---
_id: '713'
abstract:
- lang: eng
  text: To determine the dynamics of allelic-specific expression during mouse development,
    we analyzed RNA-seq data from 23 F1 tissues from different developmental stages,
    including 19 female tissues allowing X chromosome inactivation (XCI) escapers
    to also be detected. We demonstrate that allelic expression arising from genetic
    or epigenetic differences is highly tissue-specific. We find that tissue-specific
    strain-biased gene expression may be regulated by tissue-specific enhancers or
    by post-transcriptional differences in stability between the alleles. We also
    find that escape from X-inactivation is tissue-specific, with leg muscle showing
    an unexpectedly high rate of XCI escapers. By surveying a range of tissues during
    development, and performing extensive validation, we are able to provide a high
    confidence list of mouse imprinted genes including 18 novel genes. This shows
    that cluster size varies dynamically during development and can be substantially
    larger than previously thought, with the Igf2r cluster extending over 10 Mb in
    placenta.
article_number: e25125
author:
- first_name: Daniel
  full_name: Andergassen, Daniel
  last_name: Andergassen
- first_name: Christoph
  full_name: Dotter, Christoph
  id: 4C66542E-F248-11E8-B48F-1D18A9856A87
  last_name: Dotter
- first_name: Dyniel
  full_name: Wenzel, Dyniel
  last_name: Wenzel
- first_name: Verena
  full_name: Sigl, Verena
  last_name: Sigl
- first_name: Philipp
  full_name: Bammer, Philipp
  last_name: Bammer
- first_name: Markus
  full_name: Muckenhuber, Markus
  last_name: Muckenhuber
- first_name: Daniela
  full_name: Mayer, Daniela
  last_name: Mayer
- first_name: Tomasz
  full_name: Kulinski, Tomasz
  last_name: Kulinski
- first_name: Hans
  full_name: Theussl, Hans
  last_name: Theussl
- first_name: Josef
  full_name: Penninger, Josef
  last_name: Penninger
- first_name: Christoph
  full_name: Bock, Christoph
  last_name: Bock
- first_name: Denise
  full_name: Barlow, Denise
  last_name: Barlow
- first_name: Florian
  full_name: Pauler, Florian
  id: 48EA0138-F248-11E8-B48F-1D18A9856A87
  last_name: Pauler
- first_name: Quanah
  full_name: Hudson, Quanah
  last_name: Hudson
citation:
  ama: Andergassen D, Dotter C, Wenzel D, et al. Mapping the mouse Allelome reveals
    tissue specific regulation of allelic expression. <i>eLife</i>. 2017;6. doi:<a
    href="https://doi.org/10.7554/eLife.25125">10.7554/eLife.25125</a>
  apa: Andergassen, D., Dotter, C., Wenzel, D., Sigl, V., Bammer, P., Muckenhuber,
    M., … Hudson, Q. (2017). Mapping the mouse Allelome reveals tissue specific regulation
    of allelic expression. <i>ELife</i>. eLife Sciences Publications. <a href="https://doi.org/10.7554/eLife.25125">https://doi.org/10.7554/eLife.25125</a>
  chicago: Andergassen, Daniel, Christoph Dotter, Dyniel Wenzel, Verena Sigl, Philipp
    Bammer, Markus Muckenhuber, Daniela Mayer, et al. “Mapping the Mouse Allelome
    Reveals Tissue Specific Regulation of Allelic Expression.” <i>ELife</i>. eLife
    Sciences Publications, 2017. <a href="https://doi.org/10.7554/eLife.25125">https://doi.org/10.7554/eLife.25125</a>.
  ieee: D. Andergassen <i>et al.</i>, “Mapping the mouse Allelome reveals tissue specific
    regulation of allelic expression,” <i>eLife</i>, vol. 6. eLife Sciences Publications,
    2017.
  ista: Andergassen D, Dotter C, Wenzel D, Sigl V, Bammer P, Muckenhuber M, Mayer
    D, Kulinski T, Theussl H, Penninger J, Bock C, Barlow D, Pauler F, Hudson Q. 2017.
    Mapping the mouse Allelome reveals tissue specific regulation of allelic expression.
    eLife. 6, e25125.
  mla: Andergassen, Daniel, et al. “Mapping the Mouse Allelome Reveals Tissue Specific
    Regulation of Allelic Expression.” <i>ELife</i>, vol. 6, e25125, eLife Sciences
    Publications, 2017, doi:<a href="https://doi.org/10.7554/eLife.25125">10.7554/eLife.25125</a>.
  short: D. Andergassen, C. Dotter, D. Wenzel, V. Sigl, P. Bammer, M. Muckenhuber,
    D. Mayer, T. Kulinski, H. Theussl, J. Penninger, C. Bock, D. Barlow, F. Pauler,
    Q. Hudson, ELife 6 (2017).
date_created: 2018-12-11T11:48:05Z
date_published: 2017-08-14T00:00:00Z
date_updated: 2021-01-12T08:11:57Z
day: '14'
ddc:
- '576'
department:
- _id: GaNo
- _id: SiHi
doi: 10.7554/eLife.25125
file:
- access_level: open_access
  checksum: 1ace3462e64a971b9ead896091829549
  content_type: application/pdf
  creator: system
  date_created: 2018-12-12T10:13:36Z
  date_updated: 2020-07-14T12:47:50Z
  file_id: '5020'
  file_name: IST-2017-885-v1+1_elife-25125-figures-v2.pdf
  file_size: 6399510
  relation: main_file
- access_level: open_access
  checksum: 6241dc31eeb87b03facadec3a53a6827
  content_type: application/pdf
  creator: system
  date_created: 2018-12-12T10:13:36Z
  date_updated: 2020-07-14T12:47:50Z
  file_id: '5021'
  file_name: IST-2017-885-v1+2_elife-25125-v2.pdf
  file_size: 4264398
  relation: main_file
file_date_updated: 2020-07-14T12:47:50Z
has_accepted_license: '1'
intvolume: '         6'
language:
- iso: eng
month: '08'
oa: 1
oa_version: Published Version
project:
- _id: 25E9AF9E-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: P27201-B22
  name: Revealing the mechanisms underlying drug interactions
publication: eLife
publication_identifier:
  issn:
  - 2050084X
publication_status: published
publisher: eLife Sciences Publications
publist_id: '6971'
pubrep_id: '885'
quality_controlled: '1'
scopus_import: 1
status: public
title: Mapping the mouse Allelome reveals tissue specific regulation of allelic expression
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 6
year: '2017'
...
---
_id: '714'
abstract:
- lang: eng
  text: Background HIV-1 infection and drug abuse are frequently co-morbid and their
    association greatly increases the severity of HIV-1-induced neuropathology. While
    nucleus accumbens (NAcc) function is severely perturbed by drugs of abuse, little
    is known about how HIV-1 infection affects NAcc. Methods We used calcium and voltage
    imaging to investigate the effect of HIV-1 trans-activator of transcription (Tat)
    on rat NAcc. Based on previous neuronal studies, we hypothesized that Tat modulates
    intracellular Ca2+ homeostasis of NAcc neurons. Results We provide evidence that
    Tat triggers a Ca2+ signaling cascade in NAcc medium spiny neurons (MSN) expressing
    D1-like dopamine receptors leading to neuronal depolarization. Firstly, Tat induced
    inositol 1,4,5-trisphsophate (IP3) receptor-mediated Ca2+ release from endoplasmic
    reticulum, followed by Ca2+ and Na+ influx via transient receptor potential canonical
    channels. The influx of cations depolarizes the membrane promoting additional
    Ca2+ entry through voltage-gated P/Q-type Ca2+ channels and opening of tetrodotoxin-sensitive
    Na+ channels. By activating this mechanism, Tat elicits a feed-forward depolarization
    increasing the excitability of D1-phosphatidylinositol-linked NAcc MSN. We previously
    found that cocaine targets NAcc neurons directly (independent of the inhibition
    of dopamine transporter) only when IP3-generating mechanisms are concomitantly
    initiated. When tested here, cocaine produced a dose-dependent potentiation of
    the effect of Tat on cytosolic Ca2+. Conclusion We describe for the first time
    a HIV-1 Tat-triggered Ca2+ signaling in MSN of NAcc involving TRPC and depolarization
    and a potentiation of the effect of Tat by cocaine, which may be relevant for
    the reward axis in cocaine-abusing HIV-1-positive patients.
acknowledgement: This work was supported by the National Institutes of Health grants
  DA035926 (to MEA), and P30DA013429 (to EMU).
article_processing_charge: No
article_type: original
author:
- first_name: Gabriela
  full_name: Brailoiu, Gabriela
  last_name: Brailoiu
- first_name: Elena
  full_name: Deliu, Elena
  id: 37A40D7E-F248-11E8-B48F-1D18A9856A87
  last_name: Deliu
  orcid: 0000-0002-7370-5293
- first_name: Jeffrey
  full_name: Barr, Jeffrey
  last_name: Barr
- first_name: Linda
  full_name: Console Bram, Linda
  last_name: Console Bram
- first_name: Alexandra
  full_name: Ciuciu, Alexandra
  last_name: Ciuciu
- first_name: Mary
  full_name: Abood, Mary
  last_name: Abood
- first_name: Ellen
  full_name: Unterwald, Ellen
  last_name: Unterwald
- first_name: Eugen
  full_name: Brǎiloiu, Eugen
  last_name: Brǎiloiu
citation:
  ama: Brailoiu G, Deliu E, Barr J, et al. HIV Tat excites D1 receptor-like expressing
    neurons from rat nucleus accumbens. <i>Drug and Alcohol Dependence</i>. 2017;178:7-14.
    doi:<a href="https://doi.org/10.1016/j.drugalcdep.2017.04.015">10.1016/j.drugalcdep.2017.04.015</a>
  apa: Brailoiu, G., Deliu, E., Barr, J., Console Bram, L., Ciuciu, A., Abood, M.,
    … Brǎiloiu, E. (2017). HIV Tat excites D1 receptor-like expressing neurons from
    rat nucleus accumbens. <i>Drug and Alcohol Dependence</i>. Elsevier. <a href="https://doi.org/10.1016/j.drugalcdep.2017.04.015">https://doi.org/10.1016/j.drugalcdep.2017.04.015</a>
  chicago: Brailoiu, Gabriela, Elena Deliu, Jeffrey Barr, Linda Console Bram, Alexandra
    Ciuciu, Mary Abood, Ellen Unterwald, and Eugen Brǎiloiu. “HIV Tat Excites D1 Receptor-like
    Expressing Neurons from Rat Nucleus Accumbens.” <i>Drug and Alcohol Dependence</i>.
    Elsevier, 2017. <a href="https://doi.org/10.1016/j.drugalcdep.2017.04.015">https://doi.org/10.1016/j.drugalcdep.2017.04.015</a>.
  ieee: G. Brailoiu <i>et al.</i>, “HIV Tat excites D1 receptor-like expressing neurons
    from rat nucleus accumbens,” <i>Drug and Alcohol Dependence</i>, vol. 178. Elsevier,
    pp. 7–14, 2017.
  ista: Brailoiu G, Deliu E, Barr J, Console Bram L, Ciuciu A, Abood M, Unterwald
    E, Brǎiloiu E. 2017. HIV Tat excites D1 receptor-like expressing neurons from
    rat nucleus accumbens. Drug and Alcohol Dependence. 178, 7–14.
  mla: Brailoiu, Gabriela, et al. “HIV Tat Excites D1 Receptor-like Expressing Neurons
    from Rat Nucleus Accumbens.” <i>Drug and Alcohol Dependence</i>, vol. 178, Elsevier,
    2017, pp. 7–14, doi:<a href="https://doi.org/10.1016/j.drugalcdep.2017.04.015">10.1016/j.drugalcdep.2017.04.015</a>.
  short: G. Brailoiu, E. Deliu, J. Barr, L. Console Bram, A. Ciuciu, M. Abood, E.
