[{"date_created":"2023-05-05T12:48:48Z","day":"27","publication_status":"published","author":[{"orcid":"0000-0002-5621-8100","last_name":"Schlögl","id":"45BF87EE-F248-11E8-B48F-1D18A9856A87","first_name":"Alois","full_name":"Schlögl, Alois"},{"last_name":"Kiss","id":"3D3A06F8-F248-11E8-B48F-1D18A9856A87","first_name":"Janos","full_name":"Kiss, Janos"},{"last_name":"Elefante","full_name":"Elefante, Stefano","first_name":"Stefano","id":"490F40CE-F248-11E8-B48F-1D18A9856A87"}],"conference":{"name":"AHPC: Austrian HPC Meeting","start_date":"2019-02-25","end_date":"2019-02-27","location":"Grundlsee, Austria"},"page":"25","file_date_updated":"2023-05-16T07:27:09Z","department":[{"_id":"ScienComp"}],"ddc":["000"],"language":[{"iso":"eng"}],"year":"2019","oa_version":"Published Version","citation":{"chicago":"Schlögl, Alois, Janos Kiss, and Stefano Elefante. “Is Debian Suitable for Running an HPC Cluster?” In <i>AHPC19 - Austrian HPC Meeting 2019 </i>, 25. Institut für Mathematik und wissenschaftliches Rechnen der Universität Graz, 2019.","mla":"Schlögl, Alois, et al. “Is Debian Suitable for Running an HPC Cluster?” <i>AHPC19 - Austrian HPC Meeting 2019 </i>, Institut für Mathematik und wissenschaftliches Rechnen der Universität Graz, 2019, p. 25.","ama":"Schlögl A, Kiss J, Elefante S. Is Debian suitable for running an HPC Cluster? In: <i>AHPC19 - Austrian HPC Meeting 2019 </i>. Institut für Mathematik und wissenschaftliches Rechnen der Universität Graz; 2019:25.","ieee":"A. Schlögl, J. Kiss, and S. Elefante, “Is Debian suitable for running an HPC Cluster?,” in <i>AHPC19 - Austrian HPC Meeting 2019 </i>, Grundlsee, Austria, 2019, p. 25.","short":"A. Schlögl, J. Kiss, S. Elefante, in:, AHPC19 - Austrian HPC Meeting 2019 , Institut für Mathematik und wissenschaftliches Rechnen der Universität Graz, 2019, p. 25.","apa":"Schlögl, A., Kiss, J., &#38; Elefante, S. (2019). Is Debian suitable for running an HPC Cluster? In <i>AHPC19 - Austrian HPC Meeting 2019 </i> (p. 25). Grundlsee, Austria: Institut für Mathematik und wissenschaftliches Rechnen der Universität Graz.","ista":"Schlögl A, Kiss J, Elefante S. 2019. Is Debian suitable for running an HPC Cluster? AHPC19 - Austrian HPC Meeting 2019 . AHPC: Austrian HPC Meeting, 25."},"main_file_link":[{"url":"https://vsc.ac.at/fileadmin/user_upload/vsc/conferences/ahpc19/BOOKLET_AHPC19.pdf","open_access":"1"}],"has_accepted_license":"1","date_published":"2019-02-27T00:00:00Z","type":"conference_abstract","status":"public","month":"02","oa":1,"file":[{"access_level":"open_access","file_name":"2019_AHPC_Schloegl.pdf","success":1,"checksum":"acc8272027faaf30709c51ac5c58ffa4","date_updated":"2023-05-16T07:27:09Z","file_size":1097603,"content_type":"application/pdf","file_id":"12970","date_created":"2023-05-16T07:27:09Z","relation":"main_file","creator":"dernst"}],"user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","article_processing_charge":"No","publisher":"Institut für Mathematik und wissenschaftliches Rechnen der Universität Graz","date_updated":"2023-05-16T07:29:32Z","title":"Is Debian suitable for running an HPC Cluster?","publication":"AHPC19 - Austrian HPC Meeting 2019 ","_id":"12901"},{"title":"Frontiers in microfluidics, a teaching resource review","_id":"7225","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","abstract":[{"lang":"eng","text":"This is a literature teaching resource review for biologically inspired microfluidics courses\r\nor exploring the diverse applications of microfluidics. The structure is around key papers and model\r\norganisms. While courses gradually change over time, a focus remains on understanding how\r\nmicrofluidics has developed as well as what it can and cannot do for researchers. As a primary\r\nstarting point, we cover micro-fluid mechanics principles and microfabrication of devices. A variety\r\nof applications are discussed using model prokaryotic and eukaryotic organisms from the set\r\nof bacteria (Escherichia coli), trypanosomes (Trypanosoma brucei), yeast (Saccharomyces cerevisiae),\r\nslime molds (Physarum polycephalum), worms (Caenorhabditis elegans), flies (Drosophila melangoster),\r\nplants (Arabidopsis thaliana), and mouse immune cells (Mus musculus). Other engineering and\r\nbiochemical methods discussed include biomimetics, organ on a chip, inkjet, droplet microfluidics,\r\nbiotic games, and diagnostics. While we have not yet reached the end-all lab on a chip,\r\nmicrofluidics can still be used effectively for specific applications."}],"pmid":1,"status":"public","intvolume":"         6","citation":{"short":"J. Merrin, Bioengineering 6 (2019).","ieee":"J. Merrin, “Frontiers in microfluidics, a teaching resource review,” <i>Bioengineering</i>, vol. 6, no. 4. MDPI, 2019.","ista":"Merrin J. 2019. Frontiers in microfluidics, a teaching resource review. Bioengineering. 6(4), 109.","apa":"Merrin, J. (2019). Frontiers in microfluidics, a teaching resource review. <i>Bioengineering</i>. MDPI. <a href=\"https://doi.org/10.3390/bioengineering6040109\">https://doi.org/10.3390/bioengineering6040109</a>","chicago":"Merrin, Jack. “Frontiers in Microfluidics, a Teaching Resource Review.” <i>Bioengineering</i>. MDPI, 2019. <a href=\"https://doi.org/10.3390/bioengineering6040109\">https://doi.org/10.3390/bioengineering6040109</a>.","ama":"Merrin J. Frontiers in microfluidics, a teaching resource review. <i>Bioengineering</i>. 2019;6(4). doi:<a href=\"https://doi.org/10.3390/bioengineering6040109\">10.3390/bioengineering6040109</a>","mla":"Merrin, Jack. “Frontiers in Microfluidics, a Teaching Resource Review.” <i>Bioengineering</i>, vol. 6, no. 4, 109, MDPI, 2019, doi:<a href=\"https://doi.org/10.3390/bioengineering6040109\">10.3390/bioengineering6040109</a>."},"has_accepted_license":"1","scopus_import":"1","isi":1,"file_date_updated":"2020-07-14T12:47:54Z","issue":"4","publication_identifier":{"eissn":["23065354"]},"date_updated":"2023-09-06T14:52:49Z","doi":"10.3390/bioengineering6040109","publisher":"MDPI","publication":"Bioengineering","article_processing_charge":"Yes","file":[{"creator":"dernst","relation":"main_file","file_id":"7243","date_created":"2020-01-07T14:49:59Z","content_type":"application/pdf","file_size":2660780,"date_updated":"2020-07-14T12:47:54Z","checksum":"80f1499e2a4caccdf3aa54b137fd99a0","file_name":"2019_Bioengineering_Merrin.pdf","access_level":"open_access"}],"oa":1,"quality_controlled":"1","article_number":"109","month":"12","type":"journal_article","date_published":"2019-12-03T00:00:00Z","volume":6,"oa_version":"Published Version","external_id":{"isi":["000505590000024"],"pmid":["31816954"]},"article_type":"review","language":[{"iso":"eng"}],"year":"2019","ddc":["620"],"department":[{"_id":"NanoFab"}],"tmp":{"legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode","image":"/images/cc_by.png","short":"CC BY (4.0)","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)"},"author":[{"first_name":"Jack","id":"4515C308-F248-11E8-B48F-1D18A9856A87","full_name":"Merrin, Jack","orcid":"0000-0001-5145-4609","last_name":"Merrin"}],"day":"03","publication_status":"published","date_created":"2020-01-05T23:00:45Z","license":"https://creativecommons.org/licenses/by/4.0/"},{"date_published":"2019-01-15T00:00:00Z","project":[{"grant_number":"303564","_id":"25548C20-B435-11E9-9278-68D0E5697425","name":"Microbial Ion Channels for Synthetic Neurobiology","call_identifier":"FP7"},{"name":"Molecular mechanisms of endocytic cargo recognition in plants","call_identifier":"FWF","grant_number":"I03630","_id":"26538374-B435-11E9-9278-68D0E5697425"},{"grant_number":"W1232-B24","_id":"2548AE96-B435-11E9-9278-68D0E5697425","name":"Molecular Drug Targets","call_identifier":"FWF"}],"type":"journal_article","volume":312,"oa_version":"None","external_id":{"pmid":["30496761"],"isi":["000456220900013"]},"article_type":"original","article_processing_charge":"No","publisher":"Elsevier","doi":"10.1016/j.jneumeth.2018.11.018","date_updated":"2023-09-06T15:27:29Z","publication":"Journal of Neuroscience Methods","month":"01","quality_controlled":"1","author":[{"id":"3EEDE19A-F248-11E8-B48F-1D18A9856A87","first_name":"Catherine","full_name":"Mckenzie, Catherine","last_name":"Mckenzie"},{"last_name":"Spanova","id":"44A924DC-F248-11E8-B48F-1D18A9856A87","first_name":"Miroslava","full_name":"Spanova, Miroslava"},{"full_name":"Johnson, Alexander J","id":"46A62C3A-F248-11E8-B48F-1D18A9856A87","first_name":"Alexander J","last_name":"Johnson","orcid":"0000-0002-2739-8843"},{"last_name":"Kainrath","id":"32CFBA64-F248-11E8-B48F-1D18A9856A87","first_name":"Stephanie","full_name":"Kainrath, Stephanie"},{"first_name":"Vanessa","id":"39C5A68A-F248-11E8-B48F-1D18A9856A87","full_name":"Zheden, Vanessa","orcid":"0000-0002-9438-4783","last_name":"Zheden"},{"first_name":"Harald H.","full_name":"Sitte, Harald H.","last_name":"Sitte"},{"last_name":"Janovjak","orcid":"0000-0002-8023-9315","full_name":"Janovjak, Harald L","first_name":"Harald L","id":"33BA6C30-F248-11E8-B48F-1D18A9856A87"}],"page":"114-121","date_created":"2020-01-30T09:12:19Z","acknowledged_ssus":[{"_id":"Bio"},{"_id":"EM-Fac"}],"day":"15","publication_status":"published","year":"2019","language":[{"iso":"eng"}],"department":[{"_id":"HaJa"},{"_id":"Bio"}],"status":"public","ec_funded":1,"intvolume":"       312","citation":{"chicago":"Mckenzie, Catherine, Miroslava Spanova, Alexander J Johnson, Stephanie Kainrath, Vanessa Zheden, Harald H. Sitte, and Harald L Janovjak. “Isolation of Synaptic Vesicles from Genetically Engineered Cultured Neurons.” <i>Journal of Neuroscience Methods</i>. Elsevier, 2019. <a href=\"https://doi.org/10.1016/j.jneumeth.2018.11.018\">https://doi.org/10.1016/j.jneumeth.2018.11.018</a>.","mla":"Mckenzie, Catherine, et al. “Isolation of Synaptic Vesicles from Genetically Engineered Cultured Neurons.” <i>Journal of Neuroscience Methods</i>, vol. 312, Elsevier, 2019, pp. 114–21, doi:<a href=\"https://doi.org/10.1016/j.jneumeth.2018.11.018\">10.1016/j.jneumeth.2018.11.018</a>.","ama":"Mckenzie C, Spanova M, Johnson AJ, et al. Isolation of synaptic vesicles from genetically engineered cultured neurons. <i>Journal of Neuroscience Methods</i>. 2019;312:114-121. doi:<a href=\"https://doi.org/10.1016/j.jneumeth.2018.11.018\">10.1016/j.jneumeth.2018.11.018</a>","short":"C. Mckenzie, M. Spanova, A.J. Johnson, S. Kainrath, V. Zheden, H.H. Sitte, H.L. Janovjak, Journal of Neuroscience Methods 312 (2019) 114–121.","ieee":"C. Mckenzie <i>et al.</i>, “Isolation of synaptic vesicles from genetically engineered cultured neurons,” <i>Journal of Neuroscience Methods</i>, vol. 312. Elsevier, pp. 114–121, 2019.","apa":"Mckenzie, C., Spanova, M., Johnson, A. J., Kainrath, S., Zheden, V., Sitte, H. H., &#38; Janovjak, H. L. (2019). Isolation of synaptic vesicles from genetically engineered cultured neurons. <i>Journal of Neuroscience Methods</i>. Elsevier. <a href=\"https://doi.org/10.1016/j.jneumeth.2018.11.018\">https://doi.org/10.1016/j.jneumeth.2018.11.018</a>","ista":"Mckenzie C, Spanova M, Johnson AJ, Kainrath S, Zheden V, Sitte HH, Janovjak HL. 2019. Isolation of synaptic vesicles from genetically engineered cultured neurons. Journal of Neuroscience Methods. 312, 114–121."