler M, Harpsøe K, Liutkeviciute Z, Keov P, Eder T, Rattei T, Arrowsmith S, Wray S, Marek A, Elbert T, Alewood P, Gloriam D, Gruber C. 2017. Development of a human vasopressin V1a-receptor antagonist from an evolutionary-related insect neuropeptide. Scientific Reports. 7, 41002.","apa":"Di Giglio, M., Muttenthaler, M., Harpsøe, K., Liutkeviciute, Z., Keov, P., Eder, T., … Gruber, C. (2017). Development of a human vasopressin V1a-receptor antagonist from an evolutionary-related insect neuropeptide. <i>Scientific Reports</i>. Nature Publishing Group. <a href=\"https://doi.org/10.1038/srep41002\">https://doi.org/10.1038/srep41002</a>"},"intvolume":"         7","publication_status":"published","doi":"10.1038/srep41002","date_published":"2017-02-01T00:00:00Z","oa_version":"Published Version","title":"Development of a human vasopressin V1a-receptor antagonist from an evolutionary-related insect neuropeptide","publisher":"Nature Publishing Group","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","day":"01","tmp":{"short":"CC BY (4.0)","image":"/images/cc_by.png","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode"},"file":[{"file_id":"5115","creator":"system","access_level":"open_access","file_name":"IST-2017-790-v1+1_srep41002_1_.pdf","date_updated":"2018-12-12T10:14:59Z","date_created":"2018-12-12T10:14:59Z","relation":"main_file","content_type":"application/pdf","file_size":1994139}],"author":[{"full_name":"Di Giglio, Maria","last_name":"Di Giglio","first_name":"Maria"},{"full_name":"Muttenthaler, Markus","last_name":"Muttenthaler","first_name":"Markus"},{"full_name":"Harpsøe, Kasper","last_name":"Harpsøe","first_name":"Kasper"},{"full_name":"Liutkeviciute, Zita","first_name":"Zita","last_name":"Liutkeviciute"},{"last_name":"Keov","first_name":"Peter","full_name":"Keov, Peter"},{"first_name":"Thomas","last_name":"Eder","full_name":"Eder, Thomas"},{"full_name":"Rattei, Thomas","first_name":"Thomas","last_name":"Rattei"},{"full_name":"Arrowsmith, Sarah","first_name":"Sarah","last_name":"Arrowsmith"},{"last_name":"Wray","first_name":"Susan","full_name":"Wray, Susan"},{"full_name":"Marek, Ales","first_name":"Ales","last_name":"Marek"},{"first_name":"Tomas","last_name":"Elbert","full_name":"Elbert, Tomas"},{"first_name":"Paul","last_name":"Alewood","full_name":"Alewood, Paul"},{"first_name":"David","last_name":"Gloriam","full_name":"Gloriam, David"},{"last_name":"Gruber","first_name":"Christian","full_name":"Gruber, Christian"}],"isi":1,"publist_id":"6291","article_processing_charge":"No","pubrep_id":"790","external_id":{"isi":["000393163800001"]},"quality_controlled":"1","volume":7,"page":"41002","date_created":"2018-12-11T11:50:04Z","publication":"Scientific Reports","has_accepted_license":"1","language":[{"iso":"eng"}],"type":"journal_article"},{"citation":{"chicago":"Song, Baofang, Dwight Barkley, Björn Hof, and Marc Avila. “Speed and Structure of Turbulent Fronts in Pipe Flow.” <i>Journal of Fluid Mechanics</i>. Cambridge University Press, 2017. <a href=\"https://doi.org/10.1017/jfm.2017.14\">https://doi.org/10.1017/jfm.2017.14</a>.","ista":"Song B, Barkley D, Hof B, Avila M. 2017. Speed and structure of turbulent fronts in pipe flow. Journal of Fluid Mechanics. 813, 1045–1059.","apa":"Song, B., Barkley, D., Hof, B., &#38; Avila, M. (2017). Speed and structure of turbulent fronts in pipe flow. <i>Journal of Fluid Mechanics</i>. Cambridge University Press. <a href=\"https://doi.org/10.1017/jfm.2017.14\">https://doi.org/10.1017/jfm.2017.14</a>","mla":"Song, Baofang, et al. “Speed and Structure of Turbulent Fronts in Pipe Flow.” <i>Journal of Fluid Mechanics</i>, vol. 813, Cambridge University Press, 2017, pp. 1045–59, doi:<a href=\"https://doi.org/10.1017/jfm.2017.14\">10.1017/jfm.2017.14</a>.","short":"B. Song, D. Barkley, B. Hof, M. Avila, Journal of Fluid Mechanics 813 (2017) 1045–1059.","ama":"Song B, Barkley D, Hof B, Avila M. Speed and structure of turbulent fronts in pipe flow. <i>Journal of Fluid Mechanics</i>. 2017;813:1045-1059. doi:<a href=\"https://doi.org/10.1017/jfm.2017.14\">10.1017/jfm.2017.14</a>","ieee":"B. Song, D. Barkley, B. Hof, and M. Avila, “Speed and structure of turbulent fronts in pipe flow,” <i>Journal of Fluid Mechanics</i>, vol. 813. Cambridge University Press, pp. 1045–1059, 2017."