    Unterwald, E. Brǎiloiu, Drug and Alcohol Dependence 178 (2017) 7–14.
date_created: 2018-12-11T11:48:05Z
date_published: 2017-09-01T00:00:00Z
date_updated: 2021-01-12T08:12:00Z
day: '01'
department:
- _id: GaNo
doi: 10.1016/j.drugalcdep.2017.04.015
external_id:
  pmid:
  - '28623807'
intvolume: '       178'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5797705
month: '09'
oa: 1
oa_version: Submitted Version
page: 7 - 14
pmid: 1
publication: Drug and Alcohol Dependence
publication_identifier:
  issn:
  - '03768716'
publication_status: published
publisher: Elsevier
publist_id: '6967'
quality_controlled: '1'
scopus_import: 1
status: public
title: HIV Tat excites D1 receptor-like expressing neurons from rat nucleus accumbens
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 178
year: '2017'
...
---
_id: '715'
abstract:
- lang: eng
  text: D-cycloserine ameliorates breathing abnormalities and survival rate in a mouse
    model of Rett syndrome.
article_number: aao4218
author:
- first_name: Gaia
  full_name: Novarino, Gaia
  id: 3E57A680-F248-11E8-B48F-1D18A9856A87
  last_name: Novarino
  orcid: 0000-0002-7673-7178
citation:
  ama: Novarino G. More excitation for Rett syndrome. <i>Science Translational Medicine</i>.
    2017;9(405). doi:<a href="https://doi.org/10.1126/scitranslmed.aao4218">10.1126/scitranslmed.aao4218</a>
  apa: Novarino, G. (2017). More excitation for Rett syndrome. <i>Science Translational
    Medicine</i>. American Association for the Advancement of Science. <a href="https://doi.org/10.1126/scitranslmed.aao4218">https://doi.org/10.1126/scitranslmed.aao4218</a>
  chicago: Novarino, Gaia. “More Excitation for Rett Syndrome.” <i>Science Translational
    Medicine</i>. American Association for the Advancement of Science, 2017. <a href="https://doi.org/10.1126/scitranslmed.aao4218">https://doi.org/10.1126/scitranslmed.aao4218</a>.
  ieee: G. Novarino, “More excitation for Rett syndrome,” <i>Science Translational
    Medicine</i>, vol. 9, no. 405. American Association for the Advancement of Science,
    2017.
  ista: Novarino G. 2017. More excitation for Rett syndrome. Science Translational
    Medicine. 9(405), aao4218.
  mla: Novarino, Gaia. “More Excitation for Rett Syndrome.” <i>Science Translational
    Medicine</i>, vol. 9, no. 405, aao4218, American Association for the Advancement
    of Science, 2017, doi:<a href="https://doi.org/10.1126/scitranslmed.aao4218">10.1126/scitranslmed.aao4218</a>.
  short: G. Novarino, Science Translational Medicine 9 (2017).
date_created: 2018-12-11T11:48:06Z
date_published: 2017-08-30T00:00:00Z
date_updated: 2021-01-12T08:12:04Z
day: '30'
department:
- _id: GaNo
doi: 10.1126/scitranslmed.aao4218
intvolume: '         9'
issue: '405'
language:
- iso: eng
month: '08'
oa_version: None
publication: Science Translational Medicine
publication_identifier:
  issn:
  - '19466234'
publication_status: published
publisher: American Association for the Advancement of Science
publist_id: '6968'
quality_controlled: '1'
scopus_import: 1
status: public
title: More excitation for Rett syndrome
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 9
year: '2017'
...
---
_id: '716'
abstract:
- lang: eng
  text: 'Two-player games on graphs are central in many problems in formal verification
    and program analysis, such as synthesis and verification of open systems. In this
    work, we consider solving recursive game graphs (or pushdown game graphs) that
    model the control flow of sequential programs with recursion.While pushdown games
    have been studied before with qualitative objectives-such as reachability and
    ?-regular objectives- in this work, we study for the first time such games with
    the most well-studied quantitative objective, the mean-payoff objective. In pushdown
    games, two types of strategies are relevant: (1) global strategies, which depend
    on the entire global history; and (2) modular strategies, which have only local
    memory and thus do not depend on the context of invocation but rather only on
    the history of the current invocation of the module. Our main results are as follows:
    (1) One-player pushdown games with mean-payoff objectives under global strategies
    are decidable in polynomial time. (2) Two-player pushdown games with mean-payoff
    objectives under global strategies are undecidable. (3) One-player pushdown games
    with mean-payoff objectives under modular strategies are NP-hard. (4) Two-player
    pushdown games with mean-payoff objectives under modular strategies can be solved
    in NP (i.e., both one-player and two-player pushdown games with mean-payoff objectives
    under modular strategies are NP-complete). We also establish the optimal strategy
    complexity by showing that global strategies for mean-payoff objectives require
    infinite memory even in one-player pushdown games and memoryless modular strategies
    are sufficient in two-player pushdown games. Finally, we also show that all the
    problems have the same complexity if the stack boundedness condition is added,
    where along with the mean-payoff objective the player must also ensure that the
    stack height is bounded.'
article_type: original
arxiv: 1
author:
- first_name: Krishnendu
  full_name: Chatterjee, Krishnendu
  id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
  last_name: Chatterjee
  orcid: 0000-0002-4561-241X
- first_name: Yaron
  full_name: Velner, Yaron
  last_name: Velner
citation:
  ama: Chatterjee K, Velner Y. The complexity of mean-payoff pushdown games. <i>Journal
    of the ACM</i>. 2017;64(5):34. doi:<a href="https://doi.org/10.1145/3121408">10.1145/3121408</a>
  apa: Chatterjee, K., &#38; Velner, Y. (2017). The complexity of mean-payoff pushdown
    games. <i>Journal of the ACM</i>. ACM. <a href="https://doi.org/10.1145/3121408">https://doi.org/10.1145/3121408</a>
  chicago: Chatterjee, Krishnendu, and Yaron Velner. “The Complexity of Mean-Payoff
    Pushdown Games.” <i>Journal of the ACM</i>. ACM, 2017. <a href="https://doi.org/10.1145/3121408">https://doi.org/10.1145/3121408</a>.
  ieee: K. Chatterjee and Y. Velner, “The complexity of mean-payoff pushdown games,”
    <i>Journal of the ACM</i>, vol. 64, no. 5. ACM, p. 34, 2017.
  ista: Chatterjee K, Velner Y. 2017. The complexity of mean-payoff pushdown games.
    Journal of the ACM. 64(5), 34.
  mla: Chatterjee, Krishnendu, and Yaron Velner. “The Complexity of Mean-Payoff Pushdown
    Games.” <i>Journal of the ACM</i>, vol. 64, no. 5, ACM, 2017, p. 34, doi:<a href="https://doi.org/10.1145/3121408">10.1145/3121408</a>.
  short: K. Chatterjee, Y. Velner, Journal of the ACM 64 (2017) 34.
date_created: 2018-12-11T11:48:06Z
date_published: 2017-09-01T00:00:00Z
date_updated: 2021-01-12T08:12:08Z
day: '01'
department:
- _id: KrCh
doi: 10.1145/3121408
ec_funded: 1
external_id:
  arxiv:
  - '1201.2829'
intvolume: '        64'
issue: '5'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1201.2829
month: '09'
oa: 1
oa_version: Preprint
page: '34'
project:
- _id: 2584A770-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: P 23499-N23
  name: Modern Graph Algorithmic Techniques in Formal Verification
- _id: 25863FF4-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: S11407
  name: Game Theory
- _id: 2581B60A-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '279307'
  name: 'Quantitative Graph Games: Theory and Applications'
publication: Journal of the ACM
publication_identifier:
  issn:
  - '00045411'
publication_status: published
publisher: ACM
publist_id: '6964'
quality_controlled: '1'
scopus_import: 1
status: public
title: The complexity of mean-payoff pushdown games
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 64
year: '2017'
...
---
_id: '7163'
abstract:
- lang: eng
  text: The de novo genome assemblies generated for this study, and the associated
    metadata.
article_processing_charge: No
author:
- first_name: Christelle
  full_name: Fraisse, Christelle
  id: 32DF5794-F248-11E8-B48F-1D18A9856A87
  last_name: Fraisse
  orcid: 0000-0001-8441-5075
citation:
  ama: Fraisse C. Supplementary Files for “The deep conservation of the Lepidoptera
    Z chromosome suggests a non canonical origin of the W.” 2017. doi:<a href="https://doi.org/10.15479/AT:ISTA:7163">10.15479/AT:ISTA:7163</a>
  apa: Fraisse, C. (2017). Supplementary Files for “The deep conservation of the Lepidoptera
    Z chromosome suggests a non canonical origin of the W.” Institute of Science and
    Technology Austria. <a href="https://doi.org/10.15479/AT:ISTA:7163">https://doi.org/10.15479/AT:ISTA:7163</a>
  chicago: Fraisse, Christelle. “Supplementary Files for ‘The Deep Conservation of
    the Lepidoptera Z Chromosome Suggests a Non Canonical Origin of the W.’” Institute
    of Science and Technology Austria, 2017. <a href="https://doi.org/10.15479/AT:ISTA:7163">https://doi.org/10.15479/AT:ISTA:7163</a>.
  ieee: C. Fraisse, “Supplementary Files for ‘The deep conservation of the Lepidoptera
    Z chromosome suggests a non canonical origin of the W.’” Institute of Science
    and Technology Austria, 2017.
  ista: Fraisse C. 2017. Supplementary Files for ‘The deep conservation of the Lepidoptera
    Z chromosome suggests a non canonical origin of the W’, Institute of Science and
    Technology Austria, <a href="https://doi.org/10.15479/AT:ISTA:7163">10.15479/AT:ISTA:7163</a>.
  mla: Fraisse, Christelle. <i>Supplementary Files for “The Deep Conservation of the
    Lepidoptera Z Chromosome Suggests a Non Canonical Origin of the W.”</i> Institute
    of Science and Technology Austria, 2017, doi:<a href="https://doi.org/10.15479/AT:ISTA:7163">10.15479/AT:ISTA:7163</a>.
  short: C. Fraisse, (2017).
contributor:
- first_name: Christelle
  id: 32DF5794-F248-11E8-B48F-1D18A9856A87
  last_name: Fraisse
  orcid: 0000-0001-8441-5075
- first_name: Marion A L
  id: 2C921A7A-F248-11E8-B48F-1D18A9856A87
  last_name: Picard
  orcid: 0000-0002-8101-2518
- first_name: Beatriz
  id: 49E1C5C6-F248-11E8-B48F-1D18A9856A87
  last_name: Vicoso
  orcid: 0000-0002-4579-8306
date_created: 2019-12-09T23:03:03Z
date_published: 2017-12-01T00:00:00Z
date_updated: 2024-02-21T13:47:47Z
day: '01'
ddc:
- '576'
department:
- _id: BeVi
- _id: NiBa
doi: 10.15479/AT:ISTA:7163
file:
- access_level: open_access
  checksum: 3cae8a2e3cbf8703399b9c483aaba7f3
  content_type: application/zip
  creator: cfraisse
  date_created: 2019-12-10T08:46:46Z
  date_updated: 2020-07-14T12:47:50Z
  file_id: '7164'
  file_name: Vicoso_Cohridella_Ndegeerella_Tsylvina_genome_assemblies.zip
  file_size: 841375478
  relation: main_file
file_date_updated: 2020-07-14T12:47:50Z
has_accepted_license: '1'
month: '12'
oa: 1
oa_version: Published Version
project:
- _id: 250ED89C-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: P28842-B22
  name: Sex chromosome evolution under male- and female- heterogamety
publisher: Institute of Science and Technology Austria
related_material:
  record:
  - id: '614'
    relation: research_paper
    status: public
status: public
title: Supplementary Files for "The deep conservation of the Lepidoptera Z chromosome
  suggests a non canonical origin of the W"
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: research_data
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
year: '2017'
...
