@inproceedings{1136,
  abstract     = {We propose an interactive sculpting system for seamlessly editing pre-computed animations of liquid, without the need for any resimulation. The input is a sequence of meshes without correspondences representing the liquid surface over time. Our method enables the efficient selection of consistent space-time parts of this animation, such as moving waves or droplets, which we call space-time features. Once selected, a feature can be copied, edited, or duplicated and then pasted back anywhere in space and time in the same or in another liquid animation sequence. Our method circumvents tedious user interactions by automatically computing the spatial and temporal ranges of the selected feature. We also provide space-time shape editing tools for non-uniform scaling, rotation, trajectory changes, and temporal editing to locally speed up or slow down motion. Using our tools, the user can edit and progressively refine any input simulation result, possibly using a library of precomputed space-time features extracted from other animations. In contrast to the trial-and-error loop usually required to edit animation results through the tuning of indirect simulation parameters, our method gives the user full control over the edited space-time behaviors. © 2016 Copyright held by the owner/author(s).},
  author       = {Manteaux, Pierre and Vimont, Ulysse and Wojtan, Christopher J and Rohmer, Damien and Cani, Marie},
  booktitle    = {Proceedings of the 9th International Conference on Motion in Games },
  location     = {San Francisco, CA, USA},
  publisher    = {ACM},
  title        = {{Space-time sculpting of liquid animation}},
  doi          = {10.1145/2994258.2994261},
  year         = {2016},
}

@article{1137,
  abstract     = {RASGRP1 is an important guanine nucleotide exchange factor and activator of the RAS-MAPK pathway following T cell antigen receptor (TCR) signaling. The consequences of RASGRP1 mutations in humans are unknown. In a patient with recurrent bacterial and viral infections, born to healthy consanguineous parents, we used homozygosity mapping and exome sequencing to identify a biallelic stop-gain variant in RASGRP1. This variant segregated perfectly with the disease and has not been reported in genetic databases. RASGRP1 deficiency was associated in T cells and B cells with decreased phosphorylation of the extracellular-signal-regulated serine kinase ERK, which was restored following expression of wild-type RASGRP1. RASGRP1 deficiency also resulted in defective proliferation, activation and motility of T cells and B cells. RASGRP1-deficient natural killer (NK) cells exhibited impaired cytotoxicity with defective granule convergence and actin accumulation. Interaction proteomics identified the dynein light chain DYNLL1 as interacting with RASGRP1, which links RASGRP1 to cytoskeletal dynamics. RASGRP1-deficient cells showed decreased activation of the GTPase RhoA. Treatment with lenalidomide increased RhoA activity and reversed the migration and activation defects of RASGRP1-deficient lymphocytes.},
  author       = {Salzer, Elisabeth and Çaǧdaş, Deniz and Hons, Miroslav and Mace, Emily and Garncarz, Wojciech and Petronczki, Oezlem and Platzer, René and Pfajfer, Laurène and Bilic, Ivan and Ban, Sol and Willmann, Katharina and Mukherjee, Malini and Supper, Verena and Hsu, Hsiangting and Banerjee, Pinaki and Sinha, Papiya and Mcclanahan, Fabienne and Zlabinger, Gerhard and Pickl, Winfried and Gribben, John and Stockinger, Hannes and Bennett, Keiryn and Huppa, Johannes and Dupré, Loï̈C and Sanal, Özden and Jäger, Ulrich and Sixt, Michael K and Tezcan, Ilhan and Orange, Jordan and Boztug, Kaan},
  journal      = {Nature Immunology},
  number       = {12},
  pages        = {1352 -- 1360},
  publisher    = {Nature Publishing Group},
  title        = {{RASGRP1 deficiency causes immunodeficiency with impaired cytoskeletal dynamics}},
  doi          = {10.1038/ni.3575},
  volume       = {17},
  year         = {2016},
}

@inproceedings{1138,
  abstract     = {Automata with monitor counters, where the transitions do not depend on counter values, and nested weighted automata are two expressive automata-theoretic frameworks for quantitative properties. For a well-studied and wide class of quantitative functions, we establish that automata with monitor counters and nested weighted automata are equivalent. We study for the first time such quantitative automata under probabilistic semantics. We show that several problems that are undecidable for the classical questions of emptiness and universality become decidable under the probabilistic semantics. We present a complete picture of decidability for such automata, and even an almost-complete picture of computational complexity, for the probabilistic questions we consider. © 2016 ACM.},
  author       = {Chatterjee, Krishnendu and Henzinger, Thomas A and Otop, Jan},
  booktitle    = {Proceedings of the 31st Annual ACM/IEEE Symposium},
  location     = {New York, NY, USA},
  pages        = {76 -- 85},
  publisher    = {IEEE},
  title        = {{Quantitative automata under probabilistic semantics}},
  doi          = {10.1145/2933575.2933588},
  year         = {2016},
}