},"intvolume":"       813","scopus_import":"1","main_file_link":[{"url":"https://arxiv.org/abs/1603.04077","open_access":"1"}],"date_published":"2017-02-25T00:00:00Z","oa_version":"Submitted Version","doi":"10.1017/jfm.2017.14","publication_status":"published","ec_funded":1,"month":"02","oa":1,"publication_identifier":{"issn":["00221120"]},"status":"public","_id":"1087","acknowledged_ssus":[{"_id":"ScienComp"}],"year":"2017","abstract":[{"text":"Using extensive direct numerical simulations, the dynamics of laminar-turbulent fronts in pipe flow is investigated for Reynolds numbers between and 5500. We here investigate the physical distinction between the fronts of weak and strong slugs both by analysing the turbulent kinetic energy budget and by comparing the downstream front motion to the advection speed of bulk turbulent structures. Our study shows that weak downstream fronts travel slower than turbulent structures in the bulk and correspond to decaying turbulence at the front. At the downstream front speed becomes faster than the advection speed, marking the onset of strong fronts. In contrast to weak fronts, turbulent eddies are generated at strong fronts by feeding on the downstream laminar flow. Our study also suggests that temporal fluctuations of production and dissipation at the downstream laminar-turbulent front drive the dynamical switches between the two types of front observed up to.","lang":"eng"}],"date_updated":"2023-09-20T11:47:22Z","quality_controlled":"1","volume":813,"external_id":{"isi":["000394376400044"]},"article_processing_charge":"No","publist_id":"6290","department":[{"_id":"BjHo"}],"language":[{"iso":"eng"}],"type":"journal_article","publication":"Journal of Fluid Mechanics","page":"1045 - 1059","date_created":"2018-12-11T11:50:04Z","title":"Speed and structure of turbulent fronts in pipe flow","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"Cambridge University Press","author":[{"last_name":"Song","first_name":"Baofang","full_name":"Song, Baofang"},{"full_name":"Barkley, Dwight","last_name":"Barkley","first_name":"Dwight"},{"first_name":"Björn","orcid":"0000-0003-2057-2754","id":"3A374330-F248-11E8-B48F-1D18A9856A87","last_name":"Hof","full_name":"Hof, Björn"},{"first_name":"Marc","last_name":"Avila","full_name":"Avila, Marc"}],"isi":1,"project":[{"grant_number":"306589","_id":"25152F3A-B435-11E9-9278-68D0E5697425","call_identifier":"FP7","name":"Decoding the complexity of turbulence at its origin"}],"day":"25"},{"issue":"3","abstract":[{"text":"We discuss properties of distributions that are multivariate totally positive of order two (MTP2) related to conditional independence. In particular, we show that any independence model generated by an MTP2 distribution is a compositional semigraphoid which is upward-stable and singleton-transitive. In addition, we prove that any MTP2 distribution satisfying an appropriate support condition is faithful to its concentration graph. Finally, we analyze factorization properties of MTP2 distributions and discuss ways of constructing MTP2 distributions; in particular we give conditions on the log-linear parameters of a discrete distribution which ensure MTP2 and characterize conditional Gaussian distributions which satisfy MTP2.","lang":"eng"}],"date_updated":"2023-09-20T11:46:53Z","year":"2017","_id":"1089","status":"public","publication_identifier":{"issn":["00905364"]},"oa":1,"month":"06","doi":"10.1214/16-AOS1478","publication_status":"published","oa_version":"Submitted Version","date_published":"2017-06-01T00:00:00Z","main_file_link":[{"url":"https://arxiv.org/abs/1510.01290","open_access":"1"}],"scopus_import":"1","intvolume":"        45","citation":{"chicago":"Fallat, Shaun, Steffen Lauritzen, Kayvan Sadeghi, Caroline Uhler, Nanny Wermuth, and Piotr Zwiernik. “Total Positivity in Markov Structures.” <i>Annals of Statistics</i>. Institute of Mathematical Statistics, 2017. <a href=\"https://doi.org/10.1214/16-AOS1478\">https://doi.org/10.1214/16-AOS1478</a>.","ista":"Fallat S, Lauritzen S, Sadeghi K, Uhler C, Wermuth N, Zwiernik P. 2017. Total positivity in Markov structures. Annals of Statistics. 45(3), 1152–1184.","apa":"Fallat, S., Lauritzen, S., Sadeghi, K., Uhler, C., Wermuth, N., &#38; Zwiernik, P. (2017). Total positivity in Markov structures. <i>Annals of Statistics</i>. Institute of Mathematical Statistics. <a href=\"https://doi.org/10.1214/16-AOS1478\">https://doi.org/10.1214/16-AOS1478</a>","mla":"Fallat, Shaun, et al. “Total Positivity in Markov Structures.” <i>Annals of Statistics</i>, vol. 45, no. 3, Institute of Mathematical Statistics, 2017, pp. 1152–84, doi:<a href=\"https://doi.org/10.1214/16-AOS1478\">10.1214/16-AOS1478</a>.","ama":"Fallat S, Lauritzen S, Sadeghi K, Uhler C, Wermuth N, Zwiernik P. Total positivity in Markov structures. <i>Annals of Statistics</i>. 2017;45(3):1152-1184. doi:<a href=\"https://doi.org/10.1214/16-AOS1478\">10.1214/16-AOS1478</a>","ieee":"S. Fallat, S. Lauritzen, K. Sadeghi, C. Uhler, N. Wermuth, and P. Zwiernik, “Total positivity in Markov structures,” <i>Annals of Statistics</i>, vol. 45, no. 3. Institute of Mathematical Statistics, pp. 1152–1184, 2017.","short":"S. Fallat, S. Lauritzen, K. Sadeghi, C. Uhler, N. Wermuth, P. Zwiernik, Annals of Statistics 45 (2017) 1152–1184."