@misc{5577,
  abstract     = {Data on Austrian open access publication output at Emerald from 2013-2017 including data analysis.},
  author       = {Villányi, Márton},
  keywords     = {Publication analysis, Bibliography, Open Access},
  publisher    = {Institute of Science and Technology Austria},
  title        = {{Emerald Austrian Publications 2013-2017}},
  doi          = {10.15479/AT:ISTA:89},
  year         = {2018},
}

@misc{5578,
  abstract     = {Data on Austrian open access publication output at IOP from 2012-2015 including data analysis.},
  author       = {Villányi, Márton},
  keywords     = {Publication analysis, Bibliography, Open Access},
  publisher    = {Institute of Science and Technology Austria},
  title        = {{IOP Austrian Publications 2012-2015}},
  doi          = {10.15479/AT:ISTA:90},
  year         = {2018},
}

@misc{5579,
  abstract     = {Data on Austrian open access publication output at RSC from 2013-2017 including data analysis.},
  author       = {Villányi, Márton},
  keywords     = {Publication analysis, Bibliography, Open Access},
  publisher    = {Institute of Science and Technology Austria},
  title        = {{RSC Austrian Publications 2013-2017}},
  doi          = {10.15479/AT:ISTA:91},
  year         = {2018},
}

@misc{5580,
  abstract     = {Data on Austrian open access publication output at SAGE from 2013-2017 including data analysis.},
  author       = {Villányi, Márton},
  keywords     = {Publication analysis, Bibliography, Open Access},
  publisher    = {Institute of Science and Technology Austria},
  title        = {{SAGE Austrian Publications 2013-2017}},
  doi          = {10.15479/AT:ISTA:92},
  year         = {2018},
}

@misc{5581,
  abstract     = {Data on Austrian open access publication output at Springer from 2013-2016 including data analysis.},
  author       = {Villányi, Márton},
  keywords     = {Publication analysis, Bibliography, Open Access},
  publisher    = {Institute of Science and Technology Austria},
  title        = {{Springer Austrian Publications 2013-2016}},
  doi          = {10.15479/AT:ISTA:93},
  year         = {2018},
}

@misc{5582,
  abstract     = {Data on Austrian open access publication output at Taylor&Francis from 2013-2017 including data analysis.},
  author       = {Villányi, Márton},
  keywords     = {Publication analysis, Bibliography, Open Access},
  publisher    = {Institute of Science and Technology Austria},
  title        = {{Taylor&Francis Austrian Publications 2013-2017}},
  doi          = {10.15479/AT:ISTA:94},
  year         = {2018},
}

@misc{5588,
  abstract     = {Script to perform a simple exponential lifetime fit of a ROI on time stacks acquired with a FLIM X16 TCSPC detector (+example data)},
  author       = {Hauschild, Robert},
  keywords     = {FLIM, FRET, fluorescence lifetime imaging},
  publisher    = {Institute of Science and Technology Austria},
  title        = {{Fluorescence lifetime analysis of FLIM X16 TCSPC data}},
  doi          = {10.15479/AT:ISTA:0113},
  year         = {2018},
}

@techreport{5686,
  author       = {Danowski, Patrick},
  pages        = {5},
  title        = {{An Austrian proposal for the Classification of Open Access Tuples (COAT) - Distinguish different Open Access types beyond colors}},
  doi          = {10.5281/zenodo.1244154},
  year         = {2018},
}

@misc{6459,
  author       = {Petritsch, Barbara},
  keywords     = {Open Access, Publication Analysis},
  location     = {Graz, Austria},
  publisher    = {IST Austria},
  title        = {{Open Access at IST Austria 2009-2017}},
  doi          = {10.5281/zenodo.1410279},
  year         = {2018},
}

