---
_id: '5860'
abstract:
- lang: eng
  text: 'A major problem for evolutionary theory is understanding the so-called open-ended
    nature of evolutionary change, from its definition to its origins. Open-ended
    evolution (OEE) refers to the unbounded increase in complexity that seems to characterize
    evolution on multiple scales. This property seems to be a characteristic feature
    of biological and technological evolution and is strongly tied to the generative
    potential associated with combinatorics, which allows the system to grow and expand
    their available state spaces. Interestingly, many complex systems presumably displaying
    OEE, from language to proteins, share a common statistical property: the presence
    of Zipf''s Law. Given an inventory of basic items (such as words or protein domains)
    required to build more complex structures (sentences or proteins) Zipf''s Law
    tells us that most of these elements are rare whereas a few of them are extremely
    common. Using algorithmic information theory, in this paper we provide a fundamental
    definition for open-endedness, which can be understood as postulates. Its statistical
    counterpart, based on standard Shannon information theory, has the structure of
    a variational problem which is shown to lead to Zipf''s Law as the expected consequence
    of an evolutionary process displaying OEE. We further explore the problem of information
    conservation through an OEE process and we conclude that statistical information
    (standard Shannon information) is not conserved, resulting in the paradoxical
    situation in which the increase of information content has the effect of erasing
    itself. We prove that this paradox is solved if we consider non-statistical forms
    of information. This last result implies that standard information theory may
    not be a suitable theoretical framework to explore the persistence and increase
    of the information content in OEE systems.'
article_number: '20180395'
article_processing_charge: No
arxiv: 1
author:
- first_name: Bernat
  full_name: Corominas-Murtra, Bernat
  id: 43BE2298-F248-11E8-B48F-1D18A9856A87
  last_name: Corominas-Murtra
  orcid: 0000-0001-9806-5643
- first_name: Luís F.
  full_name: Seoane, Luís F.
  last_name: Seoane
- first_name: Ricard
  full_name: Solé, Ricard
  last_name: Solé
citation:
  ama: Corominas-Murtra B, Seoane LF, Solé R. Zipf’s Law, unbounded complexity and
    open-ended evolution. <i>Journal of the Royal Society Interface</i>. 2018;15(149).
    doi:<a href="https://doi.org/10.1098/rsif.2018.0395">10.1098/rsif.2018.0395</a>
  apa: Corominas-Murtra, B., Seoane, L. F., &#38; Solé, R. (2018). Zipf’s Law, unbounded
    complexity and open-ended evolution. <i>Journal of the Royal Society Interface</i>.
    Royal Society Publishing. <a href="https://doi.org/10.1098/rsif.2018.0395">https://doi.org/10.1098/rsif.2018.0395</a>
  chicago: Corominas-Murtra, Bernat, Luís F. Seoane, and Ricard Solé. “Zipf’s Law,
    Unbounded Complexity and Open-Ended Evolution.” <i>Journal of the Royal Society
    Interface</i>. Royal Society Publishing, 2018. <a href="https://doi.org/10.1098/rsif.2018.0395">https://doi.org/10.1098/rsif.2018.0395</a>.
  ieee: B. Corominas-Murtra, L. F. Seoane, and R. Solé, “Zipf’s Law, unbounded complexity
    and open-ended evolution,” <i>Journal of the Royal Society Interface</i>, vol.
    15, no. 149. Royal Society Publishing, 2018.
  ista: Corominas-Murtra B, Seoane LF, Solé R. 2018. Zipf’s Law, unbounded complexity
    and open-ended evolution. Journal of the Royal Society Interface. 15(149), 20180395.
  mla: Corominas-Murtra, Bernat, et al. “Zipf’s Law, Unbounded Complexity and Open-Ended
    Evolution.” <i>Journal of the Royal Society Interface</i>, vol. 15, no. 149, 20180395,
    Royal Society Publishing, 2018, doi:<a href="https://doi.org/10.1098/rsif.2018.0395">10.1098/rsif.2018.0395</a>.
  short: B. Corominas-Murtra, L.F. Seoane, R. Solé, Journal of the Royal Society Interface
    15 (2018).
date_created: 2019-01-20T22:59:19Z
date_published: 2018-12-12T00:00:00Z
date_updated: 2023-09-19T10:40:38Z
day: '12'
department:
- _id: EdHa
doi: 10.1098/rsif.2018.0395
external_id:
  arxiv:
  - '1612.01605'
  isi:
  - '000456783800002'
intvolume: '        15'
isi: 1
issue: '149'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1612.01605
month: '12'
oa: 1
oa_version: Preprint
publication: Journal of the Royal Society Interface
publication_identifier:
  issn:
  - '17425689'
publication_status: published
publisher: Royal Society Publishing
quality_controlled: '1'
scopus_import: '1'
status: public
title: Zipf's Law, unbounded complexity and open-ended evolution
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 15
year: '2018'
...
