@inproceedings{2973,
  abstract     = {Efficient zero-knowledge proofs of knowledge (ZK-PoK) are basic building blocks of many practical cryptographic applications such as identification schemes, group signatures, and secure multiparty computation. Currently, first applications that critically rely on ZK-PoKs are being deployed in the real world. The most prominent example is Direct Anonymous Attestation (DAA), which was adopted by the Trusted Computing Group (TCG) and implemented as one of the functionalities of the cryptographic Trusted Platform Module (TPM) chip.

Implementing systems using ZK-PoK turns out to be challenging, since ZK-PoK are, loosely speaking, significantly more complex than standard crypto primitives, such as encryption and signature schemes. As a result, implementation cycles of ZK-PoK are time-consuming and error-prone, in particular for developers with minor or no cryptographic skills. 

In this paper we report on our ongoing and future research vision with the goal to bring ZK-PoK to practice by making them accessible to crypto and security engineers. To this end we are developing compilers and related tools that support and partially automate the design, implementation, verification and secure implementation of ZK-PoK protocols.},
  author       = {Bangerter, Endre and Barzan, Stefania and Stephan Krenn and Sadeghi, Ahmad-Reza and Schneider, Thomas and Tsay, Joe-Kai},
  editor       = {Christianson, Bruce and Malcolm, James A. and Matyas, Vashek and Roe, Michael},
  pages        = {51 -- 62},
  publisher    = {Springer},
  title        = {{Bringing Zero-Knowledge Proofs of Knowledge to Practice}},
  doi          = {10.1007/978-3-642-36213-2_9},
  volume       = {7028},
  year         = {2013},
}

@article{3261,
  abstract     = {Cells in a developing embryo have no direct way of "measuring" their physical position. Through a variety of processes, however, the expression levels of multiple genes come to be correlated with position, and these expression levels thus form a code for "positional information." We show how to measure this information, in bits, using the gap genes in the Drosophila embryo as an example. Individual genes carry nearly two bits of information, twice as much as expected if the expression patterns consisted only of on/off domains separated by sharp boundaries. Taken together, four gap genes carry enough information to define a cell's location with an error bar of ~1% along the anterior-posterior axis of the embryo. This precision is nearly enough for each cell to have a unique identity, which is the maximum information the system can use, and is nearly constant along the length of the embryo. We argue that this constancy is a signature of optimality in the transmission of information from primary morphogen inputs to the output of the gap gene network.},
  author       = {Dubuis, Julien and Tkacik, Gasper and Wieschaus, Eric and Gregor, Thomas and Bialek, William},
  journal      = {PNAS},
  number       = {41},
  pages        = {16301 -- 16308},
  publisher    = {National Academy of Sciences},
  title        = {{Positional information, in bits}},
  doi          = {10.1073/pnas.1315642110},
  volume       = {110},
  year         = {2013},
}

@inproceedings{10749,
  abstract     = {Fluxoid quantization provides a direct means to study phase coherence. In cuprate superconductors, there have been observations which suggest that phase coherent superconducting fluctuations may persist at temperatures significantly above Tc. The focus of this work is to study the vortex states in mesoscopic cuprate superconducting samples to directly probe phase coherence over a wide range of temperatures. We present cantilever torque susceptometry measurements of micron and sub-micron size Bi2212 rings and disks. The high sensitivity of this technique allowed observation of transitions between different fluxoid states of a single ring, and the discrete vortex states of micron size disks. The dependence of magnetic susceptibility on diameter and wall thickness of the ring was investigated. Measurements were made at different values of the in-plane magnetic field, and over a wide range of temperatures.},
  author       = {Polshyn, Hryhoriy and Budakian, Raffi and Gu, Genda},
  booktitle    = {APS March Meeting 2013},
  issn         = {0003-0503},
  location     = {Baltimore, MD, United States},
  number       = {1},
  publisher    = {American Physical Society},
  title        = {{Cantilever micro-susceptometry of mesoscopic Bi2212 samples}},
  volume       = {58},
  year         = {2013},
}