@article{1139,
  abstract     = {Microtubules switch stochastically between phases of growth and shrinkage. The molecular mechanism responsible for the end of a growth phase, an event called catastrophe, is still not understood. The probability for a catastrophe to occur increases with microtubule age, putting constraints on the possible molecular mechanism of catastrophe induction. Here we used microfluidics-Assisted fast tubulin washout experiments to induce microtubule depolymerization in a controlled manner at different times after the start of growth. We found that aging can also be observed in this assay, providing valuable new constraints against which theoretical models of catastrophe induction can be tested. We found that the data can be quantitatively well explained by a simple kinetic threshold model that assumes an age-dependent broadening of the protective cap at the microtubule end as a result of an evolving tapered end structure; this leads to a decrease of the cap density and its stability. This analysis suggests an intuitive picture of the role of morphological changes of the protective cap for the age dependence of microtubule stability.},
  author       = {Düllberg, Christian F and Cade, Nicholas and Surrey, Thomas},
  journal      = {Molecular Biology and Evolution},
  number       = {22},
  pages        = {3563 -- 3573},
  publisher    = {Oxford University Press},
  title        = {{Microtubule aging probed by microfluidics assisted tubulin washout}},
  doi          = {10.1091/mbc.E16-07-0548},
  volume       = {27},
  year         = {2016},
}

@inproceedings{1140,
  abstract     = {Given a model of a system and an objective, the model-checking question asks whether the model satisfies the objective. We study polynomial-time problems in two classical models, graphs and Markov Decision Processes (MDPs), with respect to several fundamental -regular objectives, e.g., Rabin and Streett objectives. For many of these problems the best-known upper bounds are quadratic or cubic, yet no super-linear lower bounds are known. In this work our contributions are two-fold: First, we present several improved algorithms, and second, we present the first conditional super-linear lower bounds based on widely believed assumptions about the complexity of CNF-SAT and combinatorial Boolean matrix multiplication. A separation result for two models with respect to an objective means a conditional lower bound for one model that is strictly higher than the existing upper bound for the other model, and similarly for two objectives with respect to a model. Our results establish the following separation results: (1) A separation of models (graphs and MDPs) for disjunctive queries of reachability and Büchi objectives. (2) Two kinds of separations of objectives, both for graphs and MDPs, namely, (2a) the separation of dual objectives such as Streett/Rabin objectives, and (2b) the separation of conjunction and disjunction of multiple objectives of the same type such as safety, Büchi, and coBüchi. In summary, our results establish the first model and objective separation results for graphs and MDPs for various classical -regular objectives. Quite strikingly, we establish conditional lower bounds for the disjunction of objectives that are strictly higher than the existing upper bounds for the conjunction of the same objectives. © 2016 ACM.},
  author       = {Chatterjee, Krishnendu and Dvoák, Wolfgang and Henzinger, Monika H and Loitzenbauer, Veronika},
  booktitle    = {Proceedings of the 31st Annual ACM/IEEE Symposium on Logic in Computer Science},
  location     = {New York, NY, USA},
  pages        = {197 -- 206},
  publisher    = {IEEE},
  title        = {{Model and objective separation with conditional lower bounds: disjunction is harder than conjunction}},
  doi          = {10.1145/2933575.2935304},
  year         = {2016},
}

@article{1141,
  abstract     = {In this paper we introduce the Multiobjective Optimization Hierarchic Genetic Strategy with maturing (MO-mHGS), a meta-algorithm that performs evolutionary optimization in a hierarchy of populations. The maturing mechanism improves growth and reduces redundancy. The performance of MO-mHGS with selected state-of-the-art multiobjective evolutionary algorithms as internal algorithms is analysed on benchmark problems and their modifications for which single fitness evaluation time depends on the solution accuracy. We compare the proposed algorithm with the Island Model Genetic Algorithm as well as with single-deme methods, and discuss the impact of internal algorithms on the MO-mHGS meta-algorithm. © 2016 Elsevier B.V.},
  author       = {Łazarz, Radosław and Idzik, Michał and Gądek, Konrad and Gajda-Zagorska, Ewa P},
  journal      = {Journal of Computational Science},
  number       = {1},
  pages        = {249 -- 260},
  publisher    = {Elsevier},
  title        = {{Hierarchic genetic strategy with maturing as a generic tool for multiobjective optimization}},
  doi          = {10.1016/j.jocs.2016.03.004},
  volume       = {17},
  year         = {2016},
}