},"day":"01","isi":1,"author":[{"full_name":"Fallat, Shaun","last_name":"Fallat","first_name":"Shaun"},{"last_name":"Lauritzen","first_name":"Steffen","full_name":"Lauritzen, Steffen"},{"full_name":"Sadeghi, Kayvan","first_name":"Kayvan","last_name":"Sadeghi"},{"id":"49ADD78E-F248-11E8-B48F-1D18A9856A87","last_name":"Uhler","orcid":"0000-0002-7008-0216","first_name":"Caroline","full_name":"Uhler, Caroline"},{"full_name":"Wermuth, Nanny","last_name":"Wermuth","first_name":"Nanny"},{"first_name":"Piotr","last_name":"Zwiernik","full_name":"Zwiernik, Piotr"}],"project":[{"grant_number":"Y 903-N35","_id":"2530CA10-B435-11E9-9278-68D0E5697425","name":"Gaussian Graphical Models: Theory and Applications","call_identifier":"FWF"}],"user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"Institute of Mathematical Statistics","title":"Total positivity in Markov structures","publication":"Annals of Statistics","date_created":"2018-12-11T11:50:05Z","page":"1152 - 1184","type":"journal_article","department":[{"_id":"CaUh"}],"language":[{"iso":"eng"}],"article_processing_charge":"No","publist_id":"6288","quality_controlled":"1","volume":45,"external_id":{"isi":["000404395900008"]}},{"ddc":["571"],"scopus_import":"1","file_date_updated":"2018-12-12T10:16:06Z","citation":{"short":"S. Deny, U. Ferrari, E. Mace, P. Yger, R. Caplette, S. Picaud, G. Tkačik, O. Marre, Nature Communications 8 (2017).","ieee":"S. Deny <i>et al.</i>, “Multiplexed computations in retinal ganglion cells of a single type,” <i>Nature Communications</i>, vol. 8, no. 1. Nature Publishing Group, 2017.","ama":"Deny S, Ferrari U, Mace E, et al. Multiplexed computations in retinal ganglion cells of a single type. <i>Nature Communications</i>. 2017;8(1). doi:<a href=\"https://doi.org/10.1038/s41467-017-02159-y\">10.1038/s41467-017-02159-y</a>","ista":"Deny S, Ferrari U, Mace E, Yger P, Caplette R, Picaud S, Tkačik G, Marre O. 2017. Multiplexed computations in retinal ganglion cells of a single type. Nature Communications. 8(1), 1964.","apa":"Deny, S., Ferrari, U., Mace, E., Yger, P., Caplette, R., Picaud, S., … Marre, O. (2017). Multiplexed computations in retinal ganglion cells of a single type. <i>Nature Communications</i>. Nature Publishing Group. <a href=\"https://doi.org/10.1038/s41467-017-02159-y\">https://doi.org/10.1038/s41467-017-02159-y</a>","mla":"Deny, Stephane, et al. “Multiplexed Computations in Retinal Ganglion Cells of a Single Type.” <i>Nature Communications</i>, vol. 8, no. 1, 1964, Nature Publishing Group, 2017, doi:<a href=\"https://doi.org/10.1038/s41467-017-02159-y\">10.1038/s41467-017-02159-y</a>.","chicago":"Deny, Stephane, Ulisse Ferrari, Emilie Mace, Pierre Yger, Romain Caplette, Serge Picaud, Gašper Tkačik, and Olivier Marre. “Multiplexed Computations in Retinal Ganglion Cells of a Single Type.” <i>Nature Communications</i>. Nature Publishing Group, 2017. <a href=\"https://doi.org/10.1038/s41467-017-02159-y\">https://doi.org/10.1038/s41467-017-02159-y</a>."},"intvolume":"         8","publication_status":"published","doi":"10.1038/s41467-017-02159-y","date_published":"2017-12-06T00:00:00Z","oa_version":"Published Version","oa":1,"publication_identifier":{"issn":["20411723"]},"status":"public","ec_funded":1,"month":"12","year":"2017","date_updated":"2023-09-20T11:41:19Z","issue":"1","abstract":[{"lang":"eng","text":"In the early visual system, cells of the same type perform the same computation in different places of the visual field. How these cells code together a complex visual scene is unclear. A common assumption is that cells of a single-type extract a single-stimulus feature to form a feature map, but this has rarely been observed directly. Using large-scale recordings in the rat retina, we show that a homogeneous population of fast OFF ganglion cells simultaneously encodes two radically different features of a visual scene. Cells close to a moving object code quasilinearly for its position, while distant cells remain largely invariant to the object's position and, instead, respond nonlinearly to changes in the object's speed. We develop a quantitative model that accounts for this effect and identify a disinhibitory circuit that mediates it. Ganglion cells of a single type thus do not code for one, but two features simultaneously. This richer, flexible neural map might also be present in other sensory systems."