---
_id: '717'
abstract:
- lang: eng
  text: 'We consider finite-state and recursive game graphs with multidimensional
    mean-payoff objectives. In recursive games two types of strategies are relevant:
    global strategies and modular strategies. Our contributions are: (1) We show that
    finite-state multidimensional mean-payoff games can be solved in polynomial time
    if the number of dimensions and the maximal absolute value of weights are fixed;
    whereas for arbitrary dimensions the problem is coNP-complete. (2) We show that
    one-player recursive games with multidimensional mean-payoff objectives can be
    solved in polynomial time. Both above algorithms are based on hyperplane separation
    technique. (3) For recursive games we show that under modular strategies the multidimensional
    problem is undecidable. We show that if the number of modules, exits, and the
    maximal absolute value of the weights are fixed, then one-dimensional recursive
    mean-payoff games under modular strategies can be solved in polynomial time, whereas
    for unbounded number of exits or modules the problem is NP-hard.'
acknowledgement: 'The research was supported by Austrian Science Fund (FWF) Grant
  No. P 23499-N23, FWF NFN Grant No. S11407-N23 (RiSE), ERC Start grant (279307: Graph
  Games), Microsoft faculty fellows award, the RICH Model Toolkit (ICT COST Action
  IC0901), and was carried out in partial fulfillment of the requirements for the
  Ph.D. degree of the second author.'
author:
- first_name: Krishnendu
  full_name: Chatterjee, Krishnendu
  id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
  last_name: Chatterjee
  orcid: 0000-0002-4561-241X
- first_name: Yaron
  full_name: Velner, Yaron
  last_name: Velner
citation:
  ama: Chatterjee K, Velner Y. Hyperplane separation technique for multidimensional
    mean-payoff games. <i>Journal of Computer and System Sciences</i>. 2017;88:236-259.
    doi:<a href="https://doi.org/10.1016/j.jcss.2017.04.005">10.1016/j.jcss.2017.04.005</a>
  apa: Chatterjee, K., &#38; Velner, Y. (2017). Hyperplane separation technique for
    multidimensional mean-payoff games. <i>Journal of Computer and System Sciences</i>.
    Academic Press. <a href="https://doi.org/10.1016/j.jcss.2017.04.005">https://doi.org/10.1016/j.jcss.2017.04.005</a>
  chicago: Chatterjee, Krishnendu, and Yaron Velner. “Hyperplane Separation Technique
    for Multidimensional Mean-Payoff Games.” <i>Journal of Computer and System Sciences</i>.
    Academic Press, 2017. <a href="https://doi.org/10.1016/j.jcss.2017.04.005">https://doi.org/10.1016/j.jcss.2017.04.005</a>.
  ieee: K. Chatterjee and Y. Velner, “Hyperplane separation technique for multidimensional
    mean-payoff games,” <i>Journal of Computer and System Sciences</i>, vol. 88. Academic
    Press, pp. 236–259, 2017.
  ista: Chatterjee K, Velner Y. 2017. Hyperplane separation technique for multidimensional
    mean-payoff games. Journal of Computer and System Sciences. 88, 236–259.
  mla: Chatterjee, Krishnendu, and Yaron Velner. “Hyperplane Separation Technique
    for Multidimensional Mean-Payoff Games.” <i>Journal of Computer and System Sciences</i>,
    vol. 88, Academic Press, 2017, pp. 236–59, doi:<a href="https://doi.org/10.1016/j.jcss.2017.04.005">10.1016/j.jcss.2017.04.005</a>.
  short: K. Chatterjee, Y. Velner, Journal of Computer and System Sciences 88 (2017)
    236–259.
date_created: 2018-12-11T11:48:07Z
date_published: 2017-09-01T00:00:00Z
date_updated: 2023-02-23T10:38:15Z
day: '01'
department:
- _id: KrCh
doi: 10.1016/j.jcss.2017.04.005
ec_funded: 1
intvolume: '        88'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1210.3141
month: '09'
oa: 1
oa_version: Preprint
page: 236 - 259
project:
- _id: 2584A770-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: P 23499-N23
  name: Modern Graph Algorithmic Techniques in Formal Verification
- _id: 25863FF4-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: S11407
  name: Game Theory
- _id: 2581B60A-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '279307'
  name: 'Quantitative Graph Games: Theory and Applications'
- _id: 2587B514-B435-11E9-9278-68D0E5697425
  name: Microsoft Research Faculty Fellowship
publication: Journal of Computer and System Sciences
publication_status: published
publisher: Academic Press
publist_id: '6963'
quality_controlled: '1'
related_material:
  record:
  - id: '2329'
    relation: earlier_version
    status: public
scopus_import: 1
status: public
title: Hyperplane separation technique for multidimensional mean-payoff games
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 88
year: '2017'
...
---
_id: '718'
abstract:
- lang: eng
  text: Mapping every simplex in the Delaunay mosaic of a discrete point set to the
    radius of the smallest empty circumsphere gives a generalized discrete Morse function.
    Choosing the points from a Poisson point process in ℝ n , we study the expected
    number of simplices in the Delaunay mosaic as well as the expected number of critical
    simplices and nonsingular intervals in the corresponding generalized discrete
    gradient. Observing connections with other probabilistic models, we obtain precise
    expressions for the expected numbers in low dimensions. In particular, we obtain
    the expected numbers of simplices in the Poisson–Delaunay mosaic in dimensions
    n ≤ 4.
arxiv: 1
author:
- first_name: Herbert
  full_name: Edelsbrunner, Herbert
  id: 3FB178DA-F248-11E8-B48F-1D18A9856A87
  last_name: Edelsbrunner
  orcid: 0000-0002-9823-6833
- first_name: Anton
  full_name: Nikitenko, Anton
  id: 3E4FF1BA-F248-11E8-B48F-1D18A9856A87
  last_name: Nikitenko
  orcid: 0000-0002-0659-3201
- first_name: Matthias
  full_name: Reitzner, Matthias
  last_name: Reitzner
citation:
  ama: Edelsbrunner H, Nikitenko A, Reitzner M. Expected sizes of poisson Delaunay
    mosaics and their discrete Morse functions. <i>Advances in Applied Probability</i>.
    2017;49(3):745-767. doi:<a href="https://doi.org/10.1017/apr.2017.20">10.1017/apr.2017.20</a>
  apa: Edelsbrunner, H., Nikitenko, A., &#38; Reitzner, M. (2017). Expected sizes
    of poisson Delaunay mosaics and their discrete Morse functions. <i>Advances in
    Applied Probability</i>. Cambridge University Press. <a href="https://doi.org/10.1017/apr.2017.20">https://doi.org/10.1017/apr.2017.20</a>
  chicago: Edelsbrunner, Herbert, Anton Nikitenko, and Matthias Reitzner. “Expected
    Sizes of Poisson Delaunay Mosaics and Their Discrete Morse Functions.” <i>Advances
    in Applied Probability</i>. Cambridge University Press, 2017. <a href="https://doi.org/10.1017/apr.2017.20">https://doi.org/10.1017/apr.2017.20</a>.
  ieee: H. Edelsbrunner, A. Nikitenko, and M. Reitzner, “Expected sizes of poisson
    Delaunay mosaics and their discrete Morse functions,” <i>Advances in Applied Probability</i>,
    vol. 49, no. 3. Cambridge University Press, pp. 745–767, 2017.
  ista: Edelsbrunner H, Nikitenko A, Reitzner M. 2017. Expected sizes of poisson Delaunay
    mosaics and their discrete Morse functions. Advances in Applied Probability. 49(3),
    745–767.
  mla: Edelsbrunner, Herbert, et al. “Expected Sizes of Poisson Delaunay Mosaics and
    Their Discrete Morse Functions.” <i>Advances in Applied Probability</i>, vol.
    49, no. 3, Cambridge University Press, 2017, pp. 745–67, doi:<a href="https://doi.org/10.1017/apr.2017.20">10.1017/apr.2017.20</a>.
  short: H. Edelsbrunner, A. Nikitenko, M. Reitzner, Advances in Applied Probability
    49 (2017) 745–767.
date_created: 2018-12-11T11:48:07Z
date_published: 2017-09-01T00:00:00Z
date_updated: 2023-09-07T12:07:12Z
day: '01'
department:
- _id: HeEd
doi: 10.1017/apr.2017.20
ec_funded: 1
external_id:
  arxiv:
  - '1607.05915'
intvolume: '        49'
issue: '3'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1607.05915
month: '09'
oa: 1
oa_version: Preprint
page: 745 - 767
project:
- _id: 255D761E-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '318493'
  name: Topological Complex Systems
- _id: 2561EBF4-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: I02979-N35
  name: Persistence and stability of geometric complexes
publication: Advances in Applied Probability
publication_identifier:
  issn:
  - '00018678'
publication_status: published
publisher: Cambridge University Press
publist_id: '6962'
quality_controlled: '1'
related_material:
  record:
  - id: '6287'
    relation: dissertation_contains
    status: public
scopus_import: 1
status: public
title: Expected sizes of poisson Delaunay mosaics and their discrete Morse functions
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 49
year: '2017'
...
---
_id: '719'
abstract:
- lang: eng
  text: 'The ubiquity of computation in modern machines and devices imposes a need
    to assert the correctness of their behavior. Especially in the case of safety-critical
    systems, their designers need to take measures that enforce their safe operation.
    Formal methods has emerged as a research field that addresses this challenge:
    by rigorously proving that all system executions adhere to their specifications,
    the correctness of an implementation under concern can be assured. To achieve
    this goal, a plethora of techniques are nowadays available, all of which are optimized
    for different system types and application domains.'
author:
- first_name: Krishnendu
  full_name: Chatterjee, Krishnendu
  id: 2E5DCA20-F248-11E8-B48F-1D18A9856A87
  last_name: Chatterjee
  orcid: 0000-0002-4561-241X
- first_name: Rüdiger
  full_name: Ehlers, Rüdiger
  last_name: Ehlers
citation:
  ama: 'Chatterjee K, Ehlers R. Special issue: Synthesis and SYNT 2014. <i>Acta Informatica</i>.