---
_id: '132'
abstract:
- lang: eng
  text: Pancreas development involves a coordinated process in which an early phase
    of cell segregation is followed by a longer phase of lineage restriction, expansion,
    and tissue remodeling. By combining clonal tracing and whole-mount reconstruction
    with proliferation kinetics and single-cell transcriptional profiling, we define
    the functional basis of pancreas morphogenesis. We show that the large-scale organization
    of mouse pancreas can be traced to the activity of self-renewing precursors positioned
    at the termini of growing ducts, which act collectively to drive serial rounds
    of stochastic ductal bifurcation balanced by termination. During this phase of
    branching morphogenesis, multipotent precursors become progressively fate-restricted,
    giving rise to self-renewing acinar-committed precursors that are conveyed with
    growing ducts, as well as ductal progenitors that expand the trailing ducts and
    give rise to delaminating endocrine cells. These findings define quantitatively
    how the functional behavior and lineage progression of precursor pools determine
    the large-scale patterning of pancreatic sub-compartments.
acknowledgement: E.H. is funded by a Junior Research Fellowship from Trinity College,
  Cam-bridge, a Sir Henry Wellcome Fellowship from the Wellcome Trust, and theBettencourt-Schueller
  Young Researcher Prize for support.
article_processing_charge: No
article_type: original
author:
- first_name: Magdalena
  full_name: Sznurkowska, Magdalena
  last_name: Sznurkowska
- first_name: Edouard B
  full_name: Hannezo, Edouard B
  id: 3A9DB764-F248-11E8-B48F-1D18A9856A87
  last_name: Hannezo
  orcid: 0000-0001-6005-1561
- first_name: Roberta
  full_name: Azzarelli, Roberta
  last_name: Azzarelli
- first_name: Steffen
  full_name: Rulands, Steffen
  last_name: Rulands
- first_name: Sonia
  full_name: Nestorowa, Sonia
  last_name: Nestorowa
- first_name: Christopher
  full_name: Hindley, Christopher
  last_name: Hindley
- first_name: Jennifer
  full_name: Nichols, Jennifer
  last_name: Nichols
- first_name: Berthold
  full_name: Göttgens, Berthold
  last_name: Göttgens
- first_name: Meritxell
  full_name: Huch, Meritxell
  last_name: Huch
- first_name: Anna
  full_name: Philpott, Anna
  last_name: Philpott
- first_name: Benjamin
  full_name: Simons, Benjamin
  last_name: Simons
citation:
  ama: Sznurkowska M, Hannezo EB, Azzarelli R, et al. Defining lineage potential and
    fate behavior of precursors during pancreas development. <i>Developmental Cell</i>.
    2018;46(3):360-375. doi:<a href="https://doi.org/10.1016/j.devcel.2018.06.028">10.1016/j.devcel.2018.06.028</a>
  apa: Sznurkowska, M., Hannezo, E. B., Azzarelli, R., Rulands, S., Nestorowa, S.,
    Hindley, C., … Simons, B. (2018). Defining lineage potential and fate behavior
    of precursors during pancreas development. <i>Developmental Cell</i>. Cell Press.