},"user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","abstract":[{"lang":"eng","text":"Background\r\nSynaptic vesicles (SVs) are an integral part of the neurotransmission machinery, and isolation of SVs from their host neuron is necessary to reveal their most fundamental biochemical and functional properties in in vitro assays. Isolated SVs from neurons that have been genetically engineered, e.g. to introduce genetically encoded indicators, are not readily available but would permit new insights into SV structure and function. Furthermore, it is unclear if cultured neurons can provide sufficient starting material for SV isolation procedures.\r\n\r\nNew method\r\nHere, we demonstrate an efficient ex vivo procedure to obtain functional SVs from cultured rat cortical neurons after genetic engineering with a lentivirus.\r\n\r\nResults\r\nWe show that ∼108 plated cortical neurons allow isolation of suitable SV amounts for functional analysis and imaging. We found that SVs isolated from cultured neurons have neurotransmitter uptake comparable to that of SVs isolated from intact cortex. Using total internal reflection fluorescence (TIRF) microscopy, we visualized an exogenous SV-targeted marker protein and demonstrated the high efficiency of SV modification.\r\n\r\nComparison with existing methods\r\nObtaining SVs from genetically engineered neurons currently generally requires the availability of transgenic animals, which is constrained by technical (e.g. cost and time) and biological (e.g. developmental defects and lethality) limitations.\r\n\r\nConclusions\r\nThese results demonstrate the modification and isolation of functional SVs using cultured neurons and viral transduction. The ability to readily obtain SVs from genetically engineered neurons will permit linking in situ studies to in vitro experiments in a variety of genetic contexts."}],"title":"Isolation of synaptic vesicles from genetically engineered cultured neurons","_id":"7406","pmid":1,"publication_identifier":{"issn":["0165-0270"]},"scopus_import":"1","isi":1},{"page":"S11-S12","issue":"Supplement 6","author":[{"last_name":"Morandell","id":"4739D480-F248-11E8-B48F-1D18A9856A87","first_name":"Jasmin","full_name":"Morandell, Jasmin"},{"last_name":"Nicolas","full_name":"Nicolas, Armel","first_name":"Armel","id":"2A103192-F248-11E8-B48F-1D18A9856A87"},{"first_name":"Lena A","id":"29A8453C-F248-11E8-B48F-1D18A9856A87","full_name":"Schwarz, Lena A","last_name":"Schwarz"},{"orcid":"0000-0002-7673-7178","last_name":"Novarino","full_name":"Novarino, Gaia","id":"3E57A680-F248-11E8-B48F-1D18A9856A87","first_name":"Gaia"}],"publication_status":"published","day":"13","publication_identifier":{"issn":["0924-977X"]},"date_created":"2020-01-30T10:07:41Z","year":"2019","language":[{"iso":"eng"}],"isi":1,"department":[{"_id":"GaNo"},{"_id":"LifeSc"}],"status":"public","volume":29,"type":"journal_article","date_published":"2019-12-13T00:00:00Z","intvolume":"        29","citation":{"apa":"Morandell, J., Nicolas, A., Schwarz, L. A., &#38; Novarino, G. (2019). S.16.05 Illuminating the role of the e3 ubiquitin ligase cullin3 in brain development and autism. <i>European Neuropsychopharmacology</i>. Elsevier. <a href=\"https://doi.org/10.1016/j.euroneuro.2019.09.040\">https://doi.org/10.1016/j.euroneuro.2019.09.040</a>","ista":"Morandell J, Nicolas A, Schwarz LA, Novarino G. 2019. S.16.05 Illuminating the role of the e3 ubiquitin ligase cullin3 in brain development and autism. European Neuropsychopharmacology. 29(Supplement 6), S11–S12.","ieee":"J. Morandell, A. Nicolas, L. A. Schwarz, and G. Novarino, “S.16.05 Illuminating the role of the e3 ubiquitin ligase cullin3 in brain development and autism,” <i>European Neuropsychopharmacology</i>, vol. 29, no. Supplement 6. Elsevier, pp. S11–S12, 2019.","short":"J. Morandell, A. Nicolas, L.A. Schwarz, G. Novarino, European Neuropsychopharmacology 29 (2019) S11–S12.","mla":"Morandell, Jasmin, et al. “S.16.05 Illuminating the Role of the E3 Ubiquitin Ligase Cullin3 in Brain Development and Autism.” <i>European Neuropsychopharmacology</i>, vol. 29, no. Supplement 6, Elsevier, 2019, pp. S11–12, doi:<a href=\"https://doi.org/10.1016/j.euroneuro.2019.09.040\">10.1016/j.euroneuro.2019.09.040</a>.","ama":"Morandell J, Nicolas A, Schwarz LA, Novarino G. S.16.05 Illuminating the role of the e3 ubiquitin ligase cullin3 in brain development and autism. <i>European Neuropsychopharmacology</i>. 2019;29(Supplement 6):S11-S12. doi:<a href=\"https://doi.org/10.1016/j.euroneuro.2019.09.040\">10.1016/j.euroneuro.2019.09.040</a>","chicago":"Morandell, Jasmin, Armel Nicolas, Lena A Schwarz, and Gaia Novarino. “S.16.05 Illuminating the Role of the E3 Ubiquitin Ligase Cullin3 in Brain Development and Autism.” <i>European Neuropsychopharmacology</i>. Elsevier, 2019. <a href=\"https://doi.org/10.1016/j.euroneuro.2019.09.040\">https://doi.org/10.1016/j.euroneuro.2019.09.040</a>."},"article_type":"original","external_id":{"isi":["000502657500021"]},"oa_version":"None","publication":"European Neuropsychopharmacology","_id":"7415","publisher":"Elsevier","doi":"10.1016/j.euroneuro.2019.09.040","date_updated":"2023-09-07T14:56:17Z","title":"S.16.05 Illuminating the role of the e3 ubiquitin ligase cullin3 in brain development and autism","article_processing_charge":"No","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","quality_controlled":"1","month":"12"},{"file_date_updated":"2021-06-29T14:41:46Z","isi":1,"scopus_import":"1","issue":"3","pmid":1,"_id":"6052","title":"A practical guide to optimization in X10 expansion microscopy","abstract":[{"lang":"eng","text":"Expansion microscopy is a relatively new approach to super-resolution imaging that uses expandable hydrogels to isotropically increase the physical distance between fluorophores in biological samples such as cell cultures or tissue slices. The classic gel recipe results in an expansion factor of ~4×, with a resolution of 60–80 nm. We have recently developed X10 microscopy, which uses a gel that achieves an expansion factor of ~10×, with a resolution of ~25 nm. Here, we provide a step-by-step protocol for X10 expansion microscopy. A typical experiment consists of seven sequential stages: (i) immunostaining, (ii) anchoring, (iii) polymerization, (iv) homogenization, (v) expansion, (vi) imaging, and (vii) validation. The protocol presented here includes recommendations for optimization, pitfalls and their solutions, and detailed guidelines that should increase reproducibility. Although our protocol focuses on X10 expansion microscopy, we detail which of these suggestions are also applicable to classic fourfold expansion microscopy. We exemplify our protocol using primary hippocampal neurons from rats, but our approach can be used with other primary cells or cultured cell lines of interest. This protocol will enable any researcher with basic experience in immunostainings and access to an epifluorescence microscope to perform super-resolution microscopy with X10. The procedure takes 3 d and requires ~5 h of actively handling the sample for labeling and expansion, and another ~3 h for imaging and analysis."}],"user_id":"4359f0d1-fa6c-11eb-b949-802e58b17ae8","has_accepted_license":"1","intvolume":"        14","citation":{"ama":"Truckenbrodt SM, Sommer CM, Rizzoli SO, Danzl JG. A practical guide to optimization in X10 expansion microscopy. <i>Nature Protocols</i>. 2019;14(3):832–863. doi:<a href=\"https://doi.org/10.1038/s41596-018-0117-3\">10.1038/s41596-018-0117-3</a>","mla":"Truckenbrodt, Sven M., et al. “A Practical Guide to Optimization in X10 Expansion Microscopy.” <i>Nature Protocols</i>, vol. 14, no. 3, Nature Publishing Group, 2019, pp. 832–863, doi:<a href=\"https://doi.org/10.1038/s41596-018-0117-3\">10.1038/s41596-018-0117-3</a>.","chicago":"Truckenbrodt, Sven M, Christoph M Sommer, Silvio O Rizzoli, and Johann G Danzl. “A Practical Guide to Optimization in X10 Expansion Microscopy.” <i>Nature Protocols</i>. Nature Publishing Group, 2019. <a href=\"https://doi.org/10.1038/s41596-018-0117-3\">https://doi.org/10.1038/s41596-018-0117-3</a>.","ista":"Truckenbrodt SM, Sommer CM, Rizzoli SO, Danzl JG. 2019. A practical guide to optimization in X10 expansion microscopy. Nature Protocols. 14(3), 832–863.","apa":"Truckenbrodt, S. M., Sommer, C. M., Rizzoli, S. O., &#38; Danzl, J. G. (2019). A practical guide to optimization in X10 expansion microscopy. <i>Nature Protocols</i>. Nature Publishing Group. <a href=\"https://doi.org/10.1038/s41596-018-0117-3\">https://doi.org/10.1038/s41596-018-0117-3</a>","short":"S.M. Truckenbrodt, C.M. Sommer, S.O. Rizzoli, J.G. Danzl, Nature Protocols 14 (2019) 832–863.","ieee":"S. M. Truckenbrodt, C. M. Sommer, S. O. Rizzoli, and J. G. Danzl, “A practical guide to optimization in X10 expansion microscopy,” <i>Nature Protocols</i>, vol. 14, no. 3. Nature Publishing Group, pp. 832–863, 2019."},"ec_funded":1,"status":"public","department":[{"_id":"JoDa"},{"_id":"Bio"}],"year":"2019","language":[{"iso":"eng"}],"ddc":["570"],"publication_status":"published","day":"01","date_created":"2019-02-24T22:59:20Z","page":"832–863","author":[{"last_name":"Truckenbrodt","first_name":"Sven M","id":"45812BD4-F248-11E8-B48F-1D18A9856A87","full_name":"Truckenbrodt, Sven M"},{"first_name":"Christoph M","id":"4DF26D8C-F248-11E8-B48F-1D18A9856A87","full_name":"Sommer, Christoph M","orcid":"0000-0003-1216-9105","last_name":"Sommer"},{"first_name":"Silvio O","full_name":"Rizzoli, Silvio O","last_name":"Rizzoli"},{"last_name":"Danzl","orcid":"0000-0001-8559-3973","full_name":"Danzl, Johann G","id":"42EFD3B6-F248-11E8-B48F-1D18A9856A87","first_name":"Johann G"}],"file":[{"content_type":"application/vnd.openxmlformats-officedocument.wordprocessingml.document","file_id":"9619","date_created":"2021-06-29T14:41:46Z","relation":"main_file","creator":"kschuh","access_level":"open_access","file_name":"181031_Truckenbrodt_ExM_NatProtoc.docx","success":1,"checksum":"7efb9951e7ddf3e3dcc2fb92b859c623","date_updated":"2021-06-29T14:41:46Z","file_size":84478958}],"quality_controlled":"1","oa":1,"month":"03","publication":"Nature Protocols","date_updated":"2023-08-24T14:48:33Z","doi":"10.1038/s41596-018-0117-3","publisher":"Nature Publishing Group","article_processing_charge":"No","article_type":"original","oa_version":"Submitted Version","external_id":{"pmid":["30778205"],"isi":["000459890700008"]},"volume":14,"project":[{"call_identifier":"H2020","name":"ISTplus - Postdoctoral Fellowships","grant_number":"754411","_id":"260C2330-B435-11E9-9278-68D0E5697425"},{"name":"Optical control of synaptic function via adhesion molecules","call_identifier":"FWF","_id":"265CB4D0-B435-11E9-9278-68D0E5697425","grant_number":"I03600"}],"date_published":"2019-03-01T00:00:00Z","type":"journal_article"},{"main_file_link":[{"open_access":"1","url":"https://doi.org/10.1016/j.cell.2019.01.019"}],"intvolume":"       176","citation":{"chicago":"Xia, Peng, Daniel J Gütl, Vanessa Zheden, and Carl-Philipp J Heisenberg. “Lateral Inhibition in Cell Specification Mediated by Mechanical Signals Modulating TAZ Activity.” <i>Cell</i>. Elsevier, 2019. <a href=\"https://doi.org/10.1016/j.cell.2019.01.019\">https://doi.org/10.1016/j.cell.2019.01.019</a>.","mla":"Xia, Peng, et al. “Lateral Inhibition in Cell Specification Mediated by Mechanical Signals Modulating TAZ Activity.” <i>Cell</i>, vol. 176, no. 6, Elsevier, 2019, p. 1379–1392.e14, doi:<a href=\"https://doi.org/10.1016/j.cell.2019.01.019\">10.1016/j.cell.2019.01.019</a>.","ama":"Xia P, Gütl DJ, Zheden V, Heisenberg C-PJ. Lateral inhibition in cell specification mediated by mechanical signals modulating TAZ activity. <i>Cell</i>. 