@article{10895,
  abstract     = {Due to their sessile lifestyles, plants need to deal with the limitations and stresses imposed by the changing environment. Plants cope with these by a remarkable developmental flexibility, which is embedded in their strategy to survive. Plants can adjust their size, shape and number of organs, bend according to gravity and light, and regenerate tissues that were damaged, utilizing a coordinating, intercellular signal, the plant hormone, auxin. Another versatile signal is the cation, Ca2+, which is a crucial second messenger for many rapid cellular processes during responses to a wide range of endogenous and environmental signals, such as hormones, light, drought stress and others. Auxin is a good candidate for one of these Ca2+-activating signals. However, the role of auxin-induced Ca2+ signaling is poorly understood. Here, we will provide an overview of possible developmental and physiological roles, as well as mechanisms underlying the interconnection of Ca2+ and auxin signaling. },
  author       = {Vanneste, Steffen and Friml, Jiří},
  issn         = {2223-7747},
  journal      = {Plants},
  keywords     = {Plant Science, Ecology, Ecology, Evolution, Behavior and Systematics},
  number       = {4},
  pages        = {650--675},
  publisher    = {MDPI},
  title        = {{Calcium: The missing link in auxin action}},
  doi          = {10.3390/plants2040650},
  volume       = {2},
  year         = {2013},
}

@article{11085,
  abstract     = {During mitotic exit, missegregated chromosomes can recruit their own nuclear envelope (NE) to form micronuclei (MN). MN have reduced functioning compared to primary nuclei in the same cell, although the two compartments appear to be structurally comparable. Here we show that over 60% of MN undergo an irreversible loss of compartmentalization during interphase due to NE collapse. This disruption of the MN, which is induced by defects in nuclear lamina assembly, drastically reduces nuclear functions and can trigger massive DNA damage. MN disruption is associated with chromatin compaction and invasion of endoplasmic reticulum (ER) tubules into the chromatin. We identified disrupted MN in both major subtypes of human non-small-cell lung cancer, suggesting that disrupted MN could be a useful objective biomarker for genomic instability in solid tumors. Our study shows that NE collapse is a key event underlying MN dysfunction and establishes a link between aberrant NE organization and aneuploidy.},
  author       = {Hatch, Emily M. and Fischer, Andrew H. and Deerinck, Thomas J. and HETZER, Martin W},
  issn         = {0092-8674},
  journal      = {Cell},
  keywords     = {General Biochemistry, Genetics and Molecular Biology},
  number       = {1},
  pages        = {47--60},
  publisher    = {Elsevier},
  title        = {{Catastrophic nuclear envelope collapse in cancer cell micronuclei}},
  doi          = {10.1016/j.cell.2013.06.007},
  volume       = {154},
  year         = {2013},
}

@article{11086,
  abstract     = {Faithful execution of developmental gene expression programs occurs at multiple levels and involves many different components such as transcription factors, histone-modification enzymes, and mRNA processing proteins. Recent evidence suggests that nucleoporins, well known components that control nucleo-cytoplasmic trafficking, have wide-ranging functions in developmental gene regulation that potentially extend beyond their role in nuclear transport. Whether the unexpected role of nuclear pore proteins in transcription regulation, which initially has been described in fungi and flies, also applies to human cells is unknown. Here we show at a genome-wide level that the nuclear pore protein NUP98 associates with developmentally regulated genes active during human embryonic stem cell differentiation. Overexpression of a dominant negative fragment of NUP98 levels decreases expression levels of NUP98-bound genes. In addition, we identify two modes of developmental gene regulation by NUP98 that are differentiated by the spatial localization of NUP98 target genes. Genes in the initial stage of developmental induction can associate with NUP98 that is embedded in the nuclear pores at the nuclear periphery. Alternatively, genes that are highly induced can interact with NUP98 in the nuclear interior, away from the nuclear pores. This work demonstrates for the first time that NUP98 dynamically associates with the human genome during differentiation, revealing a role of a nuclear pore protein in regulating developmental gene expression programs.},
  author       = {Liang, Yun and Franks, Tobias M. and Marchetto, Maria C. and Gage, Fred H. and HETZER, Martin W},
  issn         = {1553-7404},
  journal      = {PLoS Genetics},
  keywords     = {Cancer Research, Genetics (clinical), Genetics, Molecular Biology, Ecology, Evolution, Behavior and Systematics},
  number       = {2},
  publisher    = {Public Library of Science},
  title        = {{Dynamic association of NUP98 with the human genome}},
  doi          = {10.1371/journal.pgen.1003308},
  volume       = {9},
  year         = {2013},
}