    <a href="https://doi.org/10.1016/j.devcel.2018.06.028">https://doi.org/10.1016/j.devcel.2018.06.028</a>
  chicago: Sznurkowska, Magdalena, Edouard B Hannezo, Roberta Azzarelli, Steffen Rulands,
    Sonia Nestorowa, Christopher Hindley, Jennifer Nichols, et al. “Defining Lineage
    Potential and Fate Behavior of Precursors during Pancreas Development.” <i>Developmental
    Cell</i>. Cell Press, 2018. <a href="https://doi.org/10.1016/j.devcel.2018.06.028">https://doi.org/10.1016/j.devcel.2018.06.028</a>.
  ieee: M. Sznurkowska <i>et al.</i>, “Defining lineage potential and fate behavior
    of precursors during pancreas development,” <i>Developmental Cell</i>, vol. 46,
    no. 3. Cell Press, pp. 360–375, 2018.
  ista: Sznurkowska M, Hannezo EB, Azzarelli R, Rulands S, Nestorowa S, Hindley C,
    Nichols J, Göttgens B, Huch M, Philpott A, Simons B. 2018. Defining lineage potential
    and fate behavior of precursors during pancreas development. Developmental Cell.
    46(3), 360–375.
  mla: Sznurkowska, Magdalena, et al. “Defining Lineage Potential and Fate Behavior
    of Precursors during Pancreas Development.” <i>Developmental Cell</i>, vol. 46,
    no. 3, Cell Press, 2018, pp. 360–75, doi:<a href="https://doi.org/10.1016/j.devcel.2018.06.028">10.1016/j.devcel.2018.06.028</a>.
  short: M. Sznurkowska, E.B. Hannezo, R. Azzarelli, S. Rulands, S. Nestorowa, C.
    Hindley, J. Nichols, B. Göttgens, M. Huch, A. Philpott, B. Simons, Developmental
    Cell 46 (2018) 360–375.
date_created: 2018-12-11T11:44:48Z
date_published: 2018-08-06T00:00:00Z
date_updated: 2023-09-11T12:52:41Z
day: '06'
ddc:
- '570'
department:
- _id: EdHa
doi: 10.1016/j.devcel.2018.06.028
external_id:
  isi:
  - '000441327300012'
file:
- access_level: open_access
  checksum: 78d2062b9e3c3b90fe71545aeb6d2f65
  content_type: application/pdf
  creator: dernst
  date_created: 2018-12-17T10:49:49Z
  date_updated: 2020-07-14T12:44:43Z
  file_id: '5694'
  file_name: 2018_DevelopmentalCell_Sznurkowska.pdf
  file_size: 8948384
  relation: main_file
file_date_updated: 2020-07-14T12:44:43Z
has_accepted_license: '1'
intvolume: '        46'
isi: 1
issue: '3'
language:
- iso: eng
month: '08'
oa: 1
oa_version: Published Version
page: 360 - 375
publication: Developmental Cell
publication_status: published
publisher: Cell Press
publist_id: '7791'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Defining lineage potential and fate behavior of precursors during pancreas
  development
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 46
year: '2018'
...
---
_id: '401'
abstract:
- lang: eng
  text: The actomyosin cytoskeleton, a key stress-producing unit in epithelial cells,
    oscillates spontaneously in a wide variety of systems. Although much of the signal
    cascade regulating myosin activity has been characterized, the origin of such
    oscillatory behavior is still unclear. Here, we show that basal myosin II oscillation
    in Drosophila ovarian epithelium is not controlled by actomyosin cortical tension,
    but instead relies on a biochemical oscillator involving ROCK and myosin phosphatase.
    Key to this oscillation is a diffusive ROCK flow, linking junctional Rho1 to medial
    actomyosin cortex, and dynamically maintained by a self-activation loop reliant
    on ROCK kinase activity. In response to the resulting myosin II recruitment, myosin
    phosphatase is locally enriched and shuts off ROCK and myosin II signals. Coupling
    Drosophila genetics, live imaging, modeling, and optogenetics, we uncover an intrinsic
    biochemical oscillator at the core of myosin II regulatory network, shedding light
    on the spatio-temporal dynamics of force generation.