2019;176(6):1379-1392.e14. doi:<a href=\"https://doi.org/10.1016/j.cell.2019.01.019\">10.1016/j.cell.2019.01.019</a>","short":"P. Xia, D.J. Gütl, V. Zheden, C.-P.J. Heisenberg, Cell 176 (2019) 1379–1392.e14.","ieee":"P. Xia, D. J. Gütl, V. Zheden, and C.-P. J. Heisenberg, “Lateral inhibition in cell specification mediated by mechanical signals modulating TAZ activity,” <i>Cell</i>, vol. 176, no. 6. Elsevier, p. 1379–1392.e14, 2019.","apa":"Xia, P., Gütl, D. J., Zheden, V., &#38; Heisenberg, C.-P. J. (2019). Lateral inhibition in cell specification mediated by mechanical signals modulating TAZ activity. <i>Cell</i>. Elsevier. <a href=\"https://doi.org/10.1016/j.cell.2019.01.019\">https://doi.org/10.1016/j.cell.2019.01.019</a>","ista":"Xia P, Gütl DJ, Zheden V, Heisenberg C-PJ. 2019. Lateral inhibition in cell specification mediated by mechanical signals modulating TAZ activity. Cell. 176(6), 1379–1392.e14."},"ec_funded":1,"status":"public","acknowledgement":"We thank Roland Dosch, Makoto Furutani-Seiki, Brian Link, Mary Mullins, and Masazumi Tada for providing transgenic and/or mutant zebrafish lines; Alexandra Schauer, Shayan Shami-Pour, and the rest of the Heisenberg lab for technical assistance and feedback on the manuscript; and the Bioimaging, Electron Microscopy, and Zebrafish facilities of IST Austria for continuous support. This work was supported by an ERC advanced grant ( MECSPEC to C.-P.H.).","pmid":1,"_id":"6087","title":"Lateral inhibition in cell specification mediated by mechanical signals modulating TAZ activity","abstract":[{"lang":"eng","text":"Cell fate specification by lateral inhibition typically involves contact signaling through the Delta-Notch signaling pathway. However, whether this is the only signaling mode mediating lateral inhibition remains unclear. Here we show that in zebrafish oogenesis, a group of cells within the granulosa cell layer at the oocyte animal pole acquire elevated levels of the transcriptional coactivator TAZ in their nuclei. One of these cells, the future micropyle precursor cell (MPC), accumulates increasingly high levels of nuclear TAZ and grows faster than its surrounding cells, mechanically compressing those cells, which ultimately lose TAZ from their nuclei. Strikingly, relieving neighbor-cell compression by MPC ablation or aspiration restores nuclear TAZ accumulation in neighboring cells, eventually leading to MPC re-specification from these cells. Conversely, MPC specification is defective in taz−/− follicles. These findings uncover a novel mode of lateral inhibition in cell fate specification based on mechanical signals controlling TAZ activity."}],"user_id":"4359f0d1-fa6c-11eb-b949-802e58b17ae8","issue":"6","isi":1,"scopus_import":"1","oa_version":"Published Version","article_type":"original","external_id":{"pmid":["30773315"],"isi":["000460509600013"]},"volume":176,"project":[{"name":"Interaction and feedback between cell mechanics and fate specification in vertebrate gastrulation","call_identifier":"H2020","_id":"260F1432-B435-11E9-9278-68D0E5697425","grant_number":"742573"}],"date_published":"2019-03-07T00:00:00Z","type":"journal_article","oa":1,"quality_controlled":"1","month":"03","publication":"Cell","date_updated":"2023-08-25T08:02:23Z","publisher":"Elsevier","doi":"10.1016/j.cell.2019.01.019","article_processing_charge":"No","publication_status":"published","day":"07","acknowledged_ssus":[{"_id":"Bio"},{"_id":"EM-Fac"},{"_id":"LifeSc"}],"date_created":"2019-03-10T22:59:19Z","page":"1379-1392.e14","author":[{"full_name":"Xia, Peng","first_name":"Peng","id":"4AB6C7D0-F248-11E8-B48F-1D18A9856A87","last_name":"Xia","orcid":"0000-0002-5419-7756"},{"id":"381929CE-F248-11E8-B48F-1D18A9856A87","first_name":"Daniel J","full_name":"Gütl, Daniel J","last_name":"Gütl"},{"full_name":"Zheden, Vanessa","id":"39C5A68A-F248-11E8-B48F-1D18A9856A87","first_name":"Vanessa","orcid":"0000-0002-9438-4783","last_name":"Zheden"},{"orcid":"0000-0002-0912-4566","last_name":"Heisenberg","first_name":"Carl-Philipp J","id":"39427864-F248-11E8-B48F-1D18A9856A87","full_name":"Heisenberg, Carl-Philipp J"}],"department":[{"_id":"CaHe"},{"_id":"EM-Fac"}],"language":[{"iso":"eng"}],"year":"2019","related_material":{"link":[{"relation":"press_release","url":"https://ist.ac.at/en/news/in-zebrafish-eggs-most-rapidly-growing-cell-inhibits-its-neighbours-through-mechanical-signals/","description":"News on IST Homepage"}]}},{"file_date_updated":"2020-07-14T12:47:19Z","scopus_import":"1","isi":1,"issue":"2","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","abstract":[{"text":"Blebs are cellular protrusions observed in migrating cells and in cells undergoing spreading, cytokinesis, and apoptosis. Here we investigate the flow of cytoplasm during bleb formation and the concurrent changes in cell volume using zebrafish primordial germ cells (PGCs) as an in vivo model. We show that bleb inflation occurs concomitantly with cytoplasmic inflow into it and that during this process the total cell volume does not change. We thus show that bleb formation in primordial germ cells results primarily from redistribution of material within the cell rather than being driven by flow of water from an external source.","lang":"eng"}],"title":"Fluid dynamics during bleb formation in migrating cells in vivo","_id":"6093","citation":{"short":"M. Goudarzi, A. Boquet-Pujadas, J.C. Olivo-Marin, E. Raz, PLOS ONE 14 (2019).","ieee":"M. Goudarzi, A. Boquet-Pujadas, J. C. Olivo-Marin, and E. Raz, “Fluid dynamics during bleb formation in migrating cells in vivo,” <i>PLOS ONE</i>, vol. 14, no. 2. Public Library of Science, 2019.","ista":"Goudarzi M, Boquet-Pujadas A, Olivo-Marin JC, Raz E. 2019. Fluid dynamics during bleb formation in migrating cells in vivo. PLOS ONE. 14(2), e0212699.","apa":"Goudarzi, M., Boquet-Pujadas, A., Olivo-Marin, J. C., &#38; Raz, E. (2019). Fluid dynamics during bleb formation in migrating cells in vivo. <i>PLOS ONE</i>. Public Library of Science. <a href=\"https://doi.org/10.1371/journal.pone.0212699\">https://doi.org/10.1371/journal.pone.0212699</a>","chicago":"Goudarzi, Mohammad, Aleix Boquet-Pujadas, Jean Christophe Olivo-Marin, and Erez Raz. “Fluid Dynamics during Bleb Formation in Migrating Cells in Vivo.” <i>PLOS ONE</i>. Public Library of Science, 2019. <a href=\"https://doi.org/10.1371/journal.pone.0212699\">https://doi.org/10.1371/journal.pone.0212699</a>.","ama":"Goudarzi M, Boquet-Pujadas A, Olivo-Marin JC, Raz E. Fluid dynamics during bleb formation in migrating cells in vivo. <i>PLOS ONE</i>. 2019;14(2). doi:<a href=\"https://doi.org/10.1371/journal.pone.0212699\">10.1371/journal.pone.0212699</a>","mla":"Goudarzi, Mohammad, et al. “Fluid Dynamics during Bleb Formation in Migrating Cells in Vivo.” <i>PLOS ONE</i>, vol. 14, no. 2, e0212699, Public Library of Science, 2019, doi:<a href=\"https://doi.org/10.1371/journal.pone.0212699\">10.1371/journal.pone.0212699</a>."},"intvolume":"        14","has_accepted_license":"1","status":"public","tmp":{"legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode","image":"/images/cc_by.png","short":"CC BY (4.0)","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)"},"department":[{"_id":"Bio"}],"ddc":["570"],"year":"2019","language":[{"iso":"eng"}],"date_created":"2019-03-10T22:59:21Z","day":"26","publication_status":"published","author":[{"id":"3384113A-F248-11E8-B48F-1D18A9856A87","first_name":"Mohammad","full_name":"Goudarzi, Mohammad","last_name":"Goudarzi"},{"last_name":"Boquet-Pujadas","first_name":"Aleix","full_name":"Boquet-Pujadas, Aleix"},{"first_name":"Jean Christophe","full_name":"Olivo-Marin, Jean Christophe","last_name":"Olivo-Marin"},{"last_name":"Raz","first_name":"Erez","full_name":"Raz, Erez"}],"month":"02","oa":1,"quality_controlled":"1","file":[{"date_updated":"2020-07-14T12:47:19Z","file_size":2967731,"access_level":"open_access","file_name":"2019_PLoSOne_Goudarzi.pdf","checksum":"b885de050ed4bb3c86f706487a47197f","creator":"dernst","relation":"main_file","content_type":"application/pdf","date_created":"2019-03-11T16:09:23Z","file_id":"6096"}],"article_number":"e0212699","article_processing_charge":"No","date_updated":"2023-09-19T14:46:47Z","doi":"10.1371/journal.pone.0212699","publisher":"Public Library of Science","publication":"PLOS ONE","oa_version":"Published Version","external_id":{"isi":["000459712100022"]},"type":"journal_article","date_published":"2019-02-26T00:00:00Z","volume":14},{"article_type":"letter_note","external_id":{"pmid":["30944468"],"isi":["000465594200050"]},"oa_version":"Submitted Version","volume":568,"project":[{"name":"Cytoskeletal force generation and force transduction of migrating leukocytes (EU)","call_identifier":"FP7","_id":"25A603A2-B435-11E9-9278-68D0E5697425","grant_number":"281556"},{"name":"Cellular navigation along spatial gradients","call_identifier":"H2020","_id":"25FE9508-B435-11E9-9278-68D0E5697425","grant_number":"724373"},{"call_identifier":"FWF","name":"Nano-Analytics of Cellular Systems","_id":"265FAEBA-B435-11E9-9278-68D0E5697425","grant_number":"W01250-B20"},{"call_identifier":"FP7","name":"International IST Postdoc Fellowship Programme","_id":"25681D80-B435-11E9-9278-68D0E5697425","grant_number":"291734"},{"name":"Molecular and system level view of immune cell migration","grant_number":"ALTF 1396-2014","_id":"25A48D24-B435-11E9-9278-68D0E5697425"}],"date_published":"2019-04-25T00:00:00Z","type":"journal_article","month":"04","oa":1,"quality_controlled":"1","article_processing_charge":"No","publication":"Nature","date_updated":"2024-03-25T23:30:22Z","doi":"10.1038/s41586-019-1087-5","publisher":"Springer Nature","acknowledged_ssus":[{"_id":"SSU"}],"date_created":"2019-04-17T06:52:28Z","publication_status":"published","day":"25","author":[{"orcid":"0000-0003-2856-3369","last_name":"Renkawitz","id":"3F0587C8-F248-11E8-B48F-1D18A9856A87","first_name":"Jörg","full_name":"Renkawitz, Jörg"},{"orcid":"0000-0002-2187-6656","last_name":"Kopf","id":"31DAC7B6-F248-11E8-B48F-1D18A9856A87","first_name":"Aglaja","full_name":"Kopf, Aglaja"},{"full_name":"Stopp, Julian A","id":"489E3F00-F248-11E8-B48F-1D18A9856A87","first_name":"Julian A","last_name":"Stopp"},{"last_name":"de Vries","full_name":"de Vries, Ingrid","id":"4C7D837E-F248-11E8-B48F-1D18A9856A87","first_name":"Ingrid"},{"last_name":"Driscoll","full_name":"Driscoll, Meghan K.","first_name":"Meghan K."},{"orcid":"0000-0001-5145-4609","last_name":"Merrin","first_name":"Jack","id":"4515C308-F248-11E8-B48F-1D18A9856A87","full_name":"Merrin, Jack"},{"last_name":"Hauschild","orcid":"0000-0001-9843-3522","full_name":"Hauschild, Robert","first_name":"Robert","id":"4E01D6B4-F248-11E8-B48F-1D18A9856A87"},{"full_name":"Welf, Erik S.","first_name":"Erik S.","last_name":"Welf"},{"last_name":"Danuser","first_name":"Gaudenz","full_name":"Danuser, Gaudenz"},{"last_name":"Fiolka","full_name":"Fiolka, Reto","first_name":"Reto"},{"full_name":"Sixt, Michael K","first_name":"Michael K","id":"41E9FBEA-F248-11E8-B48F-1D18A9856A87","orcid":"0000-0002-6620-9179","last_name":"Sixt"}],"page":"546-550","department":[{"_id":"MiSi"},{"_id":"NanoFab"},{"_id":"Bio"}],"year":"2019","language":[{"iso":"eng"}],"related_material":{"record":[{"id":"14697","status":"public","relation":"dissertation_contains"},{"id":"6891","relation":"dissertation_contains","status":"public"}],"link":[{"url":"https://ist.ac.at/en/news/leukocytes-use-their-nucleus-as-a-ruler-to-choose-path-of-least-resistance/","relation":"press_release","description":"News on IST Homepage"}]},"main_file_link":[{"open_access":"1","url":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7217284/"}],"intvolume":"       568","citation":{"ista":"Renkawitz J, Kopf A, Stopp JA, de Vries I, Driscoll MK, Merrin J, Hauschild R, Welf ES, Danuser G, Fiolka R, Sixt MK. 2019. Nuclear positioning facilitates amoeboid migration along the path of least resistance. Nature. 