}],"_id":"1104","article_number":"1964","publist_id":"6266","pubrep_id":"921","article_processing_charge":"No","external_id":{"isi":["000417241200004"]},"volume":8,"quality_controlled":"1","date_created":"2018-12-11T11:50:10Z","publication":"Nature Communications","has_accepted_license":"1","language":[{"iso":"eng"}],"department":[{"_id":"GaTk"}],"type":"journal_article","title":"Multiplexed computations in retinal ganglion cells of a single type","publisher":"Nature Publishing Group","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","tmp":{"short":"CC BY (4.0)","image":"/images/cc_by.png","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)","legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode"},"day":"06","file":[{"file_size":2872887,"content_type":"application/pdf","relation":"main_file","date_created":"2018-12-12T10:16:06Z","date_updated":"2018-12-12T10:16:06Z","file_name":"IST-2018-921-v1+1_s41467-017-02159-y.pdf","access_level":"open_access","file_id":"5191","creator":"system"}],"project":[{"name":"Localization of ion channels and receptors by two and three-dimensional immunoelectron microscopic approaches","call_identifier":"FP7","grant_number":"604102","_id":"25CD3DD2-B435-11E9-9278-68D0E5697425"},{"_id":"254D1A94-B435-11E9-9278-68D0E5697425","grant_number":"P 25651-N26","call_identifier":"FWF","name":"Sensitivity to higher-order statistics in natural scenes"}],"author":[{"last_name":"Deny","first_name":"Stephane","full_name":"Deny, Stephane"},{"full_name":"Ferrari, Ulisse","first_name":"Ulisse","last_name":"Ferrari"},{"last_name":"Mace","first_name":"Emilie","full_name":"Mace, Emilie"},{"full_name":"Yger, Pierre","first_name":"Pierre","last_name":"Yger"},{"last_name":"Caplette","first_name":"Romain","full_name":"Caplette, Romain"},{"full_name":"Picaud, Serge","last_name":"Picaud","first_name":"Serge"},{"first_name":"Gasper","orcid":"0000-0002-6699-1455","last_name":"Tkacik","id":"3D494DCA-F248-11E8-B48F-1D18A9856A87","full_name":"Tkacik, Gasper"},{"full_name":"Marre, Olivier","first_name":"Olivier","last_name":"Marre"}],"isi":1},{"month":"08","oa":1,"publication_identifier":{"issn":["2041-1723"]},"status":"public","_id":"11065","article_number":"328","year":"2017","abstract":[{"lang":"eng","text":"Premature aging disorders provide an opportunity to study the mechanisms that drive aging. In Hutchinson-Gilford progeria syndrome (HGPS), a mutant form of the nuclear scaffold protein lamin A distorts nuclei and sequesters nuclear proteins. We sought to investigate protein homeostasis in this disease. Here, we report a widespread increase in protein turnover in HGPS-derived cells compared to normal cells. We determine that global protein synthesis is elevated as a consequence of activated nucleoli and enhanced ribosome biogenesis in HGPS-derived fibroblasts. Depleting normal lamin A or inducing mutant lamin A expression are each sufficient to drive nucleolar expansion. We further show that nucleolar size correlates with donor age in primary fibroblasts derived from healthy individuals and that ribosomal RNA production increases with age, indicating that nucleolar size and activity can serve as aging biomarkers. While limiting ribosome biogenesis extends lifespan in several systems, we show that increased ribosome biogenesis and activity are a hallmark of premature aging."}],"date_updated":"2022-07-18T08:33:03Z","citation":{"chicago":"Buchwalter, Abigail, and Martin Hetzer. “Nucleolar Expansion and Elevated Protein Translation in Premature Aging.” <i>Nature Communications</i>. Springer Nature, 2017. <a href=\"https://doi.org/10.1038/s41467-017-00322-z\">https://doi.org/10.1038/s41467-017-00322-z</a>.","mla":"Buchwalter, Abigail, and Martin Hetzer. “Nucleolar Expansion and Elevated Protein Translation in Premature Aging.” <i>Nature Communications</i>, vol. 8, 328, Springer Nature, 2017, doi:<a href=\"https://doi.org/10.1038/s41467-017-00322-z\">10.1038/s41467-017-00322-z</a>.","apa":"Buchwalter, A., &#38; Hetzer, M. (2017). Nucleolar expansion and elevated protein translation in premature aging. <i>Nature Communications</i>. Springer Nature. <a href=\"https://doi.org/10.1038/s41467-017-00322-z\">https://doi.org/10.1038/s41467-017-00322-z</a>","ista":"Buchwalter A, Hetzer M. 2017. Nucleolar expansion and elevated protein translation in premature aging. Nature Communications. 8, 328.","ama":"Buchwalter A, Hetzer M. Nucleolar expansion and elevated protein translation in premature aging. <i>Nature Communications</i>. 2017;8. doi:<a href=\"https://doi.org/10.1038/s41467-017-00322-z\">10.1038/s41467-017-00322-z</a>","ieee":"A. Buchwalter and M. Hetzer, “Nucleolar expansion and elevated protein translation in premature aging,” <i>Nature Communications</i>, vol. 8. Springer Nature, 2017.","short":"A. Buchwalter, M. Hetzer, Nature Communications 8 (2017)."