@article{11087,
  abstract     = {Intracellular proteins with long lifespans have recently been linked to age-dependent defects, ranging from decreased fertility to the functional decline of neurons. Why long-lived proteins exist in metabolically active cellular environments and how they are maintained over time remains poorly understood. Here, we provide a system-wide identification of proteins with exceptional lifespans in the rat brain. These proteins are inefficiently replenished despite being translated robustly throughout adulthood. Using nucleoporins as a paradigm for long-term protein persistence, we found that nuclear pore complexes (NPCs) are maintained over a cell’s life through slow but finite exchange of even its most stable subcomplexes. This maintenance is limited, however, as some nucleoporin levels decrease during aging, providing a rationale for the previously observed age-dependent deterioration of NPC function. Our identification of a long-lived proteome reveals cellular components that are at increased risk for damage accumulation, linking long-term protein persistence to the cellular aging process.},
  author       = {Toyama, Brandon H. and Savas, Jeffrey N. and Park, Sung Kyu and Harris, Michael S. and Ingolia, Nicholas T. and Yates, John R. and HETZER, Martin W},
  issn         = {0092-8674},
  journal      = {Cell},
  keywords     = {General Biochemistry, Genetics and Molecular Biology},
  number       = {5},
  pages        = {971--982},
  publisher    = {Elsevier},
  title        = {{Identification of long-lived proteins reveals exceptional stability of essential cellular structures}},
  doi          = {10.1016/j.cell.2013.07.037},
  volume       = {154},
  year         = {2013},
}

@article{11088,
  abstract     = {The crowded intracellular environment poses a formidable challenge to experimental and theoretical analyses of intracellular transport mechanisms. Our measurements of single-particle trajectories in cytoplasm and their random-walk interpretations elucidate two of these mechanisms: molecular diffusion in crowded environments and cytoskeletal transport along microtubules. We employed acousto-optic deflector microscopy to map out the three-dimensional trajectories of microspheres migrating in the cytosolic fraction of a cellular extract. Classical Brownian motion (BM), continuous time random walk, and fractional BM were alternatively used to represent these trajectories. The comparison of the experimental and numerical data demonstrates that cytoskeletal transport along microtubules and diffusion in the cytosolic fraction exhibit anomalous (nonFickian) behavior and posses statistically distinct signatures. Among the three random-walk models used, continuous time random walk provides the best representation of diffusion, whereas microtubular transport is accurately modeled with fractional BM.},
  author       = {Regner, Benjamin M. and Vučinić, Dejan and Domnisoru, Cristina and Bartol, Thomas M. and HETZER, Martin W and Tartakovsky, Daniel M. and Sejnowski, Terrence J.},
  issn         = {0006-3495},
  journal      = {Biophysical Journal},
  keywords     = {Biophysics},
  number       = {8},
  pages        = {1652--1660},
  publisher    = {Elsevier},
  title        = {{Anomalous diffusion of single particles in cytoplasm}},
  doi          = {10.1016/j.bpj.2013.01.049},
  volume       = {104},
  year         = {2013},
}