568, 546–550.","apa":"Renkawitz, J., Kopf, A., Stopp, J. A., de Vries, I., Driscoll, M. K., Merrin, J., … Sixt, M. K. (2019). Nuclear positioning facilitates amoeboid migration along the path of least resistance. <i>Nature</i>. Springer Nature. <a href=\"https://doi.org/10.1038/s41586-019-1087-5\">https://doi.org/10.1038/s41586-019-1087-5</a>","ieee":"J. Renkawitz <i>et al.</i>, “Nuclear positioning facilitates amoeboid migration along the path of least resistance,” <i>Nature</i>, vol. 568. Springer Nature, pp. 546–550, 2019.","short":"J. Renkawitz, A. Kopf, J.A. Stopp, I. de Vries, M.K. Driscoll, J. Merrin, R. Hauschild, E.S. Welf, G. Danuser, R. Fiolka, M.K. Sixt, Nature 568 (2019) 546–550.","ama":"Renkawitz J, Kopf A, Stopp JA, et al. Nuclear positioning facilitates amoeboid migration along the path of least resistance. <i>Nature</i>. 2019;568:546-550. doi:<a href=\"https://doi.org/10.1038/s41586-019-1087-5\">10.1038/s41586-019-1087-5</a>","mla":"Renkawitz, Jörg, et al. “Nuclear Positioning Facilitates Amoeboid Migration along the Path of Least Resistance.” <i>Nature</i>, vol. 568, Springer Nature, 2019, pp. 546–50, doi:<a href=\"https://doi.org/10.1038/s41586-019-1087-5\">10.1038/s41586-019-1087-5</a>.","chicago":"Renkawitz, Jörg, Aglaja Kopf, Julian A Stopp, Ingrid de Vries, Meghan K. Driscoll, Jack Merrin, Robert Hauschild, et al. “Nuclear Positioning Facilitates Amoeboid Migration along the Path of Least Resistance.” <i>Nature</i>. Springer Nature, 2019. <a href=\"https://doi.org/10.1038/s41586-019-1087-5\">https://doi.org/10.1038/s41586-019-1087-5</a>."},"ec_funded":1,"status":"public","pmid":1,"abstract":[{"text":"During metazoan development, immune surveillance and cancer dissemination, cells migrate in complex three-dimensional microenvironments1,2,3. These spaces are crowded by cells and extracellular matrix, generating mazes with differently sized gaps that are typically smaller than the diameter of the migrating cell4,5. Most mesenchymal and epithelial cells and some—but not all—cancer cells actively generate their migratory path using pericellular tissue proteolysis6. By contrast, amoeboid cells such as leukocytes use non-destructive strategies of locomotion7, raising the question how these extremely fast cells navigate through dense tissues. Here we reveal that leukocytes sample their immediate vicinity for large pore sizes, and are thereby able to choose the path of least resistance. This allows them to circumnavigate local obstacles while effectively following global directional cues such as chemotactic gradients. Pore-size discrimination is facilitated by frontward positioning of the nucleus, which enables the cells to use their bulkiest compartment as a mechanical gauge. Once the nucleus and the closely associated microtubule organizing centre pass the largest pore, cytoplasmic protrusions still lingering in smaller pores are retracted. These retractions are coordinated by dynamic microtubules; when microtubules are disrupted, migrating cells lose coherence and frequently fragment into migratory cytoplasmic pieces. As nuclear positioning in front of the microtubule organizing centre is a typical feature of amoeboid migration, our findings link the fundamental organization of cellular polarity to the strategy of locomotion.","lang":"eng"}],"user_id":"4359f0d1-fa6c-11eb-b949-802e58b17ae8","_id":"6328","title":"Nuclear positioning facilitates amoeboid migration along the path of least resistance","isi":1,"scopus_import":"1"},{"issue":"1","file_date_updated":"2020-07-14T12:47:34Z","scopus_import":"1","isi":1,"intvolume":"         9","citation":{"ista":"Nguyen CH, Glüxam T, Schlerka A, Bauer K, Grandits AM, Hackl H, Dovey O, Zöchbauer-Müller S, Cooper JL, Vassiliou GS, Stoiber D, Wieser R, Heller G. 2019. SOCS2 is part of a highly prognostic 4-gene signature in AML and promotes disease aggressiveness. Scientific Reports. 9(1), 9139.","apa":"Nguyen, C. H., Glüxam, T., Schlerka, A., Bauer, K., Grandits, A. M., Hackl, H., … Heller, G. (2019). SOCS2 is part of a highly prognostic 4-gene signature in AML and promotes disease aggressiveness. <i>Scientific Reports</i>. Nature Publishing Group. <a href=\"https://doi.org/10.1038/s41598-019-45579-0\">https://doi.org/10.1038/s41598-019-45579-0</a>","short":"C.H. Nguyen, T. Glüxam, A. Schlerka, K. Bauer, A.M. Grandits, H. Hackl, O. Dovey, S. Zöchbauer-Müller, J.L. Cooper, G.S. Vassiliou, D. Stoiber, R. Wieser, G. Heller, Scientific Reports 9 (2019).","ieee":"C. H. Nguyen <i>et al.</i>, “SOCS2 is part of a highly prognostic 4-gene signature in AML and promotes disease aggressiveness,” <i>Scientific Reports</i>, vol. 9, no. 1. Nature Publishing Group, 2019.","ama":"Nguyen CH, Glüxam T, Schlerka A, et al. SOCS2 is part of a highly prognostic 4-gene signature in AML and promotes disease aggressiveness. <i>Scientific Reports</i>. 2019;9(1). doi:<a href=\"https://doi.org/10.1038/s41598-019-45579-0\">10.1038/s41598-019-45579-0</a>","mla":"Nguyen, Chi Huu, et al. “SOCS2 Is Part of a Highly Prognostic 4-Gene Signature in AML and Promotes Disease Aggressiveness.” <i>Scientific Reports</i>, vol. 9, no. 1, 9139, Nature Publishing Group, 2019, doi:<a href=\"https://doi.org/10.1038/s41598-019-45579-0\">10.1038/s41598-019-45579-0</a>.","chicago":"Nguyen, Chi Huu, Tobias Glüxam, Angela Schlerka, Katharina Bauer, Alexander M. Grandits, Hubert Hackl, Oliver Dovey, et al. “SOCS2 Is Part of a Highly Prognostic 4-Gene Signature in AML and Promotes Disease Aggressiveness.” <i>Scientific Reports</i>. Nature Publishing Group, 2019. <a href=\"https://doi.org/10.1038/s41598-019-45579-0\">https://doi.org/10.1038/s41598-019-45579-0</a>."},"has_accepted_license":"1","status":"public","user_id":"4359f0d1-fa6c-11eb-b949-802e58b17ae8","abstract":[{"lang":"eng","text":"Acute myeloid leukemia (AML) is a heterogeneous disease with respect to its genetic and molecular basis and to patients´ outcome. Clinical, cytogenetic, and mutational data are used to classify patients into risk groups with different survival, however, within-group heterogeneity is still an issue. Here, we used a robust likelihood-based survival modeling approach and publicly available gene expression data to identify a minimal number of genes whose combined expression values were prognostic of overall survival. The resulting gene expression signature (4-GES) consisted of 4 genes (SOCS2, IL2RA, NPDC1, PHGDH), predicted patient survival as an independent prognostic parameter in several cohorts of AML patients (total, 1272 patients), and further refined prognostication based on the European Leukemia Net classification. An oncogenic role of the top scoring gene in this signature, SOCS2, was investigated using MLL-AF9 and Flt3-ITD/NPM1c driven mouse models of AML. SOCS2 promoted leukemogenesis as well as the abundance, quiescence, and activity of AML stem cells. Overall, the 4-GES represents a highly discriminating prognostic parameter in AML, whose clinical applicability is greatly enhanced by its small number of genes. The newly established role of SOCS2 in leukemia aggressiveness and stemness raises the possibility that the signature might even be exploitable therapeutically."}],"title":"SOCS2 is part of a highly prognostic 4-gene signature in AML and promotes disease aggressiveness","_id":"6607","date_created":"2019-07-07T21:59:19Z","day":"24","publication_status":"published","author":[{"first_name":"Chi Huu","full_name":"Nguyen, Chi Huu","last_name":"Nguyen"},{"first_name":"Tobias","full_name":"Glüxam, Tobias","last_name":"Glüxam"},{"last_name":"Schlerka","first_name":"Angela","full_name":"Schlerka, Angela"},{"last_name":"Bauer","full_name":"Bauer, Katharina","id":"2ED6B14C-F248-11E8-B48F-1D18A9856A87","first_name":"Katharina"},{"last_name":"Grandits","full_name":"Grandits, Alexander M.","first_name":"Alexander M."},{"last_name":"Hackl","first_name":"Hubert","full_name":"Hackl, Hubert"},{"first_name":"Oliver","full_name":"Dovey, Oliver","last_name":"Dovey"},{"last_name":"Zöchbauer-Müller","first_name":"Sabine","full_name":"Zöchbauer-Müller, Sabine"},{"last_name":"Cooper","full_name":"Cooper, Jonathan L.","first_name":"Jonathan L."},{"first_name":"George S.","full_name":"Vassiliou, George S.","last_name":"Vassiliou"},{"first_name":"Dagmar","full_name":"Stoiber, Dagmar","last_name":"Stoiber"},{"last_name":"Wieser","full_name":"Wieser, Rotraud","first_name":"Rotraud"},{"first_name":"Gerwin","full_name":"Heller, Gerwin","last_name":"Heller"}],"department":[{"_id":"PreCl"}],"tmp":{"legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode","image":"/images/cc_by.png","short":"CC BY (4.0)","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)"},"language":[{"iso":"eng"}],"year":"2019","ddc":["576"],"external_id":{"isi":["000472597400042"]},"oa_version":"Published Version","date_published":"2019-06-24T00:00:00Z","type":"journal_article","volume":9,"month":"06","file":[{"creator":"kschuh","relation":"main_file","content_type":"application/pdf","date_created":"2019-07-08T15:15:28Z","file_id":"6623","date_updated":"2020-07-14T12:47:34Z","file_size":2017352,"access_level":"open_access","file_name":"nature_2019_Nguyen.pdf","checksum":"3283522fffadf4b5fc8c7adfe3ba4564"}],"oa":1,"quality_controlled":"1","article_number":"9139","article_processing_charge":"No","date_updated":"2023-08-28T12:26:51Z","doi":"10.1038/s41598-019-45579-0","publisher":"Nature Publishing Group","publication":"Scientific Reports"},{"status":"public","has_accepted_license":"1","intvolume":"        72","citation":{"mla":"Danowski, Patrick. “An Austrian Proposal for the Classification of Open Access Tuples (COAT) - Distinguish Different Open Access Types beyond Colors.” <i>Mitteilungen Der Vereinigung Österreichischer Bibliothekarinnen Und Bibliothekare</i>, vol. 72, no. 1, Vereinigung Österreichischer Bibliothekarinnen und Bibliothekare, 2019, pp. 59–65, doi:<a href=\"https://doi.org/10.31263/voebm.v72i1.2276\">10.31263/voebm.v72i1.2276</a>.","ama":"Danowski P. An Austrian proposal for the classification of Open Access Tuples (COAT) - distinguish different open access types beyond colors. <i>Mitteilungen der Vereinigung Österreichischer Bibliothekarinnen und Bibliothekare</i>. 2019;72(1):59-65. doi:<a href=\"https://doi.org/10.31263/voebm.v72i1.2276\">10.31263/voebm.v72i1.2276</a>","chicago":"Danowski, Patrick. “An Austrian Proposal for the Classification of Open Access Tuples (COAT) - Distinguish Different Open Access Types beyond Colors.” <i>Mitteilungen Der Vereinigung Österreichischer Bibliothekarinnen Und Bibliothekare</i>. Vereinigung Österreichischer Bibliothekarinnen und Bibliothekare, 2019. <a href=\"https://doi.org/10.31263/voebm.v72i1.2276\">https://doi.org/10.31263/voebm.v72i1.2276</a>.","apa":"Danowski, P. (2019). An Austrian proposal for the classification of Open Access Tuples (COAT) - distinguish different open access types beyond colors. <i>Mitteilungen Der Vereinigung Österreichischer Bibliothekarinnen Und Bibliothekare</i>. Vereinigung Österreichischer Bibliothekarinnen und Bibliothekare. <a href=\"https://doi.org/10.31263/voebm.v72i1.2276\">https://doi.org/10.31263/voebm.v72i1.2276</a>","ista":"Danowski P. 2019. An Austrian proposal for the classification of Open Access Tuples (COAT) - distinguish different open access types beyond colors. Mitteilungen der Vereinigung Österreichischer Bibliothekarinnen und Bibliothekare. 