@article{437,
  abstract     = {Dendritic cells (DCs) are sentinels of the adaptive immune system that reside in peripheral organs of mammals. Upon pathogen encounter, they undergo maturation and up-regulate the chemokine receptor CCR7 that guides them along gradients of its chemokine ligands CCL19 and 21 to the next draining lymph node. There, DCs present peripherally acquired antigen to naïve T cells, thereby triggering adaptive immunity.},
  author       = {Leithner, Alexander F and Renkawitz, Jörg and De Vries, Ingrid and Hauschild, Robert and Haecker, Hans and Sixt, Michael K},
  journal      = {European Journal of Immunology},
  number       = {6},
  pages        = {1074 -- 1077},
  publisher    = {Wiley-Blackwell},
  title        = {{Fast and efficient genetic engineering of hematopoietic precursor cells for the study of dendritic cell migration}},
  doi          = {10.1002/eji.201747358},
  volume       = {48},
  year         = {2018},
}

@article{442,
  abstract     = {The rapid auxin-triggered growth of the Arabidopsis hypocotyls involves the nuclear TIR1/AFB-Aux/IAA signaling and is accompanied by acidification of the apoplast and cell walls (Fendrych et al., 2016). Here, we describe in detail the method for analysis of the elongation and the TIR1/AFB-Aux/IAA-dependent auxin response in hypocotyl segments as well as the determination of relative values of the cell wall pH.},
  author       = {Li, Lanxin and Krens, Gabriel and Fendrych, Matyas and Friml, Jirí},
  issn         = {2331-8325},
  journal      = {Bio-protocol},
  number       = {1},
  publisher    = {Bio-protocol},
  title        = {{Real-time analysis of auxin response, cell wall pH and elongation in Arabidopsis thaliana Hypocotyls}},
  doi          = {10.21769/BioProtoc.2685},
  volume       = {8},
  year         = {2018},
}

@article{53,
  abstract     = {In 2013, a publication repository was implemented at IST Austria and 2015 after a thorough preparation phase a data repository was implemented - both based on the Open Source Software EPrints. In this text, designed as field report, we will reflect on our experiences with Open Source Software in general and specifically with EPrints regarding technical aspects but also regarding their characteristics of the user community. The second part is a pleading for including the end users in the process of implementation, adaption and evaluation.},
  author       = {Petritsch, Barbara and Porsche, Jana},
  journal      = {VÖB Mitteilungen},
  number       = {1},
  pages        = {199 -- 206},
  publisher    = {Vereinigung Österreichischer Bibliothekarinnen und Bibliothekare},
  title        = {{IST PubRep and IST DataRep: the institutional repositories at IST Austria}},
  doi          = {10.31263/voebm.v71i1.1993},
  volume       = {71},
  year         = {2018},
}

@article{275,
  abstract     = {Lymphatic endothelial cells (LECs) release extracellular chemokines to guide the migration of dendritic cells. In this study, we report that LECs also release basolateral exosome-rich endothelial vesicles (EEVs) that are secreted in greater numbers in the presence of inflammatory cytokines and accumulate in the perivascular stroma of small lymphatic vessels in human chronic inflammatory diseases. Proteomic analyses of EEV fractions identified > 1,700 cargo proteins and revealed a dominant motility-promoting protein signature. In vitro and ex vivo EEV fractions augmented cellular protrusion formation in a CX3CL1/fractalkine-dependent fashion and enhanced the directional migratory response of human dendritic cells along guidance cues. We conclude that perilymphatic LEC exosomes enhance exploratory behavior and thus promote directional migration of CX3CR1-expressing cells in complex tissue environments.},
  author       = {Brown, Markus and Johnson, Louise and Leone, Dario and Májek, Peter and Vaahtomeri, Kari and Senfter, Daniel and Bukosza, Nora and Schachner, Helga and Asfour, Gabriele and Langer, Brigitte and Hauschild, Robert and Parapatics, Katja and Hong, Young and Bennett, Keiryn and Kain, Renate and Detmar, Michael and Sixt, Michael K and Jackson, David and Kerjaschki, Dontscho},
  journal      = {Journal of Cell Biology},
  number       = {6},
  pages        = {2205 -- 2221},
  publisher    = {Rockefeller University Press},
  title        = {{Lymphatic exosomes promote dendritic cell migration along guidance cues}},
  doi          = {10.1083/jcb.201612051},
  volume       = {217},
  year         = {2018},
}