    2017;54(6):543-544. doi:<a href="https://doi.org/10.1007/s00236-017-0299-0">10.1007/s00236-017-0299-0</a>'
  apa: 'Chatterjee, K., &#38; Ehlers, R. (2017). Special issue: Synthesis and SYNT
    2014. <i>Acta Informatica</i>. Springer. <a href="https://doi.org/10.1007/s00236-017-0299-0">https://doi.org/10.1007/s00236-017-0299-0</a>'
  chicago: 'Chatterjee, Krishnendu, and Rüdiger Ehlers. “Special Issue: Synthesis
    and SYNT 2014.” <i>Acta Informatica</i>. Springer, 2017. <a href="https://doi.org/10.1007/s00236-017-0299-0">https://doi.org/10.1007/s00236-017-0299-0</a>.'
  ieee: 'K. Chatterjee and R. Ehlers, “Special issue: Synthesis and SYNT 2014,” <i>Acta
    Informatica</i>, vol. 54, no. 6. Springer, pp. 543–544, 2017.'
  ista: 'Chatterjee K, Ehlers R. 2017. Special issue: Synthesis and SYNT 2014. Acta
    Informatica. 54(6), 543–544.'
  mla: 'Chatterjee, Krishnendu, and Rüdiger Ehlers. “Special Issue: Synthesis and
    SYNT 2014.” <i>Acta Informatica</i>, vol. 54, no. 6, Springer, 2017, pp. 543–44,
    doi:<a href="https://doi.org/10.1007/s00236-017-0299-0">10.1007/s00236-017-0299-0</a>.'
  short: K. Chatterjee, R. Ehlers, Acta Informatica 54 (2017) 543–544.
date_created: 2018-12-11T11:48:07Z
date_published: 2017-09-01T00:00:00Z
date_updated: 2021-01-12T08:12:18Z
day: '01'
department:
- _id: KrCh
doi: 10.1007/s00236-017-0299-0
intvolume: '        54'
issue: '6'
language:
- iso: eng
month: '09'
oa_version: None
page: 543 - 544
publication: Acta Informatica
publication_identifier:
  issn:
  - '00015903'
publication_status: published
publisher: Springer
publist_id: '6961'
quality_controlled: '1'
scopus_import: 1
status: public
title: 'Special issue: Synthesis and SYNT 2014'
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 54
year: '2017'
...
---
_id: '720'
abstract:
- lang: eng
  text: 'Advances in multi-unit recordings pave the way for statistical modeling of
    activity patterns in large neural populations. Recent studies have shown that
    the summed activity of all neurons strongly shapes the population response. A
    separate recent finding has been that neural populations also exhibit criticality,
    an anomalously large dynamic range for the probabilities of different population
    activity patterns. Motivated by these two observations, we introduce a class of
    probabilistic models which takes into account the prior knowledge that the neural
    population could be globally coupled and close to critical. These models consist
    of an energy function which parametrizes interactions between small groups of
    neurons, and an arbitrary positive, strictly increasing, and twice differentiable
    function which maps the energy of a population pattern to its probability. We
    show that: 1) augmenting a pairwise Ising model with a nonlinearity yields an
    accurate description of the activity of retinal ganglion cells which outperforms
    previous models based on the summed activity of neurons; 2) prior knowledge that
    the population is critical translates to prior expectations about the shape of
    the nonlinearity; 3) the nonlinearity admits an interpretation in terms of a continuous
    latent variable globally coupling the system whose distribution we can infer from
    data. Our method is independent of the underlying system’s state space; hence,
    it can be applied to other systems such as natural scenes or amino acid sequences
    of proteins which are also known to exhibit criticality.'
article_number: e1005763
article_processing_charge: Yes
author:
- first_name: Jan
  full_name: Humplik, Jan
  id: 2E9627A8-F248-11E8-B48F-1D18A9856A87
  last_name: Humplik
- first_name: Gasper
  full_name: Tkacik, Gasper
  id: 3D494DCA-F248-11E8-B48F-1D18A9856A87
  last_name: Tkacik
  orcid: 0000-0002-6699-1455
citation:
  ama: Humplik J, Tkačik G. Probabilistic models for neural populations that naturally
    capture global coupling and criticality. <i>PLoS Computational Biology</i>. 2017;13(9).
    doi:<a href="https://doi.org/10.1371/journal.pcbi.1005763">10.1371/journal.pcbi.1005763</a>
  apa: Humplik, J., &#38; Tkačik, G. (2017). Probabilistic models for neural populations
    that naturally capture global coupling and criticality. <i>PLoS Computational
    Biology</i>. Public Library of Science. <a href="https://doi.org/10.1371/journal.pcbi.1005763">https://doi.org/10.1371/journal.pcbi.1005763</a>
  chicago: Humplik, Jan, and Gašper Tkačik. “Probabilistic Models for Neural Populations
    That Naturally Capture Global Coupling and Criticality.” <i>PLoS Computational
    Biology</i>. Public Library of Science, 2017. <a href="https://doi.org/10.1371/journal.pcbi.1005763">https://doi.org/10.1371/journal.pcbi.1005763</a>.
  ieee: J. Humplik and G. Tkačik, “Probabilistic models for neural populations that
    naturally capture global coupling and criticality,” <i>PLoS Computational Biology</i>,
    vol. 13, no. 9. Public Library of Science, 2017.
  ista: Humplik J, Tkačik G. 2017. Probabilistic models for neural populations that
    naturally capture global coupling and criticality. PLoS Computational Biology.
    13(9), e1005763.
  mla: Humplik, Jan, and Gašper Tkačik. “Probabilistic Models for Neural Populations
    That Naturally Capture Global Coupling and Criticality.” <i>PLoS Computational
    Biology</i>, vol. 13, no. 9, e1005763, Public Library of Science, 2017, doi:<a
    href="https://doi.org/10.1371/journal.pcbi.1005763">10.1371/journal.pcbi.1005763</a>.
  short: J. Humplik, G. Tkačik, PLoS Computational Biology 13 (2017).
date_created: 2018-12-11T11:48:08Z
date_published: 2017-09-19T00:00:00Z
date_updated: 2021-01-12T08:12:21Z
day: '19'
ddc:
- '530'
- '571'
department:
- _id: GaTk
doi: 10.1371/journal.pcbi.1005763
file:
- access_level: open_access
  checksum: 81107096c19771c36ddbe6f0282a3acb
  content_type: application/pdf
  creator: system
  date_created: 2018-12-12T10:18:30Z
  date_updated: 2020-07-14T12:47:53Z
  file_id: '5352'
  file_name: IST-2017-884-v1+1_journal.pcbi.1005763.pdf
  file_size: 14167050
  relation: main_file
file_date_updated: 2020-07-14T12:47:53Z
has_accepted_license: '1'
intvolume: '        13'
issue: '9'
language:
- iso: eng
month: '09'
oa: 1
oa_version: Published Version
project:
- _id: 255008E4-B435-11E9-9278-68D0E5697425
  grant_number: RGP0065/2012
  name: Information processing and computation in fish groups
- _id: 254D1A94-B435-11E9-9278-68D0E5697425
  call_identifier: FWF
  grant_number: P 25651-N26
  name: Sensitivity to higher-order statistics in natural scenes
publication: PLoS Computational Biology
publication_identifier:
  issn:
  - 1553734X
publication_status: published
publisher: Public Library of Science
publist_id: '6960'
pubrep_id: '884'
quality_controlled: '1'
scopus_import: 1
status: public
title: Probabilistic models for neural populations that naturally capture global coupling
  and criticality
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 13
year: '2017'
...
---
_id: '721'
abstract:
- lang: eng
  text: 'Let S be a positivity-preserving symmetric linear operator acting on bounded
    functions. The nonlinear equation -1/m=z+Sm with a parameter z in the complex
    upper half-plane ℍ has a unique solution m with values in ℍ. We show that the
    z-dependence of this solution can be represented as the Stieltjes transforms of
    a family of probability measures v on ℝ. Under suitable conditions on S, we show
    that v has a real analytic density apart from finitely many algebraic singularities
    of degree at most 3. Our motivation comes from large random matrices. The solution
    m determines the density of eigenvalues of two prominent matrix ensembles: (i)
    matrices with centered independent entries whose variances are given by S and
    (ii) matrices with correlated entries with a translation-invariant correlation
    structure. Our analysis shows that the limiting eigenvalue density has only square
    root singularities or cubic root cusps; no other singularities occur.'
author:
- first_name: Oskari H
  full_name: Ajanki, Oskari H
  id: 36F2FB7E-F248-11E8-B48F-1D18A9856A87
  last_name: Ajanki
- first_name: Torben H
  full_name: Krüger, Torben H
  id: 3020C786-F248-11E8-B48F-1D18A9856A87
  last_name: Krüger
  orcid: 0000-0002-4821-3297
- first_name: László
  full_name: Erdös, László
  id: 4DBD5372-F248-11E8-B48F-1D18A9856A87
  last_name: Erdös
  orcid: 0000-0001-5366-9603
citation:
  ama: Ajanki OH, Krüger TH, Erdös L. Singularities of solutions to quadratic vector
    equations on the complex upper half plane. <i>Communications on Pure and Applied
    Mathematics</i>. 2017;70(9):1672-1705. doi:<a href="https://doi.org/10.1002/cpa.21639">10.1002/cpa.21639</a>
  apa: Ajanki, O. H., Krüger, T. H., &#38; Erdös, L. (2017). Singularities of solutions
    to quadratic vector equations on the complex upper half plane. <i>Communications
    on Pure and Applied Mathematics</i>. Wiley-Blackwell. <a href="https://doi.org/10.1002/cpa.21639">https://doi.org/10.1002/cpa.21639</a>
  chicago: Ajanki, Oskari H, Torben H Krüger, and László Erdös. “Singularities of
    Solutions to Quadratic Vector Equations on the Complex Upper Half Plane.” <i>Communications
    on Pure and Applied Mathematics</i>. Wiley-Blackwell, 2017. <a href="https://doi.org/10.1002/cpa.21639">https://doi.org/10.1002/cpa.21639</a>.
  ieee: O. H. Ajanki, T. H. Krüger, and L. Erdös, “Singularities of solutions to quadratic
    vector equations on the complex upper half plane,” <i>Communications on Pure and
    Applied Mathematics</i>, vol. 70, no. 9. Wiley-Blackwell, pp. 1672–1705, 2017.
  ista: Ajanki OH, Krüger TH, Erdös L. 2017. Singularities of solutions to quadratic
    vector equations on the complex upper half plane. Communications on Pure and Applied
    Mathematics. 70(9), 1672–1705.
  mla: Ajanki, Oskari H., et al. “Singularities of Solutions to Quadratic Vector Equations
    on the Complex Upper Half Plane.” <i>Communications on Pure and Applied Mathematics</i>,
    vol. 70, no. 9, Wiley-Blackwell, 2017, pp. 1672–705, doi:<a href="https://doi.org/10.1002/cpa.21639">10.1002/cpa.21639</a>.
  short: O.H. Ajanki, T.H. Krüger, L. Erdös, Communications on Pure and Applied Mathematics
    70 (2017) 1672–1705.
date_created: 2018-12-11T11:48:08Z
date_published: 2017-09-01T00:00:00Z
date_updated: 2021-01-12T08:12:24Z
day: '01'
department:
- _id: LaEr
doi: 10.1002/cpa.21639
ec_funded: 1
intvolume: '        70'
issue: '9'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1512.03703
month: '09'
oa: 1
oa_version: Submitted Version
page: 1672 - 1705
project:
- _id: 258DCDE6-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '338804'
  name: Random matrices, universality and disordered quantum systems
publication: Communications on Pure and Applied Mathematics
publication_identifier:
  issn:
  - '00103640'
publication_status: published
publisher: Wiley-Blackwell
publist_id: '6959'
quality_controlled: '1'
scopus_import: 1
status: public
title: Singularities of solutions to quadratic vector equations on the complex upper
  half plane
type: journal_article
user_id: 4435EBFC-F248-11E8-B48F-1D18A9856A87
volume: 70
year: '2017'
...