72(1), 59–65.","ieee":"P. Danowski, “An Austrian proposal for the classification of Open Access Tuples (COAT) - distinguish different open access types beyond colors,” <i>Mitteilungen der Vereinigung Österreichischer Bibliothekarinnen und Bibliothekare</i>, vol. 72, no. 1. Vereinigung Österreichischer Bibliothekarinnen und Bibliothekare, pp. 59–65, 2019.","short":"P. Danowski, Mitteilungen Der Vereinigung Österreichischer Bibliothekarinnen Und Bibliothekare 72 (2019) 59–65."},"abstract":[{"text":"In this article a model is described how Open Access definitions can be formed on the basis of objective criteria. The common Open Access definitions such as \"gold\" and \"green\" are not exactly defined. This becomes a problem as soon as one begins to measure Open Access, for example if the development of the Open Access share should be monitored. This was discussed in the working group on Open Access Monitoring  of  the  AT2OA  project  and  the  present  model  was  developed, which is based on 5 critics with 4 characteristics: location, licence, version, embargo and conditions of the Open Access publication are taken into account. In the meantime, the model has also been tested in practice using R scripts, and the initial results are quite promising.","lang":"eng"}],"user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","_id":"6657","title":"An Austrian proposal for the classification of Open Access Tuples (COAT) - distinguish different open access types beyond colors","issue":"1","publication_identifier":{"eissn":["1022-2588"]},"scopus_import":"1","file_date_updated":"2020-07-14T12:47:35Z","volume":72,"date_published":"2019-05-17T00:00:00Z","type":"journal_article","article_type":"original","oa_version":"Published Version","article_processing_charge":"No","publication":"Mitteilungen der Vereinigung Österreichischer Bibliothekarinnen und Bibliothekare","doi":"10.31263/voebm.v72i1.2276","publisher":"Vereinigung Österreichischer Bibliothekarinnen und Bibliothekare","date_updated":"2023-10-17T11:33:58Z","month":"05","quality_controlled":"1","oa":1,"file":[{"file_id":"6661","date_created":"2019-07-22T08:45:03Z","content_type":"application/pdf","creator":"apreinsp","relation":"main_file","checksum":"c0d2695d6d0d34e62ba06fb3f0ebaaed","access_level":"open_access","file_name":"2019_MitteilungenDerVOEB_Danowski.pdf","file_size":468558,"date_updated":"2020-07-14T12:47:35Z"}],"author":[{"orcid":"0000-0002-6026-4409","last_name":"Danowski","full_name":"Danowski, Patrick","id":"2EBD1598-F248-11E8-B48F-1D18A9856A87","first_name":"Patrick"}],"page":"59-65","date_created":"2019-07-21T21:59:15Z","publication_status":"published","day":"17","language":[{"iso":"eng"}],"year":"2019","ddc":["020"],"related_material":{"record":[{"id":"5686","relation":"earlier_version","status":"public"}]},"department":[{"_id":"E-Lib"}],"tmp":{"legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode","image":"/images/cc_by.png","short":"CC BY (4.0)","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)"}},{"_id":"6819","title":"Glyphosate does not substitute for glycine in proteins of actively dividing mammalian cells","abstract":[{"text":"Glyphosate (N-phosphonomethyl glycine) and its commercial herbicide formulations have been shown to exert toxicity via various mechanisms. It has been asserted that glyphosate substitutes for glycine in polypeptide chains leading to protein misfolding and toxicity. However, as no direct evidence exists for glycine to glyphosate substitution in proteins, including in mammalian organisms, we tested this claim by conducting a proteomics analysis of MDA-MB-231 human breast cancer cells grown in the presence of 100 mg/L glyphosate for 6 days. Protein extracts from three treated and three untreated cell cultures were analysed as one TMT-6plex labelled sample, to highlight a specific pattern (+/+/+/−/−/−) of reporter intensities for peptides bearing true glyphosate treatment induced-post translational modifications as well as allowing an investigation of the total proteome.","lang":"eng"}],"user_id":"3E5EF7F0-F248-11E8-B48F-1D18A9856A87","pmid":1,"status":"public","has_accepted_license":"1","intvolume":"        12","citation":{"ieee":"M. N. Antoniou, A. Nicolas, R. Mesnage, M. Biserni, F. V. Rao, and C. V. Martin, “Glyphosate does not substitute for glycine in proteins of actively dividing mammalian cells,” <i>BMC Research Notes</i>, vol. 12. BioMed Central, 2019.","short":"M.N. Antoniou, A. Nicolas, R. Mesnage, M. Biserni, F.V. Rao, C.V. Martin, BMC Research Notes 12 (2019).","apa":"Antoniou, M. N., Nicolas, A., Mesnage, R., Biserni, M., Rao, F. V., &#38; Martin, C. V. (2019). Glyphosate does not substitute for glycine in proteins of actively dividing mammalian cells. <i>BMC Research Notes</i>. BioMed Central. <a href=\"https://doi.org/10.1186/s13104-019-4534-3\">https://doi.org/10.1186/s13104-019-4534-3</a>","ista":"Antoniou MN, Nicolas A, Mesnage R, Biserni M, Rao FV, Martin CV. 2019. Glyphosate does not substitute for glycine in proteins of actively dividing mammalian cells. BMC Research Notes. 12, 494.","chicago":"Antoniou, Michael N., Armel Nicolas, Robin Mesnage, Martina Biserni, Francesco V. Rao, and Cristina Vazquez Martin. “Glyphosate Does Not Substitute for Glycine in Proteins of Actively Dividing Mammalian Cells.” <i>BMC Research Notes</i>. BioMed Central, 2019. <a href=\"https://doi.org/10.1186/s13104-019-4534-3\">https://doi.org/10.1186/s13104-019-4534-3</a>.","mla":"Antoniou, Michael N., et al. “Glyphosate Does Not Substitute for Glycine in Proteins of Actively Dividing Mammalian Cells.” <i>BMC Research Notes</i>, vol. 12, 494, BioMed Central, 2019, doi:<a href=\"https://doi.org/10.1186/s13104-019-4534-3\">10.1186/s13104-019-4534-3</a>.","ama":"Antoniou MN, Nicolas A, Mesnage R, Biserni M, Rao FV, Martin CV. Glyphosate does not substitute for glycine in proteins of actively dividing mammalian cells. <i>BMC Research Notes</i>. 2019;12. doi:<a href=\"https://doi.org/10.1186/s13104-019-4534-3\">10.1186/s13104-019-4534-3</a>"},"scopus_import":1,"file_date_updated":"2020-07-14T12:47:40Z","publication_identifier":{"eissn":["1756-0500"]},"publication":"BMC Research Notes","publisher":"BioMed Central","date_updated":"2023-02-23T14:08:14Z","doi":"10.1186/s13104-019-4534-3","article_processing_charge":"No","article_number":"494","quality_controlled":"1","file":[{"checksum":"4a2bb7994b7f2c432bf44f5127ea3102","file_name":"2019_BMC_Antoniou.pdf","access_level":"open_access","file_size":1177482,"date_updated":"2020-07-14T12:47:40Z","file_id":"6829","date_created":"2019-08-23T11:10:35Z","content_type":"application/pdf","creator":"dernst","relation":"main_file"}],"oa":1,"month":"08","volume":12,"date_published":"2019-08-08T00:00:00Z","type":"journal_article","oa_version":"Published Version","external_id":{"pmid":["31395095"]},"language":[{"iso":"eng"}],"year":"2019","ddc":["570"],"related_material":{"record":[{"id":"9784","status":"public","relation":"research_data"}]},"tmp":{"legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode","image":"/images/cc_by.png","short":"CC BY (4.0)","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)"},"department":[{"_id":"LifeSc"}],"author":[{"last_name":"Antoniou","first_name":"Michael N.","full_name":"Antoniou, Michael N."},{"last_name":"Nicolas","first_name":"Armel","id":"2A103192-F248-11E8-B48F-1D18A9856A87","full_name":"Nicolas, Armel"},{"last_name":"Mesnage","first_name":"Robin","full_name":"Mesnage, Robin"},{"last_name":"Biserni","first_name":"Martina","full_name":"Biserni, Martina"},{"full_name":"Rao, Francesco V.","first_name":"Francesco V.","last_name":"Rao"},{"full_name":"Martin, Cristina Vazquez","first_name":"Cristina Vazquez","last_name":"Martin"}],"publication_status":"published","day":"08","date_created":"2019-08-18T22:00:39Z"},{"file_date_updated":"2020-07-14T12:47:42Z","scopus_import":"1","isi":1,"publication_identifier":{"eissn":["20452322"]},"issue":"1","pmid":1,"title":"A novel magnet-based scratch method for standardisation of wound-healing assays","_id":"6867","user_id":"4359f0d1-fa6c-11eb-b949-802e58b17ae8","abstract":[{"text":"A novel magnetic scratch method achieves repeatability, reproducibility and geometric control greater than pipette scratch assays and closely approximating the precision of cell exclusion assays while inducing the cell injury inherently necessary for wound healing assays. The magnetic scratch is affordable, easily implemented and standardisable and thus may contribute toward better comparability of data generated in different studies and laboratories.","lang":"eng"}],"intvolume":"         9","citation":{"short":"M. Fenu, T. Bettermann, C. Vogl, N. Darwish-Miranda, J. Schramel, F. Jenner, I. Ribitsch, Scientific Reports 9 (2019).","ieee":"M. Fenu <i>et al.</i>, “A novel magnet-based scratch method for standardisation of wound-healing assays,” <i>Scientific Reports</i>, vol. 9, no. 1. Springer Nature, 2019.","ista":"Fenu M, Bettermann T, Vogl C, Darwish-Miranda N, Schramel J, Jenner F, Ribitsch I. 2019. A novel magnet-based scratch method for standardisation of wound-healing assays. Scientific Reports. 9(1), 12625.","apa":"Fenu, M., Bettermann, T., Vogl, C., Darwish-Miranda, N., Schramel, J., Jenner, F., &#38; Ribitsch, I. (2019). A novel magnet-based scratch method for standardisation of wound-healing assays. <i>Scientific Reports</i>. Springer Nature. <a href=\"https://doi.org/10.1038/s41598-019-48930-7\">https://doi.org/10.1038/s41598-019-48930-7</a>","chicago":"Fenu, M., T. Bettermann, C. Vogl, Nasser Darwish-Miranda, J. Schramel, F. Jenner, and I. Ribitsch. “A Novel Magnet-Based Scratch Method for Standardisation of Wound-Healing Assays.” <i>Scientific Reports</i>. Springer Nature, 2019. <a href=\"https://doi.org/10.1038/s41598-019-48930-7\">https://doi.org/10.1038/s41598-019-48930-7</a>.","ama":"Fenu M, Bettermann T, Vogl C, et al. A novel magnet-based scratch method for standardisation of wound-healing assays. <i>Scientific Reports</i>. 2019;9(1). doi:<a href=\"https://doi.org/10.1038/s41598-019-48930-7\">10.1038/s41598-019-48930-7</a>","mla":"Fenu, M., et al. “A Novel Magnet-Based Scratch Method for Standardisation of Wound-Healing Assays.” <i>Scientific Reports</i>, vol. 9, no. 1, 12625, Springer Nature, 2019, doi:<a href=\"https://doi.org/10.1038/s41598-019-48930-7\">10.1038/s41598-019-48930-7</a>."},"has_accepted_license":"1","status":"public","department":[{"_id":"Bio"}],"tmp":{"legal_code_url":"https://creativecommons.org/licenses/by/4.0/legalcode","image":"/images/cc_by.png","short":"CC BY (4.0)","name":"Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)"},"ddc":["570"],"language":[{"iso":"eng"}],"year":"2019","day":"02","publication_status":"published","date_created":"2019-09-15T22:00:42Z","author":[{"full_name":"Fenu, M.","first_name":"M.","last_name":"Fenu"},{"last_name":"Bettermann","full_name":"Bettermann, T.","first_name":"T."},{"last_name":"Vogl","first_name":"C.","full_name":"Vogl, C."