@phdthesis{278,
  abstract     = {Consortial subscription contracts regulate the digital access to publications between publishers and scientific libraries. However, since a couple of years the tendency towards a freely accessible publishing (Open Access) intensifies. As a consequence of this trend the contractual relationship between licensor and licensee is gradually changing as well: More and more contracts exercise influence on open access publishing. The present study attempts to compare Austrian examples of consortial licence contracts, which include components of open access. It describes the difference between pure subscription contracts and differing innovative deals including open access components. Thereby it becomes obvious that for the evaluation of this licence contracts new methods are needed. An essential new element of such analyses is the evaluation of the open access publication numbers. So this study tries to carry out such publication analyses for Austrian open access deals focusing on quantitative questions: How does the number of publications evolve? How does the open access share change? Publications reports of the publishers and database queries from Scopus form the data basis. The analysis of the data points out that differing approaches of contracts result in highly divergent results: Particular deals can prioritize a saving in costs or else the increase of the open access rate. It is to be assumed that within the following years further numerous open access deals will be negotiated. The finding of this study shall provide guidance.},
  author       = {Villányi, Márton},
  pages        = {94},
  publisher    = {Universität Wien},
  title        = {{Lizenzverträge mit Open-Access-Komponenten an österreichischen Bibliotheken}},
  year         = {2018},
}

@article{308,
  abstract     = {Migrating cells penetrate tissue barriers during development, inflammatory responses, and tumor metastasis. We study if migration in vivo in such three-dimensionally confined environments requires changes in the mechanical properties of the surrounding cells using embryonic Drosophila melanogaster hemocytes, also called macrophages, as a model. We find that macrophage invasion into the germband through transient separation of the apposing ectoderm and mesoderm requires cell deformations and reductions in apical tension in the ectoderm. Interestingly, the genetic pathway governing these mechanical shifts acts downstream of the only known tumor necrosis factor superfamily member in Drosophila, Eiger, and its receptor, Grindelwald. Eiger-Grindelwald signaling reduces levels of active Myosin in the germband ectodermal cortex through the localization of a Crumbs complex component, Patj (Pals-1-associated tight junction protein). We therefore elucidate a distinct molecular pathway that controls tissue tension and demonstrate the importance of such regulation for invasive migration in vivo.},
  author       = {Ratheesh, Aparna and Biebl, Julia and Smutny, Michael and Veselá, Jana and Papusheva, Ekaterina and Krens, Gabriel and Kaufmann, Walter and György, Attila and Casano, Alessandra M and Siekhaus, Daria E},
  journal      = {Developmental Cell},
  number       = {3},
  pages        = {331 -- 346},
  publisher    = {Elsevier},
  title        = {{Drosophila TNF modulates tissue tension in the embryo to facilitate macrophage invasive migration}},
  doi          = {10.1016/j.devcel.2018.04.002},
  volume       = {45},
  year         = {2018},
}

@inproceedings{12905,
  author       = {Schlögl, Alois and Kiss, Janos},
  booktitle    = {AHPC17 – Austrian HPC Meeting 2017},
  location     = {Grundlsee, Austria},
  pages        = {28},
  publisher    = {FSP Scientific Computing},
  title        = {{Scientific Computing at IST Austria}},
  year         = {2017},
}

@article{825,
  abstract     = {What data is needed about data? Describing the process to answer this question for the institutional data repository IST DataRep.},
  author       = {Petritsch, Barbara},
  issn         = {10222588},
  journal      = {Mitteilungen der Vereinigung Österreichischer Bibliothekarinnen & Bibliothekare},
  number       = {2},
  pages        = {200 -- 207},
  publisher    = {VÖB},
  title        = {{Metadata for research data in practice}},
  doi          = {10.31263/voebm.v70i2.1678},
  volume       = {70},
  year         = {2017},
}