@article{11520,
  abstract     = {We present the spatially resolved Hα dynamics of 16 star-forming galaxies at z ∼ 0.81 using the new KMOS multi-object integral field spectrograph on the ESO Very Large Telescope. These galaxies, selected using 1.18 μm narrowband imaging from the 10 deg2 CFHT-HiZELS survey of the SA 22 hr field, are found in a ∼4 Mpc overdensity of Hα emitters and likely reside in a group/intermediate environment, but not a cluster. We confirm and identify a rich group of star-forming galaxies at z = 0.813 ± 0.003, with 13 galaxies within 1000 km s−1 of each other, and seven within a diameter of 3 Mpc. All of our galaxies are “typical” star-forming galaxies at their redshift, 0.8 ± 0.4 SFR$^*_{z = 0.8}$, spanning a range of specific star formation rates (sSFRs) of 0.2–1.1 Gyr−1 and have a median metallicity very close to solar of 12 + log(O/H) = 8.62 ± 0.06. We measure the spatially resolved Hα dynamics of the galaxies in our sample and show that 13 out of 16 galaxies can be described by rotating disks and use the data to derive inclination corrected rotation speeds of 50–275 km s−1. The fraction of disks within our sample is 75% ± 8%, consistent with previous results based on Hubble Space Telescope morphologies of Hα-selected galaxies at z ∼ 1 and confirming that disks dominate the SFR density at z ∼ 1. Our Hα galaxies are well fitted by the z ∼ 1–2 Tully–Fisher (TF) relation, confirming the evolution seen in the zero point. Apart from having, on average, higher stellar masses and lower sSFRs, our group galaxies at z = 0.81 present the same mass–metallicity and TF relation as z ∼ 1 field galaxies and are all disk galaxies.},
  author       = {Sobral, D. and Swinbank, A. M. and Stott, J. P. and Matthee, Jorryt J and Bower, R. G. and Smail, Ian and Best, P. and Geach, J. E. and Sharples, R. M.},
  issn         = {1538-4357},
  journal      = {The Astrophysical Journal},
  keywords     = {Space and Planetary Science, Astronomy and Astrophysics, galaxies: evolution – galaxies, high-redshift – galaxies, starburst},
  number       = {2},
  publisher    = {IOP Publishing},
  title        = {{The dynamics of z=0.8 H-alpha-selected star-forming galaxies from KMOS/CF-HiZELS}},
  doi          = {10.1088/0004-637x/779/2/139},
  volume       = {779},
  year         = {2013},
}

@article{11759,
  abstract     = {Matching markets play a prominent role in economic theory. A prime example of such a market is the sponsored search market. Here, as in other markets of that kind, market equilibria correspond to feasible, envy free, and bidder optimal outcomes. For settings without budgets such an outcome always exists and can be computed in polynomial-time by the so-called Hungarian Method. Moreover, every mechanism that computes such an outcome is incentive compatible. We show that the Hungarian Method can be modified so that it finds a feasible, envy free, and bidder optimal outcome for settings with budgets. We also show that in settings with budgets no mechanism that computes such an outcome can be incentive compatible for all inputs. For inputs in general position, however, the presented mechanism—as any other mechanism that computes such an outcome for settings with budgets—is incentive compatible.},
  author       = {Dütting, Paul and Henzinger, Monika H and Weber, Ingmar},
  issn         = {0020-0190},
  journal      = {Information Processing Letters},
  number       = {3},
  pages        = {67--73},
  publisher    = {Elsevier},
  title        = {{Sponsored search, market equilibria, and the Hungarian Method}},
  doi          = {10.1016/j.ipl.2012.11.006},
  volume       = {113},
  year         = {2013},
}

@inproceedings{11791,
  abstract     = {The focus of classic mechanism design has been on truthful direct-revelation mechanisms. In the context of combinatorial auctions the truthful direct-revelation mechanism that maximizes social welfare is the VCG mechanism. For many valuation spaces computing the allocation and payments of the VCG mechanism, however, is a computationally hard problem. We thus study the performance of the VCG mechanism when bidders are forced to choose bids from a subspace of the valuation space for which the VCG outcome can be computed efficiently. We prove improved upper bounds on the welfare loss for restrictions to additive bids and upper and lower bounds for restrictions to non-additive bids. These bounds show that the welfare loss increases in expressiveness. All our bounds apply to equilibrium concepts that can be computed in polynomial time as well as to learning outcomes.},
  author       = {Dütting, Paul and Henzinger, Monika H and Starnberger, Martin},
  booktitle    = {9th International Conference on Web and Internet Economics},
  isbn         = {9783642450457},
  issn         = {1611-3349},
  location     = {Cambridge, MA, USA},
  pages        = {146–159},
  publisher    = {Springer Nature},
  title        = {{Valuation compressions in VCG-based combinatorial auctions}},
  doi          = {10.1007/978-3-642-45046-4_13},
  volume       = {8289},
  year         = {2013},
}