},"intvolume":"         8","main_file_link":[{"open_access":"1","url":"https://doi.org/10.1038/s41467-017-00322-z"}],"scopus_import":"1","date_published":"2017-08-30T00:00:00Z","oa_version":"Published Version","doi":"10.1038/s41467-017-00322-z","publication_status":"published","title":"Nucleolar expansion and elevated protein translation in premature aging","user_id":"72615eeb-f1f3-11ec-aa25-d4573ddc34fd","publisher":"Springer Nature","author":[{"last_name":"Buchwalter","first_name":"Abigail","full_name":"Buchwalter, Abigail"},{"id":"86c0d31b-b4eb-11ec-ac5a-eae7b2e135ed","last_name":"HETZER","orcid":"0000-0002-2111-992X","first_name":"Martin W","full_name":"HETZER, Martin W"}],"extern":"1","day":"30","pmid":1,"volume":8,"quality_controlled":"1","keyword":["General Physics and Astronomy","General Biochemistry","Genetics and Molecular Biology","General Chemistry"],"external_id":{"pmid":["28855503"]},"article_type":"original","article_processing_charge":"No","language":[{"iso":"eng"}],"type":"journal_article","publication":"Nature Communications","date_created":"2022-04-07T07:45:50Z"},{"article_type":"original","article_processing_charge":"No","keyword":["Developmental Biology","Genetics"],"quality_controlled":"1","volume":31,"external_id":{"pmid":["29269482"]},"publication":"Genes & Development","date_created":"2022-04-07T07:45:59Z","page":"2222-2234","language":[{"iso":"eng"}],"type":"journal_article","title":"Nup98 recruits the Wdr82–Set1A/COMPASS complex to promoters to regulate H3K4 trimethylation in hematopoietic progenitor cells","user_id":"72615eeb-f1f3-11ec-aa25-d4573ddc34fd","publisher":"Cold Spring Harbor Laboratory","extern":"1","day":"21","pmid":1,"author":[{"full_name":"Franks, Tobias M.","last_name":"Franks","first_name":"Tobias M."},{"first_name":"Asako","last_name":"McCloskey","full_name":"McCloskey, Asako"},{"full_name":"Shokhirev, Maxim Nikolaievich","last_name":"Shokhirev","first_name":"Maxim Nikolaievich"},{"full_name":"Benner, Chris","last_name":"Benner","first_name":"Chris"},{"first_name":"Annie","last_name":"Rathore","full_name":"Rathore, Annie"},{"full_name":"HETZER, Martin W","last_name":"HETZER","id":"86c0d31b-b4eb-11ec-ac5a-eae7b2e135ed","orcid":"0000-0002-2111-992X","first_name":"Martin W"}],"scopus_import":"1","main_file_link":[{"open_access":"1","url":"https://doi.org/10.1101/gad.306753.117"}],"intvolume":"        31","citation":{"ieee":"T. M. Franks, A. McCloskey, M. N. Shokhirev, C. Benner, A. Rathore, and M. Hetzer, “Nup98 recruits the Wdr82–Set1A/COMPASS complex to promoters to regulate H3K4 trimethylation in hematopoietic progenitor cells,” <i>Genes &#38; Development</i>, vol. 31, no. 22. Cold Spring Harbor Laboratory, pp. 2222–2234, 2017.","ama":"Franks TM, McCloskey A, Shokhirev MN, Benner C, Rathore A, Hetzer M. Nup98 recruits the Wdr82–Set1A/COMPASS complex to promoters to regulate H3K4 trimethylation in hematopoietic progenitor cells. <i>Genes &#38; Development</i>. 2017;31(22):2222-2234. doi:<a href=\"https://doi.org/10.1101/gad.306753.117\">10.1101/gad.306753.117</a>","short":"T.M. Franks, A. McCloskey, M.N. Shokhirev, C. Benner, A. Rathore, M. Hetzer, Genes &#38; Development 31 (2017) 2222–2234.","chicago":"Franks, Tobias M., Asako McCloskey, Maxim Nikolaievich Shokhirev, Chris Benner, Annie Rathore, and Martin Hetzer. “Nup98 Recruits the Wdr82–Set1A/COMPASS Complex to Promoters to Regulate H3K4 Trimethylation in Hematopoietic Progenitor Cells.” <i>Genes &#38; Development</i>. Cold Spring Harbor Laboratory, 2017. <a href=\"https://doi.org/10.1101/gad.306753.117\">https://doi.org/10.1101/gad.306753.117</a>.","mla":"Franks, Tobias M., et al. “Nup98 Recruits the Wdr82–Set1A/COMPASS Complex to Promoters to Regulate H3K4 Trimethylation in Hematopoietic Progenitor Cells.” <i>Genes &#38; Development</i>, vol. 31, no. 22, Cold Spring Harbor Laboratory, 2017, pp. 2222–34, doi:<a href=\"https://doi.org/10.1101/gad.306753.117\">10.1101/gad.306753.117</a>.","ista":"Franks TM, McCloskey A, Shokhirev MN, Benner C, Rathore A, Hetzer M. 2017. Nup98 recruits the Wdr82–Set1A/COMPASS complex to promoters to regulate H3K4 trimethylation in hematopoietic progenitor cells. Genes &#38; Development. 31(22), 2222–2234.","apa":"Franks, T. M., McCloskey, A., Shokhirev, M. N., Benner, C., Rathore, A., &#38; Hetzer, M. (2017). Nup98 recruits the Wdr82–Set1A/COMPASS complex to promoters to regulate H3K4 trimethylation in hematopoietic progenitor cells. <i>Genes &#38; Development</i>. Cold Spring Harbor Laboratory. <a href=\"https://doi.org/10.1101/gad.306753.117\">https://doi.org/10.1101/gad.306753.117</a>"},"doi":"10.1101/gad.306753.117","publication_status":"published","date_published":"2017-12-21T00:00:00Z","oa_version":"Published Version","publication_identifier":{"issn":["0890-9369","1549-5477"]},"oa":1,"status":"public","month":"12","year":"2017","abstract":[{"lang":"eng","text":"Recent studies have shown that a