@article{693,
  abstract     = {Many central synapses contain a single presynaptic active zone and a single postsynaptic density. Vesicular release statistics at such “simple synapses” indicate that they contain a small complement of docking sites where vesicles repetitively dock and fuse. In this work, we investigate functional and morphological aspects of docking sites at simple synapses made between cerebellar parallel fibers and molecular layer interneurons. Using immunogold labeling of SDS-treated freeze-fracture replicas, we find that Cav2.1 channels form several clusters per active zone with about nine channels per cluster. The mean value and range of intersynaptic variation are similar for Cav2.1 cluster numbers and for functional estimates of docking-site numbers obtained from the maximum numbers of released vesicles per action potential. Both numbers grow in relation with synaptic size and decrease by a similar extent with age between 2 wk and 4 wk postnatal. Thus, the mean docking-site numbers were 3.15 at 2 wk (range: 1–10) and 2.03 at 4 wk (range: 1–4), whereas the mean numbers of Cav2.1 clusters were 2.84 at 2 wk (range: 1–8) and 2.37 at 4 wk (range: 1–5). These changes were accompanied by decreases of miniature current amplitude (from 93 pA to 56 pA), active-zone surface area (from 0.0427 μm2 to 0.0234 μm2), and initial success rate (from 0.609 to 0.353), indicating a tightening of synaptic transmission with development. Altogether, these results suggest a close correspondence between the number of functionally defined vesicular docking sites and that of clusters of voltage-gated calcium channels. },
  author       = {Miki, Takafumi and Kaufmann, Walter and Malagon, Gerardo and Gomez, Laura and Tabuchi, Katsuhiko and Watanabe, Masahiko and Shigemoto, Ryuichi and Marty, Alain},
  issn         = {00278424},
  journal      = {PNAS},
  number       = {26},
  pages        = {E5246 -- E5255},
  publisher    = {National Academy of Sciences},
  title        = {{Numbers of presynaptic Ca2+ channel clusters match those of functionally defined vesicular docking sites in single central synapses}},
  doi          = {10.1073/pnas.1704470114},
  volume       = {114},
  year         = {2017},
}

@article{727,
  abstract     = {Actin filaments polymerizing against membranes power endocytosis, vesicular traffic, and cell motility. In vitro reconstitution studies suggest that the structure and the dynamics of actin networks respond to mechanical forces. We demonstrate that lamellipodial actin of migrating cells responds to mechanical load when membrane tension is modulated. In a steady state, migrating cell filaments assume the canonical dendritic geometry, defined by Arp2/3-generated 70° branch points. Increased tension triggers a dense network with a broadened range of angles, whereas decreased tension causes a shift to a sparse configuration dominated by filaments growing perpendicularly to the plasma membrane. We show that these responses emerge from the geometry of branched actin: when load per filament decreases, elongation speed increases and perpendicular filaments gradually outcompete others because they polymerize the shortest distance to the membrane, where they are protected from capping. This network-intrinsic geometrical adaptation mechanism tunes protrusive force in response to mechanical load.},
  author       = {Mueller, Jan and Szep, Gregory and Nemethova, Maria and De Vries, Ingrid and Lieber, Arnon and Winkler, Christoph and Kruse, Karsten and Small, John and Schmeiser, Christian and Keren, Kinneret and Hauschild, Robert and Sixt, Michael K},
  issn         = {00928674},
  journal      = {Cell},
  number       = {1},
  pages        = {188 -- 200},
  publisher    = {Cell Press},
  title        = {{Load adaptation of lamellipodial actin networks}},
  doi          = {10.1016/j.cell.2017.07.051},
  volume       = {171},
  year         = {2017},
}

@article{807,
  abstract     = {On January the 1st, 2016 a new agreement between 32 Austrian scientific libraries and the publisher Springer took its effect: this deal covers accessing the licensed content on the one hand, and publishing open access on the other hand. More than 1000 papers by Austrian authors were published open access at Springer in the first year alone. The working group "Springer Compact Evaluierung" made the data for these articles available via the platform OpenAPC and would like to use this opportunity to give a short account of what this publishing agreement actually entails and the working group intends to do.},
  author       = {Andrae, Magdalena and Villányi, Márton},
  issn         = {10222588},
  journal      = {Mitteilungen der Vereinigung Österreichischer Bibliothekarinnen und Bibliothekare},
  number       = {2},
  pages        = {274 -- 280},
  publisher    = {VÖB},
  title        = {{Der Springer Compact-Deal – Ein erster Einblick in die Evaluierung einer Offsetting-Vereinbarung}},
  doi          = {10.31263/voebm.v70i2.1898},
  volume       = {70},
  year         = {2017},
}