article_number: '1210'
article_processing_charge: No
author:
- first_name: Xiang
  full_name: Qin, Xiang
  last_name: Qin
- first_name: Edouard B
  full_name: Hannezo, Edouard B
  id: 3A9DB764-F248-11E8-B48F-1D18A9856A87
  last_name: Hannezo
  orcid: 0000-0001-6005-1561
- first_name: Thomas
  full_name: Mangeat, Thomas
  last_name: Mangeat
- first_name: Chang
  full_name: Liu, Chang
  last_name: Liu
- first_name: Pralay
  full_name: Majumder, Pralay
  last_name: Majumder
- first_name: Jjiaying
  full_name: Liu, Jjiaying
  last_name: Liu
- first_name: Valerie
  full_name: Choesmel Cadamuro, Valerie
  last_name: Choesmel Cadamuro
- first_name: Jocelyn
  full_name: Mcdonald, Jocelyn
  last_name: Mcdonald
- first_name: Yinyao
  full_name: Liu, Yinyao
  last_name: Liu
- first_name: Bin
  full_name: Yi, Bin
  last_name: Yi
- first_name: Xiaobo
  full_name: Wang, Xiaobo
  last_name: Wang
citation:
  ama: Qin X, Hannezo EB, Mangeat T, et al. A biochemical network controlling basal
    myosin oscillation. <i>Nature Communications</i>. 2018;9(1). doi:<a href="https://doi.org/10.1038/s41467-018-03574-5">10.1038/s41467-018-03574-5</a>
  apa: Qin, X., Hannezo, E. B., Mangeat, T., Liu, C., Majumder, P., Liu, J., … Wang,
    X. (2018). A biochemical network controlling basal myosin oscillation. <i>Nature
    Communications</i>. Nature Publishing Group. <a href="https://doi.org/10.1038/s41467-018-03574-5">https://doi.org/10.1038/s41467-018-03574-5</a>
  chicago: Qin, Xiang, Edouard B Hannezo, Thomas Mangeat, Chang Liu, Pralay Majumder,
    Jjiaying Liu, Valerie Choesmel Cadamuro, et al. “A Biochemical Network Controlling
    Basal Myosin Oscillation.” <i>Nature Communications</i>. Nature Publishing Group,
    2018. <a href="https://doi.org/10.1038/s41467-018-03574-5">https://doi.org/10.1038/s41467-018-03574-5</a>.
  ieee: X. Qin <i>et al.</i>, “A biochemical network controlling basal myosin oscillation,”
    <i>Nature Communications</i>, vol. 9, no. 1. Nature Publishing Group, 2018.
  ista: Qin X, Hannezo EB, Mangeat T, Liu C, Majumder P, Liu J, Choesmel Cadamuro
    V, Mcdonald J, Liu Y, Yi B, Wang X. 2018. A biochemical network controlling basal
    myosin oscillation. Nature Communications. 9(1), 1210.
  mla: Qin, Xiang, et al. “A Biochemical Network Controlling Basal Myosin Oscillation.”
    <i>Nature Communications</i>, vol. 9, no. 1, 1210, Nature Publishing Group, 2018,
    doi:<a href="https://doi.org/10.1038/s41467-018-03574-5">10.1038/s41467-018-03574-5</a>.
  short: X. Qin, E.B. Hannezo, T. Mangeat, C. Liu, P. Majumder, J. Liu, V. Choesmel
    Cadamuro, J. Mcdonald, Y. Liu, B. Yi, X. Wang, Nature Communications 9 (2018).
date_created: 2018-12-11T11:46:16Z
date_published: 2018-03-23T00:00:00Z
date_updated: 2023-09-08T11:41:45Z
day: '23'
ddc:
- '539'
- '570'
department:
- _id: EdHa
doi: 10.1038/s41467-018-03574-5
external_id:
  isi:
  - '000428165400009'
file:
- access_level: open_access
  checksum: 87a427bc2e8724be3dd22a4efdd21a33
  content_type: application/pdf
  creator: system
  date_created: 2018-12-12T10:11:45Z
  date_updated: 2020-07-14T12:46:22Z
  file_id: '4902'
  file_name: IST-2018-996-v1+1_2018_Hannezo_A-biochemical.pdf
  file_size: 3780491
  relation: main_file
file_date_updated: 2020-07-14T12:46:22Z
has_accepted_license: '1'
intvolume: '         9'
isi: 1
issue: '1'
language:
- iso: eng
month: '03'
oa: 1
oa_version: Published Version
publication: Nature Communications
publication_status: published
publisher: Nature Publishing Group
publist_id: '7427'
pubrep_id: '996'
quality_controlled: '1'
scopus_import: '1'
status: public
title: A biochemical network controlling basal myosin oscillation
tmp:
  image: /images/cc_by.png
  legal_code_url: https://creativecommons.org/licenses/by/4.0/legalcode
  name: Creative Commons Attribution 4.0 International Public License (CC-BY 4.0)
  short: CC BY (4.0)
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 9
year: '2018'
...