subset of nucleoporins (Nups) can detach from the nuclear pore complex and move into the nuclear interior to regulate transcription. One such dynamic Nup, called Nup98, has been implicated in gene activation in healthy cells and has been shown to drive leukemogenesis when mutated in patients with acute myeloid leukemia (AML). Here we show that in hematopoietic cells, Nup98 binds predominantly to transcription start sites to recruit the Wdr82–Set1A/COMPASS (complex of proteins associated with Set1) complex, which is required for deposition of the histone 3 Lys4 trimethyl (H3K4me3)-activating mark. Depletion of Nup98 or Wdr82 abolishes Set1A recruitment to chromatin and subsequently ablates H3K4me3 at adjacent promoters. Furthermore, expression of a Nup98 fusion protein implicated in aggressive AML causes mislocalization of H3K4me3 at abnormal regions and up-regulation of associated genes. Our findings establish a function of Nup98 in hematopoietic gene activation and provide mechanistic insight into which Nup98 leukemic fusion proteins promote AML."}],"issue":"22","date_updated":"2022-07-18T08:33:05Z","_id":"11066"},{"year":"2017","issue":"5","abstract":[{"text":"Neural progenitor cells (NeuPCs) possess a unique nuclear architecture that changes during differentiation. Nucleoporins are linked with cell-type-specific gene regulation, coupling physical changes in nuclear structure to transcriptional output; but, whether and how they coordinate with key fate-determining transcription factors is unclear. Here we show that the nucleoporin Nup153 interacts with Sox2 in adult NeuPCs, where it is indispensable for their maintenance and controls neuronal differentiation. Genome-wide analyses show that Nup153 and Sox2 bind and co-regulate hundreds of genes. Binding of Nup153 to gene promoters or transcriptional end sites correlates with increased or decreased gene expression, respectively, and inhibiting Nup153 expression alters open chromatin configurations at its target genes, disrupts genomic localization of Sox2, and promotes differentiation in vitro and a gliogenic fate switch in vivo. Together, these findings reveal that nuclear structural proteins may exert bimodal transcriptional effects to control cell fate.","lang":"eng"}],"date_updated":"2022-07-18T08:33:07Z","_id":"11067","publication_identifier":{"issn":["1934-5909"]},"oa":1,"status":"public","month":"11","doi":"10.1016/j.stem.2017.08.012","publication_status":"published","date_published":"2017-11-02T00:00:00Z","oa_version":"Published Version","main_file_link":[{"url":"https://doi.org/10.1016/j.stem.2017.08.012","open_access":"1"}],"scopus_import":"1","citation":{"short":"T. Toda, J.Y. Hsu, S.B. Linker, L. Hu, S.T. Schafer, J. Mertens, F.V. Jacinto, M. Hetzer, F.H. Gage, Cell Stem Cell 21 (2017) 618–634.e7.","ama":"Toda T, Hsu JY, Linker SB, et al. Nup153 interacts with Sox2 to enable bimodal gene regulation and maintenance of neural progenitor cells. <i>Cell Stem Cell</i>. 2017;21(5):618-634.e7. doi:<a href=\"https://doi.org/10.1016/j.stem.2017.08.012\">10.1016/j.stem.2017.08.012</a>","ieee":"T. Toda <i>et al.</i>, “Nup153 interacts with Sox2 to enable bimodal gene regulation and maintenance of neural progenitor cells,” <i>Cell Stem Cell</i>, vol. 21, no. 5. Elsevier, p. 618–634.e7, 2017.","chicago":"Toda, Tomohisa, Jonathan Y. Hsu, Sara B. Linker, Lauren Hu, Simon T. Schafer, Jerome Mertens, Filipe V. Jacinto, Martin Hetzer, and Fred H. Gage. “Nup153 Interacts with Sox2 to Enable Bimodal Gene Regulation and Maintenance of Neural Progenitor Cells.” <i>Cell Stem Cell</i>. Elsevier, 2017. <a href=\"https://doi.org/10.1016/j.stem.2017.08.012\">https://doi.org/10.1016/j.stem.2017.08.012</a>.","apa":"Toda, T., Hsu, J. Y., Linker, S. B., Hu, L., Schafer, S. T., Mertens, J., … Gage, F. H. (2017). Nup153 interacts with Sox2 to enable bimodal gene regulation and maintenance of neural progenitor cells. <i>Cell Stem Cell</i>. Elsevier. <a href=\"https://doi.org/10.1016/j.stem.2017.08.012\">https://doi.org/10.1016/j.stem.2017.08.012</a>","mla":"Toda, Tomohisa, et al. “Nup153 Interacts with Sox2 to Enable Bimodal Gene Regulation and Maintenance of Neural Progenitor Cells.” <i>Cell Stem Cell</i>, vol. 21, no. 5, Elsevier, 2017, p. 618–634.e7, doi:<a href=\"https://doi.org/10.1016/j.stem.2017.08.012\">10.1016/j.stem.2017.08.012</a>.","ista":"Toda T, Hsu JY, Linker SB, Hu L, Schafer ST, Mertens J, Jacinto FV, Hetzer M, Gage FH. 2017. Nup153 interacts with Sox2 to enable bimodal gene regulation and maintenance of neural progenitor cells. Cell Stem Cell. 21(5), 618–634.e7."},"intvolume":"        21","extern":"1","day":"02","pmid":1,"author":[{"full_name":"Toda, Tomohisa","last_name":"Toda","first_name":"Tomohisa"},{"first_name":"Jonathan Y.","last_name":"Hsu","full_name":"Hsu, Jonathan Y."