---
_id: '722'
abstract:
- lang: eng
  text: Plants are sessile organisms rooted in one place. The soil resources that
    plants require are often distributed in a highly heterogeneous pattern. To aid
    foraging, plants have evolved roots whose growth and development are highly responsive
    to soil signals. As a result, 3D root architecture is shaped by myriad environmental
    signals to ensure resource capture is optimised and unfavourable environments
    are avoided. The first signals sensed by newly germinating seeds — gravity and
    light — direct root growth into the soil to aid seedling establishment. Heterogeneous
    soil resources, such as water, nitrogen and phosphate, also act as signals that
    shape 3D root growth to optimise uptake. Root architecture is also modified through
    biotic interactions that include soil fungi and neighbouring plants. This developmental
    plasticity results in a ‘custom-made’ 3D root system that is best adapted to forage
    for resources in each soil environment that a plant colonises.
author:
- first_name: Emily
  full_name: Morris, Emily
  last_name: Morris
- first_name: Marcus
  full_name: Griffiths, Marcus
  last_name: Griffiths
- first_name: Agata
  full_name: Golebiowska, Agata
  last_name: Golebiowska
- first_name: Stefan
  full_name: Mairhofer, Stefan
  last_name: Mairhofer
- first_name: Jasmine
  full_name: Burr Hersey, Jasmine
  last_name: Burr Hersey
- first_name: Tatsuaki
  full_name: Goh, Tatsuaki
  last_name: Goh
- first_name: Daniel
  full_name: Von Wangenheim, Daniel
  id: 49E91952-F248-11E8-B48F-1D18A9856A87
  last_name: Von Wangenheim
  orcid: 0000-0002-6862-1247
- first_name: Brian
  full_name: Atkinson, Brian
  last_name: Atkinson
- first_name: Craig
  full_name: Sturrock, Craig
  last_name: Sturrock
- first_name: Jonathan
  full_name: Lynch, Jonathan
  last_name: Lynch
- first_name: Kris
  full_name: Vissenberg, Kris
  last_name: Vissenberg
- first_name: Karl
  full_name: Ritz, Karl
  last_name: Ritz
- first_name: Darren
  full_name: Wells, Darren
  last_name: Wells
- first_name: Sacha
  full_name: Mooney, Sacha
  last_name: Mooney
- first_name: Malcolm
  full_name: Bennett, Malcolm
  last_name: Bennett
citation:
  ama: Morris E, Griffiths M, Golebiowska A, et al. Shaping 3D root system architecture.
    <i>Current Biology</i>. 2017;27(17):R919-R930. doi:<a href="https://doi.org/10.1016/j.cub.2017.06.043">10.1016/j.cub.2017.06.043</a>
  apa: Morris, E., Griffiths, M., Golebiowska, A., Mairhofer, S., Burr Hersey, J.,
    Goh, T., … Bennett, M. (2017). Shaping 3D root system architecture. <i>Current
    Biology</i>. Cell Press. <a href="https://doi.org/10.1016/j.cub.2017.06.043">https://doi.org/10.1016/j.cub.2017.06.043</a>
  chicago: Morris, Emily, Marcus Griffiths, Agata Golebiowska, Stefan Mairhofer, Jasmine
    Burr Hersey, Tatsuaki Goh, Daniel von Wangenheim, et al. “Shaping 3D Root System
    Architecture.” <i>Current Biology</i>. Cell Press, 2017. <a href="https://doi.org/10.1016/j.cub.2017.06.043">https://doi.org/10.1016/j.cub.2017.06.043</a>.
  ieee: E. Morris <i>et al.</i>, “Shaping 3D root system architecture,” <i>Current
    Biology</i>, vol. 27, no. 17. Cell Press, pp. R919–R930, 2017.
  ista: Morris E, Griffiths M, Golebiowska A, Mairhofer S, Burr Hersey J, Goh T, von
    Wangenheim D, Atkinson B, Sturrock C, Lynch J, Vissenberg K, Ritz K, Wells D,
    Mooney S, Bennett M. 2017. Shaping 3D root system architecture. Current Biology.
    27(17), R919–R930.
  mla: Morris, Emily, et al. “Shaping 3D Root System Architecture.” <i>Current Biology</i>,
    vol. 27, no. 17, Cell Press, 2017, pp. R919–30, doi:<a href="https://doi.org/10.1016/j.cub.2017.06.043">10.1016/j.cub.2017.06.043</a>.
  short: E. Morris, M. Griffiths, A. Golebiowska, S. Mairhofer, J. Burr Hersey, T.
    Goh, D. von Wangenheim, B. Atkinson, C. Sturrock, J. Lynch, K. Vissenberg, K.
    Ritz, D. Wells, S. Mooney, M. Bennett, Current Biology 27 (2017) R919–R930.
date_created: 2018-12-11T11:48:08Z
date_published: 2017-09-11T00:00:00Z
date_updated: 2021-01-12T08:12:29Z
day: '11'
ddc:
- '581'
department:
- _id: JiFr
doi: 10.1016/j.cub.2017.06.043
ec_funded: 1
external_id:
  pmid:
  - '28898665'
file:
- access_level: open_access
  checksum: e45588b21097b408da6276a3e5eedb2e
  content_type: application/pdf
  creator: dernst
  date_created: 2019-04-17T07:46:40Z
  date_updated: 2020-07-14T12:47:54Z
  file_id: '6332'
  file_name: 2017_CurrentBiology_Morris.pdf
  file_size: 1576593
  relation: main_file
file_date_updated: 2020-07-14T12:47:54Z
has_accepted_license: '1'
intvolume: '        27'
issue: '17'
language:
- iso: eng
month: '09'
oa: 1
oa_version: Submitted Version
page: R919 - R930
pmid: 1
project:
- _id: 25681D80-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '291734'
  name: International IST Postdoc Fellowship Programme
publication: Current Biology
publication_identifier:
  issn:
  - '09609822'
publication_status: published
publisher: Cell Press
publist_id: '6956'
pubrep_id: '982'
quality_controlled: '1'
scopus_import: 1
status: public
title: Shaping 3D root system architecture
tmp:
  image: /images/cc_by_nc_nd.png
  legal_code_url: https://creativecommons.org/licenses/by-nc-nd/4.0/legalcode
  name: Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International
    (CC BY-NC-ND 4.0)
  short: CC BY-NC-ND (4.0)
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 27
year: '2017'
...
---
_id: '724'
abstract:
- lang: eng
  text: We investigate the stationary and dynamical behavior of an Anderson localized
    chain coupled to a single central bound state. Although this coupling partially
    dilutes the Anderson localized peaks towards nearly resonant sites, the most weight
    of the original peaks remains unchanged. This leads to multifractal wave functions
    with a frozen spectrum of fractal dimensions, which is characteristic for localized
    phases in models with power-law hopping. Using a perturbative approach we identify
    two different dynamical regimes. At weak couplings to the central site, the transport
    of particles and information is logarithmic in time, a feature usually attributed
    to many-body localization. We connect such transport to the persistence of the
    Poisson statistics of level spacings in parts of the spectrum. In contrast, at
    stronger couplings the level repulsion is established in the entire spectrum,
    the problem can be mapped to the Fano resonance, and the transport is ballistic.
acknowledgement: "We  would  like  to  thank  Dmitry  Abanin,  Christophe  De\r\nBeule,
  \ Joel  Moore,  Romain  Vasseur,  and  Norman  Yao  for\r\nmany  stimulating  discussions.
  \ Financial  support  has  been\r\nprovided  by  the  Deutsche  Forschungsgemeinschaft
  \ (DFG)\r\nvia Grant No. TR950/8-1, SFB 1170 “ToCoTronics” and the\r\nENB  Graduate
  \ School  on  Topological  Insulators.  M.S.  was\r\nsupported by Gordon and Betty
  Moore Foundation’s EPiQS\r\nInitiative through Grant No. GBMF4307. F.P. acknowledges\r\nsupport
  from the DFG Research Unit FOR 1807 through Grant\r\nNo. PO 1370/2-1."
article_number: '104203'
author:
- first_name: Daniel
  full_name: Hetterich, Daniel
  last_name: Hetterich
- first_name: Maksym
  full_name: Serbyn, Maksym
  id: 47809E7E-F248-11E8-B48F-1D18A9856A87
  last_name: Serbyn
  orcid: 0000-0002-2399-5827
- first_name: Fernando
  full_name: Domínguez, Fernando
  last_name: Domínguez
- first_name: Frank
  full_name: Pollmann, Frank
  last_name: Pollmann
- first_name: Björn
  full_name: Trauzettel, Björn
  last_name: Trauzettel
citation:
  ama: Hetterich D, Serbyn M, Domínguez F, Pollmann F, Trauzettel B. Noninteracting
    central site model localization and logarithmic entanglement growth. <i>Physical
    Review B</i>. 2017;96(10). doi:<a href="https://doi.org/10.1103/PhysRevB.96.104203">10.1103/PhysRevB.96.104203</a>
  apa: Hetterich, D., Serbyn, M., Domínguez, F., Pollmann, F., &#38; Trauzettel, B.
    (2017). Noninteracting central site model localization and logarithmic entanglement
    growth. <i>Physical Review B</i>. American Physical Society. <a href="https://doi.org/10.1103/PhysRevB.96.104203">https://doi.org/10.1103/PhysRevB.96.104203</a>
  chicago: Hetterich, Daniel, Maksym Serbyn, Fernando Domínguez, Frank Pollmann, and
    Björn Trauzettel. “Noninteracting Central Site Model Localization and Logarithmic
    Entanglement Growth.” <i>Physical Review B</i>. American Physical Society, 2017.
    <a href="https://doi.org/10.1103/PhysRevB.96.104203">https://doi.org/10.1103/PhysRevB.96.104203</a>.
  ieee: D. Hetterich, M. Serbyn, F. Domínguez, F. Pollmann, and B. Trauzettel, “Noninteracting
    central site model localization and logarithmic entanglement growth,” <i>Physical
    Review B</i>, vol. 96, no. 10. American Physical Society, 2017.
  ista: Hetterich D, Serbyn M, Domínguez F, Pollmann F, Trauzettel B. 2017. Noninteracting
    central site model localization and logarithmic entanglement growth. Physical
    Review B. 96(10), 104203.
  mla: Hetterich, Daniel, et al. “Noninteracting Central Site Model Localization and
    Logarithmic Entanglement Growth.” <i>Physical Review B</i>, vol. 96, no. 10, 104203,
    American Physical Society, 2017, doi:<a href="https://doi.org/10.1103/PhysRevB.96.104203">10.1103/PhysRevB.96.104203</a>.
  short: D. Hetterich, M. Serbyn, F. Domínguez, F. Pollmann, B. Trauzettel, Physical
    Review B 96 (2017).
date_created: 2018-12-11T11:48:09Z
date_published: 2017-09-13T00:00:00Z
date_updated: 2021-01-12T08:12:35Z
day: '13'
department:
- _id: MaSe
doi: 10.1103/PhysRevB.96.104203
intvolume: '        96'
issue: '10'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1701.02744
month: '09'
oa: 1
oa_version: Submitted Version
publication: Physical Review B
publication_identifier:
  issn:
  - '24699950'
publication_status: published
publisher: American Physical Society
publist_id: '6955'
quality_controlled: '1'
scopus_import: 1
status: public
title: Noninteracting central site model localization and logarithmic entanglement
  growth
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 96
year: '2017'
...