---
_id: '421'
abstract:
- lang: eng
  text: Cell shape is determined by a balance of intrinsic properties of the cell
    as well as its mechanochemical environment. Inhomogeneous shape changes underlie
    many morphogenetic events and involve spatial gradients in active cellular forces
    induced by complex chemical signaling. Here, we introduce a mechanochemical model
    based on the notion that cell shape changes may be induced by external diffusible
    biomolecules that influence cellular contractility (or equivalently, adhesions)
    in a concentration-dependent manner—and whose spatial profile in turn is affected
    by cell shape. We map out theoretically the possible interplay between chemical
    concentration and cellular structure. Besides providing a direct route to spatial
    gradients in cell shape profiles in tissues, we show that the dependence on cell
    shape helps create robust mechanochemical gradients.
article_processing_charge: No
arxiv: 1
author:
- first_name: Kinjal
  full_name: Dasbiswas, Kinjal
  last_name: Dasbiswas
- first_name: Claude-Edouard B
  full_name: Hannezo, Claude-Edouard B
  id: 3A9DB764-F248-11E8-B48F-1D18A9856A87
  last_name: Hannezo
  orcid: 0000-0001-6005-1561
- first_name: Nir
  full_name: Gov, Nir
  last_name: Gov
citation:
  ama: Dasbiswas K, Hannezo EB, Gov N. Theory of eppithelial cell shape transitions
    induced by mechanoactive chemical gradients. <i>Biophysical Journal</i>. 2018;114(4):968-977.
    doi:<a href="https://doi.org/10.1016/j.bpj.2017.12.022">10.1016/j.bpj.2017.12.022</a>
  apa: Dasbiswas, K., Hannezo, E. B., &#38; Gov, N. (2018). Theory of eppithelial
    cell shape transitions induced by mechanoactive chemical gradients. <i>Biophysical
    Journal</i>. Biophysical Society. <a href="https://doi.org/10.1016/j.bpj.2017.12.022">https://doi.org/10.1016/j.bpj.2017.12.022</a>
  chicago: Dasbiswas, Kinjal, Edouard B Hannezo, and Nir Gov. “Theory of Eppithelial
    Cell Shape Transitions Induced by Mechanoactive Chemical Gradients.” <i>Biophysical
    Journal</i>. Biophysical Society, 2018. <a href="https://doi.org/10.1016/j.bpj.2017.12.022">https://doi.org/10.1016/j.bpj.2017.12.022</a>.
  ieee: K. Dasbiswas, E. B. Hannezo, and N. Gov, “Theory of eppithelial cell shape
    transitions induced by mechanoactive chemical gradients,” <i>Biophysical Journal</i>,
    vol. 114, no. 4. Biophysical Society, pp. 968–977, 2018.
  ista: Dasbiswas K, Hannezo EB, Gov N. 2018. Theory of eppithelial cell shape transitions
    induced by mechanoactive chemical gradients. Biophysical Journal. 114(4), 968–977.
  mla: Dasbiswas, Kinjal, et al. “Theory of Eppithelial Cell Shape Transitions Induced
    by Mechanoactive Chemical Gradients.” <i>Biophysical Journal</i>, vol. 114, no.
    4, Biophysical Society, 2018, pp. 968–77, doi:<a href="https://doi.org/10.1016/j.bpj.2017.12.022">10.1016/j.bpj.2017.12.022</a>.
  short: K. Dasbiswas, E.B. Hannezo, N. Gov, Biophysical Journal 114 (2018) 968–977.
date_created: 2018-12-11T11:46:23Z
date_published: 2018-02-27T00:00:00Z
date_updated: 2023-09-19T10:13:55Z
day: '27'
department:
- _id: EdHa
doi: 10.1016/j.bpj.2017.12.022
external_id:
  arxiv:
  - '1709.01486'
  isi:
  - '000428016700021'
intvolume: '       114'
isi: 1
issue: '4'
language:
- iso: eng
main_file_link:
- open_access: '1'
  url: https://arxiv.org/abs/1709.01486
month: '02'
oa: 1
oa_version: Submitted Version
page: 968 - 977
publication: Biophysical Journal
publication_status: published
publisher: Biophysical Society
publist_id: '7403'
quality_controlled: '1'
scopus_import: '1'
status: public
title: Theory of eppithelial cell shape transitions induced by mechanoactive chemical
  gradients
type: journal_article
user_id: c635000d-4b10-11ee-a964-aac5a93f6ac1
volume: 114
year: '2018'
...