},{"full_name":"Linker, Sara B.","first_name":"Sara B.","last_name":"Linker"},{"full_name":"Hu, Lauren","first_name":"Lauren","last_name":"Hu"},{"first_name":"Simon T.","last_name":"Schafer","full_name":"Schafer, Simon T."},{"first_name":"Jerome","last_name":"Mertens","full_name":"Mertens, Jerome"},{"full_name":"Jacinto, Filipe V.","last_name":"Jacinto","first_name":"Filipe V."},{"last_name":"HETZER","id":"86c0d31b-b4eb-11ec-ac5a-eae7b2e135ed","first_name":"Martin W","orcid":"0000-0002-2111-992X","full_name":"HETZER, Martin W"},{"full_name":"Gage, Fred H.","first_name":"Fred H.","last_name":"Gage"}],"title":"Nup153 interacts with Sox2 to enable bimodal gene regulation and maintenance of neural progenitor cells","user_id":"72615eeb-f1f3-11ec-aa25-d4573ddc34fd","publisher":"Elsevier","publication":"Cell Stem Cell","date_created":"2022-04-07T07:46:12Z","page":"618-634.e7","language":[{"iso":"eng"}],"type":"journal_article","article_processing_charge":"No","article_type":"original","volume":21,"quality_controlled":"1","keyword":["Cell Biology","Genetics","Molecular Medicine"],"external_id":{"pmid":["28919367"]}},{"file":[{"date_created":"2018-12-12T10:12:03Z","date_updated":"2018-12-12T10:12:03Z","file_name":"IST-2017-868-v1+1_1-s2.0-S0166432816309160-main.pdf","access_level":"open_access","file_id":"4921","creator":"system","file_size":2291511,"content_type":"application/pdf","relation":"main_file"}],"isi":1,"author":[{"first_name":"Martin","last_name":"Breuss","full_name":"Breuss, Martin"},{"id":"38853E16-F248-11E8-B48F-1D18A9856A87","last_name":"Hansen","first_name":"Andi H","full_name":"Hansen, Andi H"},{"full_name":"Landler, Lukas","last_name":"Landler","first_name":"Lukas"},{"last_name":"Keays","first_name":"David","full_name":"Keays, David"}],"extern":"1","tmp":{"name":"Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International (CC BY-NC-ND 4.0)","legal_code_url":"https://creativecommons.org/licenses/by-nc-nd/4.0/legalcode","short":"CC BY-NC-ND (4.0)","image":"/images/cc_by_nc_nd.png"},"day":"14","acknowledgement":"Austrian Science Fund (FWF) for funding this research [I914,P21092]","title":"Brain specific knockin of the pathogenic Tubb5 E401K allele causes defects in motor coordination and prepulse inhibition","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","publisher":"Elsevier","language":[{"iso":"eng"}],"type":"journal_article","publication":"Behavioural Brain Research","date_created":"2018-12-11T11:50:11Z","page":"47 - 55","has_accepted_license":"1","quality_controlled":"1","volume":323,"external_id":{"isi":["000397369100007"]},"article_processing_charge":"No","pubrep_id":"868","publist_id":"6262","_id":"1107","year":"2017","abstract":[{"lang":"eng","text":"The generation, migration, and differentiation of neurons requires the functional integrity of the microtubule cytoskeleton. Mutations in the tubulin gene family are known to cause various neurological diseases including lissencephaly, ocular motor disorders, polymicrogyria and amyotrophic lateral sclerosis. We have previously reported that mutations in TUBB5 cause microcephaly that is accompanied by severe intellectual impairment and motor delay. Here we present the characterization of a Tubb5 mouse model that allows for the conditional expression of the pathogenic E401K mutation. Homozygous knockin animals exhibit a severe reduction in brain size and in body weight. These animals do not show any significant impairment in general activity, anxiety, or in the acoustic startle response, however, present with notable defects in motor coordination. When assessed on the static rod apparatus mice took longer to orient and often lost their balance completely. Interestingly, mutant animals also showed defects in prepulse inhibition, a phenotype associated with sensorimotor gating and considered an endophenotype for schizophrenia. This study provides insight into the behavioral consequences of tubulin gene mutations."}],"date_updated":"2023-09-20T11:37:25Z","month":"04","oa":1,"publication_identifier":{"issn":["01664328"]},"status":"public","date_published":"2017-04-14T00:00:00Z","oa_version":"Published Version","doi":"10.1016/j.bbr.2017.01.029","publication_status":"published","intvolume":"       323","citation":{"mla":"Breuss, Martin, et al. “Brain Specific Knockin of the Pathogenic Tubb5 E401K Allele Causes Defects in Motor Coordination and Prepulse Inhibition.” <i>Behavioural Brain Research</i>, vol. 323, Elsevier, 2017, pp. 47–55, doi:<a href=\"https://doi.org/10.1016/j.bbr.2017.01.029\">10.1016/j.bbr.2017.01.029</a>.","ista":"Breuss M, Hansen AH, Landler L, Keays D. 2017. Brain specific knockin of the pathogenic Tubb5 E401K allele causes defects in motor coordination and prepulse inhibition. Behavioural Brain Research. 