---
_id: '725'
abstract:
- lang: eng
  text: Individual computations and social interactions underlying collective behavior
    in groups of animals are of great ethological, behavioral, and theoretical interest.
    While complex individual behaviors have successfully been parsed into small dictionaries
    of stereotyped behavioral modes, studies of collective behavior largely ignored
    these findings; instead, their focus was on inferring single, mode-independent
    social interaction rules that reproduced macroscopic and often qualitative features
    of group behavior. Here, we bring these two approaches together to predict individual
    swimming patterns of adult zebrafish in a group. We show that fish alternate between
    an “active” mode, in which they are sensitive to the swimming patterns of conspecifics,
    and a “passive” mode, where they ignore them. Using a model that accounts for
    these two modes explicitly, we predict behaviors of individual fish with high
    accuracy, outperforming previous approaches that assumed a single continuous computation
    by individuals and simple metric or topological weighing of neighbors’ behavior.
    At the group level, switching between active and passive modes is uncorrelated
    among fish, but correlated directional swimming behavior still emerges. Our quantitative
    approach for studying complex, multi-modal individual behavior jointly with emergent
    group behavior is readily extensible to additional behavioral modes and their
    neural correlates as well as to other species.
author:
- first_name: Roy
  full_name: Harpaz, Roy
  last_name: Harpaz
- first_name: Gasper
  full_name: Tkacik, Gasper
  id: 3D494DCA-F248-11E8-B48F-1D18A9856A87
  last_name: Tkacik
  orcid: 0000-0002-6699-1455
- first_name: Elad
  full_name: Schneidman, Elad
  last_name: Schneidman
citation:
  ama: Harpaz R, Tkačik G, Schneidman E. Discrete modes of social information processing
    predict individual behavior of fish in a group. <i>PNAS</i>. 2017;114(38):10149-10154.
    doi:<a href="https://doi.org/10.1073/pnas.1703817114">10.1073/pnas.1703817114</a>
  apa: Harpaz, R., Tkačik, G., &#38; Schneidman, E. (2017). Discrete modes of social
    information processing predict individual behavior of fish in a group. <i>PNAS</i>.
    National Academy of Sciences. <a href="https://doi.org/10.1073/pnas.1703817114">https://doi.org/10.1073/pnas.1703817114</a>
  chicago: Harpaz, Roy, Gašper Tkačik, and Elad Schneidman. “Discrete Modes of Social
    Information Processing Predict Individual Behavior of Fish in a Group.” <i>PNAS</i>.
    National Academy of Sciences, 2017. <a href="https://doi.org/10.1073/pnas.1703817114">https://doi.org/10.1073/pnas.1703817114</a>.
  ieee: R. Harpaz, G. Tkačik, and E. Schneidman, “Discrete modes of social information
    processing predict individual behavior of fish in a group,” <i>PNAS</i>, vol.
    114, no. 38. National Academy of Sciences, pp. 10149–10154, 2017.
  ista: Harpaz R, Tkačik G, Schneidman E. 2017. Discrete modes of social information
    processing predict individual behavior of fish in a group. PNAS. 114(38), 10149–10154.
  mla: Harpaz, Roy, et al. “Discrete Modes of Social Information Processing Predict
    Individual Behavior of Fish in a Group.” <i>PNAS</i>, vol. 114, no. 38, National
    Academy of Sciences, 2017, pp. 10149–54, doi:<a href="https://doi.org/10.1073/pnas.1703817114">10.1073/pnas.1703817114</a>.
  short: R. Harpaz, G. Tkačik, E. Schneidman, PNAS 114 (2017) 10149–10154.
date_created: 2018-12-11T11:48:10Z
date_published: 2017-09-19T00:00:00Z
date_updated: 2021-01-12T08:12:36Z
day: '19'
department:
- _id: GaTk
doi: 10.1073/pnas.1703817114
external_id:
  pmid:
  - '28874581'
intvolume: '       114'
issue: '38'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5617265/
month: '09'
oa: 1
oa_version: Submitted Version
page: 10149 - 10154
pmid: 1
publication: PNAS
publication_identifier:
  issn:
  - '00278424'
publication_status: published
publisher: National Academy of Sciences
publist_id: '6953'
quality_controlled: '1'
scopus_import: 1
status: public
title: Discrete modes of social information processing predict individual behavior
  of fish in a group
type: journal_article
user_id: 2DF688A6-F248-11E8-B48F-1D18A9856A87
volume: 114
year: '2017'
...
---
_id: '726'
abstract:
- lang: eng
  text: The morphogenesis of branched organs remains a subject of abiding interest.
    Although much is known about the underlying signaling pathways, it remains unclear
    how macroscopic features of branched organs, including their size, network topology,
    and spatial patterning, are encoded. Here, we show that, in mouse mammary gland,
    kidney, and human prostate, these features can be explained quantitatively within
    a single unifying framework of branching and annihilating random walks. Based
    on quantitative analyses of large-scale organ reconstructions and proliferation
    kinetics measurements, we propose that morphogenesis follows from the proliferative
    activity of equipotent tips that stochastically branch and randomly explore their
    environment but compete neutrally for space, becoming proliferatively inactive
    when in proximity with neighboring ducts. These results show that complex branched
    epithelial structures develop as a self-organized process, reliant upon a strikingly
    simple but generic rule, without recourse to a rigid and deterministic sequence
    of genetically programmed events.
article_processing_charge: No
author:
- first_name: Edouard B
  full_name: Hannezo, Edouard B
  id: 3A9DB764-F248-11E8-B48F-1D18A9856A87
  last_name: Hannezo
  orcid: 0000-0001-6005-1561
- first_name: Colinda
  full_name: Scheele, Colinda
  last_name: Scheele
- first_name: Mohammad
  full_name: Moad, Mohammad
  last_name: Moad
- first_name: Nicholas
  full_name: Drogo, Nicholas
  last_name: Drogo
- first_name: Rakesh
  full_name: Heer, Rakesh
  last_name: Heer
- first_name: Rosemary
  full_name: Sampogna, Rosemary
  last_name: Sampogna
- first_name: Jacco
  full_name: Van Rheenen, Jacco
  last_name: Van Rheenen
- first_name: Benjamin
  full_name: Simons, Benjamin
  last_name: Simons
citation:
  ama: Hannezo EB, Scheele C, Moad M, et al. A unifying theory of branching morphogenesis.
    <i>Cell</i>. 2017;171(1):242-255. doi:<a href="https://doi.org/10.1016/j.cell.2017.08.026">10.1016/j.cell.2017.08.026</a>
  apa: Hannezo, E. B., Scheele, C., Moad, M., Drogo, N., Heer, R., Sampogna, R., …
    Simons, B. (2017). A unifying theory of branching morphogenesis. <i>Cell</i>.
    Cell Press. <a href="https://doi.org/10.1016/j.cell.2017.08.026">https://doi.org/10.1016/j.cell.2017.08.026</a>
  chicago: Hannezo, Edouard B, Colinda Scheele, Mohammad Moad, Nicholas Drogo, Rakesh
    Heer, Rosemary Sampogna, Jacco Van Rheenen, and Benjamin Simons. “A Unifying Theory
    of Branching Morphogenesis.” <i>Cell</i>. Cell Press, 2017. <a href="https://doi.org/10.1016/j.cell.2017.08.026">https://doi.org/10.1016/j.cell.2017.08.026</a>.
  ieee: E. B. Hannezo <i>et al.</i>, “A unifying theory of branching morphogenesis,”
    <i>Cell</i>, vol. 171, no. 1. Cell Press, pp. 242–255, 2017.
  ista: Hannezo EB, Scheele C, Moad M, Drogo N, Heer R, Sampogna R, Van Rheenen J,
    Simons B. 2017. A unifying theory of branching morphogenesis. Cell. 171(1), 242–255.
  mla: Hannezo, Edouard B., et al. “A Unifying Theory of Branching Morphogenesis.”
    <i>Cell</i>, vol. 171, no. 1, Cell Press, 2017, pp. 242–55, doi:<a href="https://doi.org/10.1016/j.cell.2017.08.026">10.1016/j.cell.2017.08.026</a>.
  short: E.B. Hannezo, C. Scheele, M. Moad, N. Drogo, R. Heer, R. Sampogna, J. Van
    Rheenen, B. Simons, Cell 171 (2017) 242–255.
date_created: 2018-12-11T11:48:10Z
date_published: 2017-09-21T00:00:00Z
date_updated: 2023-09-28T11:34:17Z
day: '21'
ddc:
- '539'
department:
- _id: EdHa
doi: 10.1016/j.cell.2017.08.026
external_id:
  isi:
  - '000411331800024'
file:
- access_level: open_access
  checksum: 7a036d93a9e2e597af9bb504d6133aca
  content_type: application/pdf
  creator: system
  date_created: 2018-12-12T10:11:17Z
  date_updated: 2020-07-14T12:47:55Z
  file_id: '4870'
  file_name: IST-2017-883-v1+1_PIIS0092867417309510.pdf
  file_size: 12670204
  relation: main_file
file_date_updated: 2020-07-14T12:47:55Z
has_accepted_license: '1'
intvolume: '       171'
isi: 1
issue: '1'
language:
- iso: eng
month: '09'
oa: 1
oa_version: Published Version
page: 242 - 255
publication: Cell
publication_identifier:
  issn:
  - '00928674'
publication_status: published
publisher: Cell Press
publist_id: '6952'
pubrep_id: '883'
quality_controlled: '1'
scopus_import: '1'
status: public
title: A unifying theory of branching morphogenesis
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 171
year: '2017'
...
---
_id: '727'
abstract:
- lang: eng
  text: 'Actin filaments polymerizing against membranes power endocytosis, vesicular
    traffic, and cell motility. In vitro reconstitution studies suggest that the structure
    and the dynamics of actin networks respond to mechanical forces. We demonstrate
    that lamellipodial actin of migrating cells responds to mechanical load when membrane
    tension is modulated. In a steady state, migrating cell filaments assume the canonical
    dendritic geometry, defined by Arp2/3-generated 70° branch points. Increased tension
    triggers a dense network with a broadened range of angles, whereas decreased tension
    causes a shift to a sparse configuration dominated by filaments growing perpendicularly
    to the plasma membrane. We show that these responses emerge from the geometry
    of branched actin: when load per filament decreases, elongation speed increases
    and perpendicular filaments gradually outcompete others because they polymerize
    the shortest distance to the membrane, where they are protected from capping.