---
_id: '726'
abstract:
- lang: eng
  text: The morphogenesis of branched organs remains a subject of abiding interest.
    Although much is known about the underlying signaling pathways, it remains unclear
    how macroscopic features of branched organs, including their size, network topology,
    and spatial patterning, are encoded. Here, we show that, in mouse mammary gland,
    kidney, and human prostate, these features can be explained quantitatively within
    a single unifying framework of branching and annihilating random walks. Based
    on quantitative analyses of large-scale organ reconstructions and proliferation
    kinetics measurements, we propose that morphogenesis follows from the proliferative
    activity of equipotent tips that stochastically branch and randomly explore their
    environment but compete neutrally for space, becoming proliferatively inactive
    when in proximity with neighboring ducts. These results show that complex branched
    epithelial structures develop as a self-organized process, reliant upon a strikingly
    simple but generic rule, without recourse to a rigid and deterministic sequence
    of genetically programmed events.
article_processing_charge: No
author:
- first_name: Edouard B
  full_name: Hannezo, Edouard B
  id: 3A9DB764-F248-11E8-B48F-1D18A9856A87
  last_name: Hannezo
  orcid: 0000-0001-6005-1561
- first_name: Colinda
  full_name: Scheele, Colinda
  last_name: Scheele
- first_name: Mohammad
  full_name: Moad, Mohammad
  last_name: Moad
- first_name: Nicholas
  full_name: Drogo, Nicholas
  last_name: Drogo
- first_name: Rakesh
  full_name: Heer, Rakesh
  last_name: Heer
- first_name: Rosemary
  full_name: Sampogna, Rosemary
  last_name: Sampogna
- first_name: Jacco
  full_name: Van Rheenen, Jacco
  last_name: Van Rheenen
- first_name: Benjamin
  full_name: Simons, Benjamin
  last_name: Simons
citation:
  ama: Hannezo EB, Scheele C, Moad M, et al. A unifying theory of branching morphogenesis.
    <i>Cell</i>. 2017;171(1):242-255. doi:<a href="https://doi.org/10.1016/j.cell.2017.08.026">10.1016/j.cell.2017.08.026</a>
  apa: Hannezo, E. B., Scheele, C., Moad, M., Drogo, N., Heer, R., Sampogna, R., …
    Simons, B. (2017). A unifying theory of branching morphogenesis. <i>Cell</i>.
    Cell Press. <a href="https://doi.org/10.1016/j.cell.2017.08.026">https://doi.org/10.1016/j.cell.2017.08.026</a>
  chicago: Hannezo, Edouard B, Colinda Scheele, Mohammad Moad, Nicholas Drogo, Rakesh
    Heer, Rosemary Sampogna, Jacco Van Rheenen, and Benjamin Simons. “A Unifying Theory
    of Branching Morphogenesis.” <i>Cell</i>. Cell Press, 2017. <a href="https://doi.org/10.1016/j.cell.2017.08.026">https://doi.org/10.1016/j.cell.2017.08.026</a>.
  ieee: E. B. Hannezo <i>et al.</i>, “A unifying theory of branching morphogenesis,”
    <i>Cell</i>, vol. 171, no. 1. Cell Press, pp. 242–255, 2017.
  ista: Hannezo EB, Scheele C, Moad M, Drogo N, Heer R, Sampogna R, Van Rheenen J,
    Simons B. 2017. A unifying theory of branching morphogenesis. Cell. 171(1), 242–255.
  mla: Hannezo, Edouard B., et al. “A Unifying Theory of Branching Morphogenesis.”
    <i>Cell</i>, vol. 171, no. 1, Cell Press, 2017, pp. 242–55, doi:<a href="https://doi.org/10.1016/j.cell.2017.08.026">10.1016/j.cell.2017.08.026</a>.
  short: E.B. Hannezo, C. Scheele, M. Moad, N. Drogo, R. Heer, R. Sampogna, J. Van
    Rheenen, B. Simons, Cell 171 (2017) 242–255.
date_created: 2018-12-11T11:48:10Z
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title: A unifying theory of branching morphogenesis
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