@inbook{3456,
  abstract     = {L-a-amino-3-hydroxy-5-methyl-4-isoxazolepropionate receptors (AMPARs) and N-methyl-D-aspartate receptors (NMDARs) are the two major types of postsynaptic glutamate receptors (GluRs) that mediate excitatory synaptic transmission in the mammalian central nervous system (CNS). Both AMPARs and NMDARs are multimeric proteins, probably tetramers, formed by a variety of molecularly distinct subunits. AMPARs can be assembled from four types of subunits, termed GIuR-A, -B, -C, and -D (or, in an alternative nomenclature, G1uR1, G1uR2, GluR3, and G1uR4). Additional molecular diversity of AMPARs is generated by alternative splicing of the flip-flop module and RNA editing at the Q/R and R/G site. NMDARs are heteromers primarily assembled from NR1 subunits and NR2A, B, C, or D subunits. Various splice variants have been identified for the NR1 subunit, and a new NR3 subunit has been discovered recently. Considering all combinatorial possibilities, the molecular diversity of glutamate-receptor channels is considerable (HOLLMANN, this volume).},
  author       = {Monyer, Hannah and Jonas, Peter M and Rossier, Jean},
  booktitle    = {Ionotropic Glutamate Receptors in the CNS},
  editor       = {Jonas, Peter M and Monyer, Hannah},
  isbn         = {9783642085390},
  pages        = {309 -- 339},
  publisher    = {Springer},
  title        = {{Molecular determinants controlling functional properties of AMPARs and NMDARs in the mammalian CNS}},
  doi          = {10.1007/978-3-662-08022-1_9},
  volume       = {141},
  year         = {1999},
}

@inbook{3457,
  abstract     = {Principal neurons and interneurons are the two main classes of cells in cortical neuronal networks. Principal neurons (granule cells or pyramidal neurons) have transregional axonal projections and release glutamate onto their postsynaptic target cells. In contrast, interneurons have local, but often extensive, axonal arborizations and use γ-aminobutyric acid (GABA) as a transmitter. Although interneurons represent only approximately 10% of the neuronal population, they control the electrical activity of the entire network (FREUND and BUZSÁKI 1996). Interneurons forming inhibitory synapses on the somata or axon initial segments of their postsynaptic target cells are thought to set the threshold of action potential initiation (MILES et al. 1996) and can synchronize the collective activities of large principal neuron ensembles (COBB et al. 1995). In contrast, interneurons establishing inhibitory synapses mainly on dendrites could suppress dendritic Na+ or Ca2+ spikes (BUZSÁKI et al. 1996; MILES et al. 1996) and, thus, regulate plasticity at glutamatergic synapses in the cortex (DAVIES et al.1991).},
  author       = {Geiger, Jörg and Roth, Arnd and Taskin, Birol and Jonas, Peter M},
  booktitle    = {Ionotropic Glutamate Receptors in the CNS},
  editor       = {Monyer, Hannah and Jonas, Peter M},
  isbn         = {9783642085390},
  pages        = {363 -- 398},
  publisher    = {Springer},
  title        = {{Glutamate-mediated synaptic excitation of cortical interneurons}},
  doi          = {10.1007/978-3-662-08022-1_11},
  volume       = {141},
  year         = {1999},
}