    This network-intrinsic geometrical adaptation mechanism tunes protrusive force
    in response to mechanical load.'
acknowledged_ssus:
- _id: ScienComp
article_processing_charge: No
author:
- first_name: Jan
  full_name: Mueller, Jan
  last_name: Mueller
- first_name: Gregory
  full_name: Szep, Gregory
  id: 4BFB7762-F248-11E8-B48F-1D18A9856A87
  last_name: Szep
- first_name: Maria
  full_name: Nemethova, Maria
  id: 34E27F1C-F248-11E8-B48F-1D18A9856A87
  last_name: Nemethova
- first_name: Ingrid
  full_name: De Vries, Ingrid
  id: 4C7D837E-F248-11E8-B48F-1D18A9856A87
  last_name: De Vries
- first_name: Arnon
  full_name: Lieber, Arnon
  last_name: Lieber
- first_name: Christoph
  full_name: Winkler, Christoph
  last_name: Winkler
- first_name: Karsten
  full_name: Kruse, Karsten
  last_name: Kruse
- first_name: John
  full_name: Small, John
  last_name: Small
- first_name: Christian
  full_name: Schmeiser, Christian
  last_name: Schmeiser
- first_name: Kinneret
  full_name: Keren, Kinneret
  last_name: Keren
- first_name: Robert
  full_name: Hauschild, Robert
  id: 4E01D6B4-F248-11E8-B48F-1D18A9856A87
  last_name: Hauschild
  orcid: 0000-0001-9843-3522
- first_name: Michael K
  full_name: Sixt, Michael K
  id: 41E9FBEA-F248-11E8-B48F-1D18A9856A87
  last_name: Sixt
  orcid: 0000-0002-6620-9179
citation:
  ama: Mueller J, Szep G, Nemethova M, et al. Load adaptation of lamellipodial actin
    networks. <i>Cell</i>. 2017;171(1):188-200. doi:<a href="https://doi.org/10.1016/j.cell.2017.07.051">10.1016/j.cell.2017.07.051</a>
  apa: Mueller, J., Szep, G., Nemethova, M., de Vries, I., Lieber, A., Winkler, C.,
    … Sixt, M. K. (2017). Load adaptation of lamellipodial actin networks. <i>Cell</i>.
    Cell Press. <a href="https://doi.org/10.1016/j.cell.2017.07.051">https://doi.org/10.1016/j.cell.2017.07.051</a>
  chicago: Mueller, Jan, Gregory Szep, Maria Nemethova, Ingrid de Vries, Arnon Lieber,
    Christoph Winkler, Karsten Kruse, et al. “Load Adaptation of Lamellipodial Actin
    Networks.” <i>Cell</i>. Cell Press, 2017. <a href="https://doi.org/10.1016/j.cell.2017.07.051">https://doi.org/10.1016/j.cell.2017.07.051</a>.
  ieee: J. Mueller <i>et al.</i>, “Load adaptation of lamellipodial actin networks,”
    <i>Cell</i>, vol. 171, no. 1. Cell Press, pp. 188–200, 2017.
  ista: Mueller J, Szep G, Nemethova M, de Vries I, Lieber A, Winkler C, Kruse K,
    Small J, Schmeiser C, Keren K, Hauschild R, Sixt MK. 2017. Load adaptation of
    lamellipodial actin networks. Cell. 171(1), 188–200.
  mla: Mueller, Jan, et al. “Load Adaptation of Lamellipodial Actin Networks.” <i>Cell</i>,
    vol. 171, no. 1, Cell Press, 2017, pp. 188–200, doi:<a href="https://doi.org/10.1016/j.cell.2017.07.051">10.1016/j.cell.2017.07.051</a>.
  short: J. Mueller, G. Szep, M. Nemethova, I. de Vries, A. Lieber, C. Winkler, K.
    Kruse, J. Small, C. Schmeiser, K. Keren, R. Hauschild, M.K. Sixt, Cell 171 (2017)
    188–200.
date_created: 2018-12-11T11:48:10Z
date_published: 2017-09-21T00:00:00Z
date_updated: 2023-09-28T11:33:49Z
day: '21'
department:
- _id: MiSi
- _id: Bio
doi: 10.1016/j.cell.2017.07.051
ec_funded: 1
external_id:
  isi:
  - '000411331800020'
intvolume: '       171'
isi: 1
issue: '1'
language:
- iso: eng
month: '09'
oa_version: None
page: 188 - 200
project:
- _id: 25AD6156-B435-11E9-9278-68D0E5697425
  grant_number: LS13-029
  name: Modeling of Polarization and Motility of Leukocytes in Three-Dimensional Environments
- _id: 25A603A2-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '281556'
  name: Cytoskeletal force generation and force transduction of migrating leukocytes
    (EU)
publication: Cell
publication_identifier:
  issn:
  - '00928674'
publication_status: published
publisher: Cell Press
publist_id: '6951'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Load adaptation of lamellipodial actin networks
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 171
year: '2017'
...
---
_id: '728'
abstract:
- lang: eng
  text: During animal development, cell-fate-specific changes in gene expression can
    modify the material properties of a tissue and drive tissue morphogenesis. While
    mechanistic insights into the genetic control of tissue-shaping events are beginning
    to emerge, how tissue morphogenesis and mechanics can reciprocally impact cell-fate
    specification remains relatively unexplored. Here we review recent findings reporting
    how multicellular morphogenetic events and their underlying mechanical forces
    can feed back into gene regulatory pathways to specify cell fate. We further discuss
    emerging techniques that allow for the direct measurement and manipulation of
    mechanical signals in vivo, offering unprecedented access to study mechanotransduction
    during development. Examination of the mechanical control of cell fate during
    tissue morphogenesis will pave the way to an integrated understanding of the design
    principles that underlie robust tissue patterning in embryonic development.
article_processing_charge: No
author:
- first_name: Chii
  full_name: Chan, Chii
  last_name: Chan
- first_name: Carl-Philipp J
  full_name: Heisenberg, Carl-Philipp J
  id: 39427864-F248-11E8-B48F-1D18A9856A87
  last_name: Heisenberg
  orcid: 0000-0002-0912-4566
- first_name: Takashi
  full_name: Hiiragi, Takashi
  last_name: Hiiragi
citation:
  ama: Chan C, Heisenberg C-PJ, Hiiragi T. Coordination of morphogenesis and cell
    fate specification in development. <i>Current Biology</i>. 2017;27(18):R1024-R1035.
    doi:<a href="https://doi.org/10.1016/j.cub.2017.07.010">10.1016/j.cub.2017.07.010</a>
  apa: Chan, C., Heisenberg, C.-P. J., &#38; Hiiragi, T. (2017). Coordination of morphogenesis
    and cell fate specification in development. <i>Current Biology</i>. Cell Press.
    <a href="https://doi.org/10.1016/j.cub.2017.07.010">https://doi.org/10.1016/j.cub.2017.07.010</a>
  chicago: Chan, Chii, Carl-Philipp J Heisenberg, and Takashi Hiiragi. “Coordination
    of Morphogenesis and Cell Fate Specification in Development.” <i>Current Biology</i>.
    Cell Press, 2017. <a href="https://doi.org/10.1016/j.cub.2017.07.010">https://doi.org/10.1016/j.cub.2017.07.010</a>.
  ieee: C. Chan, C.-P. J. Heisenberg, and T. Hiiragi, “Coordination of morphogenesis
    and cell fate specification in development,” <i>Current Biology</i>, vol. 27,
    no. 18. Cell Press, pp. R1024–R1035, 2017.
  ista: Chan C, Heisenberg C-PJ, Hiiragi T. 2017. Coordination of morphogenesis and
    cell fate specification in development. Current Biology. 27(18), R1024–R1035.
  mla: Chan, Chii, et al. “Coordination of Morphogenesis and Cell Fate Specification
    in Development.” <i>Current Biology</i>, vol. 27, no. 18, Cell Press, 2017, pp.
    R1024–35, doi:<a href="https://doi.org/10.1016/j.cub.2017.07.010">10.1016/j.cub.2017.07.010</a>.
  short: C. Chan, C.-P.J. Heisenberg, T. Hiiragi, Current Biology 27 (2017) R1024–R1035.
date_created: 2018-12-11T11:48:11Z
date_published: 2017-09-18T00:00:00Z
date_updated: 2023-09-28T11:33:21Z
day: '18'
department:
- _id: CaHe
doi: 10.1016/j.cub.2017.07.010
external_id:
  isi:
  - '000411581800019'
intvolume: '        27'
isi: 1
issue: '18'
language:
- iso: eng
month: '09'
oa_version: None
page: R1024 - R1035
publication: Current Biology
publication_identifier:
  issn:
  - '09609822'
publication_status: published
publisher: Cell Press
publist_id: '6949'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Coordination of morphogenesis and cell fate specification in development
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 27
year: '2017'
...
---
_id: '729'
abstract:
- lang: eng
  text: The cellular mechanisms allowing tissues to efficiently regenerate are not
    fully understood. In this issue of Developmental Cell, Cao et al. (2017)) discover
    that during zebrafish heart regeneration, epicardial cells at the leading edge
    of regenerating tissue undergo endoreplication, possibly due to increased tissue
    tension, thereby boosting their regenerative capacity.
article_processing_charge: No
author:
- first_name: Zoltan P
  full_name: Spiro, Zoltan P
  id: 426AD026-F248-11E8-B48F-1D18A9856A87
  last_name: Spiro
- first_name: Carl-Philipp J
  full_name: Heisenberg, Carl-Philipp J
  id: 39427864-F248-11E8-B48F-1D18A9856A87
  last_name: Heisenberg
  orcid: 0000-0002-0912-4566
citation:
  ama: Spiro ZP, Heisenberg C-PJ. Regeneration tensed up polyploidy takes the lead.
    <i>Developmental Cell</i>. 2017;42(6):559-560. doi:<a href="https://doi.org/10.1016/j.devcel.2017.09.008">10.1016/j.devcel.2017.09.008</a>
  apa: Spiro, Z. P., &#38; Heisenberg, C.-P. J. (2017). Regeneration tensed up polyploidy
    takes the lead. <i>Developmental Cell</i>. Cell Press. <a href="https://doi.org/10.1016/j.devcel.2017.09.008">https://doi.org/10.1016/j.devcel.2017.09.008</a>
  chicago: Spiro, Zoltan P, and Carl-Philipp J Heisenberg. “Regeneration Tensed up
    Polyploidy Takes the Lead.” <i>Developmental Cell</i>. Cell Press, 2017. <a href="https://doi.org/10.1016/j.devcel.2017.09.008">https://doi.org/10.1016/j.devcel.2017.09.008</a>.
  ieee: Z. P. Spiro and C.-P. J. Heisenberg, “Regeneration tensed up polyploidy takes
    the lead,” <i>Developmental Cell</i>, vol. 42, no. 6. Cell Press, pp. 559–560,
    2017.
  ista: Spiro ZP, Heisenberg C-PJ. 2017. Regeneration tensed up polyploidy takes the
    lead. Developmental Cell. 42(6), 559–560.
  mla: Spiro, Zoltan P., and Carl-Philipp J. Heisenberg. “Regeneration Tensed up Polyploidy
    Takes the Lead.” <i>Developmental Cell</i>, vol. 42, no. 6, Cell Press, 2017,
    pp. 559–60, doi:<a href="https://doi.org/10.1016/j.devcel.2017.09.008">10.1016/j.devcel.2017.09.008</a>.
  short: Z.P. Spiro, C.-P.J. Heisenberg, Developmental Cell 42 (2017) 559–560.
date_created: 2018-12-11T11:48:11Z
date_published: 2017-01-01T00:00:00Z
date_updated: 2023-09-28T11:32:49Z
day: '01'
department:
- _id: CaHe
doi: 10.1016/j.devcel.2017.09.008
external_id:
  isi:
  - '000411582800003'
intvolume: '        42'
isi: 1
issue: '6'
language:
- iso: eng
month: '01'
oa_version: None
page: 559 - 560
publication: Developmental Cell
publication_identifier:
  issn:
  - '15345807'
publication_status: published
publisher: Cell Press
publist_id: '6948'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Regeneration tensed up polyploidy takes the lead
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 42
year: '2017'
...
---
_id: '730'
abstract:
- lang: eng
  text: Neural responses are highly structured, with population activity restricted
    to a small subset of the astronomical range of possible activity patterns. Characterizing
    these statistical regularities is important for understanding circuit computation,
    but challenging in practice. Here we review recent approaches based on the maximum
    entropy principle used for quantifying collective behavior in neural activity.