},{"full_name":"Darwish-Miranda, Nasser","id":"39CD9926-F248-11E8-B48F-1D18A9856A87","first_name":"Nasser","last_name":"Darwish-Miranda","orcid":"0000-0002-8821-8236"},{"first_name":"J.","full_name":"Schramel, J.","last_name":"Schramel"},{"last_name":"Jenner","first_name":"F.","full_name":"Jenner, F."},{"full_name":"Ribitsch, I.","first_name":"I.","last_name":"Ribitsch"}],"file":[{"relation":"main_file","creator":"dernst","file_id":"6879","date_created":"2019-09-16T12:42:40Z","content_type":"application/pdf","file_size":3523795,"date_updated":"2020-07-14T12:47:42Z","checksum":"9cfd986d4108e288cc72276ef047ab0c","access_level":"open_access","file_name":"2019_ScientificReports_Fenu.pdf"}],"quality_controlled":"1","oa":1,"article_number":"12625","month":"09","date_updated":"2023-08-29T07:55:15Z","doi":"10.1038/s41598-019-48930-7","publisher":"Springer Nature","publication":"Scientific Reports","article_processing_charge":"No","oa_version":"Published Version","external_id":{"pmid":["31477739"],"isi":["000483697800007"]},"type":"journal_article","date_published":"2019-09-02T00:00:00Z","volume":9},{"department":[{"_id":"LifeSc"}],"year":"2019","related_material":{"record":[{"id":"6819","relation":"used_in_publication","status":"public"}]},"day":"09","date_created":"2021-08-06T08:14:05Z","author":[{"full_name":"Antoniou, Michael N.","first_name":"Michael N.","last_name":"Antoniou"},{"full_name":"Nicolas, Armel","first_name":"Armel","id":"2A103192-F248-11E8-B48F-1D18A9856A87","last_name":"Nicolas"},{"first_name":"Robin","full_name":"Mesnage, Robin","last_name":"Mesnage"},{"last_name":"Biserni","full_name":"Biserni, Martina","first_name":"Martina"},{"first_name":"Francesco V.","full_name":"Rao, Francesco V.","last_name":"Rao"},{"full_name":"Martin, Cristina Vazquez","first_name":"Cristina Vazquez","last_name":"Martin"}],"oa":1,"month":"08","_id":"9784","date_updated":"2023-02-23T12:52:29Z","title":"MOESM1 of Glyphosate does not substitute for glycine in proteins of actively dividing mammalian cells","publisher":"Springer Nature","doi":"10.6084/m9.figshare.9411761.v1","abstract":[{"text":"Additional file 1: Table S1. Kinetics of MDA-MB-231 cell growth in either the presence or absence of 100Â mg/L glyphosate. Cell counts are given at day-1 of seeding flasks and following 6-days of continuous culture. Note: no differences in cell numbers were observed between negative control and glyphosate treated cultures.","lang":"eng"}],"article_processing_charge":"No","user_id":"6785fbc1-c503-11eb-8a32-93094b40e1cf","main_file_link":[{"url":"https://doi.org/10.6084/m9.figshare.9411761.v1","open_access":"1"}],"citation":{"mla":"Antoniou, Michael N., et al. <i>MOESM1 of Glyphosate Does Not Substitute for Glycine in Proteins of Actively Dividing Mammalian Cells</i>. Springer Nature, 2019, doi:<a href=\"https://doi.org/10.6084/m9.figshare.9411761.v1\">10.6084/m9.figshare.9411761.v1</a>.","ama":"Antoniou MN, Nicolas A, Mesnage R, Biserni M, Rao FV, Martin CV. MOESM1 of Glyphosate does not substitute for glycine in proteins of actively dividing mammalian cells. 2019. doi:<a href=\"https://doi.org/10.6084/m9.figshare.9411761.v1\">10.6084/m9.figshare.9411761.v1</a>","chicago":"Antoniou, Michael N., Armel Nicolas, Robin Mesnage, Martina Biserni, Francesco V. Rao, and Cristina Vazquez Martin. “MOESM1 of Glyphosate Does Not Substitute for Glycine in Proteins of Actively Dividing Mammalian Cells.” Springer Nature, 2019. <a href=\"https://doi.org/10.6084/m9.figshare.9411761.v1\">https://doi.org/10.6084/m9.figshare.9411761.v1</a>.","apa":"Antoniou, M. N., Nicolas, A., Mesnage, R., Biserni, M., Rao, F. V., &#38; Martin, C. V. (2019). MOESM1 of Glyphosate does not substitute for glycine in proteins of actively dividing mammalian cells. Springer Nature. <a href=\"https://doi.org/10.6084/m9.figshare.9411761.v1\">https://doi.org/10.6084/m9.figshare.9411761.v1</a>","ista":"Antoniou MN, Nicolas A, Mesnage R, Biserni M, Rao FV, Martin CV. 2019. MOESM1 of Glyphosate does not substitute for glycine in proteins of actively dividing mammalian cells, Springer Nature, <a href=\"https://doi.org/10.6084/m9.figshare.9411761.v1\">10.6084/m9.figshare.9411761.v1</a>.","short":"M.N. Antoniou, A. Nicolas, R. Mesnage, M. Biserni, F.V. Rao, C.V. Martin, (2019).","ieee":"M. N. Antoniou, A. Nicolas, R. Mesnage, M. Biserni, F. V. Rao, and C. V. Martin, “MOESM1 of Glyphosate does not substitute for glycine in proteins of actively dividing mammalian cells.” Springer Nature, 2019."},"oa_version":"Published Version","status":"public","date_published":"2019-08-09T00:00:00Z","type":"research_data_reference"},{"author":[{"orcid":"0000-0002-6625-3348","last_name":"Hons","id":"4167FE56-F248-11E8-B48F-1D18A9856A87","first_name":"Miroslav","full_name":"Hons, Miroslav"},{"full_name":"Kopf, Aglaja","first_name":"Aglaja","id":"31DAC7B6-F248-11E8-B48F-1D18A9856A87","last_name":"Kopf","orcid":"0000-0002-2187-6656"},{"last_name":"Hauschild","orcid":"0000-0001-9843-3522","full_name":"Hauschild, Robert","id":"4E01D6B4-F248-11E8-B48F-1D18A9856A87","first_name":"Robert"},{"last_name":"Leithner","orcid":"0000-0002-1073-744X","full_name":"Leithner, Alexander F","id":"3B1B77E4-F248-11E8-B48F-1D18A9856A87","first_name":"Alexander F"},{"last_name":"Gärtner","orcid":"0000-0001-6120-3723","full_name":"Gärtner, Florian R","id":"397A88EE-F248-11E8-B48F-1D18A9856A87","first_name":"Florian R"},{"last_name":"Abe","full_name":"Abe, Jun","first_name":"Jun"},{"last_name":"Renkawitz","orcid":"0000-0003-2856-3369","id":"3F0587C8-F248-11E8-B48F-1D18A9856A87","first_name":"Jörg","full_name":"Renkawitz, Jörg"},{"last_name":"Stein","full_name":"Stein, Jens","first_name":"Jens"},{"last_name":"Sixt","orcid":"0000-0002-6620-9179","first_name":"Michael K","id":"41E9FBEA-F248-11E8-B48F-1D18A9856A87","full_name":"Sixt, Michael K"}],"page":"606 - 616","date_created":"2018-12-11T11:44:10Z","acknowledged_ssus":[{"_id":"SSU"}],"day":"18","publication_status":"published","publist_id":"8040","related_material":{"record":[{"id":"6891","status":"public","relation":"dissertation_contains"}]},"language":[{"iso":"eng"}],"year":"2018","department":[{"_id":"MiSi"},{"_id":"Bio"}],"project":[{"name":"Cellular navigation along spatial gradients","call_identifier":"H2020","_id":"25FE9508-B435-11E9-9278-68D0E5697425","grant_number":"724373"},{"call_identifier":"H2020","name":"Mechanical Adaptation of Lamellipodial Actin Networks in Migrating Cells","_id":"260AA4E2-B435-11E9-9278-68D0E5697425","grant_number":"747687"},{"_id":"25A48D24-B435-11E9-9278-68D0E5697425","grant_number":"ALTF 1396-2014","name":"Molecular and system level view of immune cell migration"},{"_id":"25A603A2-B435-11E9-9278-68D0E5697425","grant_number":"281556","name":"Cytoskeletal force generation and force transduction of migrating leukocytes (EU)","call_identifier":"FP7"}],"type":"journal_article","date_published":"2018-05-18T00:00:00Z","volume":19,"external_id":{"pmid":["29777221"],"isi":["000433041500026"]},"oa_version":"Published Version","article_processing_charge":"No","date_updated":"2024-03-25T23:30:22Z","publisher":"Nature Publishing Group","doi":"10.1038/s41590-018-0109-z","publication":"Nature Immunology","month":"05","quality_controlled":"1","oa":1,"issue":"6","scopus_import":"1","isi":1,"status":"public","ec_funded":1,"intvolume":"        19","citation":{"ista":"Hons M, Kopf A, Hauschild R, Leithner AF, Gärtner FR, Abe J, Renkawitz J, Stein J, Sixt MK. 2018. Chemokines and integrins independently tune actin flow and substrate friction during intranodal migration of T cells. Nature Immunology. 19(6), 606–616.","apa":"Hons, M., Kopf, A., Hauschild, R., Leithner, A. F., Gärtner, F. R., Abe, J., … Sixt, M. K. (2018). Chemokines and integrins independently tune actin flow and substrate friction during intranodal migration of T cells. <i>Nature Immunology</i>. Nature Publishing Group. <a href=\"https://doi.org/10.1038/s41590-018-0109-z\">https://doi.org/10.1038/s41590-018-0109-z</a>","ieee":"M. Hons <i>et al.</i>, “Chemokines and integrins independently tune actin flow and substrate friction during intranodal migration of T cells,” <i>Nature Immunology</i>, vol. 19, no. 6. Nature Publishing Group, pp. 606–616, 2018.","short":"M. Hons, A. Kopf, R. Hauschild, A.F. Leithner, F.R. Gärtner, J. Abe, J. Renkawitz, J. Stein, M.K. Sixt, Nature Immunology 19 (2018) 606–616.","ama":"Hons M, Kopf A, Hauschild R, et al. Chemokines and integrins independently tune actin flow and substrate friction during intranodal migration of T cells. <i>Nature Immunology</i>. 2018;19(6):606-616. doi:<a href=\"https://doi.org/10.1038/s41590-018-0109-z\">10.1038/s41590-018-0109-z</a>","mla":"Hons, Miroslav, et al. “Chemokines and Integrins Independently Tune Actin Flow and Substrate Friction during Intranodal Migration of T Cells.” <i>Nature Immunology</i>, vol. 19, no. 6, Nature Publishing Group, 2018, pp. 606–16, doi:<a href=\"https://doi.org/10.1038/s41590-018-0109-z\">10.1038/s41590-018-0109-z</a>.","chicago":"Hons, Miroslav, Aglaja Kopf, Robert Hauschild, Alexander F Leithner, Florian R Gärtner, Jun Abe, Jörg Renkawitz, Jens Stein, and Michael K Sixt. “Chemokines and Integrins Independently Tune Actin Flow and Substrate Friction during Intranodal Migration of T Cells.” <i>Nature Immunology</i>. Nature Publishing Group, 2018. <a href=\"https://doi.org/10.1038/s41590-018-0109-z\">https://doi.org/10.1038/s41590-018-0109-z</a>."},"main_file_link":[{"open_access":"1","url":"https://www.ncbi.nlm.nih.gov/pubmed/29777221"}],"user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","abstract":[{"lang":"eng","text":"Although much is known about the physiological framework of T cell motility, and numerous rate-limiting molecules have been identified through loss-of-function approaches, an integrated functional concept of T cell motility is lacking. Here, we used in vivo precision morphometry together with analysis of cytoskeletal dynamics in vitro to deconstruct the basic mechanisms of T cell migration within lymphatic organs. We show that the contributions of the integrin LFA-1 and the chemokine receptor CCR7 are complementary rather than positioned in a linear pathway, as they are during leukocyte extravasation from the blood vasculature. Our data demonstrate that CCR7 controls cortical actin flows, whereas integrins mediate substrate friction that is sufficient to drive locomotion in the absence of considerable surface adhesions and plasma membrane flux."}],"title":"Chemokines and integrins independently tune actin flow and substrate friction during intranodal migration of T cells","_id":"15","pmid":1,"acknowledgement":"This work was funded by grants from the European Research Council (ERC StG 281556 and CoG 724373) and the Austrian Science Foundation (FWF) to M.S. and by Swiss National Foundation (SNF) project grants 31003A_135649, 31003A_153457 and CR23I3_156234 to J.V.S. F.G. received funding from the European Union’s Horizon 2020 research and innovation programme under the Marie Skłodowska-Curie grant agreement no. 747687, and J.R. was funded by an EMBO long-term fellowship (ALTF 1396-2014)."