@article{1142,
  abstract     = {Hemolysis drives susceptibility to bacterial infections and predicts poor outcome from sepsis. These detrimental effects are commonly considered to be a consequence of heme-iron serving as a nutrient for bacteria. We employed a Gram-negative sepsis model and found that elevated heme levels impaired the control of bacterial proliferation independently of heme-iron acquisition by pathogens. Heme strongly inhibited phagocytosis and the migration of human and mouse phagocytes by disrupting actin cytoskeletal dynamics via activation of the GTP-binding Rho family protein Cdc42 by the guanine nucleotide exchange factor DOCK8. A chemical screening approach revealed that quinine effectively prevented heme effects on the cytoskeleton, restored phagocytosis and improved survival in sepsis. These mechanistic insights provide potential therapeutic targets for patients with sepsis or hemolytic disorders.},
  author       = {Martins, Rui and Maier, Julia and Gorki, Anna and Huber, Kilian and Sharif, Omar and Starkl, Philipp and Saluzzo, Simona and Quattrone, Federica and Gawish, Riem and Lakovits, Karin and Aichinger, Michael and Radic Sarikas, Branka and Lardeau, Charles and Hladik, Anastasiya and Korosec, Ana and Brown, Markus and Vaahtomeri, Kari and Duggan, Michelle and Kerjaschki, Dontscho and Esterbauer, Harald and Colinge, Jacques and Eisenbarth, Stephanie and Decker, Thomas and Bennett, Keiryn and Kubicek, Stefan and Sixt, Michael K and Superti Furga, Giulio and Knapp, Sylvia},
  journal      = {Nature Immunology},
  number       = {12},
  pages        = {1361 -- 1372},
  publisher    = {Nature Publishing Group},
  title        = {{Heme drives hemolysis-induced susceptibility to infection via disruption of phagocyte functions}},
  doi          = {10.1038/ni.3590},
  volume       = {17},
  year         = {2016},
}

@article{1143,
  abstract     = {We study the ground state of a dilute Bose gas in a scaling limit where the Gross-Pitaevskii functional emerges. This is a repulsive nonlinear Schrödinger functional whose quartic term is proportional to the scattering length of the interparticle interaction potential. We propose a new derivation of this limit problem, with a method that bypasses some of the technical difficulties that previous derivations had to face. The new method is based on a combination of Dyson\'s lemma, the quantum de Finetti theorem and a second moment estimate for ground states of the effective Dyson Hamiltonian. It applies equally well to the case where magnetic fields or rotation are present.},
  author       = {Nam, Phan and Rougerie, Nicolas and Seiringer, Robert},
  journal      = {Analysis and PDE},
  number       = {2},
  pages        = {459 -- 485},
  publisher    = {Mathematical Sciences Publishers},
  title        = {{Ground states of large bosonic systems: The gross Pitaevskii limit revisited}},
  doi          = {10.2140/apde.2016.9.459},
  volume       = {9},
  year         = {2016},
}

@article{1145,
  abstract     = {Auxin directs plant ontogenesis via differential accumulation within tissues depending largely on the activity of PIN proteins that mediate auxin efflux from cells and its directional cell-to-cell transport. Regardless of the developmental importance of PINs, the structure of these transporters is poorly characterized. Here, we present experimental data concerning protein topology of plasma membrane-localized PINs. Utilizing approaches based on pH-dependent quenching of fluorescent reporters combined with immunolocalization techniques, we mapped the membrane topology of PINs and further cross-validated our results using available topology modeling software. We delineated the topology of PIN1 with two transmembrane (TM) bundles of five α-helices linked by a large intracellular loop and a C-terminus positioned outside the cytoplasm. Using constraints derived from our experimental data, we also provide an updated position of helical regions generating a verisimilitude model of PIN1. Since the canonical long PINs show a high degree of conservation in TM domains and auxin transport capacity has been demonstrated for Arabidopsis representatives of this group, this empirically enhanced topological model of PIN1 will be an important starting point for further studies on PIN structure–function relationships. In addition, we have established protocols that can be used to probe the topology of other plasma membrane proteins in plants. © 2016 The Authors},
  author       = {Nodzyński, Tomasz and Vanneste, Steffen and Zwiewka, Marta and Pernisová, Markéta and Hejátko, Jan and Friml, Jirí},
  journal      = {Molecular Plant},
  number       = {11},
  pages        = {1504 -- 1519},
  publisher    = {Cell Press},
  title        = {{Enquiry into the topology of plasma membrane localized PIN auxin transport components}},
  doi          = {10.1016/j.molp.2016.08.010},
  volume       = {9},
  year         = {2016},
}