@inproceedings{11792,
  abstract     = {We study the problem of maximizing a monotone submodular function with viability constraints. This problem originates from computational biology, where we are given a phylogenetic tree over a set of species and a directed graph, the so-called food web, encoding viability constraints between these species. These food webs usually have constant depth. The goal is to select a subset of k species that satisfies the viability constraints and has maximal phylogenetic diversity. As this problem is known to be NP-hard, we investigate approximation algorithm. We present the first constant factor approximation algorithm if the depth is constant. Its approximation ratio is (1−1𝑒√). This algorithm not only applies to phylogenetic trees with viability constraints but for arbitrary monotone submodular set functions with viability constraints. Second, we show that there is no (1 − 1/e + ε)-approximation algorithm for our problem setting (even for additive functions) and that there is no approximation algorithm for a slight extension of this setting.},
  author       = {Dvořák, Wolfgang and Henzinger, Monika H and Williamson, David P.},
  booktitle    = {21st Annual European Symposium on Algorithms},
  isbn         = {9783642404498},
  issn         = {1611-3349},
  location     = {Sophia Antipolis, France},
  pages        = {409 -- 420},
  publisher    = {Springer Nature},
  title        = {{Maximizing a submodular function with viability constraints}},
  doi          = {10.1007/978-3-642-40450-4_35},
  volume       = {8125},
  year         = {2013},
}

@inproceedings{11793,
  abstract     = {We study the problem of maintaining a breadth-first spanning tree (BFS tree) in partially dynamic distributed networks modeling a sequence of either failures or additions of communication links (but not both). We show (1 + ε)-approximation algorithms whose amortized time (over some number of link changes) is sublinear in D, the maximum diameter of the network. This breaks the Θ(D) time bound of recomputing “from scratch”.

Our technique also leads to a (1 + ε)-approximate incremental algorithm for single-source shortest paths (SSSP) in the sequential (usual RAM) model. Prior to our work, the state of the art was the classic exact algorithm of [9] that is optimal under some assumptions [27]. Our result is the first to show that, in the incremental setting, this bound can be beaten in certain cases if a small approximation is allowed.},
  author       = {Henzinger, Monika H and Krinninger, Sebastian and Nanongkai, Danupon},
  booktitle    = {40th International Colloquium on Automata, Languages, and Programming},
  isbn         = {9783642392115},
  issn         = {1611-3349},
  location     = {Riga, Latvia},
  pages        = {607–619},
  publisher    = {Springer Nature},
  title        = {{Sublinear-time maintenance of breadth-first spanning tree in partially dynamic networks}},
  doi          = {10.1007/978-3-642-39212-2_53},
  volume       = {7966},
  year         = {2013},
}

@article{8030,
  abstract     = {While the plasticity of excitatory synaptic connections in the brain has been widely studied, the plasticity of inhibitory connections is much less understood. Here, we present recent experimental and theoretical findings concerning the rules of spike timing-dependent inhibitory plasticity and their putative network function. This is a summary of a workshop at the COSYNE conference 2012.},
  author       = {Vogels, Tim P and Froemke, R. C. and Doyon, N. and Gilson, M. and Haas, J. S. and Liu, R. and Maffei, A. and Miller, P. and Wierenga, C. J. and Woodin, M. A. and Zenke, F. and Sprekeler, H.},
  issn         = {1662-5110},
  journal      = {Frontiers in Neural Circuits},
  publisher    = {Frontiers Media},
  title        = {{Inhibitory synaptic plasticity: Spike timing-dependence and putative network function}},
  doi          = {10.3389/fncir.2013.00119},
  volume       = {7},
  year         = {2013},
}