},{"month":"07","quality_controlled":"1","article_processing_charge":"No","publication":"Methods in Cell Biology","publisher":"Academic Press","doi":"10.1016/bs.mcb.2018.07.004","date_updated":"2023-09-13T08:56:35Z","oa_version":"None","external_id":{"pmid":["30165964"],"isi":["000452412300006"]},"volume":147,"type":"book_chapter","date_published":"2018-07-27T00:00:00Z","department":[{"_id":"MiSi"},{"_id":"NanoFab"}],"year":"2018","language":[{"iso":"eng"}],"date_created":"2018-12-11T11:44:54Z","publist_id":"7768","publication_status":"published","day":"27","author":[{"last_name":"Renkawitz","orcid":"0000-0003-2856-3369","full_name":"Renkawitz, Jörg","first_name":"Jörg","id":"3F0587C8-F248-11E8-B48F-1D18A9856A87"},{"last_name":"Reversat","orcid":"0000-0003-0666-8928","full_name":"Reversat, Anne","first_name":"Anne","id":"35B76592-F248-11E8-B48F-1D18A9856A87"},{"last_name":"Leithner","orcid":"0000-0002-1073-744X","id":"3B1B77E4-F248-11E8-B48F-1D18A9856A87","first_name":"Alexander F","full_name":"Leithner, Alexander F"},{"orcid":"0000-0001-5145-4609","last_name":"Merrin","full_name":"Merrin, Jack","first_name":"Jack","id":"4515C308-F248-11E8-B48F-1D18A9856A87"},{"id":"41E9FBEA-F248-11E8-B48F-1D18A9856A87","first_name":"Michael K","full_name":"Sixt, Michael K","orcid":"0000-0002-6620-9179","last_name":"Sixt"}],"page":"79 - 91","pmid":1,"abstract":[{"lang":"eng","text":"Cells migrating in multicellular organisms steadily traverse complex three-dimensional (3D) environments. To decipher the underlying cell biology, current experimental setups either use simplified 2D, tissue-mimetic 3D (e.g., collagen matrices) or in vivo environments. While only in vivo experiments are truly physiological, they do not allow for precise manipulation of environmental parameters. 2D in vitro experiments do allow mechanical and chemical manipulations, but increasing evidence demonstrates substantial differences of migratory mechanisms in 2D and 3D. Here, we describe simple, robust, and versatile “pillar forests” to investigate cell migration in complex but fully controllable 3D environments. Pillar forests are polydimethylsiloxane-based setups, in which two closely adjacent surfaces are interconnected by arrays of micrometer-sized pillars. Changing the pillar shape, size, height and the inter-pillar distance precisely manipulates microenvironmental parameters (e.g., pore sizes, micro-geometry, micro-topology), while being easily combined with chemotactic cues, surface coatings, diverse cell types and advanced imaging techniques. Thus, pillar forests combine the advantages of 2D cell migration assays with the precise definition of 3D environmental parameters."}],"user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","_id":"153","title":"Micro-engineered “pillar forests” to study cell migration in complex but controlled 3D environments","citation":{"ieee":"J. Renkawitz, A. Reversat, A. F. Leithner, J. Merrin, and M. K. Sixt, “Micro-engineered ‘pillar forests’ to study cell migration in complex but controlled 3D environments,” in <i>Methods in Cell Biology</i>, vol. 147, Academic Press, 2018, pp. 79–91.","short":"J. Renkawitz, A. Reversat, A.F. Leithner, J. Merrin, M.K. Sixt, in:, Methods in Cell Biology, Academic Press, 2018, pp. 79–91.","apa":"Renkawitz, J., Reversat, A., Leithner, A. F., Merrin, J., &#38; Sixt, M. K. (2018). Micro-engineered “pillar forests” to study cell migration in complex but controlled 3D environments. In <i>Methods in Cell Biology</i> (Vol. 147, pp. 79–91). Academic Press. <a href=\"https://doi.org/10.1016/bs.mcb.2018.07.004\">https://doi.org/10.1016/bs.mcb.2018.07.004</a>","ista":"Renkawitz J, Reversat A, Leithner AF, Merrin J, Sixt MK. 2018.Micro-engineered “pillar forests” to study cell migration in complex but controlled 3D environments. In: Methods in Cell Biology. vol. 147, 79–91.","chicago":"Renkawitz, Jörg, Anne Reversat, Alexander F Leithner, Jack Merrin, and Michael K Sixt. “Micro-Engineered ‘Pillar Forests’ to Study Cell Migration in Complex but Controlled 3D Environments.” In <i>Methods in Cell Biology</i>, 147:79–91. Academic Press, 2018. <a href=\"https://doi.org/10.1016/bs.mcb.2018.07.004\">https://doi.org/10.1016/bs.mcb.2018.07.004</a>.","mla":"Renkawitz, Jörg, et al. “Micro-Engineered ‘Pillar Forests’ to Study Cell Migration in Complex but Controlled 3D Environments.” <i>Methods in Cell Biology</i>, vol. 147, Academic Press, 2018, pp. 79–91, doi:<a href=\"https://doi.org/10.1016/bs.mcb.2018.07.004\">10.1016/bs.mcb.2018.07.004</a>.","ama":"Renkawitz J, Reversat A, Leithner AF, Merrin J, Sixt MK. Micro-engineered “pillar forests” to study cell migration in complex but controlled 3D environments. In: <i>Methods in Cell Biology</i>. Vol 147. Academic Press; 2018:79-91. doi:<a href=\"https://doi.org/10.1016/bs.mcb.2018.07.004\">10.1016/bs.mcb.2018.07.004</a>"},"intvolume":"       147","status":"public","isi":1,"scopus_import":"1","publication_identifier":{"issn":["0091679X"]}},{"isi":1,"scopus_import":"1","issue":"12","publication_identifier":{"issn":["0022-1554"]},"abstract":[{"text":"For ultrafast fixation of biological samples to avoid artifacts, high-pressure freezing (HPF) followed by freeze substitution (FS) is preferred over chemical fixation at room temperature. After HPF, samples are maintained at low temperature during dehydration and fixation, while avoiding damaging recrystallization. This is a notoriously slow process. McDonald and Webb demonstrated, in 2011, that sample agitation during FS dramatically reduces the necessary time. Then, in 2015, we (H.G. and S.R.) introduced an agitation module into the cryochamber of an automated FS unit and demonstrated that the preparation of algae could be shortened from days to a couple of hours. We argued that variability in the processing, reproducibility, and safety issues are better addressed using automated FS units. For dissemination, we started low-cost manufacturing of agitation modules for two of the most widely used FS units, the Automatic Freeze Substitution Systems, AFS(1) and AFS2, from Leica Microsystems, using three dimensional (3D)-printing of the major components. To test them, several labs independently used the modules on a wide variety of specimens that had previously been processed by manual agitation, or without agitation. We demonstrate that automated processing with sample agitation saves time, increases flexibility with respect to sample requirements and protocols, and produces data of at least as good quality as other approaches.","lang":"eng"}],"user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","_id":"163","title":"Agitation modules: Flexible means to accelerate automated freeze substitution","pmid":1,"status":"public","main_file_link":[{"open_access":"1","url":"https://doi.org/10.1369/0022155418786698"}],"citation":{"chicago":"Reipert, Siegfried, Helmuth Goldammer, Christine Richardson, Martin Goldberg, Timothy Hawkins, Elena Saeckl, Walter Kaufmann, Sebastian Antreich, and York Stierhof. “Agitation Modules: Flexible Means to Accelerate Automated Freeze Substitution.” <i>Journal of Histochemistry and Cytochemistry</i>. SAGE Publications, 2018. <a href=\"https://doi.org/10.1369/0022155418786698\">https://doi.org/10.1369/0022155418786698</a>.","ama":"Reipert S, Goldammer H, Richardson C, et al. Agitation modules: Flexible means to accelerate automated freeze substitution. <i>Journal of Histochemistry and Cytochemistry</i>. 2018;66(12):903-921. doi:<a href=\"https://doi.org/10.1369/0022155418786698\">10.1369/0022155418786698</a>","mla":"Reipert, Siegfried, et al. “Agitation Modules: Flexible Means to Accelerate Automated Freeze Substitution.” <i>Journal of Histochemistry and Cytochemistry</i>, vol. 66, no. 12, SAGE Publications, 2018, pp. 903–21, doi:<a href=\"https://doi.org/10.1369/0022155418786698\">10.1369/0022155418786698</a>.","short":"S. Reipert, H. Goldammer, C. Richardson, M. Goldberg, T. Hawkins, E. Saeckl, W. Kaufmann, S. Antreich, Y. Stierhof, Journal of Histochemistry and Cytochemistry 66 (2018) 903–921.","ieee":"S. Reipert <i>et al.</i>, “Agitation modules: Flexible means to accelerate automated freeze substitution,” <i>Journal of Histochemistry and Cytochemistry</i>, vol. 66, no. 12. SAGE Publications, pp. 903–921, 2018.","ista":"Reipert S, Goldammer H, Richardson C, Goldberg M, Hawkins T, Saeckl E, Kaufmann W, Antreich S, Stierhof Y. 2018. Agitation modules: Flexible means to accelerate automated freeze substitution. Journal of Histochemistry and Cytochemistry. 66(12), 903–921.","apa":"Reipert, S., Goldammer, H., Richardson, C., Goldberg, M., Hawkins, T., Saeckl, E., … Stierhof, Y. (2018). Agitation modules: Flexible means to accelerate automated freeze substitution. <i>Journal of Histochemistry and Cytochemistry</i>. SAGE Publications. <a href=\"https://doi.org/10.1369/0022155418786698\">https://doi.org/10.1369/0022155418786698</a>"},"intvolume":"        66","language":[{"iso":"eng"}],"year":"2018","department":[{"_id":"RySh"},{"_id":"EM-Fac"}],"author":[{"last_name":"Reipert","full_name":"Reipert, Siegfried","first_name":"Siegfried"},{"first_name":"Helmuth","full_name":"Goldammer, Helmuth","last_name":"Goldammer"},{"last_name":"Richardson","full_name":"Richardson, Christine","first_name":"Christine"},{"full_name":"Goldberg, Martin","first_name":"Martin","last_name":"Goldberg"},{"full_name":"Hawkins, Timothy","first_name":"Timothy","last_name":"Hawkins"},{"last_name":"Hollergschwandtner","full_name":"Hollergschwandtner, Elena","first_name":"Elena","id":"3C054040-F248-11E8-B48F-1D18A9856A87"},{"orcid":"0000-0001-9735-5315","last_name":"Kaufmann","full_name":"Kaufmann, Walter","first_name":"Walter","id":"3F99E422-F248-11E8-B48F-1D18A9856A87"},{"last_name":"Antreich","first_name":"Sebastian","full_name":"Antreich, Sebastian"},{"last_name":"Stierhof","full_name":"Stierhof, York","first_name":"York"}],"page":"903-921","date_created":"2018-12-11T11:44:57Z","publication_status":"published","day":"01","article_processing_charge":"No","publication":"Journal of Histochemistry and Cytochemistry","date_updated":"2023-10-17T08:42:24Z","publisher":"SAGE Publications","doi":"10.1369/0022155418786698","month":"12","quality_controlled":"1","oa":1,"volume":66,"type":"journal_article","date_published":"2018-12-01T00:00:00Z","oa_version":"Published Version","external_id":{"pmid":["29969056"],"isi":["000452277700005"]},"article_type":"original"},{"type":"journal_article","date_published":"2018-06-25T00:00:00Z","volume":4,"external_id":{"pmid":["29942048"],"isi":["000443221200017"]},"oa_version":"Submitted Version","article_type":"original","publisher":"Springer Nature","doi":"10.1038/s41477-018-0190-1","date_updated":"2023-09-15T12:11:03Z","publication":"Nature Plants","article_processing_charge":"No","quality_controlled":"1","oa":1,"month":"06","page":"453 - 459","author":[{"orcid":"0000-0002-9767-8699","last_name":"Fendrych","full_name":"Fendrych, Matyas","first_name":"Matyas","id":"43905548-F248-11E8-B48F-1D18A9856A87"},{"full_name":"Akhmanova, Maria","id":"3425EC26-F248-11E8-B48F-1D18A9856A87","first_name":"Maria","orcid":"0000-0003-1522-3162","last_name":"Akhmanova"},{"last_name":"Merrin","orcid":"0000-0001-5145-4609","id":"4515C308-F248-11E8-B48F-1D18A9856A87","first_name":"Jack","full_name":"Merrin, Jack"},{"first_name":"Matous","full_name":"Glanc, Matous","last_name":"Glanc"},{"first_name":"Shinya","full_name":"Hagihara, Shinya","last_name":"Hagihara"},{"last_name":"Takahashi","first_name":"Koji","full_name":"Takahashi, Koji"},{"first_name":"Naoyuki","full_name":"Uchida, Naoyuki","last_name":"Uchida"},{"full_name":"Torii, Keiko U","first_name":"Keiko U","last_name":"Torii"},{"last_name":"Friml","orcid":"0000-0002-8302-7596","id":"4159519E-F248-11E8-B48F-1D18A9856A87","first_name":"Jirí","full_name":"Friml, Jirí"}],"day":"25","publist_id":"7728","publication_status":"published","date_created":"2018-12-11T11:45:07Z","related_material":{"link":[{"description":"News on IST Homepage","url":"https://ist.ac.at/en/news/new-mechanism-for-the-plant-hormone-auxin-discovered/","relation":"press_release"}]},"language":[{"iso":"eng"}],"year":"2018","department":[{"_id":"JiFr"},{"_id":"DaSi"},{"_id":"NanoFab"}],"status":"public","intvolume":"         4","citation":{"chicago":"Fendrych, Matyas, Maria Akhmanova, Jack Merrin, Matous Glanc, Shinya Hagihara, Koji Takahashi, Naoyuki Uchida, Keiko U Torii, and Jiří Friml. “Rapid and Reversible Root Growth Inhibition by TIR1 Auxin Signalling.” <i>Nature Plants</i>. Springer Nature, 2018. <a href=\"https://doi.org/10.1038/s41477-018-0190-1\">https://doi.org/10.1038/s41477-018-0190-1</a>.","mla":"Fendrych, Matyas, et al. “Rapid and Reversible Root Growth Inhibition by TIR1 Auxin Signalling.” <i>Nature Plants</i>, vol. 4, no. 7, Springer Nature, 2018, pp. 453–59, doi:<a href=\"https://doi.org/10.1038/s41477-018-0190-1\">10.1038/s41477-018-0190-1</a>.","ama":"Fendrych M, Akhmanova M, Merrin J, et al. Rapid and reversible root growth inhibition by TIR1 auxin signalling. <i>Nature Plants</i>. 