@article{1147,
  abstract     = {Apical dominance is one of the fundamental developmental phenomena in plant biology, which determines the overall architecture of aerial plant parts. Here we show apex decapitation activated competition for dominance in adjacent upper and lower axillary buds. A two-nodal-bud pea (Pisum sativum L.) was used as a model system to monitor and assess auxin flow, auxin transport channels, and dormancy and initiation status of axillary buds. Auxin flow was manipulated by lateral stem wounds or chemically by auxin efflux inhibitors 2,3,5-triiodobenzoic acid (TIBA), 1-N-naphtylphtalamic acid (NPA), or protein synthesis inhibitor cycloheximide (CHX) treatments, which served to interfere with axillary bud competition. Redirecting auxin flow to different points influenced which bud formed the outgrowing and dominant shoot. The obtained results proved that competition between upper and lower axillary buds as secondary auxin sources is based on the same auxin canalization principle that operates between the shoot apex and axillary bud. © The Author(s) 2016.},
  author       = {Balla, Jozef and Medved'Ová, Zuzana and Kalousek, Petr and Matiješčuková, Natálie and Friml, Jirí and Reinöhl, Vilém and Procházka, Stanislav},
  journal      = {Scientific Reports},
  publisher    = {Nature Publishing Group},
  title        = {{Auxin flow mediated competition between axillary buds to restore apical dominance}},
  doi          = {10.1038/srep35955},
  volume       = {6},
  year         = {2016},
}

@article{1148,
  abstract     = {Continuous-time Markov chain (CTMC) models have become a central tool for understanding the dynamics of complex reaction networks and the importance of stochasticity in the underlying biochemical processes. When such models are employed to answer questions in applications, in order to ensure that the model provides a sufficiently accurate representation of the real system, it is of vital importance that the model parameters are inferred from real measured data. This, however, is often a formidable task and all of the existing methods fail in one case or the other, usually because the underlying CTMC model is high-dimensional and computationally difficult to analyze. The parameter inference methods that tend to scale best in the dimension of the CTMC are based on so-called moment closure approximations. However, there exists a large number of different moment closure approximations and it is typically hard to say a priori which of the approximations is the most suitable for the inference procedure. Here, we propose a moment-based parameter inference method that automatically chooses the most appropriate moment closure method. Accordingly, contrary to existing methods, the user is not required to be experienced in moment closure techniques. In addition to that, our method adaptively changes the approximation during the parameter inference to ensure that always the best approximation is used, even in cases where different approximations are best in different regions of the parameter space. © 2016 Elsevier Ireland Ltd},
  author       = {Schilling, Christian and Bogomolov, Sergiy and Henzinger, Thomas A and Podelski, Andreas and Ruess, Jakob},
  journal      = {Biosystems},
  pages        = {15 -- 25},
  publisher    = {Elsevier},
  title        = {{Adaptive moment closure for parameter inference of biochemical reaction networks}},
  doi          = {10.1016/j.biosystems.2016.07.005},
  volume       = {149},
  year         = {2016},
}