@article{8245,
  abstract     = {Background: Monoclonal antibodies (mAb), such as trastuzumab are a valuable addition to breast cancer therapy.
Data obtained from neoadjuvant settings revealed that antibody-dependent cell-mediated cytotoxicity (ADCC) is a
major mechanism of action for the mAb trastuzumab. Conflicting results still call into question whether disease
progression, prolonged treatment or concomitant chemotherapy influences ADCC and related immunological
phenomena.
Methods: We analyzed the activity of ADCC and antibody-dependent cell-mediated phagocytosis (ADCP) of
peripheral blood mononuclear cells (PBMCs) from human epidermal growth factor receptor 2 (HER2/neu) positive
breast cancer patients receiving trastuzumab therapy either in an adjuvant (n = 13) or metastatic (n = 15) setting as
well as from trastuzumab treatment-naive (t-naive) HER2/neu negative patients (n = 15). PBMCs from healthy volunteers
(n = 24) were used as controls. ADCC and ADCP activity was correlated with the expression of antibody binding
Fc-gamma receptor (FcγR)I (CD64), FcγRII (CD32) and FcγRIII (CD16) on CD14+ (monocytes) and CD56+ (NK) cells, as well as the expression of CD107a+ (LAMP-1) on CD56+ cells and the total amount of CD4+CD25+FOXP3+ (Treg) cells. In metastatic patients, markers were correlated with progression-free survival (PFS).
Results: ADCC activity was significantly down regulated in metastatic, adjuvant and t-naive patient cohorts as compared to healthy controls. Reduced ADCC activity was inversely correlated with the expression of CD107a on CD56+
cells in adjuvant patients. ADCC and ADCP activity of the patient cohorts were similar, regardless of treatment duration
or additional chemotherapy. PFS in metastatic patients inversely correlated with the number of peripheral Treg cells.
Conclusion: The reduction of ADCC in patients as compared to healthy controls calls for adjuvant strategies, such as
immune-enhancing agents, to improve the activity of trastuzumab. However, efficacy of trastuzumab-specific ADCC
and ADCP appears not to be affected by treatment duration, disease progression or concomitant chemotherapy. This
finding supports the application of trastuzumab at any stage of the disease.},
  author       = {Petricevic, Branka and Laengle, Johannes and Singer, Josef and Sachet, Monika and Fazekas, Judit and Steger, Guenther and Bartsch, Rupert and Jensen-Jarolim, Erika and Bergmann, Michael},
  issn         = {1479-5876},
  journal      = {Journal of Translational Medicine},
  publisher    = {Springer Nature},
  title        = {{Trastuzumab mediates antibody-dependent cell-mediated cytotoxicity and phagocytosis to the same extent in both adjuvant and metastatic HER2/neu breast cancer patients}},
  doi          = {10.1186/1479-5876-11-307},
  volume       = {11},
  year         = {2013},
}

@article{827,
  abstract     = {As sessile organisms, plants have to be able to adapt to a continuously changing environment. Plants that perceive some of these changes as stress signals activate signaling pathways to modulate their development and to enable them to survive. The complex responses to environmental cues are to a large extent mediated by plant hormones that together orchestrate the final plant response. The phytohormone cytokinin is involved in many plant developmental processes. Recently, it has been established that cytokinin plays an important role in stress responses, but does not act alone. Indeed, the hormonal control of plant development and stress adaptation is the outcome of a complex network of multiple synergistic and antagonistic interactions between various hormones. Here, we review the recent findings on the cytokinin function as part of this hormonal network. We focus on the importance of the crosstalk between cytokinin and other hormones, such as abscisic acid, jasmonate, salicylic acid, ethylene, and auxin in the modulation of plant development and stress adaptation. Finally, the impact of the current research in the biotechnological industry will be discussed.},
  author       = {O'Brien, José and Benková, Eva},
  journal      = {Frontiers in Plant Science},
  publisher    = {Frontiers Research Foundation},
  title        = {{Cytokinin cross talking during biotic and abiotic stress responses}},
  doi          = {10.3389/fpls.2013.00451},
  volume       = {4},
  year         = {2013},
}