2018;4(7):453-459. doi:<a href=\"https://doi.org/10.1038/s41477-018-0190-1\">10.1038/s41477-018-0190-1</a>","ieee":"M. Fendrych <i>et al.</i>, “Rapid and reversible root growth inhibition by TIR1 auxin signalling,” <i>Nature Plants</i>, vol. 4, no. 7. Springer Nature, pp. 453–459, 2018.","short":"M. Fendrych, M. Akhmanova, J. Merrin, M. Glanc, S. Hagihara, K. Takahashi, N. Uchida, K.U. Torii, J. Friml, Nature Plants 4 (2018) 453–459.","apa":"Fendrych, M., Akhmanova, M., Merrin, J., Glanc, M., Hagihara, S., Takahashi, K., … Friml, J. (2018). Rapid and reversible root growth inhibition by TIR1 auxin signalling. <i>Nature Plants</i>. Springer Nature. <a href=\"https://doi.org/10.1038/s41477-018-0190-1\">https://doi.org/10.1038/s41477-018-0190-1</a>","ista":"Fendrych M, Akhmanova M, Merrin J, Glanc M, Hagihara S, Takahashi K, Uchida N, Torii KU, Friml J. 2018. Rapid and reversible root growth inhibition by TIR1 auxin signalling. Nature Plants. 4(7), 453–459."},"main_file_link":[{"url":"https://www.ncbi.nlm.nih.gov/pubmed/29942048","open_access":"1"}],"title":"Rapid and reversible root growth inhibition by TIR1 auxin signalling","_id":"192","user_id":"c635000d-4b10-11ee-a964-aac5a93f6ac1","abstract":[{"text":"The phytohormone auxin is the information carrier in a plethora of developmental and physiological processes in plants(1). It has been firmly established that canonical, nuclear auxin signalling acts through regulation of gene transcription(2). Here, we combined microfluidics, live imaging, genetic engineering and computational modelling to reanalyse the classical case of root growth inhibition(3) by auxin. We show that Arabidopsis roots react to addition and removal of auxin by extremely rapid adaptation of growth rate. This process requires intracellular auxin perception but not transcriptional reprogramming. The formation of the canonical TIR1/AFB-Aux/IAA co-receptor complex is required for the growth regulation, hinting to a novel, non-transcriptional branch of this signalling pathway. Our results challenge the current understanding of root growth regulation by auxin and suggest another, presumably non-transcriptional, signalling output of the canonical auxin pathway.","lang":"eng"}],"pmid":1,"issue":"7","scopus_import":"1","isi":1},{"author":[{"orcid":"0000-0001-8126-0426","last_name":"Villányi","first_name":"Márton","id":"3FFCCD3A-F248-11E8-B48F-1D18A9856A87","full_name":"Villányi, Márton"}],"keyword":["Publication analysis","Bibliography","Open Access"],"datarep_id":"86","date_created":"2018-12-12T12:31:37Z","license":"https://creativecommons.org/publicdomain/zero/1.0/","day":"16","related_material":{"record":[{"status":"public","relation":"part_of_dissertation","id":"278"}]},"year":"2018","ddc":["020"],"tmp":{"short":"CC0 (1.0)","name":"Creative Commons Public Domain Dedication (CC0 1.0)","image":"/images/cc_0.png","legal_code_url":"https://creativecommons.org/publicdomain/zero/1.0/legalcode"},"department":[{"_id":"E-Lib"}],"file_date_updated":"2020-07-14T12:47:05Z","date_published":"2018-01-16T00:00:00Z","type":"research_data","status":"public","oa_version":"Submitted Version","citation":{"ama":"Villányi M. Data Check IOP Scopus vs. Publisher. 2018. doi:<a href=\"https://doi.org/10.15479/AT:ISTA:86\">10.15479/AT:ISTA:86</a>","mla":"Villányi, Márton. <i>Data Check IOP Scopus vs. Publisher</i>. Institute of Science and Technology Austria, 2018, doi:<a href=\"https://doi.org/10.15479/AT:ISTA:86\">10.15479/AT:ISTA:86</a>.","chicago":"Villányi, Márton. “Data Check IOP Scopus vs. Publisher.” Institute of Science and Technology Austria, 2018. <a href=\"https://doi.org/10.15479/AT:ISTA:86\">https://doi.org/10.15479/AT:ISTA:86</a>.","ista":"Villányi M. 2018. Data Check IOP Scopus vs. Publisher, Institute of Science and Technology Austria, <a href=\"https://doi.org/10.15479/AT:ISTA:86\">10.15479/AT:ISTA:86</a>.","apa":"Villányi, M. (2018). Data Check IOP Scopus vs. Publisher. Institute of Science and Technology Austria. <a href=\"https://doi.org/10.15479/AT:ISTA:86\">https://doi.org/10.15479/AT:ISTA:86</a>","short":"M. Villányi, (2018).","ieee":"M. Villányi, “Data Check IOP Scopus vs. Publisher.” Institute of Science and Technology Austria, 2018."},"has_accepted_license":"1","article_processing_charge":"No","user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","abstract":[{"text":"Comparison of Scopus' and publisher's data on Austrian publication output at IOP. ","lang":"ger"}],"date_updated":"2024-02-21T13:42:21Z","doi":"10.15479/AT:ISTA:86","title":"Data Check IOP Scopus vs. Publisher","publisher":"Institute of Science and Technology Austria","_id":"5574","month":"01","file":[{"content_type":"application/zip","date_created":"2018-12-12T13:05:14Z","file_id":"5642","creator":"system","relation":"main_file","file_name":"IST-2018-86-v1+1_Data_Check_IOP_Scopus_vs._Publisher.zip","access_level":"open_access","checksum":"c7a61147bd15cb4ae45878d270628c06","date_updated":"2020-07-14T12:47:05Z","file_size":12283857}],"oa":1},{"status":"public","date_published":"2018-01-16T00:00:00Z","type":"research_data","citation":{"ama":"Villányi M. Data Check RSC Scopus vs. FWF. 2018. doi:<a href=\"https://doi.org/10.15479/AT:ISTA:87\">10.15479/AT:ISTA:87</a>","mla":"Villányi, Márton. <i>Data Check RSC Scopus vs. FWF</i>. Institute of Science and Technology Austria, 2018, doi:<a href=\"https://doi.org/10.15479/AT:ISTA:87\">10.15479/AT:ISTA:87</a>.","chicago":"Villányi, Márton. “Data Check RSC Scopus vs. FWF.” Institute of Science and Technology Austria, 2018. <a href=\"https://doi.org/10.15479/AT:ISTA:87\">https://doi.org/10.15479/AT:ISTA:87</a>.","ista":"Villányi M. 2018. Data Check RSC Scopus vs. FWF, Institute of Science and Technology Austria, <a href=\"https://doi.org/10.15479/AT:ISTA:87\">10.15479/AT:ISTA:87</a>.","apa":"Villányi, M. (2018). Data Check RSC Scopus vs. FWF. Institute of Science and Technology Austria. <a href=\"https://doi.org/10.15479/AT:ISTA:87\">https://doi.org/10.15479/AT:ISTA:87</a>","ieee":"M. Villányi, “Data Check RSC Scopus vs. FWF.” Institute of Science and Technology Austria, 2018.","short":"M. Villányi, (2018)."},"has_accepted_license":"1","oa_version":"Submitted Version","doi":"10.15479/AT:ISTA:87","publisher":"Institute of Science and Technology Austria","date_updated":"2024-02-21T13:43:25Z","title":"Data Check RSC Scopus vs. FWF","_id":"5575","article_processing_charge":"No","user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","abstract":[{"text":"Comparison of Scopus' and FWF's data on Austrian publication output at RSC. ","lang":"ger"}],"file":[{"file_size":277078,"date_updated":"2020-07-14T12:47:05Z","checksum":"563cc5266c0bac354007873c92be777b","file_name":"IST-2018-87-v1+1_Data_Check_RSC_Scopus_vs._FWF.zip","access_level":"open_access","creator":"system","relation":"main_file","file_id":"5610","date_created":"2018-12-12T13:02:44Z","content_type":"application/zip"}],"oa":1,"month":"01","datarep_id":"87","keyword":["Publication analysis","Bibliography","Open Access"],"author":[{"last_name":"Villányi","orcid":"0000-0001-8126-0426","full_name":"Villányi, Márton","first_name":"Márton","id":"3FFCCD3A-F248-11E8-B48F-1D18A9856A87"}],"day":"16","date_created":"2018-12-12T12:31:37Z","related_material":{"record":[{"id":"278","relation":"part_of_dissertation","status":"public"}]},"ddc":["020"],"year":"2018","file_date_updated":"2020-07-14T12:47:05Z","tmp":{"short":"CC0 (1.0)","name":"Creative Commons Public Domain Dedication (CC0 1.0)","image":"/images/cc_0.png","legal_code_url":"https://creativecommons.org/publicdomain/zero/1.0/legalcode"},"department":[{"_id":"E-Lib"}]},{"date_created":"2018-12-12T12:31:37Z","day":"16","keyword":["Publication analysis","Bibliography","Open Access"],"author":[{"orcid":"0000-0001-8126-0426","last_name":"Villányi","id":"3FFCCD3A-F248-11E8-B48F-1D18A9856A87","first_name":"Márton","full_name":"Villányi, Márton"}],"datarep_id":"88","file_date_updated":"2020-07-14T12:47:05Z","tmp":{"short":"CC0 (1.0)","name":"Creative Commons Public Domain Dedication (CC0 1.0)","image":"/images/cc_0.png","legal_code_url":"https://creativecommons.org/publicdomain/zero/1.0/legalcode"},"department":[{"_id":"E-Lib"}],"related_material":{"record":[{"status":"public","relation":"part_of_dissertation","id":"278"}]},"ddc":["020"],"year":"2018","oa_version":"Submitted Version","citation":{"ieee":"M. Villányi, “Data Check T&#38;F Scopus vs. FWF.” Institute of Science and Technology Austria, 2018.","short":"M. Villányi, (2018).","ista":"Villányi M. 2018. Data Check T&#38;F Scopus vs. FWF, Institute of Science and Technology Austria, <a href=\"https://doi.org/10.15479/AT:ISTA:88\">10.15479/AT:ISTA:88</a>.","apa":"Villányi, M. (2018). Data Check T&#38;F Scopus vs. FWF. Institute of Science and Technology Austria. <a href=\"https://doi.org/10.15479/AT:ISTA:88\">https://doi.org/10.15479/AT:ISTA:88</a>","chicago":"Villányi, Márton. “Data Check T&#38;F Scopus vs. FWF.” Institute of Science and Technology Austria, 2018. <a href=\"https://doi.org/10.15479/AT:ISTA:88\">https://doi.org/10.15479/AT:ISTA:88</a>.","ama":"Villányi M. Data Check T&#38;F Scopus vs. FWF. 2018. doi:<a href=\"https://doi.org/10.15479/AT:ISTA:88\">10.15479/AT:ISTA:88</a>","mla":"Villányi, Márton. <i>Data Check T&#38;F Scopus vs. FWF</i>. Institute of Science and Technology Austria, 2018, doi:<a href=\"https://doi.org/10.15479/AT:ISTA:88\">10.15479/AT:ISTA:88</a>."},"has_accepted_license":"1","date_published":"2018-01-16T00:00:00Z","type":"research_data","status":"public","month":"01","file":[{"file_name":"IST-2018-88-v1+1_Data_Check_T_F_Scopus_vs._FWF.zip","access_level":"open_access","checksum":"a887246c2b41b98df90ccbc1d62b4487","date_updated":"2020-07-14T12:47:05Z","file_size":741195,"content_type":"application/zip","date_created":"2018-12-12T13:02:32Z","file_id":"5598","relation":"main_file","creator":"system"}],"oa":1,"user_id":"2DF688A6-F248-11E8-B48F-1D18A9856A87","article_processing_charge":"No","abstract":[{"text":"Comparison of Scopus' and FWF's data on Austrian publication output at T&F.","lang":"ger"}],"doi":"10.15479/AT:ISTA:88","publisher":"Institute of Science and Technology Austria","date_updated":"2024-02-21T13:43:10Z","title":"Data Check T&F Scopus vs. FWF","_id":"5576"}]