@article{1149,
  abstract     = {We study the usefulness of two most prominent publicly available rigorous ODE integrators: one provided by the CAPD group (capd.ii.uj.edu.pl), the other based on the COSY Infinity project (cosyinfinity.org). Both integrators are capable of handling entire sets of initial conditions and provide tight rigorous outer enclosures of the images under a time-T map. We conduct extensive benchmark computations using the well-known Lorenz system, and compare the computation time against the final accuracy achieved. We also discuss the effect of a few technical parameters, such as the order of the numerical integration method, the value of T, and the phase space resolution. We conclude that COSY may provide more precise results due to its ability of avoiding the variable dependency problem. However, the overall cost of computations conducted using CAPD is typically lower, especially when intervals of parameters are involved. Moreover, access to COSY is limited (registration required) and the rigorous ODE integrators are not publicly available, while CAPD is an open source free software project. Therefore, we recommend the latter integrator for this kind of computations. Nevertheless, proper choice of the various integration parameters turns out to be of even greater importance than the choice of the integrator itself. © 2016 IMACS. Published by Elsevier B.V. All rights reserved.},
  author       = {Miyaji, Tomoyuki and Pilarczyk, Pawel and Gameiro, Marcio and Kokubu, Hiroshi and Mischaikow, Konstantin},
  journal      = {Applied Numerical Mathematics},
  pages        = {34 -- 47},
  publisher    = {Elsevier},
  title        = {{A study of rigorous ODE integrators for multi scale set oriented computations}},
  doi          = {10.1016/j.apnum.2016.04.005},
  volume       = {107},
  year         = {2016},
}

@article{1150,
  abstract     = {When neutrophils infiltrate a site of inflammation, they have to stop at the right place to exert their effector function. In this issue of Developmental Cell, Wang et al. (2016) show that neutrophils sense reactive oxygen species via the TRPM2 channel to arrest migration at their target site. © 2016 Elsevier Inc.},
  author       = {Renkawitz, Jörg and Sixt, Michael K},
  journal      = {Developmental Cell},
  number       = {5},
  pages        = {448 -- 450},
  publisher    = {Cell Press},
  title        = {{A Radical Break Restraining Neutrophil Migration}},
  doi          = {10.1016/j.devcel.2016.08.017},
  volume       = {38},
  year         = {2016},
}

@article{1151,
  abstract     = {Tissue patterning in multicellular organisms is the output of precise spatio–temporal regulation of gene expression coupled with changes in hormone dynamics. In plants, the hormone auxin regulates growth and development at every stage of a plant’s life cycle. Auxin signaling occurs through binding of the auxin molecule to a TIR1/AFB F-box ubiquitin ligase, allowing interaction with Aux/IAA transcriptional repressor proteins. These are subsequently ubiquitinated and degraded via the 26S proteasome, leading to derepression of auxin response factors (ARFs). How auxin is able to elicit such a diverse range of developmental responses through a single signaling module has not yet been resolved. Here we present an alternative auxin-sensing mechanism in which the ARF ARF3/ETTIN controls gene expression through interactions with process-specific transcription factors. This noncanonical hormonesensing mechanism exhibits strong preference for the naturally occurring auxin indole 3-acetic acid (IAA) and is important for coordinating growth and patterning in diverse developmental contexts such as gynoecium morphogenesis, lateral root emergence, ovule development, and primary branch formation. Disrupting this IAA-sensing ability induces morphological aberrations with consequences for plant fitness. Therefore, our findings introduce a novel transcription factor-based mechanism of hormone perception in plants. © 2016 Simonini et al.},
  author       = {Simonini, Sara and Deb, Joyita and Moubayidin, Laila and Stephenson, Pauline and Valluru, Manoj and Freire Rios, Alejandra and Sorefan, Karim and Weijers, Dolf and Friml, Jirí and Östergaard, Lars},
  journal      = {Genes and Development},
  number       = {20},
  pages        = {2286 -- 2296},
  publisher    = {Cold Spring Harbor Laboratory Press},
  title        = {{A noncanonical auxin sensing mechanism is required for organ morphogenesis in arabidopsis}},
  doi          = {10.1101/gad.285361.116},
  volume       = {30},
  year         = {2016},
}

@article{1153,
  abstract     = {Differential cell growth enables flexible organ bending in the presence of environmental signals such as light or gravity. A prominent example of the developmental processes based on differential cell growth is the formation of the apical hook that protects the fragile shoot apical meristem when it breaks through the soil during germination. Here, we combined in silico and in vivo approaches to identify a minimal mechanism producing auxin gradient-guided differential growth during the establishment of the apical hook in the model plant Arabidopsis thaliana. Computer simulation models based on experimental data demonstrate that asymmetric expression of the PIN-FORMED auxin efflux carrier at the concave (inner) versus convex (outer) side of the hook suffices to establish an auxin maximum in the epidermis at the concave side of the apical hook. Furthermore, we propose a mechanism that translates this maximum into differential growth, and thus curvature, of the apical hook. Through a combination of experimental and in silico computational approaches, we have identified the individual contributions of differential cell elongation and proliferation to defining the apical hook and reveal the role of auxin-ethylene crosstalk in balancing these two processes. © 2016 American Society of Plant Biologists. All rights reserved.},
  author       = {Žádníková, Petra and Wabnik, Krzysztof T and Abuzeineh, Anas and Gallemí, Marçal and Van Der Straeten, Dominique and Smith, Richard and Inze, Dirk and Friml, Jirí and Prusinkiewicz, Przemysław and Benková, Eva},
  journal      = {Plant Cell},
  number       = {10},
  pages        = {2464 -- 2477},
  publisher    = {American Society of Plant Biologists},
  title        = {{A model of differential growth guided apical hook formation in plants}},
  doi          = {10.1105/tpc.15.00569},
  volume       = {28},
  year         = {2016},
}