    We highlight recent models that capture population-level statistics of neural
    data, yielding insights into the organization of the neural code and its biological
    substrate. Furthermore, the MaxEnt framework provides a general recipe for constructing
    surrogate ensembles that preserve aspects of the data, but are otherwise maximally
    unstructured. This idea can be used to generate a hierarchy of controls against
    which rigorous statistical tests are possible.
article_processing_charge: No
author:
- first_name: Cristina
  full_name: Savin, Cristina
  id: 3933349E-F248-11E8-B48F-1D18A9856A87
  last_name: Savin
- first_name: Gasper
  full_name: Tkacik, Gasper
  id: 3D494DCA-F248-11E8-B48F-1D18A9856A87
  last_name: Tkacik
  orcid: 0000-0002-6699-1455
citation:
  ama: Savin C, Tkačik G. Maximum entropy models as a tool for building precise neural
    controls. <i>Current Opinion in Neurobiology</i>. 2017;46:120-126. doi:<a href="https://doi.org/10.1016/j.conb.2017.08.001">10.1016/j.conb.2017.08.001</a>
  apa: Savin, C., &#38; Tkačik, G. (2017). Maximum entropy models as a tool for building
    precise neural controls. <i>Current Opinion in Neurobiology</i>. Elsevier. <a
    href="https://doi.org/10.1016/j.conb.2017.08.001">https://doi.org/10.1016/j.conb.2017.08.001</a>
  chicago: Savin, Cristina, and Gašper Tkačik. “Maximum Entropy Models as a Tool for
    Building Precise Neural Controls.” <i>Current Opinion in Neurobiology</i>. Elsevier,
    2017. <a href="https://doi.org/10.1016/j.conb.2017.08.001">https://doi.org/10.1016/j.conb.2017.08.001</a>.
  ieee: C. Savin and G. Tkačik, “Maximum entropy models as a tool for building precise
    neural controls,” <i>Current Opinion in Neurobiology</i>, vol. 46. Elsevier, pp.
    120–126, 2017.
  ista: Savin C, Tkačik G. 2017. Maximum entropy models as a tool for building precise
    neural controls. Current Opinion in Neurobiology. 46, 120–126.
  mla: Savin, Cristina, and Gašper Tkačik. “Maximum Entropy Models as a Tool for Building
    Precise Neural Controls.” <i>Current Opinion in Neurobiology</i>, vol. 46, Elsevier,
    2017, pp. 120–26, doi:<a href="https://doi.org/10.1016/j.conb.2017.08.001">10.1016/j.conb.2017.08.001</a>.
  short: C. Savin, G. Tkačik, Current Opinion in Neurobiology 46 (2017) 120–126.
date_created: 2018-12-11T11:48:11Z
date_published: 2017-10-01T00:00:00Z
date_updated: 2023-09-28T11:32:22Z
day: '01'
department:
- _id: GaTk
doi: 10.1016/j.conb.2017.08.001
ec_funded: 1
external_id:
  isi:
  - '000416196400016'
intvolume: '        46'
isi: 1
language:
- iso: eng
month: '10'
oa_version: None
page: 120 - 126
project:
- _id: 25681D80-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '291734'
  name: International IST Postdoc Fellowship Programme
publication: Current Opinion in Neurobiology
publication_identifier:
  issn:
  - '09594388'
publication_status: published
publisher: Elsevier
publist_id: '6943'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Maximum entropy models as a tool for building precise neural controls
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 46
year: '2017'
...
---
_id: '731'
abstract:
- lang: eng
  text: Genetic variations in the oxytocin receptor gene affect patients with ASD
    and ADHD differently.
article_number: eaap8168
author:
- first_name: Gaia
  full_name: Novarino, Gaia
  id: 3E57A680-F248-11E8-B48F-1D18A9856A87
  last_name: Novarino
  orcid: 0000-0002-7673-7178
citation:
  ama: Novarino G. The science of love in ASD and ADHD. <i>Science Translational Medicine</i>.
    2017;9(411). doi:<a href="https://doi.org/10.1126/scitranslmed.aap8168">10.1126/scitranslmed.aap8168</a>
  apa: Novarino, G. (2017). The science of love in ASD and ADHD. <i>Science Translational
    Medicine</i>. American Association for the Advancement of Science. <a href="https://doi.org/10.1126/scitranslmed.aap8168">https://doi.org/10.1126/scitranslmed.aap8168</a>
  chicago: Novarino, Gaia. “The Science of Love in ASD and ADHD.” <i>Science Translational
    Medicine</i>. American Association for the Advancement of Science, 2017. <a href="https://doi.org/10.1126/scitranslmed.aap8168">https://doi.org/10.1126/scitranslmed.aap8168</a>.
  ieee: G. Novarino, “The science of love in ASD and ADHD,” <i>Science Translational
    Medicine</i>, vol. 9, no. 411. American Association for the Advancement of Science,
    2017.
  ista: Novarino G. 2017. The science of love in ASD and ADHD. Science Translational
    Medicine. 9(411), eaap8168.
  mla: Novarino, Gaia. “The Science of Love in ASD and ADHD.” <i>Science Translational
    Medicine</i>, vol. 9, no. 411, eaap8168, American Association for the Advancement
    of Science, 2017, doi:<a href="https://doi.org/10.1126/scitranslmed.aap8168">10.1126/scitranslmed.aap8168</a>.
  short: G. Novarino, Science Translational Medicine 9 (2017).
date_created: 2018-12-11T11:48:12Z
date_published: 2017-10-11T00:00:00Z
date_updated: 2021-01-12T08:12:57Z
day: '11'
department:
- _id: GaNo
doi: 10.1126/scitranslmed.aap8168
intvolume: '         9'
issue: '411'
language:
- iso: eng
month: '10'
oa_version: None
publication: Science Translational Medicine
publication_identifier:
  issn:
  - '19466234'
publication_status: published
publisher: American Association for the Advancement of Science
publist_id: '6938'
quality_controlled: '1'
scopus_import: 1
status: public
title: The science of love in ASD and ADHD
type: journal_article
user_id: 3E5EF7F0-F248-11E8-B48F-1D18A9856A87
volume: 9
year: '2017'
...
---
_id: '732'
abstract:
- lang: eng
  text: 'Background: Social insects form densely crowded societies in environments
    with high pathogen loads, but have evolved collective defences that mitigate the
    impact of disease. However, colony-founding queens lack this protection and suffer
    high rates of mortality. The impact of pathogens may be exacerbated in species
    where queens found colonies together, as healthy individuals may contract pathogens
    from infectious co-founders. Therefore, we tested whether ant queens avoid founding
    colonies with pathogen-exposed conspecifics and how they might limit disease transmission
    from infectious individuals. Results: Using Lasius Niger queens and a naturally
    infecting fungal pathogen Metarhizium brunneum, we observed that queens were equally
    likely to found colonies with another pathogen-exposed or sham-treated queen.
    However, when one queen died, the surviving individual performed biting, burial
    and removal of the corpse. These undertaking behaviours were performed prophylactically,
    i.e. targeted equally towards non-infected and infected corpses, as well as carried
    out before infected corpses became infectious. Biting and burial reduced the risk
    of the queens contracting and dying from disease from an infectious corpse of
    a dead co-foundress. Conclusions: We show that co-founding ant queens express
    undertaking behaviours that, in mature colonies, are performed exclusively by
    workers. Such infection avoidance behaviours act before the queens can contract
    the disease and will therefore improve the overall chance of colony founding success
    in ant queens.'
article_number: '219'
article_processing_charge: Yes
article_type: original
author:
- first_name: Christopher
  full_name: Pull, Christopher
  id: 3C7F4840-F248-11E8-B48F-1D18A9856A87
  last_name: Pull
  orcid: 0000-0003-1122-3982
- first_name: Sylvia
  full_name: Cremer, Sylvia
  id: 2F64EC8C-F248-11E8-B48F-1D18A9856A87
  last_name: Cremer
  orcid: 0000-0002-2193-3868
citation:
  ama: Pull C, Cremer S. Co-founding ant queens prevent disease by performing prophylactic
    undertaking behaviour. <i>BMC Evolutionary Biology</i>. 2017;17(1). doi:<a href="https://doi.org/10.1186/s12862-017-1062-4">10.1186/s12862-017-1062-4</a>
  apa: Pull, C., &#38; Cremer, S. (2017). Co-founding ant queens prevent disease by
    performing prophylactic undertaking behaviour. <i>BMC Evolutionary Biology</i>.
    BioMed Central. <a href="https://doi.org/10.1186/s12862-017-1062-4">https://doi.org/10.1186/s12862-017-1062-4</a>
  chicago: Pull, Christopher, and Sylvia Cremer. “Co-Founding Ant Queens Prevent Disease
    by Performing Prophylactic Undertaking Behaviour.” <i>BMC Evolutionary Biology</i>.
    BioMed Central, 2017. <a href="https://doi.org/10.1186/s12862-017-1062-4">https://doi.org/10.1186/s12862-017-1062-4</a>.
  ieee: C. Pull and S. Cremer, “Co-founding ant queens prevent disease by performing
    prophylactic undertaking behaviour,” <i>BMC Evolutionary Biology</i>, vol. 17,
    no. 1. BioMed Central, 2017.
  ista: Pull C, Cremer S. 2017. Co-founding ant queens prevent disease by performing
    prophylactic undertaking behaviour. BMC Evolutionary Biology. 17(1), 219.
  mla: Pull, Christopher, and Sylvia Cremer. “Co-Founding Ant Queens Prevent Disease
    by Performing Prophylactic Undertaking Behaviour.” <i>BMC Evolutionary Biology</i>,
    vol. 17, no. 1, 219, BioMed Central, 2017, doi:<a href="https://doi.org/10.1186/s12862-017-1062-4">10.1186/s12862-017-1062-4</a>.
  short: C. Pull, S. Cremer, BMC Evolutionary Biology 17 (2017).
date_created: 2018-12-11T11:48:12Z
date_published: 2017-10-13T00:00:00Z
date_updated: 2023-09-28T11:31:32Z
day: '13'
ddc:
- '576'
- '592'
department:
- _id: SyCr
doi: 10.1186/s12862-017-1062-4
ec_funded: 1
external_id:
  isi:
  - '000412816800001'
file:
- access_level: open_access
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  creator: system
  date_created: 2018-12-12T10:17:18Z
  date_updated: 2020-07-14T12:47:55Z
  file_id: '5271'
  file_name: IST-2017-882-v1+1_12862_2017_Article_1062.pdf
  file_size: 949857
  relation: main_file
file_date_updated: 2020-07-14T12:47:55Z
has_accepted_license: '1'
intvolume: '        17'
isi: 1
issue: '1'
language:
- iso: eng
month: '10'
oa: 1
oa_version: Published Version
project:
- _id: 25DC711C-B435-11E9-9278-68D0E5697425
  call_identifier: FP7
  grant_number: '243071'
  name: 'Social Vaccination in Ant Colonies: from Individual Mechanisms to Society
    Effects'
publication: BMC Evolutionary Biology
publication_identifier:
  issn:
  - '14712148'
publication_status: published
publisher: BioMed Central
publist_id: '6937'
pubrep_id: '882'
quality_controlled: '1'
related_material:
  record:
  - id: '819'
    relation: dissertation_contains
    status: public
scopus_import: '1'
status: public
title: Co-founding ant queens prevent disease by performing prophylactic undertaking
  behaviour
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 17
year: '2017'
...