323, 47–55.","apa":"Breuss, M., Hansen, A. H., Landler, L., &#38; Keays, D. (2017). Brain specific knockin of the pathogenic Tubb5 E401K allele causes defects in motor coordination and prepulse inhibition. <i>Behavioural Brain Research</i>. Elsevier. <a href=\"https://doi.org/10.1016/j.bbr.2017.01.029\">https://doi.org/10.1016/j.bbr.2017.01.029</a>","chicago":"Breuss, Martin, Andi H Hansen, Lukas Landler, and David Keays. “Brain Specific Knockin of the Pathogenic Tubb5 E401K Allele Causes Defects in Motor Coordination and Prepulse Inhibition.” <i>Behavioural Brain Research</i>. Elsevier, 2017. <a href=\"https://doi.org/10.1016/j.bbr.2017.01.029\">https://doi.org/10.1016/j.bbr.2017.01.029</a>.","short":"M. Breuss, A.H. Hansen, L. Landler, D. Keays, Behavioural Brain Research 323 (2017) 47–55.","ama":"Breuss M, Hansen AH, Landler L, Keays D. Brain specific knockin of the pathogenic Tubb5 E401K allele causes defects in motor coordination and prepulse inhibition. <i>Behavioural Brain Research</i>. 2017;323:47-55. doi:<a href=\"https://doi.org/10.1016/j.bbr.2017.01.029\">10.1016/j.bbr.2017.01.029</a>","ieee":"M. Breuss, A. H. Hansen, L. Landler, and D. Keays, “Brain specific knockin of the pathogenic Tubb5 E401K allele causes defects in motor coordination and prepulse inhibition,” <i>Behavioural Brain Research</i>, vol. 323. Elsevier, pp. 47–55, 2017."},"ddc":["570","571"],"file_date_updated":"2018-12-12T10:12:03Z"},{"_id":"1108","year":"2017","abstract":[{"text":"In this work we study the learnability of stochastic processes with respect to the conditional risk, i.e. the existence of a learning algorithm that improves its next-step performance with the amount of observed data. We introduce a notion of pairwise discrepancy between conditional distributions at different times steps and show how certain properties of these discrepancies can be used to construct a successful learning algorithm. Our main results are two theorems that establish criteria for learnability for many classes of stochastic processes, including all special cases studied previously in the literature.","lang":"eng"}],"date_updated":"2023-10-17T10:01:12Z","ec_funded":1,"month":"04","oa":1,"status":"public","date_published":"2017-04-01T00:00:00Z","oa_version":"Submitted Version","publication_status":"published","conference":{"location":"Fort Lauderdale, FL, United States","end_date":"2017-04-22","name":"AISTATS: Artificial Intelligence and Statistics","start_date":"2017-04-20"},"citation":{"apa":"Zimin, A., &#38; Lampert, C. (2017). Learning theory for conditional risk minimization (Vol. 54, pp. 213–222). Presented at the AISTATS: Artificial Intelligence and Statistics, Fort Lauderdale, FL, United States: ML Research Press.","ista":"Zimin A, Lampert C. 2017. Learning theory for conditional risk minimization. AISTATS: Artificial Intelligence and Statistics, PMLR, vol. 54, 213–222.","mla":"Zimin, Alexander, and Christoph Lampert. <i>Learning Theory for Conditional Risk Minimization</i>. Vol. 54, ML Research Press, 2017, pp. 213–22.","chicago":"Zimin, Alexander, and Christoph Lampert. “Learning Theory for Conditional Risk Minimization,” 54:213–22. ML Research Press, 2017.","short":"A. Zimin, C. Lampert, in:, ML Research Press, 2017, pp. 213–222.","ieee":"A. Zimin and C. Lampert, “Learning theory for conditional risk minimization,” presented at the AISTATS: Artificial Intelligence and Statistics, Fort Lauderdale, FL, United States, 2017, vol. 54, pp. 213–222.","ama":"Zimin A, Lampert C. Learning theory for conditional risk minimization. In: Vol 54. ML Research Press; 2017:213-222."},"intvolume":"        54","main_file_link":[{"open_access":"1","url":"http://proceedings.mlr.press/v54/zimin17a/zimin17a.pdf"}],"author":[{"first_name":"Alexander","last_name":"Zimin","id":"37099E9C-F248-11E8-B48F-1D18A9856A87","full_name":"Zimin, Alexander"},{"first_name":"Christoph","orcid":"0000-0001-8622-7887","last_name":"Lampert","id":"40C20FD2-F248-11E8-B48F-1D18A9856A87","full_name":"Lampert, Christoph"}],"isi":1,"project":[{"_id":"2532554C-B435-11E9-9278-68D0E5697425","grant_number":"308036","call_identifier":"FP7","name":"Lifelong Learning of Visual Scene Understanding"}],"day":"01","alternative_title":["PMLR"],"title":"Learning theory for conditional risk minimization","user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","publisher":"ML Research Press","department":[{"_id":"ChLa"}],"language":[{"iso":"eng"}],"type":"conference","page":"213 - 222","date_created":"2018-12-11T11:50:11Z","volume":54,"quality_controlled":"1","external_id":{"isi":["000509368500024"]},"article_processing_charge":"No","publist_id":"6261"}]