@article{1154,
  abstract     = {Cellular locomotion is a central hallmark of eukaryotic life. It is governed by cell-extrinsic molecular factors, which can either emerge in the soluble phase or as immobilized, often adhesive ligands. To encode for direction, every cue must be present as a spatial or temporal gradient. Here, we developed a microfluidic chamber that allows measurement of cell migration in combined response to surface immobilized and soluble molecular gradients. As a proof of principle we study the response of dendritic cells to their major guidance cues, chemokines. The majority of data on chemokine gradient sensing is based on in vitro studies employing soluble gradients. Despite evidence suggesting that in vivo chemokines are often immobilized to sugar residues, limited information is available how cells respond to immobilized chemokines. We tracked migration of dendritic cells towards immobilized gradients of the chemokine CCL21 and varying superimposed soluble gradients of CCL19. Differential migratory patterns illustrate the potential of our setup to quantitatively study the competitive response to both types of gradients. Beyond chemokines our approach is broadly applicable to alternative systems of chemo- and haptotaxis such as cells migrating along gradients of adhesion receptor ligands vs. any soluble cue. 
},
  author       = {Schwarz, Jan and Bierbaum, Veronika and Merrin, Jack and Frank, Tino and Hauschild, Robert and Bollenbach, Mark Tobias and Tay, Savaş and Sixt, Michael K and Mehling, Matthias},
  journal      = {Scientific Reports},
  publisher    = {Nature Publishing Group},
  title        = {{A microfluidic device for measuring cell migration towards substrate bound and soluble chemokine gradients}},
  doi          = {10.1038/srep36440},
  volume       = {6},
  year         = {2016},
}

@article{1156,
  abstract     = {Let k, n, and r be positive integers with k < n and r≤⌊nk⌋. We determine the facets of the r-stable n, k-hypersimplex. As a result, it turns out that the r-stable n, k-hypersimplex has exactly 2n facets for every r<⌊nk⌋. We then utilize the equations of the facets to study when the r-stable hypersimplex is Gorenstein. For every k > 0 we identify an infinite collection of Gorenstein r-stable hypersimplices, consequently expanding the collection of r-stable hypersimplices known to have unimodal Ehrhart δ-vectors.},
  author       = {Hibi, Takayugi and Liam Solus},
  journal      = {Annals of Combinatorics},
  number       = {4},
  pages        = {815 -- 829},
  publisher    = {Springer},
  title        = {{Facets of the r-stable (n, k)-hypersimplex}},
  doi          = {10.1007/s00026-016-0325-x},
  volume       = {20},
  year         = {2016},
}

@article{1157,
  abstract     = {We consider sample covariance matrices of the form Q = ( σ1/2X)(σ1/2X)∗, where the sample X is an M ×N random matrix whose entries are real independent random variables with variance 1/N and whereσ is an M × M positive-definite deterministic matrix. We analyze the asymptotic fluctuations of the largest rescaled eigenvalue of Q when both M and N tend to infinity with N/M →d ϵ (0,∞). For a large class of populations σ in the sub-critical regime, we show that the distribution of the largest rescaled eigenvalue of Q is given by the type-1 Tracy-Widom distribution under the additional assumptions that (1) either the entries of X are i.i.d. Gaussians or (2) that σ is diagonal and that the entries of X have a sub-exponential decay.},
  author       = {Lee, Ji and Schnelli, Kevin},
  journal      = {Annals of Applied Probability},
  number       = {6},
  pages        = {3786 -- 3839},
  publisher    = {Institute of Mathematical Statistics},
  title        = {{Tracy-widom distribution for the largest eigenvalue of real sample covariance matrices with general population}},
  doi          = {10.1214/16-AAP1193},
  volume       = {26},
  year         = {2016},
}