@article{828,
  abstract     = {The plant root system is essential for providing anchorage to the soil, supplying minerals and water, and synthesizing metabolites. It is a dynamic organ modulated by external cues such as environmental signals, water and nutrients availability, salinity and others. Lateral roots (LRs) are initiated from the primary root post-embryonically, after which they progress through discrete developmental stages which can be independently controlled, providing a high level of plasticity during root system formation. Within this review, main contributions are presented, from the classical forward genetic screens to the more recent high-throughput approaches, combined with computer model predictions, dissecting how LRs and thereby root system architecture is established and developed.},
  author       = {Cuesta, Candela and Wabnik, Krzysztof T and Benková, Eva},
  journal      = {Frontiers in Plant Science},
  publisher    = {Frontiers Research Foundation},
  title        = {{Systems approaches to study root architecture dynamics}},
  doi          = {10.3389/fpls.2013.00537},
  volume       = {4},
  year         = {2013},
}

@article{1759,
  abstract     = {We report an electric-field-induced giant modulation of the hole g factor in SiGe nanocrystals. The observed effect is ascribed to a so-far overlooked contribution to the g factor that stems from the mixing between heavy- and light-hole wave functions. We show that the relative displacement between the confined heavy- and light-hole states, occurring upon application of the electric field, alters their mixing strength leading to a strong nonmonotonic modulation of the g factor.},
  author       = {Ares, Natalia and Golovach, Vitaly N and Georgios Katsaros and Stoffel, Mathieu and Fournel, Frank and Glazman, Leonid I and Schmidt, Oliver G and De Franceschi, Silvano},
  journal      = {Physical Review Letters},
  number       = {4},
  publisher    = {American Physical Society},
  title        = {{Nature of tunable hole g factors in quantum dots}},
  doi          = {10.1103/PhysRevLett.110.046602},
  volume       = {110},
  year         = {2013},
}

@article{1760,
  abstract     = {We report on hole g-factor measurements in three terminal SiGe self-assembled quantum dot devices with a top gate electrode positioned very close to the nanostructure. Measurements of both the perpendicular as well as the parallel g-factor reveal significant changes for a small modulation of the top gate voltage. From the observed modulations, we estimate that, for realistic experimental conditions, hole spins can be electrically manipulated with Rabi frequencies in the order of 100 MHz. This work emphasises the potential of hole-based nano-devices for efficient spin manipulation by means of the g-tensor modulation technique.},
  author       = {Ares, Natalia and Georgios Katsaros and Golovach, Vitaly N and Zhang, Jianjun and Prager, Aaron A and Glazman, Leonid I and Schmidt, Oliver G and De Franceschi, Silvano},
  journal      = {Applied Physics Letters},
  number       = {26},
  publisher    = {American Institute of Physics},
  title        = {{SiGe quantum dots for fast hole spin Rabi oscillations}},
  doi          = {10.1063/1.4858959},
  volume       = {103},
  year         = {2013},
}

@article{1786,
  abstract     = {We report the experimental observation and a theoretical explanation of collective suppression of linewidths for multiple superconducting qubits coupled to a good cavity. This demonstrates how strong qubit-cavity coupling can significantly modify the dephasing and dissipation processes that might be expected for individual qubits, and can potentially improve coherence times in many-body circuit QED.},
  author       = {Nissen, Felix and Johannes Fink and Mlynek, Jonas A and Wallraff, Andreas and Keeling, Jonathan M},
  journal      = {Physical Review Letters},
  number       = {20},
  publisher    = {American Physical Society},
  title        = {{Collective suppression of linewidths in circuit QED}},
  doi          = {10.1103/PhysRevLett.110.203602},
  volume       = {110},
  year         = {2013},
}