@article{11574,
  abstract     = {We present new results from the widest narrow-band survey search for Lyα emitters at z = 5.7, just after reionization. We survey a total of 7 deg2 spread over the COSMOS, UDS and SA22 fields. We find over 11 000 line emitters, out of which 514 are robust Lyα candidates at z = 5.7 within a volume of 6.3 × 106 Mpc3. Our Lyα emitters span a wide range in Lyα luminosities, from faint to bright (LLyα ∼ 1042.5–44 erg s−1) and rest-frame equivalent widths (EW0 ∼ 25–1000 Å) in a single, homogeneous data set. By combining all our fields, we find that the faint end slope of the z = 5.7 Lyα luminosity function is very steep, with α=−2.3+0.4−0.3⁠. We also present an updated z = 6.6 Lyα luminosity function, based on comparable volumes and obtained with the same methods, which we directly compare with that at z = 5.7. We find a significant decline of the number density of faint Lyα emitters from z = 5.7 to 6.6 (by 0.5 ± 0.1 dex), but no evolution at the bright end/no evolution in L*. Faint Lyα emitters at z = 6.6 show much more extended haloes than those at z = 5.7, suggesting that neutral Hydrogen plays an important role, increasing the scattering and leading to observations missing faint Lyα emission within the epoch of reionization. Altogether, our results suggest that we are observing patchy reionization which happens first around the brightest Lyα emitters, allowing the number densities of those sources to remain unaffected by the increase of neutral Hydrogen fraction from z ∼ 5 to 7.},
  author       = {Santos, Sérgio and Sobral, David and Matthee, Jorryt J},
  issn         = {1365-2966},
  journal      = {Monthly Notices of the Royal Astronomical Society},
  keywords     = {Space and Planetary Science, Astronomy and Astrophysics, galaxies: high-redshift, galaxies: luminosity function, mass function, cosmology: observations, dark ages, reionization, first stars},
  number       = {2},
  pages        = {1678--1691},
  publisher    = {Oxford University Press},
  title        = {{The Lyα luminosity function at z= 5.7–6.6 and the steep drop of the faint end: Implications for reionization}},
  doi          = {10.1093/mnras/stw2076},
  volume       = {463},
  year         = {2016},
}

@article{11575,
  abstract     = {We investigate correlations between different physical properties of star-forming galaxies in the ‘Evolution and Assembly of GaLaxies and their Environments’ (EAGLE) cosmological hydrodynamical simulation suite over the redshift range 0 ≤ z ≤ 4.5. A principal component analysis reveals that neutral gas fraction (fgas,neutral), stellar mass (Mstellar) and star formation rate (SFR) account for most of the variance seen in the population, with galaxies tracing a two-dimensional, nearly flat, surface in the three-dimensional space of fgas, neutral–Mstellar–SFR with little scatter. The location of this plane varies little with redshift, whereas galaxies themselves move along the plane as their fgas, neutral and SFR drop with redshift. The positions of galaxies along the plane are highly correlated with gas metallicity. The metallicity can therefore be robustly predicted from fgas, neutral, or from the Mstellar and SFR. We argue that the appearance of this ‘Fundamental Plane of star formation’ is a consequence of self-regulation, with the plane's curvature set by the dependence of the SFR on gas density and metallicity. We analyse a large compilation of observations spanning the redshift range 0 ≲ z ≲ 3, and find that such a plane is also present in the data. The properties of the observed Fundamental Plane of star formation are in good agreement with EAGLE's predictions.},
  author       = {Lagos, Claudia del P. and Theuns, Tom and Schaye, Joop and Furlong, Michelle and Bower, Richard G. and Schaller, Matthieu and Crain, Robert A. and Trayford, James W. and Matthee, Jorryt J},
  issn         = {1365-2966},
  journal      = {Monthly Notices of the Royal Astronomical Society},
  keywords     = {Space and Planetary Science, Astronomy and Astrophysics  stars: formation, ISM: evolution, galaxies: evolution, galaxies: formation, galaxies: ISM},
  number       = {3},
  pages        = {2632--2650},
  publisher    = {Oxford University Press},
  title        = {{The Fundamental Plane of star formation in galaxies revealed by the EAGLE hydrodynamical simulations}},
  doi          = {10.1093/mnras/stw717},
  volume       = {459},
  year         = {2016},
}